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■' A TEXT-BOOK
DISEASES OF THE EYE
HENRY D. NOYES, A.M., M.D.
Professor of Ophthalmology and Otology in Bellevue Hospital Medical College; Executive Surgeon to the
New York Eye and Ear Infirmary; recently President of the American Ophthalmological
Society; Member of the New York Ophthalmological Society; recently Vice-
President of the New York Academy of Medicine; Permanent Member
of the New York State Medical Society; Member of the
American Medical Association, etc., etc.
jgjecotxd and Revised ISdition
ILLUSTRATED BY 5 CHROMO-LITHOGRAPHIC PLATES, IO PLATES IN
BLACK AND COLORS, AND 269 WOOD-ENGRAVINGS
NEW YORK
WILLIAM WOOD & COMPANY
1894
NWt
Copyrighted, 1894
WILLIAM WOOD & COMPANY
PRESS OF
THE PUBLISHERS' PRINTING COMPANY
132-138 W. FOURTEENTH 8T.
NEW YORK
EBRATA.
Page 651, for Plate III. read Plate A.
Page 656, Fig. 245, for Bouillaud-Beoca
read Bouillaud-Broca.
PREFACE TO SECOND EDITION.
In revising this treatise numerous additions have been made
to render it more complete. For example, those parts of cerebral
anatomy and pathology which have a bearing upon the eye have
been discussed at unusual length for such a work; this is true
both for the motor nerves and for the optic nerves. Some chap-
ters have been considerably modified and some have been rewrit-
ten; for example, those on granular conjunctivitis, asthenopia,
sympathetic ophthalmia, glaucoma, cataract, hemianopsia, etc.
Throughout the book the attempt has been made to set forth the
most recent and well-established views in ophthalmology, and
references to authorities have been abundantly inserted that
readers may be aided in further research. The writer's judgment
on new suggestions has been freely given on points where he has
opinions. He is indebted to his friend, Dr. John E. Weeks, for the
pathological statements respecting so-called Bright's retinitis and
for his drawings which illustrate it. For preparation of the index
he again acknowledges with thanks the painstaking work of Dr.
D. W. Hunter. Additional cuts and plates have been inserted, and
attention is called to the list on p. xiii. The author has been much
gratified by the kind reception which his work has met, and has
for this reason sought in this edition to make it still more worthy
of acceptance.
PREFACE TO THE FIRST EDITION.
The present work has grown out of a treatise on diseases of the
eye, published in December, 1881, in Wood's Library of Standard
Medical Authors. The same arrangement of subjects is adhered
to: presenting in the first part the general anatomy and physi-
ology of the eye with its functional disorders, and in the second
part its inflammations and organic textural changes, and the
lesions of the accessory parts. That this order is both scientific
and natural seems evident. The spirit of the book is clinical, but
an adequate preparation for clinical and practical work demands a
wide range of preliminary knowledge. This is true of all branches
of medicine. To the study and experience needed in general medi-
cine, must be added for proper treatment of troubles of the eye, a
considerable acquaintance with physics, mathematics, and physio-
logical optics.
The knowledge which they furnish finds its chief application in
unravelling functional disorders of sight, viz., errors of refraction
and accommodation, and motility. These subjects as well as the
operative surgery of the eye will always constitute a field of special
practice. But in dealing with pathological conditions or inflamma-
tions, the physician must take his share with the ophthalmologist.
Nor can he refuse to take in hand the ophthalmoscope, because it
is not only essential to discovery of deep lesions of the organ, but
its revelations often have an important bearing upon remote
pathological conditions.
In accordance with the practical intent of the book, mathematical
formulae have been omitted; pathology and microscopic anatomy
have been presented so far as seemed helpful to an intelligent ac-
count of morbid processes; the share which micro-organisms have
in exciting diseases of the eye, has been fully recognized; no little
labor has been spent in setting forth its intimate relations to the
VI
PREFACE.
brain and nervous system, as both illustrations and descriptions
testify; the participation of the eye in numerous general diseases
or lesions, of remote organs, such as the kidneys, the uterus, the
heart, etc., and the reflex influence which the eye can sometimes
exert upon distant parts have been set forth.
The writer has quoted his own cases and experiences, and stated
his own opinions; he has familiarized himself with the work of
others, not only in their writings, but pretty largely by personal
acquaintance, and drawn freely upon their labors, as may be seen
by the references in the text, and by the bibliography.
Of the illustrations manj^ are familiar, while not a few have
either not hitherto appeared in ophthalmic text-books, or are orig-
inal.
In depicting diseases of the fundus oculi, black and white have
been used for many of the ordinary lesions, while colored plates
have been reserved for special conditions, some of which are familiar
and others unusual.
The liberality of the publishers in the number and quality of
the illustrations has been a source of gratification and will be ap-
preciated.
For the compilation of the copious indices acknowledgment is
due to the author's friend, Dr. D. W. Hunter. In them references
will be found to passages where the connection of general diseases
with eye pathology is mentioned, and one will need only to look
under the head of such diseases for the ocular complication.
To the medical profession, who have greatly honored him with
their confidence, and to the classes of Bellevue Hospital Medical
College whom he has taught and who have rewarded his efforts by
their attention and respect, the author offers this volume as an
attempt to discharge a great debt.
CONTEXTS.
PART FIRST.
CHAPTER I.
GENERAL ANATOMY OP THE GLOBE.
PAGE
Measurements—Component Structures—Vessels of the Globe and
their Relations to the Diagnosis of Diseases of the Eye, . . 1-i)
CHAPTER II.
GENERAL PHYSIOLOGY OF THE EYE.
Refraction: Cardinal Points—Optic Axis : Visual Line: Line of
Fixation : Angle Alpha : Angle Gamma—Accommodation—
Functions of the Retina: Blind Spot—Visual Acuity—Form
Sense — Test Types — Visual Field: Perimeters: Eccentric
Vision — Color Sense — Daltonism: Color Blindness — Light
Sense : Photometer of Forster : Phosphenes : Vascular Image
of Purkinje—Visual Purple,........10-34
CHAPTER III.
HOW TO EXAMINE THE EYE.
External Inspection—Oblique Illumination—Tension—Mobility—
Ana'sthetics—Functional Examination,.....35-38
CHAPTER IV.
THE OPHTHALMOSCOPE.
Instruments, their Principles and Varieties—The Fundus Oculi in
Health,............39"52
CHAPTER V.
GLASSES.
Spherical: Cylindric : Toric—Arrangement and Nomenclature—
Dioptries: Prisms,..........53-60
CHAPTER VI.
ACCOMMODATION AND ITS ERRORS.
Presbyopia-Second Sight—Spasm—Paralysis and Paresis, . 01-68
Vlll
CONTENTS.
CHAPTER VII.
ERRORS OF REFRACTION.
PAGE:
Ametropia, viz.: Hypermetropia, Myopia, Astigmatism—Diagnosis
of Refractive Errors—Subjective and Objective Methods—
Mydriatics—Ophthalmoscopic Optometry—Schmidt- Riinpler's
Method—Shadow Test—Thomson's Method. Hypermetropia:
Anatomical Characteristics : Symptoms : Complications : Diag-
" nosis : Prognosis : Treatment: Length of Axis in H. Myopia:
Length of Axis in M.: Causes—Functional Disturbances and
Pathological Anatomy—Prognosis : Diagnosis : Prophylaxis :
Treatment. Astigmatism: Definition : Varieties : Symptoms :
Dr. Thomas Young: Stokes' Lens : Diagnosis by Ophthalmo-
scope—by Shadow Test—by Ophthalmometer of Javal and
Schiotz : Disc of Placido—Anisometropia—Incidental Effects of
Glasses,............69-131
CHAPTER VIII.
BINOCULAR VISION.
Anatomy and Physiology of Ocular Muscles—Horopter—Homony-
mous and Heteronymous Images : Paralysis of Muscles : Symp-
toms : Diagnosis—Special Paralyses—Etiology : Origin of
Nerves animating Ocular Muscles: their Nuclei—Basal,
Nuclear, Orbital, Cortical Paralysis—Prognosis—Treatment—
Prisms, Tenotomy, Advancement—the Oculo-orbital Fascia—
Congenital Paralysis—Spasm of Muscles—Nystagmus—Con-
jugate Deviation,..........132-176-
CHAPTER IX.
STRABISMUS CONCOMITANS.
Varieties—Measurement—Etiology—Amblyopia—Course—Cause of
Strabismus—Treatment: Atropine, Eserine, Glasses, Opera-
tion : its Mechanism and Ultimate Result—Strabismus deorsum
vergens, and sursum vergens,........177-196
CHAPTER X.
ASTHENOPIA.
Accommodative—Properties and Uses of Prisms—Muscular—the
Metre-angle, how Measured, its Relation to Prisms—Adduction
and Abduction—General Divisions and Considerations—Sub-
jective Symptoms : Objective Symptoms—Tests by Prisms—
The Prism Carrier—Perimetric Examination—Retinal Anaes-
thesia—Nasal Disorders—Treatment—Prisms—Operations, . 197-230
PART SECOND.
CHAPTER I.
GENERAL CONSIDERATIONS.
General Treatment—Shades—Protective Glasses—Bandages—Asep-
sis : Antisepsis—Anaesthetics : Cocaine—Instruments—Mydri-
atics : Myotics—Collyria—Means of Reducing Tension, . 231.
CONTENTS.
IX
CHAPTER II.
THE EYELIDS AND CONJUNCTIVA.
Anatomy—Blephariti s m arginali s—Hordeolum—Chalazion—Phleg-
mon— Tarsitis—Eczema—Xanthelasma—Molluscum contagio-
sum—Herpes zoster Ophthalmicus — Epithelioma, Lupoid—
Naevi—Diseases of Eyelashes—Entropium—Ectropium—Bleph-
aroplasty — Blepharophimosis — Cantholysis— Canthoplasty—
Tarsoraphy—Injuries and Lacerations—Coloboma—Epicanthus
—Spasm of Orbicularis—Paralysis of Orbicularis—Ptosis—
Burns—Symblepharon—Ankyloblepharon, .... 247-291
CHAPTER III.
THE LACHRYMAL APPARATUS.
Anatomy—Diseases of Lachrymal Gland—Dacryocystitis—Chronic
—Acute—Lachrymal Fistula—Leptothrix lachrymalis, . . 292-307
CHAPTER IV.
THE CONJUNCTIVA.
Anatomy and Physiology—Conjunctivitis— Hypersemia Palpebralis
—Conjunctivitis Simplex, Catarrhalis, Symptomatic, Meta-
static, Catarrho-rheumatic—Purulenta, Neonatorum, Gonor-
rhoea—Plastica : Croupous—Diphtheritic, Acute and Chronic
—Granulosa : Trachoma — Papillary—Follicularis—Treatment
—Xeroma—Sequelae—Morbid Growths: Vernal Catarrh—Phlyc-
tenule : Herpes: Pemphigus; Traumatic Conjunctivitis—
Ecchymosis—Pterygium,.........308-370
CHAPTER V.
THE CORNEA.
Anatomy—Physiology—Pathology—Keratitis : Phlyctenula : Her-
pes : Pemphigus : Keratitis vasculosa — Malarial Keratitis :
Keratitis Interstitialis : Syphilitica—Suppurativa — Ulcera-
tiva—Purulenta discreta—Ulcus Corneae Serpens: Neuro-
paralytic Keratitis : Kerato-malacia—Ulcers—Keratitis postica,
Dendritica, or Arborescens—Mycotica, Interpalpebral De-
generation—Sclerosis—Arcus senilis: Sequelae—Opacity: Fistula
—Staphyloma, Cornea conica: Morbid Growths,.... 371-423
CHAPTER VI.
THE SCLERA.
Anatomy—Episcleritis—Scleritis—Sclero-keratitis—Staphyloma, . 424-429
CHAPTER VII.
THE IRIS.
Anatomy and Physiology — Mydriasis — Myosis — Hippus — Irido-
donesis—Congenital Defects : Albinism—Irideremia—Coloboma
Membrana pupillaris — Iritis: Serosa, Spongiosa, Plastica,
Suppurativa—Complications : Symptoms—Sequelae—Causes-
Prognosis— Treatment — Varieties: Syphilitic, Rheumatic,
Gouty, Gonorrhceal—Tumors—Tubercles—Sarcoma—Cysts, . 430-453
X
CONTENTS.
CHAPTER VIII.
OPERATIONS ON THE IRIS.
PAGE
Iridectomy—Iridotomy—Iridorhexis—Iridavulsion—Corelysis—Iri-
dodesis, . ..........454-461
CHAPTER IX.
THE CILIARY BODY.
Anatomy—Ciliary Muscle —Cyclitis : Serous, Plastic, Purulent,
Acute and Chronic : Traumatic—Hypotony—Ophthalmomala-
cia: Morbid Growths,.........462-470
CHAPTER X.
THE CRYSTALLINE LENS.
Anatomy — Development — Images of Purkinje — Arcus senilis
Lentis—Dislocation,..........471-477
CHAPTER XI.
CATARACT.
Pathology — Symptoms — Examination — Varieties — Diagnosis—
Complications — Prognosis — Treatment—Soft Cataract—Dis-
cission—Simple Linear Extraction—Extraction of Hard Cata-
ract, with Iridectomy, without Iridectomy—Secondary Cata-
ract—Glasses for Aphakia.........478-511
CHAPTER XII.
THE VITREOUS BODY.
Anatomy—Hyalitis—Persistent Hyaline Artery—Synchisis Scintil-
lans—Muscae Volitantes — Cysticercus — Membranes—Hemor-
rhage—Foreign Bodies,.........512-518
CHAPTER XIII.
WOUNDS ASD INJURIES.
Contusions and Blows—Wounds—Foreign Bodies—The Magnet—
Burns,.............519-530
CHAPTER XIV.
SYMPATHETIC OPHTHALMIA.
Irritation—Inflammation—Causes—Stages—Prognosis—Symptoms
—Neuro-retinitis—Mode of Transmission—Treatment— Iridec-
tomy— Enucleation—Optico-ciliary Neurectomy—Exenteration
—Risk of Enucleation,.........531-547
CHAPTER XV.
GLAUCOMA.
Degrees of Tension—Glaucoma Simplex—Symptoms—Development
—Glaucoma Acutum—Subacutum—Chronicum—Hemorrhagi-
eum — Secondarium — Buphthalmus — Diagnosis—Prognosis—
CONTENTS.
XI
Etiology and Pathogenesis — Pathology—Treatment—Iridec-
tomy — Sclerotomy — Posterior Sclerotomy—Malignant Glau-
coma—Cystoid Cicatrix—Rationale of Iridectomy—Results, . 548-572
CHAPTER XVI.
THE CHOROIDEA.
Anatomy—Albinism—Coloboma—Choroiditis—Pathology—Colloid
Degeneration—Symptoms and Subdivisions—Metamorphopsia
—Megalopsia—Micropsia—Treatment—Choroiditis Metastatica
—Panophthalmitis Suppurativa—Laceration of the Choroid—
Hemorrhage—Detachment—Tubercles—Tumors—Ossification, 573-593
CHAPTER XVII.
THE RETINA.
Minute Anatomy—Opaque Nerve Fibres—Coloboma of the Macula
—Hyperaemia—Pulsations of Arteries and Veins—Anaemia-
Spasm of Arteries—Ischaemia—Embolism and Thrombosis—
Hemorrhage—Retinitis Apoplectica—Miliary Aneurisms—Re-
tinitis: various appearances—Etiology—Acute Traumatic Retin-
itis—Retinitis Simplex—Albuminurica under various conditions
— Pathology — Glycosurica — Leucocy thaemica — Syphilitica—
Proliferans — Punctata Albescens—Pigment Degeneration—
Neuro-retinitis pigmentosa—Detachment—Pathogenesis —Gli-
oma—Pseudo-glioma,.........594-640
CHAPTER XVIII.
THE OPTIC NERVE.
Anatomy and Physiology of Optic Nerve, and of Brain connected
with it—Coloboma of Sheath—Opaque Nerve Fibres^Con-
nective Tissue on Disc—Hyperaemia—Anaemia—Hemorrhage—
Papillitis—Neuro-retinitis—Neuritis Hemorrhagica—Peri-neu-
ritis—Subjective Symptoms—Pathological Anatomy—Etiology
— Pathogenesis — Prognosis— Treatment — Retrobulbar Neu-
ritis: Chronic, Acute, Toxic, Alcoholic, Tobacco—Atrophy of
Optic Nerve—Morbid Anatomy........641-693
CHAPTER XIX.
AMBLYOPIA AND AMAUROSIS.
Traumatic—Concussion of Spinal Cord—By Lightning, from Hem-
orrhage—Toxic: by Quinine, by Salicylic Acid—Uraemic—Gly-
cosuria by Pregnancy—Hysterical—Dyslexia, Paralexia, Alexia
— Hemeralopia—Snow-blindness—Dental Amblyopia—Hyper-
esthesia—Hemianopia: monocular, homonymous, binocular,
nasal, bitemporal—Causes—Diagnosis—Amaurosis in Young
Children — Fndiscovered Monocular Blindness — Simulated
Blindness.............694-721
CHAPTER XX.
THE ORBIT.
Anatomy — Periostitis Orbitae— Cellulitis—Phlegmon—Inflamma-
tion of Oculo-orbital Fascia—Thrombosis of Veins and of Cav-
Xll
CONTENTS.
ernous Sinus—Tumors—Tumors of Optic Nerve—Extenteratio
Orbitae—Pulsating Exophthalmus—Enophthalmus Traumaticus
—Empyema of Frontal Sinus—Hemorrhage into Orbit—Wounds
and Injuries—Fractures—Wound of Optic Nerve — Foreign
Bodies in Orbit—Exophthalmic Goitre,.....
CHAPTER XXI.
USE OF ARTIFICIAL EYES.
Mules' Artificial Vitreous,.........768-770
CHAPTER XXII.
STATISTICS OF EYE DISEASES.
Relative Proportion of Eye Diseases—Cohn—Causes of Blindness,
Magnus, etc.,...........771-776
PAGE
722-767
Bibliography,
777-780
LIST OF ILLUSTRATIONS.
FIG- PAGE
1. Section of Eye, horizontal.............................Heitzmann. 3
2. Ciliary Region.........................................Heitzmann. 4
3. Section of Eye showing Uvea..................Merkel, G. and S. 5
4. Blood-vessels of Eye.......................................Leber. 7
5. Refraction by a spherical Surface................................. 11
6. Cardinal Points..........................................Landolt. 13
7. Angle Alpha, angle Gamma, etc..........................Landolt. 14
8. Reflex from Cornea and Lens..........................Hehnholtz. 16
9. Changes during Accommodation..........................Landolt, 17
10. Relative Accommodation..................................Loring. 19
11. Retina : Macula Lutea....................................Strieker. 20
12. Blind Spot.............................................Helmholtz. 21
13. Perimeter.........................................Priestley Smith. 25
14. Boundaries of Color Sense.......•........................Landolt. 27
15. Optogram on Retina........................................Ayers. 33
16. Oblique Illumination.......................................Noyes. 36
17. Ophthalmoscopy—direct Method..........................Noyes. 40
18. Ophthalmoscopy—indirect Method.........................Noyes. 41
19. Loring's Ophthalmoscope................................Loring. 42
20. Loring's Ophthalmoscope..................................Loring. 43
21. Noyes' Ophthalmoscope...................................Noyes. 45
22. Optic Disc Surface and Section...........................Landolt. 48
23. Blood-vessels of Retina.....................................Leber. 49
24. Varieties of spherical Lenses................................Ganot. 53
25. Cylindric Lenses............................................Noyes. 54
26. Nachet's Spectacle Frame..........................Tiemann & Co. 58
27. Emmetropia, Hypermetropia and Myopia............ ___Brown. 69
28. Course of parallel Rays in convex Lens.....................Ganot. 69
29. Parallax of Vessels in excavated Disc.............................. 75
30. Explanation of Shadow Test........,..................Nettleship. 77
31. Explanation of Shadow Test............... ...........Nettleship. 78
32. Scheiner's Test..........................................Follin. 82
33. Myopia with Crescent..................................Nettleship. 97
33a. Section of Myopic Eye.................................Nettleship. 97
34. Myopia with choroidal Lesions............................Jaeger. 98
35. Supratraction of Optic Fibres in Myopia...................Weiss. 98
36. Myopia, Choroidal Crescent................................Jaeger. 99
37. Myopia, circum-papillary Atrophy........................Jaeger. 100
38. Myopia, Lesion at Macula .................................Jaeger. 101
39, 40. Course of Rays through cylindric Lens...................Fick. 113
41, 42, 43, 44, 45. Varieties of Astigmatism.................Hartridge. 115
xiv LIST OF ILLUSTRATIONS.
FIG. PAGE
46, 47. Tests for Astigmatism...................................Green. 119
48. Tests for Astigmatism......................................Green. 120
49. Ophthalmometer of Javal and Schiotz......................Noyes. 124
50, 51. Images seen by Ophthalmometer.......................Javal. 125
33. Disc of Placido..............................................Noyes. 126
53. Axes of ocular Muscles...................................Landolt. 133
54. Converging visual Lines.................................. Follin. 136
55. Diverging visual Lines....................................Follin. 137
56, 57, 58, 59. Images in muscular Paralysis..... ...........Zehender. 142
60, 61, 62, 63. Images in muscular Paralysis................Zehender. 143
64. Section of Skull in frontal Plane through Sphenoid........Henle. 145
65. Base of Brain, Pons and Medulla................................. 146
66. Optic Commissure and Tract.......... .........Merkel, G. and S. 147
07. Oblique view of sagittal Section of Mid-brain..............Henle. 148
68. Diagram—Lesion of Pons with alternating motor Paralysis.. Starr. 149
69. Diagram—Lesion of Pons with alternating Anaesthesia.... Starr. 149
70. Section of caudal Edge of anterior Corp. Quadrigem.. Obersteiner. 150
71. Sagittal Section of Medulla modified from Edinger................ 151
72. Diagram of Nuclei in Medulla...............................Knies. 152
73. Nuclei of third and fourth Nerves...........................Perlia. 153
74. Nuclei of third and fourth Nerves........................Edinger. 153
75. A. Section of Brain on median Plane........................Starr. 156
B. Localization Centres....................................Starr. 156
76. Horizontal Section of internal Capsule.................M. Foster. 157
77. Strabismus Forceps................................................ 189
78. Strabismus Hook.................................................. 189
79. Refraction by a Prism......................................Ganot. 198
80. Displacement of Object by Prism................................. 199
81. Adduction by Prisms.................,............................ 199
82. Abduction by Prisms.............................................. 200
83. Metric Angle.................................................Nagel. 203
84, 85. Prisms in Series for testing at a remote distance........Noyes. 211
86. Prism-Carrier for testing at the working distance..........Noyes. 213
87. Speculum, flat View.......................................Noyes. 238
88. Speculum, profile View............................... ......Noyes. 239
89. Tarsi Palpebrarum.....................................Schwalbe. 247
90. Exterior of Eye and Lids................................Schwalbe. 248
91. Section through Globe and Orbit.......................Schwalbe. 249
92. Section of upper Lid...................................Schwalbe. 251
93. Eye-lid Clamp Forceps...................................Noyes. 254
94. Operation for spasmodic Entropium........................Meyer. 264
95. Hotz's Operation for Entropium.............................Hotz. 266
96. Green's Operation for Entropium...........................Green. 266
97. Ectropium at inner Canthus...........................Galezowski. 269
98, 99, 100, 101. Operations for Ectropium............,.......Meyer. 270
102, 103. Blepharoplasty........................................Noyes. 274
104, 105, 106, 107. Blepharoplasty..............................Noyes! 275
108. Blepharoplasty..............................................Noyes. 276
109, 110, 111, 112. Blepharoplasty---..........................Noves. 277
113. Laceration of upper Lid....................... ..........Lawson 279
114. Wound of lower Lid, etc....................................Noyes. 279
115, 116. Operation for Ptosis................................Wecker 28(*
LIST OF ILLUSTRATIONS. xv
FIG- PAGE
117, 118. Symblepharon.........................................Meyer. 289
119. Abscess of lachrymal Sac...................................Noyes. 296
120. Empyema of frontal Sinus.................................Noyes. 297
121, 122. Lachrymal Knives........................................... 300
123, 124, 125, 120, 127. Lachrymal Probes............................... 300
128. Gouge for lachrymal Duct..................................Noyes. 303
129. Abscess of lachrymal Sac..................................Noyes. 305
130. Desmarres' Elevators.......... ................................... 324
131. Gonococci.................................................Michel. 326
132. Trachoma Follicles..........................................Fuchs. 345
133. Forceps for follicular Trachoma...........................Noyes. 349
134. Squeezing follicular Trachoma.............................Noyes. 349
135. Gibson's Forceps for everting Lids................................ 353
136. Pterygium......................................................... 369
137. Corneal Corpuscles___...................................Strieker. 373
138, 139. Keratitis Mycotica......................................Haab. 376
140. Ulcer of Cornea........................................Galezowski. 380
141. Herpes Corneae.............................................Haab. 384
142, 143. Teeth in hereditary Syphilis and from Mal-nutrition.. Noyes. 391
144. Keratitis with Hypopyum.............................Galezowski. 397
145. Keratitis Postica.....................................Galezowski. 408
146. Keratitis Dendritica (vel Arborescens)..............Hansen-Grut. 409
147. Tattooing Needle................................................ 413
148,149,150. Staphyloma Corneae................................... 415
151. Operation for Staphyloma Corneae..........................Noyes. 418
152. Epithelioma Corneae ......................................Noyes. 423
153, 154. Membrana Pupillaris perseverans............................. 439
155. Congestion in Iritis...................................__......... 441
150. Posterior Synechiae.....................................Sichel, Jr. 441
157, 158. Occlusion and Exclusion of Pupil...................Sichel, Jr. 444
159, 160, 161. Forceps, Tyrrell's Hook, and Lance Knives.............. 455
102. Matthieu's Iris Forceps............................................ 450
-03, 104. Iridectomy.............................................Meyer. 457
165, 166. Iridectomy...................... ......................Meyer. 458
167. Wecker's Forceps-scissors...............................Wecker. 459
168, 169, 170. Iritomy Knife-needles.................................... 400
171, 172. Ciliary Muscle in H. and M... .......................Iwanoff. 463
173,174. Plastic Cyclitis ..........................................Alt. 465
175,176. Irido-Cyclitis.............................................Alt. 465
177,178. Plastic Cyclitis..........................................Alt. 465
170, 180, 181. Results of Cyclitis ................................Noyes. 468
182. Eye of embryo Calf at third Month.......................Kolliker. 471
183. Images from Cornea and Lens........................Helmholtz. 472
184. Dislocation of Lens...................................... Lawson. 474
185. Zonular (laminated) Cataract...............................Meyer. 483
186, 187, 188. Congenital Cataract...............................Wilde. 484
189, 190. Congenital Cataract ...................................Wilde. 484
191. Anterior Polar Cataract....................................Wilde. 484
192. Discission of Cataract.....................................Meyer. 490
193. Linear Extraction of Cataract upward.....................Wolfe. 492
194. Linear Extraction of Cataract outward.....................Meyer. 493
395. Section of Cornea in various Methods of Cataract Extraction.....
Sichel, Jr. 496
XVI
LIST OF ILLUSTRATIONS.
FIG. PAGE
196. Graefe's linear Extraction—making the Section.............Noyes. 497
197. Graefe's linear Extraction—making the Section ...........Meyer. 498
198. Wire Retractor for upper Lid..............................Noyes. 499
199. Cataract Knife.................................................... 504
200, 201. Cystitome and Spatula : Fixation Bident.................. 504
202. Secondary Cataract...................................Galezowski. 508
203, 204, 205. Knife, Needles, and Hook for membranous Cataract..... 509
206. Persistent hyaloid Artery...................................Little. 513
207. Connective Tissue in Vitreous..............................Jaeger. 514
208, 209. Irido-dialysis : Rupture of Sclera...................Lawson. 519
210. Wound of Cornea, Eyelid and Cheek......................Noyes. 522
211. Appliance for oblique Illumination.........................Noyes. 523
212. Iridectomy after sympathetic Ophthalmia ...............Lawson. 539
213. Myopia and Glaucoma ....................................Jaeger. 549
214, 215. Excavation of optic Nerve : Glaucoma................Jaeger. 550
216. Sclerotomy—downward or bilateral.......................Lawson. 565
217. Colloid Deposits in Choroid................................Mtiller. 575
218. Patch of Choroiditis.......................................Michel. 576
219. Exudative Choroiditis ; peripheral.....................Jaeger, j
220, 221, 222, 223, 224. Atrophic Choroiditis.................Jaeger, f Plate
225. Choroiditis atrophica in Myopia............................Jaeger. 582
226. Diagram of minute Structure of Retina..................Strieker. 595
227. Minute Anatomy of Retina......................Perrin et Poncet. 596
228. Embolism of Arteria Centralis Retinae............. .....Magnus. 602
229. Dilated Choroidal Capillaries....................Perrin et Poncet. 605
230. Section of Retina in Morbus Brightii.......................Weeks. 617
231. Retinis albuminuria......................................Weeks. 618
232. Section of Iris in Albuminuric Retinitis....................Weeks. 619
233. Choroidal Lesion in Albuminuric Retinitis.......Perrin et Poncet. 619
234. Retinitis Pigmentosa......................................Meyer. 628
235. Optic Commissure and Tract....................Merkel, G. and S. 641
236. Medulla Oblongata and Pons.............................Edinger. 642
237. Diagram of Optic Tractus Fibres, etc....................Edinger. 643
238. Scheme of Cerebral visual Apparatus................Obersteiner. 646
239. Sections of Occipital Lobe, to show visual centre........Henschen 648
240. Schweigger's Perimeter................................Schweigger. 653
241. Transverse Section of Skull.................................Henle. 654
242, 243. Cerebral Convolutions................................Gowers. 655
244. Localization Areas on Brain Surface........................Starr. 656
245. Functional Centres of Brain Surface.......................Berger. 656
246. Connective Tissue on Optic Disc........................Massillon. 659
247. Hemorrhage within Optic Nerve Sheath.........Priestley-Smith. 661
- 248. Papillitis—Section of Globe............................. Allbutt. 663
249. Distention of Optic Sheath in Neuritis .. Pagenstecher and Genth. 667
250. Optic Neuritis—Microscopic Section---Pagenstecher and Genth. 668
251. Optic Neuritis—Longitudinal Section---Edmunds and Lawford. 669
252. Optic Neuritis—Cross Section...........Edmunds and Lawford. 670
253. Optic Neuritis—Cross Section.............................Uhthoff 685
254. Optic Neuritis—Cross Section.............................Uhthoff 685
255. Atrophy of Optic Nerve....................................Jaeger. 690
256. Section of Optic Nerve, Normal..................Perrin et Poncet 691
257. Section of Optic Nerve, Atrophic................Perrin et Poncet 691
LIST OF ILLUSTRATIONS. xvii
FIG. PAGE
258. Diagram to illustrate left Hemianopia.....................Seguin. 715
259. Angioma of Orbit and neighboring Skin......................Bull. 739
260. Plastic Operation for Occlusion of Orbit....................Noyes. 742
261. Empyema of Frontal Sinus................................Noyes. 749
262. Diagram of origin of Ocular Muscles at Orbital Apex......Merkel. 755
263. Section of Muscles at Apex of Orbit.......................Merkel. 755
264. Section of Skull through Optic Canals.......Berger and Tyrman. 758
265. Patient with Foreign Body (Breech-pin) in Orbit, etc......Noyes. 762
266. The above-mentioned Foreign Body (natural size)..........Noyes. 763
267. Skull prepared to show the Position of the Breech-pin in the Orbit
and Cranial Cavity—the Separated frontal Bone Exhibits the
Place of Trephining for Cerebral Abscess................Noyes. 763
268. Exophthalmic Goitre.....................................Noyes. 764
269. Artificial Eyes ..................................................... 769
t
DISEASES OF THE EYE.
PART FIRST.
CHAPTER I.
GENERAL ANATOMY OF THE GLOBE.
The eyeball is a spheroid, rotating in the orbit, upon a cushion
of fat and fibrous tissue, and protected by the eyelids. The fibrous
membrane on which it rests, called oculo-orbital fascia or capsule
of Tenon, is shaped into a cup and moistened by serum: and in
front it is lubricated by secretion from its covering membrane, the
conjunctiva, and from the lachrj'mal glands. It consists externally
of the cornea and sclerotica or sclera. A line perpendicular to the
centre of the cornea is its antero-posterior diameter or axis; and
intersecting this at the centre of rotation we have the vertical and
the transverse axes. A plane through both vertical and antero-
posterior diameters will touch the surface on its vertical meridian.
A similar plane, passing through the transverse and antero-pos-
terior axes, will form at the surface the horizontal meridian.
The plane passing transversely through the vertical meridian forms
the equator, and the extremities of the antero-posterior axis are
the poles of the eye. All planes going through the geometrical
centre form principal meridians or great circles. Planes vertical
to the axis but not passing through this centre form lesser circles.
or those of latitude.
At birth the normal length is 17.5 mm. and the full size is not
reached until after puberty. The following table of measurements is
1
2
DISEASES OF THE EYE.
mm.
compiled from various authors, viz., Jaeger, Merkel, Reuss, etc.;
chiefly from Merkel.
Antero-posterior diameter externally,
Transverse, "
Vertical,
Sclera, thickness behind,
Cornea, thickness at apex,
" " " margin, .
" radius of front surface, .
" diameter of its base (Jaeger, 12 mm.)
" height of apex above base,
Pupil, average diameter,
Lens, thickness (axis) in repose1 (Reuss),
" equatorial diameter,
" radius of anterior surface (Reuss),
" " " posterior " (Reuss, 8.2 mm.) Aubert, 6.
Distance from outer surface of cornea to lens (Horstmann), 3.
(Apparent distance .5 mm. less.)
Depth of anterior chamber, .
Vitreous axis,.....
Retina, thickness at optic disc,
" " at fovea centralis,
" diameter of fovea centralis,
Optic disc, diameter,
Distance from centre of optic disc to centre of fovea,
Internal axis of eyeball from apex of cornea to surface of
fovea (Becker),........23.8"
24.3
23.6
23.4
1.
.9
1.1
7.7
11.6
2.6
4.
3.8
7 to 10.3
10.6
2.6
15.1
.4
.1
.4
1.4
4.0
.2 to
Absolute accuracy in all these details is not attainable; even
the length of the axis measured to the fovea centralis is not settled,
because it is not absolutely uniform among normal eyes. For
further details see Nagel2 and Becker.3
Going from before backward we find the following parts,
viz.: the cornea; the space called aqueous chamber filled by
aqueous humor, and which contains also the iris; it divides the
aqueous chamber into the anterior and posterior chambers, and
is itself perforated by an opening, the pupil; the crystalline
lens, inclosed in a capsule which by certain fibres is attached
at its edge to the tips of the ciliary processes; behind the lens
the corpus vitreum or vitreous humor; in contact with the
vitreous is the retina,* into which passes the optic nerve *
1 "Untersuchungen fiber die optische Constanten Ametropischer Augen "
Graefe's Archiv, xxiii., 4, p. 183.
2 G. u. S. " Handbuch," Bd. ii., pp. 280 to 290*.
3 Ibid., Bd. v., pp. 432 to 442.
GENERAL ANATOMY OF THE GLOBE. 3
external to the retina is the choroid, which, at a place near the
corneal edge, takes the name of ciliary body, and is raised into
folds called ciliary processes, and is also continuous with the iris ;
outside of the choroid and in front, joined to the cornea, is the sclera,
which behind is continuous with the sheath of the optic nerve. The
optic nerve passes through the sclera and choroid and joins the
retina.
Figure 1 illustrates and describes the facts mentioned.
For practical purposes it is important to understand correctly
the relations of the parts composing the anterior half of the eye.
The subjoined diagram, Fig. 2, presents them, in most respects,
satisfactorily.
The edge or limbus corneal is a ring-shaped space about 1 to 2
mm. in breadth whose borders are often very ill defined; the epi-
thelium hero increases in thickness and merges into the conjunctiva.
A practical point to be noted is that the limit of the transparent
4
DISEASES OF THE EYE.
cornea lies in front of the place from which the iris springs;
hence, the anterior chamber can be punctured through the sclera.
A clear perception of this fact is indispensable in operating at this
region. The existence of that congeries of vessels called the
canal of Schlemm or the circular venous sinus, is also to be
noted. It has important relations to the physiology of the
anterior chamber. It is the outlet by which the aqueous humor
finds its way into the circulation, and is supposed by Schwalbe
to have in its wall minute clefts for this purpose; but this is
disputed by Leber. At the angle are to be found delicate fibres
passing from the cornea across to the iris. They are insignificant
Fig. 2.
in man, and are called the pectiniform ligament. In lower
animals, viz.: in the ox and in swine, etc., they are more de-
veloped, and constitute the canal of Fontana.' Upon theories
of intraocular pressure, the parts now alluded to have important
significance.
The aqueous humor is derived from the vessels of the iris and
ciliary processes. The posterior chamber is entirely shut off from
the anterior chamber by contact of the iris with the lens and
even when the pupil is well dilated the contact continues ' The
anterior chamber appears to have much less than its real denth
which is from 2.6 mm. to 3.5 mm., for the same reason that a brook
is likely to deceive us as to the depth of water. Refraction brings the
ins nearer than it really is, and likewise betrays us into wadin-deet
er than we thought to go. In each case we may have cause for vegZ
GENERAL ANATOMY OF THE GLOBE.
5
The sphincter of the iris makes the pupillary part of the membrane
thicker than the rest of it. Another point to be observed is that
the ciliary processes do not touch the rim of the crystalline. There
is always a separation between them. The zonula of Zinn, or sus-
pensory ligament of the lens, comes from the posterior surface of the
ciliary processes, and is attached to the lens-capsule (see Fig. 1.)
It splits into fine fibres, of which more go to the anterior surface of
the lens than to the posterior.
Passing to the deep part of the eye, we have the retina, begin-
ning at the optic nerve and lining the concavity of the globe to the
posterior edge of the ciliary body. Because this edge is irregular
it is called the ora serrata. The retina is transparent and near
the optic nerve is thicker than at any other part. At a point 4
mm. from the centre of the nerve, on its temporal side, and about
1 mm. below it, is a depression called the fovea centralis. Around
it the retina has a faint yellowish or tawny color over an ill-defined
elliptical space, and this region is called the macula lutea, or yel-
low spot. Its greatest diameter, which is horizontal, is about 0.8
mm. The thickness of the retina near the nerve is 0.3 mm. The
fovea centralis is 0.2 mm. in diameter. Outside of the retina is
the choroid, which is perforated by the optic nerve, and consists
chiefly of blood-vessels and pigment and connective tissue. The
pigment is of a dark brown color,
and varies in amount in different
persons. We find a layer of hex-
agonal epithelium, filled with pig-
ment-granules and each contain-
ing a nucleus; this was formerly
assigned to the choroid, but is
now regarded as the most exte-
rior layer of the retina. In the
choroidal stroma are irregular
cells with stellate processes and
nuclei filled with pigment-gran-
ules. There is also free pigment
scattered among the vessels. The
choroidal vessels will be mentioned hereafter. At the point where
the retina terminates, or no longer possesses nerve-elements, we
have the beginning of that part of the choroid called the ciliary
body (Fig. 3). It is divided into the pars non plicata behind, and
the pars plicata in front. The plicae or folds are some seventy in
number, and of unequal length. They consist of a congeries of
vessels, which in front lift themselves up into projecting masses,
and are called the ciliary processes. The great abundance and
Fig. 3.—Cc, Cilliary process; Vv, venae
vorticosse.
6
DISEASES OF THE EYE.
plexiform arrangement of blood-vessels in the choroid and ciliary
processes is necessary to secrete the pigment and to furnish nutri-
tive material for the vitreous body and lens, which have no blood-
vessels.
Outside of the ciliary body, inserted between it and the sclera,
is a mass of muscular fibres, known as the ciliary muscle (see Fig.
2). Its most exterior fibres run in the meridians of the eye; those
which lie next, run in oblique directions whichjslant more and more
as we go deeper, until we come to the innermost set, which take a
circular direction. The whole mass, in meridional section, has a tri-
angular form, whose apex and point of attachment is near the canal
of Schlemm. For reference to the variations in its form and size,
see chapter on refraction.
The place of attachment is called by Gerlach the ligamentum
annulare. The anatomy of this region of the eye was long misun-
derstood, and there is likely to be confusion from the variety of
terms which have been employed at different periods. The inser-
tion of the ciliary muscle is upon the choroid, and its effect is to
relax the fibres which pass from the tips of the ciliary processes to
the margin of the lens, and which fibres are known as the zonula of
Zinn, or suspensory ligament of the lens. This name is also ex-
tended to a transparent membrane which lies between the ciliary
body and the vitreous. The purpose and effect of the ciliary muscle
is to permit the crystalline lens to become more convex. The space
between the ciliary processes and the margin of the lens has im-
portance in reference to the escape of fluid from the vitreous to the
canal of Schlemm and the exterior circulation of the eye. The iris
has pigment, blood-vessels, epithelium, and also muscular fibres
which regulate the size of the pupil ; iris, ciliary body, and choroid,
are together known as the uvea.
We have next to speak of the vessels of the eye, and for much
of our knowledge of the details of their arrangement we are in-
debted to Leber, whose diagram is introduced below (Fig. 4).
There are four systems of vessels, which may be distinguished
from each other: 1st, the arteria centralis retinae, which enters
the eye through the optic nerve, is destined exclusively for the
retina and optic nerve, and forms few anastomoses with other
vessels, and those chiefly at the edge of the optic disc (rami com-
municantes). At the choroidal opening through which the nerve
passes to reach the retina is found the only communication between
the retina and choroid, and the optic nerve, and it is known as the
circle of Holler. There is also communication between the sheath
of the nerve near the sclera and the above-mentioned vessels. The
retinal system is remarkably isolated, and bv Cohn is classed
as "a terminal system;" 2d,the posterior, or short ciliary arteries
GENERAL ANATOMY OF THE GLOBE.
7
which perforate the posterior part of the sclera and supply the
choroid, and, with the long ciliary arteries, are the chief source of
the elaborate vascular system of the choroid, of the ciliary body,
and of the iris; 3d, the anterior ciliary arteries derived from vessels
which come from the recti muscles and perforate the sclera about
four to six millimetres behind the cornea. They are visible to the
naked eye, more or less conspicuously, and supply the ciliary body,
the iris, and the anterior part of the sclera, and furnish the plasma
Fig. 4.
which nourishes the cornea. These vessels join with the branches
of the posterior ciliary arteries, and at the border of the cornea
send off loops, which constitute the peculiar vascularity of this re-
gion. Moreover they here anastomose with, 4th, the vessels which
have come from the ocular conjunctiva. It thus happens that for
a zone about the cornea there is a system of vessels which have
communication with the face and with the deep and the superficial
tissues of the eye. The vessels proper to the conjunctiva are of
8
DISEASES OF THE EYE.
darker hue than those more deep, and they can be moved about as
the membrane is slipped over the sclera by traction of the lids.
This statement of the anatomy of the vessels shows how untrust-
worthy is any attempt to make a diagnosis of the locality of an
inflammatory process by fixing attention chiefly on the kind of
hyperaemia. The vascular phenomena are important as auxiliary
evidence, but do not take the first rank in deciding a diagnosis.
The non-vascular structures of the globe form so much of its
bulk, that some statements respecting their nutrition and the lymph
circulation will be proper; Schoeler1 and Knies2 have elaborately
studied these questions.
The fluid to supply the aqueous, to nourish the lens and to nour-
ish the anterior part of the vitreous, comes from the ciliary processes
and posterior surface of the iris (at Schoeler's "secretory angle"),
while the deep part of the vitreous may have nourishment from the
choroid through the retina. The current for the lens goes through
the canal of Petit and does not seem to come from the vitreous (on
this point Knies and Schoeler are at variance). The cornea may be
nourished from the aqueous as well as from its adjacent vessels at
the limbus. It, like the sclera, has no true lymphatic vessels, but
numerous channels in its substance, through which fluid passes. It
admits fluid both from in front and from behind, but the epithelium
and endothelium greatly hinder the transit of fluid, as is proven
when these layers are removed either mechanically or by ulceration.
From the corneal substance the fluid passes into the veins, and also
into the canal of Schlemm; and the intraocular lymph current is
largely from behind forward to this point; while from the vitreous
it also goes out along the perivascular lymph spaces of the venae
vorticosa. Here it comes into the supra-choroidea and besides
entering the general circulation it also makes its way into the
inter-vaginal lymph space of the optic sheath.
Stilling states that fluid passes from the deep part of the vitre-
ous through the optic nerve along the space outside the effete hya-
loid artery (Canal of Cloquet).
The degree of fulness of the globe, or what is called intraocular
tension, has important physiological and pathological bearings.
Much study has been given to it. To a remarkable degree the in-
traocular circulation is independent of the systemic vessels because
shut within a special cavity, yet a slight relationship exists (Schul-
ten3). The tone of its vessels and consequently the tension of the
globe, are largely controlled by the nerves. For example, stimula-
tion of the sympathetic in the neck contracts the ocular blood-
1 Jahresbericht, Berlin, 1882, pp. 52 to 92.
2 Archives of Ophth. and Otol., vol. vii., p. 347.
B Graefe's Archiv, Bd. xxix., Abth. Ill, p. 1.
GENERAL ANATOMY OF THE GLOBE.
9
vessels, and the effect may be sufficient to cause dilatation of the
pupil. Both arteries and veins become smaller and the intraocular
tension falls. The same result ensues from irritation of the spinal
cord above the third or fourth vertebrae, but not below this point.
Irritation of sensitive nerves, as when creosote is applied to
the cornea, causes contraction of the pupil and increase of tension.
A similar increase occurs after irritation of the sciatic nerve.
If, on the other hand, the contractility of the vessels is destroyed,
they passively dilate and tension rises. Prolonged pressure on the
globe will have this result, as Donders observed, and if this be done
by injecting fluid into the globe and subsequently allowing the
pressure to fall, it does not return to the normal because the ves-
sels have been paralyzed and remain dilated. Schoeler says that
cutting the sympathetic increases the secretion of aqueous humor
and diminishes its excretion. He also says that the fifth nerve con-
tains fibres which influence secretion within the eye, and can in-
crease, hasten, and modify it more when it is cut within the cranium,
than after division of the sympathetic. He also asserts that he
found these fibres to lie on the medial fourth of the width of the
nerve; he cut the nerve three-quarters across behind the ganglion
of Gasser, and because the innermost fourth remained, no changes
occurred in the secretions of the eye. When these last fibres were
cut the changes took place.
Paracentesis of the anterior chamber is followed by paresis and
enlargement of the vessels, and the aqueous humor becomes fibrin-
ous. This proceeding, therefore, has ultimate effects which may be
directly contrary to what is desired; and a similar remark applies
to strong pressure by a bandage.
In experimental research tubes called manometers are inserted
into the eye, and the ordinary intraocular pressure is equal to
twenty-five millimetres of mercury.
CHAPTER II.
GENERAL PHYSIOLOGY OF THE EYE.
Refraction.
Like the photographer's camera, the eye consists of an arrange-
ment of lenses to throw a luminous picture upon a sensitive surface.
The former we ha ve in the cornea with the aqueous humor, in the
crystalline lens and the vitreous. The cornea and the lens are the
essential refractive or dioptric apparatus. The sensitive or per-
cipient structure is the retina.
The properties of a lens are determined by (1) the transparency
and homogeneity of its structure, (2) by its refractive power or
index, (3) by its form; and (4) its effect is varied according to its
distance from the surface on which the picture is formed.
Absolute transparency is never met with in nature, and both
the cornea and lens become visible by the light which cannot pass
through but is reflected from their substance. Neither are these
structures perfectly homogeneous and especially is this true of the
lens. Refractive power or index signifies the ratio by which light
falling on a medium, is diverted from its previous course through
air—and it is assumed that the direction is not perpendicular, and
that the surface of the medium is plane, and not curved. If air be
called 1, the refractive index of the cornea is the same as of water,
viz., 1,336, while the index of the crystalline is 1,437. All lenses
have curved surfaces, and the form of the cornea and lens have
been carefully studied. See table below. The greater the number
of surfaces in a refractive system, the more complex the problem.
As light goes from one medium to another of different refractive
index it is deviated, and every surface must be taken into account.
Moreover, the separation of the surfaces from each other has its
influence.
The first deviation is at the exterior of the cornea, and no
other takes place until we reach the surface of the lens:—because
we may neglect the thickness of the cornea, and the aqueous humor
GENERAL PHYSIOLOGY OF THE EYE.
11
has no effect because its index is the same as that of the cor-
nea. At the posterior surface of the lens another refraction occurs,
making three in all.
The following diagram represents the conditions in a simple
optical sj^stem.
Let a b represent a spherical surface separating a less refract-
ing medium from one of higher refracting power. Let n be the
centre of the curve, and draw O A through the centre and normal
to the curve at p; the line O A becomes the axis of the surface
and rays passing through this line are not deviated. All rays im-
pinging perpendicularly upon a b, like m d and m' e, will also suffer
no deviation but go to n. The point n is therefore called the nodal
point. All other rays in passing through a b will undergo refrac-
tion. If the medium on the side O be of lower index than the
medium on the side A, rays going in the direction from O to A will
be refracted toward the axis. Rays which are parallel to O A like
m
r' p[ X ^^^/" 4
Fig. 5.
c d will be refracted to a point lying upon the axis, say at F2, and
this point is called the posterior or second principal focus. If
rays pass from the side A to the side 0, and are parallel to the axis,
they will be refracted toward the axis, and will meet upon it at a
point F', which is called the anterior or first principal focus. F1 is
nearer a b than F2. The point P, where the optic axis cuts the sur-
face, is called the principal point. The anterior principal focus
F1, the posterior principal focus F2, the principal point P, and
the nodal point n, are called the cardinal points of a refractive
system.
Let us apply these principles to the human eye.
The simplest optical instrument is an eye from which the lens has
been removed. We are then presented with only one refractive sur-
face, and have to take account of its index and its curve. Rays from
c
O
12
DISEASES OF THE EYE.
a luminous point farther than twenty feet or five metres from the eye
may be considered to be parallel, because the degree of divergence
possible with the average diameter of the pupil may be discarded.
Parallel rays will be refracted by the cornea so as to unite at a focus
33.8 mm. behind it, if they fall upon its convex surface. If, however,
parallel rays are supposed to pass in the opposite direction and fall
upon the concave surface, they will unite at a point or focus 26 mm.
in front of it. The first is called the posterior principal focus, the
second is called the anterior principal focus of the cornea The
difference between these figures is exactly the length of the radius
of curve of the cornea, viz., 7.8 mm.
In the crystalline surrounded by air, we have a bi-convex lens
whose index is 1.437, and whose front surface is curved on a
radius of about 10 mm. and the back surface on a radius of 6 mm.
The principal focus is at about 50 mm., and this is the same both
for the anterior and posterior principal foci, neglecting the thick-
ness of the lens. Both for the cornea and for the crystalline sepa-
rately, it is easy to determine by simple construction or by simple
formulae the place and size of the image when the distance and size
of the object is known. The curves are assumed to be spherical
and the angles so small that they may be measured by the arc as
well as by the sine without appreciable error. The optical centre
for the cornea is the centre of curve, and this may be called its
nodal point. The optical centre for the crystalline, if both its sur-
faces had the same curve, would be midway between the extremities
of its two radii, that is at its centre, but with unequal radii it is
a little nearer the side having the shorter radius.
If now we combine the cornea and crystalline at the distance at
which they are normally separated, we have an optical problem
much more difficult when we attempt to fix the place and size of
the image. Gauss furnished its solution in a general way and ab-
stractly, while Listing made special application of the theory to
the eye. He also simplified the calculations, and made them ap-
plicable to what he called the reduced eye, in which a single refrac-
tive surface of a particular curve is substituted for the actual con-
struction. For an explanation of the cardinal points of a compound
optical system, reference must be made to other works, especially
Landolt," The Refraction and Accommodation of the Eye/' trans-
lated by Culver, Edinburgh, 1886. We shall only enumerate them
as consisting of three pairs, viz., the anterior and posterior princi-
pal foci, the anterior and posterior principal points, and the anterior
and posterior nodal points. The first two are widely separated
the one being in front of the cornea and the other at the fovea cen-
tralis retina ; the second pair are close together and situated be-
tween the cornea and lens ; the third pair are also close together
and lie just behind the lens. The nodal points are usually spoken
GENERAL PHYSIOLOGY OF THE EYE.
13
of as a single point, the posterior one being chosen. It is the opti-
cal centre where rays cross, and its position determines the size of
retinal images and the refractive quality of the eye. The subjoined
diagram from Landolt indicates the position of all these points
upon a schematic eye three times magnified.
The measurements are thus tabulated:
Fig. 6.
Distance from the apex of the cornea A of the an-
terior or first principal focus
', anterior focus ; "
posterior focus; H' H", principal points ; K' K", nodal
points; JV/, centre of motion; C, centre of cornea; BB,
base of cornea; EL, major axis of the corneal ellip-
soid; F, fovea centralis; O. point of fixation; K' O,
line of vision; M O, line of fixation; O X E, angle
alpha; O M A, angle gamma.
1 Archives of Ophthalmology (Knapp), vol. xii., p. l 1883
GENERAL PHYSIOLOGY OF THE EYE.
15
ual line. Where this intersects at X the axis of the cornea, is
found the angle alpha which represents the displacement of the
cornea from the visual line. Usually this is outward and then is
called plus; sometimes it is inward and is then called minus, or it
may not exist. In extreme cases it amounts to 12° on the horizon-
tal plane, usually not more than 5°, and it may also deviate slightly
in a vertical sense.
In the crystalline we meet with another special feature, that its
density and refractive index increase from the surface to the cen-
tre. By its arrangement in layers its refractive power is increased.
Neither is the lens alike in all its sectors, and for this reason the
image of a luminous point like a star, is never a mathematical point,
but has irregular radiations, and to each individual the stars have
dissimilar radiations.
Such are some of the irregularities of the optical structure of
the eye. It becomes the equivalent of a lens whose focal length is
15.5 mm. or -| of an inch. This is most simply expressed by the
diagrammatic eye of Donders in which the cornea has a radius of
5 mm.; the optic axis is 20 mm.; the nodal point is 5 mm. from the
cornea and 15 mm. from the retina; while the anterior principal
focus is 15 mm. in front of the cornea.
By this model it is easy to reckon the size of images on the
retina, and the results are approximately true. It is only necessary
to divide the distance of the object in millimetres by 15 to show
how much smaller is the retinal image than the object.1 A rod one
metre in height placed at 15 metres distance (15,000 mm.) gives a
retinal image 1,000 times smaller than itself and therefore 1 mm.
in size. An object 10 mm. high at 300 mm., about 12 inches from
the eye, gives an image one twentieth of its size, viz., | mm.
In the example last cited we have introduced another element
in the problem of refraction; we have brought the object near the
eye. When this is done, the image no longer falls at the same dis-
tance from the cornea, but retires to a point farther behind, in ac-
cordance with the law of conjugate foci or reciprocal relation
between object and image in all lenses. The nearer the object the
more divergent become the rays, and the degree of divergence in-
creases very rapidly as the object approaches, because measured by
angular increase. The retina cannot retire and if no modification
is made, either by increasing the convexity of the crystalline or by
advancing its position farther from the retina, there can be no
sharp and clear picture. Each luminous point of the object
will not be represented by a point on the retina, but by a circle.
The circles of dispersion often exhibit colors, chiefly blue and red,
1 See Donders1 "Accommodation and Refraction of the Eye," p. 178.
16
DISEASES OF THE EYE.
by breaking the light into its component elements. To obviate this
difficulty, the eye is provided with a means of adjustment or ac-
commodation when objects are presented near it.
Accommodation.
If in a dark room a light be held in front of the eye, its image
will be reflected from the surfaces of the cornea and of the crystal-
line. That from the cornea though small is conspicuous, those from
the lens are best seen with dilated pupil and when the light is held
at one side. Purkinje called attention to them; Helmholtzutilized
them to determine what happens when the eye adjusts itself to
view a near object. He measured their size and the changes of
ft ft
a b c a b c
a. eye in repose. b. eye during accommodation.
Fig. 8.
In A and B the light comes through square openings in a disc.
a. Image from the cornea (erect).
b. Image from the anterior surface of the lens.
c. Image from the posterior surface of the lens (inverted).
form and place which they undergo, by an instrument which he in-
vented for this purpose—the ophthalmometer.
We may sum up the changes in the eye during accommodation
as follows: the pupil becomes smaller, the front of the lens becomes
more convex, and, by advancing a little, carries with it the iris and
reduces the distance between it and the cornea; the posterior sur-
face of the lens becomes inappreciably more convex. The lens is
thus increased in thickness and its equatorial diameter lessened.
The ciliary processes swell. These changes of the ciliary processes
have been proved by inspecting the eyes of albinoes, and eyes in
which iridectomy has been performed (Coccius, Becker, Hiort).
The active agent is the ciliary muscle, and the mode of its action
is believed to be as follows: the muscle contracts, becomes thicker
and presses the ciliary processes nearer the optic axis, and enlarges
their volume. At the same time the fibres draw upon the zonula
(suspensory ligament of the lens) and release the crystalline from
the tension under which by its anatomical construction like
h'ENERAL PHYSIOLOGY OF THE EYE.
17
bundle of watch springs, it is kept, and it, by its elasticity, increases
its anterior convexity while its border grows more rounded. The
distance between lens border and ciliary processes is not altered,
neither does the lens increase in volume. The aqueous humor be-
comes slightly displaced toward the periphery of the chamber, and
the pupil diminishes. The actual increase of the axis of the lens, in
accommodating from infinity to five inches, is 0.4 mm. The radius
of the anterior surface of the lens is shortened from 10 mm. to 6
mm., the radius of the posterior surface from 6 mm. to 5-J mm.
The lens increases from a central thickness of 3.6 mm. to 4 mm. (see
Mauthner: " Vorlesungen," 1872, p. 20). The
changes are shown in Fig. 9, copied from
Landolt.
The nerves which preside over the action
of the ciliary muscle and the pupil are the
ciliary, of which some come from the ciliary
or lenticular ganglion, and. others from the
large ciliary nerves. For further remarks
see pupil, p. 432.
What is the extent and course of the c
accommodation ?
We know that in early life the degree of
accommodation is highest, and that it stead-
ily diminishes. Donders, to whom we owe
most of our knowledge on this subject,
showed that if at ten years of age the nearest
point of distinct vision is at 2.8 inches, at
twent3T it has receded to 3.9 inches, at thirty
to 5.7 inches, at fifty to 16 inches.
At first thought, the diminution which occurs at the age of thirty
does not seem important. We know that lenses are, in respect to
their power, to each other inversely as their focal length. A lens
of 4 inches focus is to one of 12 inches focus as i is to ^. The former
is three times as strong as the latter, and the difference between
them is \-^=\, that is, it equals a lens of 6 inches focus. Now, in
comparing the accommodation at ten years of age with that pres-
ent at thirty, we are to use the formula zj — &r = sj- In other
words, by thirty years of age the eye has lost one-half its power of
accommodation, at fifty years we have u - -5- = §t, which is a loss
of almost TV its original accommodative power.
The nearest point to which the eye can adjust itself is called
the near-point of accommodation, denoted by the symbol P (punc-
tumproximum). The farthest point of accommodation is denoted
by the symbol R (punctual remotum), or far-point. The breadth
or range' of accommodation is expressed by the formula, 7-^ and
18
DISEASES OF THE EYE.
is equivalent to the difference in refractive power of lenses whose
principal foci are P and R. The range of accommodation be-
comes, therefore, a lens of definite focus, whose refractive power
is expressed by f. Now, in normal eyes, up to about fifty-five years
of age, R is at an infinite distance, and the refraction is denoted by
l-i, that is, it equals the near-point. But, beyond this age, the
far-point goes still farther away than infinity, an expression not
absurd in mathematical language, and which means that the eye
can now bring to a focus rays which are slightly convergent, and,
as light from natural objects never travels in converging lines, a
convex lens is needful to enable the eye perfectly to see distant ob-
jects. The course of the accommodation is given in the subjoined
table, constructed by Donders, and taken from Nagel (G. und S.,
Bd. VI., p. 46G), and is given both in metres and in English inches:
Distance of Distance of Breadth of A.
Age in Distance of P i;i Metres. Distance of R in Bletres. P in English Inches. R in English Inches.
Years. Metres D. Inches.
10 0.071 oo 2.8 00 14 D 1: 2.8
15 0.083 oo 3.32 00 15. 1 2.3
20 0.100 00 4. oo 10. 1 4.
25 0 128 oo 5.1 00 8.5 1 5.1
30 0.143 oo 5.7 oo 7. 1 5.7
35 0.182 oo 7.2 oo 5.5 1 7.2
40 0.222 oo 8.88 00 4.5 1 8.8
45 0.286 oo 11.44 00 3.5 1 11.44
50 0.400 oo 16. 00 2.5 1 16.
55 0.666 —4. (H0.25) 26.64 —160. 1.75 1 41.
60 2. —2. (H0.5) 80. — 80. 1. 1 40.
65 —4. —1.33 (H 0.75) —160. — 57. 0.5 1 80.
70 —1. —0.8 (H 1.25) — 40. — 32. 0.25 1 160.
75 —0.571 —0.57 L (H 1.75) — 25. — 23. 0. 1 0.
80 —0.4 —0.4 (H 2.5) — 16. — 16. 0. 1 0.
Another and familiar way of exhibiting the variations of accom-
modation with age is by a diagram first employed by Donders and
reduced to dioptries by Landolt.
The above measurements relate to the accommodation of one
eye by itself; they are not strictly true when both eyes, working
simultaneously, are considered. The binocular accommodation is
rather less than the monocular. In binocular sight the visual lines
converge upon the object, and a suitable amount of A is exerted
according to the distance of the object. There is, therefore a re-
lation between convergence of visual lines and A. This relationship
is of great importance in dealing with objects near the eye, and we
speak of it as the relative accommodation. For a given angle of
convergence it is possible for the eyes to put forth a greater and also
a less degree of A than the distance of the object requires. We
illustrate by a diagram, Fig. 9, in which, upon the line AB, the
visual lines converge at a point O, which is at the same time the
GENERAL PHYSIOLOGY OF THE EYE.
19
place for which the eyes are accommodated. While the visual lines
remain at the same angle of inclination, it is possible to see O cor-
rectly when it is viewed either through a convex glass, which will
by so much diminish the effort of accommodation and place it vir-
tually at A, or through a concave glass, which will compel greater
effort of A, and make the object seem to be at C. If, with a person
fifteen years old, O be taken at 12", then a convex glass about T1r
can be used, which will carry the accommodation to 72 inches, while
a concave glass, viz., about |, will be accepted, which will bring the
Fig. 10.
accommodation to 5.33" (Donders). The former, found by the con-
vex glass, gives the negative side, and the latter, found by the con-
cave glass, gives the positive side of the relative A. With parallel
visual lines, concave glasses — TV can be overcome, which bring the
object to 11 inches. But if convergence be at 4", concave glasses
can no longer be used; only convex can be employed, and therefore
the relative A is entirely negative. The practical result of these
■ investigations is that for a given amount of convergence there
i must be a certain ratio of positive A to negative A, else the eyes
? soon grow weary. Graefe said that the positive side must be about
20
DISEASES OF THE EYE.
equal to the negative, but age and the refractive quality of the eye
make important differences. In later life when the accommodation
has become much restricted, its reserve part very greatly diminishes
and without any discomfort to the individual. While some reserve
of relative A must remain experience shows that in this matter there
is great diversity among individuals. Peculiarities of refraction
and of muscular capacity have great influence. This subject will be
again referred to when treating of muscular asthenopia.
Functions of the Retina.
The retina is made up of nerve-elements of peculiar structure, of
the fibres coming to it from the optic nerve, of epithelium, and of con-
nective tissue. The only elements we now need to consider are the
bacilli or rods and cones. They
are upon the outer surface of the
retina, next the epithelium, and
may be likened to the pile of
velvet, because they stand per-
pendicularly to its surface. At
the fovea centralis they are
most numerous and elongated,
the cones alone existing here.
The minute structure of the
retina at the fovea centralis is
shown in Fig. 11, which is taken
from Schultze's schematic sec-
tion, given in Strieker. The
fibres of the optic nerve are the
innermost of the nerve ele-
ments ; thickest around the
disc, at the fovea thej^ are thin-
nest. They convey to the brain
the impressions excited, and are
themselves not capable of being
stimulated by light. The blind
spot (Mariotte) in each visual
field is thus explained. If, with
the right eye, one look at the cross in Fig. 12 (from Helmholtz)
placed at about twelve inches distance, the circular white spot
will correspond to the size of the vacancy in the field of most
persons. The cross is above the level of the centre of the
circle, because the fovea centralis is lower than the middle of the
optic disc.
Fig. 11.
GENERAL PHYSIOLOGY OF THE EYE.
21
All parts of the retina up to the ora serrata are capable of per-
ceiving light. The impressible surface is not quite a hemisphere,
and if it stood out beyond the obstruction of the nose and the other
surroundings of the eye, it would include within its scope a corre-
sponding exterior hemisphere, or field of vision. The space which
the eye at rest can cover in vision, varies according to the facial
peculiarities of each person and the prominence of the eye from the
orbit; we shall return to the subject of the visual field farther on.
The middle of the retina is its most sensitive part, and when we give
attention to an object, i.e., look at it, we turn the eye so that the
object shall be imaged on the fovea centralis.
We have already learned the size of the image for a given dis-
tance of an object; we have now to inquire what is the smallest im-
age which the retina is able to distinguish: in other words what is
the normal acuity of vision? The problem must be taken on its
Fig. 12.
practical side, and it does not mean the perception of a point of light
like a star. We have three properties in the retina, the form sense or
acuity of vision, the color sense, and the light sense. Naturally the
last is presupposed in both the others, yet sometimes it demands
separate investigation. The determination of the form sense or
acuity of vision has been studied by Helmholtz, Aubert and others
as a problem of physiology.
It is found that the smallest perceptible angle is one wThich forms
an image covering two cones. For accurate vision the image must
reach the outer extremity of the cones. The thickness of the retina
at the fovea is .15 mm. The smallest visual angle for black lines
on a white surface is taken by Aubert at 52". This corresponds to an
image about .004 mm. long. Snellen, who first worked out the ques-
tion of visual acuity in a practical way, has taken the angle of V
(one minute) as the average degree of visual power. He has given
the formula V = p to represent it. V stands for visus; d for the
distance at which the object is placed, and D is the distance at which
it ought to be seen and which is placed above the type. He has
22
DISEASES OF THE EYE.
constructed test types with letters wiiose stroke shall at 6 metres
subtend an angle of V, and the entire letter, supposed to be contained
in a square, subtends an angle of 5'. The first editions of his test
types were constructed upon the scale of Paris feet, the recent edi-
tions are according to the metric scale, and for 20' we now take 6
metres. In the last editions the numbers formerly known as xx
and xxx have been changed for smaller letters to correspond to the
reduced distance, because 20 Paris feet = 6.5 metres.
The separation between the letters has an important influence.
In the English edition of Snellen, the interspaces are three fifths
the height of the letters. White letters upon a black surface seem
larger by irradiation. It appears that differences of illumination
have less influence upon eyes with normal acuity than upon those
with reduced acuity.
An important consideration is the age of the subject. Cohn found the
children in a village school among the mountains had, when tested by Snel-
len's " hooks " (which are the characters to take the place of letters for those
who do not know the alphabet) v = 2 in 47$ (114 eyes); v = 2 to \\ in 34$ (85
eyes); v = \\ to 1 in 15.5# (38 eyes); v = 1 in 2.8$ (7 eyes). Total of 244 eyes.
In old age the opposite condition usually appears. Cohn, in 1874 (Nagel's
Jahresbericht, Funfter Jahrgang, p. 210), examined 100 persons between 60
and 84 years, dwellers among the mountains. He found 88 eyes with v better
than 1 and 34 eyes with v = 1. It is commonly said that v = \ is normal
after 60. Among savages vision is always high. Occupation and mode of
life also have an influence. Those who are occupied upon near objects and
who do not live much in view of objects at a great distance usually have
lower acuity.
It would appear from Seggert's examinations that persons in health be-
tween 20 and 25 years, who have v greater than 1, have as an average v=^^
(I.e., p. 98) and the visual angle is therefore 4' 14". This conclusion is after
examination of 2,253 eyes. Another point to be mentioned is that binocular
is better than monocular vision. Common experience testifies to this fact;
while Seggert has shown that if one eye is inferior to the other, vision will
in 85$ be equal to that of the better eye. Reference will be made to this
hereafter, under the head of anisometropia.
Snellen's types are the usually accepted standard, and they may
be employed with the qualifications which have been set forth.
Monoyer has given a table in which the series is made to progress-
in tenths, from 1 to 0.1. This system is to be preferred over all others
because of the regularity and smallness of the intervals between
the sizes of types. (See p. 33.)
Many other test types have been published; in some the visual angle is
taken at 4'. In some the more difficult letters of Snellen are left out T)
John Green, of St. Louis, has issued a table in which he adopts a plainer form
of letter, Gothic rather than Egyptian, and he fills out gaps in the series of
Snellen's types by making the series more correctly progressive in an arith-
metical series. The spacing of his letters makes them more difficult to deci-
pher than the English edition^ Snellen. For ignorant persons Snellen r -0"
GENERAL PHYSIOLOGY OF THE EYE.
23
vided characters which the patient is to describe by telling which side of
the figure is open and whither it points. Burchardt has a set of tests
composed of dots arranged in groups, and of different sizes, and he takes the
distance much greater than Snellen. His tests are for use in the military
service and while he sets acuity higher than Snellen, it is not as easy to use
his tests as Snellen's. Seggel, after careful comparison of the two on a large
scale, gives Snellen's the preference (I.e., p. 84). Mauthner says that acuity
which with Snellen may be 1, will with Burchardt be 1£.
In testing a patient, he is placed at six metres from the card, or
if this distance is not available, at the greatest distance possible.
(Pfliiger has issued a set of letters which can be viewed by reflection
in a mirror and so gain distance). The series extends from X to
CC. If vision is better than 1 or | or H it will be perhaps ^ or 4-
On the other hand it may be less than 1 and be ^k, jz or 4 (met-
ric). If less than the last-mentioned amount, the card may be
brought to the patient and the vision given accordingly, say 4 or
if in feet ^-0 or less. The rule should always be observed to place
the actual distance of the test types as the numerator of the frac-
tion. When V is very low, say | (in feet) it is often impossible to
decide between ^ and ^ and the same uncertainty may appear
with V = £^. It is in this class of cases, that illumination has
most influence. To this must be added the influence of the size of
the pupil; because persons with low V, see with dispersion circles,
and the smaller the pupil the smaller the circles of dispersion. It
is surprising to note what sharpness of sight, or rather what "dis-
cerning power," as Nagel calls it, will sometimes be found in persons
who have slight opacities of the cornea, incipient cataract, astigma-
tism, etc. Although the retinalimage is very badly outlined, they
are able to draw inferences as to form and features which persons
who rely chiefly on the accuracy of retinal images cannot in any
degree compete with. Such persons show decidedly much better
vision for near, than for distant objects. Hence, the little value
which attaches to examinations by reading fine print. Power of
accommodation, size of the pupil and skill in deciphering obscure
characters, make such examinations untrustworthy as measures of
visual acuity; although for the patients they have great practical
importance and consolation. Certain letters are well known to be
more easily recognized than others. Cattell experimented with a
special apparatus on this matter. He viewed the letters for very
brief periods of time in uniform light, and made 270 exposures of
each letter. They were of the plainest form and the stroke of uni-
form thickness. The order of legibility he found to be W Z M D
HKNXAYOGLQISCTRPBVFTJJE. W was seen
241 times correctly; K N X A Y between 180 and 150 times; B V
F U J about 100 times; E was recognized only 63 times out of the
24 DISEASES OF THE EYE.
270. In the small letters the order of legibility was d k m q h b
pwuljtvzrofnaxyeigcs.
He found that, to read a letter, light must act upon the retina
from .001 to .0017 of a second, varying greatly in different individ-
uals and in the same individual at different times. " The Inertia of
the Eye and the Brain," Cattell, Brain, vol. 8, p. 294, 1885-86.
It is understood that Snellen's types are to be viewed by good
ordinary daylight. On cloudy days the visual power is naturally
less. To avoid this inaccuracy, Dr. H. Derby proposed that the test
should always be by artificial light. In all cases where the light
is not normal, the observer, if his own vision be good, should com-
pare the patient's vision with his own. That a much smaller visual
angle is attained by some persons and that a very bright light,
like direct sun, greatly increases acuity, is well understood.
The value of illumination in affecting vision has been elaborately studied
by Posch (Arch, of Oph. and Otol., v. III. and IV., p. 295, 1876), who form-
ulates the law that under a given degree of illumination and with one
which is 16 times greater, acuteness of vision increases in arithmetical
progression, while illumination progresses geometrically; such a ratio of in-
crease is observed, if the light be neither very feeble nor very intense.
Seggel (Graefe's Archiv f. Ophth., Bd. XXX., II., 69, 1884, has given an
analysis of his visual examinations in the German army, not employing pho-
tometric methods like Posch, but noting the differences between bright
and rainy days. His examinations were made in a barrack with windows to
the north. He found that between bright sky and rainy weather, eyes which
had v = —, or better, would show a difference represented by 6: 5. If, how-
ever, in good light v = ^ the difference would be as 4: 3. For such as had
v = |- the difference would be as 3: 2 (I.e., p. 87).
Visual Field—Indirect Vision.
Let us now study the functions of the eccentric parts of the
retina. The first fact which wTe notice is, that outside of the fovea,
acuity declines very rapidly. For instance, at 1° outside of it, acuity
of vision is reduced to $; at 2° or 3° V = | (Konigshofer). If the
fingers be spread widely, they can be counted at almost the outer
limit of the field of vision. But, for the peripheral parts of the
retina, we confine our examination to the recognition of form with-
out attempting to ascertain discriminating power. This investiga-
tion is called taking the field of vision. It is done for each eye alone
the other being covered. To do this properly, an arc of a circle
must be placed in front of the patient, which shall be not less than
90° nor more than 180° in extent. Its radius should be about 12
inches or 30 cm. The eye to be examined must be at the centre of
the circle, and fixed steadfastly upon the point directly in front
An object, the size of which will be chosen according to the accuracy
GENERAL PHYSIOLOGY OF THE EYE.
25
demanded—generally a white object, 1 inch square, is suitable—will
then be moved along the arc from its centre to its extremity, or bet-
ter, vice versa. When the perception has been determined with the
urc in one meridian, it must be turned to another, until the whole
field has been explored. Beginning with the arc in the horizontal
position, it will be carried around to the vertical position, and a de-
termination made for each meridian at intervals of 15° or 30°. The
examination is easily made by an instrument called a perimeter, in-
vented by Aubert and put in practical form by Forster. Many
others have been made, in some of which a true hemisphere is used
(Schirk, Dyer), or a quadrant is employed. In some, a diagram of
the field is automatically traced
(Stevens, McHardy), while a
simple form consisting of a
quadrant, the invention of
Priestley Smith (see Fig. 13),
has a place in front of the ob-
server for inserting a blank on
which a diagram of the field can
be quickly and easily pricked
off. This instrument, which has
been imitated by Meyrowitz, is
the most convenient of all.
In the absence of instru-
ments, it is easy to take the
field upon a flat surface for a
distance 45° each way from the
line of vision, because the tan-
gent of 45° is equal to radius.
If the eye be put at 15 inches
from the wall, a circle on the
wall with a radius of 15 inches
gives 45°. If the space be divided into three equal zones, the first
circle will be at 18° and the second at 33°. On such a surface,
scotomata may be ascertained, but it is none the less important to
investigate the peripheral parts of the field.
The outlines of the visual field are far from symmetrical. Its
greatest extent is on the temporal side. On the opposite side the
limit is determined by the height of the nose, while above, the eye-
brow, and below, the cheek, fix the extent of its boundaries. The
position of the eye in the orbit, the configuration of the face, the
size of the pupil, and the length of the optical axis, are factors
which enter into the form of the field. Usually the extent on the
temporal side is 90°; on the nasal side, 50°. Above it is 50°, and
below it is 65°. These figures are liable to great variations in dif-
ferent persons. To be sure that the full limit belonging to each
26
DISEASES OF THE EYE.
case is secured, the observer may sight across the arc from extreme
positions on the outer, inner, or upper sides, and note upon it the
place across which he is able to catch a view of the patient's pupil.
This marks the limit to which the field ought to extend, and
should be noted on the chart as the boundary to which the field
should correspond.
The numbering of the meridians has not yet been uniformly
agreed upon. By some, including Priestley Smith, the top of the
vertical meridian is the starting point, and 180° are counted each
way, the temporal side called plus and the nasal side minus. Others
begin at the left (Forster) and go around 360°, 90° being at the top.
The difference is not very important. We must always remember
that the temporal sides of the fields correspond to the crossed fibres
of the tractus optici, and the nasal sides to the uncrossed fibres.
Moreover that the right halves of the respective fields belong to the
left optic nerve and vice versa. At least three meridians should
be taken in each quadrant. To get the field on the nasal side to its
absolute limit, the eye may be turned to fix on a spot 30° to the
temporal side. In glaucoma simplex this suggestion has value..
Sometimes the light must be greatly reduced to discover either
limited defects within, or encroachments upon the periphery of the
field. Another device sometimes helpful, is to make the patient face
the window, and the glare of the light will sometimes bring out a
limitation which would not occur in a normal eye. A perimeter is
not needed to make out hemianopsia, such as happens from brain
disease, and the same is often true of cases of detachment of the
retina. The hand moved from point to point as the patient looks
in the observer's face will discover the defect.
A small and portable perimeter has lately been made by Schweigger which.
is convenient for use at patients' homes.
Measurement of the blind spot can be made by using a small
bright test object with the perimeter. It is increased in some cases
of myopia and of papillitis. It varies normally from 4° to 7° 30'.
The measurement of the angle of converging strabismus can also
be made with the perimeter by sighting across it to the eye which
does not fix.
The chief reason for the great reduction of visual acuity outside the fovea is
the inferior sensibility of the retina. For objects very peripherically situated
an additional reason would be anticipated in the distortion which images
undergo when rays fall at very oblique angles. Fick, however has shown
("Handbuchder Physiologic" Hermann, p. 80), that the position of the crys
talline and its laminated structure very largely antagonize this source of
error, and that the eye is eminently periscopic. That there is not absolute
accuracy may be seen in emmetropic eyes, which are always hypermetronic
on very oblique axes. p
GENERAL PHYSIOLOGY OF THE EYE. 27
Color Sense.
Examinations of the color sense are called for in two classes
of cases. First in those whose defect is caused by disease, and
second in those in whom it is congenital. The two methods are
unlike, and the conditions existent are also unlike. The pathologi-
cal cases simply present modifications of the normal kind of percep-
tion, and there maybe either a central region or a peripheral region
deficient. The other class of cases, viz., those which are congenital,
will be separately considered.
The capacity for recognizing colors of every hue, pertains only to
Fig. 14.
the middle region of the retina. Much depends, however, on the
purity and luminosity of the color as well as on the degree of light.
Very large and luminous colored surfaces can be recognized at the
extreme limits of the field, as for instance a red house in sunlight;
but we are dealing with small objects. The colors employed are
blue, yellow, red, and green, and we use bits of card of from 1
cm. to 4 cm. square for the field; while for central perception
we reduce the size to 2 mm. square. There is a natural boundary
of the area over which each color can be recognized, and this
varies according to the color. Landolt has given the chart, Fig.
14, which maps the limits of the colors named. While within the
:28
DISEASES OF THE EYE.
boundaries of green, all other tints can be known, and for the red,
also yellow and blue; and for the space within yellow, also the
blue; outside of yellow,only blue can be seen (Fig. 13); and outside
of blue, no color is recognizable. These color limits cannot be
held to be uniform even among normal eyes, but they have value
as a general statement.
In making the test, a bit of card-board, 2 cm. square, of the
proper tint, is put on the end of a rod and brought across the
field, always beginning from the end of the perimetric arc. It is
well to have different colors on its opposite sides, that, by turning
the card around occasionally, we may guard against mistake or
deception. One may have several rods, each having cards of differ-
ent color, and thus be more sure of correct answers.
It happens that patients may have a dimness of color-sense for
a certain hue all over the field. This will be discovered by finding
out that the fainter shades of a special color fail to be correctly
noted. But it usually happens that more than one color is dimly
discerned. It also happens that there may be a scotoma in the
field of a particular color, as for example, red. For its detection a
small card not more than two millimetres square is to be held at
the centre of fixation, and a similar one a few degrees to one side
•of it. The one at the middle ought to be equally brilliant with the
other, while in the cases designated the eccentric card seems to
have the brighter hue. The extent and boundary of the scotoma
«an, by this means, be made out, provided the test be not continued
for a period so protracted as to fatigue the color-sense. This ex-
amination belongs to cases of tobacco or alcoholic amblyopia, etc.
We find defective color sense in atrophy of the optic nerve, in
chronic glaucoma, sometimes after apoplexy and injuries of the head,
in amblyopia and amaurosis, and also in hysterical amblyopia. As a
measure of direct color perception, Oliver1 states that at five metres
one should recognize a red card of 3 mm. square, a green of 2 mm.,
yellow of 2.5 mm., and blue of 8 mm.
Congenital defect of color sense, or Daltonism, has attracted
great attention within eighteen years, and the literature of the sub-
ject is copious. For proper understanding of signals on railways
and ships, and in some other cases, defect in color perception
produces liability to mistakes whose consequences may be suf-
ficiently serious. That accidents have not occurred more fre-
quently wlien color-blind persons have beeu responsible for the in-
terpretation of signals, is due to their possessing in most instances
very acute perception of shades and degrees of light. They are
often unaware of their defect. The condition is frequently hered-
itary. Horner gives a case of transmission from grandfather to
1 Archives -of 'Ophthalmology, vol. xi., p. 65, Am. Ed.
GENERAL PHYSIOLOGY OF THE EYE. 29r
grandson during eight generations. Both eyes are affected; yet
six cases are on record where only one was concerned. The actual
frequency of color blindness is on the average 5# among men and
less than ty among women. The common error is inability to
distinguish red from green, while other colors are appreciated.
There are differences among these persons; some confound light
red with dark green, and these are red blind, while others confound
dark red with light green, and they are green blind. A much
smaller class of persons have "blue-yellow" or violet blindness.
Very searching and interesting studies have been made by Don-
ders, Koenig,1 Hirschberg and others upon the vision of these per-
sons with the solar spectrum. In place of red and green bands,,
they see only yellow of varying intensity, and recognize very
subtle shadings of light. With some, the spectrum is shortened
at the red end, with others it is not.
When candidates for employment in the service of a railway
are to be tested, the method is by the worsteds of Holmgren, which
are of all hues and tied in skeins. A mass of them, about 100, are
thrown upon a white cloth and the person examined is asked to pick
out tints which resemble the hue of certain standard test skeins.
The first test is a skein of light green, the second test is a skein of
light purple or rose, which is the complement of the green. A third
test which is only confirmatory and suitable for persons whose de-
fect is extreme, is a scarlet or brilliant red. The standard green is
put into the person's hand, and with correct perceptions he has no
difficulty in matching it with congruous colors. But if his color
sense is defective, he will be guided not by the tint, but by the lumi-
nosity of the shades. Blues and yellows he understands and avoids;.
but among tints of a greenish hue he will take up grays, light
browns, yellows, and skeins of tan and dove color, " confusion colors."
He will hesitate over uncertain colors and when he has picked out
all which he regards as similar to the green, this bunch will be put
aside and he will proceed in the same way with the second test,
the light purple or rose. In this hue red and blue are mixed, the
red predominating. The first test shows that the color defect is of,
the red-green variety; the second test will decide whether the defi-
ciency is greater with red or with green. The red-blind individual
chooses out the light reds and grays and greens, and avoids the
dark. The green-blind selects the dark and avoids the light reds
and violets. If with the second test only purple skeins are selected,
the person is only partially color-blind for red and green. In case,
besides purples, he picks out only blue and violet or one of themr
he is completely red-blind. If with purple he selects only green and
gray or one of them, he is completely green-blind.
1 Graefe's Archiv, xxx., 11, 154.
30
DISEASES OF THE EYE.
The third test applies to those who are totally red-and-green
blind and is confirmatory of the second.
A method similar to the above has been devised by Dr. Wm.
Thomson, of Philadelphia, in which the skeins are arranged upon a
frame and numbered, and the results of the examination are re-
- corded on charts. It enables the examination to be made with facil-
ity by persons who have no expert knowledge, and the results can
be sent to a central bureau.1
If the very rare case of violet or blue-yellow blindness should
appear, purple, red and orange will be confused in the second test.
Total color-blindness will be recognized by a confusion of all shades
having the same intensity of light, and is also rare.
Stilling has published isochromatic tables for detecting color-
blindness. Colored letters made up of small blocks are printed on
a ground of confusion color, and they consequently cannot be made
out by the color blind. The edition of 1880 is the best.
Donders used colored glass viewed by transmitted light. Woinow
employed Maxwell's revolving discs; the method by colored shadows,
and other methods have been used. Holmgren's worsteds are on
the whole the most satisfactory. For elaborate details on this
topic see Jeffries2 and Burnett.3
Very recently Prof. Langley (American Journal of Science, xxxvi., Nov.,
1888) has published the results of experiments upon the perception of colors
in normal eyes. He found among four persons, all of whom had supposably
normal perceptions, extraordinary differences; two were myopic and their
ability far exceeded the others. His general conclusion is that, apart from
individual peculiarities, the time required for the distinct perception of a very
faint light is about one-half second. The visual effect produced by any given
-amount of (luminous) energy varies enormously according to the color of the
light in question. For details see his paper and see also the article by Cat-
tell in Brain, vol. 8, p. 294, 1885-86.
Light Sense.
In case of an occluded pupil, and of cataract, we manifestly
are called upon to examine the degree of light perception. But
dn disease of the retina and optic nerve, and in cases of ambly-
opia it may also be important. Retinitis pigmentosa, hemeralopia,
and nyctalopia are instances in point; and so is detachment of
the retina. Opacity of the media cannot quench the light sense
with a normal retina, nor can closing the lids do it.
A rough mode of testing is by passing over the eye the shadow
of the hand or of the outspread fingers, or by throwing upon it the
light from the ophthalmoscope removed to a considerable distance.
1 Trans. Am. Oph. Soc, xvi. meeting, 1880, p. 142.
2 "Color-Blindness," Boston, 1880. 3 Archiv for Ophthal., vol. x., p. 1
GENERAL PHYSIOLOGY OF THE EYE. 31
In the latter method the patient is asked to state from what
direction the light comes, which gives the projecting power,
likewise.
If an isolated spot of the retina is deficient in light perception
we call it a scotoma, and if this be absolute, i. e., for white light, it
is known as a positive scotoma; if it refer only to the perception,
of colored light, as, for example, red, it is called a negative scotoma.
The former implies reduced or absent form sense, the latter may be
compatible with good visual acuity.
Deficient perception at the periphery has been already referred
to under perimetry.
There is no recognized standard of normal light sense. The
best known instrument for its examination is the photometer of
Forster. It is a square box in which are placed black lines equal
to Snellen LX, at one-third of a metre from the eye. A standard
candle illuminates them and the degree of light is regulated by a
window whose size is variable. When this window is two millime-
tres square we have the normal minimum and it can be enlarged to
1500 square mm.
Other methods, as by rotating discs, and by letters printed
on gray backgrounds (Bjerrum), have been suggested, but for
clinical purposes the simple methods above mentioned usually;
suffice.
The sensitiveness of the retina may be exhausted by exposure
to very strong light, thereby producing total blindness. Leaving
•out of view direct sunlight and the electric arc light, the retina will
become fatigued by prolonged exposure to light of much less inten-
sity, and for this reason an increase in the number of gas burners
beyond a certain luminosity blunts the sensibility of the retina.
Artificial light is inferior to daylight (certain forms of electric light
•excepted) because it contains fewer blue and green rays. See
remarks on p. 24.
Subjective sensations of light may be referred to. They are
produced by pressure on the eye, by the galvanic current as it is
connected or broken, by effort of accommodation in the dark. These
appearances are called phosphenes. In certain cases of choroiditis
and retinitis subjective light phenomena, like coruscations or a dif-
fused glow are very troublesome. With widely dilated pupils (my-
driasis) a diffused red light sometimes appears. A similar blue
color is sometimes seen by patients immediately after the extrac-
tion of cataract.
The retina retains the impression of light for an appreciable
time and with some persons the duration is annoying. It varies
from 3V to ^ of a second, and gives rise to the so-called after-
32 DISEASES OF THE EYE.
images. These are in colors complementary to the quality of light
received. Bright windows of painted glass excite phantom images
of opposite colors. Rapid alternations of light and shade will excite
in the retina sensations of color (Rood)1 at first green and with more
rapid alternations, red. If we steadily gaze upon a very bright ob-
ject and then close the lids, we have for a few moments a correct pic-
ture of the object in its light and shade, a positive after-image;
soon the reverse picture appears, which is the negative after-image,
and it gradually fades. (See Leconte,2 Aubert.3)
If in a dark room we look at a lamp through a pin-hole in a card,
which we move quickly to and fro across the area of the pupil, we
shall presently behold a phantom representation of the vessels of
the retina. This phenomenon is called after the name of Purkinje,
who first described it. It is caused by the displacement of the
shadows of the retinal vessels cast upon the rods and cones as the
angle at which the rays fall upon them quickly varies. It proves
that the rods and cones are the percipient light structures; and
the amount of parallax has been calculated, and the distance of the
rods and cones from the blood-vessels deduced. The same ap-
pearance can be produced by condensing on the sclera by a 2-inch
lens the light of a lamp in a dark room, and moving it rapidly over
a small surface.
The Visual Purple.
Experiments upon living animals, made first in 1876, by Prof.
Boll, of Bologna, and subsequently pursued by Prof. Kuhne, of Hei-
delberg, have demonstrated the existence of a pigmentary sub-
stance in the retina, which is called the visual purple or visual
rose. It is a secretion from the hexagonal pigment-epithelium of
the retina.
Its properties are summed up by Dr. Ayers (in the New York
Medical Journal, May 1881, p. 582), who says, that it is an albu-
minoid compound belonging to the outer segments of the rods and
not to the cones. Its extraction requires a ten-per-cent solution of
sodium chloride, or a two-per-cent solution of gall, and other steps
which a foot-note describes. It is a photo-chemical substance, sen-
sitive to light, and in man becomes bleached to a yellow hue. In
some fishes, chiefly the deep-sea varieties, it is not changed in color
by light, but remains purple. Its secretion in animals is increased
by pilocarpine and muscarine. We know of no drugs or nerves
whose action can diminish its quantity. When a person is for a
long time kept in darkness, it becomes abundant; if bright light
1 "Modern Chromatics," N. Y., 1879. 2 " Sight," D. sTAppletonTN. Y 1882
3 Grraefe und Saemisch, v. iii. 2, p. 508.
GENERAL PHYSIOLOGY OF THE EYE. 33
be admitted the eye is painfully dazzled. On the other hand, it is
bleached by light to a yellow hue, and perhaps because yellow is
the most strongly antagonistic to chemical rays of light, the retina
is not suitably stimulated and we do not see well on first entering
a dark room. The purple seems needful to the appreciation of dim
light, and its conversion into yellow may be a defence of the retina
against the injurious influence of very bright light.
It is seen that the retina, in its chemical properties, bears out the
analogy of the eye to a photographic camera in the most surprising
and complete manner. Indeed, by confining rab-
bits in darkness for a length of time and then ex-
posing them to a bright window crossed by bars,
decapitating them in a room lighted only by a so-
dium flame, and treating the retina by a solution
of alum, and in a manner similar to the usual pro-
cesses of photography, a picture or optogram can
be developed and fixed in the retina and preserved
for future study. Such a picture is given in the diagram (Fig. 15)
copied from the New York Medical Journal, March, 1881, and taken
by Dr. Avers, who worked with Prof. Kiihne in his laboratory.
It is natural to imagine that this remarkable substance has an
important relation to sight, but we cannot define its functions,
because it is wanting in the cones which give us the best vision.
The yellow material which gives name to the macula lutea, lies in
the front retinal layers and has nothing to do with the purple. It
is impossible to discover the purple by the ophthalmoscope, and it
has nothing to do with the red color of the living fundus, as has
been proved by Becker in albinos. It has been proved also that
animals whose retina has been bleached not only can see, but can
distinguish colors (Kiihne).1
Note.—In the test series of Snellen the intervals from one line
to the next are as follows:
n • . 2J) 2^ 2_0 _2J 2JL 2JL _£0. 2_0 LI
OOI ICts . X XV XX XXX XL LL LXX C CC
Intervals: fJ-iiiViAiV
The test types of Dr. John Green have less irregular intervals,
but are by no means uniform. By the decimal system (Monoyer's)
the interval between each line is ^ and the degrees go from 0.1 to
1.0. What in Snellen's series is written as f£ becomes 0.5, and
from M to f£ we have five lines instead of three. We secure
1 " Handbuch der Physiologie," dritter Band, i. Theil —Gesichtssinn, 235
to 342,1879. See also an account by W. S. Ayers, New York Medical Journal,
May, 1881.
3
34
DISEASES OF THE EYE.
greater precision in determinations of vision, at the early stages
of its decline, and also greater accuracy in correcting errors of
refraction.
For additional detail upon the physiology of sight the reader
is referred to "Text-book of Physiology" by M. Foster; Part
IV,, 1891, MacMillan & Co., sixth edition,
CHAPTER HI.
HOW TO EXAMINE THE EYE.
A. We naturally first give heed to the external parts and we
note the lachrymal sac, and press it with the finger; the lids—
their edges, the cilia, the Meibomian follicles, lachrymal puncta,
their cutaneous and their mucous surfaces, and they are to be turned
over; the width to which the lids separate, their mobility—whether
everted or inverted or thickened; the length of the palpebral slit;
the cornea—whether transparent or blurred by opacity, its shape or
curvature; the ocular conjunctiva—its color, the appearance of its
vessels; the depth of the anterior chamber; the pupil—its size and
mobility, its clearness; are both the pupils alike ? the iris—its hue
and brilliancy—is it adherent to the cornea or to the lens ? is its
periphery retracted ? is its tissue healthy or atrophied ? do both
irides look alike? the crystalline—is it clear or smoky, or posi-
tively opaque?
Critical inspection of the cornea, iris and lens is of great impor-
tance, and while marked lesions will be obvious enough, the finer
changes in the cornea and lens demand special attention. This is
true both of the cornea and of the lens.
A good light and a fair exposure of the eye are to be secured,
but great assistance is gained by resorting to two methods which
are to be mentioned, viz. :•
1st. Oblique or focal illumination by means of a convex lens
whose focus is about two inches. This may be used in ordinary day-
light, the patient being at a little distance from the window; or, still
better, the examination may be made by gaslight in a dark room.
The lens is held about two inches from the eye, condensing the
light on one side of it while the observer looks from the other side.
The focus of the lens is made to play over the eye in all directions,
deeper and more superficially as the various parts are to be exam-
ined. The contrast between the intense light of the focus and the
shadow which surrounds it constitutes the chief advantage of this
proceeding. Caution must be used not to subject cases to this
method which are likely to suffer harm by the strong glare, but
experience will soon indicate what patients are not to be thus inves-
tigated. For slight opacities or facets and in searching for foreign
bodies upon the cornea, or for studying the iris and pupil, and for
exploring the crystalline lens and anterior portion of the vitreous
humor, oblique illumination is indispensable.
36
DISEASES OF THE EYE.
One may also use a magnifying lens in addition to the illuminat-
ing lens. Brticke's dissecting spectacles are extremely useful in giv-
ing stereoscopic vision besides magnifying power. They consist of
a pair of convex glasses of 12 inches focus combined with prisms
of about 7°, set with the bases inward.
2d. Another device of value is illumination by a small plane
mirror, reflecting a dim light. If we have no other than the mir-
ror usually made for the ophthalmoscope, which is concave and of
about seven inches fo-
cus, it must be held at
twelve or fifteen inches
from the patient's eye
to properly reduce the
light. The examina-
tion must be made in a
dark room, the lamp
placed about six inches
behind the head, so as to
leave the face in shadow.
Viewed through the
hole in the mirror, at
the distance of a foot
or more, whatever opa-
cities may exist in the cornea or in the lens are easily revealed.
By little tilting movements of the mirror the light plays over the
eye, and if opacities exist they flit like shadows across the illumi-
nated pupil. A shadow appears where, before, the surface looked
transparent, and again a clear surface comes out as the shadow
glides to another spot. The same phenomena occur when the cor-
nea has lost its natural curvature, and has become conical or bulges
in any manner. The shadows caused by irregular reflection some-
times are very striking. More will be said on this point when we
deal with diseases of the cornea and of the crystalline lens.
Examine the tension of the globe by pressure with the finger—
is it elastic, yet firm, like the normal eye, or too resisting, or softer
than normal ? Does pressure cause pain, especially if made upon
any spot of the ciliary region ? Ascertain if the cornea and con-
junctiva, if touched by a fleck of cotton, or by a hair, exhibit their
proper sensibility.
Attend to the mobility of the eye, that its range of motion is
sufficient in all directions, without tremor or spasm, or lagging"
that the two eyes move in harmony, both for near and for distant
objects, and in all directions. Whether there is apparent binocular
vision, or if strabismus exist. A patient may volunteer the state-
Fig. 16.
HOW TO EA'AMINE THE EYE.
3T
ment, that he sees double, which may or may not suggest itself by
the behavior of one or both eyes, and there will follow the suitable
method of inquiry about double images and muscular paralysis.
Only by systematic use of prisms can this topic be fully considered.
Protrusion of the globe on one or both sides will not be over-
looked, and exploration of the border of the orbit with the finger,
and especially of the foramina of exit of the supra-orbital and in-
fra-orbital nerves, will not be omitted.
In a great number of cases the examination will not go farther,
because there is some obvious malady which demands immediate
action and further inquiry is superfluous.
Sometimes to accomplish what has been proposed will be diffi-
cult, because pain or spasm of the lids or photophobia or fear,
makes the patient refuse to permit a complete investigation. Es-
pecially is this the case with burns and injuries, with cases of kera-
titis and iritis and conjunctivitis.
We may often gain our end by using a 4$ solution of muriate of
cocaine several times within twenty minutes. At first it smarts,
but soon it allays distress and photophobia and also reduces hyper-
aemia. With children we may be obliged to employ anaesthetics,
and my preference with them is for chloroform. In fact, I would
use it rather than ether in many older subjects, when a brief inspec-
tion or quick manipulation is the only requirement.
The value of anaesthetics is greatly to be insisted upon in dealing
with young children who have acute conjunctivitis and acute kera-
titis. Not only are they spared the infliction of pain, but the eye is
less likely to sustain injury. It is not necessary to give anaesthet-
ics in every examination, but ofttimes efficient treatment can be
practised in no other way. I once treated a child, aged about five
years, for granular conjunctivitis, during nine months, and gave her
chloroform about eighty times. She not only got well of the disease,
but evinced no ill effects from the chloroform, either at that time or
in her adult life. The great immunity of children from evil effects
of chloroform is well known.
B. We now give attention to the functional examination of the
eye, and to the appearances to be seen by the ophthalmoscope.
A functional examination includes, 1st, the acuity of vision, for
which we need test types, and a box of trial glasses: 2d, the field
of vision, for which we want a perimeter; 3d, the color sense and
sometimes the light sense; 4th, we inquire into the function of
the muscles as to their adduction and abduction, etc., both for par-
allel axes and during convergence for the working point, and, 5th,
we investigate the accommodation.
When examining visual acuity we are obliged to correct errors
of refraction, and to discover them we have various methods at
38
DISEASES OF THE EYE.
command. The chief of these are the box of trial glasses, the
ophthalmometer, and the ophthalmoscope. The first is to a great
degree a subjective mode of examination, and liable to various
sources of error. The second is purely objective, and if available
is invaluable within its proper sphere. The third is also largely
objective in its method, and if the observer is sufficiently expert
he will often thus detect errors which trial glasses have either
failed to discover or have reported falsely.
For any knowledge of the deeper parts, viz., those behind the
crystalline, we are wholly dependent on the ophthalmoscope. To
this we shall next give attention,
CHAPTER IV.
THE OPHTHALMOSCOPE.
The invention of the ophthalmoscope by Helmholtz in 1851,
was the result of a careful study of the conditions which ordinarily
prevent the pupil from emitting light from the eye, or wh}1- it looks
black. He demonstrated that this accorded with well-known
laws of optics, of which one is that light passing through a lens
follows the same lines both when entering and wiien returning,
in case any can return. Hence, with a pupil not larger than
four millimetres, an emergent beam is very small and must go
straight to the luminous source in a path so narrow, that an ob-
server will not be able to catch it, without screening off the light
with his head. If, as Loring has pictured, an observer look through
a tube traversing a candle flame held close to his own eye, he
can then catch light reflected from another eye. Helmholtz's de-
vice was to reflect the light by a transparent mirror, consisting of
three slips of plane glass. He set them at the angle of greatest
polarization, so as to reduce the dazzling effect of reflection from
the cornea. Ruete, in 1852, proposed a perforated metallic mirror.
All the optical principles involved were fully discussed by Helm-
holtz (see " Physiologische Optik," 1867), and it has been left to
others to improve practical details and working instruments.
The mirrors are either plane or concave. The latter usually
have a focus of about seven inches; for special cases it may be
shorter. The mirror need not be large, as Wadsworth has shown,
for the useful part is very near the sight hole. The latter should
be about 3£ to 4 mm. in diameter.
Artificial light is commonly used, while by proper arrange-
ments, sunlight can be employed.
There are two methods of examination, viz., the direct and the
indirect, or that with an upright image and that with an inverted
image.
In employing the direct method, it is obvious that the surface
commanded by the eye of the observer will be most extensive if the
mirror be as close as possible to the patient, just as we would peep
into a room through the key-hole. To view satisfactorily the fun-
dus oculi—supposing its optical structure normal or as it is called
40
DISEASES OF THE EYE.
emmetropic, and with its accommodation suspended—the observer
must put his own eye into a state for reception of parallel rays, i. e.,
look as if the object were far away, notwithstanding he knows it
is only about an inch distant. He can use but one eye ; what the
other sees must be disregarded, or it must be closed. The practical
details are as follows:
We darken the room and use a single light—an Argand gas-
burner or a student's lamp. The object to be sought for is the
optic disc, and the patient is bidden to look straight forward, while
the observer looks in from the temporal side at an angle of about
15°. For the examination of the left eye the observer's left is used,
and for the right eye the observer's right; the place of the lamp
Fig. 17.
being shifted and the instrument put into the corresponding hand;
the observer comes as close to the eye as possible, and this may be
within one inch or even within fifteen millimetres. If now the eyes
of both be normal in refraction, and in both the accommodation be
entirely at rest, the details of the eye-ground will be easily seen.
The other method, called the indirect, or by the inverted image,
is as follows: the observer holds the mirror twelve or fourteen
inches from the patient, and brings before the latter's eye, and
within two inches of it, a biconvex lens of two and one-half inches
focus. This lens condenses the light from the mirror, and also
collects the emergent light into an inverted image which lies at
about two and one-half inches from the lens, between it and the
mirror. The observer examines this aerial image, and not the eye.
It is bright, small, and covers a larger surface than is to be seen
with the indirect method, and shows better the relation of the parts.
THE OPHTHALMOSCOPE.
41
To know whereto direct the light, the observer should keep both
his eyes open, and rest the upper edge of the mirror on the inner
end of the brow. When he has thrown it on the eye, he will be
attracted and embarrassed by the reflection from the cornea. This
annoyance is greatest when the region of the macula is under in-
spection. One learns, after a time, to look beside this reflection
and ignore it. When using the indirect method, the biconvex lens
furnishes in addition two reflections of the mirror as small, round
spots, and these are gotten out of the way by giving it a slight in-
clination. In this kind of examination the corneal reflex sometimes
seems to cover the whole field. A little change in the position of
the lens or mirror will remove it.
Fig. IS.
The direct, method of examination presents fewer difficulties of
instrumentation than does the indirect method, but it offers a more
complicated problem than the other, because the refractive condi-
tion of the eye must be determined, and, if erroneous, must be cor-
rected by proper glasses before the inspection of the fundus can
take place. What in the beginning is a difficulty, becomes, after a
time, a most valuable quality of the direct method.
What is the extent of field and what the magnifying power by
these respective methods ? It is assumed that both observer and
observed have normal eyes, whose nodal point is 16.6 mm. from the
retina. With the direct method the extent of field may be thus
stated: If the observer could make his own nodal point coincide
with the antei'ior principal focus of the patient's eye, he would have
a field equal in size to the patient's pupil. This would bring the
eyes within about 7 mm. of each other, which is not feasible. At
15158
i2
DISEASES OF THE EYE.
the nearest approach, with a pupil of 4 mm. we have a field of about
2 mm. or practically what covers the diameter of the optic disc.
Another limitation is the size and form of the light employed. We
see its image on the fundus long and narrow, and the bigger it is,
the larger the illuminated space. It follows that we can view only
a very small portion of the fundus at a
time. The magnifying power will be
that of a lens about 15 mm., or j inch
focus, and the distance from the eye is
unimportant. It may be stated in terms
of angles or by a conventional linear
standard. By the former it will be 5° 43%
by the latter, taking 10 inches or 250 mm.
as the distance of distinct vision, it will
be 15 times, if we take 12 inches or 300
mm. as the standard, we have 18 times
magnifying power.
By the indirect method the extent of
field is determined by the diameter of the
objective lens and its focal length; the
latter being the more important. If this
be 2\ inches or 60 mm. focus, and the
diameter be 1^ inches or 30 mm. the ex-
tent of field will be 8 mm. diameter. This,
however, is not realized because of the
shape and size of the gas or other light.
It amounts to about 5 diameters of the
disc. Magnifying power is also deter-
mined by the focal length of the objective
lens. With one of 2£ inches, we have
about 4 times, with a lens of 3 inches we
have 5 times. That is, the weaker the
lens the greater the amplification; at the
same time the smaller the field.
Intensity of illumination is brighter
with the indirect than with the direct
method.
To be prepared for the various refrac-
tive errors which will be met, and also
to correct such as may belong to the ob-
server, a series of convex and concave glasses are placed behind the
mirror. In the early instruments these wrere few in number and the
mechanical parts simple. But as the value of the instrument in diag-
nosticating refractive errors became more fully appreciated the
number of glasses has been increased and the mechanism for them
THE OPHTHALMOSCOPE.
43
more complex. No one has cultivated this kind of improvement
more successfully than Loring, yet there are many other good de-
vices, by Couper, Nettleship, Jackson and others. A moderately com-
plete set of glasses is indispensable, even though accurate diagnosis
of refractive errors is not sought. Without them one will often be
limited to the use of the inverted image in studying the pathologi-
cal changes of the fundus. In medical ophthalmoscopy this would
be a serious hindrance to a just appreciation of changes in the optic
nerve and retina. Fig. 19 shows a simple style of instrument
Fig. 20.
with 8 concave and 7 convex lenses. Still another, by Loring (see
Fig. 20), gives 24 concave and 23 convex by interposing a quadrant
on which are strong plus and minus glasses, and the series is still
farther enlarged by using what are called half numbers.
Another arrangement has been introduced by the writer to en-
able an observer to command the series of lenses without removing
the instrument from his eye. This is done by cog-wheels playing
upon two discs which contain the lenses, and besides a full series
of spherical glasses, cylindric glasses from the spectacle box can be
inserted in a spring clip. See Fig. 21, p. 45.
44
DISEASES OF THE EYE.
A long handle is a decided convenience unless a patient is in bed,
when the ivory portion may, if needful, be unscrewed.
Another form of ophthalmoscope is the binocular (Giraud-Teu-
lon) which gives stereoscopic effect and is available only for the
inverted image. It is seldom employed. There are fixed or dem-
onstrating ophthalmoscopes, Liebreich, Cusco, etc. Burke substi-
tuted a concave mirror for the convex lens in getting the inverted
image and gains greater magnifying power.
In learning to use the ophthalmoscope, the first requirement is
that both parties lay aside all efforts of accommodation. The pa-
tient usually does this, because he has no object to inspect, and his
eye is dazzled by the glare; yet too much dependence cannot safely
be placed on this assumption, as will be dwelt upon hereafter. The
inexperienced observer never does this, but looks as he always
would at a near object, and not as he would at a distant one—in
other words, he calls in play his accommodation. To prove this
and to enable him to see, he may, when using the direct method, put
behind the mirror a concave glass of 10 inches focus. Then he will
see the bottom of the eye, just as he would read a book ten inches
away. But let him weaken this glass to 20 inches, and again he
will see; and then to 40 inches, and perhaps he still will see. His
problem is to see clearly, without any glass and with no effort.
He must cultivate this habit. Let him practise looking with a
convex glass of 8" focus before one eye at a page 7£ inches away,
or as much farther as he can read, keeping the other eye open.
He will finally find whether he can, at pleasure, utterly abando'n
accommodation, or what fraction of it he is obliged to use. What-
ever that may be, he is to allow for it as his personal equation of
error. If, however, the observer do not have normal eyes, he must
put behind the mirror the glass which corrects his sight for dis-
tance, plus or minus the glass which his habit of accommodation
compels him to employ. Then he is in position to examine abnor-
mal eyes. In doing this he will have to add to his correcting-glass,
or subtract from it, the glass which corrects the error of the pa-
tient's eye. On a later page a slight modification of this statement
will be made, and more details mentioned.
The following method of conducting an examination is suggested
as being sure to cover all the points of a case.
First, illuminate the eye with the mirror from a distance of six-
teen or eighteen inches, and let the light play from side to side over
the cornea. This will show opacities in the cornea or lens and the
degree of luminosity of the fundus. If the eye be of decidedly ab-
normal refraction or ametropic, retinal blood-vessels will be visible.
They may indicate that the eye is either near-sighted or far-sighted.
If the former, the vessels will move in a direction opposite to the
THE OPHTHALMOSCOPE.
45
Fig. 31.—The mirror, besides swinging in the trunnions, may be rotated in a circular direction and
thus assume any angle. The front disc is moved by the lowest wheel and the back disc by the upper
and exposed wheel. There is a spring clip on the back of the instrument to carry a cylindric glass. In
other particulars the instrument is copied after Dr. Loring's latest model. It gives command of a
complete set of spherical glasses, both positive and negative, amounting to seventy-six in number,
and cylindric glasses may be inserted at pleasure from the spectacle-box.
46
DISEASES OF THE EYE.
motion of the observer as he moves his head from side to side, while
for far-sightedness the vessels will move in the same direction with
the motions of the observer.
Having this preliminary idea of the state of the eye, the bicon-
vex or objective lens may then be put up for the inverted image.
The lens is held by the thumb and forefinger, while the little finger
takes a support on the edge of the temple. To find the nerve, say
in the right eye, let the patient look at the observer's left ear and
vice versa; or look straight forward while the observer moves 15°
to the temporal side. The lens is to be moved a little from side to
side, which of course carries the image with it; and it will be noticed
that parts upon deeper planes, as in the case of excavation of the
optic nerve, have a greater range of movement than do the more
superficial parts. For instance, the edge of the nerve will move
less extensively than its bottom if there be excavation. The
little finger may be allowed to press on the eye, at the same time
lifting the lid, and thereby determine whether a little increase
of tension will cause pulsation of the retinal vessels. After inspect-
ing the nerve, the patient should be directed to look in every direc-
tion, to bring all parts of the eye-ground into view. The region of
the macula will also be noted, although this will often not be well
seen unless the pupil has been dilated.
Next, the eye should be inspected by the upright image, the ob-
server coming so close to the face as even to touch it, and bringing
the light to the requisite position to permit close approach. Now,
it will be needful to put behind the mirror such glasses as neutral-
ize refractive errors, and the details of the fundus will be more fully
appreciated, besides learning what is the state of refraction. I do
not mean to be understood as intimating that the diagnosis of the
state of refraction will easily be made by the beginner—on the con-
trary, he will meet not a few difficulties; yet these may be sur-
mounted by moderate ability and perseverance.
After having studied the bottom of the eye, a strong convex
lens, say of three inches focus, may be put behind the mirror to
enable one to inspect the crystalline, the anterior part of the vitre-
ous, and the cornea, the patient being told to look in different direc-
tions, to throw into view the periphery of the lens or vitreous
opacities not in the field. It will be well to turn down the light to
get a view of very faint opacities in any of the media and one will
look from various distances, by doing which the magnifying power
will be altered. Finally, turn the patient to face the light and use
the focal illumination already described (page 36). Of course re-
gard must be had to the sensitiveness of the patient's eye and* its
liability to injury by intense light. Very rarely does any result,
and this is specially true of lesions of the optic nerve, retina and
THE OPHTHALMOSCOPE.
4?
choroid. In very many cases, only the direct method need be used,
and to the fundus as thus seen we will now call attention.
The Fundus Oculi as seen by the Ophthalmoscope, and espe-
cially by the upright image—The larger the pupil the easier and
more complete will be an examination, but one will seldom need to
use atropia. If mydriasis is required, a 4$ solution of cocaine will
usually give sufficient enlargement in 20 or 30 minutes and it
passes in a few hours. The object first sought is the optic nerve,
which appears as a circular disc on which the retinal vessels are
seen. For the recognition of the anatomy of the nerve compared
with its ophthalmoscopic picture see Fig. 22. Its color varies
from pinkish-white to deep red; often the whole surface is not
of the same hue, a part being red and the rest pale, and this may
be respectively the nasal side contrasted wTith the temporal side,
or the circumference contrasted with the centre. The whiter
parts reflect light more brilliantly because they are sunken and
concave, and the paucity of fibres in the depressed part favors
the penetration of light to, and its reflection from, the lamina
cribrosa. The depression or so-called excavation often found in the
nerve may be central and small, or in extent it may exceed half its
diameter:—it may be a slope on the temporal side, or more rarely
downward; or the outer half may be almost flat and below the level
of the inner, like a step. The nerve is sometimes a true papilla, and
the highest part may be central or on the nasal side. In all cases the
tissue is translucent, so that one looks through a depth of substance,
and the limit of inspection is the lamina cribrosa. The latter when
seen is densely white, and is often mottled with dark spots. As
the nerve-fibres come through its meshes, they lose their neuri-
lemma and become transparent axis cylinders. The nerve is some-
times oval, with its long axis vertical, and, even when truly circu-
lar, may by reason of astigmatism seem to be oval in any direction.
It sometimes has an irregular outline. The border is well defined,
being sharply cut by the edge of the choroidal aperture, and often
a black pigment deposit extends more or less about it. Sometimes
the choroidal opening is appreciably larger than that in the sclera,
and a narrow ring of the latter is to be observed. If the optic
fibres are heaped together in a certain space, they will be easily
recognized as they cross the edge of the disc and extend into the
retina, sometimes to a considerable distance. In eyes deeply pig-
mented, the optic nerve is always by contrast more red, and the
nerve-fibres are more distinct. Sometimes they make a complete
fringe or aureole of hair-like radiating lines.
The conspicuous feature of the nerve is the network of vessels
which appear upon it. They emerge and enter near its centre, and
present many varieties of arrangement and subdivision. A single
48 DISEASES OF THE EYE.
arterial trunk usually comes up from the bottom of the disc and
sends branches above and below, the veins taking a course nearly
parallel with the arteries. It would be useless to attempt to de-
scribe all the varieties which the vessels present. The diagram
from Leber (Fig. 23) gives the vessels and their nomenclature.
Besides these main branches, there are many finer twigs Avhich
pass from the nerve in the horizontal meridian, and they are most
numerous on the temporal side. The number of the vessels on the
disc is exceedingly various, and sometimes they spring forward in
large curves and take a
sinuous course, or may
even curve around each
other in complete or par-
tial spirals. Such pecu-
liarities will have relation
to the vascularity of the
general system, and due
allowance must be made.
Sometimes the walls of
the arteries are of unusual
thickness for a certain
distance beyond the disc,
and then they have a
whitish border.
At the region of the
yellow spot there are never
any large vessels, but it
will be seen that from the
transverse branches above
and below numerous small
twigs are sent down which
run almost to the fovea.
So fine are these that for
a long time it was declared
that the region of the macula was the most poorly supplied with
vessels. This, however, is erroneous, as has been shown by Nettle-
ship, Becker, Loring, and others. Nettleship says: " On comparing
different parts of the retina, I find that while in an area of 5-gVo
square inch> in the yellow spot region, forty complete capillary
meshes can be counted, not more than from six to nine are included
in the same area at a spot -^ inch behind the ora serrata, the injec-
tion being equally complete in both places. The area of the fovea
centralis, which is destitute of vessels in the specimen here figured, is
equal to about ^Vir square inch, and is irregularly oblong. It is
scarcely larger than the single capillary meshes at the ora serrata."
Fig. 22.—i, Internal sheath of optic nerve; e, e, external
sheath of optic nerve; v, the intervaginal space; I, lamina
cribrosa; c, c, posterior ciliary arteries; S, S, sclera; Ch,
choroid; R, retina; t, T, tendinous or scleral ring; p, P,
choroidal ring; C, optic papilla.
THE OPHTHALMOSCOPE.
49
Another frequent anomaly is that a vessel may come out at the
margin of the optic nerve, or at a point beyond the margin, and
go back to the retina. These have been called cilio-retinal vessels
(see Nettleship: " Royal London Oph. Hosp. Reports," vol. ix., part
2, p. 161, December, 1877). Mr. N. found one such vessel in a micro-
scopic examination of the optic nerve, and proved that it passed
from the sclera, at the level of the lamina cribrosa, into the nerve
and to the retina, and such vessels seem in all cases destined to the
supply of the region of the macula lutea. The opportunity of see-
ing them is most often given in the choroidal crescent of myopic
eyes.
Fig. 23.—Blood-vessels of the Retina, ans, Arteria nasalis superior; ani, arteria nasalis inferior;
ats, ati, arteria temporalis superior and inferior; vns, vni, vena nasalis superior and inferior;yes,
vti, vena temporalis superior and inferior; ame, vme, arteria et vena mediana; am, vm, arteria et
vena macularis.
The retina is to a slight degree discernible as a tissue, notwith-
standing its transparency, and near the disc its optic nerve-layer
usually appears, with greater or less conspicuousness, as fine hair-
like lines radiating from the margin. Above and below they are
most marked, and they cluster around the principal vessels. The
visibility of the retina, as wTell as the tone of the fundus, depends
chiefly on the quantity of pigment in the epithelium and m the
choroid. In blue-eyed persons the retina seems very transparent,
and the fundus of a brilliant red. In dark-eyed, and especially in
dark-skinned persons, negroes, Indians, etc., the retina seems opal-
escent, and the hue of the fundus is dull, and of a dun or tan color.
4
50
DISEASES OF THE EYE.
The pigmentation is always deepest about the central region, be-
cause the epithelium is more saturated, while the remoter parts
permit the choroidal vessels to be seen as light-red stripes with ir-
regular islets of pigment. The surface of the retina sometimes
shows a flashy, silvery reflection, which glances along the vessels
and plays about the macula lutea. It alters in place and form, on
the slightest movement of the eye or of the mirror, and the spot
which it has left has a perfectly normal look. This is seen in dark
eyes and in young children most frequently. It is not pathological.
Another phenomenon is a circle which sometimes appears at the
middle of the fundus, around the fovea as a centre, and has a diam-
eter varying from one to two discs, as seen by the upright image.
This is also visible by the inverted method, and is evidently an
annular reflection. Probably in these cases the source of reflection
is the membrana limitans. The reason why the macula should be
the special seat of such appearances is its convexo-concave surface.
The fovea centralis often shows as a small glistening dot, more or
less completely circular as the light plays over it. Its concavity
favors its action as a reflector. In a myopic eye, where this was
seen, I have observed it to be most brilliant before the perfectly
correcting glass was employed, and that when this was used it dis-
appeared almost entirely.
It has been said that the arteries of the retina are smaller and
brighter than the veins. It must be added that they exhibit a well-
defined line of light along their centre, which, in the veins, is much
less conspicuous. This is an optical effect whose cause has been
disputed, and a most valuable paper upon it was published by Dr.
Loring in Tr. Am. Oph. Soc., 1873. That it is due to the refractive
action of the column of blood in the vessel condensing the light
which passes through it and is again reflected from the underlying
surface, has been proved to myself by two cases. In one of them
there was an effusion of blood beneath the choroid, which made a
dark patch. This was crossed by a vein on which no light-streak
was present while it traversed this dark surface, but where situated
upon the normal choroid, the usual streak was distinct. As the
blood-patch became absorbed and a white scleral surface came to
view, which was caused by rupture of the choroid, not only did the
vessel recover its usual light-streak, but this became much more
decided than upon the adjacent portions of the vessel. A second
case bearing on this point was one of extreme colloid deposit upon
the choroid, having all the brilliancy of the most marked patches of
fatty degeneration, as found in albuminuric retinitis. This glitter-
ing surface was about two discs long and one disc wide, and was
behind one of the transverse retinal arteries. As the artery crossed
this spot, the whole vessel was a bright ribbon of light—the central
THE OPHTHALMOSCOPE.
51
streak being intensified and widened so as to equal the diameter of
the vessel. On either side of this spot the artery had the usual
appearance. It is therefore evident that the light-streak depends
chiefly on the reflecting properties of the surface over wrhich the
vessels pass, and on the nature of the blood-column. That some
reflection comes from the surface of the vessel is true, but it is ex-
cessively slight, as proved by my first case while the blood-patch
was fresh and dark. The " light-streak" is, therefore, a phenome-
non of refraction and reflection, and the light must pass through
the vessel from in front and penetrate to the sclera, to be then re-
flected from the latter and again acted upon by the blood-vessel,
which condenses it into the bright, luminous streak. This is essen-
tially the view first announced by Loring (see " Trans. Amer. Oph.
Soc," 1881). Davis coincides with Loring's explanation, having re-
peated his experiments by letting the blood from the carotid of a
cat flow through fine glass tubes (Arch, of Ophth. XX. 1., 1891).
Story discusses the matter, and agrees with an opposing theory of
Schneller, ()]>hthal. Review, April, 1892; while Dimmer is quoted
by Story in favor of a totally different theory, viz.: that the reflex
in the veins is from the surface of their blood column, and in the
arteries is from the axial part of the blood stream (Ophth. Review,
May, 185)2). My observations above quoted have not been noticed
by the writer mentioned, and my views remain unchanged.
Pulsation of the veins upon the optic disc is quite common. It
is explained by Donders as the effect of the arterial tension com-
municated to the veins through the vitreous, and causing pulsatory
movement on the optic disc, because here the column of venous
blood is just escaping from the intraocular pressure. It is most
apt to be seen when the veins are large. Schoen was able to study
this in a patient whose pulse was only 16 to 23 per minute. He
concluded that the venous pulse is merely the effect of the pulse of
the artery upon the vein as the two vessels lie in juxtaposition
in the optic nerve.—Klin. Monatsbldtter (Zehender), Sept., 1881.
Pulsation of the arteries occurs when the intraocular pressure rises
to an abnormal degree, or in cases of disease of the heart (aortic
valves) or large vessels, and under some other morbid circumstances.
Pulsation of both arteries and veins can always be caused by pres-
sure with the finger, and, if it be made very strong, the circulation
can be entirely suspended.
In observing the fundus closely, if the tissues are normal and
the refraction perfectly corrected, the retinal epithelium is seen as
a granular surface, like the finest emery-paper, and its molecular
look is perfectly distinct. A few glistening dots are sometimes seen
near the macula, which appear to have no special importance.
52
DISEASES OF THE EYE.
The fovea centralis is always the most difficult spot to examine,
especially with undilated pupil. It has a dull, red look, or majT re-
turn, as above stated, a gray reflection, which may be a partial or
complete ring, which flickers at the slighest movement, and is about
one-fourth or one-sixth of a disc in diameter. In young persons it
is widest and most distinct. The very centre is so deep in color as
to be almost brown.
The degree to which the choroid can be seen varies with the
pigmentation of the «ye. In albinoes the vessels are visible, even
about the macula. In greater degrees of pigmentation, some ves-
sels may appear between the nerve and macula, and in all persons
they are distinct at the eccentric parts of the fundus. They are of
a light pink hue, appearing like flat stripes, and have a curvilinear
arrangement and interlacement in distinct meshes. No distinction
can be made in them between arteries and veins. Sometimes the
place of beginning of the venae vorticosas is recognizable. Between
the meshes of the choroidal vessels the pigment-stroma is seen in
more or less dark patches of irregular shape. The visible choroidal
vessels are always broader than the retinal trunks. Immediately
around the optic nerve the choroidal pigment is often quite abund-
ant over a considerable breadth of surface, and, as above said, the
central part of the fundus is overspread with a uniform layer, which
usually completely hides the choroidal vessels. For verification of
above description, see colored plate at the back of the book.
CHAPTER V.
GLASSES.
As we shall have to consider the proper use of glasses in correct-
ing errors of accommodation and refraction, we may say a few
words in general upon their varieties and properties.
We have to deal with glasses of spherical curvature which are
convex or concave, and we have glasses of cylindrical curvature,
also convex or concave. We also have glasses whose surfaces are
plane, but not parallel to each other, viz., prisms. Very exception-
ally glasses ground to a hyperbola have been used.
In spherical glasses we have the following forms:
The convex are called positive or collective or magnifying glasses,
and are denoted by the sign +. The concave are called negative
or dispersive or minifying glasses, and are denoted by the sign —.
The focus of a glass is the place where the rays from a given object
Fig. 24.
cross each other on the axis of the glass. For parallel rays the
place of crossing is called the principal focus, and this is understood
when no adjective is used. If an object be near enough to the lens
to emit diverging rays, these, if they cross, do so at points called
conjugate foci. For convex lenses the foci are real and positive, and
on the side of the lens opposite to the object. For concave lenses
the foci are negative, imaginary, or virtual, and on the same side
with the object. But for convex glasses, if the object be situated
at the principal focus, the rays after passing the lens will not con-
verge, but be parallel; hence there will be no focus. If the object
come still nearer, the rays will be divergent, and the focus virtual.
For concave glasses the rays become more divergent as the object
approaches the principal focus, and at this point rays cannot pass
through, because the divergence becomes too great. In Figure 24,
54
DISEASES OF THE EYE.
we have the principal forms of lenses, viz.: the plano-convex A, the
biconvex B, the convex meniscus C, also the plano-concave D, the
biconcave E, and the concave meniscus F. The first three are all
positive, and the last three are all negative lenses. The biconcave
and biconvex are supposed to have curvatures the same on each
side, but this may not be, and frequently is not, the case.
Images from plus (i.e., convex) glasses are inverted and smaller,
if the object be beyond the principal focus. If the object be at the
principal focus, no image is formed. If it be nearer than the prin-
cipal focus, the image is not real, but is virtual and erect, and larger
than the object; the lens then becomes a magnifier (loupe). Im-
ages from minus (i.e., concave) glasses are always small, erect, and
virtual, provided the object be farther than the principal focus. If
an object lie at or nearer than the principal focus, no image can
be formed.
Cylindric glasses are ground by a cylindric tool, and have a
curve whose maximum is at right angles to the axis of the cylinder,
Fig. 25.
while in the direction parallel to the axis there is no curve. Such
glasses cannot form images, although they may be said to have
foci according to the laws of spherical lenses. Cylindric glasses
are shown in Figure 25 and represented with square outlines.
In practice they are cut oval like other glasses. The axis is shown
to be along the middle of the curve and parallel to the vertical
edge.
The Arrangement and Nomenclature of Glasses.__Formerly
no better aid could be had in choosing glasses than the advice of
the optician from whom they were purchased. At present, oph-
thalmic surgeons find their function to consist largely in advice upon
this subject. They require a trial-case more or less complete, which
must contain spherical and cylindric glasses, both convex and con-
cave; also prisms from 1° up to 20°, and a suitable frame. A slip
of red glass and an opaque screen are usually added. In giving
numbers to glasses it was formerly the practice to do so upon the
assumption that the index of refraction of the material was 1.5 (3)
GLASSES.
55
and, for a double convex or double concave glass both whose curves
were alike, the focus was found by the rule that the focus was equal
to the radius of curvature.1
Another embarrassment is the want of uniformity in the inch
measure among different nations. The following are samples: the
English inch is 25.3 mm.; the Austrian is 26.34 mm.; the Prussian
is 26.15 mm.; the Paris inch is 27.07 mm. Between the English
and the Paris inch the difference is ^. In the numbering of glasses,
therefore, two things needed to be readjusted: first, the error aris-
ing from regarding the radius of a bi-spherical lens as the equiva-
lent of its focus, and vice versa; secondly, the discrepancy as to the
standard measure. The latter objection is overcome by abandon-
ing the use of inches and employing the metric system of expres-
sion. The first difficulty is obviated by numbering glasses accord-
ing to their refractive power and not according to their focus.
Refracting power is the reciprocal or inverse of the focus. Thus,
a lens of 30 inches focus has a refractive power of ■£$. This frac-
tion may be expressed in decimal form and it becomes .033. A lens
of 20 inches focus has a refractive power of ^, or .05. A lens of 4
inches focus has a refractive power of \, or .25.
The glasses in actual use began at the numbers with long foci,
and came down to those of short foci—that is, from the weak to the
strong; but there was no regularity in the progression; no com-
mon interval was observed (see column 1 of the table on page 57).
Now, for purposes of scientific study, and for convenience in exami-
nations, regularity of interval is highly convenient. Attempts
have been made to secure this desideratum, and various intervals
have been suggested, viz., the fractions $\w, T|- *V> iV When,
however, the metric measure was substituted for the inch, it was
also resolved to establish a metric interval which should become
the unit of measure and the standard of gradation between num-
bers. Facility in calculations, and uniformity, both in gradation
and in nomenclature, were the objects sought. The unit is a glass
of one metre focal length, which in English measure equals 39.37
inches, and is called a dioptry (French dioptrie) (D). In French
measure it would be 37 Paris inches. But this interval is too great,
and therefore the metre is again divided into fractions. By the old
1 This results from the formula F=2 / • in which F stands for focus, r for
radius and n—1 for index of refraction. If now r=12 and w=1.5, the formula
becomes F—2(15_1)=1I8-=12, that is, the focus is equal to the radius. It turns
out that the glass now in use does not have the index 1.5, but a higher degree
varying between 1.52 and 1.55 (Nagel). Javal assumes it to be 1.54. If we
substitute this in the formula, we have F—2 (1'^ =^^=11.1. That is, the
focus is less than 12 inches, and very nearly 11 inches.
56
DISEASES OF THE EYE.
method a lens was known by a number which was its radius of
curve, and this was assumed to be the same as its focal length. It
is now known by its refractive power, and this is expressed by the
number of dioptries contained in it. It is seen that a metric mea-
surement of glasses may be quite distinct from the system of diop-
tries. But, where the metric system has been adopted, the dioptric
interval has also been accepted. Nagel gives in Graef e and Saemisch
(B. VI., p. 310) the mode of converting the old into the new system
of measure. He assumes the index of refraction at 1.528, which is
German glass, and with this he finds the equivalent of a dioptry
to be 41.5 English inches. If the index be that of French glass, at
1.54, the dioptry becomes 42.5 English inches; with index of 1.53 it
becomes 41.7 English inches. Nagel proposes that, in transmuting
the old to the new system, 40 inches be taken as the equivalent of
the dioptry, and with this Javal concurs, the error not being very
large. Therefore, an 80 inch glass =0.5D : 40"=1.D : 20" =2.D: 16"
= 2.5 D : 10' = 4D. The table on the next page, modified from
Mauthner, gives a sufficiently complete series, and according to
both systems, accepting 40" as the dioptry.
It is seen that at the upper end of the scale the interval between
glasses is small, viz., 0.25 D, but that beyond 3.5 D (11 inches) the
interval is .5 D, and again becomes still greater. The reason for
a large interval among the strongest glasses is that a slight alter^
ation in their distance from the eye greatly modifies their refract-
ive value, and any little change can be thus effected. The special
advantage claimed on behalf of the dioptric system is the ease with
which calculations can be made in adding and subtracting lenses.
For instance, put two positive lenses, + 2 D and -4- 3 D, together,
and their result is -j- 5 D. If + 3 D and —ID are united, + 2 D re-
sults. If — 4 D and —2D unite, — 6 D results. One need only deal
with simple numbers, and remember the effect of the precedent signs
of + or —. If lenses are to be united which are designated only by
their foci, the calculation must be made in fractions, viz., + 3 D and
+ 1D, become TV+iV—o-=tV- A lens of six inches focus added to
one of twelve inches focus £ + iV=i«
The use of the system of dioptries makes calculations simple,
but it is a mistake to imagine that it makes any practical difference
in selecting glasses. We are compelled to take what the patient
actually needs, and whether we express ourselves in terms of diop-
tries, or of focal length, or of refractive power, is not of strenuous
importance. Certainly one dioptry is too large an interval, a half
dioptry is often too small, and we may be obliged to choose glasses
which do not come under this rubric at all. The essential thing is
to know the real refractive worth of our glasses, and whether we
reckon it in dioptries or by other means is indifferent.
GLASSES. 57
Focus in Number in Focus in Number in
Inches. Dioptries. Inches. Dioptries.
160 0.25 14 2.75
80 0.5 13 8.
60 (0.67) 12 3.25
50 0.75 11 3.5
40 1.00 10 4.
36 (1.11) 9 4 5
30 1.25 8 5.
24 1.5 7 5.5
(22) 1.75 6* 6.
20 2. 6 6.5
18 2.25 5* 7.5
16 2 5 5 8.
The above table shows the glasses according to the old enumer-
ation in English inches and their equivalents in dioptries. We give
below the dioptric series and its equivalent in inches.
Inches aj'prox- Indies approx- "nches approx-
Dioptries, D. imately. D.optries, D. imately. Dioptr es, D. imately.
.5 80 3.5 11 11 31
.75 50 4. 10 12 3*
1. 40 4.5 9 13 3
1.25 30 5. 8 15 2|
1.5 24 6. 64 16 2*
1.75 22 7. 5* 18 2i
0 20 8. 5 20 2
2.5 16 9. 44 40 1
3. 13 10. 4
Attempts have been made to make available a fewer number
of glasses than the list above given, by using a spectacle-frame
which may carry simultaneously a combination of three for each
eye. Dr. E. G. Loring, jr., and Dr. John Green, and Dr. Roosa,
have gotten up such a series, and where cheapness is more to be
regarded than convenience it will answer the purpose. It must,
however, be admitted that three glasses put in the place of one
single glass will not in practice be the same, however correct the
calculation, because by six reflecting surfaces the loss of light is
three times greater than by two surfaces, and for strong glasses
allowance must be made for their respective distances from the
nodal point. If, however, the choice lies between an abridged series
and no spectacle-box, the former alternative is much to be pre-
ferred. Such a box is sold by Meyrowitz Brothers in this city.
The power of glasses depends not only on their focus, but on
the distance at which they stand from the nodal point. This varies
according to the depth of the globe in the.orbit, and the height of
the nose, and the kind of frame. When, however, a glass is worn
at the anterior focal distance of the eye, which is about 13 mm.
Focus in Number in
Inches. Dioptries.
44 9.
4 10.
3f 10.5
34 11.
34 12.
3 13.
24 14.
24 16.
24 18.
2 20.
58
DISEASES OF THE EYE.
from the cornea, it has the least influence on the size of the retinal
image—an important fact first pointed out by Giraud-TeulonJ and
later by Knapp. But moved to any other place its influence is im-
portant. At this position the visual angle is not altered, but the
linear dimensions are changed. Usually the distance is about £
inch, or 2 cm. This is to be added to the power of a convex lens and
subtracted from the power of a concave lens. The practical im-
portance of this fact is chiefly felt among the stronger glasses.
One who wears a glass as high as + -§- finds that by slipping it down
upon the nose it becomes stronger, and with persons who use cata-
ract glasses this manoeuvre is often of advantage. On the other
hand, if a "deep" concave glass is worn, say—\, its power is dimin-
ished by holding it away from the eye, and increased if pushed
Fig. 2G.
nearer to it. When discussing errors of refraction, reference will
again be made to these points.
The frame in which trial glasses are placed is not an unimpor-
tant matter. There are many contrivances: some very elaborate
and likewise heavy. A pattern made by Nachet in 1866, and which
is now made by G. Tiemann & Co., New York, has served me for
more than twenty years with great satisfaction. Its various ad-
justments are readily understood (see Fig. 26).
Bi-cylindric glasses are sometimes employed in lieu of bi-spheri-
cal lenses when very high numbers are required, as, for example,
after extraction of cataract. They give a flatter field and less
aberration. In case a cataract glass must have a cylindric surface
amounting to two or more, perhaps as many as six dioptries, one
surface may be composed of the cylindric convex surfaces with their
axes at right angles, and the other surface may be spherical An
illustration is the following: a glass -f 11.D.C+ 6.DC 180° is resolved
into + 5.DS C + 6 Dc 90° + 12.DC 180°. That is because in one meri-
1 Annales d'Oculistique, Sept., Oct, 1869. "------
GLASSES.
59
dian + 17 D and in the opposite + 11 D being called for, the differ-
ence of 6 D is thrown into one surface by employing cylinders at
right angles of + 12 D and 6 D, leaving for the opposite side 5 D
(17 — 12), to be thrown into a spherical form. Such a lens is called
" toric;" it is much thinner than the usual sphero-cylinder, gives a
flatter and more correct field, a wider visual angle and better vision.
The discussion of toric lenses is found in an article by Dr. John
Green (Trans. Am. Ophth. Soc, 1890, p. 708). They are manufac-
tured by a few opticians in Philadelphia and New York.
Prisms used in ophthalmic practice are of only moderate angle,
usually below 6°, rarely as high as 10°. Rays passing through
them are deflected toward their base, and the apparent position of
an object is shifted toward their angle. The least displacement
occurs when rays fall perpendicularly upon the surface, and because
this is plane they are not collected to a focus and do not form
images. Some dispersion and decomposition of light takes place,
and in direct ratio to the angle of incidence.
For prisms of small angle the degree of displacement is equal to
half the angle—and until recently they have been designated sim-
ply by their angles. Another nomenclature has now been proposed
to put them in harmony with the system of numbering other
glasses, viz., according to their effect, and not according to their
construction. The degree of deviation is found by combining a
prism with a convex lens, and meas-
uring the amount of displacement of
the image at the focal plane. In
the following table taken from the
report of a committee (see Trans.
Am. Ophth. Soc, 1888, p. 151), the dis-
placement is given both in degrees
and linear measurement (the sine or
tangent of the angle, which for
small angles are the same).
It is proposed to number prisms
simply by the figures of the first
column, leaving out of view their
angle and the index of the glass.
A further development of the idea is the prism dioptry of Prentice
(Archives of Ophthalmology, XIX., 1,75, and XIX., 2 and 3, p. 128),
in which the unit of measure is a prism which shall cause a tangent
deflection of 1 centimetre at the distance of 1 metre, and this
prism, P.D., becomes the multiple of the scale. Its angular value
can be deduced from the preceding table, which is calculated for
one-half the distance—and 1 P.D. will be very nearly a prism of 1°.
But Mr. Prentice as a practical optician declares that he has found
Angle of Deviation. Angle of Refraction. Linear Displacement at 0.5 metre.
0.5 0.93 4.3 mm.
1. 1.85 8.7 "
1.5 2.78 13.1 "
2. 3.70 17.4 "
2.5 4.63 21.8 u
3. 5.55 26.2 "
3.5 6.48 30.6 "
4. 7.40 34.9 "
5. 9.23 43.7 "
6. 11.5 52.5 "
7. 12.58 61.4 "
8. 14 63 70.1 "
9. 15.40 79.
10. 18.18 88.1 "
15. 26.74 133.9 "
60
DISEASES OF THE EYE.
by experiment that prisms noted as of 1° produce deflections vary-
ing from 9 to 12 millimetres. Doubtless we must aim at absolute
accuracy in all kinds of glasses, and for this reason one or other
of the above systems will in the future be chosen; yet for the
present the notation by angles prevails. In the choice of prisms
we cannot yet lay down rules with as much precision as in the case
of focalizing glasses, yet they fulfil an important function, as will
be explained hereafter. They may be used simply or in combina-
tion with convex or concave glasses. The benefit of prisms is found
in connection with the functions of the ocular muscles, and the
prism dioptry has a relation to the metric angle which will be here-
after referred to,
CHAPTER VI.
ACCOMMODATION AND ITS ERRORS.
Presbyopia.—The natural abatement of accommodation which
takes place from the gradual hardening of the lens, has been already
described. It remains to speak of it clinically. The period of life
at which the ability to read ordinary print at the usual distance of
fourteen inches becomes fatiguing, varies in emmetropes between
forty-five and fifty years. At an earlier age there may be need of
assistance in feeble persons or invalids, while in the very robust
and especially in those who do not greatly tax their eyes in
near work the occurrence of presbyopia may be further deferred.
The size of the pupil has a material influence, and to its small-
ness some persons owe their immunity from glasses at the usual
age. It is also common to find that a very slight myopia has ex-
isted in some individuals who ought, but do not, require glasses in
near work. In other cases there may be incipient cataract to
account for the refusal of glasses, as will be again mentioned.
Presbyopia usually comes upon people like their gray hairs, without
announcement or anticipation. They first complain that evening
work is troublesome, the light seems dim, and if they bring the
work closer, or provide better light, the print fails to become dis-
tinct. They find it better to hold the book farther away than for-
merly; the print seems pale, letters run together; their eyes smart
and give pain. Frequently such symptoms are endured for months,
and sometimes the reading distance is pushed as far away as the
arm can conveniently stretch. On the other hand people who
recognize the probable nature of their trouble are sometimes un-
willing to have their true age suspected, and they give up reading.
It is, indeed, sometimes ungracious to tell a lady that her troubles
can be mended by using a pair of spectacles. While presbyopia
usually comes on gradually, it sometimes appears very suddenly
and also prematurely, as a result of severe nervous prostration.
The question is asked whether it is better to defer the use of
glasses as long as possible. To this, the answer should be, that as
soon as discomfort arises for need of them, they should be adopted.
It is seldom that a glass so weak as .5 D is appropriate. The glass
which usually is called for is + .75 D, -f- 53" at the beginning.
62
DISEASES OF THE EYE.
Practically many persons get on very well until they require +1
D. A certain amount of accommodative power must remain in re-
serve to permit continuous and comfortable Avork. This law gov-
erns all muscular activity. How much reserve there should be, it
is of little use to theorize, because the question is for each individual
decided, according to his own needs. The reading distance varies
greatly among emmetropes, and lies between ten and twenty inches;
hence, much latitude is to be allowed in the number of the glass,
and the occupation and requirements of the person are to be the
guide. A portrait painter, a violinist, a book-keeper with large
ledgers to run over, these and other workers use their eyes at a
range which is proper to their calling, and not that which any theory
demands. As to the amount of reserve accommodation, it need not
be especially considered. The indication is to give the weakest
glass which makes work easy at the accustomed distance. If one
reads at 12" and cannot easily see Snellen II nearer than 18" the
glass required is ■& — -fa = z^s. — ^ — i D. In metric measure it is
3D — 2D = ID. The rate at which the strength of the glass should
be increased is to be considered. About this the rule is not abso-
lute. One must be guided by symptoms, usually no addition is
needed for two years after first taking glasses; the interval may
be longer. The increase should not be by greater increments than
.75 D. When 3 D is reached, this glass can usually be maintained
for several years. There will be need sometimes of a stronger glass
by night than is used by day. In case one wears -f- 3 D for read-
ing and finds suddenly a notable failure in reading power and must
resort to a decidedly stronger glass, there is serious reason for a
careful examination; this may indicate the onset of glaucoma sim-
plex. In fact, rapid loss of accommodative power at middle life,
whether a glass be employed or not, is a symptom to be regarded
with suspicion. It is an accompaniment sometimes of diabetes.
When glasses equal to + 4 D and higher, are used, there is a de-
cided restraint in the range of accommodation. The working
point is at a nearly fixed and unchangeable distance. Hence it
is undesirable to advance to the strong numbers. This limitation
is inherent in the working of the glasses. It follows of course
that with the loss of A and the maintenance by glasses of the
working distance at a given point, that the convergence is kept
up at a uniform quantity notwithstanding the abatement of ac-
commodation. Hence some of the annoyances of using reading
glasses are connected with the disassociation between convergence
and accommodation. Usually the function of convergence adapts
itself to the state of A and no special arrangements are required.
But where strong glasses are used, their prismatic effect must be
remembered. If the visual axes pass through their inner ed°-e,
ACCOMMODATION AND ITS ERRORS. 63
they increase the adduction, while if the glasses are decentred in-
ward and the axes pass through their outer half, they act as ab-
ductive prisms. The choice between spectacle frames and eye
glasses is to be decided chiefly by considerations of convenience,
and it is always desirable to give the glasses area enough to keep
the frames practically out of the field.
Second Sight.—It happens sometimes that at or above sixty
years of age, persons lay aside reading glasses and rejoice to find
themselves restored again to what they call their youthful sight.
This occurrence is not very rare and is popularly called second sight.
I have examined many of these persons and found the explanation
in sclerosis of the lens with or without opacities, and a small pupil.
It has been clearly shown that the refractive index of the lens is
in many cases increased and a real myopia may be induced. I can
quote such instances, and many authors have written upon it, e. g.
Mauthner,1 Priestley Smith,2 etc. It does not always follow in
these cases that cataract will develop to maturity and one need
not therefore mar the happiness of the person by dismal prognos-
tications. While emmetropic persons must in the usual course of
nature seek the aid of convex glasses at a certain age, the same
necessity befalls l^peropes at a period earlier in proportion to the
degree of their error. They have already called largely upon their
accommodation, and while they enjoy more of it than others
possess, their capacity is sooner exhausted. Hence resort to read-
ing glasses at an early age is presumption of hyperopia.
On the other hand myopes will weaken the glasses which they
have been using. With them accommodation has been little taxed,
and if they have been used to working without glasses, it will be
found that their working range is always limited. If they have a
low grade of error they will be obliged to adopt convex glasses for
work and retain concave glasses for the distance. The changes to
which they will be subject are easily determined by subtracting
from their far-point the number of dioptries required for their near-
point. A patient with M = 5 D who would read at 13" or 3 D must
use 5 — 3 = 2D. One who has M = 2 D and no longer has A suf-
ficient to read at 13" or 3 D must now use + 1 D.
Spasm of Accommodation.—The structure of the ciliary mus-
cle is in some respects still an unsettled question. Composed of
meridional (Briicke's) and circular (Muller's) fibres, the precise
course which they assume is not fully determined. Some fibres
would appear to be oblique and therefore intermediate in direction
between the above sets. It is well known that the relative pro-
portion of these fibres differs in eyes of different refractive quality.
1 " Vorlesungen uber die Optischen Fehler der Augen," p. 460.
' Ophthalmic Review.
64
DISEASES OF THE EYE.
In H the circular are far more numerous than in M, while in M the
meridional exceed the number present in H. Emmert has sought
to show that the meridional fibres have an active effect in flattening
the lens, which would carry the focus farther back, but such an
assertion lacks proof and is contrary to the natural presumption.
Myopic eyes accommodate feebly, but on becoming habituated to
glasses in early life they soon acquire a normal range. Whether
ir such cases there would be found a proportionate increase in
circular fibres would be worth investigation. In myopic eyes there
is always a certain tone of accommodation which is removed by
atropia, but in hypermetropic eyes the proportion of permanent
contraction of the ciliary muscle is much higher. This amount of
effort is what may be called the tonic state of the muscle. It is
impracticable to measure its normal limits. On the other hand an
undue degree of constant effort is called spasm of accommodation.
For example, if a slight hypermetropia is changed into a myopia
of 3 D, or should a hypermetrope of 6 D refuse to accept a glass
stronger than 1.5 D. Such are extreme cases and we are called
upon to take account of them and of lesser grades whenever they
are associated with pain or asthenopia.
The causes of spasm are local, viz., such as lie in the structure
of the eye, in the condition of the motor muscles, in injuries and in
external inflammations such as conjunctivitis, keratitis, episcleritis,
etc. These are reflex causes of spasm. There are also less fre-
quent cases of spasm due to irritation of the central nervous
system, as in epilepsy and hysteria and it may even be associated
with hemiplegia; irritation of the sympathetic in the neck and
of the cilio-spinal region of the cord have also been assigned as
causes. It has been found that one eye alone has been affected,
but this is altogether contrary to the rule. An injured eye may
set up sympathetic irritation in its fellow of which a component
symptom will be spasm of accommodation. All the above-men-
tioned causes are exceptional and rare, while refractive and mus-
cular errors are by far the common and ordinary causes. That
central nervous lesions and various reflex causes have such an
effect is indubitable. We are authorized to infer it from the
frequency with which the pupil is affected by remote nervous
causes. Both the ciliary muscle and the sphincter pupillee are
under control of the third nerve and at independent centres. The
sympathetic presides over the dilatation of the pupil, and it is
claimed by Emmert that it has the same control over the meridi-
onal fibres of the ciliary muscle. This is, however, not proven. We
may have spasm of the ciliary muscle without contraction of the
pupil; and it is recognized that while the nucleus of the third nerve
on the anterior part of the floor of the fourth ventricle looks like a
single mass, it is functionally divisible into several centres the
ACCOMMODATION AND ITS ERRORS.
65
centres for the pupil and for the ciliary muscle being in its front
yet separate (see page 153).
The diagnosis of spasm rests upon the refraction of the eye
when tested by glasses for distance, compared with the refraction
proved to exist when examined objectively by the ophthalmoscope,
and with the status under the influence of mydriatics. Moreover, if
a patient has the habit of reading at a distance too near in propor-
tion to the state of refraction, this indicates spasm. Let a patient
read with convex glasses, say 3 D. He should see Snellen II from
13" to 6" if his accommodation be fa, or to 4" if it be £. If now he
can read only at 8" and brings the book to 3", the difference be-
tween fa and £ shows the amount of A which he cannot relax, i.e.
^j. This examination is not to be accounted of more significance
than it deserves, because it must be estimated in connection with
other symptoms, and the function of convergence has a special in-
fluence which must not be overlooked. For this reason it is my
habit not to use simple convex lenses in this mode of testing, but
lenses combined with abductive prisms. To these reference will be
made in speaking of asthenopia.
The condition occurs chiefly among asthenic subjects, and more
especially among young persons and students.
Examination by the ophthalmoscope sometimes shows that even
under its illumination, spasm is not relaxed; this may be evidenced
by variations in distinctness of the fundus under the observer's eye,
or it may entirely elude detection until developed by protracted
use of atropia.
Treatment.—In cases of muscular or refractive error the appro-
priate glasses will be all that is required. In what may be called
idiopathic cases, abstinence from use and the employment of my-
driatics will usually control the trouble. But in some severe cases
the artificial leech to the temples and injections of strychnine
(Nagel) have been required. The vigor with which atropia is to be
used varies considerably. In some and especially in young sub-
jects, it will have to be pushed to the verge of intoxication; of
which the symptoms are always unpleasant and may be dangerous.
It is better to keep up the remedy for one or two weeks in moderate
doses than to resort to the strong solutions. The most frequent
remedy is sulphate of atropia, and for young subjects 2 grains to
the ounce is sufficient, or at the most 4 grains to the ounce, used
thrice daily, 2 drops in the eye. The dilation of the pupil which also
occurs may compel the use of colored glasses. Other substances
may be employed in case atropia is inefficient or has unpleasant
effects. Such are duboisine sulphate and salicylate and still more
recent is hyoscyamine and its isomeric form hyoscine. These alka-
loids, while more potent than atropia, are also liable to cause toxic
66
DISEASES OF THE EYE.
constitutional effects and must be resorted to with caution. Duboi-
sine may be used in \$ doses, i.e., gr. ij. ad § i., one drop at a time
until its tolerance is proved.
It also may happen that the spasm will recur either because
the local causes return or because of constitutional conditions. In
the obstinate or recurring cases strychnia as a tonic may be used
either by the stomach or hypodermically, while special pains must
be taken to discover any co-operative cause in weakness of the
muscles or in error of refraction, such as hypermetropia or astig-
matism. If there be general debility and an asthenic condition,
special care must be taken as to food, sleep, exercise, absence of
excitement, abstinence in reading and close work.
An extremely frequent cause of spasm is insufficiency of the
recti externi muscles, a condition whose occurrence is far more
prevalent than has been recognized. To this attention will be
called and the remedy will be found either in the use of prisms or
in tenotomy of the interni (see note, p. 68).
Paralysis and Paresis of Accommodation.—A merely feeble ac-
commodation and incapability of endurance, is not what is now
referred to. We speak of a real abatement of degree irrespective
of age. The causes of these conditions may be local, but are gener-
ally found in some constitutional disorder or in some lesion of the
oculo-motor nerve, either along its track or at its origin, or of the
nerves from the ciliary ganglion. We do not take into account the
cases of total paralysis of the third nerve which will necessarily
include the accommodation. It is not even necessary that the pupil
should be implicated. The most frequent cause is diphtheria, and
the effect may follow soon after or during convalescence, or at
some remote period. It may disappear and recur. It may be the
chief symptom of persistence of the poison or it may be only one
of many other paralyses. It is asserted by Mooren and Hutchin-
son that other affections of the throat not diphtheritic, can cause
paralysis of accommodation. After fevers, typhoid and recurrent,
after articular rheumatism, in diabetes, in trichinosis, in cerebro-
spinal sclerosis, in locomotor ataxia, in essential anaemia, from de-
bilitating excesses as by masturbation or venereal indulgence, or
alcoholism; as an incident in uterine disease and especially as the
result of syphilitic affections we may have impairment or paraly-
sis of the ciliary muscle. If the affection is one-sided, the cause
will be local and we search for it anywhere along the third nerve
to its origin, and the probability will be in favor of syphilis. In-
juries of the bones of the orbit or of the eye have an effect probably
through reflex influence, and the same has been noted in neuralgia
of the dental and other branches of the fifth nerve. Out of this long
catalogue of ailments, which is still incomplete, one may well find it
ACCOMMODATION AND ITS ERRORS. 67
sometimes difficult to select the correct etiology. Diagnosis need
not be dwelt upon; the affection publishes itself by the same tokens
that we are familiar with in presbyopia by their more complete
manifestation. Prognosis is important because the local malady
may be the forerunner of a more serious disease which will com-
promise the life or general welfare of the patient. Such will be the
cases in which diabetes or an obscure syphilitic disease of the ter-
tiary type, or disseminated sclerosis, or locomotor ataxy, sometimes
general paresis and occasionally insanity, are looming up, or have
already descended upon their victim. For this reason a careful
search must be made in the whole domain of pathological clinical
research in case some obvious cause is not discovered. (See Gowers.)
The history of the patient will be thoroughly sifted. In the greater
number of cases happily the prognosis is good, as, for instance, in
diphtheria and after fevers, whether typhoid or recurrent.
Treatment is, of course, both constitutional and local. The con-
stitutional must be decided on general principles of therapeutics as
connected with causation. It is to be stated with emphasis that
all convalescents must be strenuously cautioned against overtaxing
their eyes at the peril of serious paretic and asthenopic trouble of
the ciliary muscle. To amuse themselves by reading or sewing to
pass the weary hours, is considered a harmless thing, but it too
often proves serious in its effects. Women after confinement, or
with chronic uterine trouble, patients getting well of scarlet fever,
or from severe internal inflammations, need special warning on this
matter. If the patient is going about, as strength improves the
eye power will improve. The above remark applies to abated
capacity for employment of accommodation. When we have to
deal with a real paralysis or paresis the great local remedy is
sulphate of eserine. It must not be given in concentrated doses,
but a solution one-half or one-eighth of a grain to the ounce dropped
into the eye once daily, is enough. The pupil will contract slightly
and the accommodation will soon be stimulated to action; the
near-point is not to be forced to undue proximity and the in-
fluence will last for several hours and then gradually decline.
Pilocarpine is less energetic and if eserine be unpleasant in its
effects on the conjunctiva, muriate of pilocarpine gr. iv. ad 3 i.
may be instilled once or twice daily. The constant galvanic cur-
rent may be employed with eight to twelve cells, the positive pole
over the superior cervical ganglion of the sympathetic at the
upper part of the sterno-mastoid, with the negative pole over the
closed lids. The current to be kept up for two or three minutes
and not to be interrupted. The real value of the galvanization is
difficult to estimate, while the influence of myotics is undoubted.
It is important, however, to use them in moderation, because they
68
DISEASES OF THE EYE.
merely stimulate the muscular fibres and do not act on the cause.
We wish to improve their nutrition and not exhaust them.
It is also permissible to assist persons in this condition by suita-
ble convex glasses for work or reading. If, as sometimes happens,
they are sensitive to light, a little tint of blue may be added. It is
needless to give any rules for prescribing the glasses. It is, how-
ever, proper to guard the patient against presuming upon the value
of the help thus afforded him, and venturing to use his eyes as if
they were not really crippled. Moderate and not reckless use is
all that can be permitted.
Note.—In discussing spasm of accommodation that condition has been
chiefly in view which is to a great degree unconnected with errors of refrac-
tion and motility. When such errors are the cause of spasm, it is not always
needful to direct a prolonged treatment by mydriatics. The hydrobro-
mate of homatropine, gr. xx. ad 3 i., dropped into the eye every ten minutes
for six or eight times in succession will effect relaxation and permit the
prescription of a suitable glass. As the solution of this strength is irritat-
ing, a 4% solution of cocaine may be employed previous to its use. It usually
excites considerable hyperaemia. Its effects disappear within twenty-four or
thirty-six hours. Some oculists rely much on this method. I seldom resort
to it because the ophthalmometer, the ophthalmoscope provided, if astigma-
tism has been found, with the needful cylinder in the clip, and a little patience
with trial glasses will master the large number of cases. When obstinate
spasm of accommodation confronts me I employ atrophia and with vigor,
making a small allowance for the so-called tonic accommodation, about 0.5 D.
We have no remedy which will act on the ciliary muscle without acting on
the iris and vice versa.
CHAPTER Til.
ERRORS OF REFRACTION.
The e3Te so constructed that rays from a distant object form
a perfectly distinct image on the retina is refractively normal and
is called emmetropic (E). The eye which does not collect parallel
rays to a focus on the retina is
ametropic.
Ametropia may result from
shortening or lengthening of the
visual axis, from defect or excess
in refractive power, or from want
of regularity in the curves or the
substance of the refractive media.
Various combinations may occur,
as will be seen. Among them we
have the conditions known as hy-
permetropia, myopia and astig-
matism as subdivisions of ametro-
pia.
In hypermetropia (hyperopia,
H) the image from a distant object
falls behind the retina; in myopia
(M) it falls in front of the retina; Fig 27
the place of the image in astigmat-
ism will require special explanation. If we confine ourselves to
ametropia due to alteration of the length of the visual axis, we see
it indicated in Fig. 27. A, B and C represent respectively the em-
metropic, the hypermetropic and the myopic eyes. In the next
figure a simple biconvex lens is substituted for the eye and if E be
70 DISEASES OF THE EYE.
its principal focus, H gives hypermetropia and M gives myopia.
At E parallel rays are focussed, while at H they have not yet
crossed, and at M they are already diverging. With both H and
M vision is imperfect, because in each the retina does not receive
a picture formed of accurate points of light, but formed of circles
of dispersion; hence it is blurred. It is assumed that there is no
effort of accommodation.
Diagnosis of Refractive Errors.—We ordinarily assume that
correct visual acuity precludes refractive error. It does preclude
myopia, but it does not preclude a certain degree of hypermetropia,
nor of astigmatism. We have already stated (see pp. 22-25), that
visual acuity based upon an angle of 5' is merely an average. For
fine determinations acuity should be placed as high as §£ instead of
f£, making the visual angle 4'. The age of the subject, the degree of
light, the whiteness of the paper, etc., must all be considered.
In all such examinations objective methods which are indepen-
dent of a patient's assertions are to be added to those which are
subjective.
Examinations by test types and spectacles may be called sub-
jective,—examinations by the ophthalmoscope, by JavaPs ophthal-
mometer, and b}' the so-called shadow test, and by Thomson's
modification of Schemer's experiment are objective.
We always test each eye by itself, and for distant vision first.
There is often both greater visual acuity and slight change in the
glass preferred, when both eyes are used than each eye alone. A
stronger convex and a weaker concave will often be accepted when
both are corrected.
If visual acuity is made perfect by convex glasses, we may be
sure there is hyperopia; if it is made perfect by concave glasses and
these are higher than —2D, we may justly think there is myopia,
although astigmatism in either of the above cases is not excluded.
If no spherical glass will give perfect vision, we try cylindric glasses,.
and must not only know whether to choose plus or minus, but must
also know in what position the axis is to be placed.
If, combining spherical and cylindric glasses, we still fail to ob-
tain perfect sight, we must look for irregularities in refraction,
opacities, and for deficient perceptive power in the retina or optic
nerve.
The chief obstacle to a correct result by subjective examination
is the patient's accommodation. This may be set aside by atropia,
but in practice one will resort to it only when its use is clearly in-
dicated ; as for example, when there is severe pain, when contradic-
tory results are obtained after patient trial, when subjective and
objective methods give opposite findings, when one sees by the oph-
thalmoscope that spasm of accommodation exists. With children
we resort to atropia more often than with adults. The more we
ERRORS OF REFRACTION.
71
can rely on objective methods the better, and increasing skill will
indispose one to subject patients to the inconveniences of prolonged
mydriasis by atropia. Some advocate homatropia, gr. xx. ad 11.
as a sufficient substitute for atropia and preferable because the
effect is less prolonged. In this practice I have little experience
(see p. 68). To dilate the pupil, cocaine is better than homatropine,
while to paralyze accommodation where there is reason for doing it,
we have no substitute for atropia. Duboisia may be left out of view
in this statement, because it is even more potent than atropia.
After a preliminary effort with the test types and spectacle box
we take the ophthalmoscope. The indirect method may give in-
formation as to the refraction, by using the instrument which
Schmidt-Rimpler has suggested, but it is not the most natural
and requires special apparatus. We rely on the direct method
and while some claim higher accuracy, it is usually easy to come
within 1 D of the true state of the refraction. Illumination from a
distance of twelve to twenty inches will discover high degrees of
error in the display of such retinal vessels as may lie across the
pupil. They appear with a hyperopic eye in a virtual image, and as
the observer moves his head they go in the same sense. With a
myopic eye they appear in a real and inverted image, and as the
observer moves, they travel in the contrary sense. Coming as close
as possible, the observer with both eyes open to aid in perfect re-
laxation of his own accommodation, must also know and allow
for any errors in his own refraction. Decided myopia on the part
of the observer is a disadvantage, because the lens required for
correcting a patient's myopia, added to that which the observer
uses, will sometimes be so strong as to make inspection extremely
unsatisfactory. As between observer and patient, errors of a like
kind must be added to each other, errors of an opposite kind are to
be subtracted from each other. An observer with myopia 3 D ex-
amining a patient with M 5 D will need — 8 D. If the patient have
hyperopia 5 D, the same observer will see with -f- 2 D. If the pa-
tient have hyperopia 3 D the supposed myopic observer will see
without any glass. On the other hand the hyperopic observer will
add the glass to correct his error to that of the hyperopic patient,
and will subtract it from that of the myopic. An astigmatic ob-
server should have his correction placed upon his ophthalmoscope.
Some modification of the above statements will presently be made.
What is to be taken as evidence of a proper correction ? Usu-
ally the very fine vessels are so regarded, but besides them one
should have a clear sight of the granular look which belongs to the
pigment epithelium in the region of the macula. If no glass can
give this effect, suspect astigmatism, or haziness of the media. The
former will have to be ascertained, and a cylinder placed behind the
mirror should clear the fundus. If it does not, the media are hazy,
72
DISEASES OF THE EYE.
and this will be proved by a strong convex lens, say 15 D, with feeble
light and viewing the eye from a point two to four inches away.
We first bring the optic nerve to view and turn on the strongest
convex or weakest concave glass which will clearly display the fun-
dus. In doing this, it is a decided advantage not to be obliged to
remove the instrument as one makes changes in the glasses. A
hyperopic patient may betray efforts of accommodation, in the vari-
ation of the clearness which a vessel exhibits while viewed by the
same glass. This may compel resort to atropia. On the other
hand, when inspecting myopia the observer is tempted to use a glass
which is too strong and thereby bring into play his own accommo-
dation. Finally, after patient trial, the fundus is clearly seen; the
glass may be + 4 D. Is this correct for the patient's use supposing
the observer to be emmetropic ? It will prove to be too weak by the
distance at which the observer holds it in front of the eye and the
place at which the patient wTill wear his glass. Suppose the glass is
— 8 D. This will be too strong and by the same difference. With
weak glasses the difference is unimportant, with stronger glasses,
and especially with concave glasses, it must be considered. For in-
stance, if the observer's glass is 1| inches from the patient's cornea,
and the latter wear spectacles at | inch from his eye, 1 inch must
be added to the focal length of the glass. For instance, — 8 D is a
glass of 5 inches negative focus; to 5 add 1, making 6. Thus the
fraction ^ gives — 6.66 D as the glass required. So if the glass by
the ophthalmoscope be — 15 D: reduce this to focal length, viz.: ff
=2.66. To this add 1 inch, making 3.66. To find the glass in dioptries
^*#B-=—11 D. The difference becomes large. Hyperopia seldom
becomes as high as myopia, even after extraction of cataract. If,
however, the glass by the ophthalmoscope be + 5 D, this in focal
length is -f- 8. From it subtract 1 inch, making 7 Then 4f = 5.7 D,
which will be the proper glass.
With hyperopic eyes the illumination is brighter, and the mag-
nifying power less than in emmetropia. With myopic eyes the il-
lumination is feebler, the magnifying power greater and the field
smaller than in emmetropia. Hence with extreme myopia, say of
13 D and higher, examination by the upright image is difficult and
not satisfactory. In such cases one may take advantage of the fact
that an inverted image of the fundus is formed in front of the eye
at its own far-point and may inspect it with a convex glass. For
example, if the eye be myopic 13 D there will be an inverted image
at about 3 inches in front of the cornea, which may be examined by
a plus lens of 6 or 8 inches focus. The field will be small but the
image bright, and the degree of myopia can be approximated by
measuring the distance between the observer and the patient and
subtracting the focal length of the convex glass.
ERRORS OF'REFRACTION.
73
The foregoing statements apply to anomalies of refraction cor-
rective by spherical glasses. If now we have to do with astigma-
tism, we may not only discover the fact ophthalmoscopically, but
can, within certain limits, estimate its degree by spherical glasses;
it is not practicable to have a series of cylindric glasses attached
to the ophthalmoscope. The essential quality of astigmatism is
that the degree of refraction in a given meridian is greater or less
than in a meridian diametrically opposite; the difference between
these meridians is the amount of astigmatism.
It follows that we have to discover which are the principal meri-
dians of refraction, viz.: the least and the greatest, and each must
be studied by itself.
Now, in examining the fundus by the direct method, if we
find that fine vessels in the horizontal meridian need no glass
for distinct perception, while fine vessels in the vertical meridian
need a plus glass, we have simple hyperopic astigmatism. If we
need a plus glass for any vessels and a stronger glass for other
vessels, this betokens compound hyperopic astigmatism. In the
same way we recognize simple n^opic and compound myopic astig-
matism. The degree is the difference between the two meridians.
The rule in examining hyperopic eyes, is to use the strongest con-
vex glass which is available, and, in examining myopic eyes, to use
the weakest concave glass. Now, in eyes having a decided amount
of astigmatism, viz., 2 D, a streaky appearance is produced, and
the streaks will run in the axis of the greatest ametropia; of course
the least ametropia will be at right angles. Moreover, it will be
impossible by any spherical glasses to gain a clear view of the fun-
dus. So noticeable is this fact that one is incited to examine for
haziness of the vitreous, or erroneously led to think that the retina
is infiltrated with inflammatory effusion. Such an error is obviated
by finding that visual acuity by proper correction is satisfactory,
and if the proper cylindric glass can be attached to the ophthal-
moscope out of the trial-box, obfuscation of the fundus vanishes. I
have provided the means of doing this in the ophthalmoscope figured
on page 45, and gain the advantages of learning, first, that the deep
ocular structures are or are not healthy; and, second, that the find-
ing by the trial-glasses is or is not correct. Another feature in
astigmatic eyes is that the optic disc is no longer circular; it is
elongated in the direction of greatest ametropia, and therefore is
oval. The nerve may be misshapen anatomically, presenting a dis-
tinct oval, the long axis usually more or less vertical. In such a
case the retinal vessels will show no difference of distinctness
caused by their various directions.
By the inverted image the streakiness of the fundus can well be
seen in high degrees of As, but the lines run in directions opposite
74 DISEASES OF THE EYE.
to their course when viewed by the upright image. So, too, the oval1
of the optic disc is reversed. But the objective lens must be held
from the eye at a certain distance. It has already been said that
in the upright image the optic disc is elongated in the direction of
the meridian of greatest curvature, because the magnifying power
is greater. With the inverted image the elongation corresponds
to the weakest meridian. With the emmetropic eye the size and
form of the optic disc undergo no change in the inverted image
when the objective lens is held nearer to or farther from the eye.
With the hyperopic eye, when the objective approaches it the optic
disc becomes smaller, and grows larger as the objective recedes.
With the myopic eye, when the objective approaches it, the optic
disc becomes larger and grows smaller as the objective is held far-
ther away. In both H and M the shape of the disc remains round
or oval, whatever the distance of the objective. But with astigma-
tism the location of the objective changes the size and the shape of
the disc. If with the lens near to the eye the disc be vertically
oval, the disc becomes circular if the lens be held from the eye a
distance equal to its focal length, plus the distance of the anterior
focus, viz., half an inch. If it be drawn farther awTay beyond its
focal length, the direction of the axis of the oval is reversed. These
•phenomena have been elaborately studied by Javal and by Giraud-
Teulon, and can be utilized in diagnosis, but the upright image is
by far the most available and instructive.
The principles now presented have other applications and they
may here be stated.
Certain important pathological conditions are revealed by the
employment of the ophthalmoscope as an optometer. We are en-
abled to measure the depth or height of an object by knowing the
number and nature of the glass with which we can view it. Such,
for instance, is the depth of excavation of the nerve in glaucoma,
the height of a tumor, the elevation of a detached retina, the posi-
tion of a body floating in the vitreous. For instance, we find the
edge of a glaucomatous cup is to be seen with + 24 (+ 1.50 D); its
bottom requires — 16 ( — 2.50 D), the depth of the pit is -fa + fa = fa
=fa, or 1.50 D + 2.50 D = 4 D. By referring to the table on pages
89 and 90, we find that H fa (or + 1.50 D) means shortening of axis of
0.47 mm., while myopia fa or — 4 D means lengthening of axis of 1.37..
The depth of the cup then equals 0.47 -f 1.37 = 1.84 mm. On the
other hand, swelling of the optic nerve in neuritis may permit + 8
for its summit, and the eye be emmetropic A shortening of the vis-
ual axis of ^ = 1.50 mm. which measures the amount of swelling.
The same principle applies to all other cases above cited, and by it
we are able to give precise data in the facts and progress of a ca se.
An interesting case was one of myopia of — 7 D (—-i-), which gives
( ERRORS OF REFRACTION 75
elongation of axis of 2.13 mm. In the e}Te there was detached ret-
ina, whose conspicuous part or summit was seen by 4- 7 D or + {,
which means shortening of axis of 1.76. The true elevation of the
retina therefore was 14 D: equivalent to 3.89 mm.
In cases of this kind the inverted image has some value; if we
move the objective lens from side to side, the parts of the object
which are highest and those which are lowest will not move to an
equal degree; in other words, their parallax will be unlike, and they
will appear to be displaced unequally. The top of a swollen nerve is
nearer than its bottom, and the motion of the objective lens causes
its image to have less excursion at the top than at its bottom.
The same thing, to a less degree, can be exhibited in the direct image
Fig. 29.
by moving one's head. So too, with a glaucomatous nerve, the
vessels on the edge of the disc, as the lens is moved up and down,
move in front of and faster than do those at the bottom of the
nerve. For explanation, see the diagram (Fig. 29) from Abadie.
Let b be the edge of the excavation, and a lie at its bottom, and
the images of these points along the axis of the lens will be at B and
A respectively; cA is of course shorter than cB. When the lens
is moved down, the points A and B are displaced to A' and B' as
seen in the figure. They are no longer in the same line, because
the surface of the excavation, a, b, presents itself differently ta
the lens. The point B' moves faster and farther than the point
A', and passes in front of it, because c' A' and c' B' become, as it
were, radii of arcs of circles.
A special modification of the indirect method has been made.
76 DISEASES OF THE EYE.
by Schmidt-Rimplerl for determining the state of refraction. It is
not difficult, and is moderately accurate, and the observer is not
compelled to relax his accommodation; yet its being a rather cum-
bersome apparatus has prevented its general acceptance. Recently
Warlomont and Loiseau have constructed another instrument
based on the same method.
Yet another objective method of ascertaining the state of re-
fraction is to be mentioned which was prominently brought for-
ward by Cuignet in 1873 and which has gained considerable cur-
rency in England, and is growing into use elsewhere. Cuignet called
it keratoscopy, under a false idea of what the method really is, and
various names have been given to it, viz.: pupiloscopy, retinoscopy,
skiascopy, etc. The most common name and in a certain way suita-
ble, although not truly correct, is the
Shadow Test.—When the eye is illuminated by the ophthalmo-
scope, only a small part of the fundus is covered by the light, while
all the rest is obscure. If the mirror is turned at various angles
the illuminated surface correspondingly shifts and we may see in
the pupil a portion both of the luminous and of the non-luminous
surface. The latter is spoken of as a shadow, which is physically
incorrect, but is a term convenient for use. Its formation is illus-
trated by casting upon a screen the image of a gas flame by a 2-
inch convex lens. The image is bright, but there surrounds it a
dark circle which has nearly the diameter of the lens. This non-
luminous or dark circle corresponds to the " shadow" in the test
we are considering. The lens has deflected the rays to form the
image and the surrounding space is in darkness. If now in illumi-
nating the eye we turn the mirror in oblique positions, the shadow
appearing in the pupil will have certain peculiarities which depend
upon the quality of the mirror and upon the refractive state of the
■eye.
The mirror may be plane or concave. Because of its greater sim-
plicity we will assume the mirror to be plane. The observer sits in
front of the patient at three or four feet (say one metre) distance,
the light will most conveniently be above his head, and the pupil
must be moderately large; a mydriatic is frequently required. If the
mirror is rotated upon a vertical axis, the shadow moves transversely
across the pupil presenting a vertical edge and tests the refractive
quality of the transverse meridian. With emmetropia, hyperopia
and slight myopia the shadow moves in the same sense as the mir-
ror, or " with" the mirror. With myopia higher than 1 D the
movement of the shadow is opposite to the expected effect or is
" against the mirror." To understand the phenomena see Fig. 29,
1 " Augenheilkunde und Ophthalmoskopie," Braunschweig, 1884.
ERRORS OF REFRACTION.
from Nettleship. Let L be the light and M the plane mirror. The
virtual source of light is an erect image of the flame situated as far
behind the mirror as the flame is in front of it, viz.: four to six
feet. An image of the flame is formed upon the retina at I. If the
mirror be turned to the position of M' the retinal image shifts to I',
and the shadow will appear from the left hand side as the move-
ment of the mirror would suggest, and coincide with its motion,
providing the eye is either emmetropic or hyperopic. If, however,
the eye is myopic (see 2 in Fig. 30), the image of the flame is
formed in front of the retina and the rays cross; and emerging
from the eye they form a real and inverted image between the eye
and the mirror which moves in a sense opposite to the rotation of
Fig. 30.
the mirror because the rays now cross before reaching the observer;
the shadow is therefore against the mirror. If the myopia be so
feeble that the inverted image of the luminous area of the retina
falls beyond the mirror, the shadow will move with the mirror.
The retinal vessels will appear, but are disregarded.
Suppose we use a concave mirror of 7 or 8 inches or 20 centi-
metres focus (see Fig. 31, from Nettleship). The virtual source of
light is the inverted image of the flame L found at I, and a second
image again inverted is formed at I' in the eye. This image will
be distinct and bright if the far-point of the eye is at I, but other-
wise it will be out of focus and the luminous area will be less bright.
We will suppose the eye to be myopic (No. 2, Fig. 31). It forms a
■78 DISEASES OF 1HE EYE.
real image of the illuminated surface V at F. If the^ mirror be
turned, I' will move to I'2 and the image will shift to I"2. That is,
the image seen by the observer moves in the same direction with
ithe mirror.
If the eye be hypermetropic or emmetropic, the rays coming
iXl 3 I
3C--4 Y\"3C
:-i:
Fig. 81.
.^~~T*
from the eye being divergent or parallel will not be brought to a
focus, and the observer will see a virtual erect image at I" (No. 3)
the virtual focus of V and see its movements as they actually occur,
i.e., in the same direction as the movements of the real image I' or
I'2 and therefore " against" the movements of the mirror. If the
ERRORS OF REFRACTION.
79
myopia be of low degree, viz.: about 1 D, the rays emerging from
the pupil are focussed at about one metre and if the observer
intercept them before they meet (No. 4) he will refer them to
I" and I"2 and obtain an erect virtual image of I' the movements
of which will be the same as in H or E (No. 3), viz.: against the
mirror.
To sum up: with a plane mirror the shadow in emmetropia,
hyperopia and low myopia will move icith the mirror. In myopia
greater than 1 D, the shadow moves against the mirror. With a
concave mirror the shadow in emmetropia, hyperopia and low
myopia moves against the mirror. In myopia greater than 1 D it
moves with the mirror.
In practical work a spectacle frame is put on the patient or a
disc bearing glasses is rotated in front of the patient's eye (Brailey)
and various glasses are tried until one is found which just reverses
the movement of the shadow, or which causes the shadow and the
illumination to behave as in emmetropia. This lens is very nearly
the desired glass. In hypermetropia we must subtract about 1 D
from the lowest plus lens which reverses the shadow; and in myo-
pia 1 D must be added to the lowest minus lens which reverses the
shadow, because the reversal of movement will not occur until a
slight excess in the strength of the glass has been produced.
The higher the ametropia, the less luminous will be the pupil,
and the less distinct the shadow. We ma}' give attention to some
other points besides the direction in which the image or the shadow
moves, viz.: to its brightness, to its rate of movement and to the
form, straight or crescentic, of its border. If the eye be emmetro-
pic, or nearly so, the image will be most correctly focussed and
hence at its brightest, whereas, if notably out of focus the rays are
more dispersed. For the same reason the border of the image will
in emmetropia be best defined and straighter because the flame
has nearly vertical borders—if out of focus the image has a more
rounded and blurry outline. The movement of the image is most
rapid in emmetropia.
The detection of astigmatism becomes easy by varying the
movement of the mirror about different axes—vertical, horizontal
or oblique. If, for example, the reversal of the movement of the
shadow is secured when the movement is transversely across the
pupil and ametropia still remains in the opposite meridian when
the mirror is moved in that direction, another lens must be found
which will correct the latter error. Of course the difference be-
tween the two lenses thus found is the degree of astigmatism.
Both the kind and degree of astigmatism are thus made known.
Irregular refraction or opacities in the cornea and lens will also
be brought to view. Nettleship says: "for the quick discovery
80
DISEASES OF THE EYE.
of very slight astigmatism and of the direction of the chief meridian
in astigmatism of all degrees, retinoscopy probably excels all other
methods."
In his further observations the writer fully concurs: that accu-
rate retinoscopy (skiascopy) is not quicker than measurement by
the direct ophthalmoscopic method and that in fact the latter is de-
cidedly the more rapid if one have a good instrument. " I cannot
help thinking," says Mr. N.," that the importance of retinoscopy has
been somewhat overrated, and that though in some difficult cases
it will remain our best objective test, we shall do well generally to
use it as an auxiliary, rather than as a substitute for other
methods." The ability to employ the shadow test well is worth
possessing, but it can never attain the value of direct ophthalmo-
scopic examination of the fundus, because it teaches nothing of
the actual condition of the structures.
By the methods discussed we are enabled to diagnosticate
the kind of refractive error present, and we next have to cor-
rect it. WTe resort to trial glasses. We examine at the distance
of six metres or as near to that as may be feasible, and endeavor
to bring vision up to normal standard by the strongest convex,
or weakest concave glasses which will be accepted. Some assist-
ance is gained by noting the distance at which the patient can
read fine print. If decidedly myopic, he must hold it near, say inside
of ten inches, but he will do the same if greatly hyperopic, and also
amblyopic, or if astigmatic, or if there be insufficiency of the recti
externi muscles. If to read print it must be held very near, and
distant sight be very bad, try a concave glass, whose focal length
is about the distance at which the book is held from the face; this
should greatly improve distant vision if there be myopia, while it
will be rejected in most other kinds of refractive error. Should the
book in reading be held at a distance beyond fourteen inches there
may be hyperopia, but the probabilities are that the fault is princi-
pally in the accommodation. Special details in choosing glasses
will be given under the special kinds of error to be treated.
It must be remarked that special instruments to take the place
of the box of trial glasses have been contrived, called optometers.
Some are based on the principle of the Galilean telescope, viz.:
those of Graefe, of Perrin and Mascart, of Snellen; some have a
single convex lens (Burow, Badal, Sous, etc.), and test types are con-
tained within the tube of the instrument; some have two convex
lenses placed a distance from each other equal to the sum of their
principal foci (Hirschberg, Plehn), and in using them the types
are hung upside down across the room. In some instruments the
focus of the lens coincides with the patient's nodal point as recom-
mended by Nagel (Hirschberg, Plehn), the object is to avoid, as
ERRORS OF REFRACTION
SI
much as possible, magnifying the print. Without discussing these
apparatus in detail (see article by Plehn1), it may be remarked that
they are useful when a large number of examinations must be made
rapidly, as among recruits in military service, but they are apt to
provoke efforts of accommodation and they do not serve for read-
ing. There is also an instrument based on wholly different princi-
ples, called the prismoptometer, to which the same objections apply.
Still another mode of testing vision subjectively is based upon
the experiment of Scheiner, viz.: looking through two small holes
about three or four millimetres apart in an opaque disc The
effect of this contrivance is seen in Fig. 32. If the emmetropic eye
E be focussed for the object O, it will form a sharp image, notwith-
standing the splitting of the beam of light into two small pencils,
because each will fall upon the fovea. For the hyperopic eye H
Fig. 32.
whose retina is nearer the nodal point, the small pencils will each
make a separate confused image upon it. The same will be true
of the myopic eye whose retina is too far from the nodal point,
viz.: that each pencil will make its own circle of dispersion. Both
the hyperopic eye and the myopic will therefore see the object
doubled: it being supposed that the hyperopic makes no effort
of accommodation. This device was employed by Porterfield
many years ago, but has lately been made practical by Dr. Thom-
son, as follows: If across the upper hole of the screen a red glass
be placed, and the object be at twenty feet and be a very
small gas flame, or a small dot of light, then ametropic eyes
will not only see two lights, but one will be red and the other
white. For the hyperopic eye it will be seen by the dotted lines
going through the nodal point k (see Fig. 32), that the projection
of the spot which is uppermost on the retina is below the axis, and
if the red glass be on the upper hole of the screen that image is
red. It therefore follows that wiien the red image appears to the
side of the axis opposite to the side where the red glass is placed,
this diagnosticates hyperopia. On the other hand, if the red image
be on the same side of the axis with the glass, this declares myopia.
1 Archives of Opth., xvii., 1, 74, 1888.
82
DISEASES OF THE EYE.
It is further evident that the greater the ametropia the wider
apart will be the two images. It follows that the kind of diplopia
and the interval between the images give both the kind and ap-
proximately the power of the correcting glass. To make the esti-
mate of the power of the glass fairly accurate. Dr. Thomson has so
arranged two small gas flames upon a measured bar that each
being doubled, one is moved from the other until instead of four
flames, three only are seen by the coincidence of two. This dis-
tance is read off upon the rod, which is divided into centimetres,
and the refraction is deduced. Still more simply the refraction is
given by putting in front of the screen the convex or concave glass
which causes the images to approach until they coalesce. The
objection to this is that myopics will be likely to use accommoda-
tion and get glasses too strong and hyperopics to get glasses too
weak. Dr. Thomson's use of two flames obviates this liability.1
This method is of value in some difficult cases of refraction
where the media are irregular in curve, viz., in conical cornea, or are
opaque. With dilated pupil it is easy to apply. It is always most
effective with a large pupil, when the holes may be four millimetres
asunder and can be used in a dark room and for illiterate persons.
The theory of this method and the mathematical formulas are
given by Nagel, G. and S., VI. p. 412.
For the general theory of optometers, see Nagel, 1. c, p. 315 et
seq.
We now take up special refractive errors in detail.
Hypermetropia.
Hyperopia—H.—Hypermetropia is the condition in which, with
suspended accommodation, a person requires a convex glass to get
his best acuity of sight for distance. It is essential to put aside
the accommodation, because up to a certain age and for small de-
grees it overcomes and conceals the hyperopia. But it is not to be
expected that all cases of high degrees of H will with glasses gain
V— 2JL
v — 20 •
Many who need + 10 (4 D) or stronger glasses, have V= f# or
|-8-. In fact, the greater number of strongly hyperopic persons do
not have normal acuity of sight.
It is the optical result of a shortened visual axis or of want of
the crystalline. It also appears in later life, after sixty, by flatten-
ing of the crystalline, being the outcome of advancing presbyopia.
Flattening of the cornea may also cause it through distention of
the globe, as ensues in glaucoma. The usual cause is shortening of
1 See Trans. Am. Opth. Soc, 1870, p. 93, and 1873, p. 83.
ERRORS OF REFRACTION.
83
the visual axis. It is a congenital condition. At birth it is almost
universal, viz., in 92.4$; up to the fifth year we find 84$; among
children in elementary schools 76$; among pupils in higher schools
56$. The degree of H at the second year of life averaged in 300
cases 2 D. For a summary of all recorded investigations of re-
fraction by different observers, amounting to 22,743 cases see
Randall, Bericht Int. Ophth. Congress, Heidelberg, 1888, p. 511;
also Am. Journal Medical Sciences, July, 1885.
Anatomical Characteristics.—In his classical treatise which is
the foundation of the knowledge of the present day on this subject,
Donders portrays many features which characterize the physiog-
nomy of hyperopics. That they often have narrow faces and shal-
low orbits and eyes deep set, is true, but no conclusive deductions
can be drawn from such appearances. It is evident that the axis
of the eye increases in higher ratio during the early years of
growth than do the optical parts.
Hyperopia necessitates an effort of accommodation proportion-
ate to its degree, and the result is, that if great, the ciliary muscle
becomes enlarged and in adults its size and contour have a well-
marked and conspicuous character. This has been exhibited by
Ivvanoff. If hyperopia is less than 3 D or 4 D vision generally is
normal, if higher it is rarely normal. The explanation is found
probably in the wrant of development of the retina and optic nerve.
The supposition that the same number of rods and cones as in
emmetropia are condensed into a smaller space is inherently im-
probable. Donders pointed out that the macula lutea is farther to
the temporal side than in emmetropia, and Landolt and Dobrowlsky
proved that the distance between the papilla and the macula is
greater than in normal eyes. The cornea is more decidedly decen-
tered than in emmetropia and the angles gamma and alpha are
usually very large. With the higher degrees of H the eyeball is
evidently small and proclaims its imperfect development not only
in this fact, but even in imperfect vigor of the ciliary muscle and of
the motor muscles. The optic nerve in marked degrees of H often
shows indistinctness of tissue, it may be deep red or grayish, its
edges are often striated and ill defined, especially on the nasal
side. It may be, and in high degrees it usually is, intensely red,
and because of such signs some have regarded the condition in-
flammatory. This, however, can hardly be sustained by clinical
experience. What is known as the choroidal crescent, viz.: the
interval between the edge of the choroidal and the scleral open-
ings is not infrequently observed, and it is usually on the temporal
side. Sometimes the nerve is anatomically oval, while the oval
shape due to astigmatism is also frequent. Even the retina in the
neighborhood of the nerve sometimes is hazy and seems thick.
84
DISEASES OF THE EYE.
While these appearances belong to higher degrees of H, we shall
find in all hyperopic persons having symptoms of asthenopia, that
the nerve is red, perhaps very deeply, and the veins are large—in-
dicating reflex irritation of the papillary circulation.
Symptoms.—In using their eyes, hyperopes of moderate degree,
or rather such as have adequate accommodation and good muscles,
exhibit no difference ordinarily from emmetropes. If examined by
glasses they may not accept them. In such persons the whole error is
latent. On the other hand, they may see equally well at six metres
with and without a given glass. The glass which they accept rep-
resents the " manifest H." Under atropine such a person would
show a higher degree and accept a stronger glass; the repressed
amount is the " latent H." The " total H " is of course the sum of
the manifest and the latent. It is also possible that a hyperope
requires a weak glass to give him the best vision and will also see
equally well with a stronger one. Suppose he needs + 1.5 D for
vision f and will accept + 3 D. It is evident that his manifest H
=3 D. Of this he is able to correct 1.5 D voluntarily, and this part
of the " manifest H " Donders calls " facultative." The total H can
be usually seen by the ophthalmoscope, and can be elicited by
using atropine in full dose. But my own experience agrees with
Schweigger's ("Handbuch der Augenheilkunde," p. 49, 1880) that
exceptional cases occur which do not yield up their total H to the
ophthalmoscope. See also article by Weiss (Graefe's Archiv fur
Ophthal., XXIV. ii., 90), one by Agnew (Trans. Am. Oph. Soc).
I observed one such person, a lady aged 32, for three months and
could not make out an error which finally under atropia proved to
be 2.5 D. Persons with high degrees of H, especially if they be no-
tably amblyopic, will bring print and work very close. They will
behave as if near-sighted. They will seek a strong light, will see
badly in the evening. They often have small pupils. They apply
for relief either because distant vision is indistinct, or when they
have symptoms of asthenopia. Distant vision fails when the ac-
commodation can no longer correct the error. It is evident that
with parallel axes, the hypertrope has been accustomed to exert an
undue degree of accommodation. As this faculty declines with
years, he finds himself obliged at an early age to seek the aid
of convex glasses, and he also is apt to find that a weak convex
glass improves and restores to him his former acute distant sight.
This is what usually transpires with moderate H in healthy and
vigorous persons. An attack of sickness, especially of diphtheria,
chronic ill health, notably uterine diseases, and extreme taxation of
sight, may cause the same results before the usual decline of accom-
modation would develop them. Bnt it is more common under the
influence of such causes for the subject to suffer, not from defective
ERRORS OF REFRACTION.
85
sight either for near or far, but from pain in close eye-work. This
is called accommodative asthenopia. The symptoms are that
after working for a certain period the person must stop because
the sight is blurred, there is pain in and about the eyes and some-
times remote reflex symptoms, such as headache, pain down the
spine, sickness of stomach, etc., etc. The ability to resume work
after rest, to work better under stimulus, that the periods of ability
to work grow shorter:—these are characteristics of this condition.
Many more touches might be given to this picture, but they would
serve no good purpose. It may, however, be added that palpebral
irritation, chalazia, styes, blepharitis marginalis, and hyperaemia of
the palpebral conjunctiva are frequently seen. The inflammations
of the border of the lid occur most often in young subjects and fre-
quently without any complaint of distressful sight. This is a well
established fact and was noted by Roosa and Schirmer and has long
been familiar to the writer. Palpebral conjunctivitis is almost
always present when asthenopic symptoms exist, and explains the
sensations of smarting, dryness, heat and the inclination to press
upon the globe—as well as the comfort gained by applications of
lukewarm water and soothing lotions.
Hj^permetropia makes its appearance in the order of nature be-
yond sixty or seventy years of age, and then dimness of sight for
distance, correctible by a weak convex glass, is the symptom ob-
served. Usually there is no asthenopia.
Another class of cases exhibit the effects of their error in dis-
turbances of the muscles; they are either inclined to converging
strabismus or it becomes fully established.
We have spoken of the intimate relation which in emmetropia
subsists between accommodation and convergence. For a given
number of dioptries of accommodation, say 4 D, an equal number of
metric angles must be brought into use, i.e., 4. With hypermetro-
pia the law is not the same. On the contrary, with no convergence
the subject must put forth as many dioptries of accommodation as
will equal his total error. If hyperopia = 4 D and he accommo-
date for 250 mm., he uses 4 + 4D-8D of accommodation for 4
metric angles of convergence. At once a disproportion appears,
which may easily be converted into a complete disturbance of binocu-
lar vision, if for any cause there be difficulty in distinct vision or in
convergence. The two functions are in a state of unstable equilib-
rium and are easily disassociated. It has never been shown how large
is the proportion of hypermetropes who acquire strabismus, but
Donders showed that about two-thirds of the cases of strabismus
convergens were hypermetropic. The latitude which exists in the
amplitude of convergence for a definite amount of A, and the varia-
tion in relative A for given degrees of convergence, explain why so
86
DISEASES OF THE EYE.
large a proportion of hyperopic subjects never fall into strabismus*
But that they are subject to this tendency is evident. It must also
be remarked that other refractive complications are frequent in H,
viz.: astigmatism and unequal refraction of the two eyes (anisome-
tropia). To this must be added that often there is unequal visual
power in the two eyes and inferior visual acuity of one or both
forces the adoption of a distance for near work, closer than the
degree of H would demand. It is not found that hyperopic persons
choose a wTorking point more removed than emmetropes; on the
contrary, their inclination is to adopt a closer near-point, until the
failure of accommodation brings on premature presbyopia.
The occurrence of strabismus is evidently determined by a variety
of factors and among the most potent is the actual power of the
muscles of adduction and abduction. Efforts of accommodation
stimulate the interni and the effect is reciprocal. It is a subject
worth investigating on a large scale what is the adduction and ab-
duction of hypermetropes who do not squint. (See " Die Aetiologie
der Strab. conv. hypermet." von Dr. Richard Ulrich, 1881.) The
subject will be referred to again under the head of strabismus.
Complications.—It has been said that amblyopia, astigmatism,
inequality of the eyes both as to refraction and vision, strabismus,
spasm of accommodation and asthenopia may coexist with hyper-
opia. It is recognized to be a frequent concomitant of glaucoma and
is regarded as predisposing to it, while that it may predispose to
idiopathic retinitis (Dobrowlski) is not probable.
Diagnosis.—The acceptance of a convex glass for distant vision
and that by it vision is not impaired or may be improved, is conclu-
sive of hypermetropia. Its non-acceptance does not disprove it. If
accommodation is fully paralyzed by atropine non-acceptance of
the glass disproves it. With this must be combined objective ex-
amination, viz.: by ophthalmoscopy, by Cuignet's method (shadow
test), and for decided proof it must be possible to eliminate accom-
modation both from the observer and the patient. In most cases the
upright ophthalmoscopic image will settle the matter easily, by
showing that a convex glass permits clear vision of the fine retinal
vessels near the macula, or of the retinal epithelium (the granular
look). For H less than 1 D good observers may be in doubt with
objective methods, but if needful, atropia will determine the point.
Prognosis.—The error is not curable, but is correctible. The
degree of vision will not be impaired, but as the accommodative-
power becomes exhausted, stronger glasses will be needed, and in
higher degrees of H one pair will be required for distance, and a
second for the near-point. The endurance of hyperopic eyes usually
falls below the normal amount, and as they come to use high glasses-
for work, this disability is liable to increase. They often feel the
ERRORS OF REFRACTION.
87
necessity of constantly wearing glasses to be a grievous burden,
but the prognosis is relatively good.
Treatment.—We have no occasion to deal with these persons
until some of the above-mentioned subjective symptoms cause
them to ask for relief. So long as their accommodation can with-
out conscious strain overcome their error, they need no aid.
1st. In the simplest cases the first call for help is when they find
near vision indistinct or tiresome, i.e., when A is not large enough to
easily overbalance H. We usually meet this in persons from 25 to
40 years of age. They may accept a weak glass for distant vision,
but without it may have V = f. It is in reading, etc., that they
find the print blur and the light seem bad and the eyes grow tired.
We always test each eye separately and cover the other by a
screen. The convex glass which makes reading comfortable,
whether .75 D,or 1 Dor 1.5 D,is all that they need. This they may
use at discretion, and there is no occasion for a distant glass and
atropine need not be used in the examination. Let the same person
grow older and find distant vision a little hazy, then he may take
the convex glass which restores its sharpness and at the same time
he will need a stronger working glass and will have to make habit-
ual use of it. The glass for distant sight he may use at his plea-
sure for looking at pictures or at persons in an audience, etc In
time it may come to pass that without a glass, distant sight is un-
pleasantl}' obscure and then the person will gladly take refuge in
its continuous employment. His occupation may require sharp
vision at moderate distances, say at 10 feet, and he may even need
a series of three glasses to meet all his requirements. Portrait
painters find themselves sometimes in this predicament. It is con-
venient for them to have a glass ground with double foci. For-
merly such glasses were made in two pieces (Franklin's glasses),
now separate foci can be ground upon the same piece of glass.
The stronger should be below and it should occupy less area than
the upper. At the dividing line prismatic distortion occurs, but
generally the person can soon habituate himself to the glasses.
They must be larger than the ordinary form. Sometimes a slip of
glass is stuck to the lower portion of the weaker one by Canada
balsam.
2d. The person complains of asthenopic symptoms, and they
may arise either when at work, or be constant. In such cases
the error may be small and the distress be due to feeble health,
overwork, astigmatism, feeble muscles, etc., or the error may
be large, and there may be notable amblyopia. The mode of
dealing with such a case will depend upon the age, the general con-
dition of health and upon the intensity of the asthenopic symptoms.
It is in this class of cases that we are confronted with the question,
88
DISEASES OF THE EYE.
Is atropine necessary ? The case is to be looked upon as one of ac-
commodative asthenopia, and all the elements which enter into
this morbid complex, must be taken into account. A real attempt
must be made to correctly estimate all these elements without
resorting to atropine. First, one must test distant vision and find
the strongest convex spherical glass which will be accepted. If
visual acuity remains below the normal, the next inquiry is for as-
tigmatism, which will be hereafter discussed. If sufficient success
is not attained in bringing up the visual acuity, try the objective
examination by direct ophthalmoscopy or by the shadow test, to
see what glass, or combination of glasses, is indicated. Let the
patient use these for distant vision. He may or may not gain bet-
ter or correct acuity. Perhaps he now accepts a stronger glass
than at first. This means that accommodative tension is abating.
He may not consent to the stronger or the objectively ascertained
combination. His answers may be contradictory, he may evidently
feel uncertain what he sees, and may say that the type swims, or
comes and goes. The glass, objectively ascertained, may be much
stronger or be very unlike the glass which he is willing to take.
These symptoms betoken accommodative strain. If after a little
patience in pressing upon him glasses, which approximate to the de-
gree of error objectively ascertained, he accept them and acuity be-
comes | or |, let him take these and read with them for half an hour,
provided he be a person under 25 years. Usually these glasses, if
found satisfactory in reading, will serve the purpose and a mydri-
atic need not be employed. Such a proceeding will take considera-
ble time and patience, and it is assumed that the examiner has an
unlimited supply of the latter. It will be justifiable to give the
patient these glasses with the proviso, that he be told that they
may possibly not serve, and that if they cause trouble or fail of
relief he must come again. In other cases the examination may be
repeated the next day and then the patient is likely to be better
able to control his eyes and faculties. Glasses selected in this way
necessarily leave uncorrected what may be called the normally
latent error. The age and health of the subject have a controlling
influence over the amount of this fraction, which may be called the
physiologically latent error, and it necessarily varies with the
degree of H. To young and vigorous subjects whose subjective
symptoms are not severe and for whom visual acuity f or f can
be obtained, it is proper to prescribe glasses without resort to a
mydriatic. Whether the glasses are to be used only in near work
or at all times, depends on the degree of error, on the vision without
them, and on the amount of discomfort previously existing. If
constantly worn they sometimes become thereafter indispensable,
or in other cases, they in a little time bring about relief, and may
ERRORS OF REFRACTION.
89
be laid aside for distance and used only for the working point
(punctum agendi).
3d. But another possibility presents itself: we have not attained
satisfactory visual acuity, or the difference between the glasses
objectively found, and subjectively approved is too great, and the
patient's answers show that his accommodation is under severe
strain, or his subjective symptoms are severe. Further evidence
of serious spasm of accommodation is found in the variable sharp-
ness of the fundus under the ophthalmoscope, as various glasses are
employed. Under these conditions a mydriatic must be used.
Oftentimes the prolonged mydriasis of sulph. atropia, which will
extend to seven or ten days, is a serious inconvenience, at other
times it must be regarded as a therapeutic measure, as well as
needful for diagnosis. Then sol. sulph. atropia, gr. iv. ad § i., will
be dropped into the eyes from three to ten times within one to
three days according to the susceptibility of the patient; constitu-
tional symptoms are liable to occur, and warning must be given
accordingly. With milder subjective symptoms hydrobromate of
homatropine, gr. xx. ad § i., may be dropped in, every ten minutes
until relaxation occurs, and its effects will pass in twenty-four or
thirty-six hours. Sulphate of duboisia is our most active agent,
but has no special advantages. Muriate of cocaine 4$ solution has
much less effect on accommodation than on the pupil.
With suspended accommodation the full amount of hyperopia
will be discovered, astigmatism will be sought for, and while with
dilated pupil, acuity may not reach the normal, an ophthalmoscopic
examination by the upright image with the full correction both by
spherical and cylindric glasses will show whether defect of vision
is due to a real amblyopia. Further evidence of amblyopia will
be had by testing for a small central color scotoma, which is some
times found in one eye of hyperopes who have never squinted.
If the degree of H be important, fa or greater, it will very fre-
quently be advisable to give the glass which fully corrects H.
In most cases it will be best to wear it continuously. Under what
circumstances should convex glasses be constantly worn ? Some
people answer for themselves by finding that they are wholly un-
comfortable without them, even though the degree is not strong
The comfort of the individual is of necessity the fundamental rea
son for constant use, and that only in this way can it be secured,
is not always to be anticipated. That such use is likely to be need-
ful will be probable of cases of high degrees, viz., fa and more,
whether in young or old subjects; the more advanced in age the
person, the more likely is he to require constant help. Again, the
same advice is to be given to very sensitive persons whose eyes
give them much pain. Under this head will come a large class of
90
DISEASES OF THE EYE.
semi-invalids and impressible and neuralgic persons. On the other
hand, the dull and torpid and unobservant will often be quite in-
different to the aid of glasses for distance, even though they have a
marked degree of error. Furthermore, something has to be con-
ceded to the sense of what is becoming to their personal appearance
in persons of both sexes, and, while a physician will not modify his
deliberate opinion and advice in deference to what may suit his
patient's whim, there are doubtful cases in which his abstract views
must be modified by the patient's preference. For persons with
marked H there can be no doubt of the advantage gained by
constant use of glasses, because the range of accommodation is
brought within the physiological limits, and the continued strain
on the ciliary muscle is removed. Moreover, it is a frequent obser-
vation that in this way acuity of vision decidedly improves in the
higher degrees of H. This is not simply the effect of enlargement of
retinal images, but of improved health of the retina.
If the requisite glass is as high as 4 D it often causes discomfort
when first worn, because one's estimate of distances is disturbed
and more light is gathered into the pupil. Special care must be
taken with the frames that the centres of the glasses coincide with
the visual axes. If decentered in either direction a noticeable pris-
matic deflection occurs ; this may or may not be desirable. With
high degrees of H an increase of power in the glasses required for
reading is usually needful at an early age. A serious disadvantage
belonging to strong glasses is that they greatly restrict the range
within which reading is comfortable. The book must be held at
a certain and almost fixed distance. As an instance of extreme
hyperopia, I may mention a boy ten 3Tears old who required a
glass -§- of an inch focus or 33 D. To find hyperopia of 10 D or 12 D
is not excessively rare.
On page 83 some remarks were made on the prevalence of H in
early life. The results belong to examinations for the total H.
Among adults who were largely students and soldiers and after
eliminating the statistics whose value appeared to be doubtful
Randall finds in 22,743 examinations, Em. 23.8$, M. 21.4$ and H 51.9$.
Admitting that more than half the young adults of the com-
munity may, as these figures indicate, be hypermetropic, it by no
means follows that a large proportion will suffer inconvenience if the
error remains uncorrected. The state of health, the vigor of accom-
modation, the mode of life, and the exigencies of eye-work must all be
taken into account. Donders, in 1864, " Accommodation and Refrac-
tion," p. 174, said: " In an absolutely mathematical sense no single
eye is perhaps to be called emmetropic." Randall, in 1888, makes the
same observation (loc. cit.), and adds: " Approximate emmetropia
(Am. < ± 0.5) is infrequent at all ages, probably at no epoch exceed-
ing 10$." Happily the human race is not compelled to attain mathe-
ERRORS OF REFRACTION.
91
matical perfection, to enjoy a reasonable degree of felicity. It would
be as hard to find an individual perfectly healthy in body as to find
an absolutely emmetropic eye. A limited departure from a per-
fect standard must be admitted to be consistent with a normal eye.
The rigid scrutiny which reduces emmetropia to 23.8$ and makes
hypermetropia 51.9$ conveys a wrong impression of the relative
proportion of what their experience shows to be the number of
persons with normal eyes. The definition of such an eye may be
stated to be one that has acuity |£ or f| without glasses and that
can perform all necessary work without pain or fatigue. It would
be a serious error to insist on the possession of a mathematically
faultless instrument before we could accept a standard normal eye.
In a practical sense small errors of hyperopia or astigmatism
whether in the cornea or in the lens must be disregarded. Donders
placed these insignificant errors between Tfg- and fa. At the pres-
ent time the allowable limit would be somewhat less. But the ten-
dency now is toward what seems to me to be a needless degree
of exactness. More will be said on this point in the chapter on
asthenopia. The examinations of Seggel in the soldiers of the
Bavarian army are admitted to be exact and trustworthy, and he
reports on 3,052 eyes with the result of E. 46.7$, H. 40.6$, M. 11.4$,
and astigmatic and diseased eyes 1.3$. These were young and
healthy adults who had not been given to exacting eye-work.
Graefe's Archiv, Bd. XXX., 11., p 111, 1884.
The degree of shortening of the optic axis which corresponds to
a given amount of H is given in the subjoined table from Landolt:
Table of Axial Hyperopia.
Degree of H. Amount of shortening. Total length-of Axis. Degree of H. Amount of shortening. Total length of Axis.
mm. mm. mm. mm.
0 0 22.824 (normal). 8. 2.28 20.54
0.5 0.16 22.67 8.5 2.41 20.41
1. 0.31 22.51 9. 2.53 20.29
1.5 0.47 22.35 9.5 2.66 20.16
2. 0.62 22.20 10. 2.78 20.04
2.5 0.77 22.05 10.5 2.90 19.92
3. 0.92 21.90 11. 3.02 19.80'
3.5 1.06 21.7(5 12. 3.25 19.57
4. 1.21 21.61 13. 3.47 19.35.
4.5 1.35 21.47 14. 3.69 19.13:
5. 1.50 21.32 15. 3.91 18.91
5.5 1.62 21.20 16. 4.11 18.71
6. 1.76 21.06 17. 4.32 18.50
6.5 1.90 20.92 18. 4.52 18.30
7. 2.03 20.80 19. 4.71 18.11
7.5 2.16 20.66 20. 4.90 17.92
It will be seen in the table that the axial shortening for each D;
is about 0.3 mm. up to 7 D, but that for higher errors the abate-
ment in axis sufficient to cause 1 D becomes less and less.
92 DISEASES OF THE EYE.
MYOPIA. M.
There are two kinds of myopia. In one the refractive power
-of the media is excessive, while the optic axis is of normal length.
In the other and common form, undue elongation of the optic
axis is the essence of the error. The amount of actual increase
in length of axis may in general be stated for the lower degrees,
to be about 0.3 mm. for each D. Unlike hyperopia, the increase
of axis to make one additional dioptry of myopia becomes larger as
myopia increases. Beginning for 1 D at 0.32 mm., between — 5 D
and — 10 D the average increment for a single D is 0.41 mm. Be-
tween — 10 D and — 15 D the average increment for a single D
is 0.51 mm. Between — 15 D and — 20 D the average increment
for a single D is 0.60 mm. Exceptionally it has been shown that
Table op Axial Myopia.
Degree of M. Amount of lengthening. Total length of Axis. Degree of M. Amount of lengthening. Total length of Axis.
mm. mm. mm. mm.
0 0 22.824 (normal). 8. 2.93 25.75
0.5 0.16 22.98 8.5 3.14 25.96
1. 0.32 23.14 9. 3.35 26.17
1.5 0.49 23.31 9.5 3.58 26.40
2. 0.66 23.48 10. 3.80 26.62
2.5 0.83 23.65 10.5 4.03 26.85
3. 1.01 23.83 11. 4.26 27.08
3.5 1.19 24.01 12. 4.73 27.55
4. 1.37 24.19 13. 5.23 28.05
4.5 1.55 24.37 14. 5.74 28.58
5. 1.74 24.56 15. 6.28 29.10
5.5 1.93 24.75 16. 6.83 29.65
6. 2.13 24.95 17. 7.41 30.23
6.5 2.32 25.14 18. 8.03 30.85
7. 2.52 25.34 19. 8.65 31.47
7.5 2.73 25.55 20. 9.31 32.13
eyes whose axis comes within the usual limits of 22 to 25 mm.
may be myopic. Stilling measured twTo eyes with axes of 26 and 25
mm. respectively, in each of which the myopia had during life been
determined to be exactly 5 D. It is not found that the curve of the
cornea has any regular relation to miopia. I have found the
radius both shorter and longer than normal. As to the refractive.
index and curves of the crystalline we know nothing. It has been
shown that a few cases of myopia are congenital,1 and often the
subjects belong to the poorer classes of society, while the vast
majority of cases exhibit the condition after eight years of age.
Its beginning may date from the twelfth or fifteenth year of life,
or even perhaps later. The period of most rapid development is
1 Tscherning," Studien iiber die Aetiologie der Myopie." Graefe's Archiv
xxix., 1, 201.
ERRORS OF REFRACTION.
93
between the ages of twelve and eighteen. The proportion of myopes
in the community is estimated by Tscherning (Copenhagen) at 8$.
Great attention has been given to the production of myopia in chil-
dren, and as the result of the labors of many observers we have
statistics of over 70,000 cases.
We have seen that the normal condition of the eye at birth is
hyperopia, and that increase in length of the axis brings about
emmetropia, while a further advance will occasion myopia. It is
of great importance to discriminate between the moderate degrees
of M which are simply an inconvenience with some compensating
advantages, and the higher grades which are eminently serious
and often dangerous. Donders pronounced every myopic eye a
diseased eye. Every highly myopic eye is diseased, but within late
years it has been shown that the remark conveys a false meaning
respecting a large number, and these the moderately myopic eyes.
It will be admitted that up to 3 D the cases are seldom serious, it
being generally possible to give perfect vision by proper glasses.
Above 6 D we do not expect to procure correct vision, and look
for various pathological lesions and often there is great peril to
sight. Blindness, as the effect of detachment of the retina, of
intra-ocular hemorrhage, etc., is too often the outcome of myopia.
The cases between 3 D and 6 D may or may not be of serious
character.
M3Topic persons whose error is moderate recognize distant ob-
jects tolerably well, notwithstanding when tested by the usual
methods vision may be less than fa\. Even in this respect great
differences will be observed. I have noted with the same optical
error uncorrected, say 1 D or 2 D, differences of vision varying
between f g- and fa%. Their habit is to half shut the lids, to reduce
the pupillary aperture, and the3r rely upon their familiarity with
accessory conditions of form, color, brightness, contrast, etc. For
work near at hand, reading, sewing, etc., they have the advan-
tage of less effort of accommodation and get larger images by a
close near-point. At the usual age of presbyopia they are not
obliged to use glasses, and there may be an abatement in myopia
and improved distant sight. This advantage will not be availa-
ble where M is greater than 3 D or 4 D.
Causes.—■Occurring ordinarily during school life, it has natu-
rally been inferred that unfavorable conditions in the school room
as to light, the attitude of the head, the distance of the book or
paper, the hygienic conditions, the duration of study have an im-
portant bearing on the production of myopia. Cohn, Fuchs, Horner
and others have diligently labored to correct in their respective
countries the faults to be found in school construction and arrange-
ments. There has been room and need for the improvement which
94
DISEASES OF THE EYE.
has been brought about. It now appears that under the better
conditions of certain modern institutions in Germany, an abate-
ment in the number of cases of myopia has been realized, amount-
ing to 6$ (von Hippel). This is not a large gain and it may be put
alongside the fact established by Dr. H. Derby that a course of
study in such an institution as Harvard University in this country,
where the hygienic conditions cannot be impugned, results in the
development and increase of myopia. The subjects were between
the ages of seventeen and twenty-five. Fixing upon objects at
short range, say, less than 12 inches, implies strong efforts both
of accommodation and convergence. Let the sclera be relatively
weak, and the back of the globe begins to stretch. Nor is this re-
sult limited to the juvenile stage of life. I have seen it begin in an ap-
parently robust student of twenty, during his third year in college.
Habitual occupation with small objects near the eye, as when children
first take to books, either for pleasure or study, or when learning to draw or
to sew, or they are kept at a piano in a dark corner, or the school-room has
not the full quota of light which is each child's right, or the benches and
desks are so contrived that the child sits crouched or crooked, or a heavy
lexicon compels a stooping posture, or the text is badly printed, or is in a
language whose characters are intricate and unfamiliar, like Greek, or Ger-
man, or Hebrew : such are some of the occasions of myopia.
It has been shown that accommodation alone has little effect in
causing nryopia, but being associated with convergence the two
are considered together. The evils of convergence are manifestly
due to excessive pressure. Now strabismus occurs mostly with
hypermetropia, and the apparent contradiction is explained by call-
ing to mind that the short axis eye will rotate easily, while the long
axis eye of myopia presents proportionate difficulty in rotation.
Moreover, the most hurtful conditions appear when antagonistic
forces oppose each other, which no longer arises when a weak muscle
has surrendered, as in strabismus. It is a recognized fact that op-
posing muscles aggravate myopia, certainly in causing asthenopia,
and probably in contributing to extension of the visual axis. The
following are the factors which have been dwelt upon as effective:
1. The internal and external recti cause compression under high
angles of convergence, by which the externi are stretched and the
insertion of the optic nerve is dragged upon. 2. Stilling1 lays
chief importance upon the influence of the superior oblique, which
he finds to be extremely variable in its direction and extent,
and which, when the visual lines converge, aids in adduction. If,
as he sometimes finds, its course is quite transverse and its fibres
inserted near the optic entrance, it adds materially to the compres-
1 " TJntersuchungen uber die Entstehung der Kurzsichtigkeit " Wiesba-
den, 1887. Seggel, Graefe's Archiv, xxvi., 2, s. 1, 1891.
ERRORS OF REFRACTION
95
sive action during convergence, and also drags upon the optic nerve
entrance. He adduces autopsies of 102 eyes in support of his theory
(p. 32, 1. c.) and also takes into account the shape of the orbit in so
far as thereby the pulley is placed higher or lower and permits the
muscle to lie more or less closely in contact with the globe. 3. Arlt
attributes to the inferior oblique and external rectus an indirect
effect by pressure on the venge vorticosae which promotes choroidal
stasis. 4. The same congestive tendency results from bending
forward of the head and neck. 5. Subacute inflammation of the
choroid and sclera, which Graefe designated under the name of
sclerotico-choroiditis posterior and by which he explained the
choroidal crescent adjacent to the papilla, was formerly much
dwelt upon. In this view he was, to a considerable degree, in
error, while that in some cases such an inflammatory process
occurs, is admitted. It is now accepted that both mechanical
and inflammatory or degenerative processes go on simultaneously.
In some cases the latter seem to be primary, while the former are
by far the most common, and always co-operate with the latter.
It is easy to believe that there may in some cases be a lack of resis-
tance in the posterior part of the sclera, yet anatomically this is
difficult of proof. Clinical experience has shown that suspension
of accommodation has very slight influence in abating myopia.
Stilling thinks that movement of the eyes up and down in read-
ing is an important provoking circumstance, the effect of the obliqui
being then greater. He remarks that among watchmakers myopia
is rare, while very common among instrumental musicians. Stilling
•calls attention to the prevalence of a low-roofed orbit among
myopes, and manj7 have entered on this inquiry, which seems to
have a basis of fact, although opinions vary. The effect is to make
the action of the superior oblique more compressive.
But while mechanical action explains the usual cases of mj'opia,
it does not explain certain exceptional forms, viz.: the congenital,
such as occur among illiterate people, and those who use their eyes
only for distance, and the cases where myopia suddenly and rapidly
develops in adults or old persons. These are cases of inflammatory
action akin to hydrophthalmus, and, as above remarked, many
times both causes combine.
The effect of constitutional conditions in exciting or predisposing
to myopia is forcibly presented by Dr. Batten (Oph. Review, Jan-
uary, 1892), and he lays stress on the tokens of general vascular
lesion in young persons, as proven by, 1, spontaneous hemorrhages
(epistaxis, menorrhagia, retinal hemorrhages); 2, capillary conges-
tion ; 3, cardio-vascular disease. He dwells on the full, soft pulse
of young persons, an acute rheumatism as a frequent precursor of
myopia, and that choroiditis may be both a cause and conse-
quence of the lesion.
96
DISEASES OF THE EYE.
We have thus an ordinary and an extraordinary type of myo-
pia, sometimes called the benign and the malignant, also we are to
distinguish between stationary and progressive conditions. Pro-
gressive myopia if occurring during the years of growth need not
excite very serious concern, but if progress continue beyond the
age of twenty or a sudden increase appear during adult life we
have before us a grave condition. In making the distinction be-
tween these conditions, we must be guided by the degree of visual
acuity which is obtainable by glasses, and by the ophthalmoscopic
appearances. High degrees of myopia almost never have normal
vision, and frequently we find lesions quite sufficient to account for
it; even when we do not, stretching of the fibres of the optic nerve
will explain it, or in rare cases it may be fair to assume that a con-
genital or precedent amblyopia compelling very close approxima-
tion of objects, has been the predisposing cause of the actual myo-
pia. It becomes proper next to consider the
Functional Disturbances and Pathological Anatomy.— The
error often becomes established without the consciousness of the
subject; it may reach 3 or 4 D and not attract his attention. Fre-
quently it is recognized by accident or by the questions of another.
To accidentally find myopia in one eye and not in the other is not
at all rare. On the other hand, the development may be with pain
or weariness and photophobia and inability to study. Besides the
indistinctness of distant objects there may be occasional blurrings
of print and the eyes look irritable. When of moderate degree
the myopic eye looks normal, but in the higher grades it becomes
prominent, pushes the upper lid, and in its movements betrays its
elongated form. The anterior chamber is deep, the pupil large and
slow to act, the periphery of the iris is retracted. The myope has
a vacant look and his face has little expression, he half shuts his
lids and wrinkles his forehead. He is often abstracted, inattentive
to his surroundings, perhaps easily embarrassed, or without mean-
ing it has an air of effrontery, is more fond of books than of society,
given to sedentary rather than to out-door and active pursuits.
To the ophthalmoscope the eye will in moderate cases show no
lesion, but in most cases we find a whitish crescent on the outer
side of the optic disc. This is called the choroidal crescent and
by Jaeger the conus (see Fig. 33). It is found in a few cases of
hyperopia and emmetropia, but it is almost distinctive of my-
opia. Fig. 33a shows in section the elongation and atrophy of the
choroid at the back of the eye. Loring (Trans. Internat. Med. Con-
gress, 1876) found in 2,265 eyes that the crescent existed among
emmetropics in 3.33$, among hyperopics in 3.49$, among myopics
in 20.56$. Schnabel1 among 135 cases having the crescent found
1 Graefe's Archiv fur Oph., xx., 2, p. 1.
ERRORS OF REFRACTION.
97
73$ myopic, 13$ hyperopic and 13$ emmetropic. When high de-
grees of myopia are examined the crescent becomes still more
frequent. Graefe put it at 90$. Much discussion has been held
upon the significance and origin of- this lesion. We have at
length, thanks to the observation of Nagel * and the anatomical
examinations of Weiss2 and Herzog Carl Theodor,3 facts which
determine its character and mode of formation. In 1861 Jaeger4
when describing the crescent, spoke of a peculiar look of the disc
on the nasal side, and said that such eyes anatomically examined
presented a peculiar bending of the optic nerve fibres toward the
crescent. These remarks did not excite attention. In 1880 Nagel
brought to notice a later observation of Jaeger in 1866, that the
pigment epithelium of the retina in certain cases of myopia is
Fig. 33. Fig. 33a.
dragged over the nasal edge of the disc, and he announced that in
many cases he had by the ophthalmoscope perceived that both the
retina and the choroid were thus drawrn over (see Jaeger, pp. 66, 67
and Figs. 29, 30), while on the temporal side the membranes had
been pulled away from the nerve edge and in this \x-Ay occasioned
the crescent. Two years later Weiss examined two eyes one of
which had M 5 D and the other a little less, and in which the so-
called supratraction of the choroid had been observed during life.
He subsequently examined three others and Herzog Carl Theodor
has examined another.
We will first give attention to the ophthalmoscopic appearances.
We may have a simple crescent of small size attached to the tem-
poral side of the nerve and marked by a pigmented edge (see Fig.
33). This may grow larger, and still larger (Fig. 34)t When equal
to more than half the disc we may often see specks of pigment or
choroidal vessels upon the surface. The crescent may not only ex-
tend temporal-wise, but up and down. It may surround the whole
nerve. In extreme cases the whole posterior region of the fundus
1 " Mittheilungen aus der Ophthalmiatrischen Klinik in Tubingen," 1880,
Hft. 1, p. 231.
5 Ibid.. Bd. i., Hft. 3, p. 62. 3 Ibid., Bd. ii., Hft. 1, p. 56.
4 " Ueber die Einstellung des dioptrischen Apparates im menschlichen
Auge," p. 61.
7
98
DISEASES OF THE EYE.
shows thinning of the choroid and rarefaction of the pigment.
Figure 34 indicates some of these conditions.
What has been found in a slight case of myopia where the globe
was 24| mm. long and the M probably not more than 3 or 4 D, is
figured in the cut taken from
Weiss's paper, Fig. 35 (" Mitthei-
lungen," 1884, Taf. 11, Fig. 1).
The condition may be described
as a dragging of the papilla to
the temporal side in which the
retina, choroid, nerve fibres and
sclera, the lamina cribrosa and
vessels participate. The conspi-
cuous element is the displace-
ment of the choroid. At the
edge of the opening for the pas-
sage of the optic nerve, it is at-
tached both to the sclera and
to the lamina cribrosa. In fact
the fibres of the latter belong in great measure to the choroid.
The choroidal opening is elongated in the direction of the crescent
and made oval; its nasal border reaches over the papilla and car-
ries with it the retina. The supratraction may reach the middle
of the disc. On the temporal side the choroidal edge is dragged
Fig. 34.
-*■■; &z&£*^
~s
Fig. 35.
away from the disc, drawing with it the nerve fibres and fibres of
the lamina cribrosa; the edge of the sclera becomes oblique and its
inner surface is exposed, in proportion to the extent to which the
choroid is retracted. Thus the crescent is formed partly by re-
ERRORS OF REFRACTION. 99
traction and thinning of the choroid, and partly by a perspective
view of the scleral canal, and upon it we have the commingled
fibres of the optic nerve and lamina cribrosa. In moderate degrees
of the crescent or conus, we simply have the opening out laterally
of the scleral canal and there may be more or less pigment brought
to view. But in higher grades there is real atrophy of the choroid
with irregular islands of pigment, atrophy of vessels, some of which
may remain or everything may have disappeared, except the elas-
tic layer. The retina also suffers on both sides of the disc. The
inner granule layer and lay-
ers interior to it, will be
intact, while the layers ex-
ternal to it are deficient.
The bacillary layer is de-
stroyed both on the supra-
tracted and on the crescent
side, and this explains the
enlargement of the blind
spot which characterizes
such cases. It is seen in
Fig. 35 from Weiss that on
the nasal side the retinal
pigment has been drawn
over the edge and turned
up; this will be seen by the
ophthalmoscope as a dark
border. On the temporal
side the choroidal edge
where it becomes more nor-
mal, also shows some in-
crease of pigment, and the
damage to the outer layers
of the retina is usually more
extensive than this limit. Figs. 36 and 37 from Jaeger show the
same changes with less detail and are instructive.
In advanced cases wThere a crescent has been converted into a
ring of atrophy, though no traces of it can be seen by the unaided
eye, the microscope shows the presence of supra- and also re-trac-
tion, or that the papilla has been pulled in all directions. Stilling
explains the variations in the direction of the crescent, by the trac-
tion of the fibres of the superior oblique when its tendon happens
to reach more or less to certain parts of the circumference of the
nerve. It is of course understood that a bulging of the sclera
occurs at the posterior pole which may be either broad or sharply
defined. While the mechanical displacement with textural atrophy
100
DISEASES OF THE EYE.
is going on, changes occur both in the sheath of the nerve and in
the adjoining parts of the choroid. The intervaginal space is greatly
distended and chiefly upon the nasal side (see Figs. 36, 37). To
this Donders called attention. The sclera becomes thin and the pig-
ment of the choroid is absorbed. Added to this is degeneration of
the vitreous; it becomes liquefied, either so as to form larger or
smaller cavities in its substance, or it will be separated from the
retina by a layer of fluid. The hyaloid membrane may be split into
two or more layers and the membrana limitans of the retina be-
comes brittle and easily
separated from the deeper
layers. Fibres and cells
and detritus float in the
fluid vitreous and cause the
shadows called muscoz voli-
tantes, of which myopes so
constantly complain. In its
anterior part, the retina in
severe ca ses is found to have
become, as it is called, cede-
matous, i.e., the layer of
inner granules has been dis-
tended into spaces which are
filled with a coagulable fluid.
The ciliary muscle of
the myopic eye is charac-
terized by the fewness of
its circular fibres, while the
meridional are in large
number and reach far back-
ward even beyond the ora
serrata. Herzog Carl Theo-
dor says that in some my-
opic eyes the circular fibres are in reality not wanting, but are
pushed aside and crowded together into a small space and for this
reason may be overlooked.
In this sketch are embraced most of the points of the real path-
ology of myopia. The explanation of the mode of occurrence of
the crescent, assigns to it an entirely mechanical and not an inflam
matory origin; the evidence that the phenomena are the result of
pressure enables us to impute the real cause to the extrinsic mus-
cles of the globe. The part which can be played by the ciliary
muscle is evidently nothing more than resistance to the stretching
at the posterior pole, while itself becomes elongated and' is really
caore a passive than an active agent.
Fia. 37.
ERRORS OF REFRACTION.
101
Besides degeneration of the vitreous to which allusion has been
made, we have in advanced cases, cataract, beginning by preference
at the posterior pole of the lens and evidently due to imperfect
nutrition. The whole of the choroid may take part in the atrophy
in extreme cases, the pigment cells becoming emptied of pigment,
the chorio-capillaris and the middle layer of vessels becoming par-
tially obliterated.
More damaging is the occurrence of choroidal changes at the
region of the posterior pole and even in the macula itself, where
sometimes a distinct spot or group of spots of atrophy appears (Fig.
38). The effect upon vision is generally disastrous, causing a central
scotoma. Even when very faint
traces of choroidal lesion exist,
or perhaps none can be seen, the
cones in the macula may be-
come separated and deranged
so as to cause straight lines to
appear crooked—metamorphop-
sia. Hemorrhages are not in-
frequent m advanced myopia
and too often in the region of
the macula. The most deplor-
able occurrence, and which af-
fects myopic eyes more than any
other is sub-retinal effusion, or
detachment of the retina. This
destroys vision over an extensive part of the field, or perhaps in
toto and is rarely amenable to treatment. It will be considered
when speaking of diseases of the retina.
Besides the troubles within the eyeball, there are to be men-
tioned, the frequent troubles of the muscles, inequality of the eyes
(anisometropia), and astigmatism. As to muscular incapacity, it is
the rule to find it in high degrees of myopia. It is the result of
being obliged to converge to a point too near for the muscles to
permanently maintain the effort. The longer the axis the more
difficult does it become to turn the eyeball inward. The interni
must contract extremely, the externi are stretched and wrap
around the globe and even the optic nerve in extreme conver-
gence must suffer traction on the outer side of its sheath. The
result of these hindrances is that insufficiency of the recti in-
terni and positive diverging strabismus are frequent complications.
On the other hand we also meet with strong degrees of converging
strabismus in the higher grades of M. It is possible often for
myopes to direct one eye up and another down, as shown by their
unusual power of counteracting the diplopia caused by putting a
102 DISEASES OF THE EYE.
vertical prism before one eye. Their grasp upon binocular vision is.
much feebler than that of other persons. When the globe is much
lengthened as, for instance, to 28 or even 30 mm., such an ellipsoid
manifestly rotates with difficulty in a cavity whose shape is some-
what similar. The centre of motion is displaced and in most cases
is farther from the posterior surface of the sclera than in the em-
metropic (Donders, p. 404).
These troubles will be again adverted to. An apparent strabis-
mus con vergens is often seen in myopes, because the line of fixation
is from the shape of the cornea liable to fall to the outer side of its
axis. In other words the angle gamma is either negative, or if
positive is very small. (See page 14.) Weiss presents this in a
diagram of one of the eyes which he examined. In great myopia
the radius of the cornea becomes longer than usual, in harmony
with the general distension.
In the last degree of myopic change the lens becomes partially
or wholly cataractous, and, because of fluidity of the vitreous, is
liable to be luxated backward or downward. Intraocular hemor-
rhages are prone to occur. In extreme cases the distention of the
eye attains the condition called hydrophthalmus, and it may be
difficult to say what part of the result is to be ascribed to a genuine
irido-choroiditis, and what to the processes of simple myopia.
Enucleation may then become a necessity. In external appearance
myopic eyes often attract attention by their prominence and their
observable ovoid form. Usually the pupils are large and inactive,
but in all these particulars the contrary conditions may be true.
The strictly congenital cases do not differ in essence from those
which reach a high development in later life, and we need make no
special reference to them beyond what has been said.
Prognosis.—For practical purposes we may divide cases of
myopia into classes according to their degree and their progress—
into the moderate and the extreme—into the stationary and the
progressive. The acutely progressive are by some described as
malignant myopia, a name which seems for several reasons objec-
tionable. We can recognize M amounting to 0.5 D, and as the far-
point is then at 80 inches there is a manifest lack of clear distant
vision. Even up to 3 D the error may be called slight. From 3 D
to 6 D it may be called of medium degree, while all above 6 D are
to be counted extreme and therefore serious. Horner well says,
great myopia is the more dangerous the younger is the subject.
Every practitioner can substantiate this; and he also says that the
dangers of high myopia are most threatening in the later part of
life, i.e., after 50 years of age. Then come vitreous degeneration,
cataract, and subretinal effusion.
For myopia below 4 D which has reached its maximum and is
ERRORS OF REFRACTION. 103
stationary, there need be no anxiety as to the welfare of the eye,
and both acuity and working power are often admirable. If it be-
come stationary between 4 D and 8 D acuity is often slightly defi-
cient, viz.: f or \, and the working power may be good or otherwise.
Muscular troubles and astigmatism are apt to be present, but
prognosis is not serious. When above 8 D the case is grave and
the future will be influenced greatly by the health of the person,
his surroundings and the extent to which he uses his eyes. In his
statistics of blindness Cohn sets down 10$ as due directly or in-
directly to myopia. He includes cases in which one eye only is blind.
Diagnosis.—Remarks very similar to those in discussing hyper-
metropia might be repeated. We test for distance by glasses and
found the diagnosis conditionally upon the fact that concave
glasses give normal acuity. It is not proven by this test, because
with defective acuity there may be great myopia, and with perfect
acuity thus procured, there may be no myopia. We must resort
to the objective tests; the direct ophthalmoscopic method, or to
Cuignet's method. Spasm of accommodation simulates M, and. as
said before, while it usually disappears in the dark room, in special
cases it still persists. When a doubt exists, one may more will-
ingly resort to atropia than is admissible in either H or E.
The occurrence of myopia from excessive curvature of the cornea
is not to be forgotten, and will be especially mentioned under coni-
cal cornea. The refraction is always irregular, and the error will
betray itself by the shadow test, and with absolute certainty by
the ophthalmometer of Javal and Schiotz; under oblique illumina-
tion and by direct ophthalmoscopy it will usually be discovered.
Prophylaxis.—Because myopia is for the most part an acquired
anomaly and dependent, as has been shown, on excessive use of the
eyes during the tender years of life, it is imperative to set forth the
methods which can be employed to prevent or restrain it. Efforts
in this direction are sometimes met by the objection that the error
is hereditary and therefore the attempt is useless. Loring1 ably
discusses this point; Tscherning and Landolt review what others
have written. It is very difficult to get statistics of real value, but
this maybe said, that the predisposition maybe hereditary, but
that the determining causes are acquired and largely preventible.
The cases which are congenital, and they are generally those of
high degree, are very seldom hereditary. With the clear evidence
that the error is brought about bj7- improper modes of life and of
use of the eyes, preventive measures become imperative. The first
thing is to promote a vigorous state of health, and in this all that
relates to home hygiene, to food, air, sleep, and exercise, are to be
regarded. Second, the arrangements in schools and the hours of
1 Transactions of International Med. Cong., Phil., 1876.
104
DISEASES OF THE EYE.
studv, the light, the air space, the height of benches and desks,
the distribution of study hours and play, the print of books, atti-
tude in reading and writing,—all these are potent in their influence.
On these points, Cohn and Fuchs, Berlin and Javal have writ-
ten elaborate monographs and Cohn1 gives the literature of the
subject. In the United States there is need of attention to the
conditions of school life in many particulars, although our school
houses are free from many of the evils complained of in continental
Europe. As regards the hours of study and of play respectively, we
are not so much at fault as are the Germans, but the tendency is
to over taxation and therefore myopia. Frequent intervals of rest
are most important, and children can naturally be better cared for
in small than in large classes. Imposing long study hours out of
school is another hurtful practice. Reading by dim light at home,
by firelight, at twilight, sewing and embroidery, are things to be
prevented. Certain trades and occupations have a mischievous
effect. The most conspicuous are the type setters, whose ratio of
myopia is nearly as high as that of the higher students. Watch-
makers and jewellers have been shown to be little subject to the
error because they work so much with a magnifying lens and with-
out convergence and without much movement of their eyes.
The great factor in preventing myopia is cultivation of out-door
life and its consequent promotion of health and use of eyes upon
distant objects. It is known that the English have a lower per-
centage of myopia than exists on the Continent, and it is fair to
connect their exemption with their fondness for out-door sports.
Among us as Americans the same tastes exist, and not only for this
reason, but because of our better conditions of existence, wre should
have less of the evil. In our cities we have little advantage above
other nations, although the native population in the cities is less
prone to myopia than the children of Germans (Loring).
Diirr's2 examinations found spasm of accommodation among only
11 out of 133 myopes; i.e., 7.3$. Latent A more than 1.66 D he
reckons as spasm. In 30$ there were choroidal changes. He lays
the greatest stress on the undue proportion of working hours exacted
from children. He lays, as does Yon Hippel, less stress on imperfect
arrangements in the school buildings, than upon simple overtax-
ation whether at home or at school. He compares the total number
of school hours demanded of children between the ages of 10 and
19 years in England, France and Germany; they are 16,500 19 000
20,000. Hours of recreation, are, in England, 4,500; in France. 1,300-
in Germany, 650. It is also shown by a curious table, p. 145 that
the more gifted scholars, compared to their less brilliant comrades
are near-sighted in the ratio of 32 to 38.
1 Lehrbuch der Hygiene des Auges, 1891-92. a L. c. p 141
Noyes' " Diseases of the Eye."
Plate I.
ERRORS OF REFRACTION.
105
In describing the ophthalmoscopic appearance of the optic nerve
in myopia, the situation of the choroidal atrophic crescent has
been described. We may now call attention to certain less frequent
anomalous conditions which resemble this lesion, yet must be dis-
criminated from it. They are most frequently associated with
myopia, and in many instances with myopic astigmatism and
hence may here be introduced. That we sometimes find an ap-
parent crescent on the lower side of the nerve has been referred
to. A close inspection of these cases shows that the crescent
may sometimes be divided into two parts, of which the upper part
is gray and the lower whitish. It is easy to recognize in many
cases that the crescent is an oblique surface or pit. In fact these
cases are spoken of as coloboma of the sheath, that is of the scleral
canal of the optic nerve. To this may be added a real choroidal
crescentic atrophy. The lesion is essentially congenital. It is usu-
ally associated with amblyopia. One eye only may be affected.
Of the illustrations on Plate I. instances of this type are Nos. 2, 3,
4, 5, 6, 8, 9, 10. In No. 1, a physiological excavation is unusually
situated, viz., at the lower edge of the disc, and we also have the
crescent below. In No. 7 the lower and outer half of the disc is
much flattened and merges into the crescent.
Besides the anomalies mentioned, others are to be noticed, viz.,
that in No. 1 the vessels emerge in a direction contrary to the usual
fashion, i.e., toward the nasal side instead of toward the temporal
side. In R. No. 6, the trunk comes out as three branches instead
of two. In Nos. 8 and 9 the vessels are peculiar. In 12 is a large
choroidal atrophy and the refractive state of different localities is
designated. This evidences distinct local staphyloma. In No. 11
is depicted a coloboma of the macula lutea: viz., the large central
white surface upon which are pigment spots. Near it are two small
circular patches of choroidal atrophy. Beneath the disc is a
crescent. A case of this type I have never seen. Loring1 has
figured a case, and refers to several. It will always be difficult to
decide between an acquired and a congenital lesion of this kind.
Coloboma of the optic sheath was first described b3r Liebreich:2 see
also Nieden.3
In No. 13 is presented the very rare anomaly of a mass of con-
nective tissue covering the principal part of the papilla. The above
illustrations are taken from a paper by Prof. Fuchs, Graefe's
Archivfiir Ophth., Bd. XXVIIL, Abth. 1, S. 139, 1882.
1 Loring: " Text-book of Ophthalmoscopy," p. 94.
2 " Atlas d. Ophthalmoscopies' Taf. xii., figs. 1, 2.
3 Kn;ipp's Archives of Ophthalmology, vol. viii., p. 501.
106
DISEASES OF THE EYE.
Treatment—This divides itself into (1) the hygienic, (2) the opti-
cal, and (3) the therapeutic, What has been said about prophylaxis
includes the hygienic measures to be adopted when myopia has
begun. There must be such a reduction in the hours of study and
of close application as shall tend to diminish the rate of progress.
Entire abstention might be necessary, but can seldom be enforced.
The inclination to read for amusement must be checked and every-
thing to promote health be resorted to. If there be a delicate or
strumous constitution, especial pains must be taken by food and
habits and appropriate medication to build up the tissues. It is by
no means always possible even with the most careful management
to prevent the increase of the error during the years of juvenility.
I have followed a number of such cases and not been able to pre-
vent the increase, but have probably been of service in restraining
it. If the wisest efforts can only partly control the progress of the
error we are not therefore to cease to attempt its control. Upon
students in colleges and high schools advice should be incul-
cated, and they must be convinced of its importance. Occasional
seasons of rest are of importance, say for two or three weeks.
They give time for the intraocular circulation to become regulated
and for the muscles to gain repose. Much stress has been laid upon
the value of atropine in checking myopia. But this remedy has now
few supporters and the most that can be claimed for it is that
under its use the degree abates about 1 D. This will not be a per-
manent decrease, the ordinary tension soon returns. It gives a
good opportunity for examination, because it helps to enforce ab-
stention from use, and if the far-point be inside of 20 inches, it is no
great annoyance, and I not infrequently use it. Abandonment of
near work is the essence of the benefit, not suspension of accommo-
dation.
The corrective treatment is in the selection of glasses. If with.
unaided eye any letters on the card are read at 20 feet, the degree
is not great. If no letters are read, an approximate idea is gotten
by noting at what distance the person holds the book in reading
Snellen 2. Begin with trying, on one eye at a time, the weaker
numbers. If strong glasses are being used, viz., above — 6 D, and
the vision is nearly corrected, try whether sight is helped by hold-
ing the glass nearer or farther away. If the former, it is too weak;
if the latter, it is too strong. Myopes are often sensitive to an in-
terval of fa or less. With too strong a glass they complain of
being dazzled, and shrink from the unwonted brightness of objects.
Much respect is to be paid to their impressions; but, when they
have been wearing inadequate glasses, they are liable to mistake
the surprising distinctness conferred by a proper glass, for a strain
of over-correction. It is often impossible to give them V = ^-°- ■ but
ERRORS OF REFRACTION.
107
before admitting this, careful inquiry is to be made for astigmatism,
and the state of the fundus minutely explored by the ophthalmo-
scope—especially the region of the yellow spot. Moreover, in
doubtful cases the patient should have the benefit of atropia—using
a solution gr. iv. ad 3 1. (1 to 120) several times. In seeking the
best correction the danger is of getting too strong a glass, which
will excite tension of the ciliary muscle. To this, as a rule, myopes
are very sensitive.
Among children who chose their own glasses it has been found
that in from 19$ to 37$ (Erisman, Cohn) they were too strong. The
same is not infrequent among adults. Many circumstances are to
be considered in giving concave glasses. 1st. The degree of error.
2d. The quality of sight. 3d. The occupation and necessities of the
person; the age, the sex. 4th. The state of the muscles. 5th.,
Whether the eyes have equal value. 6th. The condition of the in-
terior of the eye, which is, perhaps, included in quality of sight.
With per sons whose error is small, glasses are usually wanted only
on special occasions, and none for near work; they care only for
eye-glasses. Even with — 5 D some myopes care little for them,
and if they do use them, complain that the habit of using
them disqualifies them for as good discernment without them as
they possessed before. This is simply an alteration in mental
habits and not in sight. As a rule, however, persons having be-
tween 4 and 10 D gladly avail themselves of glasses for uninter-
rupted use. But when glasses are above 6 D some complain of
fatigue from their continuous wear. This may be due to mus-
cular fatigue, to anisometry or simply to the continuity of atten-
tion which sharp sight induces. The value of this factor can hardly
be appreciated by the emmetrope. To the myope taking off the
glasses is sometimes like going out of the blazing sun into the
shade. A large latitude must be allowed for idiosyncrasj^. Where
the very high grades are reached, viz.: above 10 D, generall}- the
person wants the full correction. Of course we must be governed
by the state of the interior of the eye and the acuity of vision. But
even when there may be a large crescent and other choroidal
lesions, if the process be substantially stationary or slowly pro-
gressive, I have found it best to give the full correction. There
is less effort with the glasses than without them. One may often
wisely impart a blue tint to the glasses, because from a large
crescent or a patch of choroidal atrophy, light is reflected and dif-
fused within the eye, the effects of which the blue color somewhat
assuages.
It need only be added that one should always be vigilant to dis-
cover astigmatism complicating myopia, because it often makes a
great difference in the acuity of sight. A glass may easily be chosen
108
DISEASES OF THE EYE.
too strong because of an undetected astigmatism. The concurrence
of the two errors is very frequent.
In regard to giving concave glasses to children some special
remarks are proper. They rarely need them for near work; for
distance they require them to see maps and the blackboard in
school and for such purposes. If the error be below 2 D they may
get along by coming to the front, but when above that they should
have spectacles and wear them most of the time. If the error is
above 4 D the glasses should be worn constantly when the subject
is 13 or 14 years old. They have an important influence in mental
habits and character; they do not, in my judgment, when well
chosen and with no special contra-indications, hasten the progress
of myopia. They keep the working point at a suitable distance
and by developing the power of accommodation assimilate the per-
son to the emmetrope. It is, of course, assumed that there must be
no tendency to unnatural approximation of the work permitted.
For most myopes it is necessary to gradually become habituated
to glasses. They may wear them for short periods until they be-
come accustomed to them.
What has been said applies for the most part to distant vision.
We are sometimes obliged to order a special glass for near vision.
This happens in persons whose accommodation is feeble either from
want of use or from age, or whose myopia is high. It may be for
playing the piano, for writing, especially with book-keepers who
must cast their eyes over two or three large account books. How
shall the glass be selected? A simple calculation will determine.
Take the distant glass as a basis. Let it be 6 D, which we will call
7". The working point is to be at 20" = 2 D. We have 6—2 =
4 D. A glass 4 D will bring the near-point to 20" and will meet
the need. Or, if the working point is to be at 1 foot which is f$ =
3| D we may give 3 D. The middle-aged myope and likewise the
younger will do near wTork without glasses provided the near-point
is not inconveniently close. It can happen with slight M that after
middle life, a convex glass is required for near work, and myopes of
higher grades reduce the strength of their glasses or abandon them
when the time for presbyopia arrives. But distant vision does not
of necessity become improved.
Myopes suffer much from muscular asthenopia and especially
from insufficiency of the internal recti. With many who have large
error no attempt is made to maintain binocular vision for the near.
They put off their glasses and use one eye only, while the other is
allowed to diverge. In this way, despite the inconvenience of hold-
ing a book very near, many read for hours in comfort. But with
others there is an attempt at binocular vision which cannot be
maintained. Hence they have pain and asthenopic irritation. Fre-
ERRORS OF REFRACTION.
109'
quently this can be completely relieved by choosing a weaker glass
suited to the working point. In other cases additional relief can be
had by decentciing the glasses from the median line. Sometimes
a combination of abductive prisms and concave glasses can be hap-
pily made. Prisms can seldom be made stronger than 5° or 7° be-
cause they become too heavy. It is important to examine when
asthenopic symptoms exist whether there be any tendency to devi-
ation of one eye in a vertical direction. This can be told by a can-
dle at 20 feet with a red glass over one eye and a strong abductive
prism over the other, held accurately in the horizontal position. If
the two flames are not on the same level, use a prism to correct the
deviation, and this prism should be incorporated in the glasses to
be worn. I have known an error of 2° make a patient extremely
uncomfortable.
But if the muscular error be of a certain amount, resort must
be had to an operation, i.e., tenotomy of the antagonist, for its
relief. To this reference will be made later. Here it is proper to
say, that the only indication which to me is sufficient for this opera-
tion is asthenopia of marked type and which other proceedings,
faithfully tested, have not relieved or seem wholly unlikely to re-
lieve. Tenotomy as a means of diminishing or arresting myopia
has been practised, but it has no warrant in the results which it
produces. It can do much harm and has done little, if any, good.
Many times the choice of glasses for a myope is an intricate
problem, demanding not only technical skill and knowledge of
pathology, but also good sense and general wisdom.
More active measures will be in order when a sudden increase
appears, and with it symptoms of retinal irritation with congestion
of the nerve, with floating bodies in the vitreous and a retraction of
the choroid which by its size and the elongated look of the vessels
on one side and their crookedness on the other shows the posterior
staphyloma to be increasing, and a subacute inflammatory condition
to be in progress or impending. Then prolonged abstinence from
work, say for six weeks or longer must be enforced. The artificial
leech, or in lieu of this two ordinary leeches, may be applied to the
temples at intervals of four to six days. This cannot be often re-
peated. The milder saline purgatives, especially the mineral waters,
may be used, and as an especially efficacious agent pilocarpine may
be given. Care must be taken with the last mentioned, if subcuta-
neously injected, not to give a dose too large. One-sixth of a grain
of the muriate of pilocarpine will generally do no harm to an adult,
but it has been known to cause great prostration by reducing the
heart's action. The value of iodide of potassium and of corrosive
sublimate will be in the ratio of visible exudations in the fundus or
may be decided by the acuity of vision.
110
DISEASES OF THE EYE.
If the vitreous be ver}- hazy and perhaps at the same time the
lens, this implies a more acute inflammatory process and might
have been mentioned above. For this Horner recommends para-
centesis of the anterior chamber, but this remedy is to be used with
caution, because the sudden abatement of intraocular pressure is
followed by a prolonged increase of congestion. The clinical acute-
ness of Prof. Horner makes the mention of the proceeding justifi-
able.
A course of treatment thus outlined will have to be modified by
the age and health of the person. If weak and young, drastic
measures would only aggravate the case. Then the use of smoked
glasses, employment of atropine to insure the greatest rest of the
eyes, dry cups to the temples and general hygiene will be all that
may be possible, without confinement to a dark room. It is also
important that persons in whom myopia makes rapid advance
should be examined every three months and if needful undergo a
series of suspensions from eye work for a month at a time. In
some cases of this kind a long sea voyage has proved eminently
useful. The cases of high myopia in very early life, especially when
it cannot be attributed to overwork, are the least susceptible to
treatment and often there are no rational indications of treatment
except of the hygienic kind. The stage of inflammation may have
given place to that of atrophy, and often we see these deplorable
subjects only in this period. At this epoch there is very little value
in medication.
A patient highly myopic and knowing something of the perils
of his position is often extremely excitable and takes alarm at tri-
fling symptoms—a slight conjunctival swelling, or a more than
usually distinct vitreous shadow, brings them in terror for advice.
Tact and gentle handling and inoffensive applications, a weak lotion
of borax, a mild stimulating liniment to the forehead, and placebos
generally are the soothing influences which are suitable.
Should a spot of hemorrhage occur, perhaps in the macula, it is
rather an index of weakness of the vessels than of undue deitermi-
nation of blood. It is most likely to occur in the cases of dissemi-
nated choroidal atrophy or general thinning of the membrane, and
in which there are pigment deposits intermingled with atrophic
spots. Treatment is chiefly rest and avoidance of light, with very
mild derivative remedies. The blood is absorbed slowly, requiring
weeks, and afterward there will remain a white spot. Scotoma
occurred at the onset of the mishap and this may grow smaller,
but will not wholly disappear, and even if it should, metamorphopsia
is likely to ensue.
Of detachment of the retina it is not now necessary to speak
in detail. Such treatment as can avail has been related when
ERRORS OF REFRACTION.
Ill
speaking of the antiphlogistic proceedings. Evacuation of the fluid
by puncture of the sclera has questionable benefit. See chapter on
this subject, p. 630.
The inequality between the two eyes which in myopia is not in-
frequent, is often a source of trouble. Sometimes a sufficient acuity
can be given to each eye b}T its proper glass to enable both to work
in harmony; at other times one is so amblyopic as to take little
part in vision, and its error may be so high as to render correction
valueless. An essential consideration is to carry the working point
farther off.
To some myopes strong light is a distress, and their glasses
may be tinted a light blue. To some the constant observation of
objects is a weariness—they prefer to take off glasses and remain
in ignorance of what is about them until their eyes are rested.
Many are sensitive to the form of the frames, their weight and ad-
justment, and the eyelashes must not touch the glass. All these
points deserve attention. Some persons affect the wearing of a
single glass which they have learned to hold in place by nipping it
with the brow. If such have two equally good eyes, which gener-
ally is not the case, such a practice is no less damaging to the eye
than offensive as a mannerism.
The above description of the possible lesions of myopia is calcu-
lated perhaps to make the picture of near-sightedness too gloomy,
because so many woeful conditions are grouped together. The very
large proportion of myopes escape all such disastrous occurrences;
but it is highly important to convey the impression that myopia
may be more than a mere inconvenience or trifling defect, because
it does embrace such sad possibilities.
ASTIGMATISM. As.
When the refraction is such that rays emanating from a single
point cannot be brought again to a focus as a point, on the retina,
this state is astigmatism; of this there are two kinds, the regular
and the irregular. The latter is caused by opacity of the cornea or
lens, and does not admit of satisfactory correction, although it can
sometimes be mitigated. The former is chiefly dependent on ab-
normal curve of the cornea or lens, or want of homogeneousness in
the lens, and is correctible by cylindric or spherico-cylindric glasses.
The defect ma3r be acquired or congenital; irregular astigmatism
in the cornea is an acquired error, and some rare cases of regular
corneal a stigmatism are acquired; but, as a rule, the regular astig-
matism of the cornea is congenital. Acquired astigmatism in the
cornea, where no opacity exists, comes from conicity of the mem-
brane, or happens after tenotomy of muscles, or after wounds of
112
DISEASES OF THE EYE.
the cornea, iridectomy and extraction of cataract. But these cases
are a minority of the whole. Of correctible astigmatism the greater
portion are congenital cases. Objection is sometimes made to this
statement because the error does not always announce itself until
middle life. The explanation is that the accommodation can
conceal a considerable degree of error until its vigor begins seri-
ously to decline. It is also to be said that a small degree is natural
to almost every one, varying from 0.25 D to 0.75 D, and because
the radius of the vertical meridian of the cornea is shorter than
that of the horizontal.
We ,have occasion now to treat only of regular astigmatism and
without regard to its locality in the lens or in the cornea. Consist-
ing as it does in a want of uniformity in the radii of the meridians of
the media, this error manifestly may complicate either emmetropia,
hypermetropia, or myopia. For this reason we have simple astig-
matism, either hyperopic or myopic; and compound astigmatism,
both hyperopic and myopic; and lastly, there may be mixed astig-
matism, in which either hyperopia or myopia may predominate. The
symbols of these several conditions are as follows, as they have
been given to us by Donders. To him we owe the systematic study
and development of this subject, which he made with as much com
pleteness as did Helmholtz the theory of the ophthalmoscope. We
have: 1st, myopic astigmatism, Am; and compound myopic astig-
matism, M + Am; 2d, hyperopic astigmatism, Ah, and compound
hyperopic astigmatism, H -f- Ah; 3d, mixed astigmatism, with pre-
valent M, viz., Amh, and with prevalent H, Ahm, or both M and H
may be alike.
Whatever may be the length of the optic axis, it is evident that
the refraction cannot be homocentric, i.e., from a luminous point
the rays cannot again be brought to a point. On the contrary, the
focus, instead of being in one plane, is stretched over a certain
length which is called the focal interval.
This may be understood by the diagram Fig. 39 taken from Fick,.
in which CD and AB represent the vertical and horizontal merid-
ians of an asymmetric cornea viewed in perspective from an oblique
position. The vertical meridian comes to a focus upon the axis at
o and thence the rays diverge. The horizontal meridian reaches
its focus less quickly, viz.: at o' and thence its rays diverge. The
space between o and o' is called the focal interval. The form of
this portion of the bundle of rays cannot be a cone, but forms a
skew surface. There is no place within it where a punctate focus is
formed, but there are linear foci, one at o and the other at o'.
These, however, are not mathematical lines, because at both these
places there are other rays which do not join with them. It is im-
possible to indicate in a diagram the form of the bundle. If, how-
ERRORS OF REFRACTION.
113
ever, we place a screen across it at the points marked 1,2,3, 4, 5,6,7,
perpendicular to the axis, we shall get the following series of lumin-
ous surfaces as shown on Fig. 40—beginning with a vertical oval,
1,2, then converted to a vertical line, 3, where the rays are focussed
which belong to the vertical meridian; then again comes a verti-
cal oval because the vertical rays are going apart; then quickly
comes a place where each meridian has equal separation from the
axis and the figure is a circle, 5; then comes a horizontal oval;
and, lastly, a horizontal line where the rays in the horizontal me-
ridian cross. All this is easily illustrated by using a spherical
lens + 6" focus and adding to it a + cylinder 12 inches focus with
which to throw the image of a flame or of a luminous round disc of
Fig. 39.
Fig. 40.
ground glass on a screen—all the above changes will be verified if
the axis of the cylinder is placed horizontal. The mathematical
theory of this condition is intricate, and was developed by Sturm
and has been further discussed by Knapp, and very lately by
Mathiessen, who modifies slightly Sturm's theory. Knapp has
made a model with silk threads of different colors which illustrates
very admirably the phenomena. Becker has also done the same
by letting fall a beam of light upon a spherico-cylindrical lens set
in a glass tank filled with water rendered milky by nitrate of
silver. The luminous track is displayed as a pencil of light ap-
pears in a dark chamber reflected from the floating particles of
dust. The form of the beam can be studied at leisure and all the
sections shown which have been described. On a large scale the
visual phenomena of astigmatism may be perfectly shown with the
8
114
DISEASES OF THE EYE.
magic lantern by adding a very weak cylinder to the objective,
viz., + 1 D, or + 0.75 D.
It has been said that differences in the radii of the vertical and
horizontal meridians of the refractive surfaces are the chief causes
of regular astigmatism. To this must be added the want of exact-
ness in the centering or collimation of the refracting media. This
affects both the cornea and the lens. The geometrical axis of the
cornea forms with the visual line the angle alpha, and moreover, as
the line of fixation causes the eye to deviate still further from the
visual line, the angle gamma (see page 14, Fig. 7) increases the
asymmetry of the several surfaces. This want of collimation occa-
sions astigmatism. It is exceptionally the case that the shortest
focus of the media is through the horizontal meridian. This is some-
times spoken of as astigmatism against the rule. Slight deviations
from the vertical are very common. Astigmatism of the lens may
increase or diminish that of the cornea. Javal1 has shown that a
very small fraction of the total astigmatism is due to the lens; he
has also shown that by accommodation the lens may partly or
wholly neutralize asymmetry of the cornea. He, and before him
Dobrowlsky, attributes this to unequal contraction of the ciliary
muscle. In some cases of pretty high degree he finds about all the
astigmatism in the cornea; what may usually be ascribed to the
lens is not more than 1.5 D. He has revived attention to the con-
trol which young subjects have over astigmatism, citing himself
as an example, that with astigmatism = —2.5 D he could at 24
years of age see the stars correctly as others do. It is a frequent
observation that slight degrees of the error first come to light
during middle life.
Latent and manifest astigmatism of course refer to the influence
of the accommodation in concealing or modifying the error. Like
hyperopia this distinction is of most importance in its slight de-
grees and chiefly among young subjects.
Simple astigmatism affects the emmetropic eye and may be
simple myopic or simple hyperopic, Am or Ah. In Am one merid-
ian is emmetropic, the opposite is myopic. In Ah one meridian is
emmetropic, the opposite is hyperopic. (See Figs. 41,42.) The con-
ditions are illustrated in the diagrams.
Compound astigmatism belongs to the ametropic eye; if myo-
pic, both principal meridians will be myopic, but one to a greater
degree than the other, i.e., the radius in that meridian will be
shorter. If the eye be hyperopic, both principal meridians will be
hyperopic, but one to a greater degree than the other; i.e., its
radius will be longer than that of the other. We have then, M -f-
Am and H+Ah. (See Figs. 43, 44.)
1 See Annales d'Oculistique, 1881, ii., 14.
ERRORS OF REFRACTION.
115
Mixed astigmatism exists in an eye whose axis is normal, but in
one meridian myopia exists, while in the opposite hyperopia exists.
In other words, in one meridian the radius of curve is shorter than
normal, in the opposite meridian it is longer than normal. Accord-
ing as myopia or hyperopia predominate, the cases may be desig-
Fig. 41.—Simple Hyperopic Astigmatism, Ah.
Fig. 42.—Simple Myopic Astigmatism, Am.
Fig. 43.—On up. und Hyperopic Astigmatism, H + Ah.
Fig. 44.—Compound Myopic Astigmatism, M + Am.
Fig. 45.—Mixed Astigmatism, H and M equal.
nated as Amh and Ahm. In Figs. 43 and 44 the form of the eye is
made myopic and hyperopic—as compared to the remaining figures
which are supposed to be emmetropic.
The above are the subdivisions originally made by Donders and
they are what we practically have to observe. In the figures H
116
DISEASES OF THE EYE.
stands for the horizontal and V for the vertical meridians. The
position of letters and of the dots in relation to the retina indicates
the kind of error; in eyes having compound astigmatism one is
represented as hyperopic (Fig. 43), the other as myopic (Fig. 44); in
each case the vertical meridian has the shorter radius. In mixed
astigmatism the axis is represented as emmetropic, while in the eye
the myopic meridian, which is the vertical, has an equal error with
the hyperopic; equality of H and M is less frequent than marked
inequality. Instead of speaking of a shorter radius, it might be
perhaps better for the present to speak of the shorter and longer
foci, as these are what the diagrams represent. Inasmuch, how-
ever, as the principal error resides in the cornea, the terms may be
used indiscriminately. It is at the same time clear that ametropia
and astigmatism are two distinct conditions, although they may
coexist.
It has been remarked that the meridian of shortest focus or of
shortest radius is usually vertical. To this rule there are numerous
exceptions, not only in obliquity of the principal meridians, but in
complete reversal of the rule, so that the horizontal may be the
strongest. Javal says of himself that he has this condition and
that in numerous members of his family among whom the error
prevails, nine out of ten have the horizontal meridian stronger
than the vertical. The direction of the meridian has an important
influence upon vision. One may understand it by putting before
his eye a convex spherical, say of 2 D, added to a convex cylindrical
of 3 D; there will then be the condition of compound myopic astig-
matism, supposing the experimenter to be emmetropic. If the axis
of the cylinder be made horizontal, the strongest meridian becomes
the vertical; then objects will have their vertical dimensions
lengthened because in this meridian rays come to a focus quicker
than in the horizontal and have crossed before they reach the plane
of the latter's focus. For this reason the ends of an object will be
fringy, while the sides will be sharp. A series of dots in a vertical
row will run into each other and make a solid line with blurry
ends. If the axes be reversed to the horizontal, objects will seem
broadened; the top and bottom will be well defined, the sides blurry.
The dots remain separated, but extend laterally into brushes.
With the axis in an oblique position, an elongation takes place in
the direction of the strongest meridian or opposite to the axis.
If glasses of an opposite type, viz., concave, are employed by which
hyperopia is induced, the phenomena will not be so distinct because
the accommodation will modify them; they will at the same time
become reversed in reference to the axis of the cylinder. With the
axis horizontal the strongest meridian also becomes horizontal and
if one accommodate for this meridian the dots will be stretched in
ERRORS OF REFRACTION.
117
a horizontal direction, and if for the opposite meridian they will be
merged into a vertical line.
From these experiments we can appreciate the symptoms of
astigmatism. There is usually indistinct vision, acuity being reduced
in accordance with the degree of error. If spherical glasses improve
it, there will not be much difference in the value of two or three nearly
equal numbers. The person may say that he has noticed a differ-
ence in the distinctness of objects according to their form. A boy
who required in one eye a cylinder +6" axis 90° and in the other
+ 8" c. axis 90°, told me that he could see the telegraph wire farther
than he could see the pole on which it was stretched. The masts
and spars of a ship show- similar differences to some persons. Some
people have noticed that they found trouble in telling time when
the hands of a watch were in certain positions. Such patients for
the same reason find difficulty in reading certain letters. For in-
stance, O on the card seems to be H, because with a strong vertical
meridian the top and bottom are hazy and the sides black. With
a reverse meridian of maximum refraction a C is imagined to be
Z or cannot be made out. Dr. Little, of Philadelphia, has put into
a card the letters which he calls confusion letters for astigmatics,
and he has given the words FOOL and NULLIFIED, which are
special stumbling blocks. In the test types such errors are to be
heeded as significant. Similar to these mistakes is the difficulty
which such persons have in reading Greek and Hebrew, because
the characters are difficult for them to decipher. It is also charac-
terisl ic that they hold books very close and their near-point is close
for all kinds of work. They complain sometimes of confusion, but
ignorant of the nature of their trouble and seldom analyzing their
perceptions, they cannot describe exactly how objects appear, but
they often complain of headache. In this respect astigmatics suffer
greatly and so common is it, that an inquny into the refraction in
cases of obstinate headache has become a routine question. Dr.
Weir Mitchelll called attention to this as a neurologist in 1874-76.
It was known to oculists long before. Want of quick perception
is also characteristic, and this is the necessary result of their
blurred retinal images. If they have chosen glasses, especially
concave glasses, they wear them often tilted forward, by which
means their sight is improved. By doing this their confusion ellipses
are converted into lines, and the change is favorable to vision. The
degree and kind of subjective symptoms will depend much upon
the peculiarities of the person, and if the temperament be nervous
and excitable the reflex and attendant phenomena are sometimes
extraordinary. Many papers have been written on this text and
elaboration is unnecessary.
1 American Journ. of Med. Sciences, April, 1876, p. 363.
118
DISEASES OF THE EYE.
The name astigmatism was given by Dr. Thomas Young, who<
carefully studied the subject and invented tests to detect and ex-
hibit it, in 1793. If two convex cylinders be combined with their
axes parallel they increase each other's power as cylinders. If they
be combined with their axes transverse they neutralize each other
as cylinders and compose in effect the equivalent of a spherical lens
(not, however, in a mathematical sense). Such a combination
gives a flatter field than a bi-spherical lens. A convex and concave
cylinder of the same radius, if combined with parallel axes, neutral-
ize each other and become the equivalent of a plate of plane glass.
If they be combined with axes transverse they add to each other's
power as a cylinder. Such a combination of a convex and concave
cylinder has been employed under the name of Stokes' lens. When
the axes cross obliquely the degree of astigmatism is between zero
and the maximum of the two combined, and it increases as the
obliquity passes into the transverse direction. The combination in.
reality is equivalent to a mixture of a spherical and a cylindrical
glass in every position save when the axes are either parallel or
transverse.
Cylindric and spherical lenses may be combined, so that we have
concave spherico-cylindric and convex spherico-cylindric. Occa-
sionally a bi-cylindric lens is ordered, as in some cases of conical
cornea, but as a rule spherico-cylindric or plano-cylindric lenses
serve the needed purpose.
Diagnosis.—We have to ascertain the fact of astigmatism, its,
kind, its degree, and the direction of the principal meridians. We
may or may not employ a mydriatic, and we resort to both sub-
jective and objective methods as for other kinds of refractive error.
We try the acuity of vision, and prove how much may be gained by
concave or convex spherical glasses. The deficit is not apt to be
large, because high degrees of astigmatism are rare. We note
whether the patient stumbles at certain letters such as O or F,.
or L or N; and if vision reach f-g- and cannot be carried beyond, a
weak cylinder of 1. or 1.5 D may be put before the spherical and
rotated to see if at some point vision notably improves. This
would prove the presence of astigmatism. But it is better to adopt
a more systematic method to elicit and analyze the error. The
best tests are those of Dr. Green, of St. Louis. Upon a diagram of
a clock dial he places discs traced with lines and dots of various
widths and arrangements. Each disc is centred upon a pivot and
can be revolved in any direction. Out of a great variety of
patterns I have learned to confine myself to two or three. The
space between each hour is of course 30°. Put before the
patient the one denoted in Fig. 46 and give the patient the
weakest spherical concave glass or the strongest spherical con-
ERRORS OF REFRACTION.
119
vex glass which he will accept, and ask him to say whether he sees
all the lines of the figure with equal distinctness. One must urge him
to give close attention and must also increase the convex or diminish
the concave glass, so as to bring out the meridian of strongest re-
fraction. This antagonizes his accommodation. If he have astig-
matism he will state that along a certain diameter the lines are
more conspicuous or stand out as the only ones of the figure.
There may be two or three, or there may be five or six. The fewer
and more prominent the greater the astigmatism. Let him point
out between what figures on the dial (hours) the lines are found.
Say they run between XII. and VI. This indicates the meridian of
strongest refraction with the glass. Substitute for this disc an-
other, like Fig. 47 and set the lines in the direction which the
Fig. 46. Fig. 47.
patient indicates. The lines are wide at their extremities and nar-
row at the centre. If the glass he wears is suited to this meridian
the patient should trace the white interval almost to the centre.
If he do not, move the disc lightly in either direction. If this
do not separate the lines more perfectly, modify the glass a
little until the lines become well defined and separated within an
inch or less of the centre. This determines the glass for this merid-
ian, and it is to be noted. Between every hour on a clock are 30
degrees, and we have found the principal meridian to be exactly
upon the vertical. Now turn the disc 90° and bring the lines to
run from IX. to III. They are indistinct, light in color, ill-defined,
not separated and the white interspace is short or invisible. Now
diminish the convex spherical or increase the concave spherical
until the pair of lines are made as distinct as before.
Suppose in the direction from XII. to VI. the glass was — 3D, and
now from IX. to III. it is — 5 D. The difference between them is, —
120
DISEASES OF THE EYE.
2 D, and this is the astigmatism. Suppose the glass at first had
been -f- 7 D and now we have + 4 D, the difference is + 3 D, and
this is the astigmatism. In the former case it is compound myopic,
in the latter case compound hyperopic. It may have been that
from XII. to VI. the glass was —2D, and when the line is reversed no
glass is accepted or uncertain answers indicate that the patient is
not sure—this is likely to be simple myopic astigmatism. There is
great liability to think that a simple myopic astigmatism exists
with the axis horizontal wrhen in truth the error is simple hyper-
opic astigmatism with the axis vertical. Such a mistake is often
made and the only assurance against it is in the use of atropine.
We have found, we will say, —2D astigmatism. Let the patient
have —3D spherical and give him another test card like Fig. 48
where 2 pairs of lines cross at right angles; set them in the proper
diameters. Then put before the
spherical a concave cylinder — 2D
with its axis transverse to the
lines from XII. to VI., and this will
make each pair of lines perfectly
distinct. For the other case of
compound hyperopic astigmatism,
let him have the + 4 D spherical
to view the crossed lines and to
this add + 3 D cylinder with its
axis corresponding to the direction
XII. to VI., and this will clear both
the lines. The same thing will be
Fig. 48. ™ , - , . ^ &
effected by using + 7 D spherical
and to it adding —3D cylindric with axis transverse to the di-
ameter XII. to VI. Now with the combined spherical and cylindri-
cal glasses let the patient try the test letters. A smile lights up
his face as he runs glibly over what he had painfully failed to
see. There is no more satisfactory experience than to witness the
success of one's efforts in a case of astigmatism.
If the examination proceed as described, the diagnosis is deter-
mined and also the glass required, but such is not always the fact.
Satisfactory vision is not always easily gained, and we must multi-
ply the tests or resort to atropia, or what is better, employ objective
methods. Mr. Bowman (see Donders, p. 490) found illumination of
the eye by the mirror at two feet distance with rapid variation of
its inclination, sometimes lead him to the discovery of regular as-
tigmatism.
What are the indications for the use of atropine ? In myopic
astigmatism, both simple and compound, it is rarely necessary In
marked compound hyperopic astigmatism it may often be dispensed
ERRORS OF REFRACTION.
121
swith. In simple Iryperopic astigmatism it will often be necessary,
and the same remark applies to mixed astigmatism. Practically
the point in a given case, is to find out to which of these classes it
belongs. Javal strongly insists that to use atropine and to correct
the whole error is wrong practice. I cannot fully concur in this
opinion. The degree of asthenopia and the visual acuity must be
taken into account. In young subjects who enjoy good accommo-
dation and who have not complained of severe symptoms the par-
tial correction may suffice. But the presumption is not so much in
favor of this decision as in the case of simple hyperopia. One must
call to his aid the objective methods, and observe what may be the
difference between the results according to them and according to
the subjective test. If the difference be great, I have no hesitation
in using atropia to full paralysis. It is true that the correction
thus obtained is likely to be too strong for the patient to accept.
But at the end of a week the subjective test may again be made
and a glass selected Avhich will be satisfactory. The suspension of
accommodation is, in my experience, a valuable therapeutic mea-
sure in many cases of astigmatism. The enforced rest is servicea-
ble and they are prepared to use their eyes and their glasses with
greater comfort.
There are other subjective tests which it is proper to mention.
A most useful one is the letters of Dr. Pray. They are capitals
composed of strokes which run in different directions for each letter.
A patient points out the letter which is most black and clear, and the
direction of the lines composing the letter indicates the direction of
the meridians. Becker gives a set of parallel lines in triplets and
placed in various meridians; also concentric circles which will ex-
hibit a sector of distinctness contrasted with remaining indistinct-
ness. There are many other ingenious test diagrams whose char-
acter is similar. (See one by Dr. Oliver in Medical News, Oct. 6,
1883, p. 373.)
Schemer's method of determining refraction as modified by Dr.
Thomson is also to be considered, and it has value in the cases
which are decidedly amblyopic, whether from conicity of the cornea
or slight haziness of it, or from true amblyopia. Reference has
been made to it on page 81. Consideration of cases in which vis-
ual acuity can at the best be only very imperfect, will be deferred
to a later page.
Objective examinations are made by inspecting the fundus by
the direct method of ophthalmoscopy, by Cuignet's method or the
shadow test, and by ophthalmometry. The first is most available,
the second has an approximative value, the third is extremely rapid
and exact so far as corneal error is concerned and would doubtless
be much used if the instrument of Javal and Schiotz were not ex-
122
DISEASES OF THE EYE.
pensive. To detect astigmatism by the ophthalmoscope we simply
take for an object a fine vessel running in the direction of one prin-
cipal meridian and choose the glass, whether plus or minus, which
makes it distinct, and then take another fine vessel running in the
opposite meridian and find the glass suitable for that. Great care
must be taken not to let accommodation either of subject or ob-
server interfere. The difference between the glasses gives the as-
tigmatism. Now set a cylinder before the patient which corrects
the astigmatism, placing the axis in the proper manner, and in the
ophthalmoscope make up the remainder of the refractive error by
turning on spherical glasses and again examine the fundus. Now
there should be a perfectly clear and bright image if the media are
transparent. And this contrasts strongly with what was possible
when only spherical lenses were employed. (It is easy to arrange
a spring clip behind the mirror to carry a cylinder from the trial
box, and this is provided in my ophthalmoscope. See Fig. 21, page
45).
One can thus satisfy one's self not only of the accuracy of the
refractive correction, but of the integrity of the membranes, which
is very important in high degrees, both of compound myopic and of
hyperopic astigmatism. If the error be high, the fundus before the
suitable glass is found will have a smeared and streaky look, as if
a brush of paint had been swept over it. The nerve will be pulled
out into an oval form and the ends of the ellipse be fringy with
the vessels drawn in the same direction; there can be no dis-
tinctness in the detail of the surface. If the error be over-cor-
rected, the disc will be elongated slightly in the opposite direction
and the lines of distortion will be reversed. Not until the cylinder
is introduced will the view be satisfactory. It is also useful to
employ the inverted image to corroborate the diagnosis. By it, if
the nerve were by the direct method oval, with the long axis verti-
cal, the long axis becomes now horizontal. For this phenomenon
to be well seen, the objective must be not less than 3" focus and it
must be held at its own focal length distant. If nearer to the eye
or farther from it, the inverted image will be less elliptical. The
lens must be held exactly vertical and without any turning on its
axis, else this will cause the disc to seem oval.
To distinguish between an optic disc which is oval anatom-
ically, from one which seems oval by reason of astigmatism, one
must note its edges at the extremities of the oval. If they are
sharp and well-defined, the shape is anatomical; if they are blurry
or fringy, astigmatism is the cause.
The shadow test (employing a concave mirror) shows astigma-
tism in the following way: If the shadow move up and down in the
same way as the mirror and with a certain rapidity, and when
ERRORS OF REFRACTION.
123*
moved transversely the shadow goes in a similar way with less
rapidity, this indicates myopia in both meridians, but a greater
degree in the vertical. If the shadow move " against" the mirror
in one meridian, and with greater rapidity than the movement
" against" the mirror in another meridian, this proves hyperopic
astigmatism. In other words, if in meridians at right angles to
each other the reflex and shadow move in different directions or
with different degrees of rapidity, there is astigmatism. A very
bright reflex and a shadow difficult to detect, but nearly linear and
traversing the pupil very rapidly and in a direction contrary to the
motion of the mirror indicates E in that meridian. The slower the
movement the higher the ametropia; the more rapid the move-
ment the nearer to E. If there be mixed astigmatism, the shadow
will in one meridian move with the mirror and in the opposite
meridian against it. A motion opposite to the mirror indicates
either E, H, or weak M. To decide which of these is present, set a
glass -f- 1 D in the trial frame before the eye. If the refraction be
emmetropic, this glass causes myopia and the shadow should move
with the mirror; if such be not the fact, the eye has H; if the
shadow does now move with the mirror, the patient has either E or
M. A glass +.5 D will decide whether there be E or M, and a
stronger -f- glass will give an idea of the degree of H, until a glass
is found which makes the shadow go opposite the movement of the
mirror. Cylindric glasses may be put before the eye as spherical
usually are until the phenomena are made to resemble those of
emmetropia. See paper by Dr. Ferguson.1
Ophthalmometry, by the instrument of Javal * and Schiotz, is
the quickest and usually most reliable mode of examination. An
experience of eight years has given me the greatest confidence in it.
The model now made differs from the original figured in the text,
and in my judgment is less desirable. It has a large disc with so
many lines and circles as to cause confusing reflections from the
cornea. A plain black disc one foot in diameter is important
and sufficient. Its essential parts are as follows. (See Fig. 49.)
Two very bright objects, one a complete parallelogram and
the other a parallelogram of the same dimensions, but with its
long side cut away in the form of steps, p. 125, are made to slide on
a curved bar and are placed before the eye so that each shall
be reflected from the cornea. These reflections are viewed by
a small telescope, and are doubled by passing through a Nicol's
prism. Four images are in view as with a stereoscope, but the
outside ones are neglected, and attention given to the two cen-
tral ones. The patient's face is placed in a frame and is steadied
1 "Retinoscopy;" American Practitioner, Dec, 1882.
• Annales d'Oculistique, Ixxxvi., July and August, 1881, p. 5; ibid., lxxxvii.,
May and June, 1882, p. 212; ibid., lxxxviii., July and August, 1882, p. 33.
124
DISEASES OF THE EYE.
by a chin and forehead rest. The telescope stands upon a tripod
which can be moved forward and backward to get the proper focus,
and has a screw adjustment for slight variations in height: When
the central images are sharply seen, the step-slider is moved until
its bottom step comes into exact contact with the lower part of the
parallelogram (see Fig. 50). The lower edges of the reflected images
are brought into a straight continuous line by turning the bar which
-carries the sliders and which revolves around the body of the tele-
Fig. 49.-In this instrument electric lights are used, and a disc has been added to protect the ob-
server. In other respects it is like Javal's model of 1882.
scope. Having accomplished this, the position of the bar is read
off upon an index which will place it, we will suppose, at 180°. Then
the bar is turned 90° and the relations of the two images noted.
If they have not changed, either by overlapping or separating, the
curve of the cornea is the same both horizontally and vertically;
if the images overlap in the vertical meridian, the radius of curve
is in this sense shorter and there is astigmatism (see Fig. 51). If
the images separate with the bar vertical, this meridian has a longer
radius than the horizontal, and again there is astigmatism. Each
ERRORS OF REFRACTION.
125
step in the step-slider represents 1 D, and consequently the amount
of astigmatism is easily read off, and can be estimated to .25 D. By
the ordinary arrangements errors of 6 D are measured, while by
special adjustments much higher degrees can be determined.
The portion of the cornea inspected is a central circle 5 mm.
in diameter. It is assumed that the chief seat of astigmatism is in
the cornea. Javal claims that between the total astigmatism as
found by trial glasses and the corneal error the difference will not
be greater than 1.5 D. It would be inadmissible to permit an error
so great as this, while as a matter of fact so great or any import-
ant discrepancy very seldom arises. It is chiefly found among
the lower degrees of error, i.e., when less than 3 D. The axis of
the principal meridian is quickly determined and will vary from
that found by trial glasses by only a few degrees, usually not at
all. We learn nothing as to the kind of ametropia nor do we know
whether the cylinder should be plus or minus. Preliminary trial
has already given an idea of the quality of error, and putting the
□ □
Fig. 50. Fig. 51.
cylinder designated into the trial frame we add plus or minus
spherical glasses until the visual acuity is raised to the normal
degree. Before testing by trial glasses the indicated cylinder may
be put behind the ophthalmoscope and the real refractive situa-
tion be objectively ascertained, and afterward the spectacle box
will soon give us the requisite combination. A good illumina-
tion is essential, and diffused surrounding light is cut off. There
should be a screen at the observer's end of the telescope to shield
his eyes. The whole problem may often be solved at a single
sitting and without atropia. For rapid work and especially
in public institutions this instrument is invaluable. For the young,
the confused, the dull, the amblyopic, it is most excellent.
It is not rare to discover, as Laqueur pointed out, that the curve
of the cornea is not constant. It sometimes changes as we look at it,
we note the images approach or separate instead of remaining still.
They exhibit these oscillations under the action of the lids, and also
if the upper lid is lifted off the globe, under the action of the motor
muscles. Drawing on the lids at the outer angle will increase the
corneal curves greatly. Hence we have an explanation of the un-
126
DISEASES OF THE EYE.
certain findings in some cases of astigmatism, apart from spasm
of accommodation. The cornea is thin and cannot resist external
pressure. The same explanation will account for variation or in-
crease of astigmatism with lapse of years: in fact, herein lies the
beginning or suggestion of conical cornea. The ophthalmometer
unerringly detects opacities and distortions in the form of the cor-
nea, and thereby explains why the attainment of perfect visual
acuity may be impossible. It also shows that the principal merid-
ians are sometimes not at right angles to each other, e. g., one may
be at 90° and another at 160°. The axis of the cylinder accepted by
the patient will usually in such a case approximate the truly verti-
cal or horizontal position. Aeuity will usually be below standard.
A simple device which roughly points out these defects in the cor-
nea is the so-called disc of Placido, which consists of a series of con-
centric circles painted on a disc
about 10 inches in diameter in
black and white. (See Fig. 52.)
It is held close to the cornea and
the reflex viewed through a cen-
tral hole with a lens 4" focus.
Javal has applied the same to his
ophthalmometer.1
It has already been said that
astigmatism exists in many per-
sons who have normal visual
acuity and make no complaints.
Especially will this be true of the
young and vigorous and some-
times with high degrees of error.
Low degrees are often unrecog-
nized until presbyopia approaches.
Moreover, if the error be hy-
peropic or myopic, with axis nearly
horizontal, in both which cases
lines nearly perpendicular are
most distinctly seen, persons with
notable error get on moderately
well, because most objects with which we deal have greater
height than breadth. Such is the case with type (Roman letters),
and with trees, men, buildings, and the majority of objects. True,
objects are exaggerated in height, but of this the person is un-
aware. But if the axis is in the contrary direction, or if it be
■oblique, or if the two eyes are unsymmetrical, trouble announces
Fig. 52.
1 Annales d'Oculist., Jan., Feb., 1883, p. 5; also Landolt, English ed., p. 328.
ERRORS OF REFRACTION.
127
itself early. An attack of illness, chronic uterine disease, exces-
sive eye-work, great grief, etc., will reveal astigmatic error, pre-
viously unsuspected. I have also seen astigmatism, which severe
uterine disease had brought to view, retire into obscurity and un-
consciousness when by an operation the uterine lesion was cured.
The glasses which the patient had with extreme reluctance con-
sented to, were put away and satisfactory use of the eyes regained
without them.
The more pronounced are the asthenopic symptoms, the more
searching must be the quest for astigmatism and the more accu-
rate its correction. Then even slight degrees, 0.5 D, must not be
neglected. No fixed rule can be given about correcting the full
amount of error, yet this will be done in the majority of cases. If
there be no asthenopic symptoms, we need only correct the mani-
fest error; with slight symptoms this may also be the general rule,
with emphatic symptoms full correction is demanded. In young
subjects, as has been said, more may be left to the accommodation
than with persons above thirty. But an error more than 1 D
should not, in my opinion, be permitted to remain, even in young
subjects. When strong cjdinders are first worn, the subject is often
annoyed in growing accustomed to them, and as with high myopia
may have to use them for short periods at first.
For near work it is especially necessary to have the correction,
and whether the glasses be the same which are used for distance
will depend on the age and refraction. For one Avho is becoming
presbyopic an increase in refracting power may be needed. For a
myope a weaker spherical concave glass may be required. The
rules in this regard are easily deduced from the ordinary require-
ments of ametropia and presbyopia. In all cases, however, the
same cylindric corrective will be demanded as for distant vision.
In certain cases the glass for distance may be reversed for the
near. For instance a person uses — 1.50cD with axis horizontal for
distance and by presbyopia needs + 1.50s for reading. Instead of
ordering + 1.50Ds 3 — 1.50 Dc, axis 180°, one may give + 1.50cD
axis 90°, which meets the same need.
Formerly cylindric glasses were cut circular and large that
errors in setting might be corrected. Such ungainly binocles be-
long to the time of unskilful opticians. It is wholly unnecessary
to make a patient conspicuous by large and round glasses. They
may be used either as spectacles or as nose glasses. Of course the
former are more steady and trustworthy, but personal choice often
decides this point. When used as eye-glasses they are apt to tip
forward, and this makes a slight increase in the refraction in the
vertical meridian. Sometimes this has to be taken into account.
128
DISEASES OF THE EYE.
With strong cylinders patients are obliged to look straight forward
through the middle of the glass. A sidewise look gives distortion
and it cannot be avoided. Therefore they have a more restrained
field of vision than ordinary ametropes.
It happens sometimes that astigmatics have very deficient ac-
commodation. I have been obliged in some cases to order for near
work a glass which represented the whole amount of A which was
needed. This has occurred in persons whose error was not cor-
rected until they had come to thirty-five years of age. In writing
a formula for spherico-cylindric glasses the following notation is
employed: say, - 3.0 s. C - 1.5 c, ax. 180°. The sign C meaning
a combination of the spherical and cylindric surfaces. For mixed
astigmatism we may have either a spherico-cylindric or a bi-cylin-
dric combination, viz., for instance: 1.0 s. C +2.5 c, ax. 180°, which
can also be expressed by - 1.0 c, ax. 90° C + L5 c, ax. 180°. The
combination of cylinders of opposite qualities with axes at right
angles gives a wider and flatter field than belongs to spherico-cyl-
indric glasses. Hence such an arrangement is to be preferred in
considerable degrees of error. Slight degrees of mixed astigmatism
are not rare.
The above notation was suggested by Donders and is consider-
ably used—nevertheless a printed form is to be preferred in which.
each kind of glass has a line for itself, as for example:
O. D. Spherical + 3 D.
Cylindrical + 2.5 D, axis 180°
Prism. base
O. S. Spherical + 2.5 D.
Cylindrical + 2.0 D, axis 170°
Prism. base
Space is given for inserting the use of a prism. The opticians
print such blanks, and often a semicircle is added, laid out in de-
grees to assure accuracy. The inter-pupillary distance, and some-
times notes as to the height of the bridge and other details are
provided for. These points are not too trivial for the oculist's
attention, although he may place reliance on the skill and careful-
ness of his optician.
Anisometkopia.
We are in the habit of examining each eye separately and choos-
ing the glass which each requires. We meet cases of inequality of
the eyes, and when a difference of 1 D exists, the name anisometry
is employed. Differences far higher are met with. As, for example,
with monocular aphakia and in myopia one eye may exceed the
ERRORS OF REFRACTION
129
other by many dioptries. I have records of many such cases. It
can also happen that the eyes may have opposite kinds of refraction,
viz.: one be hyperopic and the other be myopic; or E in one and H
or M in the other. This I have called antimetropia.1 The behavior
of dissimilar eyes may be of three kinds: they may combine in
binocular vision; one may be used for distance and the other for the
near, or one only may always be used. If they combine in binocular
vision; this may be true for distance and not for the near point.
It is remarkable how great differences can sometimes be comfort-
ably harmonized. With a difference, say of 3 D, there is necessarily
a combination of one sharp and one very blurry image in the men-
tal act. Yet, that this is possible in much higher differences, I
have seen in persons who had binocular sight, after one eye had
been operated on for cataract and the other was normal.
We meet with peculiar idiosyncrasies on this point and only
general suggestions, not absolute rules, can be offered. If in myo-
pia there be anisometropia of 3 D and vision in each eye good,
usually each may take its own correction, and this difference is to
be maintained both for near and far. For the near, a working point
is to be chosen which will correspond to the capacity of the mus-
cles in convergence, and the effort will be to carry it as far away as
may be acceptable. One must also bear in mind the power of ac-
commodation and by a simple calculation founded upon these two
factors the correct working point can be chosen, and the glasses
fitted accordingly. Testing the muscular power at six meters when
ametropia is corrected and then again for the near will often ex-
plain the asthenopia which such persons suffer. It is difficult to
give rules to apply to the great variety of cases which are possible,
but as the practical difficulties arise at the working distance, it is
here that special attention must be given. For instance, a lady
who in one eye had E, and in the other M 3 D was made perfectly
comfortable by using a plane glass on one eye and — 3D on the
other. She wore the glasses constantly for some weeks until she
became accustomed to them and afterward she needed them only
in reading. Yet, for distance she appreciated the better vision
which they conferred. She was about thirty 37ears of age and could
easily bring into play the required accommodation. In other cases
for the more erroneous eye an incomplete correction must be
chosen, because the difference is otherwise too great in the size of
the respective images. It can also happen that for the near-point
one eye may need a weak convex and the other a concave glass;
this is not, however, very common. It is usually unavailing to try
to usurp the function of accommodation unless the person is beyond
middle life. The degree of accommodative effort to be allowed to
the individual is a matter for the exercise of judgment.
1 Report of r>th Internat. Ophth. Congress, p. 165, 1877.
130
DISEASES OF THE EYE.
In hypermetropia similar problems present themselves, but not
so often do we have large differences in refraction with nearly equal
visual acuity, as in myopia. More often do we have great differ-
ence in acuity as well as in refraction, and then there is seldom a
strong impulse to binocular vision; out of such cases a large con-
tingent of converging strabismus is derived.
It is frequent for one eye to have, and the other to be free from
astigmatism. If vision be thereby impaired in one eye or if asthen-
opic symptoms exist, each eye will require its own correction. It
is not seldom to find astigmatism different in the respective eyes
and a difference in direction of meridians is very common. Some-
times a strain is experienced in combining the eyes and such modi-
fications may be needed in the axes of the glasses as the comfort
of the patient requires. For the patients who have binocular vision
for distance and monocular for near, usually exhibiting diverging
strabismus in the act of reading, the rule of conduct will be derived
from the acuity of the eyes, the degree of divergence, the age, sex
and muscular conditions. If we are asked to relieve pain, the
method will be to carry the working point farther away by concave
glasses adapted to the desired distance, and if this do not suffice,
prisms can sometimes be used, or more frequently tenotomy of the
muscles can be employed.
For patients who use one eye for distance and the other for near,
there being extreme difference between them, it is best usually to do
nothing. It is not likely that they can acquire binocular vision. It
is not rare to meet with such persons, and in some instances the opti-
cal difference is not extreme,but there may be an impairment in one
or more muscles, or one eye may be astigmatic and the other not.
The want of binocular vision is the important circumstance and
underlying this is often not only an optical hindrance but a cere-
bral inaptitude. I have sometimes spent a long time in vainly try-
ing to elicit from such persons evidence of binocular vision, when
each eye had adequate acuity and apparently each was capable of
fixation upon the desired point. Evidently the images fall upon
parts of the retina very nearly correspondent, but there was no
cerebral impulse to binocular vision. For such cases usually only
one eye is to be corrected.
For the class of patients whose ocular discrepancies are so great
that they always use only one eye, nothing is to be done save to aid
as perfectly as possible the working member in case it needs assist-
ance. The other is to be left to purely ornamental functions.
ERRORS OF REFRACTION.
131
Incidental Effects of Glasses.
It has been previously remarked that glasses worn at the an-
terior focal distance (13 mm. in front of the eye) do not alter the
visual angle. But nevertheless the linear dimensions of the image
are greater with a convex glass, and less with a concave glass.
Moreover, if a convex glass be held farther from the eye its magni-
fying power is augmented. This is true of distant objects and also
obtains for near objects, provided the eye is adapted to a point
whose distance is greater than double the focal length of the glass.
(Landolt, 1. c, p. 358.) Dropping the glass to the tip of the nose,
provided the glass be moderately strong, increases its power. Re-
moving concave glasses from the eye diminishes their power. Con-
vex glasses limit the range of accommodation—concave glasses in-
crease it. The estimation of distances is changed. Convex glasses
make objects seem nearer and they alter the sense of relief or per-
spective. Concave glasses make objects seem more remote. For
these reasons persons are often made dizzy on first wearing glasses
or on taking them off. They have difficulty in walking or in reach-
ing objects. With very strong convex glasses the field of vision is
limited and there is a zone which is determined by the diameter
of the glass, where absolutely nothing can be seen (Berlin). This
constitutes one of the sources of annoyance to patients wearing
cataract glasses. Looking obliquely through strong glasses and
especially if they be cylindric, gives distorted images—hence persons
wearing them always turn their heads to see an object. Glasses
always disturb the relations between convergence and accommoda-
tion. When first used for presbyopia, the previous coincidence be-
tween these functions in reading gives place to diminished accom-
modation with unabated convergence, and the stronger the
glasses the greater the disharmony. A myope who uses his dis-
tance glasses for reading brings accommodation into unwonted
activity with diminished convergence. These perturbations re-
quire time for adjustment, and while as a rule, discomfort soon
ceases, sometimes it continues until special adjustments are made,
which will be considered under the head of asthenopia.
CHAPTER Till.
BINOCULAR VISION AND ITS DISTURBANCES.
Under this head are to be considered such troubles as muscular
asthenopia, or insufficiencies of muscles, strabismus, paralysis of
muscles, nystagmus, and conjugate deviation. Preliminarily it will
be proper to consider the anatomy and physiology of the muscles,
and the physiological properties of binocular vision.
Anatomy and Physiology of the Ocular Muscles.—Each eye
has six muscles. They are combined in pairs, and both eyes are co-
ordinated in particular ways. In each eye we have the internal
and external, the superior and inferior recti, and the superior and
inferior obliqui. All the recti muscles take origin from the apex
of the orbit around the foramen opticum, and come forward to be
inserted into the sclera in front of the equator oculi, about 7 mm.
behind the rim of the cornea, by flat and ribbon-like tendons. The
superior oblique or trochlearis also originates at this place; but,
inasmuch as it passes over a pulley at the supero-internal angle of
the front of the orbit, this becomes its functional place of origin
and assimilates its action to that of the inferior oblique, wilich
arises from the inner part of the inferior edge of the front of the
orbit. Both muscles then pass obliquely outward and backward to
wrap around the globe in thin, fan-like tendons, the superior going
over the upper part of the globe beneath the superior rectus, and
the inferior going over the inferior part of the globe beneath the
inferior rectus. The two obliqui hold the globe, as it were, in a
sling, which is entirely to the outer side of the optic nerve. The
recti, combined in action, retract the globe into the orbit; the
obliqui, combined in acton, draw it forward. While the recti in
combination have a simple kind of action, the obliqui draw the
globe forward and turn the cornea outward. The rectus internus
(called by Merkel, rectus medialis) and the rectus externus, move
the globe about an axis which is vertical. The rectus superior and
the rectus inferior move it about an axis which is transverse to the
vertical plane, but which is also inclined so that its outer end is
more posterior, making an angle of 67° with the antero-posterior
axis of the globe. The axis about which the obliqui rotate the
globe passes from before backward and inward on a horizontal;
BINOCULAR VISION AND ITS DISTURBANCES.
133
plane at 35° with the antero-posterior axis of the globe (see Fig.
53). The obliqui thus acquire an action which moves the eyeball so
that the rim of the cornea turns like a wheel. Taken singly, the
muscles act as follows: the rectus internus turns the cornea in-
ward on the horizontal plane; the rectus externus turns the cornea
outward on the horizontal plane; the rectus superior turns the
cornea upward and slightly inward; the rectus inferior turns the
cornea downward and slightly inward; the obliquus superior turns
the cornea downward and outward, and rotates it from above
downward. The obliquus inferior turns the cornea upward and
Fig. 53.
outward, and rotates it from below upward. In effecting the move-
ments of the eyeball all the muscles co-operate: while some pre-
dominate, the rest antagonize them to give steadiness to the action.
The nerves for the muscles are the sixth or abducens for the
rectus externus, the fourth or trochlearis for the superior oblique,
and all the rest are supplied by the oculo-motor or third. The
blood-vessels are branches of the ophthalmic.
The following schedule indicates how the muscles co-operate in
effecting certain principal movements. For motion inward, i.e.,
adduction, the effective muscles are, R. interni and R. sup. and R.
inf., antagonized by R. externi and Obi. sup. and Obi. inf.
Motion Outward.—Abductors: R. externi, Obliq. sup., and Obliq.
inf., antagonized by R. int., R. sup., and R. inf.
134 DISEASES OF THE EYE.
Motion Upward.—R. sup., Obi. inf., and when the cornea passes,
a given point, the upper fibres of R. int. and R. ext. add to the
effect. The antagonists are R. inf. and Obi. sup.
Motion Dowmvard.—R. inf. and Obi. sup., while, when the cornea
gets below a given point, the lower fibres of R. int. and R. ext.
come into play. In motions upon a horizontal axis the R. sup. and
inf. incline the top of the vertical meridian respectively inward and
outward, which tendency is counterbalanced by the Obi. inferior
acting with the R. sup. and by the Obi. sup. acting with the R. inf.,
which perform the needful rotatory or wheel movement.
Taking, now, the concomitant action of the eyes, .we arrange the
muscles into groups of adductors, which turn the corneae toward
the median plane of the body; and of abductors, which turn the
corneae away from the median plane of the body. We also find
the muscles pairing off in other combinations in turning the eyes.
to the right side and left side respectively, and in the various dia-
gonal directions. The eyeballs are capable of accomplishing, with-
in a limited range, all possible combinations, but there are certain
restrictions which are imposed by the necessity that each must
direct its visual line exactly upon the object observed. Hence, for
a near object, whether on the median plane or away from it, there
must be a slight adduction, as well as an aim suited to the position
of the object. For remote objects there will be a degree of abduc-
tion, but never to transcend parallelism of the visual lines. We
are habitually more concerned with objects below the horizontal
plane than with those above it; and in the discussion of the move-
ments of the eyes, Meissner has taken an inclination of 15° below
the horizon as the primary position; others, however, assume the
horizontal plane as the primary position. The degree of mobility
of the emmetropic eye in young persons about a vertical axis is
from 42° to 51° inward, and from 44° to 49° outward (Donders). In
miopia this is much restricted.
The horopter is a line which represents the curve along which
both eyes can join in sight, and it is formed in this way: as the
eyes fix upon a given object far to the left side, and move far to
the right at the same inclination of the visual lines, they form a
triangle whose apex, as it passes from left to right, forms the
horopter curve for this plane. If the movement be in any other
plane, vertical or oblique, the horopter will be formed in the same
way for that plane. In its simplest form, as explained by Johannes
Mueller, it is a circle which passes through the centres of rotation
of each eye and through the apex of the point of fixation of the
visual lines. This statement is not strictly correct, but will suffice
for our purposes.
Physiology.—The fundamental and imperative law which gov-
BINOCULAR VISION AND ITS DISTURBANCES. 135
erns the muscles of the eyeballs is that the fovea centralis retinse
of each eye must be fixed upon the object observed. When this is
done, all objects lying in the same horopter will form images upon
the respective retinae which will lie at equal distances from the
foveas, and will, therefore, be appreciated as single, giving what is
called binocular vision. But objects beyond the horopter or inside
the horopter, will cast images on parts of the retinae not equally
distant from the foveae, and will therefore not be appreciated as
single, but create the impression of two objects, giving rise to
double vision. The maintenance of correct binocular vision is the
necessity which dominates the ocular muscles. If the back of the
eyeballs be divided into quadrants by vertical and horizontal
planes whose intersection shall be at the fovea centralis, and par-
allel to these lines we mark points one-tenth of a millimetre asun-
der, and then suppose the two retinae to be superimposed upon each
other so that the vertical and horizontal lines shall exactly coincide,
the points which we have imagined will of course also coincide.
These coincident points, which are equidistant from the centre, are
spoken of as correspondent points of the two retinae, and there are
of course as many of them as there are percipient points, i.e.,
bacilli in the two retinae. They are functionally homologated to-
gether according to the scheme just imagined. By virtue of this
arrangement binocular vision is rendered possible. It follows, of
course, that the nasal half of the left retina is linked with the tem-
poral half of the right retina; the nasal half of the right retina
with the temporal half of the left retina; the superior halves of
both retinae are linked together, and the same is true of the inferior
halves. This corresponds with the decussation of the optic tracts
in the chiasm.
Binocular vision is primarily conditioned by the supremacy in
acuteness of the fovea centralis above other parts of the retina.
But this condition is not the only factor in the function, because
experience shows that the brain must possess a certain competency,
which sometimes seems to be the quality deficient.
For all objects on which we do not fix the foveae, and which
consequently are not in the horopter, we have diplopia. If, for in-
stance, iu one hand a pin is held at sixteen inches, and in the other
another is held at eight inches, and upon the same line, when we
look at the distant pin the nearer is seen double, and vice versa.
We are not disturbed by double vision of objects on which we are
not fixing attention; the mind ignores the impression of the things
with which it does not concern itself. This is common experience
in shooting, in using the microscope, when the unused eye may be
wide open and nothing be known of what it sees. Diplopia follows
certain laws. For instance, if the left eye fix on an object and the
136
DISEASES OF THE EYE.
axis of the right cross that of the other at a point inside the object
—in other words, if there be excessive convergence, the image
which in the left falls in the fovea, and whose position in space
is projected along the visual line, will in the right eye fall to the
inner side of its fovea. Now, the position of the object in space
is decided by what the left eye sees, and the right eye has an
image which, if it were directed aright, would belong to an object
situated to the outer side of its visual line, viz., on its right-hand
side. This image is recognized, and is mentally located as if it
were on the right side of the object seen by the left eye. In other
words, if there be excessive convergence of the visual lines, there
Fig. 54.—Convergence with Homonymous Images.
will be diplopia with correspondent or homonymous images (see
Fig. 54).
If, now, while the left eye fixes an object, the visual line of the
right diverge, the image will, in the latter, fall to the outer side of
its fovea, and will be projected mentally as coming from an object
on the left side of the visual axis. Hence, for divergence of the
visual axes we have crossed or heteronymous double images (see
Fig. 55). If the left eye fix an object, and the right eye be directed
downward, the image in the right will fall below its fovea, and be
mentally projected above the image seen by the other eye. If the
left eye fix and the right eye be turned upward, the image will fall
above its fovea, and the projection of the image will be downward
below the true place of the object. A candle-flame is usually the
BINOCULAR VISION AND ITS DISTURBANCES. 137
object chosen, and a red glass is put before the fixing eye so as
readily to distinguish the presence and place of double images. If
the visual lines form a wide angle with each other, in the deviating
eye the image will fall at a great distance from the fovea, and the
result will be that the image is less distinctly perceived because
it impinges on a less sensitive part of the retina, and it will also be
projected to a greater distance from the true place of the object.
For these two reasons, the patient is then less likely to be awrare of
double images. It also happens that persons may fix wTith either
eye and ignore the image of the other. It is also true that in many
persons, and by some it is asserted that in all persons, one eye pre-
Fig. 55—Divergence with Crossed (Heteronymous) images.
vails over the other, just as one hand is more depended on, than
the other.
The possession of two eyes confers a quality of sight which is
not only more effective than with one only, but is also of a different
kind. With two eyes not only does the brain receive a double im-
pression, but each eye views an object from a different point and
receives a slightly different image. The interpupillary distance
varies in adults between 50 and 75 mm. The average may be taken
at 64 mm. If an object be within a moderate distance and of suit-
able size, say a cube a foot square, ajb 10 feet each eye sees it under
a different perspective, and while its form is evident to each alone,
the combination of the two images gives what is called the sense
of relief or depth. For moderate distance, viz., with the objects on a
table, we realize this impression very strongly and unconsciously
138 DISEASES OF THE EYE.
form our judgment of the solidity of objects by the dissimilarity
of the images portrayed on the respective retinae. We are also
aided in this judgment by the projection of objects against parts
behind them, and by running the eye along their outlines and from
one object to another. The movements which the eyes make are
regulated with extreme nicety and are capable of very fine adjust-
ments.
The acquisition of binocular vision belongs to the first months of
life. Young infants roll their eyes about in the most inconsequen-
tial fashion, and when their visual vagaries give place to binocular
fixation, an important step has been gained in ocular and cerebral
development. In some subjects this function is never acquired, in
others it may be lost after having been presumably acquired. All
cases of permanent strabismus are instances of suppressed, or of
lost, or of undeveloped binocular vision.
The limitations of binocular vision evidently exclude those
motions by which the visual lines are not directed in proper har-
mony with each other. That is, one eye may not look up and the
other at the same time look down, and the same is true of diagonal
movements—they must be harmonious. But in the horizontal, or
nearly horizontal plane, the motions of adduction and abduction
can be pushed to an extent which shall disharmonize the visual
lines. Thus, we may turn the eyes inward so that the visual lines
cross by excessive convergence, and we may turn them by abduc-
tion so that they fall into divergence. Excessive adduction is pos-
sible without artificial aid to a marked degree, excessive abduc-
tion is never great, and cannot usually be effected without the aid
of prisms. Done by their help, the movement is made to prevent
double sight. Thus, if we look at a candle-flame twenty feet away,
and put before one eye a prism of five degrees angle, with its base
inward, there will for a moment seem to be two candles, but pres-
ently they move toward each other and fuse into one. The eye-
balls go asunder by a movement of abduction to bring the fovea of
one eye inward to the spot on which the prism has deflected the
candle-flame. This power of abduction beyond parallelism reaches
to a prism of from six degrees to eight degrees in most persons,
while adduction for distant objects, say at twenty feet, extends to
thirty degrees or to fifty degrees, and by cultivation may go higher.
It has already been mentioned (page 19) that with different
angles of convergence there will be variations in the possible
amount of accommodation, and this is known as the relative accom-
modation. It is determined by employing convex and concave
spherical glasses for particular angles of convergence. We also
have for given amounts of accommodation variable degrees of con-
vergence possible, as can be shown by prisms placed with bases
BINOCULAR VISION AND ITS DISTURBANCES. 139
inward or outward. In emmetropia the relation of the functions
of accommodation and convergence is very different from what we
find in myopia and in hyperopia. In myopia with less accommoda-
tion, convergence is greater, in hyperopia with more accommoda-
tion, convergence is less. We also find great differences among in-
dividuals both by natural endowment and from disturbing causes.
Not only is an adequate amount of accommodative power essential
to comfort in near work, but equally so is a proper proportion of
adductive and abductive power. In fact, for all visual functions a
correctly adjusted and adequate muscular apparatus is indispensa-
ble to comfort and efficiency. We do not yet possess sufficient
data to state precisely the necessary physiological conditions in the
power of the muscles, but failure in this respect brings on serious
functional troubles, which are classed under the name of asthen-
opia and under this head we shall discuss the matter.
PARALYSIS OF MUSCLES.
We may have paralysis, either complete or incomplete (paresis),
affecting one muscle or many, in one eye or in both. Symptoms are:
1st. Limitation or irregularity of motion:
2d. False position of the visual axis causing squint when
motions in certain directions are attempted. The squint affects at
first only one eye, but soon extends to both, by secondary contrac-
tion of opposing or associated muscles. For example, if the left
rectus externus be paralyzed, not only will the left rectus internus,
by reason of the diminished resistance, turn the eye unduly inward,
but the rectus internus of the right (opposite) eye will undergo con-
traction, and if the left eye look straight forward, the right eye
will squint inward. This is because the right rectus internus is
associated, in all movements to the left, with the left rectus ex-
ternus. Moreover the secondary deviation will exceed the primary;
i. e., if the right eye fix on the finger at one foot, the turning inward
of the left will be less in angle, than will be that of the right when
the left fixes on the finger. In other words, contraction of the right
internus exceeds that of the left internus.
3d. Double Images.—This is the usual consequence of disturb-
ance of the pre-existing normal co-ordination. In partial paraly-
sis some persons have a singular capacity for correcting the diplopia.
This power depends upon the instinct for binocular vision, and is
called the capacity for fusion. With the same degree of deviation,
so far as can be estimated by prisms, the extent over which fusion
of double images is achieved will be much greater in some persons
than in others. Von Graefe pointed out that, apart from errors of
refraction or accommodation, or amblyopia, the capacity for fusion
140
DISEASES OF THE EYE.
is far less in cerebral paralysis than in orbital or basilar paralysis.
The reason is that binocular vision is essentially a cerebral function.
4th. Incorrect projection of the field of vision.—Because of our
habitual reliance on the muscular sense we suppose that the effort
we make is followed by the effect to which we are accustomed, but
find that our assumption of the position of objects in the field of
vision is wrong. For instance, if the left rectus externus is para-
lyzed, especially if only partially paralyzed, and the left eye attempt
to see an object to the left side, the effort of movement is so much
greater than is usually made, that the mind believes the object to
lie much farther to the left than it really does, and the hand, in
attempting to seize an object or to put the finger on the point of a
pencil, strikes to the left side of the true position, i.e., the projection
of the field is too far on the side of the action of the muscle.
5th. Dizziness, nausea, and such cerebral symptoms are often
present, and may after a time pass away. They are caused by the
contusion of images and by the dissociation which is produced be-
tween the conscious effort of the muscles and the instability and
falsity of the projected field. Objects are made unsteady, the
ground does not seem level, going up and down stairs becomes diffi-
cult, movements of the hands are ill directed, and from all these
phenomena mental confusion and vertigo result, until further expe-
rience corrects the judgment.
If one eye only be involved, the inclination is to close it.
6th. Another effect is a peculiar attitude which the head as-
sumes to obviate double images. The inclination of the head,
when this occurs, will be such as to favor the lamed muscle, and
will be in its line of action and toward its virtual or anatomical
origin. For a paralyzed rectus externus of the left eye, the head
will turn on a vertical axis to the left. For a paralyzed rectus su-
perior of the left eye, the head will turn on a horizontal axis up-
ward and a little to the right. For the obliquus inferior the ten-
dency will be the same, both being levators of the cornea, and the
head thrown back diminishes the effort in looking upward. Some-
times pain is a prominent symptom and very often it is absent.
Diagnosis.—We meet in practice with the most complex com-
binations, and sometimes it is indeed a puzzle to tell what muscles
are at fault. We place most reliance on the character and position
of the double images, but to a clear analysis it is necessary to have
an intelligent patient with two good eyes each of which shall be
quick to observe the image it receives. To complicate the problem,
secondary contractions and involuntary compensations by other
muscles, may come in to disturb the regular scheme which ought
theoretically to be observed in the behavior of the double images,
and we are left in the lurch. But in many recent cases we can tell,
BINOCULAR VISION AND ITS DISTURBANCES. Ul
without analysis of double images, what muscle is affected. The
eye refuses to move to the proper degree in the direction of the
movement of the impaired muscle, and goes too far to the opposite
side; its movements are often partial and jerky. If many muscles
are paralyzed, the situation of the globe in the orbit may be altered
i.e., exophthalmus may occur. '
To comprehend the value of double images for diagnosis of ocular
Paralysis, a few illustrations are employed, which are borrowed
Irom Zehender ("Handbuch der Augenheilkunde," 1874, p. 317) and
somewhat modified. It has already been stated that two images
on the same level, of which the right belongs to the right eye and
the left to the left eye, are called homonymous or correspondent.
Images on the same level, and of which the right belongs to the
left eye and the left to the right eye, are called crossed or heter-
onymous. The former implies impaired power of abduction, i.e.,
the eyes are convergent; and the latter implies impaired power of
adduction, that is, the axes are divergent. We have also to study
differences in height, i.e., vertical diplopia; and the higher image
belongs, as before said, to the eye which points too low, and means
impaired power of lifting the cornea, i.e., the levators are at fault-
The lower image belongs to the other eye. Again, we are to note
whether the images are parallel to each other, and for this we use
as a test a long candle or a stick about a foot long. The images
may converge or diverge at the top. The cause will lie in deflec-
tions of the vertical meridians. We always speak only of the top
of the vertical meridian. If now these meridians diverge, the
images will incline inward; if they converge, the images will diverge.
The obliqui are thus submitted to proof, while to a less degree
the same phenomena belong to the recti superiores and recti
inferiores.
The images are figured as they are seen by the patient.
The white candle denotes the image seen by the sound eye, the
dark candle that seen by the paralyzed eye. (In practice it is
better to put a red glass over the eye which fixes, and which is
generally the sound eye, so that the image seen indirectly and by
the paralyzed eye may be relatively more distinct. This sugges-
tion is not observed in the diagrams.) It is supposed that the
candle is carried to the several positions represented across the
field and the shaded part of the diagram indicates the region in
which double images arise—viz., right or left, above or below.
Fig. 56 shows the double images in, 1st, paralysis of the rectus
externus oculi sinistri, and likewise those in, 2d, paralysis of the
rectus internus oculi dextri—the one being the counterpart of the
other, except that in the former the images are homonymous, in
the latter they are crossed. If the same figure were looked at
142
DISEASES OF THE EYE.
through the paper from its back side, or were looked at as reflected
from a mirror, it would be reversed, and then would represent, as
seen in Fig. 57, 3d, paralysis of the rectus externus oculi dextri, or
4th, paralysis of the rectus internus oculi sinistri. In the third
case the images are homonymous; in the fourth, they are crossed.
The images viewed on the horizon are vertical and parallel, while
Fig. 56.
Fig. 57.
the eyes turned up cause them to diverge at the top, and, if turned
down, to converge at the top to a slight and physiological degree.
In looking upward the rectus superior predominates and causes the
vertical meridian to converge—hence, divergence of the images;
and vice versa, in looking down, the action of the rectus inferior
causes the images to converge.
Fig. 58.
Fig. 59.
Fig. 58 gives the situation and relation of the images in, 5th,
paralysis of the rectus superior oculi sinistri, and its reverse in Fig.
59 exhibits, 6th, paralysis of the rectus superior oculi dextri. It is
noticed that difference in height (Fig. 58) increases toward the left,
and obliquity increases toward the right, the reverse occurring in
Fig. 59. In both cases the images are crossed.
In Fig. 60 we have the images seen in 7th, paralysis of the rectus
inferior oculi sinistri, and reversed in Fig. 61 of, 8th, paralysis of
rectus inferior oculi dextri.
BINOCULAR VISION AND ITS DISTURBANCES. 143
In this figure again the images are crossed, and they diverge
more widely toward the side of the affected muscle, and the obliquity
diminishes toward the same side. The figures give the diplopia
only in the extreme upper and lower parts of the field, where the
difference in height is greatest; on the median line it will be less,
and at the opposite part of the field there will be single vision.
Fig. 60. Fig. 61.
*In Fig. 62 are represented the double images found in, 9th, para-
lysis of the obliquus superior oculi sinistri, where they are homony-
mous; and if reversed, as in Fig. 63, we have, 10th, paralysis of
obliquus superior oculi dextri.
In these cases the notable thing is that, besides being homony-
mous, there is difference in height and remarkable obliquity.
Fig. 62.
Fig. 63.
The vertical separation increases on the side of the sound eye, while
the obliquity increases on the side of the impaired eye. Paralysis
of the inferior oblique, which is rare, gives double images in the
upper part of the field, and with difference in height as well as
lateral displacement, the images being crossed, which is the fact in
the case of the writer who has this affection, but by most author.
ities they are called homonymous (see Trans. Am. Oph. Soc, 1879,
p. 551) and diverging at the top.
144
DISEASES OF THE EYE.
Not infrequently one image seems to be farther removed than
the other. To aid a patient in describing what he sees, it will be
well to let him have a stick in each hand, and with them to imitate
the position of the images. To determine which is the true and
which is the false image is generally easj*, because the patient will
naturally fix with the sound eye. We shall also be guided by
other symptoms in deciding upon the faulty eye. By observing
in what direction the least displacement occurs and the line in
which the images separate most widely, the erroneous eye will
soon be detected. Moreover, while a patient fixes on an object,
if the screen be quickly shifted from one eye to the other, the eye
which does not remain steady, but makes a slight movement of
adjustment, is the affected one. Patients can by their own sensa-
tions often tell which is the injured eye.
For isolated paralyses and for some cases of multiple paralysis
there will be no great difficulty in deciding which are the faulty
muscles. When all the twigs of the third nerve are thrown out of
function, we readily recognize it, because only the rectus externus
and superior oblique remain intact, and the pupil is moderately
dilated and there is ptosis. There will often be slight exophthal-
mus. We also sometimes have all the muscles of one eye paralyzed,
and it then stands immovably at the middle of the orbit and the
upper lid drops, while the globe has a tendency to exophthalmus.
We also have various combinations which are sometimes impossible
to unravel, and compensatory effects are sometimes added to the
original complications. The above schemes of double images will
often fail to be realized and are simply guides for investigation.
Having discussed the ocular phenomena and ascertained what
muscles are at fault, we are confronted with another problem in
diagnosis, viz., where the lesion is situated: whether in the orbit or
within the cranium. The sphenoidal fissure is a favorite site of
paralytic lesions. This opening, closed by a dense fibrous mem-
brane, is chiefly occupied by the cavernous sinus, on whose wall at
its medial side is a fibrous canal by which the nerves enter in a
bundle, viz., the three motor and the ophthalmic branch of the
trigeminus, while the ophthalmic vein passes out below the oculo-
motorius. An intra-cranial lesion may affect the nerves at the
base of the skull, or within the cerebrum. We find the trunks as
they enter the brain subdivided into fasciculi or rootlets, which ter-
minate in nuclei located in the mid-brain, and from these nuclei
connecting fibres proceed to the cortex. We thus subdivide intra-
cranial paralyses into, 1, basal; 2, fascicular; 3, nuclear; 4, sub-
cortical; and. 5, cortical. We must have a critical knowledge of
anatomy to be qualified to understand or investigate the intricate
problems of pathology which now challenge our study.
BINOCULAR VISION AND ITS DISTURBANCES. 145
Leaving the sphenoidal fissure, the motor nerves pass backward
alongside the sella turcica, and we find them ranged in the follow-
ing order from the median line outward: The 3d, the 4th, the
6th, and to the outside, the trunk of the 5th, with the ganglion of
Gasser lying on the anterior face of the pars petrosa.
It would be unsuitable to enter deeply into details in a treatise on ophthal-
mology, but we want at least an approximate knowledge of the place of the
lesion if it be located within the cranium. We therefore present the follow-
ing anatomical summary—relying largely on illustrations.
A transverse section through the frontal bone just behind the apices of
the orbits discloses an instructive view of the cranial cavity, as seen in Fig. 64
from Henle. It gives a section of the cavernous sinuses and of the internal
carotid arteries; the tentorium being preserved, we are reminded of the im-
portance of the dura mater,
both in supporting the oc-
cipital lobes, in determin-
ing the cause of exuda-
tions and abscesses, and
also its influence, when in-
flamed, upon the nerves
and brain tissue because of
its large enveloping surface
and penetrating septa. We
see also the large cerebral
sinuses. Another impor-
tant elementary fact of
which we may be reminded
is the arterial supply of the
base of the brain. The
vertebrals unite at the pos-
terior edge of the pons to
form the basilar artery,
and this bifurcates upon
the crura cerebri (pedun-
cles) to enclose within the
circle of Willis the corpora
albican tia (mammillaria)
, , . . , . Fig. 64.—1, Sphenoid sinuses; 2, internal carotid artery; 3,
ana tlie optic Ciliasm, inferior maxillary nerve; 4, tentorium cerebelli; 5, falx cerebri.
alongside of which come
up the internal carotid.s after their sinuous twisting; and in front of the
chiasm the circle of Willis is completed by the anterior communicating artery.
Leaving out of view the hemispheres we limit our study chiefly to the
parts about and caudad the third ventricle. A ventral view (see Fig. 65)
displays the cranial nerves and the various organs along the median line as
far as the decussation of the pyramids. It will be remembered that the pons
Varolii lies upon the basilar process and that along its median line is a furrow
for the basilar artery. Fig. 66, from which parts have been excised, may be
placed alongside Fig. 65 to complete the mental picture. We may simply
call attention to the optic tracts which part from each other at the chiasm
and pass laterally and caudad to twine around the crura cerebri and lose
themselves in the brain substance. We note the two geniculate bodies i
10
146
DISEASES OF THE EYE.
and e, of which only the external (lateral) has any connection with vision
(Fig. 65). At the heel of the optic thalamus, Th, is a nidus of gray matter
called the pulvinar (Fig. 66), which, like its neighbor the external genicu-
late body, is a primary visual ganglion. The cross X between the crura
(Fig. 65) denotes the pos-
terior perforated space, a
lamina of gray matter
pierced by small arteries,
which is the floor of the
third ventricle.
In Fig. 67 from Henle,
we have an alcoholic speci-
men, showing the third
ventricle and the beginning
of the crura: the section is
presented a little obliquely
and the tractus opticus II'
is turned aside. The crus
cerebri appears near the
optic thalamus and is cut off
just above the pons. In the
crus we distinguish the ven-
tral portion B, basis or
crusta, and the dorsal por-
tion T or tegmentum. Be-
tween is a thin ganglionic
layer, Sn, the substantia
nigra, or ganglion of Soem-
mering. In the basis of the
crus are gathered the fibres
forming the internal capsule
which have come down from
the cortex, and which are
chiefly motor. In the teg-
mentum the fibres are most"
ly sensory, besides nuclei
and gray matter. It in-
cludes very dissimilar struc-
Fig. 65.—Base of the Brain and Cranial Nerves, Pons and
Medulla. I to XII, The cranial nerves; Th, optic thalamus;
h, pituitary body; tc, tuber cinereum; a, corpora albicantia .
(mammillaria); P, pes peduneuli; i, internal geniculate tures, and extends ironi tlie
body; e, external geniculate body; PV, pons Varolii; pa,
anterior pyramid of medulla; t>, olive; d, decussation of an-
terior pyramids; ca. anterior column of spinal cord; cl,
lateral column of spinal cord; Ce, cerebellum; fl, flocculus of
cerebellum; X, locus perforatus posticus; + (on the left side),
ganglion of Gasser; + (on the right side), motor root of tri-
geminus.
thalamus to the crossing of
the pyramids. It is merely
a convenient topographical
term, viz., the roof of the
basis of the crus. Dorsad
to the tegmentum is the canal of Sylvius, and the roof of the latter is made
by the corpora quadrigemina and the velum medullare anticum (valve of
Vieussens). The substantia nigra, and cephalad to it the corpus subthal-
amicum (Luy's body), and caudad the red nucleus of the tegmentum are a
series of ganglia located between the crusta and the tegmentum, to which
we merely allude. The crura, as seen in Fig. 65, diverge from the pons to
the thalami optici, have a slightly fluted and twisted appearance, and
enclose a triangular area called the posterior perforated space. At the apex
of this triangle we have the emergence of the third nerves.
BINOCULAR VISION AND ITS DISTURBANCES. 147
Our business is chiefly with the ocular nerves, and it would be natural to
enter at once upon a description of their course andrelations in the medulla.
We cannot appreciate their characteristics and the events which may happen
when lesions attack them, without acquaintance with their environment
and the functions, so far as known, of the organs amid which they are placed.
We shall therefore not go astray if we sketch broadly the features of
the medulla and the pons. F< >r an accurate study see the article on '' Brain:
histology," by Spitzka, •' Reference Handbook Med. Sciences" (Wm. Wood
& Co., 1887). The translations of Obersteinerl and Edinger2 will be freely
used. Dr. M. A. Starr has treated the subject in a most lucid manner in an
article3 in the li Reference Handbook of the Medical Sciences." Dr. Starr
traces the several tracts which go through the medulla and enumerates
four: 1st, The tracts connecting the cerebrum and cerebellum. They are
the outer and inner thirds of each pes, and they are to a large extent lost
upon the masses of gray matter which are intermingled with them and
with the transverse fibres of the pons. They also make up the superior
P Ca
Fig. 66.—Base of the Brain to show the Emergence of the Optic Nerves. The cerebral hemi-
spheres have been removed to show the posterior portion of the optic thalamus. The peduncles
have been divided behind the corpora quadrigemhia. Cgm, Corpus geniculatum mediale; Cgl,
corpus geniculatum laterale; P, pulvinar of the thalamus; Ch, chiasma nervorum opticorum;
Cq, corpora quadrigemina; Aq, aqueductus Sylvii; Ccb, pedunculus cerebri; Cc, corpora can-
dicantia (mammillaria); Tc, tuber cinereum; .7, infundibulum; 1, tractus olfactorius separating
into its middle and lateral roots; Spa, substantia perforata anterior.
and middle peduncles of the cerebellum. The inferior peduncle of the
cerebellum is continuous with the columns of the cord. We also have in
the pons, fibres passing from one hemisphere of the cerebellum to the
other. The longitudinal fibres grow less numerous as Ave proceed caudad,
being reduced to about one-fifth in volume. Lesions in the superior and
middle peduncles of the cerebellum are not easily recognized, giving rise,
like those due to cerebellar disturbance, to loss of equilibrium.
In the 2d division we have the middle third of the fibres of the pes or
crusta which compose the great motor tracts coming from the motor areas
of the cortex. Those for the arm and leg cross at the pyramids at the lower
end of the medulla and form the lateral columns of the cord; that is, about
•"Central Nervous Organs," Phil., 1890.
■ '' Structure of the Central Nervous System," Phil., 1891.
s " Pons Varolii and Medulla Oblongata, Diagnosis of Local Lesions in,"
vol. v., p. 773.
148 DISEASES OF THE EYE.
four-fifths of the fibres decussate, and the remainder continue straight down
the anterior columns. Besides the above fibres we have in the pons special
nuclei for the facial (seventh) and hypoglossal (twelfth) nerves, and the
fibres from them also decussate, but at higher levels than the pyramids.
Hence various combinations of paralysis of face, tongue, and extremities are
possible according to the place of lesion. The figures from Starr illustrate
these possibilities (Figs. 68, 69).
The 3d set of fibres is the tract for muscular sense, called the fillet or
lemniscus, and when in the medulla called interolivary tract. It conveys
sensation upward, and for this reason we trace it in this direction. The
columns of Coll and Burdach in the dorsal surface of the spinal cord ter-
minate at the nuclei gracilis and cuneatus. The fillet starts from these
nuclei, and as it goes cephalad the two bands decussate at a point above the
pyramids (sensory crossing of Wernicke) and enter the pons. It becomes
ribbon-like, and later assumes an L shape, lying beneath the deep transverse
bundles of the pons, in front of the formatio reticularis. In the tegmentum
it lies mainly to the outer side and reaches the cortex, passing through the
caudal part of the internal capsule.
4th. The sensory tract, which transmits sensations of touch, temperature,
Fig. 67 (Henle).—Right Half of Brain turned obliquely upward—the peduncle T divided near
its entrance into the thalamus Tho; II', the optic tract turned back. I, Olfactory nerve; CcV2,
knee of the corpus callosum; SI, septum lucidum; Coa, Com, commissura anterior and media; Cn,
conarium (pineal gland); Lq, lamina of corpora quadrigemina; Ccl*, splenium of the corpus cal-
losum; Sn, substantia nigra; B, basis; Cca, peduncle of the corpora mammillaria.
and pain through the medulla and pons, is in the formatio reticularis. This
portion of the medulla and pons lies just beneath the gray matter of the
floor of the fourth ventricle, behind the tracts hitherto described. It is made
up of nerve-fibres passing in three directions : a, transversely, the commis-
sural fibres of the cranial nerve nuclei; b, from the nuclei ventrad, the fibres
of the cranial nerve-roots and arciform fibres; e, longitudinally, the sensory
tract. The longitudinal fibres can be traced from the gray matter and
various columns of the spinal cord, through the formatio reticularis to its
upper level in the tegmentum of the crus, whence they issue to enter the
posterior part of the internal capsule. In the medulla the formatio reticu-
laris of each side is divided into two parts by the line of exit of the
nerve-roots of the twelfth. The inner part, near the median line, con-
tains the continuation of the anterior and anterolateral columns of the
spinal cord, and the interolivary tract or lemniscus already described. The
outer part contains the sensory tract now under consideration. In the inner
two-thirds of this outer part the fibres pass which convey impressions of
BINOCULAR VISION AND ITS DISTURBANCES.
149
touch, temperature, and pain from the opposite half of the trunk and limbs
in the outer third of this part is found a column of peculiar structure re-
sembling the substantia gelatinosa of the posterior horn of the spinal cord
and in this column terminate the fibres of the sensory part of the trigeminal
nerve which turn downward after entering the pons Varolii, and ter-
minate at different levels in the pons and medulla (see Fig. 69). Thus the
outer position of the formatio reticularis contains the sensory tract from
the face of the same side. It is evident from the diagram that a lesion
which involves one-half of the formatio reticularis in the pons and medulla
will produce an alternating anaesthesia, i.e., loss of sensation on one side
of the body and on the other side of the face and head (lesion at B, Fig.
69). Alternating anaesthesia is as characteristic a symptom of lesions
of the formatio reticularis of the pons and medulla, as alternating pa-
ralysis is of lesions in the motor tract of the pons. In the upper part of
the pons the sensory tract from the face crosses the median line, and hence
;yjr nucuvs.
WHERve:
Y nerve
Fig. 68.
Fig.
Fig. 68.—Lesion of Pons, with Alternating Motor Paralysis. Lesion at A causes hemiplegia of side
opposite to lesion; lesion at B causes alternating paralysis, viz., body on right side, face on left side;
lesion at C causes paralysis of tongue on side of lesion and of extremities of opposite side; lesion at
D causes paralysis of extremities of opposite side; lesion at E causes paralysis of extremities of
both sides.
Fig. 69 —Lesion of Pons, with Alternating Anaesthesia. The course of the sensory tract in pons
and medulla. A, Lesion in tegmentum cruris cerebri, or upper quarter of pons, producing hemian-
sesthesia of the opposite side; B, lesion in formatio reticularis of pons or medulla, producing alter-
nating anaesthesia.
a lesion in the formatio reticularis in the upper third of the pons, or in the
crus cerebri, will produce a unilateral anaesthesia (lesion at A, Fig. 69).
A lesion involving both halves of the formatio reticularis will produce
bilateral sensory symptoms.
It is evident that a lesion of any extent, either in the pons or medulla, will
inevitably destroy one or more of these four tracts, and consequently will
produce serious symptoms of wide extent and of considerable diversity
(Starr).
If now we make a section normal to the axis of the pons through the
width of the anterior corpora quadrigemina (Fig. 70), we shall have a view
150 DISEASES OF THE EYE.
of the rootlets of the third nerve as ttiey gather together to form Its stem.
We see that they traverse and surround a rounded mass of reticular sub-
stance called the red nucleus of the tegmentum. This body extends cepha-
lad to the optic thalamus and receives fibres from the fasciculus retroflexus
(Meynert's bundle), and also connects with the cerebellum by its superior
peduncle. It has nothing to do with the oculo-motor nerve. Below and
laterally is the substantia nigra of Soemmering, into which run fibres from
the lenticular nucleus (see Fig. 67), and below that the crusta of the pes.
On the median line at Jf is a decussation of fibres, spoken of above, named
after Meynert, and below this another decussation named after Forel, F. At
Fig. 70.—Cross Section at Caudal Edge of Ant. Corp. Quad. Tho, Thalamus opticus; Cgm,
corpus geniculatum mediale; Cgl, corpus geniculatum laterale; II, tractus opticus; Pp, pes pedun
cuh; SnS, substantia nigra; Ntg, red nucleus of the tegmentum; 727, root fibres of the oculo-mo-
tor nerve; Pcm, pedunculus corporis mammillaris; M, Meynert's fountain-like tegmentum crossing;
Lm, lemniscus; Fcop, fibres in formatio reticularis which have come from the posterior commis-
sure: Vd, descending root of trigeminus; Aq, aqueduct; Ftp, fasciculus longitudinalis posterior;
Brqp, braehium from corpora quadrigemina; Nqa, nucleus of anterior corpora quadrigemina;
Sqt, sulcus transversus; Sqs, sulcus longitudinalis; F, Forel's ventral decussation of tegmentum.
Lm is a portion of the fillet, the tract for muscular sensation, and mesial to
it Fcop indicates a spot where fibres stream from the posterior commissure
in the roof of the aqueduct into the tegmentum. This locality is made up
of fine arcuate fibres giving it the name already used of formatio reticularis
tegmenti, area reticularis, etc. At the extreme lateral side of the figure we
have a portion of the thalamus and of the two geniculate bodies Cgl, Cgm.
and below is the optic tract. Below at the middle of the figure in the sulcus
is a bundle of fibres coming down from the mammillary bodies. Mesially next
the middle geniculate body is a section of the posterior commissure or
braehium conjunctivae of the corpora quadrigemina, and at Nqa is its nucleus
designated by Qa. Cephalad of the region displayed in this section are other
ganglionic masses, the corpus subthalamicum (nucleus of Luys, etc.), the
ganglion habenulaa, the mammillary bodies, and these structures must be left
out of consideration, as well as various bundles of fibres which have been
isolated and described. Lastly, it must be stated that the walls of the third
ventricle, of the aqueduct and the floor of the fourth ventricle are covered by
a layer of gray matter, which has important functions. It is conjectured
that this substance in the third ventricle has influence upon sleep; we
know that lesion of the floor of the fourth ventricle is a cause of diabetes
mellitus. The respiratory centre in the vagus nucleus will not be forgotten.
BINOCULAR VISION AND ITS DISTURBANCES.
151
The remarkable complexity of this region renders its pathological phe-
nomena exceedingly difficult to analyze.
Dismissing the parts which are only accessory to the main object of the
Yl
W
JtuclAud ■■
A/uclFa-cial
present study, we take up the nuclear origin of the ocular and associated
nerves.
We here introduce a composite (Fig. 71) from Edinger to show on a sagittal
152
DISEASES OF THE EYE.
section the relations of the parts under consideration. The black lines de-
note motor structures, the red lines sensitive structures. The lettering ex-
plains the figure, and we shall frequently refer to it.
Another cut presenting the parts in face wiU serve to aid in locating the
relations of the origins of the nerves (Knies).
3d Ventricle......
Aqueduct. Sylvii ---.._
Anterior and")""' "" "
\ Ccrp, quad
Posterior X........___w__
Pedunc. Cerebri
4th Ventricle
Accommod.
Sphincter iridis
III
Central Canal of Cord _.
Fig. 72.
The third nerve, oculo-motorius, turns inward around the crus cerebri,
approaching its fellow of the opposite side, and enters the brain at the ceph-
alic and ventral edge of the pons (see Fig. 71). The fourth nerve climbs the
outer side of the crus and enters the brain at the cephalic and dorsal edge of
the pons. It is slender and exposed for a long course. The sixth follows
down the basilar process and disappears at the caudal and ventral edge of the
pons. The nearness of the oculo-motorii to each other at the posterior per-
forated space makes them liable to be both implicated in a single lesion, such
as a tumor or hemorrhage (see Fig. 65).
We trace the third nerve into the brain, and we find it terminate in
a group of nuclei beneath the aqueduct of Sylvius lying upon the fibres of
the posterior longitudinal bundle (see Fig. 71). These nuclei have been most
successfully studied and we possess a probably accurate knowledge of
their individual functions. Kahler and Pick,1 and afterward Starr,2
arranged them in the order of their function, and the recent studies
of Perlia3 and Siemerling4 have only slightly modified their order. The
nuclei of the two oculo-motor nerves combine to form an almost regular tri-
1 Zeitschrift fur Heilkunde, 1881.
'2 Journal of Nervous and Mental Diseases, May, 1888.
3 Graefe's Archives Ophth., xxxv., iv., 287, 1889.
4 Archiv f. Psychiatrie und Nerven krankheiten, xxii., Supple, heft, 1891.
BINOCULAR VISION AND ITS DISTURBANCES. 153
angle of broad base, its sagittal length about 10 mm., and it lies between the
caudal portion of the third ventricle above the mammillary bodies and the
region of the posterior corpora quadrigemina, occupying the whole of the
floor of the iter e tertio ad quartern ventriculum (aqueduct).
There are, according to Perlia, seven clusters which can be more or less
perfectly isolated and arranged in pairs, and there is one on the median line
common to each side, making eight nuclei for each third nerve (see Fig. 73).
Of the paired nuclei, two in front are separated distinctly from those behind,
which are more closely approximated. Of the caudal pairs two are above (dor-
sal) and two below (ventral). The most caudal, which is also ventral comes
1 Sphincter iridis.
4 Rect. sup.
5 Obliq. inf.
Fig. 73.
2 Levator palp. sup.
3 Mus. ciliaris.
6 Rect. intern.
7 Rect. inf.
9 Trochlearis.
O be
2 Levator palp. sup.
3 Mus. ciliaris.
6 Rectus internus.
7 Rectum inf.
9 Trochlearis.
Fig. 74.
1 Sphincter iridis.
4 Rect. sup.
5 Obliq. inf.
close to the nucleus for the trochlearis. The figure from Perlia (72) is num-
bered according to the scheme of Knies,1 and the nuclei may be placed in
two rows, of which the dorsal are also lateral, viz., 4 and 5. See also cut
of same nuclei from Edinger (Fig. 74).
This arrangement differs from that of Kahler and Pick2 (see Starr, 1. c.) in
placing the levator palp, nearer the median line and cephalad, and places on
the median line the nucleus for convergence. The close vicinity of the levator
palp, to the rect. superior is not disturbed, a relation abundantly confirmed
by clinical experience. Other parts remain the same. The dorsal nuclei are for
the rectus superior and the obliquus inferior. The ventral are for the rectus
internus and rectus inferior. It is seen that a very natural collocation exists;
1 Archiv f. Augenheilkunde, xxiii., 44, 1891.
sZeitschrift fur Heilkunde, 1881.
154
DISEASES OF THE EYE.
that the group of adductors, R. int. (6), R. inf. (7) (both ventral), and R. sup.
(4) dorsal and overlapping, lie closely grouped and in contact with the com-
mon nucleus (8) for convergence. The levators, R. sup. (4) obliq. inf. (5) and
the depressors, R. inf. (7) and trochlearis (9) also lie in series. The muscles for
the pupil and for accommodation are together, although a little apart from
others, while accommodation and convergence take their closely related
positions. Decussation of fibres of the nuclei had been noted by Gudden
and others, but, Knies happily points out that the more accurate description
of Perlia authorizes us to say that the decussating fibres belong to the
nucleus for the obi. inferior (5), and put it in harmony with the nucleus of
the trochlearis of the same side whose roots have long been known to cross
in the velum medullare anterius (valve of Vieussens). Therefore the recti
muscles of the right side get their impulse from the nuclei of the same side
of the brain, but the obliqui of the right side take their impulse from the
opposite side of the brain, and vice versa. Hence, if a lesion involve all the
nuclei of the right half of the brain, the paralyzed muscles will be all the
recti, the pupil and accommodation on the same side, but the inferior ob-
lique of the opposite side. The trochlearis of the other side might or might
not be paralyzed.
Something further must be said respecting the connecting fibres. The
nuclei are situated, as has been said, upon the posterior longitudinal bundle
(see Fig. 71, Edinger), and Perlia (1. c, p. 302) signalizes a tangle of fibres
which hangs like a web above the whole group and spins out laterally
into the nuclei. Near the raphe they incline ventrally beneath the pos-
terior longitudinal bundle and turn cephalad in curves. Approaching the
third ventricle they bend around the peduncles and are lost in the sub-
stantia reticularis. We thus have two sets of connecting fibres, one sagit-
tal and coarse (Spitzka) and a finer set coming more or less vertically from
the gray matter of the wall of the fourth ventricle.
Perlia also calls attention to offshoots from the interpeduncular transverse
commissure of Hall and Gudden, leaving the optic tract in the anterior corpora
quadrigemina and descending to wind around the outer surface of the crus
to enter the base of the brain a little in front of the oculo-motor nerve. One
branch of this bundle goes to the nuclei of the anterior root of the 3d nerve,
and another branch traverses the middle of the nuclear group to gain the
nuclei of the trochlearis (1. c, p. 307). These connections establish relations
between the optic tract, the tubercula quadrigemina anteriora, and the nu-
clei of the ocular muscles. Such an arrangement explains Graefe's case (1856)
in which, with paralysis of all the extrinsic muscles of the globe, the reaction
of the pupil to light was destroyed, but its reaction to convergence and ac-
commodation remained. The recognition of a special centre for convergence
(8) and the existence of longitudinal fibres on the floor of the fourth ventricle
which connect the nuclei of the third (Edinger, Spitzka), make it clear why
contraction of the pupil may fail to occur under feeble stimulus of impaired
optic nerve fibres, but may occur with convergence ; and that convergence is
a more efficient agent than accommodation, as can be experimentally proved.
Just behind the oculo-motor nest of nuclei is found that for the troch-
learis. Its root fibres unlike those of the third mount upward, enter the velum
medullare (valve of Vieussens) and decussate almost if not quite completely,
so that the nucleus for the right superior oblique muscle lies on the left side
of the median plane and vice versa. We thus have the origin of each obliqae
muscle, both inferior and superior, located across the median line of the eye
designated—a singular harmony.
BINOCULAR VISION AND ITS DISTURBANCES. 155
The nucleus of the sixth, the abducens, lies beneath the floor of the ventricle
at about its middle, and the fibres go down through the body of the pons to
appear at its caudal edge (see Figs. 65 and 71). Between the abducens
nucleus and that for the internal rectus is a connecting bundle of fibres which
Siemerling claims has not been anatomically demonstrated, but which on
clinical evidence indisputably exists.
Besides the relations now described we take note of another important
fact, viz., that the abducens nucleus is included in a bend of the fibres of
the facial nerve at the point called the knee (Fig. 71). Indeed by some the
facial is said to have a nucleus of origin at or near the nucleus of the abdu-
cens, while Spitzka says it receives an accession of fibres from the posterior
fasciculus (the longitudinal bundle). The place of this remarkable relation-
ship is denoted by a slight bulge on the floor of the ventricle, the eminentia
teres. At this point originate those fibres of the facial which supply the or-
bicularis and frontal muscles, and we are enabled to understand how some-
times only the lower facial branches may be injured, as happens in bulbar
palsy, because only the lower (caudad) nucleus is involved—while the orbicu-
laris is spared. On the other hand Birdsall1 observed ophthalmoplegia ex-
terna in which the excitability of the orbicularis and frontal muscles was im-
paired. This tallies with the remark of Spitzka as to the accession of fibres
"aberrant from the oculo-motor nidus" to the facial from the posterior fasci-
culus, and with Mendel's2 experiments, Avho located the ocular facial nucleus
in the posterior part of the third nucleus; in this he coincides with Bech-
terew, 1883. This interesting relation teaches us the impulse to the act of
winking. We must also bear in mind the sensory reflex for the action of the
orbicularis, as shown in winking and blepharospasm, which brings us to the
fifth or trigeminus.
The fifth nerve is both motor and sensory, but we are chiefly interested
in its sensory portion. Its nuclear origin is very extensive, particularly in
the sensory portion, and its origins are both widely separated and remote
from the roots of other nerves ; the nearest being those of the fourth and
sixth. Tracing it from the surface of the pons, which it enters at about its
middle and remote from the median line, its sensory root passes backward
and inward to some small groups of cells lying beneath the floor of the fourth
ventricle known as the middle nucleus. The majority of the fibres, however,
turn downward through the pons and medulla under the name of the as-
cending root, being described as starting from all levels above the cervical
spinal cord as low as the second cervical nerve, and terminating in a long
column of gray matter which is continuous with the gelatinous substance of
the posterior horn (the gray tubercle of Rolando). Another portion of the
sensory root (descending) may be traced upward from the level of the mid-
dle nucleus as high as the upper part of the corpora quadrigemina, where
they arise from large round nerve-cells. They lie external to the aqueduct
of Sylvius and are arranged in the form of a crescent (see Fig. 71). These
fibres may be implicated in affections involving the ocular nuclei. The
majority of the fibres of the motor root arise from a nucleus near to and
inside of the middle sensory nucleus (see Figs. 71 and 72). Spitzka gives six
roots to the trigeminus, while Magnus indicates four.3
Following the special purposes of an ophthalmic inquiry, we touch inci-
1 Journal of Nervous and Mental Disease, Feb., 1887, p. 65.
2 Berlin. Klin. Wochen., 1887, p. 913.
3 See Edinger, p. 209, Am. Ed., also Obersteiner, p. 292, Am. Ed.
156 DISEASES OF THE EYE.
dentally on the close relations of the facial (7th) and thehypoglossus (12th) be-
cause their root fibres lie close together under the floor of the fourth ventricle
(see Fig. 71). The principal nucleus of the twelfth lies far down the medulla
and its origin is extensively distributed (see Fig. 72). It is usually with pa-
ralysis of the lower face that deflection of the tongue is linked and then the
lesion will be low down; but it may concur with impairment of the orbicu-
laris and then the lesion is high up near the eminentia teres and may com-
promise eye muscles, abducens, etc. Some singular cases in which the open-
Fig. 75.
ingof the mouth causes movement of the upper lid1 are accounted for by
assuming an abnormal connection between the facial nucleus, the upper nu-
cleus of the hypoglossus, and the motor oculi at the spot referred to.
1 See Michel, Jahresbericht, 1892, p. 354—and Hubbel, Arch, of Ophthal.
BINOCULAR VISION AND ITS DISTURBANCES. 157
We may dwell no longer upon the pons and medulla, but now state what is
held as to the cortical representation of the ocular motor nerves. On this
point our knowledge is not precise. Experimenters have reached different
conclusions. Mills1 gives a minute diagram of the separate subdivisions of
the motor region founded chiefly on the conclusions of Horsley and Schaefer.
They decide upon the second frontal convolution as the area for movement
of the head and eyes. Ferrier concurs with this view and also adds the
angular gyrus and superior temporo-sphenoidal convolution as capable of
the same effect. For details see Mills, 1. c, p. 236. Others quoted by Knies2
fix upon the lower part of the parietal lobe, also upon the visual area of the
occipital lobe, etc. It is clear that irritation of a large portion of the cortex
will excite ocular movements. And this is what we might expect, knowing
that noises, a touch, a flash of light, will make us turn our eyes to the ex-
pected point. It is to be noted that all cor-
tical stimulation gives rise to conjugate or
associate movements of the eyes, i.e., to
binocular visual direction, never to isolated
movements of one eye. For the levator
palpebral a special area is claimed at the
lower part of the parietal convolution, near
the upper facial and the hypoglossal, and
that it controls the opposite eye. But we
cannot set apart any definite area of the
cortex as dominating eye movements as we
can for the hand or thumb, etc. Knies
(1. c.) very ably discusses all the known
facts and concludes that we must assign to
the visual area the directing power for all
intentional or voluntary movements of the
eyes, while to the nuclei come fibres from
almost all parts of the cortex, exciting
purely reflex movements. Fig. 75 (Starr)
gives the latest arrangement in condensed
form.
Sub-cortical lesions are, of course, possible, and we have somewhat more
definite knowledge about them. In Forster's diagram (Fig. 76), giving a
horizontal section just dorsad of the third ventricle, we have a spot cephalad
the knee of the internal capsule marked eye, at which lesions will affect eye
movements. Again OP marks the place where the optic radiation comes in
from the occipital lobe ; here is the spot where hemianopsia may be produced.
Coming farther down the tract, in the peduncle we come upon a spot where
a lesion will cause hemiplegia of the opposite side, and paralysis of the oculo-
motor nerve of the side of the lesion. This point is one to be specially noted.
If the oculo-motor paralysis does not include the pupil and the ciliary
muscle, the lesion will be in the peduncle and rather small. If these nuclei
are included, the lesion will be at the base, or so large as to affect the third
ventricle, where the pupillary and ciliary nuclei are found One case is given
where right hemiplegia, paralysis of face and tongue, and total paralysis of
Fig. 76.
'" Cerebral Localization," Trans. Congress Am. Physicians and Surgeons,
vol. i., p. 219, 1888.
2Archiv f. Augenheilkunde, xxiii., 1, 22, 1891.
DISEASES OF THE EYE.
the left oculo-motor occurred from a small lesion at the very end of the pe-
duncle (Mauthner1).
Diagnosis.—By what has been set forth in anatomy and
physiology much has been done to aid in making- a clinical diag-
nosis, and we may now consider the question from this stand-
point. Orbital paralysis will be fairly assumed in the presence
of tumors, injuries, abscesses, or periostitis in the orbit. The
same is true of tenonitis and hemorrhage. The eyeball may,
of course, be prominent, and pain in pushing it back is an
important sign. Should the inferior oblique escape while all
other muscles are paralyzed, this is explained by its anatomi-
cal origin. On the other hand, paralysis of the inferior oblique
with paralysis of the sphincter pupillEe and of accommodation
is explained by a lesion of the motor root of the ophthalmic
ganglion (radix brevis), which supplies the parts enumerated.
Congenital absence of muscles has been reported; while most con-
genital paralyses are nuclear. A peripheral or rheumatic paraly-
sis is difficult to prove, yet we cannot always make a better diag-
nosis. We have already spoken of lesions at the sphenoidal fissure
which may impair all the nerves here passing and may even in-
volve both eyes. Symmetrical paralyzing lesions in both orbits are
recorded. Graefe, 1864, reported schirrus in each orbit, Scott2
grummata in each orbit of a child five years old. How the optic
nerve may escape when the nerves at the sphenoidal fissure are
perhaps all implicated is explained by the separation between their
respective foramina, the former above the clinoid process of the
sphenoidal wing, the latter lying under it and covered by the edge
of the tentorial portion of the dura mater, as it surrounds the
crura.
Basal Paralysis.—To make such a diagnosis probable, we must
have all the branches of the third implicated, because integrity of
the pupil and accommodation transfers the lesion to the nuclear
variety. Again, the loss of the sense of smell is in favor of a basal
lesion, although not with certainty, because that too may be
nuclear. Optic neuritis has about the same relative value; its
origin may be both basal and cerebral. Recurrent paralysis,
especially of all the third nerve, is almost certainly basal. It hap-
pens most frequently among young subjects. The periods may be
within a few weeks, or months, or at longer intervals. It took
place five times in a girl five years of age who was under my care.
The duration of each attack may be for a few days or for weeks.
■"Die ursachlichen Momente der Augenmuskel-L&hmungen," Wies-
baden, 1886, p. 393.
2 Archiv fur Augenheilkunde, vii., 94, 1878.
BINOCULAR VISION AND ITS DISTURBANCES. 159
In most cases there is headache (migraine). The two cases where
an autopsy was made disclosed in one an exudation and in the
other tubercular deposit about the stem of the oculo-motor. Both
sides may be involved, and Graefe reported five cases of paralysis
of all the eye muscles of both sides with complete recovery; he be-
lieved them to be basal and from cold, but about this localization
there may be a doubt. An enumeration of the causes of basal
paralysis will throw some light on its diagnosis. Their enumera-
tion is borrowed from Mauthner,1 whose treatise on this topic is the
most complete in existence and summarizes what is known at the
present time. They are, (1) hemorrhages; (2) circumscribed pachy-
meningitis about the nerve trunks; (3) meningitis, both simple and
tubercular, and this often in children, sometimes getting well and
at other times ending fatally; it may also be of traumatic origin,
and the prognosis in all cases is serious; (4) abscesses, especially of
otitic origin; (5) aneurisms; (6) arteritis obliterans, which impairs
the nutrition of the nerves, and is often syphilitic; (7) neoplasms,
including specific growths compressing the nerves, and sometimes
filling their foramina; (8) idiopathic lesions of the trunks, viz., a,
interstitial neuritis and perineuritis of tubercular or gummatous
character; b, lymphomatous swellings in cases of lymphomatous
disease, and c, gray degeneration consequent upon nuclear disease.
We must study collateral symptoms carefully to form a prob-
able judgment, such as variations in temperature and other signs
of inflammation—the history personal and hereditary—not only
the presence, but the absence of other cerebral disturbances. For
example, tumor in the brain may cause nerve trunk lesions as well
as if at the base, either by stoppage of the ventricles and internal
hydrocephalus, or by pressure reaching to the base, e.g., paralj'sis
of right sixth reported by Nothnagel from tumor in left hemi-
sphere. But crossed paralysis of the third with hemiplegia, or with
hemianopsia, brings us to the crus, and if the other oculo-motor is
subsequently paralyzed, we may be sure that the lesion is in the
vicinity of the crura as the third nerves enter the pons.2 Anaes-
thesia or irritation of the fifth is not a distinctive sign, it may be
basal or pontine. But anassthesia may come from a lesion higher
up, viz., in the internal capsule at its caudal part.
It is impossible writhin suitable limits to elaborate this topic; we
can only call attention to the tokens from the olfactory nerve, the
optic nerve, the auditory, the facial nerve, and to careful analysis,
not only of what muscles are at fault, but of all other symptoms,
1 "Die ursachlichen Momente der Augenmuskel-Lahmungen," 1886.
8 See analysis of one hundred and fifty cases of brain syphilis with eye
lesions, and autopsies, by W. Uhthoff—Graefe, Archivf.Oph., B. xxxix., Abth.
1, S. 1-182.
160 DISEASES OF THE EYE.
whether general or cerebral, which may be used as clues. The
rapidity of onset, spasms, the degree and location of pain, will all
be considered. Neoplasms usually cause slowly progressive symp-
toms. Yet this is not always true, because time may be required to
develop disturbance, and periods of repose and aggravation may
occur. Hemorrhages, embolism, and softening have their own
features.
Nuclear paralysis has certain characteristics. It is sometimes
sudden, is usually slow in attack, and affects successive muscles;
they may for a time act well and give out as the examination pro-
ceeds; the affection is often incomplete; is apt to be worse at night
or when fatigued; it may be unilateral or bilateral; usually there
is no pain, yet pain may be severe. A very significant symptom
is somnolence, and a most important sign is non-recognition of
double images. Affection of the extrinsic muscles is called oph thal-
moplegia externa; of the intrinsic muscles, the pupil and accom-
modation being paralyzed, is ophthalmoplegia interna. The dila-
tation of the pupil may be moderate. If one muscle after another
fall a prey to paralysis we may consider the cause basal by certain
correlative symptoms, but it may also be nuclear, and if, for ex-
ample, the muscles animated by the oculo-motor on one side are
involved at the same time with the trochlearis of the other eye,
we know this is nuclear. There may be double ptosis of nuclear
origin; single ptosis may be cortical, fascicular, or nuclear. See
Gowers.1 Pfliiger2 reports a case of double trochlearis paralysis
with partial paralysis of both oculo-motors, due to influenza or grip.
Among oculo-motor nerves, the right inferior oblique, myosis of
both sides—abated pupillary action both to light and convergence,
paresis of accommodation both sides—these lesions must be nuclear.
Paralysis of convergence without impairment of associated move-
ment to the right and left is reported by Straub.3 I have re-
cently had one such case. Seggel4 reports mydriasis of one
e3'e, there being no response to light, but contraction upon con-
vergence and accommodation, and no impairment of sight. De-
fect of levatores palpebrarum and of both superior recti of con-
genital type must be referred to the nuclei (see Gowers, 1. c).
Graefe5 reported a case of a man with paralysis of the six external
muscles of each eye, with good accommodation and normal pupils;
this, of course, was nuclear. Paralysis of either the third, sixth, or
seventh with crossed hemiplegia signifies hemorrhage into the pons.
1 "Diseases of the Nervous System," Am. ed., p. 620, 1888.
2 Graefe's Archives, xxxvi., 4, 71, 1891.
3 Archiv fur Augenheilkunde, xxiii., 3 and 4, S. 271. I have seen one case.
4Archiv fur Augenheilkunde, xxiv., 3, S. 234.
5 Archiv fur Ophth., ii., 2, 299.
BINOCULAR VISION AND ITS DISTURBANCES. 161
If the lesions be in the upper half of the pons facial palsy may be
on the same side with body palsy. The facial and sixth are some-
times affected together on the side opposite to the limbs. The face
and limbs palsied on the same side and the third on the opposite side
points to the crus. There may be a lesion so small that the nerves
escape.1
Lesion of the abducens nucleus with or without hemiplegia
damages not only the corresponding rectus externus but also the
associated rectus internus of the other eye, causing conjugate de-
viation. This subject will be dealt with more at length when
speaking of cortical lesions, which also cause this symptom.
Gowers relates paralysis of elevators of both eyes by a tumor
on the middle line of the fourth ventricle just behind the velum
(see Fig. 73 from Perlia showing the vicinity of the superior recti
and inferior oblique nuclei).
A case seen by Dr. Starr2 and later by myself is instructive.
The symptoms were temporary conjugate deviation to the left side,
analgesia of left face and of right half of body; ataxia of right
limbs without paralysis; he staggered in walking, had to be very
cautious in going down steps; contraction of left pupil, paralysis
of left trochlearis, anaesthesia of left cornea and beginning damage
to its epithelium; urine sp. gr. 1040 and contained sugar. These
signs placed the lesion in the left side of the tegmentum where the
superior peduncle of the cerebellum comes in and near the trophic
centre of the fifth nerve, very dorsad and extremely small (see Figs.
69 and 71). It touches the trochlearis nerve after its crossing and
for a time stimulated the left abducens, thereby exciting, through
its connection with the opposite rectus internus, conjugate devia-
tion to the left. The irrit ation of the pupillary nucleus had per-
sisted on the left side. The diagrams show the various possibilities
of sensory disturbances due to the peculiar and interesting course
taken by the various sensory tracts. Small hemorrhages and very
limited lesions are possible in this region because the arteries are
small, they supply very small territories, and do not inosculate with
each other. They are technically " terminal" vessels. A fact of
importance is, that the nuclei for the ciliary muscle and for the
iris are supplied by a special arterial branch which has no rela-
tion with the region in which the other nuclei are found. In this
fact lies an explanation of some cases of isolated paralysis of these
groups.
Mauthner collects a series of instructive cases (1. c. pp. 311-3-28).
See also Gowers' Lectures, 1887, Manual, 1888.
1 For illustration of such lesions with autopsies, see Uhthoff—Graefe, Arch.,
I.e.
2 Reported in full bv Dr. Starr in X. Y. Medical Record, Feb. 11th, 1893.
11
1(>2
DISEASES OF THE EYE.
The complications may be loss of smell, which is rare; loss of
sight due to optic atrophy and often syphilitic; both the motor and
the sensory portion of the trigeminus may suffer, most frequently
the latter; facial or hypoglossal palsy; bulbar paralysis, progres-
sive muscular atrophy, deafness may co-exist. The sufferings of
the poet Heine belonged to nuclear palsy. Graefe called attention
to anaesthesia of the face, while on the other hand extreme neural-
gia may co-exist, and insanity sometimes occurs. Locomotor ataxy,
hemiplegia, hemiataxia, hemianesthesia, and general paresis may
take place.
Galezowski and Duchenne have seen cases of bilateral paralysis
of the third and of the sixth in spinal disease. Lesion of the sixth is
the most common. Unilateral lesions are common. In spinal cases
the paralysis is likely to be incomplete and not to be permanent.
No other sign of spinal-cord disease may occur for a long time, and
the eye symptoms, while unsupported by others, will remain of
doubtful significance. The motor nerves of the eye often become
implicated at a late stage of spinal disease, and then the lesion is
not transitory, but permanent. Implication of the optic nerve may
be either an early or late symptom. In some autopsies (Leube), the
trunks of the motores oculorum and of the sixth have been trans-
formed into gray, thick, and hard cords.
The Argyll -Robertson symptom "of early ataxy, that the pupils
contract feebly to light and quickly to convergence and accommoda-
tion, is explained by the most recent author1 as due, not to a nuclear
lesion of the pupillary centre, but of the communicating fibres be-
tween the optic tract and the pupillary nucleus. He quotes four
cases of this symptom confined to one eye, and says that the lesion
which then causes reflex pupillary immobility on one side, with re-
tention of the consensual reflex, merely blocks the passage of the
direct fibres from the optic tract to the oculo-motor nucleus on the
same side ; while that which produces both direct and consensual
immobility of the pupil on one side (assuming that there is only
one lesion) occupies a position which blocks both the direct and the
crossed system of fibres, i.e., in the sphere of the centre of the
sphincter iridis. A lesion in the same region is usually extensive
enough to implicate the above-mentioned fibres on both sides of
the median line, i.e., the direct and the crossing, and hence the
Argyll-Robertson pupil commonly affects both eyes.
Fontan (see Mauthner) describes nicotine paralysis in which the
extrinsic muscles are incapable of function and the pupil and cili-
ary muscle irritated (myosis and spasm of A).
1 Turner: " On the Diagnostic Value of the Loss of the Pupillary Light Re-
action, with a Note on the Oculo-Facial Muscular Group."—Ophth. Hosp.
Reports, vol. xiii., partiii., 328, Dec, 1892.
BINOCULAR VISION AND ITS DISTURBANCES. 163
Near the seventh cervical vertebra is the cilio-spinal centre
which controls the vessels of the iris by the sympathetic nerve
fibres. When they become paretic the iris vessels lose tone, dilate,
and myosis ensues—the corresponding half of the face blushes and
perspires.
Orbital paralysis arises from inflammation of the connective
tissue or from periostitis, from tumors, from wounds and injuries.
Pain, tenderness on pressure or on percussion, are important signs.
One must push the finger deeply under the rim of the orbit all
around. The case will often have to be observed for some time
before a conclusion can be reached. Finally, rheumatic or per-
ipheral paralysis may occur, as from sitting in a draught, and the
sixth nerve is oftenest concerned.
Drs. Collins and L. Wilde (Am. Journal Med. Scieiices, Nov.,
1891) have examined and tabulated 120 cases of ophthalmoplegia
from which much valuable information can be gained as to their
clinical histories and events. A useful summary of the possible
causes and varieties of these cases is stated in their classification,
which closely resembles that of Mauthner, v. s.
Concluding now this imperfect sketch of causes and etiology, it
remains to be said that fully one-half of all cases of ocular paralysis
are due to syphilis and especially to the late forms. A study of
Uhthoff's paper, 1. c, shows the special lesions to include gummy
tumors and infiltrations; thickening of the meninges or periosteum;
exostoses; degeneration of vessels, with consequent softening of
brain substance, and also atrophy of the nerve trunks or nuclei;
thrombosis, partial or complete; hemorrhages. The lesions may be
single or multiple. At least two cases are given in which no lesion
was found at the autopsy; which is perhaps not surprising unless
the microscope was used, and the same writer, Howard, reports
two cases complicated by exophthalmic goitre, which recovered.1
In addition the following etiological conditions may be found:
cerebral hemorrhages, circumscribed pachymeningitis, meningitis
on the convexity of the hemispheres, both ordinary and tubercular,
abscesses at the base frequently arising from aural disease, en-
largement of arteries, aneurisms, arteritis obliterans, tumors in-
cluding gummata, idiopathic diseases of the trunks of the nerves
at the base, such as neuritis, gummy degeneration, tubercular de-
generation, lymphomata, gray degeneration. We have also pa-
ralysis from diphtheria, from grip, from diabetes mellitus, from
nicotine, from lead, and from injuries. Schoeler, out of 70 cases
observed a long time, could determine the cause in only 64^.
The chronic cases, which are the more frequent, arise from epen-
1 " Bilateral Ophthalmoplegia,1' etc., Am. Journal Med. Sciences, p. 238,
March, 1889.
164
DISEASES OF THE EYE.
dymitis with secondary affection of the gray matter of the ventricle-
(polio-encephalitis superior) multiple sclerosis, and atrophy of the
nuclei. Ocular paralysis in spinal cord disease may disappear and
return. Pell1 reports the third nerve to have been affected seven
times.
Acute cases arise from inflammation of the gray matter of the
third and fourth ventricles (polio-encephalitis superior of Wernicke)
or from hemorrhage. As would be expected, diabetes not infre-
quently exists. In acute cases, Wernicke has seen acute double
optic neuritis. In chronic cases, the optic nerves may be atrophied
or impaired or intact. Acute cases may be suddenly fatal, while a
case of Etters, 1882, involving a series of nerves from the second
to the eighth with profound paralyses, recovered almost entirely
after seven weeks. Finally, this lesion is sometimes congenital,
although some congenital defects in the eye muscles are due, not to
paralysis, but to absence of muscles or their incorrect insertion or
imperfect development.
Fuchs2 describes cases of isolated double ptosis which he at-
tributes to atrophy of the levatores muscles—i.e., a peripheral
lesion; but he evidently has not weighed the reasons for assigning
the nuclei of these muscles to the leading cephalic position which
Knies has given them, else he would have been more likely to re-
gard the pathology as nuclear in at least some of the cases.
If disease involve the anterior quadrigeminal3 bodies there is
amaurosis; in some cases the ophthalmoscope reveals absolutely
nothing. In tumors we may have choked disc or atrophy of the
optic nerve, etc. Generally the pupil does not react either way.
Naturally we would look for lesion of the trochlearis muscles and
probably of neighboring nuclei. Hemiataxia is very prone to occur,
and simulates hemiplegia, but will be easily discriminated by testing
the muscular force. It arises from interference with the superior
cerebellar peduncles and with the crossing fibres of the pons and
possibly with the vestibular nucleus of the auditory nerve.
We have yet to consider cortical and sub-cortical paralysis of
ocular muscles. (See Fig. 75.) We have stated that this form of
paralysis is always conjugate—i.e., associated movements of the
eyes are disturbed, and of course both eyes are affected. We have
remarked, without dwelling on the point, that such lesions also take
origin in the pons and can then be quite accurately localized. But
we are far less able to determine the place affected when a lesion is
cortical. The experimenters are not in agreement as to the cortical
area. Horsley and Schaefer select a space in the frontal lobe, and
1 Berlin, klin. Wochenschrift, 1890, 1.
2 Graefe, Archiv f. Oph., xxxvi., 1, 234.
3 See Goldzieher, Centralblatt f. Augenheilk., Feb., 1893.
BINOCULAR VISION AND ITS DISTURBANCES. 165
Ferrier chooses a somewhat less extensive area in the same region,
in front of the head area, which gives rise when irritated to opening
of the eyes, dilation of the pupils, and turning of the head to the
opposite side, with conjugate deviation to the same side. In this
view Mills concurs, 1888. Stimulation of the region just in front of
and including the angular gyrus causes conjugate deviation to the
opposite side. Wernicke thinks a lesion of the lower parietal
region of the opposite side is denoted by conjugate deviation when
it remains permanent. Temporary deviation occurs from stimula-
tion almost anywhere in the cortex. It is a frequent phenomenon
at the onset of cerebral apoplexj'. The rule is formulated as first
given by Prevost, that irritation of one hemisphere causes the eyes
to turn to the opposite side, an active effect—while paralysis of one
hemisphere causes deviation to the same side, a passive result,
because the opposite hemisphere excels in power. Usually the
deviation lasts only a few hours or perhaps weeks. It often pre-
vails during the comatose period and disappears as consciousness
returns. A few cases of permanent deviation are recorded from
lesion of the thalamus, but we cannot establish any connection be-
tween the symptom and any definite cortical or subcortical region.
It is very interesting that experiments on animals by Munk and
Schaefer with weak induction currents have shown that irritation
of the anterior part of the visual area produces movement of both
eyes downward, and of the posterior part of the visual area move-
ment upward. If the macula region of the visual area be stimu-
lated no movement occurs, because it then seems as if the irritation
proceeded from the point of fixation of the visual lines, and no
change of direction ensues (Obrigia1). The farther from the macula
region of the cortex (cuneus) is the part stimulated the more de-
cided the movement of the visual axes and to the opposite side.
Conjugate deviation from pontine lesion presents some varia-
tions of type and may be either acute or chronic, temporary or per-
manent. It is not admissible to enter at length into its discussion.
There is a large literature on the subject, and a very full compila-
tion is given at the close of an excellent article on eye paralysis by
the late Dr. J. A. Jeffries.2 The thesis of Prevost, 1868, and a
treatise by Hunnius, 1881, are valuable contributions, while Ross
and Gowers treat of it at some length in their works on nervous
diseases. See also Swanzy, Trans. Oph. Soc. United Kingdom, 1889,
p. 6. Drs. Collins and L. Wilde8 have collected and classified 120
cases of ophthalmoplegia, and their arrangement of them corre-
1 Archiv f. Anat. und Phys., 1890, S. 260.
2 Boston Med. and Surg. Journal, Oct. 20th and 27th, 1892.
"Am. Journal Med. Sciences, Nov., 1891.
166 DISEASES OF THE EYE.
sponds closely to that of Mauthner. Dufour1 has compiled a large
number without closely analyzing them.
Prognosis must always be guarded. Recovery is frequent, but
the possibility of progressive disease in the brain and spinal cord is
not to be overlooked. There may be a long period of exemption
from other trouble, but in the end mental disease, cerebral tumor,
progressive paralysis, or locomotor ataxy may develop. If syphilis
can be made out, treatment is more likely to be effective, yet not
always. In the 120 cases collected by Collins and Wilde (1. c.) the
recoveries were 24$; marked improvement 16$; death 24$. A
diagnosis of the seat of the lesion, as well as its quality, is the most
important element in prognosis, and justifies the elaborate discus-
sion entered into on this point.
Treatment.—We must necessarily take into account the proba-
ble cause and localization of the disease, and when this is doubtful
we fall back on general principles of therapeutics. A patient in the
early stage of his trouble, who has double images, will close one
eye, or wear over it a screen. It is well for short periods to put
the screen over the sound eye, to keep the muscles of the other in
practice. Soon after the lesion there may be headache or symp-
toms which suggest leeches or cupping, but not often is depletion
proper. Blisters by cantharidal collodion, of small extent, over the
temples or forehead, are useful as peripheral stimuiants. Iodide
of potassium would be given in small doses in non-syphilitic cases,
and in large doses in syphilitic cases according to the stage and
peculiarity of the constitutional disease. Electricity may be applied
by the faradic current or by the interrupted galvanic current, the
former preferabty—one pole upon the temple or behind the ear,
and the other by a small sponge upon the globe. Its efficacy is, to
say the least, very doubtful. Neurologists have recently been
placing reliance on hydrotherapy in nervous affections, and this
will have its application to patients in whom there may be ocular
complications.2 It needs skilful management by well-contrived
appliances and methods.
We also use strychnia in moderate doses after a few weeks have
elapsed. We chiefly rely on spontaneous absorption of the mis-
chievous exudation or hemorrhage or thickening. In case of tumors
or of organic cerebral or spinal-cord disease, our attention is neces-
sarily chiefly given to them. Michel has proposed mechanical ex-
ercise of the affected muscle by pulling the e3^e forcibly to the side
toward which it cannot turn, by fixation forceps, having first in-
stilled a solution of cocaine. This may be done once daily or once
in twro days. Bull has found good results from the proceeding.
1 Annales d'Oculist.
2 " Hydrotherapy in the Treatment of Nervous and Mental Diseases," by
Frederick Peterson. Am. Journal Med. Sciences, Feb., 1893.
BINOCULAR VISION AND ITS DISTURBANCES. 167'
When, however, no improvement takes place after the lapse of
two or three months, we have little right to expect it; but in most
cases, a degree of amendment or entire cure will have occurred.
For stationary conditions when double images are not too wide
apart, we may employ prisms. It is sometimes possible to wear
prisms as high as 8° to 10°, but beyond this they become too clumsy
to be ordinarily tolerated. In adopting them, this rule is to be
remembered: Put the base of the prism toward the image which
is to be influenced. The total number of degrees required may be
divided between the two e\~es.
The eye which deviates the most, or which is weaker in power
or in vision, will wear the stronger prism, in case, as may happen, a
difference is to be made. Frequently the muscles undergo changes,
and require corresponding alterations of the prisms. The perma-
nent use of prisms is in fact a rarity, and pertains more especially
to cases of vertical diplopia. Double vision beginning 10° or less
above, and for all the field below the horizontal meridian, or which
concerns the median region of fixation, is the most distressing, and
calls loudly for aid. The utility of prisms is usually confined to these
regions, viz., on the median line and for parts on or below the horizon.
In fact, to extend their influence over the whole field is impossible,
because the relations of the double images become entirely different
in its various parts, and it is impracticable to adapt prisms to these
changes. Prisms, like crutches, may be greatly acceptable; but
they are imperfect substitutes for sound muscles.
When, however, a case has existed for months, and is beyond
the utility of prisms, and does not improve, an operation will often
serve an admirable purpose. Operative proceedings are twofold:
1st, simple tenotomy of one or more muscles; 2d, advancement of
the impaired muscles.
For such a case, for example, as imperfect paralysis of the sixth,
or sometimes when it is wholly paralyzed, a tenotomy of one or
both interni may be indicated and give a useful result. Both must
generally be divided, because in the opposite eye the internus has
undergone secondary contraction by co-ordinated function, and the
internus of the impaired eye, by being unopposed, has passed into
a similar condition. The greatest stress in such an operation is to
be laid on the internus of the sound eye, because undue freedom in
loosening the internus of the injured eye will tend to exophthalmus,
to sinking of the caruncle, and to render the globe incapable of
sufficient movement in any direction. On the injured eye, if any
such tendency appear, a suture must at once be deeply entered, and
drawn tight to prevent undue slipping back of the tendon. The
effect on the muscles is to be measured by using a lighted candle, a
red glass and prisms, and single vision must, if possible, be secured
to a point far within the functional range of the paralyzed muscle.
168
DISEASES OF THE EYE.
The ultimate effect will be less than the immediate. If any power
remains to the damaged muscle, it gains increase of function by
being less seriously overmastered. In fact, this principle has been
applied to the advantage of a paralyzed muscle, to prevent both
the degeneration of its own tissue and extreme secondary contrac-
tion in the co-ordinated muscle, by performing tenotomy on a
secondarily contracted muscle within a brief time, say two or three
weeks after the onset of the paralysis. I have seen Dr. E. G. Lor-
ing perform such an operation, and he declared himself satisfied
with its effect. I have had no such experience, and do not know
that such practice is pursued by any one else. The degree to which
the muscle is loosened is very carefully measured and restrained,
because the tenotomy is intended to have a preventive effect, and
also to aid in the recovery of function.
For cases of marked and permanent limitation of motion, the
proper proceeding is combined advancement of the paralyzed
muscle and setting back of one or more of its opponents.
For a correct understanding of this proceeding some remarks
on the anatomy of the oculo-orbital fascia are proper. This tissue
is also known as the capsule of Tenon. If the upper and lower lids
be divided in the middle down to the fornix and the flaps be forcibly
drawn back, it will be seen, by lifting the conjunctiva on a probe or
a strabismus-hook, that there is a distinct layer of connective tissue
going forward under it to the margin of the cornea. It is also
noted that the ends of the muscles, as they reach the globe, pro-
trude through it and are clearly displayed. Pressure with the con-
vexity of the hook between the eyeball and the margin of the orbit
demonstrates that something shuts off the parts behind, and forms
a layer which adheres on the one side to the globe, and on the other
to the margin of the orbit. The structure which is thus demon-
strated is the oculo-orbital fascia. If the globe be enucleated, the
tendons and the stump of the optic nerve will be seen to stick out
through a layer of smooth fibrous membrane, which forms the cup
in which the globe rotates, and which is part of the same fascia.
The same structure enters into the eyelids and enwraps all the
muscles as they advance toward the globe. It thus appears clear
that a tendon maj^ be entirely loosened from the globe, and if its
lateral and immediate relations with the fascia are not torn up, it
still remains in connection with the eye, and can exert an active,
although reduced influence upon its movements. If, however, in
detaching the tendon, cuts be freely made in the lateral regions,
the muscle will lose its control over the globe, because it slips back
into the orbit; and if any connection remains, it will be through
the medium of some band of tissue which has escaped disruption.
Motais has also shown that, after tenotomy, the reattachment of
the muscle is more by the medium of the conjunctiva and capsule
NOYES ON THE EYE.
PLATE
/
k ljSc/— cf» i ccp c/
FIG./
FIG. 2
EXPLANATION OF PLATE No. II.
Fig. 1.—Arrangement of capsule of Tenon in man. Vertical section pass-
ing through the superior and inferior recti.
CE, CE, CE (red), aponeurosis or external capsule forming sheath of mus-
cles. CEP, CEP, deep layer of the sheath of the muscles folded backward
upon itself to cover the posterior hemisphere of the globe. CEM, superficial
layer of the sheath of the superior rectus muscle folded to form the sheath of
the levator palpebr*. LT, LT, terminal layers of the aponeurosis going to
the orbit, and tarsal cartilages. FS, FS, subconjunctival fascia.
CI, CI (blue), bulbar or internal capsule, serous membrane of the eye.
CI', internal capsule folded beneath the deep surface of the tendon and mus-
cle which it covers to the point I, where the external capsule leaves the muscle.
CI, internal capsule in front of the tendon and anterior extremity of the mus-
cle surrounding a serous bursa indicated by a dotted blue line. It stops
behind just where the external capsule leaves that muscle to go to the or-
bit ; anteriorly it is inserted into the sclerotic with the tendon and does not
reach the border of the cornea as does the subconjunctival fascia FS.
ADI, aponeurotic offshoot of the inferior rectus muscle DI, split to envelop
the inferior oblique muscle 01. DS, Superior rectus muscle. R, levator pal-
pebral. LS, superior tarso-orbital ligament. LI, inferior tarso-orbital liga-
>nent. TS, superior tarsus. TI, inferior tarsus. CON, CON, subconjunctival
bpace.
Fig. 2.—Arrangement of capsule of Tenon in man. Horizontal section pass-
ing through the internal and external recti.
CE, CE, CE, CE (red), aponeurosis or external capsule forming sheath of
muscles, CEP, CEP, deep layer of the sheath of the muscles folded back-
ward upon itself to cover the posterior hemisphere. ADE, external ligamen-
tous offshoot. ADIN, internal ligamentous offshoot. FS, IS, subconjunc-
tival fascia.
CI (blue), internal capsule. CI', internal capsule folded beneath the ten-
don and muscle to the point I. CI", CI", internal capsule in front of the ten-
don and muscle which it covers up to the point I, where the offshoot leaves
it; surrounding a serous bursa BS, BS, indicated by a blue dotted hne; it
then terminates anteriorly at the tendinous insertion I'.
DF, external rectus muscle. DI, internal rectus muscle.
BINOCULAR VISION AND ITS DISTURBANCES. 171
of Tenon than by the tendon, and that both in retro-placement
and advancement of the muscle the most important factor is the
fascia and especially its lateral prolongations.
The oculo-orbital fascia does not admit of a clear demonstration
as a membrane; it is too complex in its ramifications, and too deli-
cate in structure, besides being perforated by a multitude of organs.
It ensheaths to a greater or less degree all the organs, muscles,
vessels, nerves, etc., which pass through it. For example, the ex-
ternal sheath of the optic nerve is continuous with it, and it also
adheres to the margin of the optic foramen. The periosteum of the
orbit is continuous with it, and is sometimes spoken of as its parie-
tal portion. But the analogy of the pleura in its visceral and pul-
monary parts cannot be strictly maintained, although it is sug-
gested. For practical purposes we are to bear in mind three facts:
1st, that the fascia serves as a cup, like the acetabulum, in which
the globe revolves and makes enucleation possible without opening
the deep parts of the orbit; 2d, that it prevents effusions in the
orbit from easily finding their way into the lid, and beneath the
ocular conjunctiva; 3d, that it constitutes a secondary attachment
for the ocular muscles, renders their combined action more perfect,
and makes it possible to sever their tendinous insertions without
annulling their influence over the globe. A further remark is that
the caruncle and semilunar fold are intimately connected with the
fascia; and so is the tendon of the muscle of Horner, at the inner
canthus, while at the outer canthus the external lateral ligament
may be called a process thrown out from the periosteum. Gerlach
further calls attention to the check which certain fibres exert over
the action of the muscles, and at the inner side of the orbit the
figure which he gives shows how firm is the connection between the
fascia and the bony wall. Motais more clearly demonstrates and
emphasizes these restraining bands, especially at the inner and
outer margins of the orbit (see Plate II.). It is always somewhat
difficult to lift the caruncle in a dissection, and if this is done at an
operation it is liable to retract and cause an unpleasant appear-
ance. This has a practical bearing on the operation for converg-
ing squint.
Advancement of the ocular muscles is called for in three differ-
ent groups of cases—viz., when the duration of the optic axes is
due, 1st, to an injury or an operation; 2d, to spontaneous dissocia-
tion from loss of sight in one or both eyes; 3d, to paralysis. In the
first two the muscles retain a degree of contractility, in the last
group there ma}T be none at all.
Up to the present time I have made trial of many modes of
operating, viz., those proposed by Critchett, by Agnew, by Weber,
by Wecker, by Prince; and have modified them as circumstances de-
manded, I have finally come to a method which has nothing but
172
DISEASES OF THE EYE.
its simplicity to commend it and with which I am entirely contented.
It is attended by very little reaction, which is far from being true
of some other methods; it is exact and is capable of being grad-
uated to a desired effect, whether this be large or small. The neces-
sary instruments are a wire speculum, two pairs of strong fixation
forceps, a good pair of scissors (Stevens pattern is the best), two
strabismus hooks, a speculum, and three sutures of fine black strong
silk with a curved needle at each end, and a needle-holder. The
important thing is to have the needles fine, "half-curved," about
| inch long, and ground so sharp that they can as easily penetrate
the cornea as a discission needle. On the fineness, sharpness, tem-
per, and curve of the needles, success chiefly depends. Suppose the
right rectus internus is to be advanced. The right rectus externus
is first divided; then seize the insertion of the rectus internus with
fixation forceps, taking a deep bite to include all that can be lifted;
sever the insertion freely and cut above and below into the con-
junctiva to the extent of 10 to 15 mm.; leave the forceps fast to the
tissues by shutting the spring catch, lay it aside, and then remove
a vertical oval of conjunctiva in front of the insertion, leaving a
strip 6 mm. wide next the cornea (this step may be deferred to a
later period, in some cases depending on the laxity of the conjunc-
tiva). Lift the muscle and pass a curved needle from within out-
ward at its middle and as far back as the proposed effect will
demand. With the needle in place cut off superfluous material
lying in front of it; then draw it through. Insert another
needle in a similar way, and before drawing it through cut away
superfluous material. Do the same with the third needle and
cut off needless material as before. The object of cutting off re-
dundant substance in successive parts as the needles are inserted
is, to be certain not to sever the sutures. We now have three
threads through muscle and fascia and conjunctiva. The needles
at the other end of the threads are next to be passed forward
beneath the remaining conjunctival strip, taking hold of the
outer layer of the sclera so that the points emerge at the lim-
bus corneae; the middle one must first be fixed in situ. To get
them through without breaking, they must be seized at the middle
and pushed without any lever action. If the globe tends to rotate
it may be steadied by a bident. I have seen the points sometimes
appear in the anterior chamber, but this is of course too deep.
What is essential is to have firm hold on the sclera. In tightening
the threads begin with the middle, and care must be used not to
pull too hard and to act first on one and then on another, deferring
the second knot until each has been pulled up as far as it will bear
without breaking. If there is much crumpling of tissue it must be
cut away and the parts be left smooth—perhaps some additional
and superficial stitches will be needed. I know of nothing more
BINOCULAR VISION AND ITS DISTURBANCES. 173
simple than this proceeding. Its effectiveness depends on the firm
hold obtained by piercing the sclera close to the cornea, and to this
end the needles must be adapted. The stitches will remain from four
to seven days. When the utmost effect is desired the externus of the
fellow eye must be divided. A bandage is applied for twenty-four
or forty-eight hours, and afterward cold-water compresses em-
ployed. It is rare to have chemosis or any important reaction.
The degree of effect must be in excess of what will be ultimately
required, because the eye will slip around some 5 or 10 degrees.
In case we have to do with an eye which has had a too liberal
tenotomy as the cause of the deviation—the dissection of the
muscle is more difficult, but the method is the same—I find no
need of the hitching thread of Dr. Agnew, nor of the clamp forceps
of Wecker, nor of the anchoring thread of Prince. The ordinary
fixation forceps, aided sometimes by a second one, each having a
spring catch, gives perfect control of the muscle. The operation
on the rectus externus is much easier of execution than on the-
rectus internus. Strabismus hooks are used in exploration and as
accessories, not as essential implements.
This mode of operating is especially, but not exclusively, suited
to strabismus paralyticus, and hence is now described. Still other
proceedings will be referred to, adapted to ordinary kinds of stra-
bismus. Advancement does not confer contractility upon a
totally paralyzed muscle, but simply gives the globe a more pleas-
ing position. It may give rise to annoying diplopia, but this is
usually temporary.
Congenital paralysis of ocular muscles sometimes demands
relief, and its most frequent form involves the levator palpebrse
superioris and the rectus superior together, causing ptosis and
dropping of the eyes. I have seen excessive contraction of the in-
ferior rectus drawing the axis of one eye far below the position
of its fellow, while in both the above-mentioned paralysis existed.
Usually both eyes are symmetrically affected. I have also seen
congenital paralysis with contraction drawing one eye downward
and outward, while the other was normal. Some cases are doubt-
less nuclear, while it has been shown that in some the muscles are
not properly developed. Surgical relief is in some cases possible,
in other cases quite unsatisfactory. If, as I have seen, the superior
recti and the levatores palpebrarum are wholly incapable of action,
only slight improvement and that of little value may be possible.
If some power remains to the muscles there will be more encourage-
ment.
SPASMS OF OCULAR MUSCLES, NYSTAGMUS.
An oscillatory movement of both eyes, quick and jerky, greater
at some times and in some positions than in others, is the charac-
teristic of this disease. In very rare cases one eye alone is affected..
174
DISEASES OF THE EYE.
Such a case was reported by Dr. St. John to the New York Oph-
thalmological Society, in December, 1882. The condition is usually
congenital. It is almost always associated with amblyopia, while it
of necessity much impairs the available acuity of sight. We often
find it with congenital cataract, both partial and total, also after
ophthalmia neonatorum with central opacity of the corneas; it is
almost invariable in albinoes, and we see it in cases of extreme
hyperopia, and sometimes with congenital choroiditis at the macula.
Frequently there is convergent strabismus. The movement may
be lateral, vertical, or rotatory, or all combined. I saw, by ^he
kindness of Dr. H. W. Williams, of Boston, a man who had ac-
quired the power of voluntary nystagmus after having been for
some eye trouble confined for several weeks in a dark room. A
form of n3Tstagmus, lately noticed, affects individuals among high
mountains, and especially those who work in mines. It comes in
adult life, is most noticeable toward night, is periodic or parox-
ysmal, is induced by looking in certain directions, and apt to be
attended with vertigo. Nystagmus among the English miners has
been described by Oglesby, and is attributed to their unhealthy
surroundings and the awkward posture of the head and the
straining of their eyes upward as they work. By refraining from
work some seem to get well, only to relapse on returning to
the mines. Commonly the patients are not aware of the oscilla-
tions, except by the effect upon sight. The movement ceases
during sleep. Rarely there is movement of the upper lid syn-
chronous with the eye: this happens usually with vertical nystag-
mus. Some persons, despite this trouble, have highly useful vision.
They are apt to be myopic, and distant vision is below the standard,
but near work may be prosecuted with great success. In New
York I have known two notable cases—one a distinguished musical
composer and teacher, and the other a well-known practical
chemist. Both of them were albinoes.
This condition is sometimes dependent on brain-lesions of recent
occurrence. For example, it has been seen to follow blows on the
head, also apoplexies, but with no definite localization, and in soft-
ening, as well as in hemorrhagic pachymeningitis. In some chronic
brain diseases it has been noted, and the matter has been summed
up by Robin (" Des Troubles Oculaires dans les Malad. de FEnce-
phale," 1880). "Nystagmus, unilateral or double, permanent or
temporary, exhibiting itself with other convulsive or with paralytic
symptoms, indicates an encephalic lesion. In general this will be
at the base or on the convexity behind the fissure of Sylvius (region
of the angular gyrus). In the former case, it will often be com-
plicated with paralysis of the motor nerves of the eye or of the
optic; in the latter case (when on the convexity), there will be
BINOCULAR VISION AND ITS DISTURBANCES. 175
epileptic attacks, hemiplegia, etc., but we cannot venture on any
exact localization." Irritation of the peduncles has caused this
symptom in experiments by Schiff. It occurs among the insane and
the neurotic. It is very frequent in disseminated sclerosis of the
brain and cord. With locomotor ataxy it is very rare. A not in-
frequent picture in a case of brain disease of the kind now noted, is
the concurrence of rotation of the head, conjugate deviation of the
eyes, and nystagmus; these phenomena evidently point to irritation
of the region of the third and fourth ventricles. Nystagmus may
be seen in cases of aphasia and of labio-glosso-larjmgeal paralysis.
It thus becomes evident that, while most cases exhibit a complex
causation, consisting both of defective sight and of irregular inner-
vation of the muscles, other cases depend alone upon lesion of inner-
vation of central origin. As to the former class of cases, it can-
not be doubted that the irregular movements are, in very many,
due simply to the want of motive for correct binocular fixation, i.e.,
to lack of predominance of the macula lutea.
Treatment of these cases is of little service. For some the cor-
rection of optical errors, so far as it can be accomplished under
the difficulties of the examination, is valuable. For those with
strabismus con vergens, tenotomy of one or both intern i is advisable.
I have done tenotomy of the interni when no strabismus existed,
but because the lateral movements were excessive, and found ben-
efit ensue. The degree of tremor was abated; but, as a rule, an
operation is not fitting. Albinotic patients wear dark glasses, and
preferably those with side-pieces to cut off the glare of light; and
all nystagmic patients hold fine objects close, and have some choice
position of the head in which their trouble is less annoying. Ex-
citement greatly aggravates the tremor, and it usually remains
unaltered through life. For an exhaustive study of nystagmus, see
an article by Raehlmann: Arch, fur Oph., XXIV., 4, pp. 237-317.
His conclusions tend to locate the cause of the disease in the brain,
but at what region is undetermined. Another elaborate article is
by R. P. Oglesby: Brain, vol. ii., July, 1880. (See Gowers, "Dis-
eases of the Nervous System," 1888.)
A paper by Alfred Graefe (Bericht Siebenter Internat. Ophthal-
mologen Congress, 1888, Wiesbaden, p. 30) discusses cases of palsy
of lateral movements with unimpaired convergence ; that is, only
the associated movements of interni and externi are abolished.
I have seen two cases of the affection of a chronic type ; one was for four
and a half months under observation. It seemed to point to a growth in the
vicinity of. the left abducens nucleus.
1882 May 6th Henry R, aged 17, Brooklyn, ship carpenter; came to
New York Eve and Ear Infirmary. Father living and healthy, mother died
of cancer eighteen months ago. General health good, denies and has no signs
of svphilis ; never had much headache. Seven weeks ago, had diplopia on
176
DISEASES OF THE EYE.
median line and more noticeably on left side. Was treated by iodide of po-
tassium and electricity. Now cannot bring either eye to median line, each
turns to the right and stops about 10° to right of it: the head turned a little
to right. On the left side has homonymous images, which implies that left
externus is more faulty than right internus. It is found that right internus
turns farther toward median line with effort of accommodation. On June
16th did tenotomy of right externus; this improved position to slight degree.
On June 26th tenotomy of left internus, but eyes could not be moved beyond
median line. Meanwhile electricity and full doses of iodide of potassium were
kept up. Vision from the beginning normal in O.D. and § % in O.S. The
temporal halves of each nerve look suspiciously white. July 7th, upper lid of
left eye does not readily shut (suggests indication of facial nucleus which is
near nucleus of sixth nerve) and sleep became bad; no headache. Begins to
be light-headed; walks well with closed lids. On July 14th was sick at stom-
ach after drinking ice water; after a week again felt perfectly well. Sept.
15th has almost complete facial palsy of left side; movement of left eye up and
down is free, but lateral movement outward extremely limited. Right eye
moves well, up and down, but cannot reach median line, and excursion to its
temporal side is limited. Head trembles, gait frequently unsteady; some-
times is dizzy; no other symptom. Not seen after this date. Diagnosis is
tumor in left side of pons, involving nucleus of sixth and later of seventh
nerves. Patient was seen by Dr. Allan McLane Hamilton, who concurred in
this opinion.
I have seen a child 2i years old with somewhat similar conditions. In the
beginning, after fever and vomiting, the eyes were turned upward for two
days and then settled down to the lateral dextral deviation and were parallel,
although capable of convergence. She had enlarged lymphatic glands in the
neck and face. I saw her for only a short time and know nothing of the ulti-
mate issue.
I witnessed the autopsy of a man 40 years old who had this symptom and
who was under care of Dr. Janeway. He entered Bellevue Hospital uncon-
scious ; both eyes and the head were turned to the right. He was hump-
backed. At the autopsy the lesions found were abundant small tubercular
deposits over the whole surface of the brain and more numerous at its base;
acute meningitis of orbital surfaces of both frontal lobes and each in equal
degree. Tubercles numerous on under surface of cerebellum and along spinal
cord; the bodies of the vertebrae absorbed. Brain congested and oedematous,
not soft, no apoplexies, nothing in ventricles nor in pons or in fourth ventri-
cle. All the lesions were on the surface and mostly basal and anterior. Dur-
ing life no choked discs. Such a case resembles more the epileptoid cases
than those with distinct focal lesions.
CHAPTER IX.
STRABISMUS CONCOMITANS.
This term denotes a condition in which either eye can fix upon
an object in all parts of the field, but binocular fixation fails. The
defect is not in the motility of each eye singly, but in the lack of
co-ordinating power with its fellow. Associated movements are
performed, but the deviation between the visual lines is always
maintained, although its degree may vary according to the dis-
tance and position of the object. Sometimes binocular fixation is
possible for extreme distance, but is lost when the object ap-
proaches. In strabismus paralyticus the essence of the lesion is
loss of power, while in strabismus concomitans the muscular ac-
tivity is perverted, not materially diminished. Again double vision
is exceptional in strabismus, and is the rule in paralysis for certain
parts of the field.
Strabismus is either permanent or occasional; it sometimes is
truly intermittent. It increases as the object approaches, that is,
with efforts of accommodation, and, as said, it may only then ap-
pear. This is always characteristic of converging strabismus,
while with diverging strabismus the rule does not always hold good.
According to the direction of the deviation we have S. conver-
gens, S. divergens, S. sursum-vergens (upward), S. deorsum-vergens
(downward). With converging squint the eye often turns up as
well as inwTard. The affection is usually bilateral (concomitant),
sometimes it is confined to one e3^e and is monolateral. Sometimes
with bilateral squint the person will fix indifferently with either
eye; more frequentry one is preferred to the other, and it may even
be impossible to employ the eye which habitually deviates, for more
than a few minutes. Of course with monolateral squint the devi-
ating eye never voluntarily fixes and often cannot fix accurately
and centrally.
To decide between monolateral and alternating squint, a screen
or the hand is placed obliquely over one eye, and while the other
looks at the finger held near it, we note the behavior of the cov-
ered eye; by trying each eye in succession we discover whether
one or both is distorted. Frequently the angle of deviation is
greater in one than in the other. It must be admitted that in
ordinary squint, especially converging, there will be some limita-
tion in mobilit}', but this is far less considerable than in paralysis.
Patients are often distressingly conscious of paralysis, usually they
give little attention to subjective symptoms in strabismus.
12
178
DISEASES OF THE EYE.
Measurement of the degree of strabismus has been made by in-
struments which give the amount of turning in millimeters or lines
along the border of the lower lid. This is of course very inexact,
and the only correct method is in terms of angles. To do this with
precision one may, as Landolt suggests, use the perimeter. Place
the squinting eye in front of the centre of the arc, let the other fix
an object at ten or more feet distance nearly on the line of the
centre of the arc—then carrj^ a small flame along the arc until it
shall be reflected from the summit of the deviating cornea and
read off the angle on the perimeter. If the eye deviates so far in-
ward as to be behind the nose, a prism with angle inward may be
interposed and half of its angle added to the number of degrees
given by the perimeter.
Another and simpler way given by Hirschberg is that the ob-
server sit facing the patient and hold a lighted candle about one
foot in front of him, screening his own eye from the light. From
each eye is seen the reflex of the flame on the cornea. The eye at
the centre of whose pupil the reflex appears is the one which fixes
—on the other cornea the reflex is eccentric. Its place may vary
as the patient gazes at the candle or afar off. The point at which
the reflex appears will give a measure of the angle of the squint,
and five degrees may be distinguished if the pupil be supposed to be
3..5 mm. in diameter. If the reflex be only a little way removed
from the centre, i.e., about half wray to the pupillary edge, the devi-
ation will be less than 10° (varying with the angle alpha as will
be explained). If at the pupillary edge, the angle will be 12° to 15°.
If at a point about midway between the pupillary edge and limbus,
the deviation will be about 25°. If at the edge of the cornea, the
quantity will be 45° to 50°. If outside the cornea, the reflex will be
blurred or multiple and the angle may reach 60° to 80°.
With normal fixation of both eyes, there may seem to be diverg-
ence, if the corneal axes lie to the outer side of the visual lines—
that is, if the angle gamma is large (see page 15) and positive. On
the other hand, if the corneal axis lie to the inner side of the visual
line, which occurs in high degrees of myopia, there will be an ap-
pearance of convergence. In these cases the angle (gamma) is un-
usually large and in the first instance is positive, in the second is
negative.
(The angle in question is really formed between the axis of the
cornea and the line of fixation, which passes through the centre
of rotation of the globe and is not identical with the visual line.
This is the angle gamma. The angle formed between the axis of
the cornea and the visual line is the angle alpha. Donders speaks
of the latter and when his treatise was written the above distinc-
tion was not made.—Woinow.)
STRABISMUS CONCOMITANS.
179
To determine the error in terms of angles both Hirchberg and
Landolt have constructed diagrams to be hung on the wall, in
which vertical and horizontal lines are drawn at the distances
corresponding to the tangents of angles from 5° to 60°.
Priestley Smith achieves the same result by the ophthalmoscopic mirror
and a tape measure. The light is put above the patient's head. He holds
one end of a tape line one metre long against the cheek below the sound eye.
The observer sits in front and the tape is held tense by a ring through which
is slipped the handle of the ophthalmoscope. The good eye fixes on the mir-
ror and the observer notes the position of the corneal reflex, which should
be near the centre of the pupil. The light is shifted to the other eye and the
position of the reflex on its cornea noted. The patient is directed to follow
the finger of the observer's free hand as it passes outward until the corneal
reflex on the squinting eye occupies the same position in the pupil that it did
on the fixing eye when it looked at the mirror. The distance between mirror
and finger is the tangent of the angle of deviation, and is denoted by figures
on another tape of which one end is fast to the ring on the handle of the
ophthalmoscope and which slips through the fingers of the moving hand.
A tolerably precise mode of detecting deviations of the ocular axes, or
lack of fixation by one eye, is furnished by the ophthalmoscopic mirror.
Priestley Smith draws attention to it (Ophthalmic Review, Feb., 1892),
but most experienced observers have probably employed it. The patient
looks at the mirror or at the observer's forehead; the place of the corneal
reflex within the illuminated pupil is noted ; without any change of the posi-
tion of the observer or of the patient, the mirror is rotated to the other eye,
and the place occupied by the reflex within that pupillary area is noted;
the light is quickly flashed from one eye to the other, and a comparison
of the relative situations of the corneal reflex will soon determine whether
both eyes are fixing on the same point. Normally the reflex is a little nearer
the inner edge of the pupil than the outer edge, because the line of fixa-
tion does not coincide with the axis of the cornea {vide supra). A markedly
non-symmetrical position of the two reflexes denotes failure to fix on the
same point. If the discrepancy remains always the same and affects each
eye alternately we may conclude that while both eyes do not fix on the
same point each has the power of fixation, but fusion is absent. If how-
ever one eye fixes correctly when the patient is told to look at the light,
and the other persistently deviates in spite of the urgency to fix the light,
we conclude that there is extremely defective sight in the wandering eye
(probably a central scotoma) and that binocular correct fixation will be
impossible, even by operation and other means.
Etiology.—It has already been remarked that we are to look
for the effective cause of functional strabismus, in errors of sight
and not primarily in lesion of the muscles. The errors are those
both of refraction and of perception. That the muscles themselves
have a part to perform in causation must also be recognized. Their
influence was formerly exaggerated, it has until lately been unduly
depreciated.
That hypermetropia is found in three-fourths of the cases of
strabismus con vergens, was one of the brilliant facts made known by
Donders. He set forth what is now so well understood, that in-
creased convergence makes augmentation of accommodation more
easy. If in a case of hypermetropia the renunciation of binocular
180
DISEASES OF THE EYE.
sight were a lesser ill, than the strain on the accommodation with
binocular sight, then converging squint would follow. This would
take place when the two eyes differed greatly in refraction, if one
were highly astigmatic, or if in one there were opacity of the
cornea or lens, or if in one there were great amblyopia. Again
Donders took into the category of causes decided weakness of the
recti externi. Such are some of the conditions which caused him
to say that hypermetropia is the dominant and sufficient cause of
strabismus convergens. On the other hand he also showed that
strabismus divergens is in the large proportion of cases associated
with myopia. The cause here lies in the inability to maintain the
needful convergence for a very near point, and such is largely the
true explanation.
That hypermetropia does stand related to strabismus conver-
gens in a most intimate way is indubitable. That the connection is
so simple as Donders' theory makes it, is not now accepted. The
difficulty lies in the fact that a large number of hypermetropes,
even when their eyes are unequally erroneous, do not squint. The
number of those who do not squint is far higher than of those who
do. Still more must we study the question with care when it is re-
membered that converging strabismus belongs chiefly to the early
age of life, and that at this time it is almost the absolute rule to have
hypermetropia. It is also true that hyperopics learn to accommo-
date with small effort of convergence. Why in some of them con-
vergence should so far go ahead of accommodation, remains to be
explained. It is admitted that the strabismic cases are not found
chiefly among the strongly hyperopic, but among those who have
medium and slight degrees. Donders assumed that the greatly
hyperopic patients see so badly that they give up the effort to im-
prove their vision. But we do find them converging very strongly
in trying to see as well as possible.
Defects of perception have been mentioned. Monocular ambly-
opia is very common in strabismus convergens and not infrequent in
S. divergens. This may or may not be associated with high degrees
of hyperopia or with astigmatism, possibly irregular. We meet
with it where the degree of ametropia differs little from that of the
eye with good vision, and in a very large proportion, perhaps in the
majority of cases, no lesion can be found with the ophthalmoscope.
On this point it is important to bestow careful attention. ,No small
number of cases exhibit what are evidently congenital abnormities
in the papilla. In my records are such conditions as follows: An
extraordinary amount of pigment deposit along the border; the
presence of connective tissue on edge of nerve and running along
the vessels (not to be confounded with opaque nerve fibres); a dull
or slaty-colored and opaque disc with hazy edges; extreme hyper-
aemia both of capillaries and veins; the nerve swollen as in papillitis
STRABISMUS CONCOMITANS. , 181
a dark gray or slaty spot upon the disc and the rest of the surface
an opaque white; coloboma of the sheath of the nerve or a very
deep and irregular excavation which was so interpreted. Besides,
one must carefully scrutinize the macula and it must be done with
dilated pupil. Not rarely will one find minute specks, white, yel-
low or glistening, clustered here, which indicate lesion either of the
choroid or retina. There may be one or more marked pigment
specks which will denote a previous inflammatory lesion. A nota-
ble number of cases, and the majority, will not reveal any visible
lesion. In the examination of the visual field we are often pre-
vented irom attaining exact knowledge by the extreme youth of
the subjects. When, however, they are sufficiently intelligent, we
frequently find that the amblyopia is central and a defined scotoma
for red may be sometimes mapped out, provided a small card 5
mm. square and dim light be employed. The scotoma may be very
small and will be better discovered on a dark plane surface than by
the perimeter. It may often be detected by the fact that out of a
row of letters at 20', say tlie line O H SU E or 0.3, the S pointed
out by the physician's finger is less clearly seen than the adjacent
letters on either side. Sometimes a patient will say that over a
small space, not the blind spot of Mariotte, a small candle flame is
not perceived. This means a small absolute scotoma. In one case
I found nasal (medial) amblyopia with the line of demarcation ver-
tical. It was not difficult to show the decided difference in percep-
tive power of the respective halves of the retina—this might be
called hemiamblyopia. It was of course natural for this patient
to have converging strabismus because then the better half of the
retina was put to use.
The amblyopia of strabismus has been attributed to non-em-
ployment of the squinting eye, and amblyopia ex anopsia is often
set down as a sufficient statement of the facts. The exclusive
use of one eye does not in monocular cataract nor in extreme
monocular astigmatism bring about amblyopia. Neither could dis-
use occasion a central or localized scotoma, neither could a clearly
defined hemiamblyopia which corresponded exactly to the fasciculus
non-cruciatus of the optic tract, be caused merely by disuse. In
truth this assumption rests upon no evidence. On the other hand,
congenital amblyopia is not at all rare; witness its frequency
in congenital cataract. Neither is congenital monocular ambly-
opia rare. On this point Schweigger'sl statistics are eminently
forcible, page 91. He collected ninety-eight cases of congenital
amblyopia who did not squint. There were all possible refractive
errors in every possible combination; in some, one eye was normal,
and the other amblyopic. Out of them 47$ were hypermetropic
—yet none of these patients squinted. On the other hand (pp.
1 " Klinische Untersuchungen tiber das Schielen." Berlin. 1881.
182 m DISEASES OF THE EYE.
99, 100 1. c), out of 247 cases of strabismus both converging and
a few of diverging, which were at the same time hyperopic, he sets
apart as amblyopic, those whose vision is less than \ and they are
87, that is, 35.2$ of the whole. He also divides these cases of hy-
peropic squint into two classes, viz.: those in whom H is less than
fj = 177, and those in whom it is above ^, i.e., 3 D = 70. In these two
classes the chosen degree of amblyopia, viz., v = \ and less, exists
in the first in 31.6$ and in the second in 44.2$. This increase in
the ratio of amblyopia with the increase of the degree of hyperopia
points unerringly to the dependence of amblyopia upon congenital
defect and not upon disuse.
My own attention has been carefully given to this question and
I have on record a very large number of hyperopics who have
monocular amblyopia and have never had squint. They at the
same time have adequate muscles and binocular vision. The same
is true of astigmatics, and my conviction is settled, that amblyopia
is, with very few exceptions, precedent to squint and is not its effect.1
In a limited sense disuse operates unfavorably upon visual
acuity. The power of fixation is much impaired, there is imperfect
control of the accommodation (I have the record of one unmistak-
able case of severe monocular spasm of accommodation in converg-
ing squint) and the retina is easily fatigued. Perhaps the temporary
scotomata and limitations which Wilbrand describes in asthenopia
(1. c.) also occur. These conditions are readily admitted and they
account for some of the feebleness and for the variability in the
visual acuity. Beyond this degree, amblyopia ex anopsia has no
proof to rest upon and it cannot be accepted as an explanation.
With this opinion, which Schweigger first forcibly enunciated, agree
Alfred Graefe, Ulrich, Segger, Landolt, Wadsworth and others.
It therefore follows that amblyopia congenita is entitled to a place
alongside of hypermetropia in the production of converging squint.
But while the proportion of H in squint is about 75$ excluding those
less than 2 D, the remaining 25$ must be accounted for by other
causes. And we have seen that the great number of hyperopics do
not squint, hence still another factor besides refractive error and
amblyopia must be found. This evidently lies in the condition of
the muscles. To this point Schell,2 Ulrich3 and Segger4 have given
attention. Schell studied the ratio of abduction to adduction in a
1 See Wadsworth, " The Amblyopia of Squint," Boston Medical and Sur-
gical Journal, Jan. 20, 1887.
2" Cause and Prevention of Squint." Amer. Journ. Med. Sci., Oct., 1-878
p. 418.
8"Zur Aetiologie des Strabismus convergens." Klin. Monatsbiatter,
xviii., 156, 1880. " Die Aetiologie des Strabismus convergens hypermetropi
cus," Kassel, 1881.
4 " Statistischer und Casuistischer Beitrag zur Aetiologie des Strab. con
verg." Klin. Monatsblatter, xviii., 439, 1880.
STRABISMUS CONCOMITANS.
18:1
small number of cases of emmetropia and hypermetropia. For 20
cases of E he found abduction was to adduction in the ratio of 28
to 100; while in 16 cases of H he found the ratio to be 48 to 100.
The hyperopic cases did not squint. And the reason which Schell
assigned was the relatively high capacity for abduction which they
possessed. The contrary condition he assumed would favor the
production of squint. Ulrich and Segger agree in the same view.
Ulrich (1. c, p. 26) puts the ratio between abduction for 10 inches
(M. 0.25) and abduction for 6 M. (parallelism) in E at 1: 5.7, in hy-
peropia at 1 :.3 That is in hyperopia, abduction in the relations in
which he compares it, is nearly twice as strong as in emmetropia,
among those who do not squint. The results of Schell and Ulrich
correspond with sufficient accuracy. With these three factors,
viz., hyperopia, amblyopia and inadequate abduction, we have the
combination which suffices to explain the larger number of cases
of strabismus convergens. We may also include among them the
cases of monocular ametropia, or opacity of the cornea and monoc-
ular cataract. Even when the vision of each eye is good we meet
with converging squint, and in these cases we may assume that
the abduction has been abnormally weak. Such for instance is the
explanation of many cases in whom we are told that the error came
after scarlet fever, measles, or diphtheria, etc., or after an attack of
acute inflammation of the eyes with blepharospasm. The assump-
tion that it was produced by imitation of a squinting person, or by
looking at a bright light, or a hanging lock of hair, etc., is of doubt-
ful value, but need not be rejected as absolutely worthless provided
other conditions concurred.
Something must be said as to the phenomena of vision in persons
who squint. That they do not complain of double images is not
surprising when there is decided monolateral amblyopia—neither
is it surprising after the full establishment of the deviation, even
when both eyes have tolerably good vision, say better than |. But
it is not true that in converging squint they make no use of the de-
viating eye in conjunction with the other. Schweigger showed that
they can always perceive the light of a candle if reflected into it by
a small mirror placed beside the nose; a slip of plane glass will
suffice. While ordinary binocular vision is of course impossible,
there is by help of the faulty eye enlargement of the field, and in
some cases it is proven that a real co-ordination between the two
eyes is established by which the macula of the sound eye is coupled
with some other spot of the retina of the deviating eye, which takes
on the usual functions of the macula. In these cases prisms with
the angle vertical, placed over the squinting eye cause diplopia, by
the stereoscope the two figures are combined, and if by tenotomy the
deviation be corrected, there may be crossed diplopia notwithstand-
184
DISEASES OF THE EYE.
ing the ocular axes are in correct position. These are very puzzling
facts in the physiology of vision. On the other hand if vision of
the two eyes be nearly equal, the beginning of strabismus is at-
tended with diplopia or there is a conflict in the impression of the
two eyes which leads to the mental suppression of one image. This
act of mental exclusion is familiar to microscopists and watch-
makers, and it can be more or less perfectly realized in strabismus.
This is the only explanation which can be offered of certain cases
and it is not inconsistent with remarks before made, to assume that
the mental impression of one eye becomes habitually weakened and
in this sense vision may be said to be impaired by disuse. Some
strabismic patients have double images when they give attention
to both eyes. In most, it is very difficult to excite them. An
oblique posture of the head is common with converging squint and
it may persist after an operation. Some persons who use each eye
alternately will employ the right for objects on the left side, and
the left eye for objects on the right side, and change the obliquity
of the head correspondingly. In doing this they evidently favor
the weak externi. A distinct tremor when the eye turns in the
direction of the weak muscles is not uncommon, it may be a real
nystagmus and it may be monolateral. It is not infrequent to find
symptoms of asthenopia, and these may be of a pronounced type,
in some cases attended by photophobia. Fatigue in eye work is
very common. Many interesting questions arise in connection with
the vision of persons having strabismus, and for their discussion the
reader is referred to the authorities quoted by Alfred Graefe in
Graefe and Saemisch," Handbuch," B. VI., p. 242, and to authorities
quoted by Landolt, article Strabismus, " Dictionnaire encyclope-
dique des sciences medicales," Dr. Dechambre, which is brought
down to 1882. Von Graefe, Donders, Javal and Alfred Graefe have
especially interested themselves in this matter.
Course of Strabismus.—Beginning usually at the time when
steadfast application begins, it at first is occasional, and months, or
years, generally elapse before it becomes permanent. It may show
itself in rare cases at birth or under one year of age, generally it
appears at two to six years. That it can occur in later life has
been remarked. It may spontaneously disappear and this not ex-
clusively in cases whose degree is small. Binocular vision is not
established and critical examination may discover that there has
been only a great abatement of the fault, not its complete disap-
pearance. Usually the deviation lasts many years, and the rule is
that when once established it permanently remains. We have no
information upon the anatomical condition of the muscles in per-
manent strabismus and it is desirable to fill this void. That the
contracted and the enfeebled muscles undergo organic changes of
STRABISMUS CONCOMITANS.
185
tissue is extremely likely. It is often found in operating that the
muscles seem rigid as well as hypertrophied—but inasmuch as we
come in relation only with the tendon we usually discover nothing
of the status of the muscle itself.
The remarks hitherto made apply chiefly to strabismus conver-
gens. Schweigger gives the following statistics as to the refraction.
He classifies the cases according to the refraction of the fixing eye,
and mentions, but does not specify, anisometropia among them.
Permanent Periodic
Strab. conv. Strab. conv.
. 85 13
Emmetropia,.
Myopia,
Hyperopia,
Emmetropia,
Myopia,
Hyperopia, .
. 44 10
. 196 98
325 121
Permanent
Strab. divergens.
. 37
. 59
4
446
Periodic
Strab. divergens.
28
50
5
100 83
183
629
Out of Horner's clinic Isler1 collected 359 cases of strabismus.
Strabismus convergens 236;
Emmetropia, . 4
Myopia, . . 11
Antimetropia, . 13
Hyperopia, . . 208
Strabismus divergens 133
Emmetropia, . . 3
Myopia, ... 62
Antimetropia, . 30
Hyperopia, . . 38
236
133
In Isler's statistics the ratio of hyperopia is higher than in
Schweigger's, because he not only takes in H less than 2 D which
Schweigger excludes, but he comprises latent as well as manifest H.
The discrepancy in regard to the ratio of E is partly explained by
the fact that Schweigger's classification is based only on the fixing
eye. He finds a higher ratio of myopia in converging squint than
does Isler, viz., 8.6$, as against 4.6$. It is, however, very noteworthy
that myopia takes the first rank in the causation of diverging stra-
bismus. Schweigger gives 59.5$ and Isler, when the cases of anisome-
tropia of which one eye was myopic (1. c, p. 28) are added to those
of myopia, viz., 22 + 62 = 84, gives 63.1$ to myopia. In the latter's
1 " Studien fiber die Abhangigkeit des Strabismus von Refraction."
Inaug. Dissert., Zurich, 1880, "Walter Isler.
186 DISEASES OF THE EYE.
statistics an unusually high proportion of cases of hyperopia is
found among diverging strabismus, viz., 28.6$. These are mostly
of moderate,degree, each eye usually had good vision, and the cause
was found in weakness of the interni. In some cases this ensued
after the occurrence of debilitating diseases, in other cases the
weakness was inherent and original.
Another class of cases is those in which one eye is so nearly
blind that it wanders outward simply in obedience to the tendency
impressed upon it by the outward direction of the axis of the orbit.
Here, too, there may be latent weakness of the interni. Divergent,
begins at a later period of life than convergent squint—when the
latter is connected with myopia it is usually of high degree. It may
be alternating, but usually the patient gives the preference to one
eye. Very often there is marked irregularity in vision or in refrac-
tion. The tendency of myopia to divergence is caused by the high
degree of convergence required for near vision and by the absence
of impulse to accommodation to aid it—a situation which is the exact
reverse of what obtains in converging strabismus with hyperme-
tropia. In both classes of cases there must coexist a lack of power
in either the externi or the interni to render the deformity possible,
while refractive conditions play a role which has been sufficiently
explained.
Treatment.—In the incipient stage of converging strabismus
we may sometimes prevent its establishment by employing the
glasses needful for correcting ametropia. Serious amblyopia will
be a hinderance to success and when the subjects are very young
there are obvious objections to their use from the danger of acci-
dents by breakage. But when in the house, young children can
wear spectacles and, when at active out-door play there is less ten-
dency to close convergence. It has been proposed to use eserine
sulphate to assist the accommodation (Ulrich) and take off strain
on the interni and to resort to this when glasses cannot be worn.
On the other hand, the entire suspension of accommodation by
atropia has been much recommended and employed from precisely
the opposite indication, viz., to relieve the interni by abolishing the
accommodation. A degree of benefit can be gained by this pro-
ceeding, but no permanent gain without using the needful correcting
glasses. The resort to atropine from time to time as the tendency
to squint becomes more pronounced, while the patient is habitually
wearing correcting glasses will not seldom prevent the establish-
ment of the error. It is decidedly worthy of trial because the
externi are strengthened and the habit of binocular vision is pro-
moted. When this is not feasible, and a patient shows a decided
tendency to prefer one eye in fixation—this may for some hours
daily be tied up (screening it by a shade will not serve the purpose)
STRABISMUS CONCOMITANS.
is;
in order to compel the use of the other. By so doing the muscles
of that eye are kept in better training, although the covered eye
continues to squint. The use of the poorer eye to improve its vision
is a pious hope rather than an assured expectation—notwithstand-
ing the contrary opinion largely prevails. Any other treatment
than tenotom}' is of little value after a permanent squint has been
produced.
In diverging strabismus, associated with myopia, the greatest
assistance is to be derived from proper concave glasses, to be em-
ployed both for near and distant work. They should be chosen
with reference to the degree of myopia and to the accommodation
of the subject and to the distance of the working point. Very
often they will be one-half or one-third less strong than the glasses
required for the far-point. The rules for this have already been
discussed. The use of glasses by young myopic persons includes
this very important advantage, that control over the recti interni
is greatly aided. This suggestion is also applicable to cases of
great dissimilarity in the degree of mj^opia of the two eyes even
when one of them may not admit of the glass which its refraction
indicates, and the correction is applied only to the better eye.
Atropine has less efficacy in these cases than in those previously
considered. In incipient divergence not only are glasses to correct
ametropia important, but they can often be usefully combined with
prisms. Whatever relieves the strain on the interni at the near-
point, is to be adopted. The subjects are usually old enough to
permit a satisfactory examination, which with convergent squint
is often impossible. In the large majority of cases of convergence
and in many of divergence we must resort to an operation.
The operative treatment has in view not only the correction of
deformity, but the improvement of the working ability of the e3Tes,
and if possible the procurement of binocular vision. We practise
tenotomy upon the contracted muscle to abate its power and we
advance the insertion of the weaker muscle to increase its power.
The former is the most frequently employed, while the latter is in
certain cases combined with it. A practical question arises in con-
verging squint, how early in life shall tenotomy be practised ? If
the deformity is not large it is better not to operate until five to
seven years of age. But if the squint be pronounced, a tenotomy
at an earlier age may be indicated to keep the muscles in better
balance, but its effect will not be complete and it should not be
done except as a palliative measure. At a latter period another
operation is likely to be needful. Should one eye be operated on or
both ? Graefe taught the importance of dividing the effect between
both eyes, but since his time some have advocated going back to the
practice of early times and confining the proceeding to the eye
188 DISEASES OF THE EYE.
which deviates most. As a matter of fact, in marked deviations
the muscles of both eyes are at fault and I have no hesitation in say-
ing that each eye should in such cases be operated on. For a devia-
tion of 3 to 5 mm., 5° to 15°, only one eye is to be operated on; for 5 to
6 mm., 15° to 30°, both will require it, but there should be two weeks
interval before the second operation; for squint of 10 mm., 30° to 45°,
both may be dealt with at the same sitting. It may be assumed that
one tenotomy will effect a change of about 3 mm., or about 15°. An
absolute result cannot be predicted because the elasticity of the
opponent is variable, and we find the ultimate position frequently
not the same as that which appeared after the first week. Inas-
much as the patients especially seek an improvement in their
personal appearance, the rule is to leave a slight degree of conver-
gence rather than to risk the slightest amount of divergence. If
there be fairly good vision in both eyes and if there be hyperopia
of 3 D or more, we may put considerable confidence in the tendency
of the eyes to a correct position. In such cases the effort is to
establish binocular vision, and if there be a divergence of 1 or 2 mm.
this will usually not be permanent. It is not, however, to be as-
sumed that a free dissection of the tendon is to be allowed. This
was the method at the first introduction of strabotoniy by Dieffen-
bach, and from the unwise practice of that day came a large crop
of cases who needed a subsequent and serious operation to correct
the divergence. Such cases are occasionally met with at the
present time.
The operation is as follows: For young subjects ether may have
to be given, for older ones instillation of 4$ solution of salicylate
or hydrochlorate of cocaine three times within twenty minutes
will suitably benumb the tissues. The local anaesthetic is much to
be preferred, because we need the aid of the patient to enable us to
decide how much has been accomplished. The lids are separated
by the spring speculum; for converging squint the eye is pushed
to the outer angle by a fixation forceps which seizes the conjunctiva
near the outer border of the cornea and if the forceps be rather
heavy it may be left to hang and its weight will keep the eye in
position. Employ forceps with a stiff spring whose teeth project
well forward, take up the conjunctiva and tendon at the latter's
insertion and snip it with slender scissors whose points are a little
rounded. The wound must be small and vertical and at the middle
of the tendon. If an opening has been made through the tendon
the slender scissors may be slipped astride the lower and upper
portions of the tendon in succession and accomplish most of the
cutting. The rest of the insertion may be caught up by the hooks
and divided. If the tendon has not been opened, the forceps will be
thrust through the conjunctival opening to seize it and be followed
by the scissors. The sharp projecting teeth are designed for this
STRABISMUS CONCOMITANS. 189
purpose (see Fig. 78). Snip through the tendon and passing through
the hole a small blunt hook catch the upper half of the insertion.
Keep the scissors flat on the sclera and cut at the insertion.
Keep hold of the tissues by the hook and insert a second hook
under the undivided lower half of the tendon and cut it. Then
examine above and below whether the whole insertion has been
separated from the sclera. The cutting is partly subconjunctival
and partly visible in the wound. A little oarb on the blunt point
of the hooks materially facilitates the search for undivided
fibres and gives greater hold to the hook (Theobald) (see *"">
Fig. 77). The conjunctival wound will unavoid-
ably be stretched by these manoeuvres, and
some blood will be effused beneath the tissues.
It is a disadvantage to have a large thrombus
because it increases the effect and renders the
operation less certain. In putting the hook
under the tendon carry the beak straight back
and keep the tip upon the sclera as it is rotated
either upward or downward. When it is well
engaged the handle is carried across the root of
the nose and the tendon pushed into view. Now
the tendon is cut between the hook and the
sclera and of course it is not shortened, but is
only detached.
There are other methods of operating. Some
make the incision of the conjunctiva below and
parallel to the edge of the muscle. Graefe ad-
vised making it close to the cornea. Arlt uses
scissors alone to divide the muscle without the
help of the hook. For myself the method de-
scribed secures the complete division of the ten-
don with the least disturbance of the structures and through
the smallest wounds. The conjunctival wound is always united
by a stitch of very fine black silk. The bleeding is to be wiped
away with absorbent cotton. Sponges are objectionable unless
very fine and soaked in corrosive sublimate solution 1: 2,000. It
is better to use salicylated absorbent cotton. Before putting in
the stitch the effect of the operation is to be ascertained. The
associated movements should be well performed, and if the de-
sired effect is apparently reached, the patient should be able to
converge easil.y to a point five inches distant. If sufficient
effect is not secured, incisions may be made above and below
the insertion into the capsule of Tenon. To dissect the con-
junctiva more freely away from the outer surface of the muscle
by undermining the caruncle will also increase the effect, but
at the expense of an unpleasant retraction of the caruncle.
190
DISEASES OF THE EYE.
Another mode of increasing the effect is to gather up a fold
of the conjunctiva on the other side of the globe in a large
suture (Knapp). Better than this, is to attach a suture to the con-
junctiva on the outer side of the globe near the cornea and carry
both ends through the skin beyond the outer canthus and tie them
over a bit of stick. Such a guy will put the globe into any desired
position. The thread must not be left more than five or six hours
and both eyes must be well bandaged to prevent pain. I have
seldom resorted to these sutures. To restrain the operative effect,
a stitch may be carried to a greater or less depth into the divided
muscle and brought out near the cornea horizontally, or in an oblique
direction. A change of 2 mm. is easily obtained. In rare cases when
I have sought to slightly increase the effect and the conjunctival
wound has been small I have altogether omitted the suture. To
do this, is to risk undesirable retraction of the caruncle and the
sprouting of granulations in the wound.
Sinking of the caruncle is liable to occur in slight degree even
after operations carefully done. It comes as a gradual result of con-
traction of the cicatricial connective tissue, and depends upon the de-
gree to which the oculo-orbital fascia has been invaded and loosened.
It is always proper to lay upon the usually squinting eye the
larger effect of the operation. If the deviation is chiefly monolat-
eral, one may combine tenotomy of the internus with advancement
of the externus of the same eye. If this do not suffice, tenotomy of
the internus of the other eye may be done at a later time. The
advisability of advancement may be estimated by the capacity of
the eye to turn toward the outer canthus. In marked monolateral
squint this will be feeble and the externus will have been so. much
stretched that it cannot pull the eye around even when the internus
is loosened. In one case of this kind where one eye had slight myopia
and the other an extreme degree, notwithstanding a good position
of the axes was obtained, it could only be held by wearing full cor-
recting glasses and practising daily with prisms for three months.
Wecker has recently advocated what he calls advancement of Ten-
on's capsule at the insertion of the antagonist, as an adjuvant to
tenotomy. The operation consists in bringing forward the inclos-
ing sheath and connective tissue as one would do in certain modes
of advancement of the tendon (see chapter on Asthenopia), but the
tendon is not loosened. A well and neatly conducted advancement
seems to me is to be preferred. As a matter of fact the chief ele-
ment in tenotomy as well as in advancement, is the altered tension
and attachments of the sheath and capsule—combined with loosen-
ing the insertion of the muscle.
The ultimate result of an operation is apt to be greater than
that which exists after two or three weeks.
STRABISMUS CONCOMITANS. 191
For ordinary cases very simple after treatment suffices, the
eyes are bandaged for twelve hours and then cold or warm water
applied as the patient prefers. The conjunctival stitch may be
taken out in two days or, if left to itself, it will drop out in a week
and it causes no appreciable irritation. Subconjunctival ecchymo-
sis will remain for ten days. If granulation spring up, it must be
cut off with scissors, not touched with caustic.
For diverging strabismus the mode of operating is essentially
the same, but the tendon of the externus is 7 to 8 mm. from
the cornea, while that of the internus is 6 mm. The effect of the
tenotomy is usually adequate in simple cases of myopia, but in em-
metropia and hyperopia it is less than ordina^. Advancement
of one or both interni may be advisable in extreme cases, or a tenot-
omy may be done the second time. For a young lady on whom I
had done two tenotomies of each external rectus without a perfect
cure, the result was obtained b}r causing her to practise daily with
adductive prisms to the utmost of her ability. She had good vision,
and was emmetropic in each eye. She gradually improved in power
of adduction and on one occasion felt something snap in one eye,
after which she acquired a sufficient power of convergence and has
always retained it. A suggestion of Dr. Gruening has proved of
great value in considerable degrees of divergence when no paralysis
exists. After dividing one or both externi, the two e3^es are coupled
together by a suture which is attached to the inner side of the globe
and carried across the nose. It takes its hold on the conjunctiva
vertically near the cornea, and when tied, both eyes are held im-
movable and in convergence. It is left in situ for twelve or twenty-
four hours. Care must be used to get no more effect than is re-
quired. An excess of 10° or 15° convergence at the beginning is
desirable, for this amount soon disappears. With extreme diver-
gence Dr. G. shortens the tendons of the externi.
Hardly less important than a correct operation is the subse-
quent management of the eyes. As soon as the reaction has dis-
appeared, say within four to six days, the propriety of using glasses
must be decided.
If there be much hyperopia and the externi are weak, not only
should glasses be worn, but possibly it will be needful to resort to
atropine. In other cases only the manifest H will need correction.
If there be tendency to over-effect, then no glasses are to be given.
Of course, astigmatism is to be corrected. Sometimes practice with
prisms and a candle flame, or the use of the stereoscope will aid in
confirming correct attitude of the eyes. Very many patients can
be made to see two images after an operation who found the great-
est difficulty in doing it before, and they can be taught to closely
approximate or to fuse them. Special test cards must be provided
for practice with the stereoscope, viz.: on one side a vertical and on
192
DISEASES OF THE EYE.
the other a horizontal line, or on one side a dot and on the other
dots in horizontal series which are to be numbered, or on one side
a capital L and on the other a capital F, which combined make E
(Green) or other similar devices by which it will be certain that each
eye sees and takes a correct position. With the usual stereoscopic
pictures there can be little assurance that proper vision is being
practised. Of course such exercises require intelligence, the subject
must not be too young and vision must have a certain acuity.
Javal employs an ingenious reflecting stereoscope which admits of
variation in the angle of reflection and consequently of conver-
gence.
Mechanism of the Operation and its Ultimate Results.—There
is a notable lack of accurate and well ascertained information on
this subject. With patients the object sought is cosmetic, and if a
passable result is obtained they are content and likewise too often
is the surgeon. It has been claimed that decided improvement of
vision is procured by the operation and aiso that binocular vision
is a result frequently secured; v. Graefe and Knapp claim 50$. A
good cosmetic result is rarely difficult of attainment, by combina-
tion of methods and by sometimes doing a series of operations.
In cases of decidedly monolateral converging squint with great
weakness of the externus and also in cases of divergent squint,
where one eye has turned aside simply because of extreme defect
of sight, both retirement of one tendon by tenotomy and advance-
ment of the opponent is required. Such is also apt to be the case
with divergence ensuing from marked debility of the interni in
cases of E and H notwithstanding vision of each eye may be good.
Here, as already said, simple tenotomy is apt not to suffice but must
be combined with advancement. With ordinary convergence cos-
metic success is secured by carefully avoiding unnecessary disturb-
ance of the surroundings of the tendon, whether lateral or between
it and the conjunctiva, and by not attempting to secure too much
by one operation, whether upon one or upon two muscles. It must
never be forgotten that the mechanism of the proceeding consists
in letting the tendon slip back and take a new attachment to the
globe behind its original insertion. For the degree of retirement
which may be safely permitted we must take into account the
length of the internus and the degree to which it is shortened. Its
length including the tendon is about 40 mm. With a squint of 5
mm. it is reduced to 35 mm. when in the squinting position. The
externus has a normal length of 49 mm. and this will be stretched
to 54 mm. The internus in the normal position of the eye lies in
contact with the globe for about 7 mm. This is of course shortened
in the squinting position, by the degree to which the eye turns in-
ward. Where the insertion is displaced backward, the muscle at
STRABISMUS CONCOMITANS.
193
its new hold still has a favorable position for action because this
point is now turned outward and will permit the muscle to exert
its force in a tangential direction. Evidently, however, if the in-
sertion be pushed back beyond a certain extent, the action of the
muscle becomes more and more embarrassed. Every tenotomy
signifies a limitation in the mobility of the globe—this may within
certain limits, in the case of antagonist muscles which have not
become seriously degenerated, amount simply to a displacement of
the arc of rotation. But in very many cases the arc of rotation is
shortened, because the externus is incapable of carrying the eye
outward to the degree by which the arc has been diminished on the
inner side. In this statement lies the reason for the rule, that it is
proper to let the muscles have time to adjust themselves to the
new relations, provided one operation fails to procure the desired
effect. The occurrence of relapses is also thus understood if it be
found that the externi prove to be too feeble to maintain the advan-
tage which tenotomy has given them. At the same time the abso-
lute unwisdom (to use a mild phrase) of striving by free dissection
to effect the purpose before a suitable adjustment of the muscular
forces has been secured, is obvious. No surgeon, however careful,
can claim absolutely immunity from the liability to slight over-ef-
fect in some cases, because it is impossible to control or acquaint
ourselves with all the conditions of the problem. Divergence,
amounting to 3 mm. or more when looking at a distance, is to be
deplored, and such a degree of deformity reflects discredit upon
the surgeon.
The value of the operation consists in improved appearance, in
more comfort in using eyes, in enlarged binocular field, in the inci-
dental correction by glasses of refractive errors, sometimes normal
binocular vision is gained. That it permanently improves visual
acuity is seldom true. An exceptionally good and permanent
restoration of sight is given by Johnson, Trans. Am. Oph. Soc.,
1893. In discussing this point we must choose patients whose age
and intelligence enable them to give us trustworthy information.
I have at hand the records of forty operations of which thirty-six
were for strabismus conv. and four for strabismus div. Twenty-
three patients who had convergence I regard as capable of giving
reliable information—their ages are as follows:
From 6 to 10 years = 6
« n " 15 " =5
" 16 " 20 " =5
" 21 " 42 " = 7—23
From these, eight cases are taken out who had V = f£ or §£ in each
eye both before and after the operation, some of them requiring
13
194
DISEASES OF THE EYE.
correcting glasses. In the remaining fifteen cases before the opera-
tion, vision in the poor eye was from -£& to yfo. In three cases the
ophthalmoscope showed defects which would necessarily prevent
visual improvement. In the thirteen which are left, five showed
absolutely no improvement in visual acuity. We are reduced then
to seven which are fairly proper for investigating and testing the
point. 1. A physician, aet. forty, had compound hyperopic astig-
matism in the poor eye; when corrected vision improved from $fo
to £■#, but the operation had no share in the benefit. 2. A young
lady, aet. sixteen, had astigmatism in both eyes; when corrected the
poor eye had V = f#. Six months after the operation, having been
subjected to the use of atropine and having used glasses, and hav-
ing also covered the good eye for certain periods so as to compel
employment of the other, the vision of the poor eye remained f-g,
and without a glass was T2^. 3. Young lady, ast. fourteen, before
the operation the poor eye with + 3 D counted figures at three feet,
after the operation counted figures at six feet. 4. Young lady, ast.
seventeen, the' poorer eye had with -f 1.5 D V = f{ and there was
hemiopic amblyopia, but this vision six months later was -^\. 5.
Male, aet. nine, in the poorer eye with + 6.s O — 0.75c 180° V = f#.
]3y tenotomy and optical treatment binocular vision was obtained,
but in both eyes the vision remained the same for fourteen months.
G. Male, aet. eleven, with -f- 2.s V = f# before operation; after ten-
otomy and use of atropine and glasses with -4- 3.s V = f-§-. Binocu-
lar vision was obtained, but no betterment of acuity was procured
in the faulty eye. Numerous specks were visible about the macula.
7. Male aet. twenty-two, in poor eye V = -f (|. Had an operation on
both eyes ten years before I saw him. I did tenotomy on one and
obtained binocular vision, which was known to remain for four
months, but no improvement in acuity ensued in the amblyopic
eye.
These few cases do not justify a broad generalization, but they
correspond to my much more extended observations, and in the
conclusion to which they point, my opinion is in accord with
Schweigger's. It remains to consider how often we obtain binocu-
lar vision. On this point experience has taught me that the num-
ber of well-tested cases in which this can be proved, provided we
demand the same kind of binocular vision of which normal eyes are
capable, is excessively small. I have not gathered large statistics,
but out of the small number of records at this moment at my
disposition, viz.: thirty-six cases 6f convergence operated on,
only four gained binocular vision, which is 11$. Out of the thirty-
six there were ten who had in each eye better vision than f #, of
those only one gained binocular vision. Of the three others who
secured it, the poor eye in all had V = £#. It does not need large
STRABISMUS CONCOMITANS. 195
statistics to show that if among ten patients in whom each eye has
. nearly normal sight and who by tenotomy and optical treatment
\ are put in a position to acquire binocular vision, nine fail to do it,
that there lies behind the ocular conditions, a something which con-
stitutes an insurmountable obstacle. This is indeed the fact, and
to it Hansen and Krenchel have especially called attention. It is the
lack of that cerebral co-ordinating faculty which is the essence of
binocular fusion. It would appear that when this faculty is once
lost or perhaps has never been developed, it can rarely be acquired
after the very early years of life. I do not assert that in only 11$
of all cases can binocular vision be obtained. I have not studied
all my records, but I am certain that the number is less than 20$.
That a certain kind of co-ordination can occur between two previ-
ously strabismic eyes, which do not each direct its macula upon the
object must be admitted. It is often possible after an operation
to elicit double images when it was before not feasible. But this is
a factitious and not genuine binocular vision. One who argues on
these points must explain what kind of vision he claims to exist. A
valuable kind of binocular vision exists in persons who have a high
degree of anisometropia and in whom it is impossible for the images
to be similar and who in reading will use only one eye. This occurs
in persons who have had one eye operated on for cataract; while
this gives sharp vision they cannot dispense with the assistance
which the other unoperated and imperfectly cataractous eye gives
them for purposes of general vision. This vision is really binocular.
Yet there also arise cases of squint where vision is performed by
one eye at the macula and by the other at a point many degrees
aside from the macula. These persons will sometimes be found to
have acquired a kind of binocular sight in which dissimilar parts
of the two retinae have learned to become associated. This kind of
retinal incongruity does sometimes exist, and is not within the scope
of our usual physiological explanation.
In conclusion I strongly urge the importance of exactness in the
performance of the requisite operations and of care and persever-
ance in the subsequent optical treatment to secure the best result.
The reaction after tenotomy is usually slight. In only three or
four instances out of several hundred have I seen important in-
flammation occur, and this soon subsided. Once after tenotomy of
both interni diphtheritic inflammation attacked the wpunds, the
patient having been allowed to go home. The eyes were saved, but
excessive divergence eventuated. The operation for advancement
has been discussed under the head of Strabismus paralyticus.
The treatment of periodic or as it may be termed incipient stra-
bismus by selection of correcting glasses and persistence in using
them will not infrequently cure the deformity and also bring about
196
DISEASES OF THE EYE.
binocular vision. This point has obvious practical importance and
its verity is proven by my records.
Intermittent strabismus is extremely rare. A case reported by
Dr. Harlan in the Transactions of the American Ophthalmological
Society, 1881, p. 277, may be referred to. A child three years old
exhibited concomitant convergent squint every other day for one
year, and it usually came, on awaking from sleep and would last
during the day. It passed over into permanent squint. The re-
fraction was emmetropic as decided by the ophthalmoscope.
Nothing is said of the degree of vision or of the possibility of diplo-
pia. When the strabismus was fully established it varied from
time to time within very wide limits. It was under observation
four years.
Strabismus Deorsum Vergens, or Sursum Vergens,
is to be treated by operating on the rectus inferior, or on the rectus
superior. The obliqui are not suitable for interference. Landolt
has devised a method of tenotomy for the inferior oblique; but the
occasions for it are extremely rare.
It has lately been stated (Eperon1) that advancement of the
weak muscle is to be preferred to tenotomy of the deflecting
muscle. I think favorably of this statement, knowing that ten-
otomy of superior or inferior rectus is not as certain in results as
of the external or internal rectus. Both proceedings may some-
times be combined.
Paralysis of superior oblique is best remedied by dividing the
rectus inferior of the opposite eye, and subsequently, if needful,
dividing the rectus internus of the affected eye and later the in-
ternus of the fellow-eye (Alfred Graefe). Each case will have its
special features and careful estimate of the conditions will be
demanded. Knapp has reported cases.
It is to be remembered that division of the superior rectus acts
by association on the levator palpebrae superioris, and is followed,
not only by a depression of the globe, but by lifting of the upper lid,
by which an unusual amount of sclera will be exposed above the cor-
nea. This fact may be utilized, if there be partial ptosis, both to
aid the levator of the lid as well as the depressor of the globe.
Correct vision below the horizon is of much greater value than
above it. For strabismus convergens or divergens, with upward
or downward deviation, it is proper to cut the adjacent tissues
rather freely on that side of the vertical meridian to which the eye
most deviates.
1 Arch. d. Ophthal., March, April, 1889, p. 115. Condensed in Ophthalmia
Review, July, 1889, p. 205.
CHAPTER X.
ASTHENOPIA.
This term and its synonyms, hebetudo visus, kopiopia,painful
vision, express the fact that exertion of the eyes is wearisome or pain-
ful, and there are often besides ocular, other symptoms, viz., head-
ache, pain in the back, nausea, dizziness, as well as numerous remote
disturbances. The term is convenient, but not definite. To give to
it positive character, we must discover the error or condition on which
it depends. The following subdivisions are clinically separable. As-
thenopia from: 1,refractive errors, causing overtaxed accommoda-
tion; 2, from muscular errors; 3, from neurasthenia. To these has
been added 4, asthenopia as a reflex effect from the nasal mucous
membrane, from which arises often a decided and unusual degree of
conjunctival irritation, and was noted by Schweigger. I have long
recognized an intimate relation between nasal catarrh and chronic
conjunctivitis, and that with it asthenopic symptoms are frequent.
In a somewhat similar sense there are asthenopic symptoms with
incipient cataract, with progressive myopia, with early presbyopia,
but such cases need no consideration under this head. Neither
is there need to refer to cases of pure neuralgia of twigs of the
fifth nerve: the supra-orbital is most frequently at fault, and from
malaria. Hyperaesthesia retinae has been set down as a kind of
asthenopia, but it is commonly only one of its symptoms, and has
for its cause some of the conditions above mentioned.
Accommodative asthenopia need not detain us long. It is the
subjective side of various refractive errors, such as hyperopia, astig-
matism, anisometropia, etc. Upon this Donders laid special stress
and thus set apart an important class of cases. If they have any
typical subjective symptom, it is likely to be indistinctness of vision
after prolonged work on near objects. Besides the blur of print,
there will be pain in the eyes, and especially headache, either frontal
or temporal or general; there may be dizziness, nausea, and other
remote symptoms.
In investigating etiology we may meet large refractive errors,
but it must be emphasized that in susceptible persons small errors,
especially hyperopic astigmatism calling for a cylinder with axis
nearly vertical, or even small degrees of hyperopia, demand exact
198
DISEASES OF THE EYE.
correction. As a fact we have often to deal with a susceptible organ-
ism, and because it is easily set ajar, we must remove even minute
sources of disquietude. For this reason we are called upon to use
atropia very often for such subjects. They may have notable
photophobia, and this at the beginning may be aggravated by myd-
riasis, but perseverance until all spasmodic and painful accom-
modation is abolished will also remove photophobia. It sometimes
happens among these subjects, that atropia causes headache, and
it may be very severe.
If the case, as will in rare instances occur, be one of merely
feeble power of accommodation, useful local treatment is to drop
sol. muriate of pilocarpine, gr. ij. ad oz. i., into the eye once daily;
or, sol. sulphate of eserine, gr. -J ad oz. i. But general tonics and
rest of the eyes will be the chief reliance.
These cases may have other complications, and there are thera-
peutic suggestions to be made which will be referred to hereafter.
It is important at this point to discuss the properties and uses of prisms
before entering upon the subject of the conditions for whose detection and
relief they are employed, viz., errors of the ocular muscles. Their physical
properties are extremely simple. A prism is a transparent substance in-
cluded between two plane surfaces inclined to each other. The thin edge or
angle is called the apex, the thick border is the base ; a line drawn from the
Fig. ra
apex perpendicularly to the base is called the base-apex line. The position
of a prism may be described by referring either to its apex or its base, but
by custom we usually choose the base and say that the base is placed in one
direction or another relatively to the eyes. Refraction through prisms is
shown in Fig. 79. On the surface AC of a prism let fall a ray HK; it is
deflected toward the perpendicular at K, emerges at D, is there deflected
away from the perpendicular to 0. The apex of the prism is A, its base is
CB. The deflection of light is always toward the base. The rays which
impinge at oblique angles are deflected more than those which strike ver-
tically to the plane of incidence. Hence the so-called minimum deviation
implies that rays fall vertically. Beyond a certain obliquity of incidence
rays cannot escape from a prism, but undergo total reflection from the in-
terior and opposite surface. The angle of total reflection varies with the
refractive index. For spectacle glass (crown 1.53) it is 40° 49'. Except for
purposes of examination only prisms of low angles (below 10°) are used in
ophthalmic practice. There is no focus and they form no image. They de-
ASTHENOPIA.
199
compose light into its elements, and this property of "dispersion" gives
them their value in spectrum analysis and is their disadvantage in physio-
logical optics. They produce colored fringes on the edges of objects by this
" chromatic aberration.1'
Objects viewed through a prism are seen in a false position; for example
0'
0
Fig. 80.
an object at 0 seen through the prism appears to be situated at another
spot, viz., O'. The stronger the angle of the prism the greater the displace-
ment, and for prisms of low angles the " deviating angle " is half the angle of
the prism. The displacement of the object is always toward the apex. If a
prism be placed before each eye and the base of each be to the right the
Fig. 81.
object will be displaced to the left, but the visual lines will not be altered in
their angular relation. If, however, the prisms be placed with their bases
the one to the right and the other to the left, or "outward," the object
viewed by the right eye will be displaced to the left, and that viewed by the
200
DISEASES OF THE EYE.
left eye to the right, and diplopia (crossed) would ensue if the visual lines
did not turn toward each other in convergence. Hence prisms with bases
outward are called adducting. If now the bases be turned toward the
median line the opposite effect follows—the object is displaced outward and
again would diplopia (homonymous) occur, did not the visual lines make a
corresponding divergence. Hence prisms with bases inward are termed ab-
ducting. If in Fig. 81 with prisms bases outward the eyes view the object
0, the displacement for each will be toward the median line and double
images will appear projected for L to the right of O and for R to the left of
0, i.e., crossed until the visual lines have converged so as to make the two
images at their intersection at 0' unite into one image. The effect is an ap-
parent approximation of the object, its reduction in size, and a conscious
converging effort accompanied by accommodation.
Now reverse the prisms, as in Fig. 82. Let the object be nearer, say at O.
The displacement is for each eye outward, and homonymous diplopia occurs
until the visual lines diverge far enough to intersect at the point O'. The
effect is an apparent removal of the object and a conscious relaxation of con-
vergence and accommodation. Still other effects occur which will be re-
Fig. 82.
ferred to later. The extent to which convergence is possible greatly exceeds
the possible divergence.
If in the above experiments the object be at G metres or at 20 feet dis-
tance (the visual lines practically parallel), they may by adductive prisms be
forced into extreme convergence and by abductive prisms into a moderate
amount of divergence.
It has been said that for spectacles only weak prisms are used, and that
for them the deviating angle is about one-half the refracting angle. The de-
mand for accuracy in ophthalmic methods has led to suggestions for a more
ASTHENOPIA.
201
perfect system of notation. The reason is that as found in the shops prisms
seldom conform precisely to the number marked as their angle, and it is also
desired to make prisms conform to the principle now universal with other
glasses, of numbering them by their power and not by a purely optical prop-
erty, and in doing this to establish a simple and uniform ratio among them.
Dr. Jackson proposed to number them by their deviating power in angles
and to mark this number on the prism, using the letter d after the number,
e.g., for a prism of 4° angle, 2°d ; of 2° angle, l°d. See Trans. Am. Oph.
Soc, 1888, p. 150. Dr. Dennett proposes a new method based on a very
modern conception in physics, the radian, which is an arc whose length
equals its radius of curvature, viz., 57.295°, of which he would take the hun-
dredth part, the centrad. For special description see Trans. Am. Oph. Soc,
1889, and comments on it by Dr. Burnett, Ophthalmic Review, January,
1891.
An optician, Mr. Prentice (Arch, of Ophth., xix., Jan., 1890, April and
July, 1890), has brought forward a nomenclature which is certainly simple,
accurate, and in accord with our customary practice with other glasses. He
proposes to measure prisms by their displacing effect measured on a plane
(tangent) at 1 metre distant from the prism, and the unit to be a prism which
shall at this distance cause a displacement of 1 centimetre. It happens very
remarkably and fortunately that the prism of 1° and index 1.53 causes this
quantity of displacement within a negligible fraction. This unit he calls the
prism dioptry, PD. The value of any prism can be easily found by drawing
upon a card placed at a convenient distance, say 6 metres, a series of parallel
lines 6 centimetres apart, and looking through the prism to be examined, the
number of spaces and fractions which a long line or a candle flame is dis-
placed, denotes the number of the prism. Mr. Prentice remarks that the
average deflection of our commercial prisms from 1° to 5° corresponds very
closely to this scale up to the fifth division. It therefore follows that the
terms are almost convertible, and our old nomenclature by degrees, differs
little from prism dioptries. The following table gives the differences (taken
from De Schweinitz):
Prism
Dioptries PD.
1.
2.0001
3.0013
4.0028
5.0045
6.0063
7.0115
Refracting Angle of
Prism Required.
1.06°
2.16
3.24
4.32
5.40
6.47
7.54
Prism Dioptries PD. Refracting Angle of Prism Required.
8.0172 8.62
9.0244 9.68
10.0333 10.73
15.114 16.1
20.270 21.13
42.288 39.0073
Prism dioptries are easily convertible into metric angles (see p. 188). By
assuming the average adult interpupillary distance at 64 mm., one MA = 32
mm. deviation, which is about one-third of one PD (i.e., 100 mm.). Therefore
1 PD = £ MA; or 1 MA = 3 PD; or 4 MA = 12 PD, or a prism 12.89° angle.
Another point for consideration is the prismatic effect of decentering lenses.
Mr. Prentice has shown that a lens decentered one centimetre gives a pris-
matic deviation of as many dioptries as there are dioptries in the lens. A
lens _|_ 4 d decentered one centimetre gives a displacement of 4 PD, and
if decentered one-half a centimetre a displacement of 2 PD. Moreover,
DISEASES OF THE EYE.
the centrad of Dr. Dennett and the prism dioptry are almost identical.
It thus appears that the decentration of strong lenses is an important cir-
cumstance, while for lenses less than 2D the deviations are small and seldom
important, except by extreme amounts of decentration. For example, a 2 D
lens must be decentered one centimetre, or j", to get the effect of a prism of 2°.
A table elaborated to millimetres is given by Maddox, "Clinical Use of
Prisms, etc.," Win. Wood & Co., Medical and Surgical Monographs, vol. ix.,
No. 2, Feb., 1891, p. 291.
We shall permit ourselves to speak in the old nomenclature of designating
prisms by their angles, because for those with which we shall chiefly deal
the errors are small. Moreover, in muscular asthenopia not only do we use
weak prisms, but their effect in a physiological sense is far higher than their
optical values would suggest.
Muscular Asthenopia ; Muscular Insufficiency ; Dynamic
Squint.
To the subject of muscular insufficiency and more particularly
of the internal recti in myopic eyes, von Graefe was the first to call
attention. See Arch. f. Ophth., viii., Abth. 11, 314,1861. He did not
omit to mention its existence under other refractive conditions, but
since his time the field of inquiry has greatly widened. We always
take account of the refractive state, but while myopia undoubtedly
carries with it many and serious conditions of muscular trouble, this
fault is exceedingly common both in other forms of ametropia and
in emmetropia. We have to do chiefly with disturbances of adduc-
tion and abduction, while a small number of cases exhibit errors in
the movements upward and downward. We have referred to the
intimate relation which subsists between accommodation and con-
vergence, and we know how greatly this is modified by the refrac-
tion. Attention has been chiefly directed to muscular errors as
they are developed at the working point, and it is here that the
chief strain occurs, but it is found that their study at the far-point
is more often of controlling importance. If the working point, as
in myopia or amblyopia, be very close, the high angle of conver-
gence aggravates the strain in rapidly increasing ratio. What-
ever be the degree of adductive effort, it is needful to have a cer-
tain amount in reserve, and it is also needful to possess a sufficient,
abductive capacity to balance adduction.
Landolt declares that there must be converging power in reserve
twice as great as that which is being employed, but this fails to,
take account of cases where defect of abductive power leaves ad-
duction almost unchecked.
We employ prisms in measuring adduction and abduction, but another
method has been introduced by Nagel, which has been adopted by Landolt
ASTHENOPIA. 203
and to some extent by others, and which, because it is scientifically accurate,
may be explained. It is founded upon a unit called by Nagel the metre
angle, which is analogous to the metre-lens
or the dioptry, which was also introduced
by Nagel. The metre-angle is the angle
formed at one metre distance by the inter-
section of the visual line with the median
plane. In Fig. 83, let the inter-ocular dis-
tance be represented by the base line EE.
Let M be the centre and draw the line MH.
Let EA represent the optic axis of the left
eye when looking at a distance. It is then
parallel to the median plane MH. Now
let the eye E fix upon an object distant one
metre and draw EM' equal to one metre.
The angle AEM' is the deflection of the
optic axis to the median line for the dis-
tance of one metre. This is the metric an-
gle or angle of deflection or adduction for
one metre. Let another point be taken
calling for an additional and equal deflec-
tion ; this will intersect the median line at
M"; add a third equal angle of deflection
and we meet the median plane at the point
M'". The distance from M' to infinity is
one metric angle, from M" to infinity is 2
metric angles, and from M'" is 3 metric
angles. The effort of accommodation to
M'" is 3 D ; to M" is 2 D ; to M' is 1 D.
We see a perfect coincidence in the ex-
pression for the two functions.
The metric angle is manifestly larger
when the base line is longer, i.e., when the
inter-ocular distance is greater. This dis-
tance we measure between the centres of
the pupils, or, more correctly, between the
centres of rotation of the globes. Nagel
gives a table of the value of the metric an:
gle for various inter-ocular distances going
from 50 mm. to 75 mm. In children the dis-
tance may be assumed to be on an average
58 mm., and in adults 64 mm. For the former the metre-angle will be 1° 39',
99', or say 100'; for adults it will be 1° 50'or 110' (minutes). In measuring prac-
tically we may employ an arrangement of Landolfs, viz., a metric tape line
upon which metre-angles are marked, one end of which is held to the temple
and the other end is attached to a little lantern inclosing a candle and which
is the object of fixation. The lantern has a hole in the side and the patient
holds it as close to the eye as he can without seeing it double; this point
read off on the tape gives the number of angles of convergence. To measure
what can be done in divergence beyond parallel visual lines, prisms with the
bases inward are required.
To translate the finding of prisms into metric angles we insert a table
from Prentice (Arch. of Ophth., xix., 1, 1890), where the sine, tangent, and
204 DISEASES OF THE EYE.
angle are accepted as equal, showing the value of the angle of convergence
when the interpupillary distance is taken at 64 mm.
Metric Angles. Dioptries, PD. Degrees of Arc, Sine.
1 3.2 1'50'
2 6.4 3 40
3 9.6 5 30
4 12.8 7 20
5 16. 9 10
10 32. 18 20
20 64. 36 40
In this table the error up to 5 metric angles is unimportant, and we need
not concern ourselves about higher degrees of disturbance, because for such
cases prisms are never applicable. But for persons whose interpupillary dis-
tance is other than 64 mm. we need a rule for determining the metric angle
and the corresponding prisms. Prentice gives the rule: " Read the inter-
pupillary distance in centimetres, when half of it will indicate the prism
dioptries required to substitute one metre-angle for each eye." For 60 mm.
the base line is 30 mm., and the metric angle will be 3 PD. For 50 mm. the
base line is 25 mm. and the MA = 2.5 PD. A reference back to p. 201 will give
the actual angular equivalents of the prisms.
If now we add the number of metric angles for the near, to those obtained
at a distance, we get the total so-called amplitude of convergence; which
may be put into formulae precisely as we discuss dioptries of accommodation.
Before discussing erroneous conditions we must have some
standard of the normal status of the muscular apparatus. The
total lack of binocular vision has been referred to under paralysis
and strabismus; we now consider the conditions under which its
maintenance is difficult. It has been shown that in all persons
there exists a considerable power of forcing the visual lines beyond
the point of binocular fixation, and that the possession of a certain
surplus power is necessary to comfortable vision. If we fix on a
small gas flame at 20 feet distance, we can preserve single vision,
even though prisms with bases outward be interposed amounting
to from 30° to 50°. We can also see a single flame if prisms with
bases inward amounting to from 6° to 12° be interposed. In these
performances the adducting and abducting muscles are forced to
unusual effort, and the limit of their capacity is denoted by the
prisms, which finally bring on uncontrollable double sight—we
see two flames. In doing this we look forward on the median
plane, and the effect consists in forced contraction of all ocular
muscles with predominating effort of certain groups. We may
say very broadly, admitting numerous exceptions, that normal
adduction is 25° to 45° and normal abduction 6° to 8°. Abduc-
tion is more nearly constant than adduction. This may be ex-
pressed in metric angles if one please, but the facts are not altered.
Deviations from the horizontal plane are physiologically more re-
ASTHENOPIA 205
stricted; we can seldom do more than overcome a prism of 2° or 3°
with base either upward or downward. If the head is turned
laterally and the eyes are directed obliquely at the flame the co-
ordination of the muscles is changed and the findings with "eyes
front" will no longer hold good; both adduction and abduction will
change and in diverse ways, not yet much investigated.
If now we attempt to determine the total adductive power we
must not only bring an object close to the face but often resort to
prisms with bases out to ascertain the totality. Many persons
can squint inward to excess and beyond measure. It is of little
value to learn the amount of this ability, and we therefore attempt
to discover what flexibility, so to speak, there may be in the mus-
cular power when we are at the ordinary working distance. This,
may be at from 6 to 20 inches according to the state of refraction
and the habit and age of the person. But if we make proper cor-
rection by glasses, and if we permit certain persons to choose
special distances according to their requirements, we may for the
large multitude assume 13" or one third of a metre as a conven-
ient point for measurement, because it closely approximates 16", the
usual distance of reading, writing, sewing, etc. We have now in
use 3 dioptries of accommodation and 3 metric angles of convergence,
which may be represented by 9.6° PD for the average adult inter-
pupillary distance of C4 mm. Each eye is inclined toward the me-
dian plane a quantity represented by a prism of about 10° angle,
making a total of 20° for both eyes. What additional amount of
adduction will now be possible? Here again wre find great varia-
tions. It will reach from 10° to 50°. As to abduction, we would
naturally count on at least 20° to establish parallelism of the visual
lines, but we must remember that an accommodative effort of 3D
is being maintained, and that this hinders the free use of the abduc-
tive capacity. We find that the range is from 10° to 20°. For
the punctum agendi the average capacity is: Adduction, 20° to
40°; abduction, 15° to 20°. We have at 13" even less freedom in
vertical planes than we have at a remote point.
In the above statements it is seen that we cannot apply the pre-
cise rules to which we are accustomed in refractive problems to
the conditions now discussed, and for obvious reasons. The very
essence of the organs is mobility and variable energy. To Graefe
we owe a very ingenious device to attempt to discover what are the
conditions of dynamic repose of the two eyes. Creating uncontrol-
lable diplopia by a prism with its base-apex line vertical, he con-
ceived that the eyes would adjust themselves to the status of re-
pose and reveal either a normal or abnormal condition. Experience
has proven the untrust worthiness of this test as a single reliance,
but it does create a condition which gives valuable hints. Graefe's
206
DISEASES OF THE EYE.
large dot and fine vertical line are better substituted by a small
white dot on a black card at 13", while the vertical prism test requires
for its distant object the same small flame at 20 feet. Under these
tests the normal and usual results are for 20' a position of the
flames varying not more than 1° or 2° either way from the perpen-
dicular, and for 13" it is most common to have 5° of divergence from
a perpendicular.
As already said, the statements now made about the usual be-
havior and capacity of the ocular muscles under the tests de-
scribed must be taken as very general, and subject to very numer-
ous exceptions. The departures from the status described are often
very startling.
General Divisions and Considerations.—We are testing what
is called the " capacity for fusion " of the separate retinal images.
This is a cerebral function; it is capable of cultivation, it depends
on a variety of factors. Some of them are, 1st, approximate
equality in the refraction and perceptive function of the two eyes,
or, in other words, the absence of high refractive errors or of
marked defects in visual acuity. For these reasons cases of anisome-
tropia, of muscular amblyopia, of loss or defect of sight in one eye,
of decided myopia in each eye, of hyperopia, of visual obtuseness,
always predispose to or present notable variations from a normal
standard of fusion. Again, 2d, we find actual muscular anomalies,
in spasm of certain muscles, or in weakness of muscles which may
be due to faulty insertion, or to lack of muscular fibre. 3d. Doubt-
less variations in the density and arrangement of the oculo-
orbital fascia give rise to greater or less degrees of effective-
ness in the muscular contractions, because it holds them steady and
makes an auxiliary insertion for their tendons. Especially is this
true of the recti. The importance of this fibrous membrane has been
too little considered both as a causative factor and in operations.
4th. The complexity of action of the twelve muscles concerned,
the necessity for rapid, exact, and smooth performance, and that
during waking hours or the maintenance of the visual act, they are
perpetuall}' on duty, and that their combinations must be adjusted
to such variable distances, angles, and cover so wide a field, and
that the mental consciousness relies so absolutely and instinctively
on their work in its judgments respecting the external world—these
considerations make it clear that their function is important, and
that we ought to expect the same variations in their conduct which
we are not surprised to find in what we call the temperament, the
constitution, the idiosyncrasy of different individuals. 5th. We
are brought by the last statement to consider the condition which
more than all others is the presiding factor in muscular asthenopia.
This is somewhat difficult to express, but may be called the quality
ASTHENOPIA.
207
■of nerve energy. The health of the efferent nerves, of their nuclei
in the medulla oblongata, and of those portions of the cortex,
with which they are associated, in fine the vigor and quality
of the whole nervous system enter into the problem. This con-
sideration explains why in some persons large muscular anoma-
lies have little or no disturbing influence and in other persons small
errors are most harassing. This explains why asthenopic symp-
toms appear after attacks of sickness, during debilitating condi-
tions, as the consequence of grief, of shock, of worry, as the accom-
paniment of many chronic diseases having no direct connection
with eyes or brain. 6th. Overwork and inadequate nutrition bring
about muscular asthenopia. On this point we do not dwell. Lastly,
if we have not catalogued all the conditions which account for the
presence or absence of symptoms of muscular asthenopia, it is clear
that among those mentioned numerous combinations of local and
general causes can take place which will give rise to startling and
perplexing and distressing situations. The judicious and skilful
treatment of cases of muscular asthenopia with all the complica-
tions which may co-exist is the highest problem in ophthalmology.
We divide cases of muscular asthenopia into two broad classes,
and these together make a third, which is naturally the largest.
We have the myotic and the neurotic. Under the myotic are in-
cluded those in which muscular anomalies are the great and pre-
dominating factor; the neurotic are such as exhibit such anoma-
lies, but their influence is made potential by the dominating neu-
rotic condition. In the first class, refractive errors may play
a, part by inducing muscular disturbances, and their elimination
often completely removes the muscular troubles; in other words,
the correction of astigmatism, of hyperopia, of anisometropia,
will often leave no occasion for dealing with muscular faults be-
cause the strain on the muscles disappears. (It is pertinent to say
that these errors express their hurtful influence through disturb-
ances of a muscle, viz., the ciliary, and the intimate association of
this with the motor muscles makes the concurrence natural.) On
the other hand, a minority of cases of refractive error do not find
relief by focalizing glasses, but demand substantial aid for com-
plaining muscles in addition. These may furthermore be compli-
cated by neurotic conditions which must have full recognition, or
treatment will not, and perhaps cannot, bring relief.
Furthermore, numerous cases present themselves in whom the
manifestly conspicuous feature is the neurotic disturbance; it may
be excitability or intensity of action, or exhaustion from any
cause. In these cases muscular defects are common, either in a
greatly reduced power of fusion, feeble muscularity in all directions,
or in spasm of convergence, or in the hurtful effect of minor errors
208 DISEASES OF THE EYE.
whether of one sort or another. It is of course frequent to find
small refractive errors, and how marvellous may be the benefit
gained by their correction, has been already noticed. It is here
that the potency of the .25 D is observed. It is also among these
people, mostly women, that the very weak prisms find their utility.
To such a point (a vanishing point) has this prescription been
carried, that a paper has been written on the value of the prism
.25 P. D. Modern civilization, the exigencies and urgencies of so-
cial and business life, hereditary tendencies, and the American
temperament with its stimulation of the mental faculties makes
neurotic asthenopia in its various phases a disease widely prev-
alent in our community. How this general tendency in some
cases reacts upon the retina will be mentioned later.
These general statements will, I trust, make it clear that my
standpoint in viewing a case of muscular asthenopia is to regard
the general condition of the patient as the controlling circumstance,,
and deal with the ocular conditions in subordination to that. I hold
firmly to the necessity of rigid, complete, and exhaustive investiga-
tion of all refractive, muscular, and other functional errors, and
apply the proper correction, but the purpose is to include these
particulars in a judicious estimate of what relates to general health
and temperament and surroundings. A corollary to this statement
is that while with neurotic people small errors must receive un-
usual consideration, one must not forget with them the influence
of mental impressions, and the cures wrought by suggestion, hyp-
notism, metallotherapy, electricity, etc. We may now particu-
larize some of the usual exciting causes. We have to note over-
taxation by reading on railway trains and in carriages; by reading
when lying down, which convalescents and chronic invalids often
find out too late; by attempting difficult work, such as embroidery,
sewing on black, fine painting, decoration on china, etc.; bending
over the work and bringing it too near the eyes; by the study of
languages whose text is intricate, such as Greek, German, Hebrew,
etc. Want of vigor, whether by congenital conditions of health, by
too rapid growth, by malaria, by any debilitating causes, by shock,
grief, etc., are to be duly considered; especially all forms of uterine
disease, hemorrhage, fevers, chronic anaemia, instigate muscular
asthenopia. It will often happen that the depressing agencies
mentioned, are simply exciting causes of a disorder whose real
progenitor is an essential muscular weakness, which may long
have been latent, but is now made potential.
Some cases seem to have congestion at the base of the brain
and there is tenderness over the middle and upper cervical verte-
brae. Nasal catarrh is both a complication and a cause, while the
same is true of chronic conjunctivitis of the lids and blepharitis.
ASTHENOPIA.
209
Subjective Symptoms.—Pain in using or fixing the eyes is the
conspicuous symptom. This appears in all kinds of near work,
reading, writing, etc.; it may also exist in distant vision, in look-
ing at a crowd, or at the stage in a theatre, looking out of a car-
riage or from the window of a railway car, etc. There may be
great photophobia. Seldom is there blur of sight, while unsteadi-
ness of letters or work, which is caused by a tendency to diplopia,
resembles the blur of refractive error, and is sometimes thus
spoken of. The pain is generally in the eyeballs and inclines the
patient to press on them for relief; but it is often temporal, fron-
tal, occipital, or at the vertex. In truth, not a small percentage of
obstinate headaches, especially " sick headaches," originate in dis-
orders of ocular muscles. A frequent symptom, and one not easy
of explanation, is headache on first waking from sleep in the morn-
ing. Sometimes dizziness occurs. When the general health is fee-
ble, or the subject neurotic, we may have the most erratic and
intense remote symptoms: aphonia, palpitations of the heart,
pain in the ovaries, diarrhoea, rectal irritation, etc. Asthenopia in
some subjects is almost as protean as hysteria, and the two go
hand in hand. It has been asserted that chorea is caused by this
condition, but my observation has been that usually the order is
otherwise, and that it is chorea which gives rise to debility and
irregular action of ocular muscles as one of its manifestations.
Epilepsy has been asserted to depend on this cause. I cannot deny
that, in a few cases, eye strain may have been demonstrated to
be an exciting cause or occasion, but there has been behind it a
deeper lesion of the general nervous system. This statement is quite
consistent with the disappearance of epileptic fits in case the mus-
cular eye trouble is cured. Well-marked relief in a case of epilepsy
took place in the practice of Dr. Ranney of which I had personal
knowledge. The great relief, effected by tenotomies, was only
partially maintained afterward, but the health improved and the
frequency of the fits was much reduced as late as three years after
the operations were begun. For further discussion on this point see
Report of Committee.1
Objective Symptoms.—These are as follows: while both eyes
within certain limits seem to move in harmony, for certain extreme
positions to the right or left, or up or down, the}* become tremu-
lous or one will deviate; in the median line and at moderate dis-
tances there may seem to be no fault, but if an object be brought
very close to the nose a deviation may occur. If no evidence is
'Journal of Nervous and Mental Disease, Nov., 1889, p. 657. On the sub-
ject of the general disturbances of the nervous system due to ocular derange-
ments see Anstie, "Neuralgia." 1872, p. 169, the writings of Weir Mitchell,
and " Functional Nervous Diseases,-' Stevens, New York, 1887.
14
210
DISEASES OF THE EYE.
thus obtained, repeat the same experiments, bidding the patient
regard the finger as it is carried to various extreme positions, and
while he fixes upon it, put a card before one eye and note whether,
when binocular sight is thus abolished, the covered eye may not
deviate from its correct position. Especially useful is this test
when searching for weakness of adductive muscles, the finger
being brought within a few inches of the nose, and each eye alter-
nately covered by the card. There are other ways of determining
the so-called static condition of the eyes, but they only give hints
and not measurements. The parallax test (Duane) is one, which
shows a deviation in fixation by the difference in localizing the gas
flame with each eye alternately, the other being screened.
Within a few years much has been written, in this country
especially, on tests for muscular errors, and various refinements
have been brought forward by Duane, N. Y. Medical Journal,
Aug. 3, 1889; by Stevens, the stenopaic spherical lens, Ophthalmic
Record, vol. i., 7 and 8, 1892, Nashville, Tenn.; by Maddox, the rod
test, and the double prism test. Several instruments have been
invented, viz., the phorometer, by Stevens; an apparatus by Ris-
ley, Med. and Surg. Reporter, Phil., Dec. 5, 1891; an apparatus by
Gradle of Chicago, and other contrivances. Long practice in using
simple contrivances and the habit of searching repeatedly and
with much care have enabled me to accomplish so much that I
have not cared to be cumbered with elaborate apparatus. The
necessary appliances for my work are squared prisms about 1£
inches long on the side and varying from £° to 15° and some twenty
in number; prisms mounted in series or piles, two sets, one having
even numbers from |° to 20°, the other having odd numbers from
1° to 21°; a combination of prisms 5,° 15°, 10°; prisms mounted in
spectacle frames of low degrees, 1° to 3°, with bases in or out; a
Maddox rod mounted, a square double prism (Maddox), a disc of
red glass. These serve for the examination at 20 feet. For the
working distance I employ a simple prism holder shown in Fig. 86,
which serves likewise for detecting spasm of accommodation, and
will permit the use of correcting glasses as well. The test for the
near ought never to be omitted. I discard instruments like the
revolving double prism of Cretes because they tempt the patient
to put forth efforts which are only temporary and misleading to
the oculist. Hence in the piles of prisms the intervals are 2°, so as
to learn more correctly what the patient can do. To keep the
square prisms level is very easy, and I avoid in an examination the
use of trial frames, or of anything placed on the face until high de-
grees of prisms are reached. The argument on behalf of Stevens'
phorometer, that by being placed several inches from the patient
it removes the liability to cause stimulation of muscular errors, in
ASTHENOPIA.
211
my experience is not well founded. The phorometer is widely
used, is a good piece of mechanism, but its tendency seems to me to
be to develop and exaggerate small errors which are unimportant.
I need not remark that refractive errors are corrected by suit-
able glasses, and sometimes atropia is used. If refractive error be
above 2 D the correction is always interposed; if less than 2 D I am
not careful about it, because large and careful investigation has
shown that small errors seldom make impor-
tant modification of the muscle findings. In all
muscular tests a red glass before one eye ex-
aggerates the conditions observed, by diminish-
ing the tendency to fusion.
The first test object will be the small gas
flame at 20 feet, against a dark background.
For errors of the oblique muscles a heavy black
line, one foot long, on a white card is useful. A
small gas jet may be put at the middle of a
Fig. 84. Fig. 85.
card on which are lines drawn parallel and 6 centimetres apart,
and the card made to revolve, to permit comparison of the dis-
placement caused at different positions of the prism.
The patient stands, the test box is at his right, the observer
faces the box and the patient; he holds before the left eye a 5° or
8° prism with base upward; vertical diplopia ensues, which perhaps
the patient does not immediately notice—the flame seen by the left
eye is projected below. If the two images are not plumb, the pile
of prisms (Fig. 85) is brought before the right eye with base on the
side where the top image is said to stand, and moved up until the
images are made plumb. A. red glass will develop the error to a
212
DISEASES OF THE EYE.
greater degree, whatever its character. Maddox' glass rod held
horizontally before the left eye gives rise to a vertical (or perhaps
oblique) streak of light on the level of the true flame, which it may
intersect or lie either to the left (convergence), or to the right (di-
vergence). The double prism of Maddox (advantageously modi-
fied by Savage) consists of two prisms, each of 4°, placed base to
base, and the patient looks through them at their junction. One
eye (always the left, unless this is highly amblyopic) sees two
displaced images and between them the true flame. These three
flames are to be brought in line by the pile of prisms above men-
tioned.
The test now made has decided value, but it does not measure
anything precise or which has commanding importance; it is
merely one factor among many. To speak of this finding as the
measure of the so-called insufficiency is to fall into error. There is
no exact measure of " insufficiency," and the term is one of general
significance, not of precise notation.
The next test is to measure the degree of abduction. Hold in
one hand a square prism of 5°, base inward, and if the light re-
mains single put a prism pile before the other eye with base in-
ward, until the extreme capacity for fusion is found. If the light
become double, turn the base of the prism pile outward and re-
duce the effect of the 5° prism until the correct result is obtained.
At this stage another use may be made of the abductive prisms.
Place before one eye a prism of 10°, base in, and if this be not
enough to excite diplopia, add 5° or 10° to the fellow eye. Hold the
prisms horizontal and inquire if the double images are on the same
level; if not, there is a tendency to vertical error (hyperphoria). It
is not at all rare to find small errors. If we rotate the patient's
head on a vertical axis, the sidelong fixation will many times
bring out vertical error. So common is this experience as to be
almost physiological. The reason is, that in a movement to the
left, for example, the left eye easily preserves the vertical meridian
vertical, but the right eye under the influence of the superior and
inferior recti as sinistral rotators must be exactly balanced by the
superior and inferior oblique to keep its vertical meridian vertical,
and this implies an equilibrium of forces very likely not possible.
It must be remembered that the strong prisms have abolished
the instinct for fusion, and the test object (the gas light) is so
small as not to furnish such an inducement to fusion as would a
larger one or as would a rod or line. In discovering vertical errors
it is of the utmost value to make the patient move the head back-
ward and forward as well as sidewise, while the prism induces ho-
monymous images; the evidence is the fact that at certain positions
there is a greater or less amount of displacement than in other
ASTHENOPIA. 213
positions. An additional proceeding is to hold a 5° prism base up
before one eye and then place it before the other, or reverse it be-
fore the same eye. If there be a notable discrepancy in the separa-
tion of the images, this marks hyperphoria. It will be noted that
none of these experiments stimulate a tendency to vertical move-
ment; they simply elicit the tendency to want of parallelism of the
visual lines in the vertical plane.
The measure of the amount is made by placing before one e3Te a
prism with base toward the image, until a level is established.
This prism will vary from £° to 3° or 5°, while I have corrected
a case of 17° vertical error. In this kind of measurement (for
hyperphoria) the phorometer of Dr. Stevens is exceedingly con-
venient and accurate. As to the practical value of the measure-
ment, comment will be made hereafter.
The next step is measurement of the adductive power. We
expect this to equal 25° to 60°. Most people will show less than 30°
Fig. 86.
until educated to more. The compound prism, Fig. 84, may be held
in the left hand with the base out. Employ the 5° next the
10° and before the other eye place one of the piles and carry it up
to its maximum ; then put the 15° before the left and again run
through the pile until its bottom prism is reached, or until the
patient "balks" and declares that he cannot unite the images.
One must coax and urge and persuade and "chaff" the patient to
do his best and never lose patience, and be ready to try, try again.
If the adducting faculty is higher than 32° put on a pair of square
prisms, each 10°, in spectacle frames, and begin again as before.
It is a useful check test in some cases to try the adduction in side-
long positions as well as with eyes front; it will always be lower
in amount, and sometimes extraordinarily.
It is proper to make mention at this point of the nomenclature for latent
muscular errors (called by Graefe dynamic squint) invented by Dr. Gr. T.
Stevens (Arch, of Ophthal., XVI., No. 2, 149. 1887), and which has gained ex-
tensive currency. The terms are convenient as general designations. But
214
DISEASES OF THE EYE.
the excessive elaboration which has been proposed, and the intensely sym-
bolic writing which some have employed like a cipher code, mystifies and
obscures the facts.
I. Generic Terms. Orthophoria: a tending of the visual lines to parallel-
ism ; heterophoria: a tending of these lines in some other way.
II. Specific Terms. Heterophoria may be divided into: (1) esophoria: a
tending of the lines inward; (2) exophoria: a tending of the lines outward ;
(3) hyperphoria: a tending of the right or left visual line above its fellow.
This term does not imply that the line to which it is referred is too high, but
that it is higher than the other, without indicating which may be at fault.
III. Compound Terms. Tendencies in oblique directions may be expressed,
as hyperesophoria: a tending upward and inward; or hyperexophoria: a
tending upward and outward. The designation " right" or " left " must be
applied to these terms.
We next examine the same functions at the patient's working
distance, and to do this I have long found it convenient to use a
holder which consists of a central stem about 0.5 metre long, gradu-
ated in inches or centimetres and metric angles, on which is a slider
carrying test cards, and which contains three cells before each eye
into which corrective glasses and squared prisms may be dropped
(see Fig. 86). It will contain prisms amounting to 50°, and is more
convenient than the trial frame. If, as sometimes happens, a pa-
tient cannot put forth his real energy when lookipg into an appara-
tus, let him make a few trials without it, holding the test card in
the hand. We usually begin with Graefe's so-called equilibrium
test, employing a prism of 10° with base vertical. He used a large
dot upon a very fine perpendicular line; I use a white dot upon a
black card as less liable to stimulate efforts of fusion. Some use
fine letters. It is most common to find that the images do not
stand vertical to each other, but that there is a deviation in the
sense of divergence of about 5°; this cannot be called abnormal.
More than this must be noted. A deviation in the sense of con-
vergence is always abnormal, and directs suspicion upon the ex-
terni. Next try the abduction and the adduction by prisms suitably
placed. It is best to begin with abduction. Often a patient will
show only 6° or 8°, because of involuntary convergence; hold the
finger between him and the test card and bid him follow it down to
13"; he gets single vision of the finger, and as this drops out of sight
the dot is single. Add another prism, say 2°, and shift the prisms
from one side to the other so as to make the increments small,
usually by 2° or 3° at a time. Gradual coaxing will bring the
abduction to its maximum. This device is extremely important in
the numerous cases where vertical diplopia at 13" displays marked
esophoria, Another trick is to push the slider half-way up to the
face and gradually withdraw it. It is absolutely necessary to give
the patient every possible chance to exhibit all the abduction he
possesses. In trying to find all the adductive power, we cannot do
ASTHENOPIA.
215
so much to aid the patient as for abduction; nevertheless pushing
up* the slider and slowly withdrawing it will often help him do his
best.
In assuming 13" as the proving distance, allowance must be
made for high refractive errors, especially myopia, and the distance
may be modified to the requirements of the case. It is also obvious
that the glass needed for the working distance must be provided;
this applies to presbyopia as well as to all refractive errors. It
will to many be a surprise to learn how low is the adductive power
with presbj^opic correction, and in how few instances inconvenience
is felt. On the other hand the displacement of the relative accom-
modation explains the annoyance caused by using too strong glasses
for presbyopia, and sometimes, when the glass is weak.
It is true, as is now generally recognized, that our findings at
the working point are subordinate in importance to the conditions
elicited at 20', but they have no little significance in the general
estimate which we make. Very often the indications corroborate
the diagnosis of spasm of convergence in the strongest way, or
they teach us that we have to do with overtaxed and incapable
muscles for which rest and improved general energy are most
necessary. The prescription of prisms or of tenotomy on the basis
of what tests at 13" show is seldom advantageous, or even per-
missible.
In cases where marked esophoria shows excessive convergence
for the whole range of fusion, we must look closely for spasni of
accommodation. Often in these cases there is mild li3Tperopia or
astigmatism, or both; we want to know if the}^ alone condition the
muscular disturbance. Before using atropine, or even if it has been
used and we want to find out if its work has been fully achieved,
we may resort to a test on which I have relied for approxima tely
accurate results for twenty years. I slip into the holder a pair of
spherical prisms 4-3DC prisms 6° bases inward. An emmetrope
ought theoretically to read S. 1 (the very fine capital letters N P
r T v) at 13" with -f- 3 D. But this will only be true with monocular
vision and with relaxed interni, conditions seldom presented in the
consulting room; hence we must cause relaxation of the interni by
interposing abductive prisms. The convex glass and the prism com-
bine to the same result. As a fact, with such a glass (an approxi-
mation to Briicke's dissecting spectacles) the point of fusion of the
visual lines for the average inter-ocular distance of 64 mm. is not
far from 2 M. A. In reality, as the conditions are various in dif-
ferent persons, the intersection of the visual lines is at 4' to 6' dis-
tance, while the accommodation is at 13". Some enlargement of
the image is caused by the glass. Reading S. 1 at 12" is not to be
regarded with suspicion, but when the point must be pushed to 10"
216
DISEASES OF THE EYE.
or 8" we have to diagnosticate spasm or amblyopia, and act accord-
ingly. If vision be feasible at 11£" to 13" we need not be concerned
about atropine unless other evidence compels it, such as the oph-
thalmoscope, the shadow test, etc.
An indication of vertical error may often be found by the tip-
ping of a patient's head; he carries it bent to the shoulder and
perhaps a little rotated. Close inspection will often show asym-
metry of the head and face—one ear will be higher than the other;
the floors of the orbits are not horizontal; the cranium will be lop-
sided. In such cases, which are common enough, one will usually
find hyperphoria, and sometimes, but by no means always, will it
demand correction. Observations of this character have been
recorded for many years among my notes. So decided are my
convictions on this matter that I may cite instances of evident
paresis of the superior oblique muscle examined and treated more
than twenty years ago in which the best results were gained not
by correcting the difference of the images in height, but on the
working and the horizontal plane. The writer is himself an in-
stance of the innocuousness of a vertical error when it develops
itself 15° above the horizon; this he has from traumatic paresis of
right obliquus inferior (see Trans. Am. Oph. Soc, 1877). It must,
moreover, be emphasized that examinations at the working point
should be made not on a horizontal but on a plane 15° below the
horizon, for this is the physiological primary position of fixation
(Aubert, G. and S.). It cannot, however, be denied that cases exist
in which hyperphoria throws other sets of muscles into spasm and
irregular action, giving incongruous findings with prisms, and re
lief is only gained upon removal of the discord in the muscles of
elevation or depression (see Stevens, 1. c, p. 165),
It may be well to remark here upon a special symptom seen
rarely and in severe neurasthenic cases, viz., retinal anaesthesia. It
consists in limitation of the visual field, rarely in hemianopsia, and
still more rarely in general reduction of light perception. There are
no abnormal appearances with the ophthalmoscope. Wilbrand1
called attention to it, and Priestley Smith2 has related cases. If the
visual field be examined by the perimeter more than once, the field
will be found smaller on each successive trial, and Priestley Smith
therefore refers to this as a spiral limitation, and* calls it a reflex
amblyopia due to an impoverished state of the blood, and excitation
of the vaso-motor nerves causing arterial contraction. I have ob-
served a few cases, and clinically they differ from migraine in being
caused by fatigue of the eyes. See case by Stewart.3
1 Archives of Ophthalm., xii., 428, 1884.
2 Ophthalmic Review, vol. hi., p. 140, 1884.
3 American Journal of Ophthal, Vol. 7, 184, Julv. 1888.
ASTHENOPIA.
217
Asthenopia associated with disorders of the nasal cavity deserves special
mention—the connection between chronic palpebral conjunctivitis and nasal
catarrh has been referred to, and for a long time it has been the occasion in my
own practice for treating nasal catarrh to cure the palpebral disease. With-
in a few years several authors have written upon it (see Nieden1), but some-
thing less simple and obvious sometimes appears in the phenomena of pro-
nounced asthenopia. To these cases Nieden refers in a brief paragraph (1. c,
p. 419), and a very marked instance recently occurred in the person of a medi-
cal friend, which may be briefly described.
Dr. T. E. S., aged 44, a hard-working and distinguished practitioner of New
York, given to microscopy and book making, found his eyes give out in 1886.
He consulted a competent oculist, who gave him various glasses with incom-
plete success. He went away for a vacation and under advice of another able
oculist be underwent the use of atropia for five weeks, and received a modified
formula for glasses: viz., O.D. + 1.25s DC-i-0.25cD 90° visusf^; O.S. + 1.25c D
3 + 0.50c 90° visus f|}. A note by the physician also stated that there was
want of energy in the internal recti muscles, and excessive power in the supe-
rior rectus of the left; that headaches were due to excessive strain of accom-
modation. He wore glasses in accordance with the above formula with the
addition of a prism 1° base down for O. S. For reading + 0.75 D was added to
the above. His distressing symptoms had continued two years.-consisting of
pains in the head, heat at the vertex, insomnia, inability to use his eyes and
the effort would be followed by facial neuralgia. He had intense photopho-
bia and had worn dark blue glasses for months. He had had much mental
strain outside of his professional work, and his case was evidently a complex
of local eye troubles, with retractive and muscular and general nerve exhaus-
tion. A marked symptom was extreme palpebral congestion, and tendency to
lachrymation on exposure to light and attempting eye work. So pre-emi-
nent was this feature that I was led to inquire into the condition of the nasal
cavity. I found the passages narrow with slight protuberance of the sep-
tum from undue thickening, decided congestion, tenderness on being touched,
and anaesthesia by cocaine afforded relief in some measure to the eye symp-
toms.
Careful examination of refractive and muscular conditions showed that at
18 adduction = 15c, abduction = 7°; the ophthalmometer gave astigmatism,O.D.
1.50 D 75" ± 165°; O.S. 1.25 D 90° ± 180V It was evident from the variability of his
answers to the muscular and refractive tests that there was much spasm both
of the extrinsic and ciliary muscles. Guided by his previous treatment and
the present symptoms, I determined to attack the nasal disease and made
several applications of spray. Relief was experienced, and on hearing that he
suffered from asthmatic attacks, that his breathing at night was much embar-
rassed and must be done with open mouth, I determined to enlarge the nasal
aperture, both to get rid of a hypersensitive surface and to afford more air space.
I removed a projection of the septum on the left side with the saw. Marked
improvement quickly took place. He took horseback exercise with enjoy-
ment ; within a month reported that he " felt young and frisky.'' The glasses
were slightly modified—the photophobia soon abated and practically disap-
peared. Muscular spasm ceased, all headache and neuralgia vanished.
Within four months he laid aside glasses, was restored to comfort, and is
1 Arch, of Ophth , vol. xvi., No. 4, 1-SST, p. 416.
2 The si.M DISEASES OF THE EYE.
a weak convex glass, or a weak prism 2° or 3° for each eye, with
base inward, is given. Each day the period of reading is increased
by one minute or by two minutes, and the most scrupulous exact-
ness is insisted on. In place of reading, other work may be substi-
tuted, but the great matter is to regulate and systematize the eye
work. Combined with this proceeding, the galvanic battery, either
the constant or interrupted current for a few minutes, with one
pole to the closed eyes and one pole on the temple, has some, yet
small, value. Stimulating liniments to the forehead and temples
of aconite, or of chloral and camphor, etc., are useful when there is
neuralgia. The douche or spray of cold water, or mild lotions to
the eyes, viz., borax and camphor-water, are all helpful. In some
cases, notwithstanding refractive errors are slight, it is best to pre-
scribe the wearing of glasses, especially if convex or cylindric, all
the time. The behavior of the muscles, both with and without the
glasses, will help to decide this point.
Another mode of invigorating the eye-muscles, and which is
especially suited to the cases where all the muscles are feeble, is by
using prisms as means of gymnastic training. Dyer's method deals
with muscular action and accommodation together; by gymnastic
prisms the extrinsic muscles alone are acted upon. The patient is
provided with prisms of 2£°, 5°, two of 10°, and one of 15°, with
squared outlines. He takes a candle-flame or door-knob at twenty
feet for his object, and performs the efforts of adduction and abduc-
tion by means of these prisms. He begins, say with adduction, and
at first holds the prism of 5° with base out, in front of one eye, then
substitutes the 10°, then before the other eye, places 5°, making a
total of 15°; then, if practicable, substitutes the other prism of 10°
for the 5°, and so climbs up the ladder of adductive prisms l\y such
steps as he can make. If the interval of 5° becomes too great, he
may take that of 2^°. On the other hand, he will in a similar way
train the abductive muscles by putting before one eye with its base
inward, the prism of 2^°, then that of 5°, then one before each eye,
and finally, may possibly reach the 10°. To reach an adductive
power of 42|° and an abductive power of 10° will require sometimes
several weeks, and when attained should be practised once or twice
daily. The daily session need not occupy more than ten minutes,
and need not be more frequent than twice. There are few cases to
which this method is well suited, and one need not spend much
time over it. Improvement in adduction is often gained, but sel-
dom in abduction. Some special disharmony will usually be dis-
covered. Sometimes it will be preferable for the physician to
superintend the prism practice, both for its beneficial effect and to
elucidate the diagnosis.
If muscular defect be combined with important refractive error,
ASTHENOPIA.
221
we have most frequently myopia with insufficiency of the interni,
which may be relieved either, 1st, by wearing the full optical cor-
rection continually; or, 2d, by using for near work a glass which
pushes out the near-point to 8", to 12", or to 14", and which may
be of about half the power of the full correction; or, 3d, with the
glass just mentioned a prism may be combined, or the glasses may
perhaps be given an adequate prismatic quality by having them
set in the frame with their centres outside the visual lines. This
brings the inner thick edge of the glass into use, whereby it will
have a low prismatic effect. For the rare cases of myopia with in-
sufficiency of the externi, optical corrections alone are not often
available. With hyperopia similar methods of proceeding may be
adopted, but with such adjustment of prisms as the kind of muscu-
lar error calls for. With emmetropia one finds less certainty in the
helpfulness of prisms to aid the performance of near work. They
sometimes are utterly intolerable, even with decided muscular error.
In deciding how strong the prisms are to be, we first decide the
proper working distance, and the correcting-glass, which for this
point is required, and with it ascertain the muscular error. To
give prisms equal to one-half the amount of error is usually suffi-
cient. If we estimate the insufficiency at say 10°, we may order
the prisms each 3°, one before each eye. It is only when error is
decidedly more on one eye than on the other that the prisms are
made unequal. But for quantities so large, tenotomy is preferable.
The cases of muscular asthenopia without any or any important
refractive error are much more frequent than has been supposed.
By far the largest quota present insufficiency of the external recti.
To these cases my attention has been pointedly called within a few
37ears;1 and they become in my experience more and more con-
spicuous. They are recognized by the tests at a distance rather
than by those for the near.
To form some idea of the refractive conditions in muscular
asthenopia, one hundred consecutive cases for which prisms were
deemed suitable are quoted. They are taken from my series of
records of muscular error noted from C 826 to C 969, in all 144
cases. The 44 rejected were either not counted suitable for prisms
or were not observed long enough to be valuable; some were purely
neurotic cases, some exhibited weakness of all the ocular muscles,
and some were operative cases. Emmetropia including those with
H not exceeding + 0.50 D = 52; H from .75 D to 2.75 D = 24; Ash
from + 0.75 D to + 2.00 D = 3; mixed astig. 4.50 = 1. Total, 100.
Of the 24 cases of H, 11 were of 0.75 D, giving 63 cases in which
1 See paper "On the Tests for Muscular Asthenopia, and on Insufficiency
of the External Recti Muscles." Trans, of Eighth Session of the Interna-
tional Medical Congress, Copenhagen, 1884,
222
DISEASES OF THE EYE.
the amount of II was insignificant and was certainly not the im-
portant factor. Alongside of these may be placed 11 cases of Ash
of + 0.75 D, although in them more stress is to be laid upon the re-
fractive error than for simple H. Astigmatism of all kinds claims
21 cases; and myopia 3. A further interest attaches to the kind
of muscular error, and herein my views published in 1884, and
quoted in the 1st edition of this text-book, p. 201,1890, are fully cor-
roborated by additional experience. Insufficiency of recti externi
= 73; insufficiency of recti interni = 26; vertical error = 1; total
= 100. The similarity of results is remarkable because in 1884 I
found 74$ of recti externi, and in 1892 I find 73$ of the same. A
further analysis giving the refractive conditions under the separate
types of muscular error shows the following:
Insuf. externi, E error not exceeding +.50 36
H 19
M 2
Ash 13
Asm 2
Antimetropia 1
73
Emmetropia, hyperopia, and hyperopic astigmatism include 68
cases, and while the refractive error was in most cases corrected,
this alone did not give relief. It is quite admissible to suppose that
spasm of the interni is more at fault than defect of the externi, and
this is doubtless the true pathogenesis of most cases, and that the
weakness of externi is secondary. The result of treatment in the
abandonment of prisms after a certain time, often demonstrates
this to be true. It might in some cases, perhaps, have been pos-
sible to break the morbid chain by prescribing atropine for three
to six weeks, but cui bono ? Prisms meet the indications.
Insuf. interni, E error not exceeding + 0.50 16
H 5
Ash 4
Asm 1
26
Among these cases spasm is necessarily excluded, and they fur-
nish a sufficient answer to the objection made against my views
of these cases, that they represent simply minor degrees of uncor-
rected hyperopic error rather than real muscular faults. Among
them emmetropia and hyperopia and hyperopic astigmatism
ASTHENOPIA.
223
count for 25$. I need not press the logical value of these cases;
they eliminate all that might be urged in opposition on the ground
of personal bias. To complete the cases there was one of vertical
error—giving this form of trouble (hyperphoria) the ver}7 modest
showing of 1$. It is right to add that among the 44 rejected cases
were 6 in which vertical error was noted, making out of 144 cases
of all kinds of muscular trouble almost 5$ of this type.
It is common to find that wearing prisms a little while increases
the manifest amount of muscular weakness. I speak particularly
of the externi, in other words the abductive power is less. The
explanation simply is that the overtasked muscles give up the
struggle and lean on the glasses. Hence it frequently follows that
prisms are a prelude to tenotomy. This comes about when pain
returns, in spite of prisms. I once increased the angle of adductive
prisms for weak externi up to 7° for each eye in obedience to the
demands of a patient, who refused tenotomy,'but for whom it was
finally done with most brilliant result.
That prisms so weak as 1|° are capable of producing positive and bene-
ficial effects is not only attested abundantly by experience, but a sufficient
reason is found in the degree of convergence which they induce. Instead of
parallelism we have an abatement of one-half a metric angle for the inter-
ocular distance of 60 mm. And this is in reality one-sixth of the effort re-
quired at the assumed working point of \ metre or 3 metric angles. A con-
stant abatement of abduction amounting to 16$ of this requirement is
certainly not insignificant.
If there be insufficiency of the interni for distance, we may uni-
formly expect the same at the working point. It may, however,
not exist at the remote end, and exist at the proximal end of the
binocular visual line. On the other hand, insufficiency of the ex-
terni may occur at the distal end and be greater at the proximal,
or it may change to insufficiency of the interni. We may some-
times properly give prisms of different degrees for the respective
positions, but we ought not to give adductive prisms for distance
and abductive for near, until the full benefit of the former has been
developed by wearing them many weeks. It may also be remarked
that weak convex glasses sometimes take the place of adductive
prisms for near work, but not often in subjects with vigorous ac-
commodation. My conclusion from ample experience is strongly
in favor of the helpfulness of weak prisms continuously worn, for
moderate degrees of muscular error, and they have in my practice
largely taken the place of Dyer's method, of gymnastic prisms and
of the various palliative proceedings above referred to.
In giving prisms the rule may be formulated that the base
should be placed toward the image whose position is to be cor-
rected, and this corresponds to the weak muscle, provided the phy-
224
DISEASES OF THE EYE.
siological or functional action of the muscle is regarded. The apex
of the prism like a knife edge indicates the muscle which should be
weakened and the base denotes the muscle to be strengthened.
The resort to operation is next to be discussed. It was em-
ployed by French surgeons,1 Bonnet, Guerin, Cunier, so long ago as
1841 for the cases now under consideration, but intelligent adapta-
tion of it was first proposed by v. Graefe2 in 1869. He especially de-
veloped its employment for deficient adduction in myopia. We
have learned by large and ofttimes deplorable experience that
very great discrimination must be used when we employ surgical
means for a functional muscular error. We must clearly recognize
the distinction already indicated between cases purely muscular,
and cases where muscular errors are symptomatic of nervous dis-
turbances. These latter are of obscure pathology and a simple
mechanical correction may utterly fail of relief. It may be tem-
porary or partial, or it may issue in diplopia excessively distress-
ing, or in aggravation of previous sufferings. It is very difficult to
formulate rules for these cases, because we meet with such diverse
conditions.
The operations may have uncertain results, and must alwaj'S
be done without general anaesthesia; happily cocaine renders
this entirely feasible. The effect must be measured immediately
by prisms, and be controlled at least as soon as the following
day, because it may be in excess or deficient. Generally complete
tenotomy has been done; by some partial tenotomy has been em-
ployed (Alfred Graefe, Abadie, etc.), while Stevens employs " grad-
uated tenotomy," which seems to be almost a full tenotomy, 1. c,
p. 173; but he also seems to practise frequent partial tenotomies
at intervals of weeks or months, until the desired result is achieved.
With complete tenotomy the effect depends upon the extent to
which the oculo-orbital fascia is loosened (capsule of Tenon), and
this tissue varies much in density in different persons. As supple-
mentary to tenotomy, or as a substitute for it, Wecker has pro-
posed advancement of the oculo-orbital fascia; while advancement
of the tendon of the weak muscle is sometimes done.
The cases in which undoubted benefit is to be expected are those
in which the fault, whether of adduction or abduction, is of the same
quality both for distance and near, and in which there is a strong
impulse to binocular vision. With decided myopia, with anisome-
tropia, with monocular amblyopia, the latter condition is imper-
fectly fulfilled or is wanting. An operation may be advisable, but
the result will be less certain. When, however, the cases are of the
1 Annales d'Oculistique, tome v., p. 139, and tome vii., p. 73.
2 Klinische Monatsblatter, 1869.
ASTHENOPIA.
225
suitable kind, the surprising result is often seen, that the amount
gained bjr the weak muscles is greatly in excess of the amount
subtracted from the robust muscles; particularly is this true with
insufficiency of the externi. This proves that we deal with func-
tional conditions, viz., spasm and not exclusively with mechanical
data.
The effects of an operation may be satisfactory for some time
and be followed by renewal of asthenopia and of the evidences of
muscular error. It can also occur that the muscular discordances
may after months be re-established, while no asthenopic symptoms
have recurred.
Cases which show a marked abridgment of muscular power at
both ends of the visual line are unsuited to an operation. For
them, prisms, regulated exercise, and general tonic measures are all
that can be safely tried and some of these patients are in a pitiable
condition. For them, advancement of weak muscles has been
proposed, and Landolt records some successes. He also narrates,
with commendable candor, grievous failures.1 There is always risk
in advancement, of twisting the vertical meridian and introducing
a new and most serious element of asthenopic disturbance. Yet
by extremely careful operation and after-treatment this may be
avoided.
The results of a tenotomy may be satisfactory both mechanically
and functionally for months, and by gradual relaxation of the cica-
trix, diplopia may at length ensue and great annoyance. The field
of diplopia may be lateral, not nearer than 25° from the median
plane, and yet occasion great discomfort. If concave or prismatic
or other glasses are worn, the error may be partially or sometimes
wholly obviated. If, however, the field of double vision come close
to the median plane, great disturbance follows. Sometimes a pa-
tient learns to ignore the image of one eye, but the situation is re-
grettable.
It may be accepted as settled that tenotomy with free dissection
of surrounding tissue is not fitting in muscular asthenopia; that
with high degrees it is better to divide the operation between two
eyes and at a considerable interval; the incision should be exactly
at the implantation of the tendon and not in front of it. Graefe's
rule of operating applied almost exclusively to cases of marked
myopia, principally for insufficiency of the interni, and was to the
effect that the eyes must be in equilibrium for a point about 20° on
the side of the divided muscle, and on a plane 15° below the horizon.
If for example, the left externus is divided, the patient, with a red
1 " Refractive Accommodation of the Eye," translation, Edinburgh, 188G,
226
DISEASES OF THE EYE.
glass before one eye, should see a candle flame singly when held to
the right 20° from the median plane of the face, and depressed
about 15° below the horizon. A prism with base vertical is to be
put in front of one eye, and the two flames must stand perpendicu-
larly when the candle is put in the place of election above desig-
nated. An error of 3° in excess is to be corrected by a suture which
shall include the conjunctiva and more or less of the tendon.
This proceeding is oftentimes too liberal and liable to leave
diplopia, or such weakness of externi as to introduce another kind
of asthenopia. He assumed that the muscle to be divided must
always have a superfluity of energy, whereas experience shows
that this may be far otherwise. It is safer to test the result of the
operation on the median plane, and the weakened muscle must
always be capable of exertion. In this position an excess of 2° or
3° is not serious, is often allowable, but the due preponderance of
the interni must always be respected, and some abductive power,
say 2° or 3°, should always remain.
The test by a vertical prism is to some degree fallacious. It
does not always teach the true place of equilibrium and if it show
remaining error, while tests of adduction and abduction are satis-
factory, the evidence of the latter is to be preferred until future
developments indicate what should be done.
My preference has been to perform complete tenotomy and con-
trol its effect by a suture. Partial1 tenotomies have in my experi-
ence been extremely uncertain and usually ineffective. With their
frequent repetition at intervals of one or several weeks, as done by
Stevens, I have had no experience. In operating, use a 4$ solution
of cocaine. The wound is to be over the middle of the tendon and
it is best to use forceps with projecting teeth which shall seize both
tendon and conjunctiva. The scissors should be sharp-pointed and
curved on the flat, the tendon is cut close at the sclera (a very
useful scissors has been devised by Stevens, with blades made
narrow for about one-third of an inch from the slightly blunted
points; they are curved on the flat); carry under it a very small
blunt hook, and cut off the insertion of the upper half of the tendon,
one blade of the scissors being in front of, and one behind the inser-
tion; put a second hook under the lower half of the tendon and sim-
ilarly sever that. Keep close to the insertion and endeavor to avoid
bleeding. Dr. Stevens' scissors with much reduced tips are well
suited to making small wounds and may render the use of hooks
unnecessary. Immediately test the effect by prisms and the candle,
and modify the proceeding either for increase or diminution. En-
deavor to make the wounds so small as not to need a suture, but if
1 See Standish, Trans. Am. Oph. Soc, 1889, p. 386, who practised partial
tenotomy, with good effect, but the amount divided was not small.
ASTHENOPIA.
227
too much effect has ensued, it must be employed. A light bandage
may or may not be used, and usually 10 to 20 grains of phenacetine
will be needed in two hours. There will be in normal cases a re-
duction of effect during the next two weeks; sometimes this needs
to be opposed by wearing prisms suitably placed. The immediate
result of an operation will vary from 6° to 15°, and each case must
be regulated according to its own peculiarities. It may happen
that very little effect ensues, while on the contrary for apparently
equal amounts of interference a large deviation may follow. This
uncertainty depends both on the muscular power of the antagonist
and on the density of the capsule of Tenon. Effects so small as 5°
may easily be secured. Partial tenotomy may give effects varying
from 0° to 3°. If it do good, as is alleged, the effect must largely
depend on setting aside a state of muscular spasm, about whose
existence I think, with Loring, there can be no doubt.—See Trans.
Am. Oph. Soc,
If the interni are to be relieved in cases of myopia by division of
the externi, it is wise not to have more than 3° convergence on the
median line. This will after a few weeks permit of a second opera-
tion at which a closer correction can be secured. The ultimate
result after six or twelve months is liable to be quite different, either
by return of the original disproportion, or free tenotomy may
eventuate in permanent homonymous diplopia. For this reason it
is wise to proceed by careful steps as indications arise. Partial
alleviation of symptoms is more frequent than complete cure.
In operating on the interni for relief of the externi, one may aim
at an immediate abduction of 8° or 12° according to the power of
adduction. The higher the adduction, say if 50° and more, the
greater will be the permissible amount of abduction procured, even
to 15°. But with adduction not above 30° caution must be used and
the resulting abduction kept below 10°. Sometimes only a slight
effect is immediately gained, which increases within four days. In
other cases the maximum is at once attained. Increased effect can
be secured by wearing appropriate prisms. In all cases close
attention must be given to the daily progress of events, and when
needful, interference promptly employed. If an excessive effect
appear, a correcting stitch can be inserted on the second or third
day, b}T opening the wound with a fine strabismus hook.
It is hard to resist the temptation to quote cases, but that would open
too wide a gate. Without abating the general approval which is intended
to be placed upon tenotomy for the relief of headache, eye-strain, and irrita-
tions, both local and general, and placing myself squarely among its staunch
advocates, some results of experience which have been contrary to just ex-
pectations must be candidly mentioned. I have in three cases done teno-
tomy of the interni for extreme headache in persons who had marked de-
228
DISEASES OF THE EYE.
grees of insufficiency of the externi. They were highly neurotic. There
ensued great relief of headache, but the capacity for eye work was deplor-
ably abridged, although diplopia was not produced. Schweigger's small
perimeter is the best instrument for ascertaining the lateral limits of binoc-
ular fixation. On the other hand my present attitude on this subject is
justified by the case of a gentleman having myopia and weak externi, seen
first in 1863, who was then forty years of age. He had myopic astigmatism
— ^s.q — ^c. V=liL. Always had headaches. Was treated by cups to
temples, mustard footbaths, bichloride hyd., and gained no relief from head-
ache by glasses or by treatment. In 1888 was found to have commencing
sclerosis of lens in 0. S. and vision very defective. In 0. D.— 4.50s.C — 1.00c.
90°. V=0.6. In 1890 O.D. about same; O. S., V = fingers at two feet. Con-
tinues to complain of headache, often waking with it in the morning; cannot
wear distance glasses with comfort. Find that he has homonymous diplopia
at 18', both with and without glasses, requiring prism 9°. Ordered prisms
O. D. 5°, O. S. 4°. In five days convergence increased to 15° at 18', while with-
out glasses at 10" has 9° divergence; with — 2D at 13" has equilibrium. After
wearing prisms sixteen days reported almost complete relief of headache.
One week later found at 18' convergence of 15° permanent with diplopia and
did tenotomy of rectus internus O. S. The effect was at first controlled by
stitches, but these were removed, and finally at 18' a convergence of 7" was
left. This on the next day was reduced to 3°. In four days the convergence
at 18' was 1°. For twelve days kept a patch over operated eye. For 18' has
equilibrium, but very little muscular flexibility, to which the extremely dim
vision of O. S. (cataract) contributes. For the near point has crossed di-
plopia (divergence) of about 50°. By wearing full correction this is removed;;
with his glasses at 18' abd. = 0°, add. = 8°. Has considerable difficulty in bi-
nocular vision for the near. Eight months after the operation reported that
there had been great diminution in the frequency of headaches; they would
sometimes come and were severe. Cannot use eyes any more than he did
previous to the tenotomy. Still has some diplopia, but images at 18' very
close together. This case was a rare one of weak externi in myopic eyes.
Not recognized when first seen in 1863, and not until twenty-seven years had
passed, when patient had become sixty-nine years old. The nervous strain
remained, the muscles were necessarily very weak, and the partial response
to their relaxation by both prisms and tenotomy is a convincing argument
in favor of the views presented. Perhaps a more careful tenotomy followed
by another operation after a few months would have given better results.
Another case of exceptional type, but having the guarantee of success by
tenotomy after eighteen years' experience, is one of hyperopia with insuffi-
ciency of interni. E. N. B., a boy, thirteen years, with O. D. -f- J, V = §£; O. S.
-f-£, V=f#. Had severe pains and occasional diplopia. For six months wore
4" v C prism 4° base inward. In January, 1869, did tenotomy of both externi at
one sitting. The measurements of muscular error are not complete. It appears
that with his glasses and vertical diplopia there was 6° divergence at 20' and
14° divergence at 12". Six weeks later had with glasses at 20' add. 10°, abd. 3°. In
1873 was examined again, and vision had improved. O. D. -f-tV* V = ^; O. S.
-f- ttjS- - Ac 180°, V = J£. With glasses at 20' add. = 16, abd. = O, with v.d.
4° convergence; with glasses at 12" v. d. 7° divergence. Is preparing for col-
lege and reads three to four hours daily without pain. In 1887 was in-
formed that he is practising law in Ohio and eyes never give any trouble.
In 1865 a boy, fifteen years, with M i and insufficiency of interni, had com-
plained of pain and asthenopia for four years. Material relief was gained
ASTHENOPIA.
229
by — tV C prisms 4° bases inward. The amount of error is given only for 12",
viz, 12° divergence with vertical prism. The rectus externus 0. D. was
divided. The consequent homonymous diplopia continued for a short time
and gave place to normal motility. Symptoms disappeared. After two
years, viz., in 1868, trouble again came on, bringing about the discovery
and correction of an important amount of astigmatism. He was ordered:
0. D. - |s. C TV<5- 180°c. V = f» -f,
O- S. - TV». C tVc 180°c. V = fft +.
For reading:
O. D. - TVs. C - tVc 180°.
O. S. -ts-CAc. 180°.
Up to 1889 the correcting glass remained almost unchanged and visual
acuity was normal. He went through college, became a lawyer, always
read prodigiously and rapidly. The muscular conditions then (1889) deter-
mined were:
At 18' with glasses abd. = 5°, add. = 15°, with v. d.-^-.
At 13" with glasses abd. = 15°, add. = 18°, with v. d. = 5° div.
In March, 1892, the record was substantially the same. He has never made
any complaint about his eyes and continues to use them unsparingly. In
his case the cause of trouble was the muscular disturbance. Later the astig-
matism came to the front. The preservation of healthful and vigorous eyes
with notable and unequal degrees of myopia throughout a period of twenty-
seven years under observation is an encouraging and noteworthy fact. It
is right to say that during twenty years he employed one pair of glasses for
all work ; that there has not been any choroidal crescent about the nerves ;
that in 1873 he had the power of overcoming a prism of 10° with base vertical.
My experience on this subject within ten years has been very large, and
to cases within this period I make no reference. Examinations have been
made more precise and interference has been ventured on in cases for which
formerly nothing operative would have been done. The combination of
prudence and boldness is absolutely essential in all these cases.
Reference has been made to errors in a vertical plane as well as
in simple adduction and abduction. Dr. Stevens1 finds these quite
frequent and operates for them when not greater than 1° or 3°.
I have sometimes corrected them by prisms and have in a few in-
stances operated. I have found in most cases that correction of
errors of adduction and abduction carried with it the adjustment
of the error in the vertical meridian and that patients desired
nothing more.
A few words may be said about advancement as contributory to
better equilibrium of muscles. My experience with this proceeding
for asthenopia is recent and limited. I have had to do it for the
undue effects left after tenotomy done both by myself and others,
and have also done it to reinforce tenotomy of antagonists. If one
secures small curved needles, the so-called "quarter round," and
has them sharpened as if they were meant to penetrate the cornea,
there will be little difficulty in accurately dosing the effect of the
1 Archives for Ophthal., xvi., 2, June, 1887, p. 149.
'.30
DISEASES OF THE EYE.
proceeding. A sufficient crescentic piece of conjunctiva must be
excised right over the site of the insertion, the stitches must include
the tendon and superjacent fascia, and the thread must have a
needle at each end. To get a good anterior hold, the needle will go
through the sclera easily and deeply enough, to answer all required
traction, while the approximation of the wound will be done by
forceps seizing the parts if there be much separation. One may
use two or three sutures and I have found no puckering and very
little reaction. In two instances I have taken a piece out of the
tendon and stitched the distal part to a bit of the insertion left as a
stump. Dr. Stevens practises an operation of this kind with very
little dissection of tissues. Experience is as yet so limited, that one
can only suggest rather than recommend.
The topic we have thus considered is one which is at the present
time actively discussed among ophthalmologists. Opinions vary,
and the writer, while frankly avowing his own views, founded on a
large experience, has only stated what thoughtful and careful ob-
servation has taught him. Many things have been left unsaid,
because in this field clinical experience is wonderfully diversified
and more detail would hardly be suited to a text-book. Even the
insertion of illustrative cases, to which there is strong temptation,
would demand more space for adequate presentation of the varied
phases of the conditions we meet than can be afforded.
See Burnett, " Contributions to the Study of Heterophoria,"
Trans. Am. Oph. Soc, 1891, p. 217—a well-digested statement of
cases.
PART SECOND.
CHAPTER I.
GENERAL TREATMENT OF DISEASES OF THE EYE.
We first speak of its proper protection from hurtful influences,
viz.: from dust, smoke, glaring light, and extreme heat, by colored
or transparent glasses, by shades, by seeking another locality, by a
bandage, by seclusion in a dark room or in bed. Protective glasses
are known usually as coquilles, are shaped like a watch-glass, and
tinted either London smoke or blue, in various shades, known by
letters A, B, C, D, etc. Very dark shades are objectionable in most
cases, because they so diminish the light that the eyes are strained
in gropinir about. The neutral tint is generally better than the
blue. Blue glasses improve the distinctness of sight to some de-
gree, in certain conditions. Workmen exposed to injury by chips
of metal may wear large glasses of mica, if they will, but they are
seldom inclined to accept them. Eye-shades may be single or
double; they should be shaped according to their purpose; if to cut
off light from above, as in reading, they should flare like a cap-
front; to cut off light in all directions, they should lie flat and come
around well on the temple. To lie flat, they should have a notch
for the nose, be three inches wide, come to the temples, and will be
kept flat by having the strings fastened three-fourths of an inch
below the corners; these must go twice around the head. A mon-
ocular shade to keep the lids closed, should be an oval whose length
should be about two and one-half inches, and breadth one and three-
fourths inches—the string fastened at the ends, and to go obliquely
over the forehead and under the corresponding ear. If required, a
packing of cotton may be put under it. A bandage should be made
of thin flannel (i.e., merino, Avhich is a texture of both wool and
cotton), be three and one-half yards long, and one and one-half
inches wide, for an adult. In summer, muslin gauze may be substi-
tuted. The width will be less in some cases, and always less
for children. It goes about the head like a figure eight, and presses
232
DISEASES OF THE EYE.
the eyeballs through a packing of absorbent cotton laid upon
patches of muslin. To adjust a single or double bandage smoothly
and firmly, requires a little practice. It is usually employed
where some pressure is to be exerted on the eye. Some oph-
thalmic surgeons prefer silk plaster which may be white or black
and is laid on in strips or in one patch. When patients are
kept in dark rooms, it is important not to have streaks of bright
light at the edges of the shades or in the shutters. It hardly
need be said that a patient wearing a bandage need not be im-
prisoned in a dark room; the moral influence is bad, and the phy-
sical effect on his attendants equally bad. I have known delirium
produced by no other cause, in old people, after cataract extrac-
tion. With dark rooms, unusual care must be given to ventilation
and cleanliness. Many serious eye diseases require confinement to
bed, and often it is difficult to make a patient submit to the hard-
ship. The object is quietude of the whole body and absolute rest
of the eyes, which a patient sitting in a chair or walking about
under a bandage will not and cannot so perfectly maintain. I ad-
vocate this only during the active period of acute disease—never in
case the general health suffers or is unfavorably influencing the
eye trouble. Even photophobia, which is usually the symptom
necessitating seclusion in darkness, is sometimes aggravated by
such confinement, especially in hysterical persons, in weakly or
scrofulous children, and when the fear of light has outlasted the
cause which originally excited it. To this point Dr. Agnew has
.called especial attention. Such persons must be provided with
smoked glasses, and sent outdoors to navigate for themselves. A
proper understanding of hygiene and of the conditions of healthy
nutrition in food, clothing, exercise, and air and occupation, is of
the utmost importance in ophthalmic treatment. I shall have to
emphasize this repeatedly.
Protection from contagion may be secured by mechanical means
such as bandages, and other devices, but special regard is to be
paid to asepsis and antisepsis both in ocular pathology and in ocu-
lar surgery. Besides the exposure to atmospheric germs which in
tenement houses, asylums, hospitals, and barracks may become very
grave, we always have bacteria and cocci of various kinds contained
in the conjunctival secretion. They abound in the nasal cavity and
with flagrant intensity in ozaena, and may be transmitted to the
eye from the nasal discharges or by the lachrymal puncta. In
the eyelashes, eyebrows, on the skin of the face, both with and
without eruptive diseases, by the hands, by handkerchiefs, towels,
rags, etc., we have ready means of contagion. It is needless to
specify all that may be possible, one must inspect the wThole body
and know a patient's habits and surroundings to find and remove
GENERAL CONSIDERATIONS.
233
-all the sources of contagion. Complete and strict cleanliness of
person, clothing, arid surroundings is the first requirement in asep-
sis. Disinfection of rooms and wards, by vapor of burning sulphur
(with attention to the free supply at the same time of watery vapor
without which it is almost inert, but with which it is most efficacious
(Squibb)), and the addition of carbolic acid or corrosive sublimate to
the water used in scrubbing walls and floors, are important agents.
In applications to the eye we are obliged to exclude some recognized
agents or greatl}7 dilute them, because the organ is too sensitive to
bear them in effective strength. We therefore rely more on asep-
sis, of which cleanliness is the chief condition, than on antisepsis,
when surgical operations are to be done, and reserve antiseptics for
pathological conditions. We shall first speak of the latter. We
•employ them especially in diseases of the conjunctiva and cornea,
and while the utility of some of them has long been empirically
known, our better knowledge of their mode of action helps us to
use them more intelligently. Carbolic acid is little employed, be-
cause it must be diluted to 1$ or 2$ solution. Boric acid, whose
solubility is 4$, has wide application, because, while not really an-
tiseptic, it is soothing and can therefore be employed freely to wash
out morbid secretions. We separate the lids and lift them from
the globe, perhaps by elevators or even a speculum, or by nipping
the skin of the lid with the fingers, and wash out the conjunctival
sac with a rubber bulb holding about two ounces; the whole cavity
must be flooded. If there be much swelling of the lid or che-
mosis, this may not be feasible to the full extent. Solution of cor-
rosive sublimate 1:10,000 does not irritate, and 1:5,000 is easily
borne. In severe morbid conditions even 1: 2,000 may be toler-
ated, but the quantity will be small and a dropper employed instead
of a bulb. A normal eye will sometimes show unpleasant reaction
to solution 1:10,000. A third agent, and which completes the list
of those most in use and commonly resorted to as antiseptics is
chlorine water, or, as usually dispensed, the liquor sodas chlorinataa
(Labarraque's solution), 1 part to 7 or 10. This is used with a drop-
per. Next we mention the actual cautery employed often with sig-
nal success in suppurations and ulcers of the cornea, less frequently
in trachoma, either as the thermo-cautery of Paquelin or as the
galvano-cautery. It will be referred to again.
Hydrochinon and resorcin in Sfo solution have been employed
with no special superiority over the fluids above mentioned.
Salicylic acid is not much used except upon dressings. It is com-
bined sometimes with borax, viz., a mixture of the two, each in 5$
solution. Benzoate of sodium, 5#, is counted antiseptic, and thymol,
1 : 1,200, yet they are rarely used in eye surgery.
Iodoform in very fine powder—a point to be insisted on—is bv
234
DISEASES OF THE EYE.
some much esteemed, yet by others is scarcely employed. Its
efficacy is slow, and it must therefore be kept in contact with the
tissues for a long time. It may also be mixed with vaseline and a
bandage applied. Used chiefly in corneal ulceration and suppura-
tion, it has been also lauded in purulent conjunctivitis, especially
by English practitioners. Aristol is equally effective and is free
from objectionable odor. Pyoktanin or methyl violet, 1 to 500 or 1
to 5,000, has value but not to a remarkable degree. Nitrate of sil-
ver plays a great part in treating external diseases of the eye, and
some of its potency is due to its antiseptic properties. Marpman
says that in putrefying solutions of albumen, decomposition is
arrested by solutions so weak as not to cause coagulation. These
will be much less than one per cent.
We might enumerate other substances habitually resorted to>
whose efficacy in fact resides in their antiseptic qualities, such as
yellow oxide of mercury, acetate of lead, etc., but there is no need.
In operative work, of which extraction of cataract may be-
taken as the type, and in which the cornea and conjunctiva are
presumably normal, the employment of antiseptics has become
thoroughly established. Nevertheless the same rigor is not prof-
itable as in general surgery. Spray is never used; solutions which
are in vogue are: boric acid, 3< or Vr. corrosive sublimate,
1:5,000 or 1:10,000; and biniodide of mercury, 1:20,000 to which
a little alcohol is added (Panas' solution), and boiled water.
As to the need for them we may bear in mind the investigations
of Fick,1 that in 49 perfectly normal conjunctival sacs, he found
only 12$ without bacilli, and 36 affected with slight catarrh contained
them. He found bacilli, cocci, and tetrades or masses of sarcini. He
enumerates seven different bacilli, three kinds of cocci, including the
staphylococcus aureus, the pyogenic form, and sarcini (p. 54, I.e.).
But more notable are the experiments of Gayet, of Lyons, who
after carefully disinfecting the conjunctival sacs of his cataract
patients, found by cultivations that microbes remained in 75<, and
from 213 test-tube cultures, he concludes that antiseptic or aseptic
fluids have very little influence over germs in the conjunctiva. It
follows that copious washing is an essential factor, and because
mercurial salts coagulate secretions, and Panas' solution, viz., hy-
drargyri biniodidum 1 : 20,000 is entirely unirritating, these are
to be preferred when slight catarrh exists. In other cases the
fluid must simply be itself free from germs and, as already said, be
freely used. Hence the utility of boiled water.
We have also to bear in mind that wounds and manipula-
tions must be made with the least possible bruising and violence,
and here lies the crowning importance of neat and skilful operat-
1 " Ueber Micro-Organismen lm Conjunctivalsack/' Wiesbaden, 1887.
GENERAL CONSIDERATIONS. 235
ing. If there be failure here, the soil is prepared in which germs
delight to multiply. We must also avoid furnishing the fruitful
soil in another way, viz., we must not operate if we can help it,
when the constitutional conditions are unfavorable, i.e., in presence
of rheumatism, syphilis, or any severe dyscrasiae; or if the disease be
incurable, like diabetes, we must select the most favorable time. For
similar reasons, conditions especially promotive of germ growth,
like trachoma, pterygium, lachrymal diseases, chronic catarrh, must
be cured, if possible, before operating. The most scrupulous care
about disinfecting instruments, sponges, cotton, dressings, the hands,
of all who have to do with the patient, and also the surface of the
patient, his clothing, bedding, etc., must not be in the least remitted
or abated. For the hands nothing equals soap and water applied
with a stiff brush and with the addition of powdered borax to pene-
trate under the nails. For instruments, soaking for thirty minutes
in 5$ carbolic-acid solution; while boiling water, for scissors, spec-
ula, forceps, and all instruments having joints and teeth is the
best. Flat porcelain trays can be had in which to soak instru-
ments, and some may be well brushed. The cutting edges of knives
are liable to be dulled by carbolic acid, and they may be wiped with
a clean rag after short immersion and then inspected with a mag-
nifying glass. Immersion in alcohol helps to insure cleanliness by
removing grease, but it is not antiseptic. Careful wiping by a
moist and then by a dry rag is most essential. No specks of rust
or stains should be tolerated, any more than a dull edge. In test-
ing the edge and point, put several thicknesses of fine leather shav-
ings or of gold beater's skin on the drum—a click or creak is the
signal for rejection. If a point be merely turned, it may perhaps
be straightened on the thumb nail. Test scissors on wet paper or
on fibres of cotton; be specially careful about their points, and the
edges should glide smoothly as the blades close.
This introduces us to the operative treatment of the eye. It
has always commanded great attention, and its scope has been
largely extended. A better knowledge of pathology, the invention
of new methods of operating, and higher skill in the manufacture
and adaptation of instruments have conspired to make the opera-
tive surgery of the eye one of the most brilliant chapters in medi-
cine.
Some general remarks are here in place. Shall anaesthetics be
used ?
Since 1884, when Dr. Carl Koiler called attention to the anaes-
thetic effect of muriate of cocaine dropped upon the eyeball, gen-
eral anaesthesia has been relegated to exceptional instances, in
ophthalmic surgery. A summary of the effects of cocaine upon the
eye is as follows; A 2$ solution, of which several drops are used,.
236
DISEASES OF THE EYE.
causes at first a slight burning and smarting, then the lids open to
an unusual extent, anaesthesia appears in from ten to twenty min-
utes, the eye becomes pale by reduction of the size of the vessels,
the pupil dilates moderately and the accommodation is slightly im-
paired. A 4$ solution acts more speedily and affects the pupil and
ciliary muscle more decidedly. The anaesthesia lasts about ten
minutes. If the solution is dropped in again after three or four
minutes, the effects are more speedy and last for fifteen to twenty
minutes. Two or three instillations of 4< solution at intervals of
ten minutes is commonly used for cataract operations. For stra-
bismus the solution must also be dropped into the wound and will
often sufficiently obviate pain. For enucleation the same has been
done, but with less success, and deep injections have been practised
into the orbital tissue of 2$ solution, but some risk of toxic effect is
incurred. For iridectomy, if the iris be prolapsed, or a drop injected
into the anterior chamber, pain upon excision of the iris may be
abolished; otherwise not, unless a full dilatation of the pupil has
been obtained, which implies its absorption and more or less decided
anaesthetic effect. Besides the above effects, tension of the globe is
reduced in most instances. Contrary effects, viz., increase of ten-
sion, have been exceptionally noted when there was a tendency to
glaucoma and the exaggeration has been caused by mydriasis. In
almost all cases tension becomes subnormal.
Its effects are exerted on both the sensory and sympathetic
nerve fibres and hence its constricting influence on the vessels, and
reduction of intraocular tension. It also constricts the lymphatics
and hence dries the cornea, and frequently causes exfoliations and
slight erosions of its epithelium. Indeed the remarkable effect was
seen by Dr. Gruening of the whole epithelial covering of the cornea
being lifted in a bleb, by exosmosis of the aqueous in a case of
glaucoma for which he ventured to use cocaine preparatory to
iridectomy. For advanced chronic glaucoma it is inexpedient to
use it; for glaucoma simplex and for acute glaucoma it is admissi-
ble. In operations on the lachrymal apparatus it has very limited
effect and to reach the duct effectively it must be injected by a
syringe. In operations on the lids, cystic tumors, etc., hypodermic
injection within the area inclosed by a clamp forceps makes it per-
fect master of the situation (2£ solution); without the clamp it
speedily diffuses and is less satisfactory. For all operations on the
cornea it is invaluable, especially in removing foreign bodies, in
scraping ulcers, using the actual cautery, making punctures and
incisions. Even when general anaesthesia is required because of the
high excitability of the patient or of the severity of the operation,
it is a useful adjuvant. It has no prejudical effect on the healing
process and rarely causes toxic effects.
GENERA L CONSIDER A TIONS.
237
Its application in inflammatory troubles is wide, viz., in chronic
conjunctivitis, ulcers and phlyctenule of the cornea, in the pain of
iritis, in combination with atropine, and in combination with eserine
in especial cases of glaucoma where an operation is unadvisable.
Its power of temporarily controlling hyperaemia as well as sensibil-
ity, suggests many opportunities for its employment. Although
temporary in its effect, it gives opportunity for examinations and
applications of more positive value. Where great hyperaemia ex-
ists it is less efficacious, and its hurtful effects on the corneal
epithelium are less. In facial neuralgia a solution of 10# to 20£
dissolved in oleic acid or mixed with lanolin and rubbed along the
painful nerve has a controlling effect.
As spray in 5$ or 10$ solution it may be applied to trachoma,
when about to be cauterized, or a fleck of absorbent cotton laid
under the lid soaked in 10$ solution will so much control sensibility
that some patients will even bear the actual cautery in the cases
suited for its employment.
Something must be said upon its toxic constitutional effects.
As with all other drugs, persons exhibit most various degrees of
susceptibility. The usual constitutional symptoms are imperfect
and sighing breathing, pallor, indifference, unconsciousness or
coma, seldom delirium, rapid and weak pulse, which often does not
go above 90, but I have seen it go to 120, and may intermit and
even go much higher. Less frequently there is giddiness, nausea
and vomiting, slow speech, extreme sweating, a red rash, spasms
of the limbs. The direct effects are on the heart and respiration.
The antidotes are whiskey, ammonia, nitrite of amyl, digitalis, sin-
apisms, friction and heat, hypodermic injection of morphine. In
very sensitive persons and for prolonged operations we must use
ether and chloroform. Therefore plastic operations, many lid oper-
ations, enucleation, neurotomy, and sometimes tenotomy and
often advancement of mucles will need general anaesthesia. As
to operations which enter the globe: paracentesis seldom needs
it; iridectomy may often be done without it, but it is more satis-
factory to have the patient passive by general anaesthesia.
Dr. Norris, of Philadelphia, has lately called attention to cases
of fatal results of etherization in patients having Bright's disease.
Experience has shown the value of this caution. The fatal result may
not occur until after forty-eight hours, and it is to be heeded especi-
ally in the fibrous kidney. For children under ten, I use chloroform,
for older persons, ether by preference, but not seldom chloroform.
When a long operation is expected, and the person is feeble, ether is
to be chosen. For a quick operation I often administer chloroform.
The primary stage of anaesthesia, before muscular relaxation has
come and consciousness is not fully destroyed, and which lasts only
238
DISEASES OF THE EYE.
part of a minute, requires a very small quantity of either ether or
chloroform, and can often be seized as the happy instant for making
an incision which will perhaps be all of the operation that the patient
would not be well able to bear. Mr. Priestley Smith has suggested
a full dose of bromide of potassium an hour before an operation, as
a means of allaying excitement, and rendering a patient more sub-
missive to the anaesthetic. I often give sodii bromid. 3 ss.-i., chloral
hydrate gr. xv., the previous night, and repeat the dose an hour
before the operation. The anaesthetic is more willingly accepted,
and vomiting is less liable to ensue. In eye operations the ill-effects
of vomiting are more serious than in general surgery, by promot-
ing prolapse of iris, loss of vitreous, and intraocular hemorrhage.
.For several years I have used an ether-inhaler which is valuable
Fig. 87.
because it takes up little space and offers the least obstruction in
operating about the eye. It has a rubber face-piece, and has a
dried bladder at the distal end of the box, which affords space for
vapor.
Eye operations should be done with the patient upon a table or a
narrow bed. Operating chairs are by some preferred and may be
taken into the patient's room: this applies to hospital practice. A
liead-rest or clamp is useful, but an assistant can do this service.
An operator who is ambidextrous will always sit behind the
patient's head, on whichever eye iie may operate. But he will
usually have to take his right hand for scissors, and sometimes,
therefore, come to the front. One who is not equally apt with both
hands, will change his place as the position of the eye or the place
of operation requires. It is a great convenience to enjoy perfect
use of both hands, but to many the accomplishment is never suffi-
ciently realized, to warrant risking a patient's sight by a clumsy
hand. As to brilliant display before spectators, no conscientious
man would harbor the thought to the peril of his patient. How to
gain needful skill ? There must be an original endowment of facil-
ity of hand and a mechanical bent of mind. Practice upon fresh
cadavers will teach something, but in them the eyes are too soft to
GENERAL CONSIDERATIONS.
239
be suitable. Pigs' eyes mounted in an operating mask, or, in lack
of this, fastened into the mouth of a bottle by a section of rubber
tubing of proper size, or by strings, will teach one how to manipu-
late in the anterior chamber, and the resistance in cutting the
cornea. A light touch and steady hand, and sensitive appreciation
of weight and resistance, are essential qualities.
It is desirable to have the least number of assistants. In most
cases but one is needed; sometimes a second, to give an anaesthetic
and keep the head steady, is desirable. In manipulating the eye
the operator should steady it by fixation forceps, and not let the
assistant do it when avoidable. He thus keeps the command, and
can co-ordinate his hands with accuracy. A fit speculum to keep
the lids apart is an important instrument. It must open them ad
maximum; it must not press on the globe; it must be out of the
Fig. 88
way of the operator. I have experimented extensively with these
contrivances, and find none perfectly adapted to all cases. The
form which I prefer is shown in Figs. 87, 88.
One which opens from the nasal side is sometimes convenient,
especially if the globe is very deep, and gives a large field for work.
But for deep eyes all such contrivances are imperfect, and one
should have a smaller one for children and a larger one for
adults. In case a speculum is impracticable, the operator may
lift the upper lid himself by the point of his index finger. He draws
up the lid by the skin as far as may be, then places the tip of his
index beneath the edge of the lid, and pushes it back into the orbit.
He does not drag or lift, but presses it under the orbital roof as he
would push a sliding cover into the grooves of a box. If he does
not choose to do this because the finger takes up room, he may use
Desmarres' elevator, which is often necessary in examinations of
the eye in children. It is made in sets of two and three sizes. An-
other elevator which I use in operating when a speculum has been
taken out, or when I wish to expose the upper part of the globe to
the fullest extent, is made of fine steel wire and presses the lid
farther under the orbital roof than anything else can, and it need
not make pressure upon or even touch the globe. These instruments
are figured in the catalogue of Geo. Tiemann & Co., New York.
A simple strabismus hook will often be valuable as an elevator.
Fixation-forceps are made with and without a spring catch.
240
DISEASES OF THE EYE.
They should be used so as to turn the eye, not to drag it; the line
of push must be at a tangent to the globe. If, for instance, the eye
is to be turned down, the forceps will be attached just below the
corneal margin, and be held perpendicularly to the globe while the
latter is rotated down and the forceps take a direction approaching
a tangent. Another way, often useful, and which is very conveni-
ent for the operator, is to apply the forceps at the same place, to
turn the eye by the same manoeuvre, and then bring the top of the
forceps up to the root of the nose, and a light push keeps the eye
down and exerts the least pressure. With anaesthetics, the forceps
may even be left to fall obliquely over the supra-orbital notch and
keep the eye down while the operator uses his hand for another
purpose. He may give the forceps to an assistant, and its position
will not need to be altered. The conjunctiva is liable to tear, and
the patient must be persuaded rather than forced to turn the eye
as desired, while the forceps simply maintain the position aimed at.
Regarding other instruments, they will be considered when
their special uses are to be described.
Medicines which have a special applicability to the eye, are those
which act on the pupil and the ciliary muscle, viz., mydriatics and
myotics. Of the former we have atropiae sulphas, duboisia, homa-
tropiae hydrobromas, daturiae sulphas, hydriodate of hyoscine. All
of them are poisonous, and can exert toxic effects when used in
sufficient strength as collyria, because they go through the cornea
by endosmosis and enter the circulation by solution in the aqueous.
humor. They also pass down the tear-passages to the throat, and
are there absorbed. Sulphate of atropia is the most common of
these remedies. It affects the dilator iridis before it affects the cil-
iary muscle. It likewise is an anodyne to the sensitive nerves of
the cornea and iris. It is used in solutions from one-fourth grain to
sixteen grains to the ounce. It was erroneously thought by Graefe
to relieve intra-ocular tension ; when it fully paralyzes the cil-
iary muscle and iris, the eye often feels much relief. On the con-
trary, all mydriatics, even cocaine muriate at times, intensify intra-
ocular pressure simply because the iris is pushed toward the angle
of the anterior chamber (Holtze and Graser), while myotics dimin-
ish the pressure because the iris is pulled away from the angle.
Atropia sometimes irritates the conjunctiva and after long contin-
uance excites papillary conjunctivitis. Occasionally even the skin
of the lids becomes erythematous. To render unpleasant consti-
tutional effects less probable, atropia may be mixed with vaseline,
or dissolved in castor oil (Green). If toxic effects appear, they will
come as dryness of the fauces, which need not be heeded; but more
important are, quickening and weakness of the pulse, flushing of
the face, palpitation of the heart, headache, nausea, prostration*
GENERAL CONSIDERATIONS.
241
garrulous delirium, desire to urinate, and sometimes muscular vio-
lence. The antidote will be brandy and morphia, and in urgent
cases hypodermic injections of muriate of pilocarpine, gr. \, every
fifteen minutes. Duboisia has more effect on the eye than atro-
pia and it does not irritate the conjunctiva. Its toxic effects come
more quickly and are more alarming, the prostration being ex-
treme. Homatropia, a derivative of atropia, acts more feebly than
either of the preceding. It dilates the pupil, if used in the strength
of gr. iv. ad oz. i., in about half an hour, and has moderate effect
on the accommodation; but in twenty-four hours its influence is
gone. It is serviceable for purely ophthalmoscopic work, but must be
taken at gr. xx. ad % i. for refractive determinations. The full effect
of atropia, whenever obtained, will last for from seven to twelve
days. Of daturine nothing need be said. Equal parts of hydro-
bromate of homatropine and sugar of milk have been used by Mit-
tendorf, dusted in the eye with a brush. Hydriodate of hyoscine is
our most powerful mydriatic. Hirschberg reports that \i> solution
is liable to cause toxic symptoms. Emmert used it with safety in
■jV # solution, i.e., gr. ^ ad § i.
The important myotics are the preparations of Calabar bean,
the sulphate and salicylate of eserine, and the alkaloid of jabo-
randi, hydrochlorate of pilocarpine. The eserine preparations should
be used in solution, gr. ss. or gr. i. ad § i.; if stronger, they become
very painful by exciting spasm of the ciliary muscle. They stimu-
late the sphincter iridis and the ciliary muscle, and irritate the con-
junctiva. Their effects are more fugitive than those of atropia in
equal strength. In strong solution they excite pain in the eye and
around the orbit, and can even cause clonic spasms of the extrinsic
muscles. They reduce ocular tension and it is claimed by some
that they reduce the calibre of the vessels. They are used in in-
flammations of the cornea with much confidence. The solution
of the sulphate undergoes change to a reddish color, which some-
what abates its efficacy.
A second myotic, but far inferior to eserine, is the alkaloid of
jaborandi, viz., pilocarpine, of which the hydrochlorate and the sali-
cylate are the preparations in use. Its value in treating diseases
of the eye rests more upon its constitutional than on its local
effects. As a topical application its minimum strength is 1 to 400,
and if stronger, its influence does not appear to increase (Jaarsma,
Thesis 1880, quoted by Landolt). It is usually prescribed in
strength gr. £ or gr. ij. It contracts the pupil moderately and
approximates the near-point and the far-point slightly. Its effect
on the pupil appears in about thirty minutes and lasts for twenty-
four hours. On the accommodation the effect begins in two and a
half hours and lasts two or three hours. It has therefore only
16
242
DISEASES OF THE EYE.
limited value in ordinary needs, but because it is not irritating and
topically devoid of unpleasant effects, it serves a good purpose in
mydriasis caused by paralysis of the third nerve, and in feeble ac-
commodation. Its constitutional uses are extensive and will be
duly considered. Its poisonous effects should be mentioned. Its
normal results are salivation and sweating, but added to these
are temporary increase of urine, thirst, vomiting, nausea, belching,
colic, and diarrhoea. The pulse is at first increased, then becomes
normal or too slow. The action on the heart is through the pneu-
mogastric nerve; prostration is caused by nausea and vomiting more
than by a specific influence. But it is recognized to be a most capri-
cious remedy and must be given internally in small doses, viz., 0.01
or 0.02, i.e., ^ Sr- to i gr. Its antidote is atropine and homatropine,
besides general stimulation, galvanization, etc. The hypodermic
injection of the muriate of pilocarpine (gr. ^ to gr. |) has seemed to
do good under certain peculiar conditions; for instance, in the late
stages of chronic keratitis or scleritis, especially in gouty subjects,
and also in the late period of gouty or rheumatic iritis, and in
serous uveitis. Virtue is claimed for it in subretinal effusion. On
the whole, the remedy has seemed to me to be overrated, although
its powerful action on the salivary glands and on the skin gives it
influence over local disease which doubtless can be sometimes suc-
cessfully applied. As yet the indications for its use are not pre-
cisely formulated. A case is recorded where by mistake a 20$
solution was hypodermically injected (Sziklaix). The case was one
of absolute glaucoma in which sclerotomy had been done and 2$
injections of pilocarpine employed. When the tenfold dose was
given, the symptoms were abundant salivation and sweating for
five hours. There was copious evacuation from the bladder and
from the bowels, vomiting and belching, from time to time squeez-
ing and tearing pain in the eyes. Afterward great prostration.
Treatment is not related. Vision said to have been impaired and
so remained for two years, but to what degree is not stated.
The Turkish bath is a similar measure, and is to be employed
in similar conditions. . It has decided value, but it is also capable of
mischief if not properly regulated.
Another myotic is muscarine, the alkaloid of Amanita muscaria,
which is little employed, but may be mentioned because, unlike eser-
ine, it causes slight contraction of the pupil, but strong spasm of
accommodation. It is very powerful and acts more on the punctum
remotum than on the punctum proximum. It may be used in
solution 1 to 400 or 1 to 100. Its antidote is atropine, but not vice
versa.
There are other mydriatics which might be mentioned, as gelse-
1 Jahresbericht fib? Ophth., 1881, p. 2G3.
GENERAL CONSIDERATIONS.
243
mine, daturine, which is identical with atropine, nitro-atropine, and
nitro-daturine, but they are seldom useful. Cocaine has slight
mydriatic effect, and a paper was written on this property of it
by Von Aurep in Archiv fur gesammte Physiologie, XXI., p. 38,
in 1879. Unfortunately its peculiar anaesthetic properties were
not then discovered.
As a summary of the whole: Among mydriatics we choose
atropine sulphate or salicylate, in cases where a prolonged effect on
the pupil and the accommodation is required. It is the mydriatic by
far most frequently used for therapeutic purposes. In case it cause
disagreeable effects, either local or general, we substitute duboisine
sulphate or salicylate. For a less prolonged but vigorous effect on
the pupil and accommodation we use duboisine gr. ss. ad oz. i., but
must be watchful against toxic influences. Where a brief effect on
the pupil and accommodation is desired, we use hydrobromate of
homatropine 1$, or muriate of cocaine 4$. The one most likely to
cause unpleasant constitutional effects is duboisine; that most
likely to produce conjunctival irritation is atropine. All are liable
with glaucomatous eyes to produce an acute attack, and in the
ratio of their energy. Of the other therapeutic uses of these rem-
edies it is not intended here to speak.
Of myotics the best is eserine salicylate or sulphate 0.1$ or gr.
ss. ad oz. i. It contracts the pupil, causes spasm of accommoda-
tion, and reduces intraocular tension. It also has other valuable
therapeutic effects in inflammations of the cornea, to be referred to
under that head. For a less energetic effect hydrochlorate of pilo-
carpine may be employed. While watery solutions are most com-
monly employed, mixtures with vaseline are very convenient, and
Dr. Mittendorf has introduced 1$ triturations which do not spoil
by keeping, are easily applied, and are very convenient. He uses
of either the mydriatic or the myotic 1; pulveris gum acac, 50;
pulv. sacchar. lactis, 50; M., to be dusted into the eye with a camel's-
hair pencil.
We are called upon to apply leeches, as, for example, for
severe inflammations, and for inflammations of the deep textures.
In reality they are not frequently employed. They should be
placed on the temple, and not too near the lids—never on the lids
or in their near vicinity. The artificial leech of Heurteloup is a cup-
ping instrument which draws blood rapidly, and is useful for deep-
seated congestions. It has quite superseded the ordinary cupping
apparatus. As a matter of fact, the abstraction of blood is re-
sorted to, in visible ocular inflammations to a much less degree
than formerly, and only in those which are attended by great pain
and hyperaemia. For deep seated diseases it is used in a way ad-
vised by Graefe. From one to two ounces of blood are withdrawn
244
DISEASES OF THE EYE.
rapidly from the temple, and the patient remains in a dark room
for twenty-four hours afterward. This proceeding is repeated once
in three, seven, or fourteen days, according to the character of the
case.
Blisters and external stimulants, such as tincture of iodine, are
not as much used as they formerly were. Their value as antiphlo-
gistics is almost nil, and they were formerly in favor because the
cases were too often incorrectly diagnosticated. As remedies for
neuralgia they sometimes are useful, and in a few other special
conditions.
Of external applications none is so common as water of various
temperatures, and its effect is modified in the most remarkable
manner by the mode of its use. For violent inflammatory attacks,
as after wounds or in severe purulent conjunctivitis, a block of ice
is kept beside the patient, and bits of muslin transferred from the
ice to the eye every minute so long as the symptoms demand
such extreme cold. We may use the water of higher tempera-
ture until it has no effect upon the surface, but serves merely
to soften the secretions. From this point we may go until we
get to 106° F. or 114° F. To keep the water cold the compresses
must be constantly renewed; so, too, in attempting to keep it
warm. To avoid such frequent change various contrivances have
been adopted. I sometimes let a patient hold a small piece of ice,
wrapped in muslin, upon the eye as long as it feels agreeable, and I
have used a small rubber bag as large as a hen's egg, filled with
ice, and stopped by a cork; but neither of these is very satisfactory.
Contrivances for keeping up continuous irrigation, by coils of rub-
ber or tin tubing, have been made (Becker, Chamberlain). For
most cases we need moist cold or moist heat, and this we get best by
compresses wrung out of water. Eye-douches are useful for certain
chronic cases and are easily contrived, and may be for warm or cold
water. They are used for only a few minutes at a time. For con-
tinuous moist heat, a good appliance is a poultice of ground slippery
elm (ulmus flava) bark. Spongio-piline dipped in hot water, covered
by oiled silk, is cleanly and serviceable. A bunch of absorbent cot-
ton is exceedingly serviceable. It is an old rule which holds good
to-day that applications to the eye should be of such temperature as
shall be grateful to the patient. This cannot be accepted abso-
lutely. For example while to the early stage of many external in-
flammations hot water is a relief, if kept up for several hours or if,
as too often is done, a hot poultice be bound on the eye, an cedema-
tous effusion is promoted which ensues in possible ulceration of the
cornea and in such relaxation of tissues as to protract the attack.
Some cases reject all moist applications; these are apt to be such
as have little or no secretion except tears by reflex irritation, viz.,.
GENERAL CONSIDERATIONS. 245
scleritis and iritis. Dry heat by a folded and warm napkin is often
most satisfactory. On the other hand, when secretion is abundant,
moist applications wash it away and by their temperature control
the exudation to some degree, as they influence the contractility of
the vessels. It is for the great majority of cases proper to use
local applications for only a portion of the time—sa\~ for ten minutes
or for thirty minutes three, four, six, ten times a day. Intermittent
use is the rule in moderate cases. Continuous use applies only to
severe cases. Details in this matter will come up in special dis-
eases.
We next come to the so-called collyria, whose name is legion,
and whose utility is regarded by the public as of the highest
moment. They are to be given almost exclusively in cases of con-
junctival disease. They are soothing, stimulating, astringent, and,
caustic. The indication for them will be found in the presence of
secretion which comes ordinarily from the conjunctiva, although the
primary lesion may be in another tissue. This secretion is serum,
epithelium, fibrin, pus- and blood-cells. The remedies are chosen
according to their power of causing contraction of the vessels and
coagulation of the secretion, or as they soothe the irritated nerve-
fibres. We do not know enough of the modus operandi of medi-
cines to reason exactly on this subject, and we act according to
the results of experience. It is simply my purpose in this place to
speak a warning against the misapplication of such remedies. To
apply to iritis, cyclitis, and pure scleritis, such remedies as tannin
or alum, or nitrate of silver, or sulphate of zinc, is utterly mischiev-
ous. So, too, they do harm in many, if not in almost all cases of
:acute keratitis. Before any " drops" are ordered, a diagnosis of
the disease must be made, and if this be not made, no drops capa-
ble of mischief are to be thought of; better temporize by lukewarm
water, or a weak solution of borax, or, best of all, frankly state the
difficulties of diagnosis, and seek further light. Such conduct will
save many an eye which rashness or false pride would ruin.
An indication of the highest importance in diseases of the eye 's
the regulation of its tension, especially to reduce it when excessive.
The cases in which it is below par are usually of a chronic charac-
ter, and are less amenable to improvement. To reduce increased
tension we have, first, eserine as a medicinal agent. But the chief
means are mechanical, viz., puncture of the cornea, and often not
more than two drops of aqueous fluid will be removed. Again, free
division of the cornea to let off all the aqueous fluid, and with it
morbid products like pus or lymph in the anterior chamber.
Thirdly, we have sclerotomy, which is done at the margin of the
anterior chamber by a peculiar method, and also at the equator.
Fourthly, we have iridectomy, which is done at the sclero-corneal
246
DISEASES OF THE EYE.
junction, and includes excision of a piece of iris. Fifthly, under
special conditions after chronic iritis and loss of the lens, division
of a mass of agglutinated tissue (iridotomy) relieves extreme ten-
sion. Sixthly, I have seen two cases where removal of the whole
iris through a small wound reduced the size and tension of a
staphylomatous globe. In ordinary practice, paracentesis of the
cornea and section of the cornea are proceedings which may be
adopted by physicians who do not regard themselves as skilled
operators, provided they cannot refer their patients to more ex-
perienced hands. The other proceedings need surgical training be-
fore they should be attempted.
Paracentesis cornece is liable to be followed in certain cases by
increased intra-ocular congestion and therefore the indications for
it must be definitely recognized.
CHAPTER II.
THE EYELIDS AND CONJUNCTIVA.
Anatomy.
The eyelids are formed at about the second month of embryonic
life as folds of skin which grow toward each other, and coming into
contact at about the end of the third month, adhere at their mar-
gins by continuity of their epithelium. They remain closed until a
short time before birth. The upper lid is much the broader, and
in the substance of each a smooth firm portion can be distinguished
which is known as the tarsus, and is composed of condensed fibrous
tissue. It was because of its stiffness formerly regarded errone-
ously as cartilaginous. The tarsi may
be spoken of as the frame-work of the lids;
where they meet they are thick, while their
orbital edges are thin. That of the upper
lid is about ten millimetres wide at its
middle, and that of the lower lid is about
five millimetres wide. Their general form
is exhibited in the diagram (see Fig. 89).
The space between them is called the
palpebral fissure. Its temporal end is
acute, while the nasal extremity is round-
ed. The tarsi are united to each other at
their extremities and also bound to the
subjacent bone by internal and external palpebral ligaments. When
open to its full extent the palpebral fissure is more rounded at its
inner than at its outer extremity and is likened to the shape of an
almond. At the inner angle (canthus) we find a fleshy mass called
the caruncle (caruncula lachrymalis); just exterior to it are the
openings of the tear passages (puncta lachrymalia) situated upon
little eminences; that of the upper lid is usually the more elevated.
The length of the palpebral fissure varies materially in different
persons; it may be taken as 30 mm. in men. Its width at the mid-
dle when looking straight forward is about 12 mm., and the border
of the upper lid covers the upper edge of the cornea for 1 or 2 mm.
The outer canthus stands at a level 3 to 6 mm. higher than the
inner canthus when the lids are open, and when closed the
Fig. 89.—The Tarsi seen from be-
hind. They have been isolated from
other tissues and remain joined at
the external and internal angles by
the lateral ligaments, external and
internal (or medial). 1, Posterior
surface of tarsus superior — on its
edge the openings of the Meibomian
follicles; 2, tarsus inferior; 3 and 4,
punctum lachrymale superior and
inferior; 5, external or lateral angle;
6, internal or medial angle of the
eyelids.
248
DISEASES OF THE EYE.
fissure is neither horizontal nor straight. When the eye looks up-
ward the palpebral opening increases to about 15 mm. in width,
and when it looks down the opening decreases to 7 or 9 mm. The
borders of the lids are fringed with short, stiff hairs (cilia or eye-
lashes) which are thicker and longer in the upper than in the lower
lid. They are slightly curved and the respective rows oppose their
convexities to each other. (See Fig. 90.)
When the lids are open we have in the upper lid a deep fold
caused by the retirement of the upper edge of its tarsus into the
orbit, and called the sulcus orbito-palpebralis superior. Above it
the skin is more prominent and is known as the orbital portion of
the lid. Similar peculiarities exist in the lower lid, but are less
conspicuous. We have the sul-
cus orbito-palpebralis inferior,
and in addition another less em-
phatic line, the sulcus palpebro-
malaris (Arlt). In certain per-
sons and particularly in the obese
and after middle life, the above-
named sulci may be strongly
marked. These features present
in different persons the widest
variations. Above the upper
lids we have the eyebrows (su-
percilia) situated at the upper
edges of the orbits (see. Fig. 90).
The inner surface of the lids
applies itself closely to the eye-
ball, and is lined by a membrane which is called the conjunctiva be-
cause it joins them to each other. Its description will be given here-
after. The lids are furnished with muscles to open and close them,
and with several varieties of glands. The closure of the lids is effected
by the orbicularis muscle, which lies just beneath the skin, to which
it adheres loosely by connective tissue and without the intervention
of any subcutaneous fat. Its fibres are more or less circular, con-
stituting a sphincter, and extend over a part of the superciliary,
the temporal, and the malar regions. They are inserted into a
tendon which adheres to the lachrymal bone and are also inserted
directly into the adjacent bony wall. The tendon crosses the
lachrymal sac at about its middle and contributes to the internal
palpebral ligament. Those fibres of the orbicularis which lie upon
the tarsi are paler than the remainder, and certain bundles which
lie close to the lid border and near the conjunctival surface are
known as the ciliary muscle of Riolani; see figure, p. 251. The or-
bicularis is supplied by the seventh or facial nerve.
Fio. go.—Right Eye and surroundings. 1, super-
cillum or eyebrow; 2, sulcus orbito-palpebralis;
3, papilla lachrymalis superior; 4, papilla lach-
rymalis inferior; 5, canthus externus (lateralis);
6, canthus internus (medialis); 7, caruncula lach-
rymalis; 8, plica semilunaris; 9, sulcus orbito-pal-
pebralis inferior: 10,11, sulcus palpebro-malaris.
THE EYELIDS AND CONJUNCTIVA. 249
The levator palpebrae superioris originates at the apex of the
orbit, lies close to its upper wall, grows wider as it comes forward,
and has a threefold insertion into and about the upper edge of the
tarsus (see Fig. 91.) The most anterior part of its tendon runs as a
layer of fibrous tissue down upon its anterior surface and merges
with the aponeurotic layer which comes down from the upper border
Fig. 91.—Vertical Section through the Globe and Orbit in the direction of the orbital axis, with
closed Lids. 1, skin of upper eyelid; 2,2, musculus orbicularis palpebrarum; 3, fascia palpebralis
superior; 4, border of frontal bone; 5. tarsus superior, schematically represented; 6, musculus le-
vator palpebne superioris; 6a. its principal tendon which spreads out between the tarsus and mus-
culus orbicularis; 66, the smooth musculus palpebralis superior; 6c, conjoined insertion of the mus-
culus levator palpebrae and musculus rectus superior going to the conjunctivae; 7, musculus rectus
superior; 8. 8, Tenon's capsule; 9, tendon of musculus rectus superior, passing through Tenon s
space 10, limit between inner orbital fat, 11, and supravaginal space 12; 13, musculus rectus oculi
inferior; 13a, extremity of its fascia, going to lower lid; 14, cross section of musculus obliquus infe-
rior: 15, skin of lower lid; 16, tarsus inferior, schematically represented; 17, fascia palpebralis infe-
rior; 18, 18, periorbita; 19, 20, fornix conjunctivae; a, optic nerve; b, vitreous; c, lens; d, cornea.
of the orbit and pushes into the fibres of the orbicularis muscle, thus
binding together all these structures. The middle layer is inserted
into the upper edge of the tarsus and consists largely of smooth
muscular fibres. A third portion of the tendon dips back to mingle
with fibrous prolongations of the insertion of the rectus superior
250
DISEASES OF THE EYE.
and goes to the superior fornix of the conjunctiva and sends lateral
off-shoots to be attached to the outer and inner walls of the orbit.
By this arrangement the movements of the upper lid and of the
globe when looking upward are co-ordinated, and a layer of dense
membrane shuts in the contents of the orbit above the eyeball.
The levator palpebral superioris is supplied by a twig from the third
nerve. It may here be remarked that the rectus inferior, after its
insertion into the globe, sends a tendinous prolongation, in a man-
ner similar to the arrangement of the levator of the upper lid, to
the edge of the inferior tarsus and to the inferior conjunctival for-
nix l (Schwalbe) and to the fascia palpebralis inferior. (See Fig. 91.)
Still another muscle is to be mentioned which lies behind the
lachr3rmal sac arising from the crista lachrymalis and bifurcating
into two tendons, of which one is inserted into the border of the
upper and lower lids respectively. It is called musculus lachry-
malis posterior or muscle of Horner.
The glandular structures of the lids are numerous. In the skin
are sweat glands and xery fine scattered hairs. The follicles of the
cilia are furnished with sebaceous glands, the glands of Moll, and
in each tarsus is an important series of glands, arranged like cur-
rants on a stem, known as the Meibomian, which run vertically in
their substance near their posterior surface and open by minute
orifices upon the free border of the lids behind the rows of cilia. A
section parallel to and about one millimetre above the free border
of the lids will cross the hair follicles, the glands of Moll and of
Meibomius, and reveal a number so great as will be likely to sur-
prise one who has not before looked at such a section. The eye-
lashes of the upper lid are from 8 to 12 mm. long and said to have a
life varying from 100 to 150 days (Donders). The relations of parts
in the upper lid will be best understood by the figure (see Fig. 92).
Still other glands are to be found in close relation to the con-
junctiva, viz., the acino-tubular glands of Krause, which lie at the
border of the tarsi near the fornix, more numerous in the upper
than in the lower lid, and other similar glands, very few in number,
imbedded in the tarsal conjunctiva and in the tarsus. These are
regarded as accessory lachrymal glands. In the tarsal conjunctiva
are certain follicular cavities formed by irregular involutions of its
epithelium which are called the glands of Henle and will be re-
ferred to again when describing the conjunctiva.
The large number of glandular structures thus mentioned give
rise, as would be expected, to many and various pathological condi-
tions to which attention will be called, and some of them are obsti-
nate and distressing.
The function of the lids is to protect the eye both from mechan-
1 " Lehrbueh der Anatomie des Sinnesorganes," p. 242, 1885.
THE EYELIDS AND CONJUNCTIVA.
251
ical injury and from excessive light, and to distribute over it the
moisture furnished by the numerous glands. The movement of the
lids is both voluntary and involuntary or reflex. The latter is de-
termined by the fibres of the fifth nerve which supply the cornea
and ocular conjunctiva acting
upon the orbicularis and es-
pecially upon those of its fibres
which traverse the tarsal por-
tion. The persistent opening
of the lids during waking hours
is provided for by the exist-
ence in the levator palpebral
superioris of certain unstriped
fibres (H. Miiller) to which ref-
erence has been made. The
eyelids follow, as has been said,
the movements of the cornea
up and down, and in so doing
the palpebral opening varies in
width, becoming larger in look-
ing up, and narrower in look-
ing down. The opening of the
lids is performed almost wholly
by the lifting of the upper lid,
but in looking down the lower
lid is also made to descend by
the indirect attachment to its
tarsus of the tendon of the rec-
tus inferior. (See Fig. 91, page
249.) Under special impulses
the separation of the lids can
be notably increased and so
much, as to show a border of
sclera both above and below
the cornea. This occurs under
emotions of surprise, of fright,
of earnest attention, and ap-
pears in exophthalmic goitre
(Basedow's disease). Droop-
ing or falling of the lids comes from fatigue, from paralysis of the
levator, and from mechanical hindrance such as thickening of the
conjunctiva, etc.
Fig. 92. —Sagittal Section through the Upper Eye-
lid. 1, skin; 2, palpebral portion of the musculus or-
bicularis oculi; 2a, its inner portion, designated as
the musculus ciliaris Riolani; 3, cilia; 4, gland of
Moll, opening into a hair follicle; 5, Meibomian
gland; 5a, its orifice; 6, indication of the ill-defined
limit of the tarsus; 7, loose connective tissue be-
tween tarsus and anterior insertion of the tendon of
the musculus levator palpebrae superioris; 8, anterior
connective-tissue-like insertion of the tendon of the
musculus levator palpebrae superioris; 9, its middle
layer non-muscular, called the musculus palpebralis
superior. (H. Miiller.)
252
DISEASES OF THE EYE.
Blepharitis Marginalis. Ophthalmia Tarsi. Blepharo-
Adenitis.
We have various degrees and kinds of this affection; for in-
stance : 1st, chronic hyperasmia of the border with slight thicken-
ing; 2d, some redness wTith an accumulation of yellowish, fatty
material at the base of the lashes, the hypersecretion of the glan-
dules, a kind of seborrhoea; 3d, ulceration, minute abscesses, crusts
gluing the lashes together and sometimes severe inflammation of
the whole border; 4th, after long continuance the lid border becomes
smooth, red, glazed, everted, thickened, weeping, and destitute of
lashes (lippitudo). It is characteristic that the hair follicles atro-
phy, the lashes dwindle, become pale or curl up and fall out. Some-
times decided ectropium, and eversion or occlusion of the lachrymal
puncta, takes place, this more frequently in the aged or the un-
cleanly.
The disease occurs most often in the young with delicate skin
and light hair, and in the strumous. It is sometimes a kind of
eczema." In very many cases it is associated with some refractive
or muscular error and is only an expression of functional strain,
for which no local remedies will avail, until proper glasses or other
correction are employed (Roosa) (Schirmer).
Chronic conjunctivitis, trachoma, phlyctenula, are frequent con-
comitants. The ailment is apt to be chronic, but except in the in-
veterate form mentioned as the fourth type, will usually yield to
proper measures.
Treatment.— The first two forms require soothing lotions, warm
water or warm milk and water, and for the seborrhoea it should be
made a little alkaline with bicarbonate of potash; at night a mix-
ture of boracic acid powder and vaseline, gr. xxx. ad oz. i., may be
applied, or soft oxide of zinc ointment. For the ulcerative form the
crusts are to be softened, and as much as possible removed, and the
following ointments may be used: two grains of hydrarg. oxid. flavse
to one drachm of vaseline or amylo-glycerin; or, ung. citrini, gr. x.
vel. xx., vaselini, 3 i., to be applied night and morning, or, in bad
cases, more frequently. In a large number of cases, the best
method is to pick off the crusts with fine forceps or the finger-
nails, and cauterize the exposed ulcers with a fine point of nitrate
of silver. It often bleeds, and the caustic hurts. In cases of ex-
tensive incrustation, and especially in young children, the lashes
may be cut off with scissors to facilitate the denudation and cau-
terization of the ulcers. The subsequent use of stimulating salve
will then control the disease. But if the person be the subject of
error of refraction, or of other error which causes eye strain, the
"removal of the blephharitis will not only demand the usual local
THE EYELIDS AND CONJUNCTIVA. 253
treatment, but also that the error be corrected. (See Part I. of
this treatise.)
In specially obstinate cases the evulsion of the lashes may be
demanded (epilation) with use of lotions of acetate of lead (Liquor
plumbi subacetatis, 3 i.; Aquae, § viij. M.) and the ointments
above mentioned, especially the yellow oxide of mercury. Treat-
ment of conjunctival disease by nitrate of silver, gr. ij.-v. ad oz. i.,
must not be neglected. For the chronic thickening with eversion
and loss of eyelashes (madarosis, tylosis), squamous blepharitis,.
stimulating ointments of more intensity may be used, such as an
ointment of Hebra's:
IJ Emplast. diachylon co.,1
Olei olivae,.....q. s.
M.
Or,
5 Olei cadini,.....1
Vaselini,......2.
M.
Apply every night.
The crystal of sulphate of copper may be applied daily, or the
nitrate of silver, pure stick, pro re nata.
Sometimes constitutional treatment of scrofulous conditions is
not to be omitted.
Hordeolum or Stye.
This affection is a phlegmonous inflammation at the tarsal edge,
which forms a small and generally painful lump. It is apt to be
associated with chronic blepharitis or conjunctivitis, and often de-
pends on general debility. In its inception it may sometimes be
checked by applying a bit of ice wrapped in muslin for a few min-
utes repeatedly, or by pulling the cilium which passes through it.
One is apt to follow another in succession. When suppuration is
unavoidable, a poultice of ground slippery elm bark (ulmus flava)
is most comforting, and a puncture should be made at an early
period. General tonics and mild astringents are the proper reme-
dies to prevent their recurrence; but it is important also to inves-
tigate the state of refraction, because what causes eye strain will
provoke styes. Another frequent concomitant and favoring condi-
tion, is nasal catarrh, which will also need attention.
1 Emplastrum diachylon co. is made as follows: Emplast. litharge, 12
parts; flour, 1 J- parts; ammoniac, galbanum, turpentine, each 1 part.
:254
DISEASES OF THE EYE.
Chalazion, or Cystic Tumors.
Obstruction and distention of some of the follicles of the tarsus,
more frequently of the Meibomian, is the origin of these tumors.
They are painless, imbedded in the tarsus, and the skin is freely
movable over them. They vary in size, and are apt to come in
crops. The sac wall is usually thin, and as the tumor enlarges it
causes a reddish or yellowish projection on the conjunctival sur-
face, and sometimes presents granulations. The contents are a
glairy, mucilaginous substance. Microscopic examinations have
shown that nutritive disturbance in a Meibomian folli-
cle, excites chronic inflammation in the surrounding
connective tissue which leads to an infiltration with
small cells. By confluence of several foci of infiltra-
tion, a nodule is formed composed of granulation tissue
and giant cells. This results in mucoid softening and
its escape by ulceration (Fuchs). Cocci are also found
within them. Fluctuation is never felt, and I have
sometimes found a solid, fibrous tumor when I expected
to meet a cyst, and at times the cyst wall has been
extremely thickened. When small, the tumors are not
troublesome, and they occasionally disappear. If they
reach a size to be annoying, they must be excised. We
may do this on the skin surface, through a wound par-
allel to the lid-border, and no perceptible scar is left.
The cyst may be opened on the inside surface if it pro-
ject notably in this direction, and the contents scooped
out with a sharp spoon. Sometimes the tumor runs
for some distance up the lid along the line of a Meibo-
mian duct: it will be apt to point at the lid border.
Here a deep puncture may be made with a narrow
knife and a sharp curette pushed up into the tubular cyst to scrape
its walls and evacuate its contents. Special forceps have been con-
trived by Desmarres, Snellen, Prout, and Knapp, to inclose the
tumor in a clamp to prevent bleeding. Before applying it, drop
into the conjunctival sac a 4$ solution of cocaine and wait for its
effect. Then put on the clamp forceps, screw it tight, and inject
hypodermically three drops of 4$ cocaine solution alongside the
tumor. In a few minutes the dissection can be made painlessly
and almost without bleeding. Most of the clamps have a broad
plate of metal or horn for one blade and this is sometimes useful.
A simpler form will usually answer. (See Fig. 93.) In lack of
clamps, a flat spatula or the operator's forefinger slipped under it,
will hold the lid tense and check bleeding; the tumor is to be ex-
posed and may be seized with a sharp hook by an assistant and
THE EYELIDS AND CONJUNCTIVA. 255
excised, or if without an assistant, pointed scissors curved on the
flat will remove the projecting part, and when thus opened the re-
maining portion can be scraped out. Should a small perforation
of the lid occur, no harm is done. For very large cysts with thick
posterior wall, it may sometimes be well to touch it lightly with
a point of lunar caustic instead of relying on scraping alone. To
guard against recurrences, remove chronic palpebral conjunctivitis
and correct eye strain, and in some instances cod-liver oil and
means to improve nutrition, especially if there be a strumous dia-
thesis, are important.
Phlegmon of the Lid.
If suppuration occur in the connective tissue of the lid, as may
happen after debilitating disease, or in strumous children, or with-
>■ out recognizable cause, there will be great swelling, and fluctuation
will be detected early. It may come with very little pain and but
little redness. It is also important to remember, that a general
inflammation of the lids may occur in delicate children, and not re-
sult in suppuration: there may be great oedema and slight redness,
and the whole may disappear by resolution. On the other hand, the
connective tissue may become gangrenous in cachectic subjects. If
suppuration occur, the pus must have vent early, by a free incision,
parallel to the border of the lid. The best knife is a Beer's cata-
ract knife, or a very narrow, sharp pointed and curved bistoury.
Stand behind the patient, pierce the skin, and run the point along
with a quick, steady thrust. The earlier the incision is made, the
less will be the likelihood of deformity after the abscess heals.
In cases of erysipelas of the face, if there be much induration of
the lids, care must be taken to watch for suppuration. It is very
liable to occur, and considerable destruction of tissue may take
place, which early incision would obviate. In the severe forms of
the disease, it sometimes becomes needful to make deep incisions
when there is no evidence of pus, to save sphacelation of the tissue.
A very remarkable and fortunately rare occurrence is sponta-
neous gangrene of the skin of the lid. Two cases of this character
were reported to the New York Ophthalmological Society by Dr.
Rushinore, of Brooklyn, in 1883, and another by Dr. R. H. Derby in
1SS4. See case by Hilbert: Centralblatt fur Augenheilkunde,
Oct., 1883, p. 293. Deformity will ensue for which a plastic opera-
tion may be required.
Inflammation of the Tarsus.
Tarsitis.—This does not occur very often. It is usually syphili-
tic, yet maj7 be simple or idiopathic. It is very slow in progress
and not specially painful; it is attended by great thickening of the
256
DISEASES OF THE EYE.
tarsus, abscesses are apt to form in its substance and they break
at the tarsal border. Hence, there will be ulcers, crusts and yellow
projecting points, the eye-lashes will fall and their follicles atrophy.
It will appear like blepharitis marginalis, but the great thicken-
ing of the tarsus declares its character. A diffuse redness spreads
over the lid border and the appearance may be very unpleasing.
If, as is usual, syphilitic, constitutional treatment will be essen-
tial; if simple, it will be difficult to control. Hot fomentations may
be applied and the stimulating ointments above mentioned, while in
the case of a young lady whom I treated for over a year the only
control over the minute abscess and the thickening, was by inserting
a red hot needle alongside a hair, or passing in a platinum needle
dipped in strong nitric acid. (See Bull, Trans. Am. Oph. Soc, 1878,
p. 405.)
All varieties of diseases of the skin may appear on the lids and
the following may be singled out: eczema, xanthelasma, molluscum
contagiosum, herpes zoster ophthalmicus.
Eczema appears in the acute and chronic form. It is common
among children in connection with acute conjunctivitis and kerati-
tis, presenting crusts and ulcerations and discharge. The scaly
and hypertrophic form appears chiefly among adults and especially
in the aged. For the former, complete and careful washing away
of scabs, and application of vaseline, or boric acid and vaseline, or
yellow oxide of mercurj7 ointment, may suffice. But in many cases,
especially among children of the poor, it is better to give chloro-
form, remove the scabs, dry the bleeding with absorbent cotton
and go over the surface with pure nitrate of silver. After this the
above ointments will complete the cure. Starch powder may be
dusted on the surface if needful. Commonly similar crusts exist
about the nostrils and perhaps at the angles of the mouth; all such
spots should be cauterized. Treatment of conjunctival inflamma-
tion w7ill at the same time be attended to.
The squamous and hypertrophic eczema may be limited to the
eyelids and vicinity, and may or may not be complicated with acute
conjunctivitis. If the condition be chronic, the stimulating reme-
dies may be used, viz., Olei cadini, 1 part; vaselini, 2 to 4 parts; or
the diachylon ointment (vide p. 253). In acute conditions with
serous effusion and, as may happen, with the whole face involved
and the conjunctiva acutely inflamed, the milder ointments of oxide
of zinc, vaseline and boric acid, of white precipitate of mercury, or
dilute citrine ointment are to be preferred. Wet applications are
generally decidedly unacceptable. Sometimes the irritation is ex-
treme and may call for bromides and preparations of opium or
other anodynes internally, as well as hypnotics: antipyrine, gr. x.;
hyoscyamine, gr. jfo or gr. -g^ in tablets, chloral, phenacetine, etc.
THE EYELIDS AND CONJUNCTIVA. 257
Such astringents, as tannin and alum, are better for the con-
junctivitis than nitrate of silver, and cocaine may be employed to
advantage.
Xanthelasma or xanthoma is a fatty degeneration of the con-
nective tissue of the skin, which has a predilection for the eyelids,
although it occurs elsewhere. Yellow or straw-colored, slightly
nodular patches appear at the inner extremities of the lids, usually
symmetrically, and both upper and lower lids may be affected.
They gradually extend and may become large and prominent welts.
They come oftener in women than in men and after middle life.
They are easily removed by excision, which is the only mode of
relief; but I have been disappointed to find the disease return
within a year in one case. Being without danger and simply a
slight blemish, few persons care to submit to their excision.
Molluscum contagiosum appears anywhere on the body and
often about the lids. The little tumors may be as large as peas or
hempseeds. The top is cupped and a little opening leads into the
middle of the tumor. The sebaceous glands are probably the seat
of the disease and they contain altered epithelial cells and peculiar
bodies called molluscum corpuscles which are of a fatty nature.
The contents may be squeezed out between the thumb nails through
the little opening above mentioned; or, if needful, they may be
opened with a knife. Evacuation cures them. That they are con-
tagious does not seem to be well founded.
Herpes Zoster Ophthalmicus
exhibits conspicuous and important features. It is called by the
French zona ophthalmique, and has been extensively described
by Hybord, and previously by Mr. Hutchinson. It is, in truth, a
neuropathic affection having its cause in degeneration of the gan-
glion of Gasser, or of the branches of the trigeminus, or of both.
Any of the branches of the fifth pair may be thus affected, and the
eruption appears along the distribution of the diseased nerve-twigs.
It therefore happens that vesicles may occur on the eyeball as well
as upon the skin, and both ulceration of the cornea, acute conjunc-
tivitis and acute iritis may take place. It may even cause loss
of the eye by irido-cyclitis. It is also said that small abscesses
have been found in the ocular muscles. The mode of occurrence, as
illustrated in a boy ten years of age, was as follows: the supra-
orbital nerve was the one affected. The initial symptom was in-
tense pain along this nerve at the supra-orbital notch, around the
lachrymal sac and side of the nose, upon the forehead, and up to
the vertex. In a few hours the skin of the forehead became red
and swollen, tender to touch, and a few vesicles appeared above
17
258
DISEASES OF THE EYE.
the inner end of the brow. While the right half of the forehead,
red and swollen, presented the look of erysipelas, the left half re-
mained natural. The hair could not be combed because the scalp
was tender, and a few vesicles were there discovered. The eyelids
swelled, a slight conjunctivitis appeared, chiefly affecting the pal-
pebral surfaces, and there was great photophobia. The pulse was
quickened; it reached ninety, and some febrile reaction occurred.
The urgent symptom was the pain, which continued day and night.
A few vesicles appeared on the side of the nose; none whatever
showed themselves across the median line.
The treatment consisted in keeping the boy in bed and dropping
into the eye every two hours a solution of sulph. atropia, gr. ij. ad
3 L, to abate the pain (cocaine would have been proper), using
upon the forehead hot fomentations without intermission, and giv-
ing full doses of morphia and quinia sulphate three times daily.
By the fourth day there was decided mitigation of the symptoms,
but it was not until the twenty-fourth day that the patient could
go out. No lesion of the cornea took place. In case the latter
should occur, it would be much longer before the patient would be
well. When there is an eruption on the cornea its surface is mark-
edly anaBsthetic. This suggests a reason for the long continuance
of the affection in some cases, and also the need of keeping the eye
bound up so long as the irritation continues. The special treat-
ment suitable to cases of ophthalmic shingles, in which the cornea
or iris may be involved, will be found under the chapters which
treat of these troubles respectively. I have seen one case in which,
while one eye was destroyed by the direct mischief of the disease,
the other was also lost through sympathetic irido-choroiditis. I
have notes of a case in which both sides of the forehead were
attacked. Permanent scars remain, which may be recognized by
their rounded form, and by a slight depression of the surface.
The disease may take place at any age, and it is most hurtful
to the aged and feeble. It is very apt to be regarded as simple
erysipelas, but from this it may be discriminated by the intense
neuralgic pain following certain nerve-twigs, by the strict localiza-
tion of the skin trouble, and by the vesicles. The lesion may go
down to the tip of the nose, or upon any part of the distribution of
the trigeminus. The treatment, as above specified, should be both
local and constitutional, the latter being such as may control neu-
ralgia, the former to soothe the local inflammation. For a severe
attack in a man who was nearly eighty years old, ten-grain doses
of quinine were given at intervals of two hours until fifty grains
were taken daily, with marked benefit and perfect tolerance of the
drug. It is said that when the vesicles appear on the nose, the
cornea is most likely to be involved. I cannot support this state-
THE EYELIDS AND CONJUNCTIVA.
259
ment because I have found the corneal affection both with and with-
out implication of the nasal twigs. See cases by Jeffries and
Mathewson, Trans. Am. Oph. Soc, 1874, pp. 221, 228. Mathewson
used constant (?) galvanic current with great relief to pain in
five cases. Severe optic neuritis followed by atrophy has been seen
(Daguenet, Rec. d'Ophth., 1877, 177); paralysis of accommodation
and mydriasis have been repeatedly seen.
Syphilitic ulcerations are sometimes found upon the lids—they
may be chancres or secondary ulcerations; although the latter are
more likely to appear on the mucous surface. It is hardly neces-
sary to say anything about the recognition and treatment of these
conditions. They only need to be mentioned (see paper by Dr. Bull,
Trans. Am. Ophth. Soc, p. 408, 1878). French literature furnishes
the greatest number (see paper by Dr. Beck, Trans. Am. Ophth.
Soc, 1886; who has collected 94 cases).
Epithelial Cancer and Lupoid Growths
are quite often situated upon and near the eyelids. A discrimina-
tion between them is hardly needful for practical purposes. If a
nodular, irregular elevation appears on the lid border, or on the
skin, and is covered by a dark crust which, when picked off, exposes
a bleeding surface, and if this continue for months or years, some-
times healing and again breaking out, but never going entirely
away, this neoplasm, although quite painless, had better be excised.
The true epithelioma is more rapid in development than a lupoid
growth, and both may result in ulceration. In either case the
neighboring lymphatic glands are not likely to be enlarged, except
at a late date. The gland which we look for is that in front of
the tragus—the pre-auricular gland. Grow7ths such as we are now
considering, occur during and after middle age, and usually remain
unheeded for a long time. Sometimes soothing lotions will pro-
cure healing of the ulcer. The solution of chlorinated soda (Labar-
raque's), diluted with five parts of water, will often be followed by
perfect cicatrization of a suspicious and extensive ulceration. It is
applied for twenty minutes by a piece of lint six or eight times a
day. I have made this observation many times during the last fifteen
years.
For almost all cases, the best method of treatment is an opera-
tion, and not caustics. If the latter be applied to the lids, deformity
will follow which will necessitate an operation, while if the knife be
resorted to, the deformity ma}7 be at once remedied by a suitable
plastic proceeding. If there be a spot of ulceration on the cheek
or temple or nose, covered with a thin brown crust, which leaves,
when removed, a bleeding depressed surface; if, too, the skin be
hard and infiltrated at this spot, the case is probably lupus non
200
DISEASES OF THE EYE.
exedens. Scraping out with a sharp spoon, or thorough burning
by actual cautery (Paquelin's thermo-cautery) will be likely to cure
it. Escharotic plasters are to be eschewed; they cause great
pain and wide destruction. In many instances of probable epithe-
lioma upon which I have operated, there has been no return of
the disease for man}- years. The prognosis is encouraging when a
thorough removal is performed. If a relapse demands a second
operation, even then, as I have seen, the disease may not recur.
I have seen one case of amyloid tumor upon the border of the
lid. The patient w7as a young woman under the care of Dr. Prout,
of Brooklyn.
Papillomata or warts are not uncommon on the border of the
lids. They may be snipped off, or accurately touched with nitric
acid applied by a platinum probe, or by a small and pointed stick.
Horny growths have been known to occur on the lids. One in-
stance I have had in my own practice.
Milium presents a perfectly white tumor, not larger than the
head of a pin. It is a retention tumor of a sebaceous follicle and
simply needs puncture.
For an account of very rare cases of adenoma of the Meibomian
glands and of the glands of Krause, see Salzmann, Arch, filr
Ophthal., XX., 3, p. 380, 1891.
Njeyi and Teleangiectatic Tumors.
Several varieties of vascular growths occur on or about the lids;
they may be simple red patches, or slightly elevated and flattened
patches, or they may be conspicuous and lobulated masses contain-
ing large vessels as well as capillaries, and are called cavernous
tumors. The diagnosis is simple and no extended description is
required.
Treatment.— Excision is advisable in the early stage of these
tumors, and the lid clamp of Snellen or Knapp can often be used to
check bleeding. Destruction of the tissue may be effected by punc-
ture with red-hot needles (shoemakers' sewing awls are sometimes
convenient). The dental blast lamp gives the requisite heat, if a
Bunsen's gas burner or large alcohol flame does not suffice. A suc-
cession of operations will be required. For a considerable number
of tumors excision ma}7 be successfully practised, because they will
be found inclosed in a distinct fibrous capsule, and if this be re-
spected, no serious hemorrhage will occur. Care must afterward be
taken to keep the cavity well closed and to treat it antiseptically.
For certain large growths which may perhaps extend into the
orbit, electrolysis offers a sufficiently safe and effectual method.
The purpose is to coagulate the blood, not to destroy the tissue.
A number of needles connected with the positive pole may be
THE EYELIDS AND CONJUNCTIVA. 261
plunged into the tumor and the negative pole applied by a sponge
to the temple. The needles should be of platinum to prevent oxi-
dation. The current must not be strong, usually four small cells
suffice. See Figure 230, under chapter on the Orbit.
In all operations care must be taken not to excite severe re-
action lest deformity ensue. I have seen a case in an infant for
which the common carotid had to be tied. The tumor disap-
peared.
Injection of persulphate of iron is too severe, of alcohol has been
lately recommended. Threads may be run through to excite sup-
puration. In some instances small and even large vascular tumors
have been known to disappear of themselves.
Minute vascular growths, like little red warts, sometimes appear
on the border of the lid. They may be easily tied or burnt off.
Moles or brown patches may occur congenitally either upon
the lids or in the neighborhood. They should be excised, and after-
ward a proper plastic operation performed. I was called upon to
do this for a young lady, whom I saw again after several years,
and found that similar pigment-nodules had appeared upon the
neighboring skin which had previously been healthy. The primary
growth was congenital, was set with stiff hairs and seemed to be
innocuous, although a decided blemish. The subsequent pigmenta-
tion showed no malignant or ulcerating tendency.
Diseases of the Eyelashes.
They may fall out as the result of chronic marginal blepharitis,
and when one of the symptoms of secondary syphilis without any
noticeable inflammation; while at the same time the eyebrows will
be shed. The condition is called madarosis or tylosis.
Canities is the name given to decoloration of the lashes. There
may be a cluster of white cilia on only one eyelid. I have seen
all the cilia of one lid perfectly white and the cilia of the remaining
lids dark; in the same lady there was a wisp of white in the
midst of the dark brown hair of the head.
Phtheiriasis signifies the presence of crab lice (pediculuspubis)
among the lashes. Their eggs adhere in rows to the cilia, and the
crawling of the creatures provokes itching. Mercurial ointment
destroys them, locally applied.
Distichiasis means that there is a double row of lashes, one of
which touches the globe. As many as two displaced rows have
been seen. As a rarity the condition is congenital, usually it is
acquired.
Trichiasis differs from the above in the irregular position and
shape of the lashes which come in contact with the eye. A few or
a j?reat number may be inverted, and they curl in various direc-
262
DISEASES OF THE EYE.
tions. Many of them will be atrophied. There may be thickening
of the tarsal border, but the tarsus is not bent or notably deformed.
The state of the tarsus makes the distinction between trichiasis
and entropium, although the former insensibly shades into the
latter. Trichiasis is caused by blepharitis marginalis, by tra-
choma, by burns, etc. We may practically distinguish between
partial and complete trichiasis. Either the upper or the lower lid
may be affected and the effects upon the cornea may be more or
less severe.
Treatment.—For partial trichiasis the methods available are 1,
epilation; 2, snaring them with a thread; 3, destruction by hot
needles or by electrolysis; 4, excision of the follicles. Spasm of the
orbicularis and conjunctival irritation often coexist and if the bor-
der of the lids be forcibly inverted, spasmodic entropium.
1. Pulling out the hairs by forceps gives temporary relief and
must be repeated every week or two. Often the offending lashes.
are very fine and difficult to seize, and a patient will much complain
if the " short hairs " are overlooked. After operations some errant
cilia may remain and their evulsion be preferred to any other pro-
ceeding, and will often be done by some member of the family.
2. Ensnaring the cilia in a loop of thread (Snellen) is done by
entering a needle, through whose eye both ends of a fine silk thread
have been passed, and as the loop is drawn to the base of the hair,,
the latter is put within it and dragged up into the substance of the
lid. This proceeding applies to single or a very few hairs. A
thread may also be used to destroy a group of hairs by carrying it
into the substance of the tarsus up, across, and then down, inclos-
ing them in its bight, and tying down hard upon the lid border to
crush the follicles and set up destructive suppuration.
3. Destruction by red hot needles, by a platinum needle dipped in
caustic potash, or by electrolysis is better suited to the above con-
ditions. The last was proposed by Michel, of St. Louis. A trian-
gular gold or platinum needle is pushed into the follicle and con-
nected with the negative pole of a constant battery of from eight
to twenty elements; the sponge of the positive pole is placed on
the temple or held in the patient's hand. When the circuit is
closed, minute bubbles of gas are disengaged and the tissue
whitens about the base of the cilium. There is considerable pain
and about a minute is needed to destroy the follicle. The treat-
ment sometimes fails.
4. Excision of the follicles is done by taking out a rectangu-
lar portion of the tarsus, without encroaching on the Meibomian
follicles. The superjacent skin is dissected up in a little flap, and
this may be extended upward, and after the removal of the bit of
tarsus the flap be dragged clown, its tip cut off, turned in to cover
the lid border, and held in place by a suture at each corner. Slight
THE EYELIDS AND CONJUNCTIVA. 263
traction is thus maintained and the gap is filled up (Anagnostakis).
When trichiasis is more extensive, the mode of proceeding will
depend upon collateral conditions. As already said, the cases
merge insensibly into those called entropium, there being in both
classes lesion of the tarsus, but in the latter it is more severe.
The choice of method will depend upon the length of the palpebral
slit, the quantity of substance in the lid, and especially on the state
of the tarsus.
It is convenient to deal with the subject under the head of en-
tropium and indicate what modifications of method are suited to
varying morbid conditions.
Entropium.
Besides simple inversion of the lashes, we find in old cases
shortening of the palpebral slit, thickening and incurvation of
the tarsus, as readily seen by the furrow along its middle when
the lid is turned over, the tarsal border becomes sharp and thin,
and often the lids hug the eyeball tightly. This last circum-
stance produces almost as great mischief as the presence of the
inverted lashes, by fretting the cornea and keeping up the super-
ficial inflammation. The effect of entropium is, opacity of the
cornea, and if the lids are tight, the softened structure loses its
proper curve and may even become staphylomatous. Entropium
may appear during the progress of trachoma, but is usually one of
its sequelae, as will be hereafter described.
We have, besides the cases above referred to, two other forms
of entropium, viz., the senile and the spasmodic Senile entropium
ensues from relaxation of the tissues. The skin becomes folded and
droops, and the ciliary border turns inward, the orbicularis aiding
in the effect.
For relief of the senile variety, the removal of a properly pro-
portioned piece of skin is all that is usually required. Threads run
vertically beneath the skin and tied tightly down to cut their way
out and reef up the tissues by the cicatrices, are objectionable from
the puffy state in which the parts are left. It is easier to effect
the object in the upper than in the lower lid. Entropium of the
lower lid often complicates the treatment of cataract extraction,
being both spasmodic and due to relaxation. Sometimes a piece of
caoutchouc plaster or the application of contractile collodion will
draw the lid down, but generally a portion of skin will need
removal. This will run parallel to the border and vary from six
to twelve millimetres in width.
Spasmodic entropium of the lower lid happens in chronic kera-
titis and in other conditions. A suitable operation for an obsti-
nate case is indicated in the diagram (Graefe), (see Fig. 94),
264
DISEASES OF THE EYE.
where the flaps being undermined, are brought together over
the open wound. For a case of trichiasis at the outer third of
the lower lid with entropium which was maintained by tracho-
matous cicatrices of the conjunctiva I did the following: At
four millimetres below and parallel to the lid, I raised a flap
about six millimetres wide and equal in length to the lid. It was
left attached at its temporal extremity and about one-half of
it cut off. The remaining piece was trimmed and turned up to
be imbedded in the lid border where the erring lashes grew, and
stitched fast. Like a piece of tape it held the lid in permanent
eversion. The wound below the lid border was closed by sutures.
The girl disappeared from view for seven years after the parts
healed. I then cut away the bridge of skin. The deformity was
cured, the cornea was healthy,
and no return of the trouble took
place. Probably a month would
have sufficed to effect the object.
Still other methods of dealing
with these cases exist, as by su-
tures. (Saemisch, Wecker.)
The methods of treating ordi-
nary entropium are almost in-
numerable. One must make
choice according to the needs of
a given case. It must be remembered that its essential cause is
deformity of the tarsus and all operations must be adapted to
modify and correct its malposition.
The operations of Arlt, Jaesche, Flarer, and others begin by
splitting the tarsus along the border into two layers for a depth of
about three millimetres, and the ciliary border is drawn up after ex-
cising a narrow strip of skin above it. Another proceeding is not to
throw away the strip, but to leave it attached at each extremity,
to draw it by the middle below the ciliary flap, and make it take
the place of the latter on the lid border (Gayet, Dianoux1). Sw7an-
zey figures the operation, but it hardly seems possible to avoid a
clumsy and unpleasing result by such a device.
Where no serious deformity of the tarsus exists, Arlt's method
of transplantation of the loosened lid border, which is well known,
serves an excellent purpose. If the incisions inclosing the semilu-
nar flap are carried a little beyond the outer and inner canthi, in-
verted lashes at the extremities will not be omitted in the effect.
The excised flap of skin will be from four to six millimetres wide at
its middle. The displaced marginal strip will be about four milli-
metres wide and may or may not be fully loosened at its upper
Fig. 94.
1 Annales d'Oculistique, 1882, xxxviii., p. 132.
THE EYELIDS AND CONJUNCTIVA.
265
■edge. The exposed surface at the margin of the lid is left to gran-
ulate. It is better to depend on a spatula by which an assistant
lifts the lid and keeps it tense, than upon a clamp. It restrains
bleeding sufficiently.
Acting upon another principle are the methods of Streatfield,
Snellen, Pope, in which a deep groove is cut into the tarsus on its
front surface just above the lid border, or the tarsus substantially
dissected out; a strip of skin with subjacent muscular fibres is next
removed, and the wound closed by sutures. A canthotomy may
also be done. These proceedings are suited to cases where shrink-
ing of the tarsus has begun, yet not advanced very far, and w7hile
often effective, more confidence can be placed upon another opera-
tion devised by Dr. John Green which will be presently described.
Mention must be first made of Hotz's operation, which is care-
fully described in Knapp's Archives, 1879. The theory of it con-
sists in making the integument so adhere to the upper edge of
the tarsus and the tarso-orbital fascia, that its tension shall draw
the ciliary border outward. (See Anatomy, page 247.) The mode
of performance is as follows: an assistant fixes the skin of the
brow against the orbital edge, the surgeon draws down the lid
at its middle with his thumb and finger or by forceps, he incises
the skin horizontally along the whole length of the lid on a line
which begins and ends two millimetres above the outer and inner
canthi. The lid being stretched and drawn down at its middle,
this line, although made horizontally, becomes a curve when the
lid is let go, parallel to the upper border of the tarsus. The as-
sistant pulls down the lower edge of the wound with forceps and
the operator thoroughly dissects off the muscular fibres which
cover the upper third of the tarsus. When bleeding stops, black
sutures, three or four, are inserted by a curved needle through the
skin at the lower edge of the wound, then through the superficial
fibrous tissue of the upper part of the tarsus and made to dip into
the aponeurosis just above its upper edge and finally emerge
through the skin at the upper side of the wound. An assistant
draws up this edge meanwhile. No muscular fibres must be in-
cluded. The loop, when tightened by a surgeon's knot, draws the
skin both above and below to the upper border of the tarsus and
the aponeurosis, and fastens it down upon it. A lever action is
thus exerted wrhich tips up the ciliary border. Unless this effect
ensues, the method has not been correctly followed, or the tarsus
has undergone so much distortion as to make it inapplicable. Con-
siderable reaction follows: the sutures remain in situ two days^
never more than three, for suppuration must be avoided. Some-
times, when very redundant, a narrow strip of skin is removed, but
usually this is needless. It may be requisite sometimes to simulta-
neously perform canthoplasty. See Fig. 95. There is no doubt
266
DISEASES OF THE EYE.
about the great value of this method, and it shares with Green's
operation well-deserved confidence. Its fundamental idea was em-
bodied in an operation by Anagnostakis in 1857, but less satisfac-
torily than by Hotz.
With Green's1 operation my own experience has been highly
satisfactory. The lid is everted and held by the fingers, and an
incision made through the
entire thickness of the tarsus
upon its conjunctival side,
parallel to the lid border and
about two millimetres above
it, and extending from end
to end. A round - pointed
scalpel is used by Green. I
usually take a Beer's catar-
act knife and push the point
through. Next a strip of skin
not more than one and a half
or two millimetres wide is
removed along a line about
one and a half millimetres
above the cilia. Muscular
fibres are left intact, to aid
in maintaining by their vascularity the vitality of the lid border.
By a curved needle A (see Fig. 96), the sutures are carried out as
shown in the diagram from the conjunctival side of the cilia
through the free edge of the tarsus just above the lower border
of the skin wound. The thread is drawn through, the needle re-
entered through the muscular fibres upward along the outer surface
of the tarsus, at the point B, going in deeply, and emerges about one
centimetre or more (about half an inch), higher up, through the skin.
When the sutures, usually three in number, are tied, the skin wound
is closed and the ciliary border is everted. An additional security,
on which, in my experience, great stress is to be laid,
consists in turning the eyelashes back upon the skin
and holding them down by collodion spread upon a
few fibres of cotton laid parallel to the lid border.
This dressing of cotton fibres and collodion rigidly
holds the lashes in the desired position, closes the
wound hermetically, and permits in many cases the
withdrawal of one or more sutures when the collodion
is dry. All sutures may be removed in twenty-four hours and a
fresh dressing of collodion and cotton applied. The tarsal wound
gapes widely and fills in a few days with granulations. Cantho-
plasty may be combined with the operation.
Fig. 95.—Represents a Vertical Section of the Upper
Eye-lid. s, supra orbital margin; to, fascia tarso-or-
bitalis; po, pars orbitalis; pc, pars ciliaris of orbicularis
muscle; £, tarsus; c, »»ye-lash; /. lower border, a, upper
border of the wound; a, b, passage of suture through
aponeurosis.
• Trans. Awer. Ophth. Soc, 1880, p. 167.
THE EYELIDS AND CONJUNCTIVA. 267
Green's operation is suited to cases of all grades. It does not
involve risk of sloughing of the ciliary border as sometimes happens
with Arlt's operation. Hotz's operation is available after other
methods have failed of success and especially if, as too frequently
happens, considerable skin has previously been removed. It can be
applied to the lower lid, but should not be done upon both upper and
lower lids at the same sitting, because reaction would be severe.
From Green's operation little reaction ensues, and it likewise is.
effective on the lower lids. A choice between these two methods,
which in the writer's view are superior to all others, is to be de-
cided by the peculiarities of a given case and by the preference of
the surgeon.
Very many methods besides the above have been devised and are figured
in text-books. Among them Snellen's is certainly good, but the writer finds
those described adequate to all needs. He may be allowed, however, to men-
tion a proceeding once employed in a desperate case, where great shrinking
and shortening and tightness of the lid, from which all lashes had been re-
moved, produced by its friction extreme vascularity and opacity of the cornea
with distortion of its curve. Various operations had been done. The in-
dication was to loosen the nip of the upper lid. The forefinger was pushed
under the lid to the top of the scanty conjunctival sac. A narrow knife was
thrust under the skin on the middle line of the lid flat-wise and when the
point had got above the upper border of the tarsus the edge was turned to-
ward the operator's finger and the point caught in the nail, then in drawing
down, the tarsus was split in the vertical line into two halves and they sprang
asunder several millimetres. The pressure on the cornea was relieved, no
deformity ensued, for the skin was not cut, and many months afterward the
cornea continued free from vascularity and irritation.
The total ablation of the ciliary border has been formerly much
practised, and it may in extreme cases be appropriate; but " scalp-
ing the lids " leaves hopeless deformity and is a slur upon surgery.
Only absolute necessity justifies its employment
Ectropium.
Permanent eversion of the lids arises, 1st, from chronic in-
flammation and hypertrophy of the conjunctiva, especially of
the lower lid in old persons—a condition due to relaxation of the
skin with spasm of the orbicularis muscle. It sometimes occurs
during recovery from operations, as well as spontaneously. Treat-
ment consists in excising a suitable strip of thickened conjunctiva
close to the ciliary border, and perhaps removing a V-shaped por-
tion of the lower lid at the outer canthus, to draw the lid up to the
globe as well as to replace it. The excision at the outer angle is
only needed when the lid droops and the amount removed will de-
pend on the degree to which it falls. Sometimes the destruction of
'268
DISEASES OF THE EYE.
conjunctiva by Paquelin's cautery is all that is required, and the
slough is left to separate. No bleeding happens and by the help of
■cocaine, 10$ solution, the operation is very simple and easy. A fine
point must be used and a deep furrow made.
The second and more fuequent type of ectropium is caused by
wounds, by burns, by caries of the edge of the orbit with adhesion
of the skin, by removal of tumors, etc. The amount of deformity
is extremely variable and after burns and explosions may be
frightful.
The following general suggestions in treatment are appropriate:
While some kind of operation will be required, none which involves
transplantation of skin should be done, until all tendency to con-
traction of cicatrices has disappeared. While ulcerated surfaces
are in process of repair, no operation is feasible, except the intro-
duction of a flap either with or without a pedicle. Usually we wait
until healing has occurred. The exceptions will be after severe burns.
Yet it might sometimes mitigate deformity and protect the cornea,
to pare the edges of the upper and lower lids behind the cilia and
stitch them fast, so as to obliterate the palpebral slit, save at its
extremities. The tendency to ectropium would in some measure
be thus counteracted until healing had become complete and con-
traction of scars had done its worst. It is easy to separate them
when the time for operation arrives.
Scars of moderate extent will yield to persistent traction; if
attached they become looser and stretch. Subcutaneous division
will not release them, because adhesion will be re-established; but
it may render traction more effective.
In a case of great deformity by loss of a large portion of the
lower edge of the orbit at its temporal side, the deep hollow was suc-
cessfully filled by inserting a thick flap shaped like a finger from
the temple and covering it over by the thin integument of the
diseased locality. An incision was made parallel to the border,
the skin undermined freely and united by sutures above the
buried flap. Union was satisfactory.
Certain general rules are to be observed in plastic surgery.
Adherent scars should be excised, or they should be buried under a
flap of skin; if not, adhesion will return. Scar tissue should not be
included in a flap unless the scar is very superficial and the true
skin has not been destroyed. An apparent exception is in the case
of the Wharton Jones sliding flap, where the whole ciliary border
furnishes vascularity, yet sloughing of the tip of the flap is the risk
of the proceeding. The existence of a syphilitic dyscrasia is preju-
dicial to success. I once met a sad disappointment in a young girl
who from hereditary syphilis had lost the nasal bones; attempting
THE EYELIDS AND CONJUNCTIVA. 269'
to make a new nose by flaps from the cheeks, the wounds showed no
disposition to unite and underwent a torpid and unhealthy process
of suppuration; the flaps shrivelled and the condition was worse
than at the first.
In constructing and placing flaps, allowance must be made for
shrinking, which will amount to about one-fifth in length, and further
shrinking will occur after the healing. Lines of union must be so
planned, if possible, as not to reproduce the deformity as shrinking
of the flaps slowly proceeds, but act in the contrary manner.
Again, flaps will sometimes grow thicker during subsequent weeks
if they be very loose. In plastic surgery of the face, one must fit
parts as a tailor fits his cloth and not fear to cut out redundant
material or to smooth down elevations. By a little ingenuity
puckers and welts need rarely be left.
In ectropium the ciliary border will be elongated and must often
be shortened, and this is done by excision of a triangle of skin
whose base is at the free border
of the lid and preferably at the
outer angle, while sometimes it
may be done at the middle. The
operation to be performed in a
given case will of course depend
on the conditions to be met, and
each case must be studied by it-
self. Wounds at the inner angle
dragging down the lower lid are
difficult to repair perfectly. In
such a case as is shown in Fig.
97 the scar on the side of the nose should be undermined, the
lid fully loosened and lifted higher up, a portion excised and the
cut edge joined to the inner canthus; then to fill the gap a flap
may be brought from the forehead with pedicle at the root of the
nose and by properly managed lines, very little wrinkling need be
made: vide infra. A small flap could be had beneath the brow. In
both cases the base of the flap is above, or at any rate not below
the site of the gap, and contraction tends to correct deformity. In
such a case a flap from the side of the nose would be unfeasible be-
cause the part beyond the cicatrix would slough. The sutures
must be very fine and numerous and the skin thin.
Fig. 98 shows ectropium of the outer part of the lower lid by
cicatrization without adhesion to the bone. The mode of operating
is clearly indicated. It will be noted that pin sutures are used.
They should be very fine, be inserted well back from the wound,
and are strongly to be commended. In the dissection of the flap
and in bringing together the gap below the lid, the skin must be
270 DISEASES OF THE EYE.
freely undermined on all sides. The cutaneous incision may be ex-
tended right and left close to the border of the lid. The same pro-
ceeding may be applied at the outer canthus and the lines be
Fig. 98. Fig. 99.
made more oblique outward and downward. This is a most im-
portant suggestion. (See Fig. 99.)
Another principle applies to more extensive deformities of this
kind, both in the lower and upper lids. It is the operation of Whar-
ton Jones, who intended it for the upper lid. (See Figs. 100 and 101.)
The length of the flap is determined by the degree of deformity,
and its dissection toward the conjunctiva must not be carried too
far lest sloughing occur. In the figures the dissection does not go
as far as it might. It is suited only to cases of moderate degrees of
ectropium, and is to be chosen when the skin has not been deeply
burned. The insertion of flaps is advisable for the greater degrees
Fig. 100. FlG. 10i.
of deformity. Von Ammon figures the same method on a larger
scale. I must again repeat the caution that the method must not
be pushed too far.
There are many devices which may be employed in dealing with
THE EYELIDS AND CONJUNCTIVA. 271
ectropium. A most ingenious one involving the formation of two
flaps, one of which fills the opening made in bringing the lid border
to its place, and the other fills the vacancy whence the first flap
was dissected, we owe to Richet. It meets cases of caries at the
outer edge of the orbit, resulting in dragging down the outer can-
thus besides ectropium of the lower lid. To supplement it, the
edges of the lids are pared and sewed together, not, however, de-
stroying the cilia. See Meyer on Diseases of the Eye.
Blepharoplasty.
The transition from restoration of the lid by some method of
displacement to its reconstruction by introduction of new skin, is
most natural. Many cases of ectropium require the latter method,
and for its accomplishment we may, 1st, use a flap from the adja-
cent parts kept in connection with the general circulation by a
pedicle; 2d, we may transport a very thin piece of skin from a re-
mote part and apply it in a wound (Wolfe's method); 3d, we may
make use of small bits of skin, grafting them into the wound as a
mosaic, the method of Reverdin; 4th, we may put in a thin shaving
of skin by Thiersch's method.
The last two methods are suitable where the skin has been
widely destroyed and healthy material for flaps is not available.
They can also be happily employed during the granulation stage of
burns to prevent or mitigate deformity. Partial closure of the
lids by tarsoraphy may simultaneously be resorted to. The pieces
(Reverdin) must be thin, say about two millimetres square, and as
numerous as occasion demands. When in place they should be
covered with gold-beater's skin, over this a pad of lint, and all held
firm by plaster or a bandage. The outer dressings may be removed
once daily, and the state of the grafts will be seen through the
gold-beater's skin. They take on a white, sodden appearance, then
become red and seem lost in granulation, but soon a healthy cica-
trix, i.e., epidermis appears. The proceeding may be repeated.
Analogous to grafting is the transplantation of a considerable
piece of skin from a remote part. This proposal emanated from
Mr. Wolfe, of Glasgow, 1875. The skin may be taken from the
chest or the inner side of the arm. A pattern the size of the place
to be filled, is to be laid down, and the outline of the piece to be
transported must exceed it by eight to ten millimetres all around.
Subcutaneous connective tissue is to be completely removed and
this is most easily done while the piece is being lifted. When put
in situ all bleeding must have stopped and fine sutures introduced;
cover it with gold-beater's skin, then with collodion, and over all
cotton or a bandage. The parts must not be disturbed, although
272
DISEASES OF THE EYE.
the bandage may be taken off in two days. The gold-beater's skin
and collodion will remain unchanged for many days. The epider-
mis comes off and the piece looks very white; sometimes it becomes
red, moist, and like ordinary granulation tissue. It at length as-
sumes a firmer character and may correct the deformity. Such
material is, however, subject to an extreme amount of contraction.
A flap 3^ inches long by If inches broad has after three months
been reduced to 1| x f inches. The tarsal margins may be united
for several months to counteract the shrinking. It is both inter-
esting and surprising that in so many instances (at least twenty
are recorded) the flap survives and gives desired relief, yet it some-
times sloughs, as twice has happened in my own experience. The
conditions of health are extremely important, and that the flap be
free from fat and connective tissue, that it fit accurately, that no
hemorrhage shall occur beneath it, and that it lie absolutely undis-
turbed for a week. If the operation fail, some other method may
be tried, or it could even be repeated. The place from which the
skin is taken may take several weeks in healing. The flap, accord-
ing to Thiersch's method, is less liable to shrink, because the shav-
ing is thinner, being taken by a razor, and is no more prone to
slough. It is more simple.
In a large class of cases, particularly where neoplasms are re-
moved and more or less of the lid is sacrificed, other methods for im-
mediate repair must be employed. Without attempting to describe
all the possible ways of meeting difficulties, certain proceedings will
be illustrated which have been found effective in my own experience.
There are several situations from which flaps may advantageously
be taken: 1st, from the temple; 2d, from the forehead on the median
line; 3d, from the side of the nose, the naso-buccal flap. All these
are available for both the upper and lower lids. 4th, a flap may also
be taken from the region in front of the ear, whose base shall be
above and near the zygoma. This is in a direction opposed to the
course of the blood-vessels and in so far is disadvantageous, yet if
necessity compel, it may be employed. A vertical flap from the
temple whose base is below, may be allowed to run a little distance
into the hair, and the hairs will to a great extent fall out and dis-
appear by obliteration of the follicles. Sometimes the place from
which the flap is taken, in the temple for instance, cannot be closed
by sliding the adjacent skin—in the scalp this is easily remedied by
moving forward a second flap to fill the gap, and the vacancy thus
caused may be left to granulate. The scar which results is pushed
so far back as to be covered by hair and be unobjectionable. Even
on the face this device may sometimes be practised.
A flap from the temple, when designed for the lower lid, should
THE EYELIDS AND CONJUNCTIVA. 273
be more or less vertical and the pedicle be a little higher than
the level of the lid. The cut on its lower or anterior side should
run farther back and down than on the upper, and if much of a
wrinkle occurs in turning it down, a triangle may be excised, and
sufficient breadth of pedicle is to be allowed to permit it. Small
trimmings should always be done at the time of operating, and ex-
tensive undermining practised to liberate the neighboring integu-
ment. The shortening of a flap after being lifted will usually
amount to about one fourth its length. Accurate coaptation by
numerous very fine black silk sutures and by fine pins, so-called
insect pins, where any tension occurs is indispensable. All bleed-
ing must first be staunched. The lines of union may be covered
with contractile collodion mingled with fibres of absorbent cotton,
which makes an air-tight, inflexible dressing of the utmost value.
Adhesive straps are wholly needless. I never use iodoform and, if
antiseptic dressing is required, depend on absorbent cotton soaked
with sublimate solution 1: 3,000, retained by a flannel bandage; it
is kept constantly wet and need not be renewed oftener than twice
in twenty-four hours. Sutures are allowed to remain three to seven
days; pins to be removed on the second or third day. If any ten-
dency to suppuration appear about the pins or sutures, remove
them, wash well with sublimate solution, and either continue to
apply it, or sometimes the collodion and cotton dressing may be
reapplied. If at an early date the wound reopen for a short space,
insert a silk suture. Such are some of the suggestions suited to all
plastic operations.
Another mode of forming a lower lid is by horizontally sliding
a flap forward, from the region of the zygoma (Knapp), and it may
if needful, be met by one from the side of the nose. An instance is
represented in Figs. 102 and 103. The divergence of the lines of in-
cision at their base is very important. The case was one of can-
cerous growth.
The naso-buccal flap is proper for repair of the inner portion of
the lower lid, and less satisfactorily for the upper lid. If the tissues
are very loose, a considerable loss of substance can be repaired at
the nasal side of the lower lid without making any flap. In Fig. 104,
page 275, excision of an epithelioma removed the inner half of the
lower lid and skin over the region of the lachrymal sac. The
deficiency was supplied by merely sliding up the cheek which was
very lax. A vertical cut was made down to the ala nasi along
the naso-buccal furrow; an incision close to the border of the
lower lid was carried horizontally backward almost to the root
of the zygoma. The whole cheek was undermined, taking care to
avoid the duct of Steno, and carried forward and inward and at-
tached to the inner canthus. No conspicuous scars were left.
18
274
DISEASES OF THE EYE.
The photograph was taken very soon after recovery, before swell-
ing had subsided. Sometimes the incision backward may begin at
the outer canthus and the remaining part of the lower lid be carried
inward with the loosened cheek. Free division of the external can-
thai ligament must always be practised when the lid is slidden.
If the inner part of the upper lid is also involved, we must take
a flap from the glabella, or may slide one across the root of the
nose, relying on the vessels of the other half of the face. Figs.
106 and 107 represent a case before and after operation where slid-
Fig. 102.
ing the cheek inward and upward was combined with a flap turned
down from the glabella. The ultimate result was comely. I
once combined the operation of sliding the cheek with the intro-
duction of a transported flap (Wolfe's method) to fill the lack at
the inner part of the upper lid, but the piece sloughed and I was
obliged to slide a flap from above the root of the nose.
The naso-buccal flap can be made to form an upper lid. See
figures illustrating case reported in "Trans. Fifth International
Congress of Ophthalmology," 1876. (See page 277, Figs. 109 to 112.)
A flap from the middle of the forehead can be utilized for the
upper lid and for defects at the inner canthus and of the inner part
of the lower lid. Fig. 105. represents a case where epithelioma had
Fig. 103.
THE EYELIDS AND CONJUNCTIVA.
Fig. 104.
Fig. 105.
Fig. 106.
Fig. 107.
276
DISEASES OF THE EYE.
attacked the region of the inner canthus and begun to penetrate
the orbit; the inner half of the lower lid, the caruncle and inner
third of the upper lid had to be removed. The flap taken from the
forehead was so arranged that its base was laid upon the opposite
side of the root of the nose; the arterial supply coming from the
trochlear branches and angular artery of that side. Before it was
sewed down the wound in the forehead was closed after undermin-
ing the skin freely, the brow7s wrere slidden to each other, the gap
in the upper lid was easily closed, and then skin was removed to
make a bed in which the flap
could lie smoothly and with-
out traction. By carrying
down the incisions for the flap
to the opposite side of the na-
sal bones, the objectionable
wrinkling of the flap is entirely
avoided and it lies so smoothly
that it is hard to be detected
after a few months. This man
was exempt for five years from
a return of the disease, but
now, seven years afterward, it
has reappeared extensively
and entered the orbit.
How such a flap may serve
for the upper lid is shown in
Fig. 108. The photograph was
taken seventeen days after the
operation and the same plan of
reaching across the nose can be
recognized. The relief was com-
plete. The ectropium had been
total and had lasted nine years.
Various other methods must be resorted to as exigencies arise,
and they are described in text-books, but I have stated the means
on which I rely.
Blepharophimosis, or narrowing of the palpebral opening, is
generally the result of chronic trachoma. Its hurtful effect is by
pressure of the lids on the cornea, and it is to be relieved by two
methods of operating. The more usual one consists in splitting the
lids apart at the outer angle and releasing them from their attach-
ment to the bone by clips of the scissors through the canthal liga-
ment, and finally uniting the conjunctiva to the skin by three fine
sutures. This is canthoplasty as designed by Von Ammon, and
it is often done as auxiliary to operations for entropium. Instead
THE EYELIDS AND CONJUNCTIVA. 277
Fig. 109. Fig. 110.
Fig. 111. Fig. 112.
Fig. 109.—The man's head and face had been burned by kerosene oil. There
were numerous ulcers in the scalp and on the forehead and such deep cica-
tricial tissue on the forehead that the best available material was by the naso-
buccal flap. The blood-vessels to nourish it were chiefly those from the op-
posite side of the median line. The ectropium of the upper lid was complete,
and the flap had to be about four inches long. Besides inserting the flap and
uniting the gap in the face, the edges of the upper and lower lids were fresh-
ened by paring their edges behind the cilia for two-thirds their length and
then uniting by sutures. Primary union was obtained to a sufficient degree
and the cornea was covered. As seen in Fig. Ill, considerable thickening
and irregularity remained, which was greatly remedied by a subsequent
operation.
Had not the lid borders been united, the deformity would have largely re-
turned. Many months after, the appearance was seen in Fig. 112. The outline
of the incisions is shown in Fig. 110. A shorter flap will sometimes suffice,
but the incisions are the same in all such cases. The dotted line under the
lower lid shows how far this cut was carried, and the whole cheek was laid
back. The tip of the flap was cut off to give it proper shape.
278
DISEASES OF THE EYE.
of cutting the lids apart with scissors, it is better to separate them
by a speculum and push a Beer's cataract knife or Aery narrow
bistoury into the edge of the lid at the outer angle, splitting up and
down along the border, and then cut the skin in a straight line out-
ward, but leave the conjunctiva undivided. The external canthal
ligament will be severed by a few strokes of the scissors to free the
lids, and then the conjunctiva united to the skin.
In very severe cases of shrinking of the conjunctiva with
blepharophimosis and perhaps with entropium, a plastic operation
may be needed and this I have called canthoplasty, while the above
operation might be known as cantholysis. The lids are split apart
for half an inch and a flap, with apex upward, is inserted; to obvi-
ate wrinkling, the cut is extended downward as required, both on
the anterior and posterior sides.
Tarsoraphy, or the uniting of the edges of the lids, has been re-
ferred to. It is called for in total paralysis of the facial nerve
including the orbicularis muscle, and may unite one-third of the
length of the lids at the outer angle. Complete union of the edges
of the lids is done as an auxiliary sometimes to operations for ectro-
pium. The method is simple, viz., paring off the posterior angle of
the tarsal borders by a Beer's cataract knife and uniting by stitches.
If the lids are to be again separated, a point must be left ununited.
Injuries and Lacerations of the Lids.
Powder burns occur by accidents in blasting, and may be com-
plicated with other and extensive injuries, as we find among miners;
boys often suffer the penalty of playing with gunpowder. The pain
and inflammation are relieved by cold lotions, while the removal of
the unburn t powder is to be effected by patiently picking out each
little granule with a spud or cataract needle. The process is tedious
and rather painful. Much of the powder will come out with the
desquamating skin, while what lies below the epidermis must be
picked out bit by bit, soon after the burn is produced. Blistering
after the skin has recovered, has been suggested to bring out the
remaining granules, but it does not seem promising.
Lacerations and wounds through the tarsal border may, when
fresh, be easily united by putting a fine pin through the free border
of the tarsus, carefully stitching the conjunctiva and putting in the
skin as many silk sutures as needful. Over all apply the collodion
cotton dressing. If the injury is old and the parts have healed,
perhaps, with ectropium, the edges of the cut must be freshened,
the normal position restored, and the same treatment adopted.
(See Fig. 113 from Lawson; a probe is inserted in the canaliculus.)
A case recently under treatment is illustrated by Fig. 114. An
THE EYELIDS AND CONJUNCTIVA. 279
Italian was cut in the face by a razor. The wound involved the
border of the upper lid, split the cornea and passed down through
the lower lid three inches upon the cheek. The wound of the face
was sewed up before he entered the N. Y. Eye and Ear Infirmary.
There was prolapse of the iris and the lens was partially cataract-
ous. The prolapse of iris was excised. In healing, adhesion of the
inner border of the pupil, as well as of the lens capsule to the cornea,
took place. Gradually by contraction of the thick scar on the cheek
which had united with suppuration, the lower lid became everted.
This was after many weeks relieved by excising the whole length
of the cicatrix, undermining the cheek on either side of it, and at its
upper extremity making incisions right and left at a point about
one quarter of an inch below the lid border, stretching up the lid
Fig. 113.
to open out the horizontal wounds and pulling them together from
the sides. This raised the tarsal edge to place and brought the
lid in apposition with the globe.
More than once I have known the lid torn away from the inner
canthus by a button-hook which a young child had thrust under
its own lid. The tarsus is detached from the superior conjunctival
sinus, and in replacing it, sutures must be first inserted on the
inner surface through the tarsus and conjunctiva to secure its re-
placement, and as a separate step the wound of the skin united.
In a recent case no deformity need remain. Very recently a girl
four years old inflicted the same injury upon her lower lid, tear-
ing it off for two-thirds its length, and when brought to me ten
days later, suppuration was established. It was necessary to
freshen the edges of the wound, to extend it farther outward along
the zygoma to compensate for the shortening due to its having
flapped loosely for so long, and carry it well inward. Fine silk
Fig. 114.
280
DISEASES OF THE EYE.
sutures and the collodion cotton dressing held the parts immov-
able for a week, and despite the tardiness of effective surgical
help, union per primam was obtained with no deformity. Such
accidents happen by bursting of soda-water bottles, by falling upon
butchers' meat-hooks, by the thrust of a stick or of the horn of a
cow, etc. Sometimes the levator palpebral is torn off, and the
walls of the orbit may be fractured. Treatment must be adapted
to the various exigencies which arise. If a canaliculus is torn, one
may have to search for the remaining portion and open a way into
the sac, which probing a few times with a lachrymal sound will
keep patulous. If this be impracticable, the uninjured canaliculus
will usually fulfil the physiological requirements.
A blow or fall upon the brow often produces a wound of the skin
which does not externally indicate its real severity. It may seem
small and likely to heal by first intention, but suppuration and
great swelling often occur because the skin is cut beneath by the
sharp edge of the bone. Sutures are of little use and indeed may
be harmful; warm poultices and antiseptic lotions are proper until
the reaction subsides.
Ecchymosis of the lids happens by blows and falls and some-
times from straining in whooping cough or from senile degenera-
tion of the vessels. If moderate in amount, stimulating lotions may
in the beginning do good, such as alcohol and water, or as follows:
1> Tinct. arnicae, 3iv.; Liq. ammonii acetatis, 1 i.; Aquae, ad 1 viij.
If there be a large clot, the practice of the prize ring is to tap it
with a lancet and squeeze or suck the blood out. The proceeding is
not strictly antiseptic, but evacuation of part of the clot is some-
times good practice, care being taken to suture the wound or close
it with collodion and cotton. After a few days no applications can
be useful, save the artist's skill who " paints your eye while you
wait."
Coloboma of the lids is a congenital deformity which rarely
occurs and usually affects the upper lid. It may be on the middle
or nearer the inner end, as in a case which fell under my notice.
There may be no other defect of structure. But much more exten-
sive deformities are seen with arrest of development of the face.
Treatment by paring the edges and uniting the opening by su-
tures, as in wounds, is obvious.
Epicanthus, sometimes called encanthus, is also a congenital
malformation whose name is derived from crescentic folds of skin
which run downward from the inner end of each brow, skirting the
inner canthus and disappear over the lachrymal sac But in many
cases several other conditions coincide with this and are of more
serious moment. These are, deficient development or absence of the
levator palpebrae superioris muscle, narrowness of the palpebral
THE EYELIDS AND CONJUNCTIVA. 281
slits, with a tendency to slope upward at their outer extremities,
great flattening of the ossa nasi. The deformity varies in degree,
is apt to be hereditary, and to be associated with defects in the de-
velopment of the eye, such as hypermetropia, or albinism, or nys-
tagmus. I have seen three cases in one family. A striking case is
figured by Von Ammon. The frontal portion of the occipito-fron-
talis muscle is constantly called into play as in cases of paralytic
ptosis, and the head has to be thrown backward to look at objects
which lie even a little below the horizontal meridian. In some in-
stances nothing can be done, or requires to be done, while in other
cases the wrinkle at the inner canthus is unpleasantly conspicuous,
and can be remedied to some degree by excising from the root of
the nose an elliptic piece of skin, and undermining the adjacent folds
very freely, so as to loosen them from their periosteal connections.
The wound is closed by harelip or fine pin-sutures, and a vertical
scar remains on the median line. Besides this, it is sometimes use-
ful to perform the usual operation of canthoplasty at the outer
angles. I have a photograph of a child who had a high degree of
this deformity and aggravated converging squint. Nothing was
ever done for her, because her drunken mother found the pitiful
condition of the child a means of profitable mendicancy.
Affections of the Muscles of the Lids.
Spasm of the orbicularis occurs in two forms: the tonic or
partial, and the clonic The first affects only a few fibres, is hardly
perceptible, and is merely a trifling annoyance. It comes from
strained accommodation, or slight conjunctival irritation; rest and
general tonics are appropriate.
The clonic form is sometimes called facial tic and is discussed in
treatises on nervous diseases (see Gowers). It is paroxysmal and
painful and the spasm often seizes upon other facial muscles. I have
seen several examples of this affection. The face is thrown into
ludicrous and painful grimaces, and the spasm is excited by very
slight irritations. In the case of a car-driver who had two attacks
of it under my observation, it once seemed to be produced by the
severe cold of the winter wind. Only one side of the face was af-
fected, and when the attack came, it would be thrown into extreme
convulsions: the skin would become red, the mouth be drawn up,
the lids lightly shut, and he would suffer severe pain. Such
turns would happen many times a day, and they recurred during
several weeks. He appeared to derive benefit from full doses of
bromide of potassium. I have seen a clergyman whose whole face
was thus contorted in interrupted paroxysms, and for whom reme-
dies were of little value, but after several years I was happy to
282
DISEASES OF THE EYE.
find that his affection had become almost imperceptible. Aconitine
and gelsemium are remedies relied on, but materia medica is often
useless. A less serious spasm, which is a kind of nictitation, may
occur as an unconscious habit or trick. It may be confined entirely
to the eyelids, and be a congenital and life-long peculiarity. A dis-
tinguished sculptor of my acquaintance is thus affected, and has a
little impediment of speech. A lady friend had it for seventy years,
and her powers of speech were not impaired. Sometimes there will
be a point of tenderness over the supra- or infra-orbital nerves, or
on the temple, and perhaps at the back of the neck, or the cause of
re Ilex irritation may be a defective tooth. Should such tender spots
be found, they suggest indications of treatment, such as neurotomy,
local anaesthesia, counter-irritation, hypodermic injection of mor-
phia at the place of tenderness, etc. The disease is often cerebral,
either cortical or nuclear. See Gowers, p. 660, Am. Ed. In many
cases no treatment avails.
It is important to bear in mind that sometimes the intensity of
the symptoms can be greatly relieved by removing optical and
muscular errors. While there may be some positive nerve or cen-
tral lesion, removal of peripheral exciting causes may greatly abate
the distressing symptoms. The analogy of such cases with as-
thenopia and epilepsy is obvious. The following case is in point
and more briefly is referred to on page 209.
Dr. D. Yv., 47, first seen Dec. 29th, 1889. His father's grandmother became
insane after the accidental death of her husband. His father has been twice
confined in an insane asylum. The doctor himself resembles his mother, in
whose family there is no insanity. No history of chorea or epilepsy on either
side. Dr. W. has always been easily excited, quick to take offence, but is
now much better able to control himself than in his younger days. Never
had syphilis, rheumatism, or gout. Children strong and healthy.
Dr. W. has suffered much from malaria, which manifests itself particularly
in hemicrania and frontal headache, recurring every two weeks, but con-
trolled by quinine.
In the summer of 1881 he first noticed twitching of the right orbicularis
muscle and consulted Dr. Weir Mitchell who suggested nerve section, but
referred him to Dr. Thomson, who thought the trouble was reflex and cor-
rected his refractive error under duboisia, viz.,
0. D. + 1.00s. C + 1.00c. ax. 10° V. = l.
O. S. + 1.75s. . . ax. 180° V. = 1.
The correction was ordered for reading only. "While using duboisia he
was free from the spasmodic action of the orbicularis and was somewhat re-
lieved by the use of the glasses for a time. Later on the trouble increased,
so that now, Dec, 1882, all the muscles of the right, side of the face twitch,
the twitching being most marked in the orbicularis, wing of nose, and angle
of the mouth in the order mentioned. There is tremor of the ocular muscles,
producing nystagmus which prevents a view of the fundus of the right eye.
There has been diplopia. After prolonged twitching there is numbness of
the right side of the face. There is now tenderness over the supra-orbital,
THE EYELIDS AND CONJUNCTIVA. 283
infra-orbital, malar, mental, and palatine twigs of the right fifth nerve and
also over the styloid process, with hyperesthesia of the right side of the face.
Without atropine,
0. D. + ^sc. ax. 10° V. = f£.
O. S. + ^c. ax. 20° V. = U- '
The above glasses were ordered for distance with an additional + ^s. for
reading. R Pot. iod. in increasing doses.
Mar. 5th, 18^:5.—Has been much relieved by distance glasses. Less tender-
ness over branches of supra- and infra-orbital nerves, although the spasms
occur at night with considerable severity. Had tried pot. iod., hydriodic
acid, and galvanism without effect. Has seen Dr. Janeway, who thinks the
lesion may be thickening of a nerve sheath.
Feb. 15th, 1888.—The glasses greatly relieve the spasm and he is unable to
get along without them. The twitching is excessive if he leaves them off,
and is confined to the right side of the face as before; it is greatly aggravated
by any excitement. The eyes tire more quickly in reading than from any
other cause and this provokes the spasm. He is often obliged to close the
right eye in reading. The twitching is at times so excessive that he has great
difficulty in getting to sleep. At times there is twitching of all the muscles
of the right side of the face with slight numbness. Occasionally there is
ptosis (right) and considerable rubbing and manipulation is required before
he is able to open the right eye. Diplopia is an occasional symptom. Drugs
have proved almost useless, although the bromide of ethyl and a 4£ solution
of cocaine are, however, of temporary benefit. He has also been somewhat
relieved by galvanism.
Out of doors he rarely has any trouble. The nose is prominent, limiting
the field of binocular vision to about 45°. Movement of O. D. outward is
limited and irregular; movement of O.S. outward is also limited. Prisms
with bases out render objects much more steady and distinct; without them
he sees the gas jet as if through a fog.
18' abd. = 3° add. = 13° v. d. = 5* latent conv.
13" gl. abd. = 10°+ add. = 15° v. d. = 3° conv.
Javal's ophthalmometer shows:
O. D. 0.5 ± ax. 55°and 145°.
O. S. 0.5 ± ax. 90° and 180°.
3 O. D. 4- 0.75c. ax. 10°CPrism H°, base out.
O. S. + 0.50s. 3 + 0.75c ax. 20" C prism 1|, base out.
June 18th, 1888.—Has found above glasses " wonderfully restful." Before
wearing them could hardly get to sleep on account of the twitching, and then
only by pinching up the skin over the right temple. Now has no trouble in
getting to sleep unless very tired. Has much less twitching and less ptosis
than formerly. Sept., 1890.—Still relies on glasses. Twitching grows worse.
Paralysis of the Orbicularis Muscle, or Lagophthalmus—
causes annoyance by exposure of the cornea. The eye is fretted
by external irritation and overflows with tears which cannot be di-
rected into the lachrymal puncta. Both the tensor tarsi and or-
bicularis are flaccid and the lids fail to hug the globe. Chronic
conjunctivitis, and even inflammation of the cornea are to be ex-
284
DISEASES OF THE EYE
pected. The cause lies in lesion of the seventh or facial nerve. It
is not uncommon for the orbicularis to escape when other muscles
supplied by the inferior branches of the facial plexus are involved;
but, if the orbicularis is paralyzed, all other muscles are also apt to
suffer. The causes of facial paralysis are peripheral, or lie along
the track of the nerve, or are in the brain. From the crookedness
of its course, and the variety of tissues which it traverses, the nerve
is greatly exposed to injury and it may be wholly or partially af-
fected. One need only remember that diseases of the ear, and of
the lymphatic glands, aud of the parotid, are all liable to do mis-
chief to the facial nerve. As to the cases of cerebral disease, Eulen-
burg says that facial paralysis, originating from lesions of the pons,
involves the orbicularis; while, if it proceed from the cerebral ped-
uncles, or from the central ganglia, or from progressive paralysis
of cranial nerves, or from spinal cord affections, the orbicularis is
likely to escape.x Peripheral paralysis of the seventh nerve usually
includes the orbicularis. The cases of partial impairment are most
common. I have seen two instances of total paralysis caused by
wound in the space between the angle of the jaw and the mastoid
process. In the case of a mason the wound was inflicted by falling
on his own trowel. I have seen this patient sixteen years after the
wounding and found partial recovery of function in the facial mus-
cles. For seven years the outer half of the palpebral fissure was
closed by tarsoraphy and then they were fully reopened. At this
date (January, 1889) he has power to close the lids enough to pro-
tect the cornea. Union of the ends of the nerve has doubtless oc-
curred.
Treatment will be governed by the supposed cause of the lesion.
The remedies to be used will suggest themselves. If the cornea be
much exposed, it may be necessary to wear a bandage, or to partly
close the lid by a strip of plaster near the outer canthus. During
sleep the lid will drop a little from its own weight, because the leva-
tor is relaxed and the cornea turns up so as to be covered, even
when paralysis is total. One of the aggravations of the trouble
comes from the frequent wiping of the eye to get rid of the tears,
and the lower lid is dragged down to an additional degree, and may
pass into permanent ectropium. When this state arrives, relief
will be afforded by paring the edges of the lids for ten to fifteen
millimetres at the outer canthus, and uniting them by sutures to
shorten the palpebral opening (tarsoraphy). The cilia are left
untouched. The sutures should be left in situ from four to six
days. I have lately seen a man for whom I did this operation seven
years ago, and the relief it gave continued until within a few
months. The continued paralysis and the drag of the lax tissues
1 See Gowers1 " Diseases of Nervous System," p. 657.
THE EYELIDS AND CONJUNCTIVA. 285
finally brought on troublesome ectropium of the lower lid at the
inner canthus, with a return of the former epiphora. I per-
formed another operation of tarsoraphy at the inner canthus,
in a different manner, as follows: I dissected up a parallelo-
gram of skin above and below the canaliculi, for a space which
reached from the commissure to three millimetres beyond the
puncta. I turned the raw surfaces of the little flaps, raised from
the respective lids, against each other and stitched through them.
The puncta were thus turned inward and out of sight. The edges
united, and the palpebral slit was left as a narrow oval through
which the pupil could peep, and the annoyance of the epiphora was
removed.
Ptosis.—There is a so-called spurious ptosis or drooping of the up-
per lid due to chronic trachoma and which remains after the disease
is practically cured. The lid can be raised, but not to the full height.
The levator is not paralyzed, but its action is hindered. Genuine
ptosis indicates partial or complete paralysis of the levator palpe-
bral superioris. It may concur with impairment of other muscles
supplied by the third nerve, or be isolated. If complete, the upper
lid covers nearly all the cornea, and is raised only by extreme con-
traction of the occipito-frontalis, lifting the brow, and by trac-
tion on the skin pulling, the lid up enough to enable the patient to
peep under it, when he throws his head backward. The attitude of
the head, when such patients attempt to use their eye, is highly
characteristic. The causes of the disease are peripheral or central;
the most frequent is syphilis. It may be well to remark that a
little drooping of the lids may be congenital, and may be confined
to one eye or affect both. The true action of the levator is con-
spicuously suggested, when the only muscle able to act on the lid is
the occipito-frontalis. The latter simply stretches the skin, and if
lax, can exert but little effect, while the levator pulls from the cavity
of the orbit and rolls the lid over the convexity of the globe, and at
the same time causes a furrow in the skin.
The treatment of ptosis consists, first, in combating the cause if
this be ascertained; second, in stimulating the muscle by the fara-
dic current of electricity; thirdly in operating. As to the first in-
dication, we give iodide of potassium in small doses, gr. v. ter in die,
for supposed rheumatic cases, and in larger doses with mercurials
in syphilitic cases. This treatment should be held to, for four or
six weeks. After the first week or two, the battery may be used
for a few minutes, once daily, or as often as practicable. With one
pole behind the ear, the other is placed on the lid, and the current
should be only of moderate strength.
After the lapse of three or four months without adequate im-
provement we may employ an operation. The removal of a por-
286
DISEASES OF THE EYE.
tion of skin and orbicularis fibres is the old mode. Pagenstecher,
1881, suggested the introduction of sutures beneath the skin to run
up to the forehead above the brow, to be tied tightly, and by the
resulting cicatrices after they have cut out, reef up the lid. Wecker
has combined excision with sutures in a manner portrayed in the
figures. A portion of skin and muscular fibres is removed from the
upper half of the tarsus and its breadth will depend on the fulness
of the lid. A strong thread is entered at a above the brow, pushed
beneath it and the skin of the lid, keeping close to the tarso-orbital
fascia, and coming out at the upper edge of the wound; it glides
over the muscular fibres and then dips under the skin and muscle
at the lower edge of the wound, emerging is carried transversely
Fig. 115. Fig. 116.
for a space of about five millimetres and then travels back on a
reverse course up to b. Two such sutures are introduced. They
are tied with a bow knot over a roll of plaster and tightened from
time to time as they grow slack. The wound is thus pulled together
and the due amount of effect is to be attended to. As the sutures
cut through the tissues, cicatrices are formed which hold the lid
up permanently. The proceeding is ingenious and superior to
simple excision of a flap. I have done it with benefit in a case of
ophthalmoplegia exterior bilateralis, where not only ptosis but
paralysis of all the extrinsic muscles existed. The case was con-
genital and the lesion nuclear. If other branches of the third
nerve are paralyzed, it may be inexpedient to cure the ptosis
because the patient will be subjected to the distressing annoy-
ance of double vision.
The drooping of the lid succeeding trachoma may be relieved by
THE EYELIDS AND CONJUNCTIVA. 28?
sutures employed in a manner similar to Hotz's operation for en-
tropium (Gruening). An incision is made just below the upper edge
of the tarsus and parallel to it. Some orbicularis fibres may be ex-
cised. With a sharply curved needle the thread is pushed along
the surface and upper edge of the tarsus through the conjunctiva,
until it loops up the cul-de-sac and returns upon itself beneath the
skin to come out at the upper part of the wound. It never traverses
the skin. Three sutures may be used. They are tied tightly and
allowed to remain two to five days, according to the degree of re-
action. The skin wound is closed as usual. By the same proceed-
ing, if the needles are carried up to the edge of the orbit and en-
gage the periosteum a much greater effect will ensue and superior
to that secured by Wecker's operation just described. Drooping
of the lid from redundancy of skin is easily cured by removing a
suitable flap. The proposal to advance the tendon of the levator
has not met with general adoption; it is not effective enough to be
useful. An attempt has been made to enable a patient to open the
eye by using a cord of india-rubber (Von Bibber), one end of which
was fastened by plaster to the lid, and the other to the forehead.
With the same object a spring clamp to pinch up a fold of skin has
been devised, to be worn habitually. Neither the clamp nor the
rubber band commend themselves in point of elegance and not
much in utility. A moderate degree of ptosis is one of the symp-
toms of disease of the sympathetic in the neck which was first
pointed out by Horner. It will be referred to later. See page 469.
Two cases of lipomata of both eyelids causing ptosis are reported
by Schell.
Burns of the Eye.
Superficial burns of the tegumentary surface of the lids need not
detain us, but those of the conjunctival surface are important be-
cause they often lead to adhesions of the lids to the globe or to each
other. Caustic materials or melted metal are the usual causes of
the accident: for instance, fresh mortar, lime, nitric or other acids,
ammonia, melted lead or iron; and red pepper may be mentioned,
which, while not destructive, is extremely irritating.
When lime gets into the eye, it must be washed copiously with
water, but the only effective means of removal is by forceps, or a
spud or curette; wiping out the coarser masses with a bit of rag
will serve, but the little pieces which remain, eat their way into the
tissue and become incorporated with it. At the inner canthus, and
in the cul-de-sac, it will be lodged, and must be dug out patiently
and thoroughly. Often ether must be administered. After com-
plete removal, syringe away all particles with warm water. The
relief for pain is cocaine and cold water. Of course an anodyne
may be given. The danger is of adhesion of the lids to the globe,
2S8
DISEASES OF THE EYE.
and of deep opacity of the cornea. The lids must be pulled away
from the eye and adhesions torn several times daily, and in mild
cases good will follow. Nothing can prevent the formation of
attachments in case the burn is deep, as the resulting ulcer slowly
granulates and contracts. Shields of lead, and dressing with lint
and sweet oil, and pulling the lid away, are then unavailing, but a
shell of glass resembling that used as an artificial eye, transparent
and convex enough to leave the cornea untouched has lately been
tried with some success. I should advise a mixture of iodoform and
vaseline on the cornea, say 3 i. ad 3 L, in bad cases where its infil-
tration threatens.
Fulminate of silver or mercury, which are used in percussion
caps and in boys' torpedoes, besides their terrific explosive power,
have a peculiar destructiveness in the kind of burn produced. The
conjunctiva becomes covered by a deep gray thick exudation, which
can be pulled off in sheets, leaving a raw, bleeding surface, and is
speedily reproduced. It resembles the worst forms of diphtheritic
exudation and is attended by severe inflammation. The cornea
may share in the process, and if it do not, is most liable to ulcera-
tion. The destruction of the conjunctiva leads to adhesions of the
lids to the globe. I have seen two accidents of this nature, and
their results were deplorable.
Burns by melted metal are, often less severe than those due to
lime, because when the metal cools it is taken out as a cup, and
there is no continuously destructive chemical action. Nitric, sul-
phuric, and acetic acids, can do great mischief. They are to be
washed out with cold water freely applied, and the case treated
for subsequent reaction. Ammonia causes a more superficial es-
char, and is exquisitely painful. Besides cocaine we may use castor
oil, vaseline, ice, etc. For red pepper, however dastardly its inten-
tional use, it may be said that it does not cauterize. The ulcers
caused by burns are damaging to sight in the ratio in which the
cornea has been involved. But a better result can generally be
expected than the first look of the case suggests. Several weeks
or months will be needed for recovery.
Symblepharon. — When the ultimate contraction has been
reached, we have adhesion of the lid to the globe, called symble-
pharon, and the question of repair is to be considered. The diffi-
culties are in proportion to the extent of adhesion. For columnar
bands, good results are possible; for total attachment of the whole
lid-surface, the difficulties of an operation are great. No good at
all is to be had by simply dissecting the tarsus and globe asunder—
the lid is sure to go back to its old site. Naturally, the lower lid
is the most frequent sufferer. Frequent traction on the cicatrix will
in time stretch it to a greater or less degree. The cure is attained
THE EYELIDS AND CONJUNCTIVA. 289
by means which shall not only separate the lid from the globe,
but prevent readaptation. For columnar attachments, the old pro-
posal of inserting a lead wire through the mass at the fornix is of
value. It has to be worn until a permanent hole is formed, which
shall be lined by a kind of epithelium; then the adhesions may be
:ut, and the parts can be kept from growing together. But this
operation is superseded by one of Arlt's (Fig. 118). He dissects down
the frenum, beginning on the globe, until he lifts it up to the fornix.
Through its free end a thread armed with two needles is passed,
and these are pushed through the cul-de-sac to the surface of the
skin, and the thread tied over a roll of plaster or bit of wood; by
this device the outer surface of the column is laid against the globe.
The raw surface is now covered over by bringing down flaps of
conjunctiva from either side. A glass shell may be worn over the
Fig. 117. Fig. 118.
globe which will aid in preventing contraction. If the new tissue
overlap the cornea, it may be left in situ and the dissection made
where the edge of the cornea should be, as in Fig 117.
For more extensive degrees of symblepharon, another operation
is available. The adhesions are dissected down to the fornix, then
the vacancy on the globe is to be filled, while the lid is left to itself.
Instead of sliding flaps of conjunctiva, as may be done in mild
cases, a kind of sling is made in this way: from near one side of
the vacancy a curved incision in the conjunctiva is carried around,
just outside the cornea, to the opposite side of the vacancy.
Then another incision, concentric to and outside this, is carried
around, but its extremities must not come nearer to the vacancy
than five to seven millimetres. It may even go up to the fornix,
and with the first incision it incloses a band which may be from
five to eight millimetres wide. Care is taken to make the ends
of the band the widest part, by turning the extremities of the
upper wound upward. The band is then loosened, except at its
ends, and slipped down over the cornea to take its place in the
19
290
DISEASES OF THE EYE.
gap made by removal of the adherent cicatrix. It is convenient
to put threads into the edge next the cornea before making the
outer incision: they serve to hold and draw down the flap, and
are used to fasten it in place. They may be carried entirely
through the lid and tied on the skin. Some readjustment of
intervening and adjacent conjunctiva is required, while the flap is
carefully fastened by fine sutures in its bed. The spot from which
it has been taken is left to itself. That in time, by granulation, be-
comes covered by a tissue which perfectly resembles normal con-
junctiva. The result of the operation is most fortunate, and I have
done it several times with great satisfaction. Operations by put-
ting in separate flaps from either side, twisting them down, and
uniting them by suture at their free ends, have been done by Knapp,
Teale, and others. Mr. Teale, who first suggested conjunctival flaps,
applied one to the inner surface of the lid after it had been dissected
off and applied another flap to the eyeball, so that the epithelial
surfaces should be in contact. For cases which may be too bad for
this proceeding, grafting of bits of conjunctiva from the rabbit is
available, and results in benefit. In most of these cases, the loosen-
ing of the adherent scar is demanded, for relief of pain and discom-
fort, without regard to sight. In many others additional opera-
tions, chiefly iridectomy, may be needed to gain better vision.
The transplantation of portions of conjunctiva from the rabbit
is serviceable in extreme cases, and aided by wearing a glass shell
will often, as my own experience has shown, give success. It is
also the only available method in attempting to make a conjunc-
tival cavity to permit the insertion of an artificial eye. I have
lately seen a boy on whom I operated eleven years ago, when he
was twelve years old. The eye was lost and reduced to a stump,
and extensive symblepharon existed. It was impossible to wear
an artificial eye. In March the adhesions were dissected away and
as large a strip as could be taken from the rabbit's eye, inserted.
Soon after a very small shell was worn and the size gradually in-
creased. In May following, the operation was repeated, and larger
shells employed. In four months longer, a suitable artificial eye
could be worn, and now, 1889, he continues to wear it and the con-
junctival cavity is capacious and perfectly healthy. Pieces of mu-
cous membrane can be taken from the lower lip and transported.
The skin of the lower lid or of the temple may also be employed,
as has been done by Harlan and Snellen (see Oph. Review, Dec,
1890). I have lately employed skin grafts of large size (Thiersch's
method) with good result for preparing a cavity in which to wear
a glass eye. Similar grafts might be used to line the raw surface
of a lid when dissected from the globe, stretched in place by sut-
ures, dressed with aristol, and the whole covered with bichloride
gauze, 1 to 1,000.
THE EYELIDS AND CONJUNCTIVA. 291
Ankyloblepharon is the adhesion together of the tarsal bor-
ders, and is a result of burns and of wounds. It is easily remedied
if there be any free spot or hole from which to start in separating
the lids. If not, the attempt is useless. One must establish a per-
manent hole by wearing a metal ring or other method. But such
a necessity has never occurred to me. Sometimes the lids are both
completely attached to the globe, and also adhere to each other at
their margins. For such cases no interference is proper. Such a
case is exhibited in the illustration, where complete occlusion was
caused by a piece of red-hot iron, both searing and sealing up the
eyeball. The same result is sometimes procured when for cancer-
ous disease both the globe and part of the lids must be removed.
Under such conditions all of the conjunctiva, both palpebral and
bulbar, must be dissected off and the edges of the lids pared. Such
an operation was necessary in the case of epithelial disease of the
lids which ultimately passed to the eyeball figured on Colored
Plate No. VI.
OHAPTEE III.
DISEASES OF THE LACHRYMAL APPARATUS.
Anatomy.—We have to do with the secretory and with the ex-
cretory parts of the apparatus. The former, which supplies the
tears, consists of a series of small follicles situated in the superior
conjunctival cul-de-sac, and the lachrymal gland, while the conjunc-
tiva itself secretes moisture, which may be counted part of the
lachrymal fluid.
The lachrymal gland is lodged in a fossa at the upper and outer
angle of the orbit, and may be felt by the finger indistinctly
under its overhanging rim. It is an acinous gland like the parotid,
subdivided into a smaller and a larger lobule, which are separated
by a septum of fascia. The smaller is sometimes called an acces-
sory gland. There are numerous isolated acini lying near the prin-
cipal masses. The size of the chief gland is variable, but may be
stated at twenty millimetres in length, eleven to twelve millimetres
from before backward (breadth), and five millimetres in thickness.
It is concavo-convex, and lies against the periosteum. Numerous
ducts, whose orifices are from ten to twelve in number, give exit
to the secretion at the temporal side of the superior fornix. The
tears contain 1.25 per cent of sodium chloride and 0.5 per cent of
albumen.
The excretory apparatus begins as minute openings (the puncta),
about six millimetres from the inner angle of the lids, which lead
into small canals (canaliculi), and they unite to empty by a com-
mon orifice into the side of the lachrymal sac. The sac rises a little
above the place of entrance of the canaliculi, and is continuous
below with the lachrymo-nasal duct, which empties into the inferior
nasal fossa, behind the tip of the inferior turbinated bone. The
total length of the sac and duct is about one inch (twenty-five mil-
limetres). Its section is ovoidal, with the long axis from before and
outward, backward and inward. Its calibre varies greatly, and its
shape may also vary. In the same skull, from which the soft parts
have been cleared, I have seen the duct on one side to be round,
and not more than three millimetres in diameter, and on the other
to be oval in section, with its major axis six millimetres long.
The membrane lining the duct and sac is like that of the nostrils,
DISEASES OF THE LACHRYMAL APPARATUS. 293
being both a periosteal and a mucous membrane. It is highly vas-
cular, thick, and covered by cylindric epithelium, lying on several
layers of spheroidal cells. The cylindric cells are by some declared
to be ciliated. Next the bone the membrane is spongy and erec-
tile. It is thrown into folds at two or three points, viz., at the
junction of the sac and duct, which corresponds with the beginning
of the bony portion of the tube in the ascending process of the
superior maxillary bone, and also at the lowermost part, where it
communicates with the nostril. There is also, sometimes, a less
distinct fold at its middle. The lining membrane of the canaliculi
is thin and pale, and the puncta are a little whiter than the neigh-
boring membrane. They point toward and rest in contact with the
globe. Muscular fibres surround these openings like sphincters,
and they are held in apposition with the eye by the action of the
orbicularis and tensor tarsi muscles.
The latter lies behind the lachrymal sac, and the tendon of the
former crosses in front of it and is sometimes called the tendo oculi.
It is brought into relief by pulling upon the lids at the outer can-
thus. The orbicularis has additional insertion into the lachrymal
bone, by bundle's of fibres which go to it directly. The tears are
forced into the excretory passages by the action of the muscles just
mentioned, aided by a kind of suction caused by the muscular fibres
of the puncta and canaliculi (Klein). Unless the puncta are kept
in tonic contact- with the eye, the tears cannot enter. The quantity
of fluid is usually so small that evaporation and secretion balance,
and nothing passes down to the nose. With any irritation of the
eye, a larger flux occurs, and frequently the capacity of the tubes
is overtaxed and tears brim over the lids (epiphora). Usually the
follicles in the superior fornix and the conjunctiva furnish all the
needed moisture, but on unusual demand the lachrymal gland
comes into play.
Diseases of the Lachrymal Gland.
Acute inflammation, dacryo-adenitis, occurs in rare instances. I
have in one case seen both glands inflamed at the same time. The
symptoms are, swelling, by which the gland is pushed down out of
its fossa and can be recognized on turning up tlie lid; there is oedema
of the lid, tenderness of the gland and of the adjacent bony edge,
together with dull pain. The amount of swelling can be great, and
it is sometimes difficult to exclude periostitis or abscess of the lid.
There ma}- be suppuration in the surrounding connective tissue,
while often the inflammation resolves without suppuration. The
treatment consists in warm fomentations and incision into acute
inflammatory swelling. Constitutional treatment is not often
294
DISEASES OF THE EYE.
needed, although the possibility of a syphilitic cause is not to be
ignored.
The gland may be the seat of neoplasms, such as sarcoma and
other tumors, and of cystoid degeneration, and it is liable to chronic
hypertrophy; but these conditions need no special consideration.
Its extirpation to cure epiphora was practised by Mr. Lawrence,
but is not now approved.
Dislocation of the lachrymal gland came under my notice in a
young girl and seemed to be due to slow relaxation of the inclos-
ing capsule. The gland presented itself beneath the ocular con-
junctiva over the insertion of the rectus externus muscle and was
affected by slight degree of inflammation which was not, how-
ever, the cause of the displacement. The opposite eye was phthisi-
cal and there too the degenerated and atrophied lachrymal gland
had descended below its proper place.
Diseases of the Excretory Apparatus.
We have eversion and stoppage of the puncta, occlusion of the
canaliculi, catarrh of the sac, and obstruction of the duct. We also
have acute dacryocystitis, chronic distention of the sac, and fistula
lachrymalis. Sometimes there are two canaliculi in each lid.
Eversion of the puncta is the consequence of chronic blepharitis
marginalis or of chronic conjunctivitis, or it follows from paralysis
of the orbicularis muscle in lesions of the facial nerve, and neces-
sarily accompanies ectropium. In the first class of cases the orifice
is apt to be made smaller; in the paralytic cases the punctum may
be uncommonly prominent as a papilla, and while the lower one
sags down, the upper also fails to lie upon the globe.
The canaliculi are sometimes the seat of stricture, and in a few
cases chalky concretions have been found in them. Leptothrix, one
of the microscopic algae, has been found in them.
Dacryocystitis or catarrh of the sac and duct is a lesion not
often presented to us at an early stage, because people are apt to
avoid the surgeon until the disease has lasted so long that simple
catarrh has become complicated with obstruction. There is prac-
tically no real distinction to be made between these conditions. In
dacryocystitis we have swelling of the mucous membrane, h}Tper-
trophy of its epithelium, and papillary growth—sometimes a state
precisely like granular conjunctivitis, and with this a muco-purulent,
glairy, somewhat tenacious secretion, which fills the cavity and is
there retained. The calibre of the nasal portion of the passage
speedily becomes choked, and the morbid secretion cannot find out-
let; hence, the sac-wall undergoes distention. The three factors of
thickening of the mucous membrane, excess of secretion, and dis-
DISEASES OF THE LACHRYMAL APPARATUS. 295
tent ion of the sac, gradually conspire to bring about a more or less
aggravated condition, in which the lachrymal tumor becomes larger
and the stricture smaller. The skin, after a long period, becomes
thin, and may even get to be translucent. It may in very old cases
happen, that the lachrymal bone becomes diseased. The constant
and annoying effect of this state of things, at almost any period of
its existence, whether early or late, is to cause an undue quantity
of tears to be formed; they overflow the lid or stand ready to drip
over. On exposure to wind or to cold air, the eye waters, uncom-
fortably, and the fluid sweeping over the cornea makes vision
misty, and continuous use of the eye is sometimes, and more espe-
cially at night, greatly embarrassed. The tears which thus flow
too liberally are called forth, it is true, by a hypersecretion of the
lachrymal gland; but they are likewise mingled with the products
of the irritated conjunctiva and its glands. The universal concom-
itant of dacryocystitis is palpebral conjunctivitis, sometimes severe,
and not infrequently blepharitis marginalis coexists. The caruncle
and semilunar fold are swollen and injected, and aid in hindering
the entrance of fluid into the puncta. The patient is constantly
using his handkerchief, and thus materially aggravates his troubles.
But he may learn, and this should be taught by the physician, to
keep the sac empty by squeezing its contents into the nose, if the
passage be permeable, or the secretion gushes out of the puncta upon
the eye. Wherever it goes, keeping the sac empty affords some
relief. But when the disease has lasted long, the secretion acquires
irritating qualities, especially if it be permitted to stay long unex-
pelled from the sac. Then its contact with the eye sets up decided
conjunctivitis, and the fluid may even have an offensive odor. The
fluid then is sticky and unpleasant; especially is it mischievous if
the eye is submitted to an operation. The pus has an infectious
quality, and is extremely apt to cause suppuration in a corneal
Wound. The reason of this pernicious quality is the populous-
ness of micro-organisms of many varieties in the secretion. With
the exception of the gonococcus none are known to be more hurtful
to the tissues of the eye. It follows that cases of cataract, or cases
which require iridectomy, should be first relieved from any lachry-
mal trouble.
The disease is of slow progress, and often for a long time causes
little annoyance. Even after a tumor appears at the inner can-
thus, the swollen sac may not cause great discomfort. But, if it be
impracticable to empty it by pressure, the stricture is close and the
condition will be both obstinate and troublesome.
A most unpleasant complication in the progress of the disease
is the occurrence of acute phlegmonous inflammation and abscess.
This is severely painful, and may cause extreme swelling of the
296
DISEASES OF THE EYE.
lids and neighboring parts. The tumor will be red, shiny, and
tense. If not large, it will be very tender to the touch, and the
conjunctiva will be hyperaemic. It is quite characteristic to find
the swelling in the /old of the lower lid and sometimes there is so
much infiltration of the skin as to suggest erysipelas. If the pro-
cess be left to itself, the matter finally escapes by ulceration, and in
this case a fistula lachrymalis is quite liable to ensue. The opening
will be below the tendon of the orbicular, and may be large or
small. (See Fig. 119.)
In cases which have been long neglected, the subjacent bone
may become carious, and a passage may even take place into the
Fig. 119.
superior nasal fossa, or into the cells of the ethmoid. In general,
the disease will either remain stationary or grow worse__it does
not get well. It may be tolerated for years with slight discomfort,
or it may prove unpleasantly exasperating. (I have seen one case
of congenital lachrymal fistula affecting both eyes. It was re-
ported by Dr. Agnew *.)
Before entering upon the consideration of treatment, a few
words may be given to a condition which causes epiphora and is
apparently not associated with the morbid lesions above described.
I have seen a few patients who were annoyed by an accumula-
tion of tears, in whom I could find no swelling of the sac nor ten-
derness over it, nor could I elicit any discharge. At the inner can-
'Trans. Am. Oph. Soc., 1874, p. 209.
DISEASES OF THE LACHRYMAL APPARATUS. 297
thus there was swelling of the semilunar fold, and turgescence of
the caruncle; the puncta were prominent, but not everted nor
choked, neither was there obstruction of the canaliculi. The cause
of the epiphora seemed to be the swollen state of the caruncle and
of adjacent parts; this irritation excited hypersecretion of tears,
while the prominence at the canthus served to obstruct entrance of
the fluid into the puncta. This rare condition has been noted by
Graefe, and I have seen it a few times.
Diagnosis.—We have epiphora and a swelling over the lachry-
mal sac The tumor will be effaced by pressure of the finger, and
its contents will either flow over the eye through the puncta, or else
Fig. 120.
pass through the nose. If by pressure the tumor do not wholly
subside, the sac-wall may be very thick, or the stricture be very
tight. If very large and the walls thin, its bluish color may sug-
gest a cyst; but the history of epiphora will settle the doubt. The
caruncle is red and apt to be swollen, and the puncta also to be
swollen and red, and of unusual size. In some very quiescent cases
no tumor appears, but pressure will force fluid into the nose. These
varieties depend on the duration of the malady, and on the amount
of secretion and the degree of obstruction. We sometimes meet
cystic or solid tumors of the skin overlying the sac, they will be
recognized by the possibility of grasping them with the thumb and
finger, or by their mobility and the absence of other signs. A
mucocele or chronic abscess of the ethmoid cells sometimes points
in the lachrymal region. Its position will be higher than the sac,
298
DISEASES OF THE EYE.
and above the tendon of the orbicularis instead of beneath or be-
hind it (see Fig. 120).
Causes.—The prevalent cause is catarrh of the nasal mucous
membrane. This cannot, however, always be discovered. We have
frequently the scrofulous diathesis and also frequently constitu-
tional syphilis. A local periostitis at the nasal outlet of the canal
may be the starting-point. In most cases the disease is essentially
a periostitis and, remembering this characteristic, its special and
pertinacious behavior will not be surprising.
Prognosis, as has been intimated, is that recovery will be slow,
requiring months. Six weeks would be a satisfactory period—six
months not infrequent. Sometimes relief can only be partial, yet it
can be absolute and complete. The pathological conditions are so
varied that the expectation of cure must be determined by the fea-
tures of each case.
Treatment.—We have the palliative and curative. A consider-
able number of persons are not greatly disturbed by their lachry-
mal trouble; another portion are too timid to submit to surgical
proceedings, and others are unwilling to spare the time which effec-
tual treatment demands. For these patients, only palliative pro-
eedings can be used, and they are as follows:
To keep the sac empty by pressing on it with the tip of the finger
from above, down, and backward, so as to force the fluid, if possible,
into the nose, with firm slow pressure. A certain knack is often
acquired by the patient which the physician cannot imitate. If
the fluid must be disgorged on the eye, the handkerchief must be
in hand to absorb the fluid at once without needless rubbing of the
lids, and at all times the eye should be gently pressed, and not
wiped. The sac must never be allowed to approach distention.
The use of astringent drops or of a lotion upon the lids, or occa-
sional astringent applications to the palpebral conjunctiva, e. g.,
Argent, nitrat., gr. ij.-v. ad | i., as this surface may become more
congested, will do good service. Moreover, the state of the nasal
cavity must be inspected, and duly dealt with. Washing out the
nostrils with warm salt water by a syringe, the application of
depurating and astringent fluids by an atomizing apparatus, or
by the blowing of powders into the nostrils, in the manner called
for in the treatment of nasal catarrh, will be well worth doing
Dobell's formula is widely employed.
ty Sodae biboratis,.......3 iv,
Glycerini,........31.
Sodae bicarbonatis,......3 ss.
Acid, carbolici,.......3 ss.
Aquas,......... | yi.
M.
DISEASES OF THE LACHRYMAL APPARATUS. 299
For powders, among many which may be chosen are the follow-
ing:
IJ Bismuthi trisnitrat.,
Gum. acacias, . . . . . . aa 3 i.
Pulv. cubebae,.......gr. x.
M.
1} Acid, boracic pulv.,......q.s.
Under such management, some persons get along fairly well
and are satisfied. Many do nothing more than keep the sac empty,
and expect when they get a coryza to have more trouble—and so
they do.
The curative treatment involves a careful discrimination of the
state of the sac and duct, and the suitable adaptation of means.
If the lachrymal tumor is easily emptied into the nose—and this
implies that the case is recent—external applications may suffice.
In children of a strumous quality it may be only possible to use
probes by giving chloroform, and often the cleansing of the nostrils
by a camel's-hair pencil, and the use of cod-liver oil, iodide of iron,
etc., will bring about recovery. Carefully wipe out the nostrils
with cotton on a holder, and apply vaseline twice daily, and a
solution of nitrate of silver, gr. x. ad § i., twice or thrice weekly,
or the powdered boracic acid once daily.
But the common run of cases call for treatment of stricture of
the nasal duct. Becker uses conical probes with which he stretches
the punctum and canaliculus and reaches the duct. The proceed-
ing is painful and permits the introduction of probes of only mod-
erate size, which are inadequate in many instances.
The first step is to slit the canaliculus, which Mr. Bowman
taught to be the best mode of approaching the sac. My preference
is for the lower one. I also choose a beaked knife, with a blade
wider than is generally used (see Fig. 121), attached to a long and
stiff, but malleable shank. Sometimes a narrow blade is useful
(Agnew). (See Fig. 122.) For a case of no long duration it may
be needful to do no more than slit the canaliculus. The surgeon,
if operating on the right eye, will stand behind the patient, hold-
ing the head against his own body, use the left hand to draw
the lower lid out and keep it tight, and insert the beak of the
knife perpendicularly into the lower punctum. Sometimes this
is partially occluded. The point of a pin or a Bowman's direc-
tor (see Fig. 123) will usually open it for the tip of the instru-
ment. When well engaged, bring the hand to the horizontal
position, and push the blade with cutting edge inclined inward
and upward into the sac until the tip is felt to strike the lach-
rymal bone; keeping the point firmly against the bone, raise the
300
DISEASES OF THE EYE.
handle up, and also lift up the blade so as to incise as freely as
possible the conjunctival wall of the sac. Many surgeons stop at
this point and let the patient apply cold water, and on the next day
attempt to introduce a probe. In the greater number of cases I do
Fig. 121.
Fig. 122.
Fig. 123. Fig. 124.
Fig. 125.
S.TIEMANN &C0
Fig. 126.
Fig. 12?.
not follow this mode of proceeding, but at the first operation carry
the knife down into the nasal duct and divide the stricture. I
make two or three incisions upon different sides of the duct, to gain
the greatest enlargement. Blood issuing from the nostrils is proof
that the passage has been opened. When the stricture is divided
DISEASES OF THE LACHRYMAL APPARATUS. 301
as Stilling recommended, a large instrument should be inserted,
viz., the larger end of Weber's conical probe (see Fig. 124), and
afterward the probes of large sizes. Cases must be dealt with
according to the calibre which is normal to each, and the fullest
possible expansion obtained. The first operation may perhaps be
done under an anaesthetic, and often two or three days will elapse
before another introduction of a probe. Meanwhile the canaliculus
must not be allowed to close. The introduction of probes is always
painful and cocaine does not serve any useful purpose, no matter
in what method employed. Usually the proceeding is less painful
as the cure progresses.
The probe may be used three times weekly, and be left in place
from ten to thirty minutes. Progress can, in some cases, be made
rapidly; others will permit only a gradual increase. The amount
of reaction after probing will regulate the frequency of introduction
and the rate of enlargement. In passing the probe carry it hori-
zontally into the sac, and when its point impinges on the bony wall,
bring it to a perpendicular and attempt to follow the axis of the
duct. The direction is downward, outward, and backward, toward
the wing of the nostril. The aim must be to get behind the edge
of the opening into the superior maxilla, and until this is gained
the probe must be handled with delicacy, and in the exercise of a
nice sense of touch. Caution at this point is indispensable, and a
moderate degree of it will avoid making a false passage. After
this opening is gained, the probe may be firmly sent down until
it reaches the nasal fossa. It should be left in place for ten min-
utes, and then withdrawn. This exploration will indicate what
kind of stricture we have in hand, and what instrument will best
dilate it.
I have, during many years, made use of Theobald's probes (Fig.
125), and find them exceedingly satisfactory. They go up to large
sizes, No. 16 being the maximum. Dr. T. has advocated the use of
large probes in a paper in Arch, of Ophth.,vi., and in Trans. Am. Oph.
Soc, 1879, and was not aware that Dr. E. Williams, of Cincinnati,
myself, and others had, for many years, sought to secure the full-
est dilation which the anatomical and pathological conditions make
possible. Dr. H. W. Williams, of Boston, has introduced probes with
bulbous tips and elastic necks (Fig. 126) which, while stiff enough to
handle easily, find their way around projecting obstacles or through
sinuous passages better than straight instruments. I have often
had occasion to be pleased with their qualities. But my ultimate
resort is to a large instrument, smooth, with conical point, which
must press its way through the inflammatory deposit—not with
violence, but with some force; and this is to be left in situ from
ten to thirty minutes, but not long if its pressure be extremely
302
DISEASES OF THE EYE.
painful. Making haste slowly is the password to success with
these cases, but I am convinced that the gate must be opened
widely and made to stay open, to get full relief. Dr. Theobald's
probes are of the following sizes: beginning with the diameter of
one quarter of a millimetre, advancing by increase in diameter of
one-quarter of a millimetre from No. 1 to 16, the last being four
millimetres in diameter. I have found them so well contrived that
I have adopted them almost to the exclusion of all others. The
largest sizes are now made of aluminum with the advantage of
lightness and greater smoothness.
As the result of probing, abatement of the catarrhal secretion
is soon manifest. In most case, nothing more than probing and
treatment of palpebral conjunctivitis is needful. In a certain num-
ber, secretion is copious, and does not measurably diminish. The
syringe must then be employed with salt and water 2$ or with a
weak solution of argent, nitratis, gr. v. ad \ i., or gr. x. ad § i.
Perforated probes have been devised for this object, but a small,
hard-rubber dental syringe can be readily adapted to the purpose
by bending its nozzle in hot water to an obtuse angle. After the
probe has been withdrawn, the syringe may be used. It will not
require protracted employment.
In cases where persons cannot spend the weeks or months with
the surgeon which treatment requires, the plan may be adopted of
putting in a leaden wire, about size No. 6 or 4 Bowman, which shall
lie in the duct with its upper end properly bent downward and in-
ward at the inner canthus. This style may be worn as in old times
Scarpa's nail was worn, for two months, more or less, and it will
then be found to have brought about absorption of the stricture.
It excites considerable secretion, is not agreeable to wear, but an-
swers fairly well. Granulations are liable to spring up at the en-
trance into the sac, and when the style is taken out, the opening
soon contracts and is difficult to find.
I have another suggestion to make in this matter. Some cases
permit dilatation of the stricture with reasonable rapidity and to a
satisfactory degree, but the annoying epiphora does not stop, and
the patient does not find the pain of the treatment compensated
by good results. It must be remembered that there may be an-
other stricture at the bottom of the duct where it enters the nose.
Here I have many times found a nodular projection from the side
of the canal, or a decided narrowing of its calibre. To overcome
this stricture the common probe is futile. I have had a form made
which is a repetition of a very old instrument, with a bulbous tip and
of unusual length (see Fig. 127). It is carried down to the lower end
of the canal in the ordinary way, and then, to get it into the nose
the flat handle must be rotated toward the temple so as to turn the
DISEASES OF THE LACHRYMAL APPARATUS.
303
point backward, and then push it onward. It will go down almost
an inch farther, and it may so far penetrate the nostril as to touch
the place of junction of the hard and soft palate. Some obstinate
cases of epiphora have been cured by ascertaining the presence of
this hidden stricture, and resorting to the instrument thus de-
scribed. In cases where the obstruction at the bottom is osteoid, I
have used a narrow gouge with a cutting end, and have bored a
way into the nose (see Fig. 128). Afterward steady probing is
needed to prevent the return of the obstruction.
I have found Theobald's probes frequently able to cope with the
cases just cited, because of their well-fashioned tips and greater
length. But they deserve special notice, and the probe I have de-
vised will sometimes be necessary.
There remains another class of cases in which the passage can-
not be restored to its normal state: either because of excessive
Fro. 128.
thickening of the lachrymal sac, or the duct is almost occluded by
osteoid growth, and is practically impermeable, or there may be
caries. The older writers proposed opening into the nasal fossa by
perforating the lachrymal bone. The modern treatment is the ob-
literation of the lachrymal sac and duct. This is done by dissect-
ing out the hypertrophied sac, or by destroying it either by the
actual or potential cautery. Excision of the sac may be combined
with the cautery. After dissecting out with scissors as much
of the sac as can be removed, the Paquelin cautery is thrust
into the duct. Usually, fuming nitric acid is the agent employed.
The sac is freely exposed by an incision in the skin, and when the
bleeding stops, a bit of wood—the untipped end of a match—with
some fibres of cotton on it, is charged with it and freely applied to
the mucous surface. Care must be taken to protect the eye, and
the edges of the wound must be held asunder by sharp hooks. This
operation has been done by Dr. Agnew through an incision upon
the mucous side of the sac, with simultaneous division of the canal-
iculi, and he reports good results. I have not followed his method;
and although, as commonly performed, a scar is left upon the skin,
I have not found it a. conspicuous thing or a deformity. Still an-
other mode of destroying the sac is by putting into it pieces of
nitrate of silver. They cause prolonged pain, and are less effectual
304 DISEASES OF THE EYE.
than the Paquelin or the nitric acid. After cauterization the
sac is stuffed with lint, and cold water dressings applied. It will
take two or three weeks for the wound to close by granulation.
When the cavity is obliterated, the success which follows in reliev-
ing the epiphora, depends on the fact that there is no longer an
irritation in the sac to stimulate a superabundant flow. The ob-
struction of the excretory passage causes no inconvenience, except
when some special occasion for weeping arises, such as keen winds
or mental emotion. In fact, however, it is not easy to perfectly
obliterate the sac and duct, and hence this treatment does not give
uniform results; but it is a great amelioration of the previous con-
dition. In very young children a probe may be passed by the help
of chloroform. I have seen lachrymal abscess with stricture, on
both sides, in a child six months old, and treated it successfully by
the usual method. I have sometimes instructed a patient to use
the probe for himself, when he had reached the proper size, and
simply needed to maintain the enlargement.
Added to the above suggestions for local treatment, the possi-
bility of syphilitic infection must be borne in mind and the suitable
medical treatment adopted. In late tertiary forms I have found
this condition. The iodide of potassium and corrosive sublimate
will do the same service as in any case of specific periosteal inflam-
mation. In all cases where nasal catarrh shows decided symptoms,
this must receive attention.
Phlegmonous Inflammation of the Lachrymal Sac.
This takes place as an incident during the progress of a chronic
dacryocystitis. The attack is always painful, may be ushered in
by a chill, and varies greatly in severity. Swelling, tenderness,
and hardening of the sac are always present, while sometimes the
lids become puffy, especially the lower lid along the furrow which
lies below it and in a few cases the oedema of surrounding parts has
simulated severe orbital cellulitis. Even though the swelling be
small and circumscribed, the patient commonly suffers much pain,
and the reason is the same as in the case of any subperiosteal in-
flammation, viz., the effusions are compressed by dense membranes
and the nerves are numerous.
Treatment.—It is rarely of any use to do anything else than to
make an incision into the sac If the case be seen early, this may
be done by way of the canaliculus and slitting freely the sides of
the sac, thus preparing for the probe at a future period, when
treatment of the original stricture shall be in order. But if much
swelling have taken place, the knife should be put perpendicularly
upon the skin over the middle of the sac and thrust through it to
DISEASES OF THE LACHRYMAL APPARATUS. 305
the bone, and then with one sweep carry the incision down for half
an inch, more or less, according to the extent of the tumor. The
best surgery is an early and a free incision. By doing this the oc-
currence of fistula is almost certain to be avoided, while it is very
likely to be the disagreeable consequence of permitting the abscess
to " break of itself." After opening the abscess, warm water dress-
ings and poultices will be applied until the attack subsides. The
cut will be kept open by a bit of lint.
It is not denied that sometimes, when phlegmonous inflamma-
tion begins, resolution may occur and this is best promoted by the
continuous use of hot poultices, of which the ground slippery elm
bark is the most eligible.
Lachrymal fistula is occasioned by the imperfect healing of an
abscess, and implies the existence of a permanent stricture. This
Fig. 129.
lesion is not seen as frequently as once it was, nor do lachrymal
diseases often attain the extremity which the older writers describe.
Surgical aid is better and more ready to be instituted than in
the older day. Hence, a bad case of caries or of fistula does not
often get an opportunity for development. It is needless to describe
the condition—it declares itself; and if dead bone be present, the
probe will soon discover it, if the foul odor and discharge do not
betray it. Fig. 129 shows a case of lachrymal abscess and fistula
during the regressive period. The fistulous opening afterward
20
306 DISEASES OF THE EYE.
closed spontaneously, while the stricture was at a later period
divided and treated by probes.
For a bad case, cleansing by the syringe through the fistula
may sometimes be proper, together with attempts to restore the
calibre of the duct. If there be dead bone, this may be left to
gradual elimination or be removed by a small gouge. For such
cases destruction of the sac will generally be a necessity. In gen-
eral, it is better for the great majority, to slit the canaliculus and
deal with them as if there were no fistula. So soon as a route can
be established for the secretions to make their way into the nostril,
the fistula will heal. In case it prove sluggish, the process of clo-
sure may be hastened by stimulating it with a pointed crayon of
nitrate of silver (Squibb's caustic points). Cure of the stricture
carries with it cure of the fistula. If the stricture be incurable,
the obliteration of the sac is the alternative, in the manner above
described.
It may be remarked, in summing up the whole matter of lach-
rymal troubles, that the larger number may be completely cured,
another proportion are relieved of special annoyance, and the re-
mainder gain some benefit, but still have trouble. That a perfect
cure should always ensue, it would be unreasonable to expect; that
palliation is better than no relief, is evident, while patient continu-
ance and careful discrimination of the precise lesion, are indispens-
able to success. Moreover, in no cases more than these, is the tactile
address of the surgeon an element of value to win confidence and
spare needless pain, and thereby contribute to success.
A case reported by Dr. Bull (Am. Jour. Med, Sci., July, 1880) is
worth remembering, where caries of the ethmoid bone caused a
pre-lachrymal abscess, and on opening it the lachrymal sac was not
involved, nor its cavity entered. In such cases there will be no
epiphora. Treatment will simply be to provide for the escape of
the discharge by washing out the cavity with antiseptic and slightly
stimulating solutions.
Leptothrix lachrymalis sometimes makes its home in the canal-
iculi, especially in the inferior. The symptoms are slight inflam-
mation and swelling about the punctum, suggesting a stye, and at
the orifice a stringy mucoidal pus presents and can be squeezed out.
There is swelling of the caruncle and semilunar fold and persistent
annoying "angular" conjunctivitis. The somewhat tenacious se-
cretion can be seen issuing from the puncta spontaneously; the
apertures will be widely open and a sucking or pumping motion
may perhaps be noticed. The secretion collects in flakes and small
masses and beginning at the inner angle spreads over all the in-
ferior cul-de-sac. While on the one hand the mistake may be made
of regarding the condition as simply a hordeolum, on the other the
case may be considered a mild dacryocystitis, There will be no dis-
DISEASES OF THE LACHRYMAL APPARATUS. 307
tention of the sac, and the chronic character of the trouble excludes
the diagnosis of a stye. The condition is somewhat rare, but occa-
sionally appears. A very precise and ample description of it was
given by Von Graefe1 in 1854, and again in 1855. By pressure a
slimy yellow substance is brought out, but the swelling may not
entirely disappear, because a number of small calcareous granules
may remain. Microscopical examination shows a bacillary micro-
organism, a fungus with jointed threads and spores, akin to the
leptothrix buccalis which is found on the tartar of teeth and has
the property of secreting lime salts. See Foster's " Encyclopaedic
Medical Dictionary," Vol. III., 1892. Treatment consists in slitting
the canaliculus to expose the cavity, emptying it, and applying a
solution of nitrate of silver gr. v.-x. ad § i. for a day or two, and
using a weak antiseptic solution as a lotion. So soon as the fungous
character of the trouble is perceived, treatment will be readily un-
derstood. A point to be noted is that only one eye is affected.
Excessive lachrymation is caused sometimes by undue secretion
of tears unassociated with disease of the excretory apparatus.
This is by far more rare than the conditions above described. The
epiphora is not constant, but appears when cold winds or similar
irritation stimulates great conjunctival secretion. It may give no
annoyance in summer or within the house, and be extremely trouble-
some when facing a cold wintry wind. There may not be very
marked congestion of the ocular or of the tarsal conjunctiva. There
will be no swelling of the caruncle as uniformly attends catarrh of
the sac, nor pouting of the puncta, nor can an}7 fluid be expressed
from the sac The sedative effect of cocaine temporarily stops the
annoyance, and if the upper lid be everted so far as the cul-de-sac
(sometimes needing a forceps for the purpose and the aid of cocaine)
a band of swollen conjunctiva will be found in or near the fornix
betraying the enlarged and irritated glands of Krause, the isolated
lachrymal follicles which are the source of the hypersecretion.
Diagnosis is included in the above description, and treatment
consists in applying solution argent, nitrat. gr. v. ad § i. to the site
of the trouble twice weekly, or perhaps the more heroic method of
slight touching with the actual cautery (Paquelin or a small gal-
vano-cautery) will be admissible and more prompt. Nasal catarrh
must be duly remembered. Extirpation of the lachrymal gland is
scarcely to be thought of, although it was once practised by Mr.
Laurence. This gland is not the source of the trouble, but the
smaller ones referred to in the fornix.
1 Graefe, Archivf, Ophthal., Bd. 1, Abth- 1, S. 284, alsoBd, ii., Abth, 1, S. 224.
CHAPTER IV.
THE CONJUNCTIVA.
Anatomy and Physiology.—The membrane presents for consid-
eration its tarsal portion, the fornix or sinus, and the ocular por-
tion. The tarsal or palpebral part is closely and smoothly adherent
to the tarsi, and permits the Meibomian follicles to be seen through
it. It has a faintly yellow color and a few vessels. The fornix, or
sinus, or cul-de-sac, or fold of transition, is very loosely attached
to the parts beneath, and slips freely back and forth; it has numer-
ous folds, is of a turgid dark color, and has many glands. The
depth of the fornix varies according to age and individual peculiar-
ities. Sometimes the whole of the superior fornix can be exposed
to view, by everting the lid, and often no effort will display it. The
inferior fornix can always be fully seen. The ocular conjunctiva
lies smoothly upon the globe, but is loosely attached, and can be
moved back and forth by pulling upon the lids. At the outer can-
thus the conjunctival sinus is deep, especially toward the lachrymal
gland; at the inner canthus, on the contrary, the sinus, or fornix,
is shallow both above and below. At the inner end of the palpe-
bral slit we have the congeries of glandular follicles, called the car-
uncle, and between it and the margin of the cornea is a slight fold
of the membrane running nearly vertically, yet somewhat crescen-
tic, called the plica semilunaris. It is bound rather firmly to the
parts beneath, and is the analogue of the third eyelid of some
animals, and in them it often contains a plate of cartilage.
The caruncula lachrymalis is an isolated and modified portion
of skin tissue covered by epithelium, containing fine hairs with their
tributary follicles, and modified sweat glands resembling the glands
of Moll. It also contains connective tissue and a little adipose sub-
stance; it is dense and firm and of a pale red color. Both it and the
semilunar fold are connected by fibrous tissue to the subjacent
structures and in the movement of the eyeball inward, they are
drawn inward with the action of the rectus internus muscle. This
relation is too often made unpleasantly obvious by the sinking
which occurs after tenotomy for converging strabismus.
The different portions of the conjunctiva vary in structure. The
tarsal part seems on casual inspection to be perfectly smooth, but
THE CONJUNCTIVA.
309
closely examined, it is found to have a slightly velvety surface, and
this is produced by numerous fine grooves and pits which reticulate
irregularly, and constitute so-called papillae. The papillae are
larger near the orbital edge of each tarsus. Their distribution
is quite irregular. They are covered by epithelium whose super-
ficial layer is cylindric and the deep layer flat. As the epithelium
goes into the depressions between the papillae, it is in several
layers and distinctly cylindric These depressions are irregular
and complex in form. There is a layer of fibrous tissue beneath
it, rich in elastic fibres and closely adherent to the substance of
the tarsus. In the meshes of the fibres is diffuse lymphoid (ade-
noid) infiltration which grows more abundant toward the orbital
portion. Lymphoid tissue is gathered into distinct masses (Henle)
and called lymphoid follicles, and is normal in many domestic
animals, and whether their occurrence in man is to be counted
normal or pathological is a subject of dispute. It is admitted that
they do not appear in the young. That they may, when in moder-
ate quantity, be considered normal in man, seems to be the best
opinion. This question has a bearing upon the pathology of tra-
choma. When the grooves and furrows of the tarsal conjunctiva
are very deep and intricate, they form crypts and follicles lined by
epithelium and may thus take on the appearance of glands. If
their orifices at the free surface become occluded, they may resemble
ductless glands. In young subjects the reticulations and furrows
do not exist.
The orbital part of the conjunctiva, the sinus or fornix, is loose
and plicated, and both variable in extent and imperfectly defined in
limits, except at the tarsal side. Its tunica propria contains abun-
dance of elastic fibres. Its epithelium is in many layers, the sur-
face cells are cylindric, those beneath are rounded and there are
very few of the depressions described as present on the tarsal por-
tion. The only glands are the acino-tubular of Krause near the
upper edge of the tarsi, and there is no lymphoid or adenoid sub-
stance. There is an abundant and loose submucous tissue.
The ocular part of the conjunctiva has a flat epithelium in
numerous layers and in this respect resembles that of the cornea.
The tunica propria has no papillae and contains among its fibres
many leucocytes. At the margin of the cornea the conjunctiva
becomes closely united to the subjacent structure and its epithelium
is more dense. This part is known as the limbus corneal, and it
is about 2 mm. wide, but varies. The sub-conjunctival connective
tissue is loose and elastic, although the membrane is held smoothly
in place, and especially by fibres which run into it from the inser-
tions of the recti muscles.
The blood-vessels are very numerous in the region of the limbus,
310
DISEASES OF THE EYE.
about the caruncle, and in portions of the tarsal conjunctiva, espe-
cially at the outer and inner angles. The large veins of the ocular
portion are to be noticed, especially those which go to the muscular
twigs and empty into the ciliary body.
The sensitive nerves of the conjunctiva are numerous, and come
from the fifth pair. They form a special meshwork about the
limbus, and in the tarsal portion special tactile bodies have been
described by Krause. For this reason the presence of a foreign
body beneath the lid is so intolerable, and reflex action between the
orbicularis and the conjunctiva so prompt.
The function of the conjunctiva is to act as a lubricating sur-
face and in this it resembles the serous membranes. The fluid
for ordinary needs comes by transudation from its vessels and glands
and no demand is made upon the lachrymal gland except under
special circumstances.
The eversion of the upper lid may be most easily effected in the
following way: The patient sits facing the operator, holds his head
erect and looks at his lap, or the floor. Place the left thumb on the
brow with its tip at the upper edge of the tarsus, while the other
fingers rest on the forehead: gently press down the skin and with
the other hand seize the border of the lid and bend it over the
thumb nail as one would fold a piece of paper. When a patient
offers no resistance one may turn the lid readily upon a pencil or a
probe laid transversely across its orbital portion, but the thumb
gives aid in forcing down the brow and skin and after eversion
helps to extrude the fornix to view. A fuller exposure of the fornix
is gained by pressing the eyeball backward through the lower lid
as the cornea turns down and the eversion is maintained. As the
globe is pushed backward the folds evert beautifully. If the lid
be very short or swollen, or the patient rebellious, place one thumb
at the upper edge of the tarsus, cover the tip of the other with a
bit of muslin and pressing against the lid border push it up and
over—one thumb aids the other. The muslin prevents slipping and
the upper thumb catches the tarsal edge. For young children who
make great resistance, lay them on the back in the mother's lap
with the head between the operator's knees. Use the point of the
forefinger as a fulcrum and with the other pull the lid over it. A
bit of muslin will often be of service. It may be necessary to use
an anaesthetic. Eversion of the inferior cul-de-sac is usually easy,
but its complete exposure is secured by first having the patient look
far down, then with the finger depress the tarsal edge and finally
bid the patient look up—the cul-de-sac at once rolls out.
Diseases of the Conjunctiva.
According to Cohn's tables, 30$ of the diseases of the eye are
furnished by the conjunctiva, and of these almost all are inflamma-
THE CONJUNCTIVA.
311
tory. The phases of conjunctivitis are various, and numerous sub-
divisions can be made according to the principles used in classifica-
tion. It is better to do this so far as possible according to the
pathological types presented. The general features of conjunctivi-
tis (syndesmitis) are redness, cedematous swelling both in and be-
neath the membrane, increase of secretion, which will consist of
tears, serum, mucus, cast-off epithelium, and a greater or less
amount of pus cells; there may be coagulable exudation upon or in
the membrane; the lymphoid elements will be increased both in
size and quantity, the papillary structure may be hypertrophied.
It may be said that congestion and undue and abnormal secre-
tion always exist in this inflammation and on the latter character-
istic particular stress must be laid. It is also to be emphasized,
that in general the secretions of conjunctivitis are contagious. For
the milder forms this is measurably true, while in acute and florid
types the contagiousness is extreme and the effect liable to be
severe. In certain cases, viz., in most of the purulent and plastic
types, the contagious element is a micrococcus of a distinct char-
acter. Communicability resides not only in the secretions, but in
some forms, seems to pervade the atmosphere, giving rise to endemic
inflammations in asylums, homes, and hospitals, and in them the
disease is prone to exhibit or degenerate into various pathological
phases grouped under the name of granular conjunctivitis.
We may make the following subdivisions: 1st hyperaemia of the
conjunctiva; 2d, conjunc simplex or catarrhalis; 3d, conj. purulenta
or blennorrhoica; 4th, conj. crouposa; 5th, conj. diphtheritica; 6th,
conj. granulosa; 7th, conj. phlyctenulosa. To these inflammatory
types we add, 8th, xeroma; 9th, lymphangiomata; 10th, hemor-
rhage; 11th, wounds and burns; 12th, tumors and ulcerations;
13th, subconjunctival emphysema.
Conjunctivitis.
1st. Hyperaemia palpebralis is frequent, may be acute or
chronic, and is usually symptomatic. Mere hyperaemia of the ocu-
lar part of the membrane is rare and apt to be traumatic. Yet
there are persons who get a mild attack of this sort for which they
apply a cold compress at night and find their eyes quite well in the
morning. They are usually affected with nasal catarrh. Hyper-
aemia of the tarsal conjunctiva is not seldom incorrectly called
granular conjunctivitis, because the surface may be slightly more
velvety than usual. The outer and inner angles always show the
deepest congestion, while along the middle of the tarsus the vessels,
both the large and fine, give a decidedly pink hue to the usually
yellowish-pink surface. There is commonly only a trifling increase
of secretion, almost no increase of thickness, and the papillary struc-
312 DISEASES OF THE EYE.
ture is developed no more than may be compared to the surface of
fine emery paper, and the transparency is not lost. Complaint is
made of dryness, of smarting, pricking, or gritty sensations: per-
haps the lids cannot be kept fully open, there may be some photo-
phobia. The symptoms are worse at night and are aggravated by
use of the eyes. The most annoying cases of this kind are those
which are caused by various forms of eye strain. Whatever makes
vision difficult, excites this hyperaemia. We see it in those who use
their eyes to excess, for long hours, or late at night, in those who
have wept extremely; in those who have errors of refraction, such
as beginning myopia, or hyperopia, or astigmatism, or anisometropia
or spasm of accommodation, or beginning presbyopia; in those who
suffer from fatigue of the motor muscles (muscular asthenopia in
various types). We see it with faint opacity of the middle of the
cornea, with incipient cataract, or diffused haziness of the vitreous.
It accompanies chronic ophthalmia tarsi, which is itself often occa-
sioned by refractive or muscular errors. It is often coincident with
chronic nasal catarrh, and if this develop at times into acute activity
as "hay fever" or "rose cold," etc., the palpebral hyperaemia cor-
respondingly increases and may grow to active conjunctivitis.
Those who work in dust, such as millers or street sweepers, etc.,
naturally have this condition, and it always attends the lodgment
of a foreign body under the lid. There is a chronic type of the dis-
ease found chiefly in old persons whose eyelids are baggy and who
may have ectropium. It always co-exists with catarrh of the lach-
rymal sac and stricture of the nasal duct. Among all these sub-
jects those who complain most will be the asthenopic persons, and
the more excitable they are, the more will they emphasize their
troubles.
Treatment.—The important point is to ascertain and remove
the cause. It may disappear upon receiving suitable glasses for
refractive or muscular errors, upon the removal of a foreign body,
upon the subsidence of a stye, the removal of a cyst of the tarsus,
or the cure of nasal catarrh. Concurrently with the proceedings
hinted at, direct remedies may be called for which will be both
soothing and mildly stimulating. Weak solutions of common salt,
half a teaspoonful in a pint of water, the addition of a few drops of
brandy to a tumbler of water, or as the fashion now is, of " Pond's
extract" (fl. extr. hamamelis) in quantity varying from a few drops
to a teaspoonful in a glass of water, are in popular use. The
cold douche from a fine rose jet arranged as may be convenient or
spray of cold water from a double-bulb spray apparatus, are often
grateful and to it the addition of a few drops of bay rum gives
more efficacy. Some of the usual prescriptions are as follows: I>
Sodae biboratis pulv., 3i.; aquae camph., § vi. 5 Acidi borici 3i.-
THE CONJUNCTIVA.
313
aquae, ? vi. $ Zinci sulph., gr. ij.; aquae, 5 iv. B Fl. ext. opii., 3 ij.;
aquae, \ iv. These mixtures are to be dropped between the lids by
a dropper or may be put into an eye cup which is to be held to the
eye and the lids being opened, the fluid remains for half a minute
or more in contact with the globe. Afterward it may be' applied
by a compress to the outer surface of the lids for ten minutes. Such
applications are made morning and evening, or at such times as the
patient prefers.
If such measures do not remove the irritation, applications
directly to the tarsal surface will be in order, and the surgeon
must evert the lids. For this purpose a solution of nitrate of
silver, gr. ss.-ij. to water 3 i. or in chronic cases, tannin, gr. x.,
glycerin, 3 i., or a smooth crystal of alum may be empoyed every
second or third day. It is sometimes useful to apply to the tarsal
border at night a weak mercurial ointment, such as, 1> hydrarg.
oxid. flav., gr. ij.; vaselini, 3 i.; or t> Unguenti citrini, gr. ij.; vase-
lini, 3 i.; vel cosmolini, 3 i., in lieu of the "touching" of the lids and
often in connection with some of the above-named collyria. The
mixture of borax and camphor water is most frequent and is widely
known. Solution of tannin in glycerin, gr. xx. ad 3 L, may be ap-
plied by a spray apparatus (Agnew).
2., Conjunctivitis simplex vel catarrhalis.—It is necessary to
allow considerable latitude to the definition of this inflammation,
because we meet it in various degree and phases. One might make
of it three subdivisions, viz., C. Simplex, C. CEdematosa and C.
Catarrhalis. The first is attended by little swelling and presents
chiefly hyperaemia. The second is not frequent and exhibits little
redness, but abundant serous effusion in and beneath the conjunc-
tiva and in the lids. It occurs in young subjects of delicate and
lymphatic habit, is not much painful and there is little sticky secre-
tion. The membrane shows little vascularity and the prevailing
tone of color is a tawny yellow. This occurs idiopathically, while
precisely similar conditions accompany some cases of periostitis
and other orbital inflammations. The third is the typical and usual
form of catarrhal conjunctivitis and has the following appearances.
Increase of vascularity both palpebral and ocular; on the globe
the mesh-work of vessels forms close and irregular spaces and is
somewhat closer near the cornea, the hyperaemia is nearly equal in
intensity over all parts of the eye. There will be more or less effu-
sion in and beneath the membrane, and pressure with the finger
through the lid will move it about. There will be swelling of the
lids and inability to fully open them, and some redness along their
margins. But the striking and special symptom is the abnormal
secretion which glues the lashes into bundles and the edges of the
lids together after sleep, which is flocculent and turbid, a mixture
314
DISEASES OF THE EYE.
of serum, tears, mucous epithelium and of some pus-cells. It collects
in flakes and spreads in a thin layer upon the tarsal surfaces.
When everted, the palpebral surface is deeply and uniformly red,
succulent, and velvety. At the edge of the cornea there are often
minute erosions and a fringe of vessels may after a time develop
around its whole circumference and reach one or two millimetres
into the transparent cornea. Sometimes there are minute hemor-
rhages in the ocular conjunctiva and the color of the hyperaemia
varies from a bright scarlet to a deep mahogany.
The subjective symptoms vary according to the severity of the
attack and the sensitiveness of the patient. There is heat, and
burning pain, with pricking and itching and the constant sense of
heaviness and of sand in the eyes. At the outer and inner angles
these feelings are most pronounced and often the skin after a time
becomes ulcerated at these sites, to the aggravation of the sensa-
tions. The flow of scalding tears and the persistent collection of
the secretion provokes constant use of a handkerchief and there is
often marked photophobia. The symptoms are apt to be worse at
night and better in the morning. Usually both eyes are affected.
A form of this inflammation called by English writers catarrho-
rheumatic ophthalmia seems to be a mixture of conjunctival and
scleral inflammation. The secretion is more watery than sticky,
there is a deep as well as superficial vascularity, and then there is
acute pain in the globe, the temples, and forehead, added to the
usual burning sensations. The eyeballs are tender to the touch,
there is usually extreme photophobia and the sufferings of the
patient are out of proportion to the apparent severity of the dis-
ease. Such persons are apt to be gouty or rheumatic.
Many times there is little discomfort experienced with catarrhal
conjunctivitis, and especally in healthy persons, and frequently the
services of a physician are not called for.
The disease is idiopathic or symptomatic and in a few cases it
seems to be metastatic. It comes from atmospheric causes, from
the foul air of ill-ventilated rooms, from exposure to dust, smoke
and heat. It is apt to occur at the seaside in the summer, from heat,
glare, and dampness. Workers at the forge, millers, cigar-makers,
moulders are its especial subjects. It is frequent in orphan asylums
and crowded eleemosynary establishments. It is often part of the
attack in acute coryza and in hay-fever or rose cold. Sometimes
epidemics occur and are thought to be more frequent in the spring
and autumn.
A special type of acute conjunctivitis, usually called papillary,
sometimes follicular, both which names indicate the differing phases
set up by the same cause, comes in certain persons from a solution
of sulphate of atropia. (Note previous remarks, p. 240.) With a
THE CONJUNCTIVA.
315
few who are remarkably susceptible a single instillation suffices.
Generally it must have been kept up for some time before this re-
sult appears. There is often erythematous inflammation of the
skin of the lid and of the cheek. The conjunctiva will exhibit besides
redness and hypersecretion, enlargement of the follicles in the tar-
sal folds. The attack promptly abates on withdrawal of the atropia.
Furthermore we have epidemics of catarrhal conjunctivitis,
usually of mild type with moderate quantity of secretion from the
eyes and attended by coryza. The attack lasts usually from three
to ten days and may be widespread in the community. It is pop-
ularly known as "pink eye" and evidently resembles the "distem-
per " of horses and other animals. Dr. John E. Weeks has studied
its micrography and found it to be a germ disease communica-
ble by contagion. He has isolated its special bacillus in pure
cultures.1
We have mild conjunctivitis during extreme summer heat, espe-
cially if combined with exposure to the sand of the desert, or the
beach, or the arid dust of the plains. It happens to travellers
among arctic snows. Facing the glare of the electric light will
produce it: if the light is properly diffused no harm results.
The symptomatic or secondary forms of the disease are numer-
ous, as in dacryocystitis, acute and chronic, in hordeolum and ab-
scess of the lid, in erysipelas, in eczema, in herpes zoster frontalis,
in all the exanthemata, as measles, scarlatina, variola, and vari-
cella. It is a serious and most annoying complication In acute
eczema of the face, especially in old persons, and is utterly rebel-
lious until the subsidence of the skin disease. It is an early token
in measles, while in scarlet fever it comes during the later stages
of the eruption, and similarly with small-pox.
Certain fugitive attacks in cases of gonorrhoea (Haltenhoff)
seem to be metastatic, expressly excluding their production by the
conveyance of secretion, and this opinion is founded upon the nature
of the attack, its mildness, its short duration, the absence of the
purulent secretion, and (of obviously less value as evidence) upon
the negation by the patient of any carelessness on his own part.
There may "be coincident inflammation of the joints (gonorrheal).
Such occurrences are rare. Cases have been observed in which a
mild conjunctivitis has preceded each menstruation. Dr. W. O.
Moore related such a case to the New York Ophthalmological
Society.
Duration, Complications, and Sequelae,.—There is a notable
tendency to spontaneous recovery, and the duration of the disease
will be from a few days to three weeks. If, however, the surround-
ings be unfavorable, or if no care be taken or the person be cachectic
or of bad habits, it may become a chronic malady. This is apt to
'Knapp, Arch, of Ophth., xv., p. 441, 1886.
31G
DISEASES OF THE EYE.
be the case among tenement houses, and in badly managed institu-
tions, where a seemingly mild attack refuses to get well, and passes
over into a state of thickening and infiltration of the tarsal and
orbital portions of the membrane, with hypertrophy of the papillae
and of the epithelium; a condition conveniently called granular
conjunctivitis, and it is extremely obstinate although it may not
seem to be severe. Again a simple attack in old persons is apt to
degenerate into a chronic condition, and hard drinkers have been
noted since the time of Solomon for " redness of eyes." It is rare
that the cornea becomes affected, save in the moderate way above
denoted. After measles the eyes are liable to remain irritable for
a long period, and while no vigorous treatment seems needful, care
in abstaining from use is required. It is generally true that the
eyes are apt to be irritable for several weeks, even when a case has
seemed to recover well. The possibility of granular conjunctivitis
ensuing under special conditions has been mentioned and it is also
possible for persons who give no attention to cleanliness, especially
if aged, to have chronic blepharitis, lippitudo and ectropium, and
perhaps impairment of the integrity of the cornea.
Treatment.—The first and chief consideration is scrupulous
cleanliness, and where an endemic has broken out in an institution,
the removal of the affected into large and well-ventilated rooms,
and giving each abundant air space. Simple cleansing of the eye
from all secretion by mild lotions is often all that is required. The
first question always is, what should be the temperature of the fluid?
That may be left to the sensations of the patient, whether it shall
be lukewarm or cool, but as to the mode of application instruction
must be given that it shall remove the secretion and not retain it
within the lids. Hence, poultices, tight compresses, bandages, raw
oysters and a farrago of disgusting popular remedies must be pro-
hibited. It is also prudent to discard sponges, because they are
apt to become means of contagion. A simple compress of linen or
muslin, or a pad of absorbent cotton wetted in the fluid, may be
applied for fifteen minutes or more at intervals of one or more hours
according to the seriousness of the case. It should be wetted afresh
every three or five minutes and the secretion gently wiped away
from the edges of the lids. The purpose is to keep the eye clean, to
remove and antagonize septic elements and to exert by the lotion a
moderate control over inflammatory action. For mild cases a cool
lotion, for more severe a cold lotion will be chosen. Nothing serves
so well in the great majority of cases as a 3$ solution of boric acid.
It need not be accurately prepared, but the patient may be told to
put a teaspoonful of the crystallized acid in half a pint of water and
use it copiously. If the amount of secretion require, it may be
squirted between the lids by a dropping tube ora small bulb. It is
THE CONJUNCTIVA.
317
grateful to the inflamed surface and is of undoubted value. For a
very large number of cases it fully meets the purpose and it has
grown to be the stock prescription in the practice of the New York
Eye and Ear Infirmary. It must not be forgotten that in all mild
cases of conjunctivitis the diseased condition is of short duration,
and that amid healthy surroundings a complete and prompt recov-
ery is the natural order of events. Many persons prescribe for
themselves weak sulphate of zinc solution dropped in the eyes
at night. We therefore will carefully avoid unnecessary treatment,
and especially any strenuous or harsh measures, which while they
exhibit zeal, are in reality evidence of indiscretion. Under this cat-
egory may be put leeches and blisters and the use of irritating
" drops," etc. The treatment will be aimed at the uncomfortable
symptoms. Under this head may be classed the cleansing of the
eye, as has already been stated: before going to sleep the edges of
the lids may be smeared with simple cerate, or unperfumed cold-
cream, or vaseline (the last melts and disappears very soon), the
same may be rubbed over the skin of the cheek or lids if the surface
is irritated by frequent wetting. If there be much discomfort from
feelings of grittiness or heat or smarting, a 2f0 solution of hydro-
chlorate of cocaine may be dropped between the lids at intervals of
ten minutes until the sensations are allayed. This remedy has also
the happy property of contracting the blood-vessels, thus serving a
double purpose. It is, however, not to be used except to relieve
discomfort and not with great frequency.
Many cases will require no other measures and a few days will
see the eyes restored to health. For cases of more severity with a
little swelling of the lids, slight sub-conjunctival effusion, more
copious secretion and more pain, it may be needful to employ in •
addition astringent solutions. Among the oldest is a mixture of
acetate of lead with infusion of opium, a drachm of the former to a
pint of the latter. It doubtless has useful properties, but it must
never be used when there is any tendency to ulceration of the
cornea, because the lead is liable to be precipitated upon it. Its
utility is in the early stage of a somewhat acute attack, and then
is most grateful if warm. The mixture should be strained and
made clear. It may be made also as follows: r> Liq. plumbi sub-
acetatis, 3 ij.; Fl. ext. oph. deodorat., I i.; Aquae, 3 vi. M. A solution
of alum, a drachm to the pint, is with many a favorite application
both inside and outside of the lids. Mr. Tweedy, of the Royal London
Ophthalmic Hospital (Moorfields), and some others have asserted
that it has a tendency to increase ulcerations of the cornea in virtue
of a solvent effect upon its cementum. He thinks he has seen de-
cidedly mischievous effects of this kind. For myself I have almost
discarded alum in favor of boric acid, and when the quantity of
31S
DISEASES OF THE EYE.
secretion or the intense redness, or the degree of effusion call for
active interference, the nitrate of silver is the most satisfactory
remedy. A solution, two grains to the ounce, £#, may be dropped
into the eye twice daily from a dropper. If the tenderness and
swelling do not prevent, the same solution, or one five grains to the
ounce, lfc, may be applied with a small brush to the everted lids
once daily, or be dropped into the eye. It causes a littie pain,
for which a solution of cocaine may be used, and compresses dipped
in iced water sedulously applied until the reaction subsides.
For very nervous patients it may be needful to resort to bro-
mides, and possibly to mild closes of morphine or Dover's powder at
night, or better still to phenacetine, gr. x., once in two hours.
It is sometimes well to add sulphate of zinc to boric acid in the
later stages of a moderately severe attack, as for instance: $ Zinci
sulphatis, gr. iij.; Acidi boric, 3 i.; Aquae, 3" vi. One may resort to
solutions of corrosive sublimate, from 1 to 3,000 to 10,000, as was
formerly done, and this would be suited to the mild epidemic cases,
and to hospital or tenement-house out-breaks. It is irritating
to many patients, but it is effective.
For cases where cedematous infiltration is great and secretion
moderate, as in delicate children and sometimes in old people, solu-
tion of tannin, gr. v. ad 1 i., or the liquor plumbi subacetat., 3 ij. ad
3 viij., is well adapted.
For severe cases of so-called " catarrho-rheumatic ophthalmia "
it may be needful at the outset to apply two or four leeches to the
temple to allay pain, to make very hot fomentations for the first
day or two, to employ sol. sulphat. atropiae, gr. ij. ad 3 i., two or
three times daily in the eye, and also perhaps a 2$ solution of coca-
ine. No astringents or irritating drops are to be used until muco-
purulent secretion is set up, and the temperature of all applications
must be comforting to the patient. A saline purgative and often
small doses of Rochelle salt frequently given for diuretic effect, will
be valuable. In these cases we have to do no doubt with a general
dyscrasia. Especially must one look for symptoms of a gouty dia-
thesis, of which the signs may be slight and the tendencies not
readily admitted by the patient.
As the acute attack subsides, if there be left a state of chronic
hyperaemia, one may touch the everted lids once daily with solu-
tion of nitrate of silver, gr. ss.-ij. ad 3 i., or with a smooth crystal
of alum. Or if a patient cannot be so frequently seen he may drop
into his eye a solution of zinc sulphate and boric acid. A patient
never should have a solution of nitrate of silver given to him for
this purpose. It soon decomposes, and its prolonged use causes
brownish discoloration of the conjunctiva (argyria conjunctivae).
It need hardly be said that during an acute attack a patient
THE CONJUNCTIVA.
319
must not read or write or smoke, and will remain in a dimly lighted,
well-ventilated room. Even when recovery has been attained, cau-
tion must be given against prolonged use of the eyes, especially at
night, and against exposure to wind, dust, smoke and foul air.
Often colored glasses (light smoked or blue) will have to be worn
in bright light.
In healthy persons with favorable surroundings and with proper
care, the outcome of acute catarrhal conjunctivitis is perfect recov-
ery within periods varying from five days to three weeks. On the
other hand, there may be ulceration of the cornea with its various
possibilities, or there may be chronic thickening of the palpebral
conjunctiva, ectropium, pterygium, etc., etc. These conditions have
been or will be considered under proper heads. Not a few cases of
so-called granular lids have originated in an acute conjunctivitis.
3. Conjunctivitis purulenta, Blennorrhcea, Pyorrhea.— This
phase of acute conjunctivitis is of more severity than the foregoing
and present itself at different ages: first it occurs in infants at or
soon after birth, and secondly it occurs in adults. In both classes
the disease is essentially the same, and originates from contagion
in the great majority of cases, but it is convenient and customary
to consider them separately. The first is commonly called
Ophthalmia Neonatorum.—At birth the eyelids are always ag-
glutinated by the parturient secretions. It is common too for the
lids to remain red and sticky for a day or two. The nurse washes
them off with warm milk and water, and soon the eyes are clear.
A little attention may be required for several days, the principal
point being the careful removal of secretion as fast as it may appear,
and soon all is well. Should there be a little swelling of the lids,
and secretion be rather troublesome, a compress wetted with a
solution of borax or of alum may be laid on the eyes for fifteen
minutes, once in two or four hours, and the edges of the lids smeared
with simple cerate or vaseline. The great proportion of the cases of
this disease will not require serious attention, and will not cause
the physician any anxiety. A physician in large obstetric practice
told me that he had had only one serious case in an experience of
twenty years. But there are cases even in private practice which
need active attention. Of these I have seen two varieties.
The less frequent are some which have more the character of
granular than of purulent conjunctivitis. There is very little
swelling of the lids—secretion is almost wholly watery. There is
little or no hyperaemia of the ocular conjunctiva, while the palpe-
bral conjunctiva is reddened, and at the fornix is considerably
thickened and swollen. This hypertrophy is the essence of the
trouble. It is truly a form of follicular conjunctivitis occurring at
an exceptional age. While this continues the eye keeps watering
320
DISEASES OF THE EYE.
and a little sticky, and the condition goes on for weeks. The cornea
does not get hazy, and there is no special danger. For the milder
cases, where an anxious mother insists on something being done for
the baby, I deprecate anything more than washing with a solution
of boracic acid several times a day. But if a child is a month old, and
the discharge continues and the fornix exhibits decided swelling, I
have been obliged to use solutions of tannin and glycerin as strong
as 3ij. ad 3 i. before the condition would yield. I had tried nitrate
of silver in mild solution, and, unwilling to make it more caustic,
had taken a solution of tannin, gr. x. ad glycerin, § i.; but this had
only a temporary good effect, and the disease was not subdued
until the strong solution was applied. It was done every second
day to the everted lid, and was of course quite painful.
Much more important is the purulent conjunctivitis which is
generally spoken of as ophthalmia neonatorum. It presents itself
under various degrees of intensity, with swelling of the lids, with
yellow, thick secretion issuing from the eye, or, if dried upon the
tarsal edges, it glues the lids to each other, and the conjunctiva,
both ocular and palpebral, is reddened and swollen; at the begm-
ning the secretion is comparatively thin, but soon it becomes
thick and creamy. This quality indicates abatement in the ac-
tivity of the process. There may be chemosis, and the palpebral
conjunctiva be thickened and intensely red and spongy, with
ridges and prominences, and be cleft by fissures between the en-
larged papillae. The tumidity of the palpebral conjunctiva, which
increases up to the fornix, is a notable feature. The cornea may
remain clear, but its integrity is the object of anxiety. Its inva-
sion may show, as the first sigh, a diffused haziness, or a single spot
of purulent infiltration, or an ulceration; while in weakly infants,
of whom premature births and foundlings are often extreme exam-
ples, the cornea may suddenly break down with general infiltration,
and become a mass of yellow putrilage. The place of ulceration in
the cornea may be anywhere; if upon the periphery, and if it per-
forate, the iris falls into the opening and becomes permanently
adherent, and from this a partial staphyloma may ensue; or, in
case of less extensive destruction, nothing more than a distortion
or concealment of the pupil. Should the ulcer be central, and not
too large, so that if it perforate and the sphincter iridis cannot be
drawn into the opening, the lens will then come forward and rest
in contact with the posterior surface of the cornea. Sometimes a
fistula will give rise to prolonged contact of the lens-capsule with
the cornea. As a consequence, there will be an opacity reaching a
little into the centre of the lens, at its middle a small white dot and
around it will be a fainter zone, and the whole will be apparently
two or three millimetres in diameter. Sometimes, long after the
THE CONJUNCTIVA. 321
eye is well, a fine thread is seen running from the lens to the mid-
dle of the cornea, which is the attenuated vestige of the material
deposited while the surfaces were in contact. The opacity belongs
to the capsule chiefly, and is both upon it and overlaid by it, some-
times having a distinct pyramidal form. It will be again referred
to in speaking of cataract. In cases so marked as the above the
secretion will be thick, creamy, and copious. The patients do not
seem to suffer much pain, and usually nurse well. The skin of the
cheek is apt to be excoriated by the discharge.
Pi-ophylaxis.—The importance of preventing this disease will
be appreciated from the statistics given by Haussmann.1 Of the
inmates of blind asylums the number made blind by this disease
was in Copenhagen, 8$, in Berlin, 20$, Vienna, 30$, in Paris among
208 blind young subjects, 45$. In 1876 it was shown that among
the young persons admitted to the blind institutions of Germany
and Austria, 33$ had been made sightless by this disease. In dif-
ferent countries the variation was from 20$ to 79$ (Horner). In
Philadelphia, Dr. Harlan found in 1871, out of 167 inmates of the
blind asylum about 20$ had been admitted for this cause. In oph-
thalmic clinics, Hirschberg had among 21,040 new cases 314 of this
kind; Scholer among 10,000,156 of this kind. Horner in 1862 found
among 10,000 cases, 161 of this variety and he gives this interesting
analysis. Of recent cases there were 108, and of old cases in whom
the disease had terminated, there were 53. Of the last named 53,
14 Avere blind in both eyes, 24 blind of one eye, and 15 had more or
less serious corneal opacities. Of the 108 active cases only 24 were
brought during the first week of the disease, and the remainder,
viz., 84, came at later periods. Of the 24 cases seen during the first
week, 2 already had corneal trouble; one healed with a trifling opac-
ity, the other died before the end of the disease;—22 recovered
with perfect sight. Out of the 84 less recent cases, 38 had disease
of the cornea, and 46 did not have it. Of the 46, 3 acquired corneal
ulcerat ion, but in none of these patients was the eye lost. For the
statistics of lying-in hospitals on this subject, reference may be
made to Haussmann, 1. c, and to Crede, "Die Verhiitung der
Augenentziindung der Neugeborenen," Archiv f. Gyndkol, XVII.,
1, S. 50 (also in separate form, Berlin, 18S4). The attention which
of late years has been given to prevention of contagion, has proved
by its success that to this, we are to look in the great majority
of cases for its cause.
The origin of the contagion is the morbid vaginal secretions.
The healthy secretions of the parturient state do not produce the
eye trouble, at any rate there is strong reason for this belief, both
from general experience and because direct inoculation of healthy
1 "Die Bindehaut-Infection der Neugeborenen," Stuttgart, 1882.
21
322
DISEASES OF THE EYE.
secretion has been practised without evil effects. Microscopic
study has found that both in the morbid vaginal secretions and in
the pus flowing from the eye a special organism appears which was
first pointed out by Neisser, of Breslau, in 1879 (Centralblatt fur
Med. Wissensch., No. 28), and called by him the gonococcus, because
it is characteristic of gonorrhoea of the urethra. This kind of infec-
tion is in the great proportion of cases the cause of the infantile
disease. Other infectious causes cannot perhaps be absolutely ex-
cluded, such as the lochial secretion, or that from a suppurating
navel, while from the nature of the case few investigations are
made on this point, and it is also impossible to prove a negative.
The eye disease appears usually about the third day, but may be
delayed as late as the eighth day.
The prophylactic measures to be employed are on the one hand
the washing of the vagina for some days before parturition, and
while labor is going on, with a 3$ solution of carbolic acid in all
cases where the secretions are of a suspicious character. On the
other hand attention is to be given to the eyes of the infant. By
some,washing the outer (not the inner) surface of the lids and the
eyelashes with a 3$ carbolic solution is practised, while in accord-
ance with the recommendation of Crede a single drop of a 2$ solu-
tion (gr. x. ad 3 i.) of nitrate of silver is dropped between the lids of
each eye by a glass rod. The effect of these measures has been
to reduce the frequency of the infantile disease in the lying-in hos-
pital of Halle from 12$ to 3$. In the lying-in hospital of Leipsic,
where Crede instituted his own method, the cases fell from 7.5$
to 0.5$.
It is impossible to resist the force of these facts and it becomes
imperative to employ such measures in all cases where any suspi-
cion may arise. This holds good especially in hospitals and lying-in
wards, but it is applicable also to practice among the uncleanly
and the poor. The solution of nitrate of silver in such strength, 2$,
excites irritation, but a very small quantity is intended to be used
(a single drop) and it neutralizes the poison. A 1$ solution will
certainly suffice, and the rigorous proceedings of a public lying-in
hospital do not fully apply to private practice. For infants where
only the usual secretions are present, cleansing the lids and eyes
with a solution of borax or with any mild antiseptic solution or
with warm water will suffice. It is seldom but that both eyes are
infected at the same time. In case one only is inflamed, the other
should be hermetically closed, and this can best be done by strips
of india-rubber plaster, which may be left on for forty-eight hours
and then renewed. The disease is usually more severe in the eye
first affected.
Treatment.—If seen at the very beginning when the swelling of
THE CONJUNCTIVA.
323
the lids and a watery secretion are the chief symptoms, nothing
but cold lotions and assiduous cleansing are to be employed. Be-
sides the attested clinical value of cold lotions, Weeks found that
a low temperature destroyed the vitality of the germs which he
isolated in simple catarrhal conjunctivitis; the same is true of
the more virulent types now considered. For the cleansing, boric
acid 3$ solution may be used. With the head upon the lap the
solutions is to be squeezed from a rag or from a mass of absorb-
ent cotton between the separated lids, and if needful is wiped away
with a camel's-hair brush. The wetted cotton may be pressed
lightly on the closed lids to squeeze out the flocculent secretion,
and this is to be repeated as often as accumulation occurs. The
cold application consists in laying linen or well-washed cotton
compresses in iced water or on a block of ice, and transferring
them every few minutes to the eye. In moderately severe cases
the water need not be so cold. But the lotion is to be kept up day
and night. As soon as the secretion grows a little thicker and the
swelling of the lid grows less, so that it can be everted, we are to
resort to nitrate of silver in strength varying from 1$ to 2$, gr. v.
-x. ad 3 i. This is not to be dropped between the lids because it
will thus endanger the cornea. But the upper lid is first to be
everted, and if difficult to do, as often is true, the tip of the finger
may be wrapped in a bit of rag or in the corner of the towel to aid
in pushing or pulling the tarsal edge against the finger of the other
hand which pushes down the brow. The physician has the back of
the child's head between his knees and is provided with water and
bits of rag. All secretion is to be wiped away and the caustic solu-
tion pencilled over the exposed palpebral conjunctiva, carefully
avoiding the cornea. Afterward the lower lid is to be similarly
treated. The effect of the solution is in proportion to the quantity
applied and the lid should be washed with water afterward. If
there be a little delay in the washing, the caustic effect is greater.
The degree of effect can be estimated by the degree to which
the surface is whitened, and repeated pencillings will intensify it.
With weak solutions one may obtain various degrees of caustic
action. Such a proceeding is to be gone through once in twenty-
four hours, very seldom will it be required once in twelve hours.
Immediately after, iced cloths are to be applied and very frequently
renewed until the cries of the child cease and the pain has presum-
ably disappeared. For several hours the secretion is held in check,
although the swelling increases, and during all the time of its in-
crease and of the painfulness of the eye, the cold is to be continued;
this may be for two or four hours. After a time flocculent portions
of the eschar and renewed secretion appear. Now cleansing is in
order and the lotions will be kept up with less assiduity as the
324
DISEASES OF THE EYE.
swelling abates. If the daily personal attendance of the physician
is not possible, a solution gr. v. ad 3 i. may be dropped between the
lids three times daily, taking pains to insure its entrance beneath
the upper lid by lifting it off the globe and at the same time avoid-
ing contact of the caustic with the cornea. It is not easy and is
very important to inspect the cornea, to watch the first tokens of
its invasion. To separate the lids, Desmarres' elevators are inval-
uable (See Fig. 130).
In skilful hands what is known as the mitigated caustic stick
(one part nitrate of silver with two parts of nitrate of potash) may
be usefully employed. It is more energetic than solutions, it can
be more accurately applied and finds its special utility in the later
stages of the disease when papilliform swelling is extreme, and the
secretion is very thick. Tlie cornea must be absolutely avoided
Fig. 130.
and the excess of caustic washed away. In ophthalmic clinics this
is in considerable use.
Frequently the conjunctiva bleeds under the handling; it is not,
however, needful to resort to scarifications, nor is canthotomy to
be practised. In infants, despite the great swelling, there is little
danger of strangulation of the tissues, and leeches are never to be
employed.
In the event of the cornea having become affected, whether by
opacity or by superficial ulceration, the caustic treatment is not to
be interrupted, neither is it to be made more vigorous. If deep
ulceration take place a solution of atropia, gr. ij. ad 3 i., may be
dropped into the eye three times daily or oftener, to secure dilatation
of the pupil. For marginal ulcers, eserine sulphate gr. i. ad 3 i.
may be used twice daily according to effect, and atropine during
the interim. If the ulcer threaten to perforate, a careful paracen-
tesis at its thinnest point will sometimes do good, but one rarely
has an opportunity to try it and it is always a delicate and difficult
thing to do. On the other hand, if perforation spontaneously occur,
there is generally an improvement in the condition. It is not
meant that spontaneous perforation with the resulting prolapse of
the iris is not to be deplored. Temporarily it affords relief, but it
brings about permanent damage to sight. It is unwise to interfere
THE CONJUNCTIVA.
325
with prolapse of iris unless it projects in a conspicuous prominence,
when sometimes careful puncture, holding the needle at a tangent
to the summit, will abate it. Excision is not to be practised. In
l)ad cases the whole cornea may rapidly melt down, the iris exten-
sively prolapse, the tissues rupture, and the lens escape. A staphy-
loma of the cornea or phthisis of the globe may eventuate. Some
writers have reported favorably upon finely powdered iodoform
dusted over the everted lids (Grossman1 and others). It is said to
check the discharge and have a favorable effect on the cornea after
infiltration has begun. It is applied once or twice daily. Since
its announcement in 1882, little has been said about it.
The duration of treatment in favorable cases will be from four
to six weeks, while in bad cases it may go on much longer. The
length of treatment and the prognosis as to result are greatly in-
fluenced for good or bad by the health of the subject and by effi-
ciency as well as early commencement of the treatment. But even
in cases which seem most unpromising the wonderful recuperative
powers of early life, will eventually bring about a degree of im-
provement for which there seemed to be no prospect.
Sometimes the conjunctiva takes on extreme hypertrophy; the
purulent secretion almost ceases, the cornea may be clear, the activ-
ity of the disease is at an end, but there has not been subsidence of
the swelling, and a constant eversion of the lids remains (paraphimo-
sis.) It does little good to apply caustics or stimulants or astrin-
gents. The lids may be inverted, but in a moment or certainly as
soon as the child cries, the deformity returns. Nothing relieves this
condition but mechanical methods. The lids must be inverted and
so retained by strips of india-rubber plaster, and their ends may be
held down by laying over them a few fibres of cotton and saturat-
ing this with contractile collodion. In fact such a dressing may
cover the whole of the lids save a spot for secretion to escape.
Should this fail after a fair trial, and it is not likely to fail, one
might in a severe case carefully pare the edges of the lids over the
middle third without doing injury to the cilia and stitch them to
each other. They will grow fast and may so remain for several
weeks until the hypertrophy has disappeared. I should give sev-
.eral weeks' trial to the other method, provided the dressing could
be kept in place without irritating the skin. It must be renewed
every three or four days.
Purulent conjunctivitis in adults (gonorrhoealophthalmia), is
essentially the same disease as above described in infants. In a
few instances it seems to be of spontaneous origin, but these are
very rare, while in the vast majority some source of contagion can
be traced. This will be from an acute or chronic gonorrhoea either
1 Ophthalmic Review, Vol. i., 1882, p. 214.
326 DISEASES OF THE EYE.
in the male or female, by communication from a similarly diseased
eye, by indirect contact through the fingers or handkerchiefs, tow-
els, clothing or rags. The secretions from diphtheritic conjunctivitis
or from vaginal leucorrhoea cause it. A very minute quantity is
sufficient and that derived from an old gleet remains effective. The
right eye, for obvious reasons, is oftener concerned than the left.
More frequently than with infants is one eye only involved.
Reference has been made to the gonococcus Neisseri as the effi-
cient agent in this disease. The micro-organism appears usually
as diplococci in twos or fours within the pus-cells, or in isolated
groups, and are also found at considerable depth in the epithelium
and in the tissue of the conjunctiva. Their malignant influence has
been unquestionably demonstrated by the fact that inoculation with
pure cultures causes urethritis in the human subject. Moreover,
Deutschmann reports a case of arthritis gonorrhoica in an infant
with purulent ophthalmia (Graefe's Archiv, XXXVL, 1, 114, 1890).
The cocci were in the conjunctival secretion and in the fluid ob-
Fig. 131.
tained from the joints by puncture. In the eye, the more active
the gonorrhoea at the time of infection, the more violent will be the
inflammation. The period of incubation is said to be about forty-
eight hours. A physician must always be careful lest he incur
infection. Sometimes pus spurts out in a jet as the lids are sepa-
rated. Immediate washing and neutralizing by a drop of 2$ solu-
tion of nitrate of silver will be indicated. The mystery which
sometimes attaches to the transmission of the inflammation to a
healthy eye is lessened, when it is known that the pus diluted 1 to
1,000 still retains decided contagious properties. It ought also to be
stated that nitrate of silver, by its power of coagulating albumen
antidotes in high degree the noxious character of the pus, and it
has been shown that a one-fourth-per-cent solution, or gr. £ ad z i.
renders it innocuous. This fact explains why so few cases of con-
tagion occur in public clinics, where the same brush may be used
for different patients, when it has passed through a solution of
nitrate of silver. I instance this fact, not to encourage economy
THE CONJUNCTIVA.
327
in the use of brushes, but as a point worth knowing. Every patient
needing an application to his lids, should have his own brush; and
purulent cases should be most vigorously quarantined from com-
munication in any manner with other patients.
The symptoms are at the outset hyperaemia of the ocular con-
junctiva, swelling and closure of the lids, and a thin and ichorous
discharge; there will be smarting and burning, which soon rises to
pain both in and around the eye, oedematous infiltration increases
until the lids become hard, tense, and shine with a dusky red, and
the upper overhangs the lower. A similar infiltration takes place
beneath the ocular conjunctiva, lifting it from the globe and making
it mount over the edge of the cornea (chemosis). The secretion in
a little time becomes more and more purulent and soon seems
wholly of this quality. The temperature of the parts is elevated,
and the whole picture is that of an excessively severe inflammatory
process. If the lids can be separated, the conjunctiva is not only
red, but is often speckled with hemorrhages; it is covered with
pus, and especially on the palpebral surface during the early period,
there will also often be a layer of plastic exudation, which will
come off in rolls or may adhere rather closely. When wiped away
the surface beneath it bleeds. It is oftentimes impossible to sepa-
rate the lids enough to view the eye properly, and the attempt
gives so much pain that the patient involuntarily and strenuously
resists it.
The great danger is mischief to the cornea. This may come
during the full activity of the disease or not until we begin to felic-
itate ourselves that the climax of the attack has passed. There
may be a variety of ways in its manifestation. Either the whole
surface may have a uniform opacity of varying density, or this
may appear at the centre or periphery. Ulceration may occur in
company with it or soon follow it, or may be the first invasion. A
furrow at the margin of the cornea, beneath the overhanging che-
mosis, may completely or partially encircle it, and it may eat
through the cornea in a very few days. Ulceration at the centre,
or at any point, is equally prone to occur. The degree of corneal
mischief can never be predicted. It is the product of three factors,
namely, the pressure of the swollen tissues, the corrosive action of
the secretion, including the invasion of the gonococci, and direct con-
tinuity of inflammation to the substance of the cornea. It is also
favored by unhealthy constitutional conditions, and by bad hygienic
surroundings. It is impossible to assign to each factor its exact
measure of influence, or to assert that a peculiar lesion of the cor-
nea must be attributed to predominance of one or the other of
them; in most cases, all concur in the disastrous effect.
The culmination of the active symptoms commonly arrives
328
DISEASES OF THE EYE.
within ten days, and the duration of the disease is from four to
twelve weeks. The final result upon vision, in case the whole cor-
nea is not destroyed or the eye escapes suppuration, is apt to be
better than would seem possible during the severity of the symp-
toms. So much of the cornea as may remain will become far more
transparent and serviceable than may seem at all likely.
Treatment.—From the beginning active measures must be
adopted, and the patient should go to bed. In robust subjects, or
with intense initial swelling or pain, four to six leeches may be put
upon the temple. Bits of muslin taken from iced water, or from a
block of ice, should be kept upon the eye constantly, and changed
every minute or two as they grow warm. In some cases a bit of
ice, wrapped in muslin, may be held upon the eye if its weight can
be endured. Continuous cold, to the degree which can be tolerated,
is the rule. Absolute cleanliness of the inside of the lids is equally
imperative. To effect it, the lids must be drawn apart and the
secretion removed by a soft camel's-hair pencil inserted between
them, or by dropping an antiseptic solution upon the eye from a
small sponge, or by injecting such a solution with a small bulb
syringe. Some one of these methods may be chosen, according to
convenience. This process will be repeated every five or fifteen
minutes during the days and nights of active secretion, and at
longer intervals as the discharge lessens. The antiseptic fluid may
be sol. corrosive sublimate, 1 to 3,000-10,000, or boric acid 4$, or of
chlorinated water 10$. It is sometimes well to use an elevator in
separating the lids, but by sensitive persons this will not be al-
lowed, and extreme delicacy is needful in all manipulations, espe-
cially when ulceration has appeared upon the cornea. In certain
cases in which the discharge is not thick and creamy, but ichorous,
gruel-like, and thin, and in which the attack is moderate in severity,
nothing but this constant cleansing, by antiseptic lotions and the
iced water, are needful; no caustic need be applied. I have seen a
remarkably good recovery of a case like this, in a young man who
acquired the disease from gleet. Mr. Grossmann of Manchester has
recommended the ointment of yellow oxide of mercury in vaseline.
Dr. Wilson of Bridgeport reported success by a mixture of boric
acid and vaseline. Experience has shown that putting vaseline
hourly between the lids is an important aid in getting rid of secre-
tion and preserving the corneal epithelium. This practice is now
generally adopted in New York. The fidelity and thoroughness
of the cleansing by night and day have the greatest influence in the
fight for preservation of the eye. Should the cornea become in-
vaded, a solution of sulphate of atropia, gr. ij. ad § i., should be in-
stilled every three to six hours. It will be seen that, to perform
this laborious nursing, not less than two attendants, both strong
and faithful, are necessary,
THE CONJUNCTIVA.
329
Another indication is to be borne in mind in severe cases,
viz.: the relief of pressure by incising the swollen tissues and un-
loading the vessels. Mr. Tyrrell laid stress on scarification of
the chemotic conjunctiva, the infiltration does not flow away
however deep the cuts, because it is of a plastic and coagulable
quality, but the depletion is of use, and the incision may be re-
peated. Canthotomy freely done unloads the vessels and lessens
the pressure of the lids by weakening the orbicularis and giving
room for the infiltration to spread. I consider this proceeding in-
dispensable when great swelling of the lids exists, and do it with
little swelling if the cornea be threatened. It may be need-
ful to again snip the tissues with scissors in the site of the
cut, after a week or more, because healing quickly takes place, and
conditions of partial strangulation by the lids may be reproduced.
In resorting to such proceedings, a surgeon will require to act with
discretion as well as boldness, appreciating the significance of the
symptoms, the danger to the eye, and the general health of the
patient. As against the propriety of incisions, the partial inter-
ruption of the process of cleansing is to be considered; but this
need not be wholly suspended; its method may be so modified that
the lid need not be greatly disturbed for the next twenty-four
hours.
Mr. George Critehett once resorted to medial vertical division of the upper
lid in an infant and thereby as he thought saved the eye. The writer be-
lieves that such a proceeding might judiciously be sometimes done in adults,
because the disastrous results of the disease are deplorably frequent and the
deformity to the lid could be easily repaired. The advantage is obvious
both by relief of pressure and in facility of cleansing. Exposure of the cor-
nea is the one thing to be guarded against.
Next, we have to consider the use of nitrate of silver. It will have
no place during the period of invasion, and of copious thin, gruel-like,
puriform secretion. But when the secretion becomes creamy and
distinctly purulent, and the conjunctiva velvety, a solution gr. v. vel
x. ad 3 i., may be tried on the everted lids. The longer the brush is
held in contact with the surface, the greater is its effect, and this
may not be small. Pain will ensue for an hour, or longer; for a
time the secretion is suspended, or becomes watery, and if after
twenty-four hours the swelling is less, and the secretion not so
copious, the remedy has been well chosen and may be repeated. It
must be remembered that the first application is tentative, and tG
be carefully watched in its effects, especially on the cornea. The
2$ solution is generally to be preferred (gr. x. ad 3 i.). Formerly
much stronger solutions were in vogue, but stronger than 4$ they
are not to be commended. Thorough contact with all of the con-
junctiva is very difficult even when the lids can be fully everted,
330
DISEASES OF THE EYE.
but an effort must be made to effect it by thrusting the brush up
to the fornix. The solution is by some dropped upon the ocular
conjunctiva, but care must be taken to avoid the cornea; it may be
dropped in three times daily when chemosis is extreme. The ap-
plication of caustic to the lids will usually be made once in twelve
or twenty-four hours, seldom oftener and frequently but once a
day. The recurrence of the purulent secretion is the signal for
repetition.' It is at this point that the good judgment of the
physician is tested. That copious use of vaseline is a valuable co-
adjutor is indubitable.
No remedy has such general acceptance in this disease as nitrate
of silver. Recently peroxide of hydrogen in watery solution con-
taining 3$ by weight has been proposed by Landolt as of value as
an antiseptic. It acts by the rapidity with which it liberates oxy-
gen, and it has the effect of abating the quantity of pus, but exerts no
special control over the inflammation. It must be kept at a tem-
perature below 60° F., and in a bottle tightly corked. The tissues
remain swollen and soggy and the cornea is no less liable to be in-
jured. It does not supersede the caustic It is not irritating pro-
vided in its preparation it is free from sulphuric acid. Iodoform
has been dusted into the eyes, but iced water and other remedies
have at the same time been employed, and we are not authorized to
assign special value to it. It may do well for cases of mild type
and with persons who will not submit to confinement and constant
treatment. The powder must be extremely fine and be dusted
freely over the everted lids. When corneal ulceration exists it may
be used, but the remedy is less in favor now than it was. Recent
authors, Heyl, Freyer, have substituted hot water, of a tempera-
ture as high as 112° to 120° Fahrenheit, for iced water and reported
useful results. Heyl uses it for one-half an hour three times daily,
while Freyer employs it from six to eight hours. Heyl confesses
want of success in dealing with adult cases, and his experience has
chiefly been among infants. With them the disease, if attacked
early enough, is seldom destructive to vision, and whether by hot
water its duration is briefer than by iced applications remains to
be corroborated. Freyer's claim of better success in the adult cases
also awaits corroboration.
Very recently corrosive sublimate has been again brought into use and
in two different methods. The first is applications of solutions 1 to 500 to the
everted lids with such frequency as will hold the purulent secretion in check.
The strength of the solution may even be increased if the physician is con-
stantly at hand and does the work himself; this is feasible only in hospital
practice. Great pain is produced, and all usual precautions of cleanliness
and cold lotions are maintained. Another method is to scarify the con-
junctiva both palpebral and ocular by incisions 2 or 3 mm. apart and brush
it moderately with a solution 1 to 1,000. An anaesthetic must be given. A
THE CONJUNCTIVA.
331
common tooth-brush is used, and canthotomy may be done. A remarkable
abatement of secretion takes place, although the lids may swell. The same
proceeding may have to be repeated at intervals of three days for two or three
occasions. The germicidal value of the remedy suggests the treatment. I
have employed it in five cases with good effect, and purpose to use it further.
An unpleasant complication is the ultimate production of more or less ex-
tensive and irregular adhesions between the lids and the globe. The cases
were patients of the infirmary who could not be taken under hospital care,
and for conditions so unfavorable I was glad to find an effective treatment,
although painful and attended by the drawbacks mentioned. In no instance
was serious damage done to the cornea. The customary auxiliary proceed-
ings were insisted on. The depth to which the gonococcus penetrates the
tissues and the amount of oedema determines the need of scarifications and
the method and vigor of the brushing.
When infiltration of the cornea appears, a solution of atropia
should be used and especially for central ulcerations. It is to be
remembered that iritis is very liable to occur and hence atropia
has an important indication in all corneal complications. Should
there be a marginal ulcer, a weak solution of sulphate of eserine,
gr. ss.-i. ad § i., may be used twice daily. But it does not exclude
the intercurrent use of atropia. If strong eserine solutions are
used, they may provoke pupillary adhesions. In many cases we
are not allowed to debate the propriety of this treatment because
of the rapid advance of ulceration. If it threaten to perforate, we
may perform paracentesis—for actual prolapsus iridis we are to do
nothing with the iris: excision does harm. While in simple corneal
ulcerations without blennorrhcea, we derive advantage from the
actual cautery such is not the fact in the situation now considered.
The tentative use of very hot water, 110° to 120° F., may be sug-
gested on general principles because of the perilous predicament,
but the writer has no experience on the point.
The issue of an attack may be in perfect restoration of the con-
junctiva to its normal texture, in case it were healthy before, but
it not seldom passes through a period of papillary hypertrophy
(granulations) which may continue for weeks. The case may be
under treatment for this condition long after the ocular conjunctiva
has recovered its normal state, and at this stage, sulphate of cop-
per crystal, tannin and glycerin, 3 i. ad 3 i. or stronger, will be the
most useful applications. In the event of the cornea being dam-
aged, all eventualities are possible, between a slight degree of opac-
ity, almost amounting to transparency on the one hand, and sta-
phyloma corneae, or atrophy of the globe, on the other. A partial
staphyloma, or anterior synechia, for which an artificial pupil may
be practicable, is exceedingly common. It is of course possible that
both eyes may be lost, but the right is the one most often affected;
and it is common, when a second eye is attacked, for it to be less
332
DISEASES OF THE EYE.
severely inflamed than the first. The reason is, because the secre-
tions are less noxious during the decline of the inflammation. For
this reason, the pus of a florid urethritis will cause much more
severe inflammation than that from the stage of gleet. The
fatality to sight of this disease is great, and is set down by Klein
as follows: Out of 40 eyes, 16 became totally blind, 9 retained some
vision, and 5 had useful vision, or could obtain it by operation: in 30
the cornea was involved, while in only 10 did it escape. That is,
corneal trouble occurred in three-fourths of the eyes, while 40$ be-
came blind.
The protection of the fellow-eye, in case it be intact, is of the
utmost importance, and my experience has been that the endeavor
to seal it hermetically by a bandage or plasters, is ineffectual. It
cannot be carried out rigorously, because of the wretchedness which
it inflicts in making the patient for the time wholly helpless, and
because of the excoriation of the skin of the cheeks and lid, and of
the impossibility of preventing the patient from slipping off the
covering during the night, and unwittingly exposing himself to
contagion by his soiled fingers or accidental communication from
the other eye. In fact, the need of handling the eye to change the
dressings, which should be done twice daily, involves more exposure
than to warn the patient and attendants of the need of the utmost
caution, and to insist that the patient sleep, lying on his back, or
on the side of the affected eye. A device, whose authorship belongs
to Dr. Buffer, of Montreal, has answered well, viz., to cover the
sound eye with a watch-glass, attached by means of rubber plas-
ter. It need not be removed oftener than once in several days, as
secretion may demand.
4. Plastic Conjunctivitis, viz.: a. croupous and b. diphther-
itic.—Under the description of purulent conjunctivitis, we have
mentioned that sometimes the secretion is to a degree plastic
either forming shreds, or adhering to the mucous surface. No
special significance is to be attached to this fact, but cases some-
times arise where the plastic quality of the secretion is a distinctive
peculiarity, predominating over all other features of the case, and,
therefore they deserve special mention. Various grades of this
quality appear, and if we find that the layer of false membrane
can be easily rubbed from the conjunctiva, there may be no need
of regarding the case as differing essentially from a blennorrhoea
because it will soon exhibit the features of the latter by transfor-
mation of the plastic substance into ordinary purulent secretion.
On the other hand, we meet cases in which a plastic exudation is
the dominant fact. It will appear in greater or less degree and
while the moderate cases differ so widely from the most severe as
THE CONJUNCTIVA.
333
to warrant separate designations for each, we also meet interme-
diate degrees of severity which it is difficult to bring under an exact
classification. Some authors insist upon describing croupous and
diphtheritic conjunctivitis as separate diseases. In the former
(croupous) the eyelids alone are attacked, the exudation is moder-
ately adherent because superficial; by rather severe friction with
a rag it can be rubbed off and exposes a bleeding surface. The
deposit may be in patches or spread over the whole surface. The
general reaction is moderate, the lids not much swollen, the ocular
conjunctiva red and somewhat oedematous, the cornea seldom in-
volved either in opacity or ulceration. On the other hand, diph-
theritic conjunctivitis is described as a condition in which both the
palpebral and ocular conjunctiva is covered by plastic exudation,
which is dense and gray, penetrates deeply into the tissue, involves
the whole mucous membrane, and in the lids attacks so great a
depth of tissue as to make them hard, stiff, and brawny. The
material cannot be wiped away and the structures are deprived of
blood by the intrusion of the plastic substance. The cornea is very
liable to ulceration or necrosis. There will be fever and general
prostration. The first distinct portrayal of this condition was in
one of the earlier papers of Von Graefe: and it was limned with
a master-hand.
There is no doubt that under the above description of croupous
conjunctivitis many cases may be ranged with correctness and that
the disease is purely local. But if we attempt to be thus precise in
our designations we fall into error. I have treated a case in which
the plastic exudation was confined to the lids, but was not superficial,
it was incorporated with the deep texture and never could be wiped
away; and finally it disappeared by absorption or simple melting
away, as I have seen similar material behave when exuded upon
the tonsils and fauces in a fatal case of diphtheria. Again Nettle-
ship describes (St. Thomas' Hosp. Reports, Vol. XIV., 1886) two
cases of fatal diphtheria beginning in one instance on the conjunc-
tiva of one eye and spreading to the nose, throat, and opposite eye,
and in which both lids of the eye first affected, were lined with a
moderately adherent membrane, but the substance of the lids was
not infiltrated nor swollen; the ocular conjunctiva was infiltrated
and the cornea was represented by a thick, white, soft slough. The
fauces were covered with membrane. The other eye became af-
fected, but the hard, brawny, rigid, and bloodless condition said to
be typical of diphtheritic conjunctivitis was never present. Yet this
case, by its complications and fatal issue, was unquestionably one of
diphtheria and the ocular disease was diphtheritic conjunctivitis.
Mr. Nettleship also reports another case of " muco-purulent oph-
thalmia of the right eye" with membrane on the lower lid, both its
334
DISEASES OF THE EYE.
mucous and cutaneous surfaces, also on the free border of the upper
lid, but not on its palpebral surface. The other eye was normal.
The pillars of the fauces on both sides, the tonsils, uvula, and soft
palate were covered with a thick, gray, ashy membrane; the glands
about the jaw on both sides were enlarged. This was of course
diphtheria, and the eye considerably impaired; the other eye re-
mained free, and on the tenth day the child died.
It seems more logical to speak of such cases in a general way
as plastic or diphtheritic conjunctivitis, and not to expose ourselves,
by a rigid definition, to a false description of a certain number.
We may meet with all the types which have been referred to, and
we may choose to call the lighter forms croupous, but it would be
less erroneous to style them mild degrees of plastic conjunctivitis,
and we may call the graver forms intense degrees of plastic con-
junctivitis, or when constitutional symptoms appear we may cor-
rectly call the cases diphtheritic irrespective of the depth or extent
of the plastic infiltration of the conjunctiva.
It may be proper to quote from Flint's " Practice of Medicine,"
1881, p. 38, a passage which describes the pathological anatomy
and which will also serve to explain the cause of the various forms
of the disease we are discussing. He says:
" The terms croupous and diphtheritic are applied to fibrinous exudations
upon mucous membranes. Unlike the inflammations of serous membranes, the
ordinary inflammations of mucous membranes are not accompanied by a
fibrous exudation. The term catarrhal is sometimes applied to those simple
inflammations of mucous membranes characterized by an exudation of
serum, mucus, and some pus-cells. According to the careful investigations
of Weigert, fibrin is present in the inflammations of mucous membranes only
when the epithelial covering is partly or wholly destroyed. The epithelium
may be destroyed from various causes, among the most important of which
is coagulation necrosis. The necrosis may extend deeper than the epithelium
into the subjacent tissues. When only the epithelium is destroyed, the
fibrinous exudation lies upon the membrana propria of the mucous mem-
brane, from which it can readily be stripped off without loss of substance.
This form of exudation is called croupous. When the primary necrosis in-
volves the tissue-cells as well as the epithelium, the fibrinous exudation ex-
tends from the surface into the tissue of the mucous membrane, and cannot
be removed without loss of substance. This second form of exudation is de-
nominated diphtheritic. When the fibrinous exudation adheres closely to
the mucous membrane without really infiltrating it, it is called pseudo-
diphtheritic. It is to be observed that croupous and diphtheritic exudations
require destruction of the epithelium only in one place, and that they may
extend themselves over the surface of the surrounding intact epithelium.
The fibrin in croupous and diphtheritic inflammations is derived partly from
the blood, partly from metamorphosis of the epithelial and other cells, and
perhaps, partly from fibrinoid degeneration of the intercellular substance
(Neumann)."
The remark made in this quotation " that croupous and diph-
theritic exudations require destruction of the epithelium only in
THE CONJUNCTIVA.
335
one place and that they may extend themselves over the surface
of the surrounding intact epithelium " was emphatically exempli-
fied in a patient whom I saw with Dr. R. H. Derby; a young child
with a thick coating of plastic exudation over the upper lid as the
special feature of a severe inflammatory condition. It could be
easily pulled off as a whole, and displayed at one point a mass of
sprouting granulations as large as a pea where the mucous mem-
brane seemed to be perforated. Observation for successive days
proved this spot to be the focus whence the exudation extended.
When by cauterization these granulations were destroyed, the ten-
dency to diffuse plastic exudation speedily disappeared. The case
continued for several weeks.
It must be added that cases are recorded in which a film of
plastic material continued to form and be thrown off for many
months, not only upon the palpebral, but on the contiguous ocular
conjunctiva, while the reaction was moderate both in swelling and
other inflammatory features, and finally the eye recovered. In
other words, plastic conjunctivitis appears rarely as a chronic af-
fection. Cases which persisted as long as five months are given by
Nettleship, St. Thomas' Hospital Reports, Vol. X., 1880, and by
Critchett and Juler, Trans. Oph. Soc of United Kingdom, Vol. III.,
p. 1, 1883, with colored plate.
Sometimes one only, but usually both eyes are affected. While
usually spontaneous, it may occur after operations upon the eye,
as happened once in my experience in a boy ten years of age oper-
ated upon for strabismus; the dense gray plastic infiltration ex-
tended from the wounds over the ocular and then over the palpe-
bral surfaces, with the typical characteristics of stiff and bloodless
structures. It sometimes occurs after the exanthemata, measles, or
scarlet fever, and broncho-pneumonia; it may go up from the throat
and nose: chronic inflammatory conditions of the conjunctiva pre-
dispose to it. Epidemics may occur. At its outset it may be ac-
companied by severe swelling and great redness of the lids, almost
erysipelatous, but ordinarily the swelling is moderate and the dis-
charge slight.
In certain instances, which seldom occur, the lids are firm, stiff,
hard, difficult to evert because of the depth of the infiltration.
Their temperature is increased, and handling produces much pain;
it ma}r be impossible to evert them. The contrast between the
abundant plastic infiltration and the absence of secretion in these
severe cases is remarkable. The cornea is liable to opacity at an
early period, and may easily succumb either by ulceration or by
diffused infiltration. When cut into, the lid is gray, lardaceous,
and little disposed to bleed. The tissues are filled with micro-
organisms. Not infrequently diphtheritic patches are found in the
336
DISEASES OF THE EYE.
nostrils. The period of infiltration lasts from six to twelve days,
when the membrane begins to be dissolved and comes off or is ab-
sorbed; reddish streaks and patches appear'in the white deposit,
the lids become softer, the conjunctival tissue is more succu-
lent and velvety, showing a papillary outgrowth, and a discharge,
which gradually becomes purulent, sets in; after a time the case
takes on the aspect of an ordinary blennorrhcea. During the process
of healing of severe cases, an abundance of cicatricial tissue is de-
veloped, giving rise to atrophy and shrinking of the conjunctiva.
The prognosis is in these cases extremely grave, and life itself is
sometimes at issue.
It must be added to the above description that a membranous
exudation may form on the cutaneous surface and edges of the lids,
and the conjunctiva present simply a blennOrrhoeal condition. Even
sloughing of the skin has been observed (Nettleship) and there may
also be ulcerations and exudations or herpetic eruptions on the
cheeks about the nostrils and lips, and perhaps exudation in the
throat.
The health of the patient and his surroundings are of great im-
portance in contending with this malady.
Treatment.—For the milder cases cleanliness and not very cold
lotions are sufficient; boric-acid solution may be employed, and the
disease will readily yield within a few7 days. As the tendency to
production of plastic membranes abates, a weak nitrate of silver
solution, 2 or 5 grs. ad 3 i., may be employed once in twenty-four
hours. There are, however, exceptional cases, of which within a year
I have seen one, where the disease is more obstinate. The exudation,
though confined to the lids, penetrates the membrane, cannot be
wiped away, and persists for weeks; at the same time some ulcer-
ation may take place at the edge of the cornea. In the instance in
my mind, applications of cold with cleanliness and mild remedies
had no controlling influence. The best result was at length ob-
tained when a solution of corrosive sublimate, 1 to 2,000, was pen-
cilled over the everted lid several times a day, and a solution, 1 to
5,000, used as an external wash.
Antiseptic and germicidal substances are obviously indicated.
In selecting them and their degree of concentration we must be
guided by the intensity of the process and its stage. The very
grave cases are fortunately rare, and in them the dense infiltration
imperils the vitality of the tissues, threatening gangrene. If this
confronts us we may use hot instead of cold lotions, and resort to
antiseptics cautiously. A trial of corrosive sublimate, 1 to 1,000, may
be made when some secretion appears, and if not too greatly re-
sented may be repeated, carefully watching its effects. In fact
the treatment must be tentative and the case frequently inspected.
THE CONJUNCTIVA. 337
If swelling be great we will use cold, even iced lotions, and employ
mild antiseptics cautiously to remove secretion. The germicidal
use of corrosive sublimate, meaning in strength 1 to 500 or 1 to 2,000,
requires caution and judgment. Its antiseptic use 1 to 5,000 will be
persistent. One may employ as antiseptics aqua chlorinat., sol.
sodae chlorinat. (Labarraque), 1 to 5, sol. potass, permanganat 2r;.
Finely powdered iodoform, dusted into the eye, or mixed with vase-
line, 3 i. ad 3 i., is to be suggested.
If swelling be so great as to make canthotomy advisable, the
cut surfaces will be invaded by the exudation. Wolfring recom-
mended the ung. hydrarg. oxid. flav. with vaseline 3$ rubbed oh the
mucous surface once or twice daily with energy, but its value has
not been sustained, although approved by Schmidt-Rimpler, 1885.
When the plastic material begins to disappear and a red and
succulent surface emerges, the nitrate of silver will find place, but
never until this stage. Its utility is precisely the same as in cases
of purulent inflammation, and the rules for its employment will be
the same. As the disease abates, the cold lotions and the cleansing
will be less frequent. Leeches and depressing remedies are out of
place.
Constitutional treatment may not in many cases be neglected.
Foremost is the necessity of sustaining the nutritive power by
careful feeding, by iron in full doses, by quinine, and perhaps by
stimulants. Milk will be largely employed, and all the measures
called for by other complications, whether in the nostrils or the
throat, on the face or in the chest, will be resorted to. By v. Graefe
and by some of his followers mercurials have been given, but they
are not to be relied upon.
The disease may be very serious and the great danger is to the
cornea, while in certain types the conjunctiva undergoes atrophy,
sometimes causing broad adhesions or fraena at the retro-tarsal
folds, and may eventuate in entropium or trichiasis. The risk to life
has been referred to, but this is not often met with. The cornea
in some instances suddenly and totally breaks down with yellow
infiltration, while usually the damage comes by ulceration and per-
foration beginning at a particular spot. Graefe, out of 40, lost 9
eyes; Hirschfeld, out of 94, lost 34 eyes; Jacobson, out of 22, lost 5
eyes. The fatality in epidemics is apt to be most severe.
5. Granular Conjunctivitis—Trachoma.—Under this name is
grouped a variety of conditions apparently quite dissimilar, whose
chief characteristic is hypertrophy of tissue, and which has received
its name from the presence of certain granules or follicles which
under one form or another are considered essential to it. It may
or may not be attended by acute inflammation. The name tra-
338
DISEASES OF THE EYE.
choma was popularized in ophthalmology by Stellwag, who called
attention to the granules as marking the disease, and they are often
called trachoma granules, but have nothing in common with the
granulations of a healing wound. In many cases of granular con-
junctivitis they are not recognizable to the naked eye because im-
bedded in the tissue; in some cases they do not exist, in some cases
they are few and hard, in other cases they are most exuberant and
very soft. We find them as semi-transparent, seed-like bodies,
compared to grains of boiled sago or the spawn of frogs; they
may, in old cases, be dull red and in hard, flattened masses. They
often appear in a ridge along the orbital edge of the tarsus as a
semi-opaque infiltration, and their seat of election is the fornix of
the upper lid and not much less frequently of the lower lid. In
these localities they exhibit clefts and prominences like a cauli-
flower, owing to the laxity of the tissue.
Besides these granules we have more or less hypertrophy of tis-
sue, which may appear as fine or conspicuous papillary outgrowth;
the membrane is deep red, usually rough, sometimes is smooth,
and occasionally is enormously thickened. After long continuance
fibrillae of connective tissue appear, and in the end it supersedes all
other textures, bringing about atrophy and various degenerations.
Under certain phases there is secretion, either watery or mucoidal
or puriform—in most cases, and these the chronic ones, there is
little or no secretion.
While of necessity a source of discomfort, the chief misery and
mischief are through the lesions of the cornea. This becomes hazy,
vascularized, ulcerated, densely opaque, sclerosed, perhaps perfora-
ted and staphylomatous. The vascular haziness is called pannus
and may be in most various degrees of density, called p. tenuis
(thin), or p. crassus (fleshy). The upper portion of the cornea may
alone be affected, the whole may be implicated. The pannus may
occur during the period of hypertrophy; its worst forms are found
in the last sad stage of conjunctival atrophy.
The disease affects the upper lid more than the lower; very
rarely does it attack the ocular conjunctiva, save in the form of
follicular trachoma, and then by continuity from the fornix. It is
chronic in its duration, liable to intercurrent inflammations, affects
chiefly those in poverty or living in unhealthy conditions; is a pest
in asylums, orphanages, barracks, and poor-houses; is found among
children (seldom in infants) and adults of all ages; in later life is
seldom begun, but merely continued. It has general geographical
limitations, in the sense that in certain countries it is far more
severe and prevalent than in others. For example, in Ireland
Holland, Belgium, Turkey, and Egypt it is the curse of the common
people. In Russia it abounds, in middle Germany it prevails less
THE CONJUNCTIVA.
339
than in southern Germany and Austria. In the United States it
affects the dwellers in tenement-houses, and goes with unhygienic
conditions in large cities. It prevails in the Western prairies and
is found scattered widely over the country. A high altitude is un-
favorable to its production, as proven in Switzerland. See Chibret.1
The negrcr race are said to be exempt (Burnett), although mulat-
toes of every grade are its victims. It occurs among the Western
Indians. Most important is the favoring influence of the so-called
lymphatic (scrofulous) temperament. On this point sufficient stress
has not until lately been laid, although clinicians have always rec-
ognized the fact in practice. This accounts for its appearance oc-
casionally among the wealthy and well-cared-for, and it is not less
obstinate among them than their poorer fellow-creatures. See
True2 For its dispersion another cause must be assigned, viz., a
degree of contagiousness. On this point careful discrimination is
requisite. Those phases accompanied by secretion are undoubtedly
contagious, and in the degree of its copiousness. For example, the
so-called Egyptian ophthalmia, which is a mixed condition; the dis-
ease as found in public institutions, educational and eleemosynary;
the so-called acute trachoma, some cases of follicular trachoma—
all are attended by muco-purulent or other secretion, and in all a
great variety of microbes are to be found. The means of contagion
are in Egypt flies and other unsavory media (Howe), use of towels
and toilet articles in common, washing in the same water, and the
innumerable modes of conveying filth practised by filthy people.
The contagium may adhere to the walls and floors of the places
where the patients live (Cohn3), it being assumed that these places
are prone to be dirty and the inmates will always be more or
less crowded. Accidental conveyance to the eye of a cleanly and
healthy person by the secretion is of course possible. On the
other hand, the large proportion of chronic cases, of whatever
variety, do not communicate the disease to others, because the
quantity of secretion is a minimum. A single person in a cleanly
household does not spread the disease, and if there be several pa-
tients its extension may be prevented by strict care and attention
to hygiene.
It has been stated that numerous micro-organisms are found
in the secretion, and they are of many varieties, sometimes includ-
ing even the gonococcus, while the usual pus-producing organisms
and the streptococcus can always be detected. This fact explains
easily the rapid and insidious extension of the disease in suitable
localities and among susceptible persons, and does away with the
1 Annales d'Oculistique, t. cv., Jan. Fev., 1891, p. 22.
8 Annales d'Oculistique, t. cvi., Aout, 1801, p. 81.
s Lehrbuch der Hygiene des Auges, p. 139, 1891.
340
DISEASES OF THE EYE.
mystery which has seemed to conceal its mode of propagation, be-
cause the air as well as numerous recognizable agencies become
the ready means of transmission. Yet another supposed possibility
of contagion is to be mentioned, viz., a micro-organism peculiar to
and residing in the follicles of trachoma, a coccus first described
by Sattler1 in 1882, and by Michel,21885. It is a diplococcus smaller
than the gonococcus and appears in groups like sarcinae. Some
observers have confirmed this discovery, while not a few deny that
its culture can give rise to trachoma. Contrary opinions yet pre-
vail, but the belief in the important influence of micro-organisms
is universal, because antiseptic treatment has become so efficient
and widespread.
We have now given an account of the disease in its general
features, and we must set forth its subdivisions. We designate
them from a clinical standpoint, and in so doing point out the con-
spicuous features of each variety, admitting that sharp divisions
are impossible because one type shades into another, and this is
true not only in external appearance, but in course of development,
and in pathological anatomy. We may speak of: 1, Papillary
trachoma; 2, Acute trachoma; 3, Follicular conjunctivitis, fol-
licular trachoma, foil iculo sis; 4, Chronic trachoma; 5, Sequelce.
(1) Papillary trachoma or chronic blenorrhcea presents itself
under two phases. The first is a condition of velvety hypertrophy
of the palpebral conjunctiva with moderate injection and no se-
cretion, and not thick enough to conceal entirely the normal yellow
tint of the tarsus. It is the outcome of mild but chronic conjuncti-
vitis; no granules are ever to be found; the papillae are very fine
and short. The second phase exhibits greater thickening, resem-
bling plush rather than closely shaven velvet—the papillae com-
pletely conceal the color of the tarsus, they stand thick and high,
may present clefts and prominences; the whole tarsal surface and
the fornices are involved; the semi-lunar fold and the ocular con-
junctiva are injected and more or less swollen. There is constant,
perhaps copious secretion with photophobia, erosions of the corneal
epithelium, vascularity at the limbus. The condition follows
acute, usually purulent conjunctivitis, and has been strenuously
separated from " true trachoma " because the fibrous degeneration
rarely occurs. Nevertheless the follicles are to be found imbedded
in the tissue, although not visible during the distinctive papillary
period and perhaps never seen by the physician. The cornea may
be superficially hazy, but eventually clears up if proper treatment
is used.
(2) Acute trachoma, a condition in which there is great swelling
1 Archives of Ophth., xv., 452, 1886.
2 Bericht der Congress Heidelberg, 1882.
THE CONJUNCTIVA.
341
of the whole thickness of the lids, great hypertrophy of the con-
junctiva, which comes on rapidly, accompanied by severe pain,
heat, injection of the ocular conjunctiva and discharge of an almost
purely watery quality. E version of the lids is difficult, and the palpe-
bral conjunctiva has a dotted look, is intensely red and shiny, and
does not exhibit special papillary prominences or trachoma gran-
ules. After the subsidence of the acute symptoms, viz., in from
one to three weeks, trachoma granules and moderate papillary
hypertrophy will be seen. This condition is not very liable to cica-
tricial degeneration, and the cornea usually escapes. It is some-
times evidently dependent upon an acute exacerbation of hyper-
trophic nasal catarrh. It may begin without previous conscious
trouble, it may come as an exacerbation of chronic trachoma, es-
pecially in lymphatic subjects. It passes into the chronic type as
the acute symptoms decline.
(3) Follicular Trachoma—Folliculosis.—The subject is usually
young, apt to be of lymphatic temperament. For some time has
been unable to use eyes with comfort, either one or both. The lids
droop, perhaps seem swollen; no redness of ocular conjunctiva, but
it is moist. There is moderate sticky secretion. The patient shuns
the light, is much irritated by wind and dust, but has no pain;
cornea clear. The lids are easily everted, and if pains be taken to
unfold the fornices (see p. 310) a most striking picture is sometimes
displayed. We find the tarsal surfaces dotted with small seedlike
bodies, semi-transparent, perhaps as large as hemp-seeds; they lie
in and upon the pale pinkish-gray membrane, and as we pass to the
folds of reflection they increase greatly, perhaps enormously in
number, crowding forward in great, succulent, gelatinous-looking
masses. The conjunctiva at the culs-de-sac is pale, yellowish-pink,
but thrown into transverse ridges. The conditions vary in inten-
sity, but it is not rare to meet a case of prodigious redundancy of
follicular masses of which no serious complaint had been made and
accompanied by no important symptoms. The condition has de-
veloped slowly, is slow to disappear except by surgical treatment.
It may pass over into the usual phase of chronic trachoma with
fibrous tissue formation (Raehlmann,1 Reich2), but this issue is not
frequent; nevertheless, the attested fact proves the cousinship of
the disease. The cornea is not affected unless the case be neglected.
It yields readily to proper treatment. By some writers this con-
dition is regarded as standing apart from trachoma. The evidence
of recent investigations, clinical and pathological, is in favor of its
kinship, although it is far less serious than chronic trachoma.
1 Graefe's Archiv, xxix., 2, 73, 1882; also, Wiener med. Wochenschrift, 41,
1890.
2 Nagel's Jahresbericht, p. 283, 1S89.
342 DISEASES OF THE EYE.
(4) Chronic or Mixed Trachoma.—We meet with extremely
different degrees of this condition—the characteristic of all being
the presence in varying quantities at some period of their duration
of the trachoma follicles, or, as it is also called, of lymphoid infil-
tration. We classify the different degrees into the following:
(a) Isolated deposits of granules with little or no thickening of
the conjunctiva. These are found almost exclusively in the folds of
reflection; they may be single or in groups. They cause slight or
sometimes no irritation and no secretion. Frequently no history of
either previous or present inflammation can be elicited. Yet it
may have occurred.
(b) Lymphoid infiltration disseminated in granules, or forming
a ridge at the orbital margin of the tarsus, and sometimes pre-
senting also thick deposits in the culs-de-sac and accompanied by
thickening and more or less papillary hypertrophy of the con-
junctiva. This condition is most pronounced in the upper lid, while
the lower may not escape. The ocular conjunctiva is very rarely
implicated. There is little secretion, but there may be lachryma-
tion; the amount depends on the activity of the attendant inflam-
matory action—it may be almost wanting. There will be photo-
phobia, and to an intense degree, if the cornea have become
vascular. Serious implication of the cornea is the tendency of the
lesion. Occasionally lymphoid infiltration occurs in its substance.
The diversified appearances of the palpebral conjunctiva cannot
be fully described. One must imagine a very great variety of pos-
sible pictures, both in quantity and distribution of the infiltration.
After a certain duration, with or without abatement of thickening
grayish lines of fibrous tissue make their appearance; they form a
mesh, and patches, and sometimes hard masses of lymphoid sub-
stance are left projecting above intervening spaces of dense and
shrunken conjunctiva. The shrinking after development of fibrous
tissue sometimes affects the membrane uniformly and leaves a
glazed, red surface or a patchy piebald mixture of red and yellow-
ish spots, with a concave tarsus which has shrivelled in breadth
and length, sometimes reduced to a solid ridge. Meanwhile the
folds of reflection have been shortened or possibly obliterated and
the conjunctiva may spring out into vertical ridges when the lid
is everted. The degenerative process in the final stages reduces
the conjunctiva to a dry cuticular membrane, xerosis. Reference
has already been made to the possible damage which may be en-
tailed upon the cornea, and this feature commands the constant
vigilance of the physician. Without dwelling on the desperately
bad conditions which are possible, it must be emphasized that hazi-
ness of an apparently unimportant degree will sadly reduce vision
so that V=-iV may be considered fortunate, while to the naked eye
THE CONJUNCTIVA.
343
the corneal tissue is pretty clear. Add to this the depreciation due
to change of curve under the softening tendencies of the process
and the pressure of the lids, and we find irregularities which defy
correction by any glasses. I have recently seen a patient, observed
twelve years ago, who requires cylinders — 20.D at oblique axes,
and gains vision of 0.2 in one eye and 0.05 in the other. She is now
forty-five and dates the lesion to her childhood. She is on the whole
fortunate.
(c) Still another phase of chronic trachoma is that in which
enormous hypertrophy of the palpebral conjunctiva takes place,
increasing to a uniform thickness of 5 to 7 mm., as I have often
witnessed. There is marked ptosis; eversion is very difficult, for
the tissue is hard and brawny; the conjunctiva when exposed is
smooth and glazed and very red; there are no granules visible, or
they are deeply set and scarcely discernible. I have known such
cases looked upon as amyloid or tuberculous, or syphilitic. In fact,
a condition called hyaline degeneration of the conjunctiva has been
described by Raehlmann and by Kansocki and Vossius.1 It cor-
responds very closely to the sketch above given; the pathologi-
cal examination does not exclude the possibility of its origin in
trachoma.
The duration of cases of chronic trachoma may be months and
years, perhaps a score of them. Much depends on the attention be-
stowed and the wisdom of this attention. Too often patients lose
heart and cease trying to get well, or their exigencies compel them
to turn to work as soon as matters begin to mend. Hence with
increasing trachoma they are liable to relapses and continue to be
the patrons of public clinics for long years or decades.
Results and Sequelos.—Under this title is included what we or-
dinarily mean by prognosis and considerable more. It has already
been stated that the milder types may under suitable handling be
fully controlled without injury to sight and with no important
changes in the conjunctiva. The time required will vary from one
to several months. With the softer and more papillomatous vari-
eties, provided lymphoid infiltration be not great, and including
some cases of true trachoma, the same good result may be achieved,
only after a longer fight. I may call attention to a condition which
I have never seen mentioned, presented at the very late stage of
the cases now considered when most of the hypertrophy has been
removed. In the pockets and clefts of the tissue, and to some de-
gree imbedded in it, I have sometimes seen white specks of soft
molecular substance, suggesting calcareous matter but probably
mere detritus of degenerated cells, which keep up troublesome
irritation. Under cocaine they may be picked out with a cataract
1 Bericht Ophth. Gesell, Heidelberg, 1889, pp. 108, 114.
344
DISEASES OF THE EYE.
needle, and after two or three sittings the irritation has been en-
tirely removed. The result of the disease, too often aggravated by
heroic treatment, is in severe cases the development in the conjunc-
tiva of connective tissue to excessive amount, altering its texture
and impairing its secreting functions. The more moderate cases
exhibit smooth red patches or lines, or a satiny surface; a higher
grade is indicated by fraena and ridges at the folds of reflection; be-
yond this stadium the fornix becomes shallow and loses its loose
texture in a smooth scar surface, and the last stage is xerosis with
posterior symblepharon, and the ocular conjunctiva is reduced to
the same cuticular condition, while the cornea has become dry and
densely opaque.
The connective-tissue growth and resultant contraction enters
also into the tarsus, thickening it, bending it inward, and causing
the free border to press against the cornea, and on eversion exhibits
a furrow along its whole length about 4 or 5 mm. from its edge.
The deformity in extreme cases reduces the tarsus to a thick cord
which shows a ridge on the exterior surface of the lid and at the
same time causes shortening of the palpebral fissure and partial
ptosis. Coincident with these changes the numerous glands and
follicles of the lid border and tarsus become obstructed, atrophied,
obliterated, and in the hair follicles the effect is loss of cilia, trichia-
sis, and entropium.
It is superfluous to dwell on the mischief wrought upon the
cornea, because this is obvious, and it has been already more than
once referred to. To prevent it, to mitigate it, to cure it is the
great end of our treatment, and our success in this particular is the
gauge of the wisdom of our method and the value of our remedies.
To shorten the duration of the malady is a great desideratum, but
to protect or restore or to save as much sight as possible is of
course infinitely more important.
Pathological Anatomy.—Much has been written on this topic,
and the chief authorities of the last ten years are Raehlmann, Reich,
Sattler, Michel, Noiszewski,1 Mutermilch,2 and Reid.3 For more
authorities consult Cohn, '•' Hygiene des Auges," p. 780. As to the
trachoma coccus, about which something has already been said
(see p. 340), the last word is to the effect that the described organ-
ism does not have the specific qualities ascribed to it, but that the
generative cause of trachoma is a vegetable fungus called micro-
sporon trachomatosum s. Jagium, which bears very close analogy
to the microsporon furfur, which is the cause of Pityriasis versi-
color, and can be seen by a low magnifying power (Noiszewski, 1. c).
1 Centralblatt f. Augenheil, March, 1891.
sAnnal. d'Oculistique, Oct., 1891, and May, 1892.
8 Trans. Oph. Soc. United King., x., 1890, p. 57, with plates.
THE CONJUNCTIVA.
345
The author declares that he has produced trachoma by pure cul-
tures of this fungus in calves and rabbits. Dr. German has traced
three Cases of acute trachoma to the poisonous effect of the soil
which entered the eyes. The patients were farm laborers and with
dirty hands and clothing easily infected themselves (St. Peters-
burg med. Wochensch., 1890, 29). Evidently the end of this inves-
tigation is not yet.
Almost all authorities have bent their attention upon the
trachoma follicles and their various appearances as the typical ele-
ments of the disease, and have classified cases accordingly. What
gives birth to them has not been clearly set forth, and little interest
attaches to the shades of difference which follicles from follicular
trachoma or from chronic trachoma exhibit. They consist of lym-
phoid cells, some within, dead and shrivelled, others without, liv-
ing and active, enveloped in a capsule of adenoid tissue, and the
mass penetrated by connective tissue, fibres and blood-vessels. The
Fig. 132.
interior of the glomerule undergoes further degeneration by either
hardening or liquefaction, and the capsule is thick or thin accord-
ing to age and natural abundance of adenoid tissue. There are
also numerous papillae, covered with a thick layer of epithelium in
various* stages of proliferation. As they are crowded together, are
ulcerated, and adhere more or less perfectly, pockets and clefts and
tubules lined by epithelium are produced, and sometimes cystoid
cavities are developed. The epithelium easily breaks down and by
its metamorphosis gives rise to goblet cells which undergo various
regressive changes, and the epithelium may assume a stellate form
after its nuclei have become vacuolated. Combine with this the
development of connective tissue and the presence of a large
amount of adenoid tissue, and the picture as usually given is
complete.
To the above nothing would be added but for the fact that a
paper by Mutermilch (1. c, 1891-92) describes an investigation be-
ginning with the earliest phases of conjunctivitis and tracing the
346
DISEASES OF THE EYE.
process step by step to the various manifestations of trachoma, and
shows how follicles, papillae, epithelial degenerations, fibrous tissue,
etc., make their appearance: it shows up the whole panorama. It
is a study in pathogenesis as well as in pathology. It were too
long to attempt to reproduce this most masterly description even
greatly condensed. This may be stated: he traces all the morbid
changes to the epithelium and its alterations. Of it he gives a
careful description with its superficial mucous corpuscles and its
deep layers which send fine processes into the subjacent adenoid
tissue, which is a cellular tissue infiltrated by lymphatic cells. De-
scribing what transpires with ordinary chronic and acute inflam-
mation, he finds the follicles an accidental production dependent
on the amount of adenoid substance and the quantity of lymphoid
infiltration. He shows how the papillae appear, and the various
phases with which we are familiar. But the dominating and novel
feature of his exposition is the importance given to the role of the
epithelium. He makes this new and striking assertion, that the
success of treatment lies in destroying the hyaline lacquer of fused
and obsolete cells and in bringing to the surface the living epithe-
lium which has been buried beneath effete inflammatory cells
and material, and therefore our remedies must not be too harsh.
Moreover that pannus appears when the epithelium has been de-
stroyed, and corresponds to the degree of its destruction. That
the roughness of the palpebral conjunctiva is not its cause, but the
absence of living epithelium. Moreover that disappearance of
pannus attends the regeneration of healthy epithelium. As to
trachoma follicles, he traces in them the effect of a low vitality of
constitution with imperfect oxygenation as the predisposing cause
why lymphoid cells exuded in inflammatory action cannot main-
tain their activity, are soon devitalized and incapable of amoeboid
movements, and become grouped into globules by the mechanical
influence of the loose adenoid cellular tissue beneath the epithelium.
Such enfeebled cells undergo regressive changes and absorption
and may quite disappear, but if in large masses they form the be-
ginnings of the future follicles. It is inexpedient to go extensively
into this fascinating exposition, but the writer finds no essential
difference between the follicles of follicular trachoma and those of
the more chronic malady. He recognizes that when formed and
solidified they become foreign bodies liable to provoke irritation
and while themselves the product of an arrested or imperfectly
resolved inflammation they may be the exciting cause of persistent
or renewed inflammation. He also makes clear how the inflamma-
tory products of acute trachoma and of the occasional enormous
hypertrophic forms of hyaline material are not anomalous but are
in accord with the usual phenomena of inflammation. He makes
THE CONJUNCTIVA. 34;
it reasonably clear that granular trachoma is no strange exception
in pathology, but simply a special appearance due to special local
conditions of tissue and interesting because easily observed.
Prophylaxis.—A few words are needful as to the measures to
protect the healthy from infection, and to put the diseased in the
best conditions for recovery. We face this requirement in families,
in orphanages, barracks, and places where many persons congre-
gate under unfavorable hygienic conditions. It will be remem-
bered that the conspicuously dangerous element is the secretion
—that this contains numerous pernicious germs, that the air may
be foul with them and the walls and clothing may be tainted by
them. Hence the rigor of precautionary measures will be in
direct ratio to the number of affected persons. In crowded insti-
tutions the inmates may have to be scattered, or put into tents,
or assigned to other and larger quarters. In schools the piti-
ful meagreness of the toilet economy, the few and numerously
used wash-basins, the vile roller-towel, and other easily imagined
vehicles of filth like rags and handkerchiefs and fingers, et id genus
omne, must be thoroughly and peremptorily destroyed, abolished, or
purified. Cleanliness, proper air space, sleeping-rooms healthful and
well ventilated, good nutrition, out-door life, plenty of water, rea-
sonable separation or even isolation are the prime requisites. Next
to these come antiseptic lotions, of boric acid, corrosive sublimate,
and permanganate of potash (see Cohn, 1. c, p. 139).
Treatment.—For the sake of convenience we shall depart from
the order observed in describing trachoma and begin with the
acute conditions. We consider attacks of primary acute trachoma
and the acute aggravations which may take place during chronic
trachoma.
If there be acute pain and photophobia, more or less swelling,
the ocular conjunctiva injected and possibly vascularity with or
without ulceration of the cornea, we must employ atropia, cocaine,
and cold lotions vigorously. The pupil must be kept dilated; ano-
dynes may be required. It is always best to use an antiseptic,
and while boric acid has value, corrosive sublimate, 1 to 2,000 to
1 to 5,000, is most worthy of favor (the old lotio flava of sol.
corrosive sublimate and lime-water is nowadays fully justified).
Scarifications of the conjunctiva may be serviceable. Frequently
the aggravated situation depends on the bad (strumous) condition
of the patient. The best hygienic surroundings and confinement to
bed are necessary. The scratching sensations may be mitigated by
free use of vaseline under the lids. If the secretions are copious
and watery, medicated applications avail little; so soon as a muco-
purulent material appears, sol. nit. arg., gr. v. vel. x. ad § i., may
be tried, but with caution. It is always imprudent to employ irri-
348
DISEASES OF THE EYE.
tating proceedings during an acute inflammatory stage. It may
require from three days to three weeks for this period to pass. It
is imperative to keep the pupil dilated, and a 4^ solution of cocaine
muriate has useful effects in relieving pain, causing the vessels to
shrink, and in reducing tension. Hippel and Sattler recommend
sol. corrosive sublimate, 1 to 1,000, applied with a brush to the lid,
as soon as any granular prominences appear. It is to be tried
with caution, and though painful does prove remarkably useful.
If an acute attack supervene during the chronic period, we must
abandon the usual remedies of nitrate of silver and sulphate of
copper, and endeavor to find out the cause of the outbreak, which
may be found in a severe nasal catarrh or in a depreciated state of
health. If the former exist, sol. nit. arg., gr. xx. or xl. ad § i., or in
less acute conditions chromic acid inside the nares will help us. For
the low health, food, change of scene, tonics, cod-liver oil, iron, fresh
air, regulated baths will be our reliance. As to the local treat-
ment, what has been said above need not be repeated.
Papillary trachoma requires moderation in treatment; touch-
ing the lids lightly once a day with a crystal of sulphate of
copper or alum, with solutions of tannin and glycerin (gr. x.-xxx.
ad 3 i.), or with a solution of nitrate of silver (gr. v. ad § L). The
patient, if unable to see a physician, may himself make use of an
ointment consisting of three grains of sulphate of copper to the
ounce of vaseline. As the parts become accustomed to one rem-
edy, another must be substituted. Patients may often be pro-
vided with astringent washes of alum, of boric acid with sulphate
of zinc, and, if no corneal complication exists, of sugar of lead.
Powdered boric acid may be dusted on the everted lid.
It must be added that for even this class of cases one may make
trial of the solutions of corrosive sublimate, 1 to 1,000plus or minus,
believing that this potent remedy will do for them what it has so
signally done for other cases to be presently related.
Follicular Trachoma.—The annoying secretion may be washed
away by lotions of boric acid 3$, or of acetate of lead 1%. But there
is no reason to lose time on such merely palliative methods. The
effective treatment is to destroy and squeeze out the follicles
whether they are in isolated masses or in such exuberance as to re-
semble the everted rectum of the horse during defecation. Since this
method was proposed, I have practised it with much success and to
the exclusion of every other proceeding. The patient lies on his
back. I use two forceps (see Fig. 133), one in each hand, and hav-
ing thoroughly cocainized the surface by a pledget of cotton soaked
in 10# solution, evert the lid and hold one end of the tarsus with
one forceps and applying the other, pull against the first, thus
squeezing and stripping out the granules (see Fig. 134). The for-
THE CONJUNCTIVA. 349
ceps enable one to pick up the deepest parts of the membrane,
and the line of demarcation between ocular and orbital conjunctiva
is usually well marked. I have seen the follicles on the ocular
membrane. Bleeding is rather free and must be wiped away with
cotton and by drenching with sol. sublimate, 1 to 3,000. All the
morbid material can be removed at one sitting.
Fig. 133.
Some and occasionally smart reaction occurs, requiring cold
lotions. This subsides in a few days. For a certain period appli-
cations of nitrate of silver, gr. ij.-v. ad 3 i., will be made once a
day or every second day and simple lotions employed. Some cases
are perfectly cured in ten days; others require a month, according
to the severity of the case. I have occasionally seen reproduction
of the follicles requiring repetition of the operation.
Efficient and truly curative treatment consists in excision of
Fig. 134.
the redundant folds at the cul-de-sac. For many years I have, in
selected cases, done this, and fully recognized the importance of
not doing it to excess, and of careful judgment in the selection of
cases. I strongly deprecate resort to such a method in ordinary
cases, but for some it is adapted. Galezowski has followed this
350
DISEASES OF THE EYE.
practice. It is only when the folds are very loose that excision is
proper. It may be done with forceps and scissors, leaving sufficient
tissue not to restrain free movements of the conjunctiva after
cicatrization shall have occurred. It is also important not to go
so deep as to damage the thin expanding tendon of the levator.
The effectiveness of the squeezing process, of which the method is
indicated in the sketch (Fig. 134), renders excision unnecessary. I
may add that the forceps depicted are more workable and will give
access to the semi-lunar fold and to the recesses at the angles
much better than either ring forceps or a roller forceps. One must
always employ two and often give an anaesthetic, because the
proceeding is severely painful.
Chronic Trachoma.—We have to consider first the simple con-
dition where isolated granules or groups of them lie imbedded
in an apparently normal membrane, or we may have the case of
an old trachoma with a few hard and red deposits lying like
boulders on a devastated plain where the fury of a fire has been
spent. To both these classes of cases similar treatment is suitable.
In the former instance there may be no history of previous trouble,
in the latter there has been a long-standing lesion of which the ex-
isting status is the remnant. In this the slight irritation will be
soothed by lotions of boric acid 3$, or of sublimate, 1 to 10,000, or po-
tass chlorat. 2$. But the effective remedy is to pick out with a needle
or squeeze out with cilia forceps the imbedded granules. If this
cannot be effectively done, the forceps previously described, or the
ring forceps of Prince, or a roller forceps of Knapp may be em-
ployed. When the expulsion is accomplished, sol. nit. arg. gr. ij.-iij.
ad 3 i. may be used every second or third day in addition to the
lotions. For the second class of cases more rigorous means are
needful, and the best success is by burning the separate masses
with the galvano-cautery, or the fine point of a thermo-cautery
(Paquelin), or by a hot needle. With cocaine several may be de-
stroyed at once and all at two or three sittings. After-treatment
will be indicated by the conditions—perhaps light touching with
sulph. copper or alum stick, or solutions of sublimate, 1 to 3,000, as
may be suitable.
Before discussing local treatment further, it must be insisted
on that severe cases, i.e., those with considerable pannus or thick-
ening of the conjunctiva, with hyperaemia of the bulbar membrane,
with acute photophobia and lachrymation, cannot be successfully
dealt with unless placed in good hygienic conditions. They need
protection from wind, dust, smoke, and bad air. They may have
to go to hospitals, but the utmost pains must then be taken to
secure free ventilation, good food, and absolute cleanliness in hands
and person, clothing, utensils, habits, and surroundings. When
THE CONJUNCTIVA.
351
such conditions are fulfilled, and they are difficult to fully realize
both in hospitals and in the houses of the poor, the battle is half
won. Frequently a patient must stay in bed a good part of the
day to permit the continuous fomentations; but sufficient exercise
must be given to secure good digestion and assimilation. Some-
times with absolutely perfect hygienic conditions the disease as-
sumes an acute type, and then we have to contend with a feeble
or strumous constitution. It is very rare for trachoma to develop
acute and obstinate forms among healthy persons in healthy con-
ditions.
Next we discuss the cases of chronic or mixed trachoma. The
hitherto established treatment consists in sol. nit. arg., gr. v.-x. ad
3 i., every second or third day to the everted lids, if possible reach-
ing the retro-tarsal folds, or the more painful sulphate of copper in
crystal, or for mild cases the daily use of a crystal of alum. By
some the yellow oxide of mercury ointment, gr. v. ad 3 i., or pow-
dered boric acid is rubbed on the everted lid with the finger: tan-
nin and slycerine, gr. xx. ad 3 i. or stronger, is employed during
the late stage when cicatrization and patches of hyperaemia ap-
pear. As a lotion, to be used several times daily, nothing equals
sol. corrosive sublimate, 1 to 3.000, pro re nata. Under the plan thus
sketched cases get well after many months, with liabilities to acute
exacerbation and prolongation of the disease for years. Because
the poor are the usual victims, their necessities compel them to omit
treatment as soon as they can return to work. Soon they present
themselves again, and the process must be begun de novo.
In view of the obstinacy of the malady, and the conviction that
its malign character is found in the presence of micro-organisms
which provoke cell growths and other changes, and guided by the
beneficent effect of corrosive sublimate in collyrium, a vigorous and
effective employment of this remedy has of late years been widely
adopted. In strong solution, 1 to 500, it is painful and caustic. This
has been used in France and Italy after cocaine anaesthesia, once
and even three times daily to the everted lid, while a solution, 1 to
120 (Arnaut's), has been twice a week employed. Von Hippel, whose
judgment and experience are worthy of respect, reports good re-
sults in 300 cases, by vigorously rubbing the surface with sublimate
sol., 1 to 2,000, once daily by a mop of cotton. The severity of the
friction is proportioned to the density of the tissue—if soft and much
swollen it will be light, if hard and the follicles firm it will be ener-
getic. The endeavor is to rub out the contents of the follicles and
the lymphoid infiltration. In some cases of diffused thickening and
infiltration this is impracticable, but even in them the treatment is
greatly effective. Cocaine is needful, but on repetition the treat-
ment is less painful than at first. With hyperaemic and soft tissues
352
DISEASES OF THE EYE.
the reaction may be sharp and require cold lotions for several hours.
On the following day there will be a thin layer of gray exudation,
which is easily removed, and the proceeding may be renewed every
day during the first week. In old cases with beginning atrophy
exudation does not appear. Pannus and slight erosions of the
cornea respond favorably, unless the conditions are severe or there
be co-existing iritis. Heisrath, commenting on this treatment, re-
ported good results, but stated that he had seen instances where
the healthy cornea became ulcerated and infiltrated (Berlin. Klin.
Wochensch., 1891, No. 5). When the follicles disappear treatment
of the chronic catarrh which remains " requires lotions of acetate
of lead, 1 i, and discontinuance of the rubbing." Some caution must
be given as to the use of lead lotions at any period of such cases.
Alum or weaker sublimate solution or boric acid will be safer, and
sol. nit. arg., 1 i, or light use of copper will be proper. Von Hippel
does not make extraordinary claims for this treatment, but holds
it superior to our old methods, and in this he is supported by
others. To his statements1 so much space has been given because
his experience leads up to another plan which embodies the same
ideas and which has been practised and witnessed by myself ex-
tensively during the past year.
The fundamental ideas are to get rid of the follicles and lym-
phoid infiltration, to apply sublimate solution in effective but not
destructive strength, to avoid such violence either mechanical or
medicinal as will provoke development of fibrous tissue; when
the follicles have disappeared to treat the case as one of chronic
conjunctivitis. Severe reaction, if present in the cornea, must first
be made to subside and must not be excited by treatment. Es-
pecially must iritis be respected and brought under control by
atropia, and all irritants until then be avoided.
The removal of the follicles is best done by the two pairs of for-
ceps referred to on p. 348. I find them superior in most cases to
roller forceps. But if the tissue be somewhat firm and the lym-
phoid infiltration cannot be sufficiently expressed, a more heroic
method is practised, which was first instituted in Abadies clinique
in Paris. An anaesthetic is given; the lid is rolled completely out-
ward by a small pair of dressing forceps. (A special forceps has
been contrived by Dr. Gibson which is of decided advantage in not
cutting the edge of the lid and in securing full control—Fig. 135).
The fornix is fully exposed and the membrane lightly incised by a
three-bladed scarificator in parallel lines about 2 mm. apart. With
a rather soft tooth-brush the corrosive sublimate, 1 to 500, is rubbed
into the lid and the bleeding checked by wads of cotton dipped in
the same solution. The epithelium is of course considerably de-
1 Bericht Ophth. Gesell., Heidelberg, 1892, p. 91.
THE CONJUNCTIVA.
353
stroyed and wiped away, but one must not deal harshly with the
tissue. The depth of the scarifications, their number, and the vigor
of the brushing must be in proportion to the induration and thick-
ness of the structures. The eyes will be covered with cotton soaked
in the same solution and bandaged firmly for a few hours to pre-
vent undue swelling. In a markedly lymphatic patient reaction
will be severe. There is always considerable secretion the next
day and the surface will be covered with a gray pellicle. Some
swelling will occur and cold lotions be necessary. Sublimate
solution, 1 to 500, will be applied daily to the everted lids by a little
mop, taking care to reach the fornix and prevent formation of ad-
hesions. If severe reaction contra-indicate the sublimate, the
manipulation will not be omitted. A patient will remain in hos-
pital about a week, less or more.
As already intimated, the cases suitable for the above proceed-
ing (" grattage ") are those in whom squeezing by forceps cannot
sufficiently remove the lymphoid infiltration because it is buried
amid great general thickening, which also prevents the effective
contact of the sublimate with the tissue. Experience has shown
Fig. 135.
that while a rapid removal of the granular thickening is effected
and the hazy cornea clears up, there is a tendency to production of
connective tissue which may easily induce unpleasant deformity of
the lids, causing bridles, ridges, and degrees of symblepharon. In
numerous instances it has been found that the superior fornix, six
months after a judiciously mild grattage, has been obliterated.
This condition may not be important, but one must not let zeal for
a rapid cure run into rashness which may occasion deformity.
Such cautions seem needful lest enthusiasm in behalf of a valuable
method of treatment overstep the limits of prudence. In public
institutions where children are assembled there is especial fitness
in the sublimate treatment, and it soon establishes a practically
aseptic condition among the patients which is the great desidera-
tum. It is also to be remembered that steady treatment must be
continued by usual remedies for weeks after the first favorable im-
pression by squeezing or sublimate, or both, has been made.
Cases of less duration and severity, and especially such as con-
sist largely of papillary hypertrophy, do well enough under the
nitrate of silver gr. v.-x. ad 3 i., where there is decided secretion,
23
354
DISEASES OF THE EYE.
or sulphate of copper where there is little of it, but the present
tendency is to use sublimate solutions in all cases for which me-
chanical means are not indicated. My most recent experience
favors solutions of sublimate 1 to 600 or 1 to 1,200 rubbed upon the
lid with a small cotton mop daily or once in two or three days, in
moderate cases, as the most satisfactory proceeding. " Grattage "
will be reserved for the more obstinate. In them a canthotomy is.
often requisite to sufficiently expose the field of operation. A repe-
tition of squeezing or sometimes of grattage may be necessary.
In former days cantholysis was often employed to help alleviate
the corneal lesion; we now resort to it only when deformity of the
lids, " phimosis," indicates its necessity. It will be referred to later.
I have in a few cases of extreme and general thickening of the
conjunctiva used the Paquelin thermo-cautery once in ten days or
two weeks and with considerable vigor. It has served me well, and
the subsequent fibroid degeneration was not excessive, although it
was considerable. The cases were exceptionally severe, and the
remarkable feature was the moderate reaction after extensive
burning.
As the thickening of the conjunctiva subsides, the corneal trou-
ble will, pari passu, abate; when, however, the cornea has become
very opaque and vascular, the vessels thick, the epithelium dense
and the surface insensitive, the sulphate of copper may be applied
directly to it, and with advantage. Excision of the conjunctiva for
the breadth of one-fourth of an inch around the entire circumfer-
ence of the cornea has been employed under the name of peritomy,
and has been of some service, but it is far less resorted to now than
in former days. Curetting the corneal surface by a small knife
has been done by Dr. Gruening, and with improvement and little
reaction (see Trans. Am. Oph. Soc, 1889).
In these extreme cases, and also in cases less severe, improve-
ment of vision has been sought for, especially in Belgium, by resort-
ing to inoculation with blennorrhoeal pus. In doing this an acute
purulent conjunctivitis is set up which is left to run its course with
but little interference, occupying from four to six weeks, and in
some cases valuable improvement in the cornea has been obtained;
there is, however, always risk of the destruction of the cornea and
of sight. This proceeding is at the present time little employed.
Within a few years an infusion of the jequirity bean, a Brazilian
plant, abrus precatorius, has been extensively employed in treating
not only the advanced and extreme cases of granular lids, but all
forms of the disease. Brought to notice by Wecker, of Paris, in
1882, it has had large trial, and experience up to the present time
seems to show that its chief value is during the later stages and
when the cornea has become seriously vascular. The bean is to be
THE CONJUNCTIVA.
355
powdered and macerated for three hours in water of the ordinary
temperature in the concentration of about 3$ (Wecker).1 The in-
fusion when old is not as effective as when recent. With a brush it
is applied two or three times to the lids; within twenty-four hours
reaction should appear; if it do not, the application may be re-
peated. The mucous surface becomes covered with a grayish
membrane which adheres for days. The lids swell, copious secretion
occurs and the cornea is apt to become very hazy. The patients
suffer pain, and iced water is to be freely employed. The inflamma-
tion will run for two weeks or more, and in favorable cases not only
does the cornea clear up, but the conjunctiva recovers a much more
healthy appearance and old cicatrices become softened. The secre-
tions from this inflammation are not contagious.
The active principle of the plant is called abrine and is regarded
as a ferment; there is no specific microscopic germ associated
with it.
A patient who has once been treated by jequirity, though less
susceptible, may be again brought under its influence. If it be em-
ployed in unsuitable cases, or with too great freedom, suppurative
inflammation or even destruction of the eye may follow. The rem*
edy is to be used with safety only in inveterate cases where other
remedies have failed and where vision is seriously impaired. Its
non-contagiousness permits its use when one eye only is affected.
It is never right to inoculate both eyes at once. Coppez, who
treats in Belgium a great many cases of trachoma, sometimes uses
the infusion as strong as 10$. But the cases must be extremely
severe and unyielding to warrant the proceeding. Similarly the
jequirity infusion is employed in so-called scrofulous pannus of the
cornea not caused by granular lids, and also in sclerosed conditions
of the cornea after parenchymatous keratitis. It has been tried to
a limited extent in torpid ulcers, but not with advantage.
It is imperative to know that for cases of simple chronic blennor-
rhoea, or chronic conjunctivitis, its use is contraindicated, because it
is likely to do serious mischief. So, too, in recent trachoma with
succulent hypertrophy and moderate secretion it is not a safe
remedy. When the conjunctiva is hard, nodular, or cicatricial and
the cornea decidedly opaque, it becomes a useful agent. It is
employed by some surgeons (Roosa, Cheatham: see also Widmark,
" Beitrage zur Ophth.," Inaug. Diss., 1884-1891) at the present time,
but it is far less in vogue than five years ago. It sets up a very
uncomfortable reaction and threatening appearances. The methods
previously described are in my esteem to be preferred.
In summing up the treatment, while old and well-established
1 Others have used a smaller quantity, say one bean to an ounce of water
and infused for twenty-four hours.
35G
DISEASES OF THE EYE.
principles and methods have been reiterated, certain new proceed-
ings are set forth, viz., the squeezing by forceps of flabby, gelatin-
ous, extremely redundant masses of granules; exceptionally they
may be excised; the picking out the contents of the granules with
a broad needle, scraping the tarsal conjunctiva with a sharp curette
or by a fine rake so as to get rid of moderate prominences.
The same effect is attained by touching the granules with a hot
needle. For large masses, whether left as isolated projections in
old cases or occurring as general and extreme hypertrophy, a more
decided use of the actual cautery is proper, viz., the thermo-cautery
of Paquelin. For this and sometimes for the use of the forceps an
anaesthetic as well as cocaine will be needful. (Mem. Ether is in-
flammable, chloroform is not.)
For chronic cases, grattage, with or without expression and
canthotomy, is a recent valuable resort. Like all surgical methods
it demands good judgment. The infusion of jequirity will im-
prove vision in desperate cases, while it, too, demands care and
discrimination.
It happens that patients cannot always remain under the hands
of the surgeon for the completion of the treatment of this tedious
disease, and it becomes advisable to instruct a friend in the manip-
ulation of turning the lids and applying either sol. tannin and
glycerin, alum crystal, or the sulphate of copper crystal. In case
the patient must depend wholly on himself, he may be supplied
with an ointment of sulph. cupri, gr. v.-x., ad vaselinum, § i. The
vaseline may in warm weather be stiffened with powdered gum
arabic or starch. The usefulness of corrosive sublimate solution
will vindicate itself, and an ointment of hydrarg. oxid. flav., gr. x.
ad 3 i., once or twice daily between the lids is well worth trial.
The necessity of remitting active treatment when relapses of
inflammation occur, as they may frequently, must not be forgotten,
and then warm water and atropine will be the best relief. The use
of the latter is often kept up during the continuance of the local
stimulants. Sometimes with prominent eyes the lids in the atro-
phic stage of trachoma are left so shortened as to press disagree-
ably on the cornea. Canthoplasty may have done all that it can,
and the skin operation above described (see p. 278) may not be
desirable. In such cases, continual pulling at the skin of the tem-
ple, forehead and cheek, a kind of massage, will, in time, loosen the
subcutaneous connective tissue and secure some relief.
Xeroma of the conjunctiva, which is the stage of consummate
atrophy of the membrane, only admits of palliation by emollients,
such as vaseline or olive oil, several times daily. An admirable
summary of treatment of trachoma has been published by Sattler,
" Die Trachombehandlung einst und jetzt," Berlin, 1891, p. 44.
THE CONJUNCTIVA.
35?
Sequelae of Granular Conjunctivitis.—The secretory glands
become obliterated, the conjunctiva cannot supply the proper
amount of fluid, and hence there is an unpleasant dryness and irri-
tation of the lids. By the shrinking of the tissues the hair follicles
are distorted, the hairs become few and some or many or all of
them may be turned upon the globe. In other words we have
trichiasis or entropium. In the latter condition the palpebral
fissure is shortened, the lids cannot be sufficiently separated, the
tarsi become prominent and are shrivelled into dense ridges. The
cornea loses transparency and its curve becomes altered in irregu-
lar forms. To the casual observer it may seem normal, but a test
of the acuity of vision and the failure of glasses to improve it will
show how serious is the mischief. If examined by the ophthalmo-
scopic mirror (preferably by the plane mirror) or still better by
the ophthalmometer of Javal, the corneal irregularity becomes
apparent. It may become conical and no satisfactory view with
the upright image will be possible.
Treatment of some of the above conditions has already been
described when discussing affections of the lids (see Entropium
and Trichiasis, etc., pp. 262, 276, 278), and those which concern f.he
cornea will be hereafter referred to. Sometimes during the progress
of the treatment of trachoma, the lids become tight or even short-
ened, the cornea is greatly irritated, and the operations of can-
tholysis or canthoplasty are resorted to to check the distressing
symptoms. A. few years ago these proceedings were employed
very frequently, but their use is now more limited, yet occasionally
suitable. The writer's experience in one extremely bad case for
which relief was gained by subcutaneous division of the superior
tarsus on its median line may be again referred to (see page 267).
To operate during acute symptoms will often aggravate them,
while the free use of cocaine and other measures, such as cold lo-
tions, etc., will control the condition.
Partial ptosis is not infrequent. It may be corrected by deep
sutures from the orbital margin of the tarsus, carried up under the
edge of the orbit by a curved needle, and tying the sutures tight to
remain several days. A similar operation might also be done on
the conjunctival surface with excision of some of the fornix.
6. Morbid Growths on the Conjunctiva.—Under this head are
included syphilitic lesions, tubercular deposits, epithelial, lupoid,
and cancerous disease, amyloid degeneration, cystoid growths,
congenital fibrous growths, papillomata, even a bony growth has
been observed and calcarous deposits occur; we also have angio-
mata, and pigment patches.
The syphilitic lesions of the conjunctiva occur oftenest in the
358
DISEASES OF THE EYE.
papebral portion, under the form of chancre or of mucous patches.
On the bulbar portion gummy tumors have been seen, and while
they often include subjacent structures, sometimes their mobility
shows that only the conjunctiva is affected. The diagnosis cannot
with certainty be made from local appearances, but rests also upon
constitutional symptoms. Mucous patches have also been seen on
the ocular conjunctiva. In all these lesions it is usual to find swell-
ing of the pre-auricular lymphatic gland of the corresponding side.
As has been said, we may have upon the conjunctiva either the
primary, the secondary, or the tertiary lesions of syphilis. Cases
of this kind are rare, yet they must not be overlooked. Among
many contributions to the subject are papers by Bull,1 and by De
Beck 2 in which the literature is extensively quoted.
Local treatment will be the same as would be proper if the
lesion were on another locality, save that cauterization of an ulcer
must be done so as not to harm the cornea, and the liability of this
structure to suffer mischief will modify the proceedings. Soothing
applications will be preferred, such as atropia and the milder anti-
septics, boric acid, diluted chlorine wrater, iodoform, etc. The chief
dependence will be on constitutional treatment.
Within the last few years attention has been much called to
tubercular disease of the eye, which may invade any part of it, and
has been found in the conjunctiva, both of the lids and of the globe.
The tendency of the disease is to attack in its progress deeper
structures, and it presents ordinarily an open ulcer, with grayish
surface, from which nodules spring up either within its area or upon
its margin. The disease more frequently seizes upon the inner
than upon the outer structures of the eye primarily, and reference
will be made to it again. The chief importance attaches to diag-
nosis, and we have to distinguish tubercular deposit from granuloma
and from lupus. The former (granuloma) is a firm, reddish, highly
vascular mass of uniform character, and the neighboring lymphatic
glands are not swollen. Persons of any age may be affected. A
tubercular deposit is a mixture of nodules and ulceration, it in-
volves all the tissues of the part affected, its edges are beset with
trachoma-looking masses. In lupus the skin is generally also
affected; Arlt, 1863, speaks of two cases in which the disease began
on the conjunctiva bulbi, and the eye was attacked by continuity
of invasion. In tubercle we find giant cells, and the peculiar bacilli.
In lupus we also find bacilli closely resembling those of tubercle,
and at the present time the two diseases are coming to be regarded
as essentially the same.
1 " Syphilis of the Conjunctiva," by C. S. Bull, Anier. Journal of Med.
Sciences, Oct., 1878, p. 405.
2 " Hard Chancre of the Eyelids and Conjunctiva," by David DeBeck,
Cincinnati, Ohio, 1886. Press of Robert Clarke & Co.
THE CONJUNCTIVA.
359
There is no satisfactory treatment of tubercular deposit in the
lids, and if it occur upon the globe, enucleation will be the ultimate
resort. The suitableness of operating for tuberculous disease of
the lids will depend on the stage of the disease and on the state of
the patient. He is usually young. If possible, thorough excision
should be done, to guard against general infection. There are
numerous cases recorded in literature, but one which is typical and
carefully described is by Baumgarten.1
Epithelial disease and lupus attack the lids quite frequently,
beginning at their margin. They may, if neglected, extend to the
globe (see Plate VI., Fig. 18, colored illustrations). We also some-
times find an epithelioma as a reddish lobulated mass growing at
the limbus corneae. A tumor of this kind which displayed the char-
acteristic microscopic structure I removed from a man fifty-seven
years old, and it has not recurred for nine years. I have also re-
moved a pigmented (melanotic) epithelioma from the same region,
and with no recurrence up to the present time. For details of the
case and references to the literature, see A rchives of Ophthal. and
Otology for 1879. Cancerous ulcerations may attack the ocular
conjunctiva. I have notes of such a case.
Sarcoma,either white or pigmented, may also appear, and while
removal of the disease without sacrificing the globe is to be pre-
ferred, such a course is not always possible. Complete removal is
imperative and that will often compel the loss of the globe. Papil-
loma is found at the inner canthus as a favorite site and is easily
recognized by its softness, its clefts, and its small pedicle.
In the lesions now considered other organs, such as the lymph
glands, the lungs, the liver and other viscera, are to be scrutinized
to learn the true meaning of the case.
Cysts sometimes appear beneath the conjunctiva, usually on the
globe, sometimes over the caruncle. Their contents are commonly
watery, sometimes oily or sebaceous. Young persons are more
often the subjects. The sac may grow to hold as much as two
drachms of fluid and sometimes it reaches back into the orbit.
(The occurrence of true orbital cysts is not meant.) Their walls
are usually very thin, and their transparency suggests their char-
acter. Greater firmness has, however, been observed. Treatment
requires either simple puncture, or besides this a silk seton, or if
obstinate, cauterizing the wall with nitrate of silver. See an article
by Dr. C. S. Bull.2
Fibroid tumors, or more properly dermoid growths, appear on
the ocular conjunctiva, and especially as a congenital formation.
1 " Ein Fall von tuberculosen Greschwiiren der Lid-Conjunctiva," Graefe's
Archives fur Ophthal, Bd. xxiv., Abth. hi., S. 225.
3 " A Study of Sub-conjunctival Serous Cysts," Amer. Journal of Medical
Sciences, Jan., 1878, p. 85.
360
DISEASES OF THE EYE.
There may be more than one, and there may be stiff hairs growing
upon them. A not infrequent condition is to find at the outer angle
a dense plate of hard tissue with thick white covering pushing for-
ward from the orbit usually on the temporal side upon the sclera,
half-way perhaps to the cornea, with a thin, rounded edge. It is
imperfectly movable, but with a rigid base, and resembles to some
extent the plica semilunaris of rabbits and sheep. Between it and
the cornea may be small hard tumors. This is a congenital mal-
formation. A careful description of such a case and an account of
others is given by Hirschberg.1 A tumor of similar kind was
described by Graefe as coming from the orbit between the rectus
superior and rectus externus muscles from the superior fornix.
I have notes of the case of a man who had three hard sessile
dermoid tumors growing upon the limbus corneae about equidistant
from each other. The outer angle of the lids was tied by a band of
fibrous tissue which stretched across it and also adhered to the
conjunctiva bulbi. A similar band ran across the inner angle be-
tween the lids. These bands were quite dense and prevented the
lifting of the lid. The condition was congenital. The other eye was
normal.
Tumors like the above may be removed to get rid of a blemish,
but if they reach into the orbit, the rules of antiseptic surgery must
be strictly observed, because the proceeding becomes serious.
Pinguecula is the name given to a small yellowish elevation be-
tween the semilunar fold and the edge of the cornea. It consists
of connective tissue, elastic fibres, and epithelium, and contrary to
the import of its name it does not contain fat. Most persons in
adult life or at its later periods will present this little elevation
more or less conspicuously. It causes no harm, never becomes
large, and is not to be meddled with.
Pigment patches of a brown or even deeper hue are sometimes
seen on the conjunctiva; they may be stationary and innocuous, or
they may increase, and they may be associated with co-existing
pigmented malignant growths.
Angioma sometimes occurs. Its most frequent seat is the car-
uncle. In the same region we find more frequently polypoid
growths or granulomata. In gouty subjects cretaceous deposit
is sometimes found in the conjunctiva. Loring2 has reported the
remarkable fact of a bony growth.
Xerosis of the conjunctiva or its extreme degeneration and
atrophy is brought about by trachoma, is a result of pemphigus
1 Centralblatt fur Augenheilkunde, Jahrg. vii., S. 295, 1883.
4 " Case of osteoma of the conjunctiva," by Dr. E. G. Loring, New York
Medical Journal, xxxvii., p. 12_, 1883.
THE CONJUNCTIVA.
361
also appears in small glistening patches three or four millimetres
across upon the inter-palpebral ocular conjunctiva, and lastly hap-
pens idiopathically to the production of complete symblepharon
when it reaches its ultimate development.
Taking up the third form, we remark that these satiny white
spots were described by Kuschbert and Neisser and also by
Leber,3 and have a special interest because bacilli and cocci
are found in them. In some instances the patients have the
form of amblyopia called hemeralopia. The connection between
the two facts is not explained, nor do they always concur. When
these patches exist there is also a little foamy secretion gathered
along the edges of the lids due to fatty degeneration of the cells,
and in some instances the cornea becomes infected and ulcerated
by the penetration of the microbes. The disease occurs mostly
among children and especially among the poorly nourished. It
was observed as early as 1874 by Bezold, and by Horner in 1877:
the lesion of the cornea was severe and the dryness of the conjunc-
tiva was regarded as incidental. Leber considers degeneration
of the epithelium the first step, followed by loss of sensibility
through injury to the terminal nerve twigs; the consequence is
imperfect closure of the lids and additional dryness of the conjunc-
tiva—naturally the cornea becomes most seriously exposed to
ulceration. The same kind of degeneration takes place in the epi-
thelium which clothes the pelvis of the kidney, and the same micro-
organisms are found as in the conjunctival epithelium. Cultivation
of these was found to cause the corneal lesion and also the degen-
eration of the conjunctiva. For cases among negro children, see
Kollock, Trans. Amer. Oph. Soc, 1890, p. 626.
Leber proposes to give the name xerophthalmus to the altera-
tion which follows trachoma and which includes atrophy of the
whole membrane, but he points out that this condition may arise
without trachoma, and both he and Arlt have noticed that it may
be attended with atrophy of the lachrymal gland. There is almost
complete absence of normal secretion, which can be only imper-
fectly supplied by water containing \, Descemet's membrane; E„ epithelium covering the sides of the ulcer; G, base of
ulcer with infiltration; J, infiltration beneath bottom of ulcer. X 20.
age, except as the necessities of cleansing and of the above treat-
ment require its removal. Sometimes it will be taken off once in
two hours; sometimes only twice daily. If hypopyum and iritis
appear, warm fomentations of boric acid will be necessary, with
atropia and other measures to be subsequently mentioned as suita-
ble for these conditions. The disease seldom recurs, although Arlt
and Horner have seen instances.
A somewhat rare and peculiar complication of superficial inflam-
mation has recently attracted attention under the name of fila-
mentous keratitis (faedchen keratitis). Small shreddy threads
grow from the surface of the cornea, usually quite short, but in
one instance (Czermak) 4 mm. long. They appear after erosions
or wounds, and also spontaneously. They present a broad or some-
times funnel-shaped attachment; the little filament is twisted like
a cord and usually terminates in a coil or knob. Leber described
1 " Krankheiten des Auges im Kindesalter," p. 333, 1883.
THE CORNEA. 385
them in 1882, and Czermak,1 Nuel,2 and ILjss3 have given draw-
ings and microscopic studies of them. Hess presented specimens
at the Heidelberg Ophthalmological Congress in 1892, and the
pathological character of the outgrowths was generally conceded
to be an excessively active proliferation of epithelium, which could
find no other mode of expansion, and begins either from a vesicle
as in a herpetic process, or from a wound or an ulcer; for example,
several observers noted them upon corneae specked with powder
or dynamite granules springing from the minute ulcers during
the period of acute inflammation. Sometimes the filaments dis-
appear in a few days, sometimes they obstinately persist and grow,
according to the nature of the etiological process. They have been
seen in the puncture made for discission of cataract and mistaken
for prolapse of capsule or of vitreous. Microscopic examination
would decide the diagnosis. No special treatment is to be em-
ployed on their account.
Under the name of herpitiform keratitis, Decker (Klin. Mon-
atsbldtter filr Augenheilkunde, Oct., 1890, p. 409) brings together
various but kindred types of inflammation, which have been de-
scribed within recent years by various authors, characterized by
isolated, nodular, or rounded spots in the parenchyma, sometimes
with vesicles on the surface. There is often bad nasal catarrh and
constitutional dyscrasia; the rate of progress is slow, the sub-
jective symptoms may be acute or torpid. In some instances they
have followed erysipelas or some distinctly neuralgic affection.
Fuchs in his text-book, p. 189, and also in Wiener Klin. Wochen-
schrift, No. 44, 1890, describes a usually chronic disease, lasting
months or even two years with exacerbations, situated in the su-
perficial layers and presenting numerous small spots with inter-
vening haziness. Stellwag described what seems to be a different
affection, "nummular keratitis," with larger spots, perhaps 1.5
mm. in diameter, situated at various depths, sometimes attended
by ulcerations, sometimes by iritis. It runs its course within two
or three weeks and may have acute symptoms (see Oph. Rev.,
March, 1890, p. 78).
Pemphigus Corner, Keratitis Bullosa.
Yet another form of eruptive disease appears on the cornea, as
it does also on the conjunctiva (see page 336), which, notwithstand-
ing its rarity, may be briefly referred to. A well-marked case was
described by Hasner4 in 1860, in a patient who had had severe
1 Klin. Monatsbiatter, July, 1891.
2 Archives d'Ophthalmologie, October, 1892.
3 Graefe's Archiv f. Ophth., xxxviii., 1, 160, 1892.
4 "BeitrSge tiber Augenheilkunde," p. 196, Prag.
25
386
DISEASES OF THE EYE.
kerato-iritis. A paper by M. Landesberg1 cites the literature and
describes seven cases seen by himself. In one of these, as well as
in cases described by other authors, glaucoma preceded this affec-
tion. In all of Landesberg's cases increased ocular tension at-
tended the acute stage. He regards the disease as a peculiar
phase of parenchymatous keratitis of which the lifting of some
corneal layers and the epithelium is an incident. Intense pain and
photophobia, great rapidity in development, a tendency to recur-
rence, acute congestion of the eye, immunity on the part of the iris
and choroid, and deep ulceration w7ith dense grayish opacity, are
features of the disease. Arlt2 describes the disease as a variety of
keratitis interstitialis consecutiva, thereby meaning that some
other disease, such as irido-cyclitis or glaucoma, or deep corneal
lesion must precede this outbreak. In this respect he differs from
Landesberg. Brugger3 had the opportunity of examining an eye
in which this disease supervened upon glaucoma. He shows that
the first step is infiltration of the corneal substance with serum
and nutritive fluid which cannot escape by the usual channels at
the limbus. New tissue is formed between Bowman's membrane
and the epithelium, which speedily degenerates, and the lamellae of
the cornea are dissevered and the fluid pushes forward the super-
jacent layers into one or more vesicles.
Treatment of these cases has consisted in the usual antiphlogis-
tic remedies, to which have been added, excision of part of the infil-
trated cornea (Hasner), incisions into the base of the ulcer (Lan-
desberg), iridectomy and sometimes enucleation. The nature of
each case will suggest the appropriate remedies.
Keratitis Vasculosa.
Under the above title we may designate a condition which is
not primary, but secondary to some other form of disease. We
meet it so often that a special designation is convenient. The sur-
face of the cornea is rough with erosions and proliferations of
epithelium, has a grayish hue and is covered by a mesh work of fine
blood-vessels. The vessels (1st) may appear in a streak and run
to a spot of infiltration or ulceration; (2d) they may form a well-
defined fringe at the margin occupying a part or the whole of the
periphery and stopping abruptly at about two millimetres from
the edge; (3d) they may cover the upper half, or constitute a patch
on some other part of the cornea; (4th) they may overspread the
1 Archives of Ophthalmology and Otology, vol. vi., p. 135, 1877.
2 " Klinische Darstellung der Krankheiten des Auges," S. 125, 1881.
3Monatsblatter fur Augenheil., xxiv. Jahrgang, S. 500, 1886.
THE CORNEA.
38?
entire surface. In some of the above phases there will be conjunc-
tival and scleral hyperaemia. Persistent phlyctenular inflamma-
tion, and granular conjunctivitis are the chief causes of the condi-
tions described. Commonly the disease is of long duration. In
certain cases, especially when in isolated streaks or patches, the
blood-vessels are significant of, and adjuvant to a process of repair.
By their aid an ulcer is being filled up with new tissue, or the
opaque elements of an infiltration are being absorbed.
In other instances the blood-vessels and the attendant lesions
merely denote the irritating effect of some severe provocation—
such as inverted eyelashes, the roughened palpebral conjunctiva,
exposure to the air by imperfect closure of the lids, etc. When the
cornea is vascularized extensively, we may have a partial, a total,
a dense or a thin form of pannus.
It is common enough to have the curve of the cornea distorted
in old cases of pannus; often the adjacent scleral and conjunctival
tissue will be thickened and vascular; sometimes in advanced
stages a yellowish dense opacity creeps into the cornea from its
edge, seeming to involve its entire thickness and assimilating both
sclera and cornea to each other in appearance, and is designated
as sclerosis of the cornea. In the severe forms of vascular kerati-
tis, iritis is common, and naturally the deeper structures will not
always escape. As a matter of course vision is seriously damaged
and may be reduced to perception of light; there will be pain, lach-
rymation, photophobia, etc. The condition is necessarily chronic.
Treatment.—It will be understood that in the above description
a distinction must be made between the conditions of vascularity
which are reparative in their nature and those which are not. It
ma}' not be necessary to interfere with the milder forms of repara-
tive vascularity, while in certain cases the blood-vessels continue
long after they have fulfilled any useful purpose. This we find when
a leash or streak of vessels runs from the sclera into the cornea
after an attack of phlyctenula. Excision of the vessels or scarifi-
cation of them has been practised, but with not much advantage.
Dusting calomel into the eye is useful in mild cases; but rubbing
into the cornea the yellow oxide of mercury ointment once daily
and with rather severe friction for several minutes is the most
efficient remedy. The strength will vary from gr. i. ad 3 i. to gr.
viij. ad 3 i. In some cases eserine sulphate, gr. i. ad 1 i., decidedly
aids the healing, used once or twice daily. But one must be watch-
ful against pupillary adhesions and occasionally use atropia. If
the vascularicy is severe, atropia will be exclusively used. To the
above treatment warm fomentations for a half hour, three times
daily, will be helpful. The eyes will be guarded from extreme light
and the general health will be well attended to. The useful effects
388
DISEASES OF THE EYE.
of cocaine will not be forgotten, and it may be used three or four
times daily in a 2$ or Af0 solution.
For the extensive and dense forms of pannus, the yellow oxide
ointment and warm fomentations are beneficial, but the first con-
sideration is to remove, if possible, the cause. We must pluck
offending eyelashes; cure by operation entropium of the lid; we may
have to do cantholysis or canthoplasty (see p. 357), to relieve the
pressure of shortened eyelids. In a large number of cases we can
do nothing directly for the cornea, but give attention to the care
of granular conjunctivitis.
The division of the tarsus vertically through its middle with-
out going through the skin is available in extreme cases. We
are on the same ground now as was discussed under trachoma
(see p. 357). A remedy not mentioned in that connection is in
obstinate and insensitive cases of pannus to apply to the cornea a
drop of turpentine.
The proposals of curetting the cornea, of peritomy, of inoculating
with infusion of jequirity have been already made.
Should the cornea have become soft and be inclined to de-
velop a staphyloma, an iridectomy may be performed, despite the
existence of pannus, and it should be broad. The existence of gran-
ulations is not a contra-indication, provided they are not very lux-
uriant—as is not likely to be the case under such circumstances,
and the situation will, of course, be serious, if not desperate as to
the chances for useful vision. The effect of iridectomy in mild
cases of either opacity or vascularity of the cornea is to hasten de-
cidedly the restoration to a better state. But the enlargement of
the pupil is disadvantageous to sharp sight, and inasmuch as some
opacity or want of homogeneity is most likely to remain, the ultimate
effect of the iridectomy becomes hurtful. It must not, therefore,
be resorted to while by other means the cornea may be made to
clear up, and one must have signs of increased tension and devel-
oping staphyloma, before resorting to it in the cases above referred
to.
A form of superficial keratitis dependent on malaria has at-
tracted my notice for many years. Some of my cases were pub-
lished by Dr. Jas. L. Minor in Am. Journal of Med. Sciences, 1881.
The lesion is confined to the epithelium and anterior layers, it is
attended by ulceration, rarely by much infiltration, although this
may become suppurative. The conspicuous feature is the blunted
sensibility of the cornea and the consequent slight photophobia.
The touch of a lock of cotton will scarcely be noticed. There will
almost always be tenderness of the supra orbital nerve at its notch
and pain in its radiations. There will also be a history of malaria
and probably of the chronic type. Dr. Kipp, of Newark, N. J., has
THE CORNEA.
389
reported observations of the same character and like myself is
firmly persuaded of the malarial etiology of the disease. He has
called attention to peculiar irregular streaks, which he has seen in
some of these cases which correspond exactly to lesions published
by Emmert under the name of keratitis mycotica dendritica (see be-
low). The same type of disease was published by Hansen-Grut, and
is represented in Fig. 138, p. 376. I recognize the disease in the cor-
nea and doubt not its mycotic character. That it is pathognomonic
of malaria seems to me improbable, because I have seen many
cases of malarial keratitis without it and have seen one extremely
typical case in which the cause certainly was not malaria, but ex-
posure to a fearful snow storm. An Irishman was engaged in dig-
ging out the Long Island Railroad from the snow-drifts of the
famous "blizzard" of March, 1888, and after working three days
was brought with several comrades to the N. Y. Eye and Ear In-
firmary suffering with so-called snow blindness. I found the above
streaks running through both his corneae. Under cocaine I applied
a very fine galvano-cautery, and relief from the intense photopho-
bia and other symptoms speedily followed; while a weak bichloride
solution procured healing of the furrows and subsidence of the in-
flammation within less than a week. This case was purely local.
But true malarial keratitis, while it may have the nodular branch-
ing streaks, will not be cured without quinine or arsenic. Local
and if mycotic striations occur, antiseptic methods and remedies
will be added (see Kipp, Trans. Am. Oph. Soc, 1889, p. 331).
Keratitis Interstitialis, Diffusa, Parenchymatosa.
In this affection the substantia propria is the part of the cornea
primarily involved, although the posterior layers, including the en-
dothelium, very often are implicated. But there is no impairment
of the epithelium save that in some instances it has a dull, finely
molecular appearance, like the surface of ground glass; ulcerations
and purulent infiltration are absent. The disease belongs to the
early periods of life. It commonly appears about puberty, yet
often shows itself at a much younger age, and I have seen a case
whose appearance was delayed until thirty-three years of age. The
cases may be mild or severe; they begin gradually, and their dura-
tion may be from several weeks to many months, or even five years
(Mooren). One eye alone may be involved, or both simultane-
ously or successively.
Etiology.—The disease has a constitutional origin, and in the
larger number the cause is hereditary syphilis. It is probable
390
DISEASES OF THE EYE.
that a few cases owe their origin to secondary, i.e., to acquired
syphilis. My own conviction is in favor of acute interstitial kera-
titis as a symptom of secondary syphilis, but I admit the difficulty
of proof and that the cases are rare. More will be said on this
point. It is also true, that a contingent of cases of interstitial
keratitis show no distinct indications of syphilitic taint, but are
classed as scrofulous subjects. In them we cannot always dis-
criminate or know, how far unhealthiness of tissue is owing to
bad hygiene, or to remote ancestral syphilitic lesion or to other
hereditary dyscrasiae. In all cases we have to do with unhealthy
subjects, notwithstanding that many do in certain particulars
show signs of blooming health. Not a few young girls with un-
mistakable signs of hereditary syphilis have plump and well-
rounded forms and rosy cheeks and declare that they feel entirely
well. Without actual statistics to support the statement it is
my strong belief that girls are more frequently affected than
boys. (Hutchinson in his original paper, I find, made this state-
ment, founded on statistics giving the ratio as boys 1 to girls
1.8. Oph. Hos. Reports, ii., p. 94.) The proportion of cases
which have a syphilitic origin has been indicated by Horner.1
Among fifty-one cases, twenty-six had hereditary syphilis; two
had acquired syphilis; in ten, syphilis was strongly suspected,
making nearly two-thirds of the whole number. Among the re-
mainder a scrofulous constitution accounted for a large proportion,
and Arlt relates a few cases in his long experience which were due
to malaria. On this point something more will be said. There
remain other cases of traumatic and rheumatic, or gouty origin
and we also have interstitial keratitis resulting from chronic irido-
choroiditis.
Symptoms.'—The outbreak is usually in haziness of the cornea,
beginning either at the margin or at its centre, and it may be con-
fined to a portion or overspread the whole surface; it is likely to be
in minute spots. The polish is not perfect, because of minute de-
pressions like pin punctures which give a dull or grayish hue. The
circum-corneal hyperaemia is much less than the degree of opacity
would seem to demand. Gradually the opacity grows more dense
and at an early date vision is extremely reduced. Photophobia
and pain will often be moderate, there is no sticky secretion and
lachrymation is slight. Such are the features of mild cases. Under
more severe types, we have blood-vessels appearing in the deep
and also in the superficial parts of the cornea; they form a close
network and sometimes the whole structure is absolutely red.
The tissue softens and may yield its curve under the pressure of
'Thesis by Jackowlewa Pulcheria: "Ueber keratitis interstitialis dif-
fusa," Zurich, 1873.
THE CORNEA. 391
the lids. There will be more intense pain, ciliary injection, and
photophobia, and copious lachrymation. An abundance of vessels
in the cornea bespeaks a protracted case. Often iritis adds its
quota of misery and mischief. One may sometimes discern through
the fog by the ophthalmoscope, dark streaks and numerous dots
which denote the participation of the deep and posterior layers.
Seldom can the pupil be illuminated, even in mild cases. A few
days may suffice for the whole cornea to become hazy. The dura-
tion is from three to twelve months. Perfect recovery may occur
or a most damaging opacity may remain. Seldom in the fortunate
cases, is the cornea restored to a perfectly normal structure, even
though it appear clear.
The symptoms which indicate a syphilitic taint are glandular
enlargements, epecially in the neck, nodes on the bones (the tibiae,
clavicles, sternum, ulna), a peculiar facies in which the nose is
sunken and the upper jaws are imperfectly developed. The teeth
will command careful inspection. The incisors will be small, coni-
cal, with wide interspaces, and the edges either bevelled off or
notched at the middle. The central incisors of the upper jaw are
the distinctive teeth. They are not apt to be carious, but while
often of bad color, they may be very white. The lateral incisors may
be similarly affected. The incisors of the lower jaw may or may
not be affected. No absolute uniformity is to be predicated of the
characteristics of the teeth, but they are ill-developed, may be
irregular, by their notches show a crumbling disposition, or if regu-
lar are dwarfed. The cut, Fig. 142, is from a plaster cast of teeth
belonging to a young girl in whom the syphilitic symptoms were
unmistakable, and the keratitis of the typical sort. In scrofulous
children the front teeth sometimes look as if part of the crown
had been filed off at about the middle and parallel to the gum,
leaving the distal end much thinner than the basal part with
an abrupt transverse ridge running across it. They have been
described as terraced. They are dark, are seamed and soft, but
392 DISEASES OF THE EYE.
they do not indicate syphilis. They are declared by Horner and
others to denote rickets or scrofula: while this may sometimes
be true, it is not the only explanation. The accompanying Fig.
143 is taken from a plaster cast of the teeth of a young lady who
never had keratitis, and her father, a physician, knows that there
has been neither syphilis nor scrofula nor rickets, or any heredi-
tary taint in the family for three generations. The deformity of
the teeth was caused by an attack of sickness during the second
dentition, bv which their development was arrested and impaired.
Radiating- fissures or scars are apt to be found on the lips or at
the angles of the mouth. The skin is usually coarse and flabby,
although exceptions occur. There is generally anaemia. The flat-
tening of the nose is noteworthy. There is nasal catarrh, often
amounting to ozaena. The vault of the hard palate is narrow and
the roof of the mouth is thrown into a high and narrow arch. We
may be told of the existence during childhood of snuffles and per-
haps of eruptions about the anus or elsewhere.
The patients are also prone to deafness, either by subacute or
chronic otitis media, or by suppuration of the middle ear with dis-
charge. I have seen a patient become totally deaf after passing
through the eye trouble with fortunate result. If the patient be
one of many children in the family, he will often be the first or
second born. There may have been previous still-births, and it is
always important to inquire about miscarriages, and about chil-
dren who may have died young. Hutchinson found that 53 mothers
who had borne 371 children whose average age was 9| years, had
lost 192. Sometimes a clear statement and admission of syphilis
can be obtained from the parents. Mooren1 found instances where
the syphilis seemed to have been derived only from the grand-
parents. Out of 64 cases, Mr. Hutchinson found 53 to have the
attack in both eyes, although more frequently in succession and
not together. Although seldom, a second attack may happen to
the same eye. My belief that acquired syphilis can cause parenchy-
matous keratitis is corroborated by Alexander,2 who mentions, p. 41,
having seen thirteen cases and others have reported isolated cases;
see Mauthner and others in the literature quoted by Alexander. It
comes as a late symptom, viz., two or three years after the initial
lesion, and resembles that due to hereditary disease. Ulceration
does not occur, but by the softening of the corneal tissue it may
undergo serious change of curve and even become staphylomatous
(Symons, Oph. Soc. United Kingdom, 1886, July 2d). The disease will
yield only to antisyphilitic treatment.
Prognosis turns much upon the general state of health and
1 "Ftinf Lustren Ophthalmolog. Wirksamkeit," p. Ill, Wiesbaden, 1882.
2 " Syphilis und Auge, Wiesbaden," 1888.
THE CORNEA.
393
upon the intensity of the syphilitic symptoms. Should the case
belong to the scrofulous rather than to the syphilitic type, the out-
look is scarcely more favorable, perhaps it is less favorable. It is
surprising how notably a densely opaque cornea may clear up, but
the contrary is too often the result. A highly vascular cornea with
yellow or buff infiltration of its deep layers, is not likely to give
useful vision. A word as to the next generation. I have seen
the child of a mother who had had interstitial keratitis with char-
acteristic symptoms of hereditary syphilis, not have any tokens
of syphilitic taint. But its health was delicate, digestion feeble,
nervous system highly excitable; these conditions were aggravated
by the indiscreet indulgences of the mother, regarding the child's
habits of eating and living.
Treatment.—Both local and constitutional methods are essen-
tial. Local treatment is the steady, prolonged, unvarying use of
hot fomentations or poultices for three hours to eight hours daily;
making periods say of two hours each, or of one hour each at equal
intervals. The more severe the case the longer must be the dura-
tion of fomentations. A bunch of absorbent cotton, or of soft rag
wrung out of water whose temperature is 104° F. or 108° F. (39° C.
to 49° C.) is to be held on the eye until it feels cool and then dipped
and replaced. Much higher temperature will be accepted, or even
demanded, after prolonged use. I have known a young girl to in-
sist on having the water at 120° F. No treatment can take the
place of this. It is wearisome, but the patient can have no other
occupation, even if only one eye be concerned. It is wise to use
atropia to guard against adhesions of the pupil, but it does little
good to the cornea. No irritating remedies are in order, until the
hyperaemia of the ciliary region has greatly declined, and then the
yellow oxide of mercury ointment will do good service, rubbed in
once daily.
Anti-syphilitic treatment must in the proper cases be instituted
at the outset. If there be a doubt as to the existence of this taint,
it is often safe to assume its presence. Mercurials are well borne
by children and in my judgment they are more important than
iodides. With them, iron, cod-liver oil, and other tonics are to be
combined. The best and most nutritious food, and attention to
the digestive functions, the use of meat and milk, and in suitable
cases insistance on outdoor exercise, protecting the eyes with
smoked glasses, are all matters to be cared for. Mercurials are
best given by inunction, using either the blue ointment or the 20$
mixture of the oleate of mercury. The latter is more energetic
than the former and more liable to irritate the skin. In some
cases a strip of flannel smeared with blue ointment may be worn
about the body. Hydr. bichlor., gr. i.; aquae, § viij., a teaspoonful
394
DISEASES OF THE EYE.
three times daily, is an easy formula for children. Most writers
advise the addition of iodides to the mercurials, but for many years
I used the latter without the former and with satisfactory results.
Ptyalism must be carefully avoided. Syrup of the iodide of iron,
Blancard's pills of iodide of iron, a combination of mass, hydrarg.,
gr. iij. vel v.; ferri sulphat. exsiccat., gr. i. Fiant pilulae, one three
times daily, are all useful remedies. In very feeble subjects quinine
and pyrophosphate of iron or similar combinations are indicated.
Only when a certain degree of strength is present can solutions of
iodide of potassium or sodium be borne. Mr. Hutchinson was in the
habit of prescribing mild mercurial ointment to be rubbed into the
axillae, on the neck, etc., at bedtime, and also a mixture containing
iodide of potassium, iodide of iron, and tincture of nux vomica.
It must be remarked that very weak or cachectic subjects will
not bear specific remedies. For them vigorous tonic and nutritive
measures take the first and exclusive place. In deciding upon the
line of proceedings, one must take all the conditions of the patient
into consideration. This holds good even though syphilitic taint be
certain.
For cases which exhibit no specific symptoms, local measures
and attention to general health will alone be required.
Keratitis Suppurativa and Ulcerations of the Cornea.
The conditions spoken of do not invariably accompany each
other, but are so usually coupled together as to make it convenient
to discuss them under one head. Suppuration most frequently
begins at the surface and leads to ulceration. If an ulcer exist, the
loss of substance has in many cases been preceded by suppuration.
Besides superficial suppuration, we meet with purulent infiltration
originating in the deep layers, and we also have cases of total sup-
puration of the whole structure.
The process may be traumatic, it may come from purulent con-
junctivitis, it may be derived from an active suppuration in deeper
parts of the eye, in some cases it comes with great rapidity in a
marasmic child (kerato-malacia), it attends upon anaesthesia of the
cornea and is combined with ulceration, and it may be both trau-
matic and infectious. Within a few years we have learned the im-
measurably momentous influence of micro-organisms in causing
corneal suppuration. They are its immediate promoters in the
large majority of cases. This is eminently, if not exclusively, true
of the superficial varieties. We cannot always account for their
presence, but we have no difficulty in accounting for them in many
cases of injury by foreign bodies, or when the eye is exposed to
the air through anaesthesia of its surface, or when the secretions
THE CORNEA. 395
•
of an inflamed lachrymal sac flow over the eye, etc. They.may
be conveyed by metastasis through the general circulation, and
this occasions the most intense and overwhelming forms of suppu-
ration. In 1873, Stromeyerx published experimental researches into
the causes of hypopyon-keratitis. In the same year Leber2 wrote
on inflammation of the cornea by septic infection. In 1875 Horner3
presented specimens of an ulcer of the cornea which had occurred
in a case of erysipelas of the face and which were filled with bac-
teria, and he called the disease mycotic keratitis. Two years later
he exhibited to the Heidelberg Congress a similar lesion from a
child with marasmic ulceration of the cornea
He did not venture then to call the bacteria the exciting agents
of the process, but since then we have accumulated abundant evi-
dence in favor of this belief. It has been repeatedly shown that
the inoculation of the cornea with pure cultures of certain bacteria,
will surely bring on suppuration and ulceration. It has been shown
that this occurs if putrefying matter is used, if diphtheritic exuda-
tion is used, and with the aspergillus glaucus (Leber4) which ranks
higher in the scale of life than the cocci. A wound made by a knife
tainted with certain bacteria will suppurate, if made with a steril-
ized knife it will not. Erysipelas is occasioned by a micrococcus in
the skin, the same can cause ulceration of the cornea. See Haab,
1. c, p. 777, who gives an illustration of such a mycotic keratitis.
It is also true that a suppurative action may be set up by organ-
isms floating in the blood, as in cases of pyaemia and similar dis-
eases. The subject is most suggestive and practical in its nature
and leads to important conclusions both as to prevention and treat-
ment. Bearing these facts in mind, we are greatly aided in under-
standing and in dealing with ulcerations and suppurations of the
cornea.
In depicting the various phases of corneal suppuration we can
refer to only a few conditions; such as; 1st, small specks of super-
ficial infiltration; 2d, hypopyon keratitis, or idcus corneal serpens
(Saemisch); 3d, neuro-paralytic keratitis; 4th, kerato-malacia; 5th,
keratitis xerotica; 6th, consecutive ulceration as in conjunctivitis
or in deep inflammations of the eye.
1 Graefe's Archiv, xix., Abth. 2, S. 1.
2 Centralblatt fiir die Mediz. Wissenschaft., S. 129, 1873.
8 Klin. Monatsblatt fiir Augenheilkunde, xiii., p. 442.
iGraefe's Arch, fiir Oph., Bd. xxv., Abth. ii., S. 285, 1879.
396 DISEASES OF THE EYE.
1. Small superficial Infiltrations. Keratitis superficialis
purulenta discreta.
We frequently see purulent deposits on the cornea which are
from one to three millimetres in diameter with a yellow centre, a
hazy border and sometimes with whitish streaks running into them.
Severe forms of phlyctenulae take on this type and are most fre-
quent among children, but we find them among adults as the result
of infection.
The most common cause assigned for them by patients is the
intrusion of foreign bodies; but while they may often enough do
the mischief, other sources of infection are plentiful; such as wip-
ing the eyes with dirty fingers, with soiled rags and handkerchiefs;
the patients will sometimes have ozaena or ulcers of the septum
nasi, or will have lachrymal trouble, etc., etc.
There will be circum-corneal redness, pain, photophobia, and
lachrymation, and dimness of sight. The subjective symptoms
vary greatly in intensity. When they are severe, the frequent use
of 4$ or 8$ solution of hydrochlorate of cocaine gives great relief,
and to this atropia sulphate, gr. ij. ad § i., may be added once or
twice daily. Hot fomentations are grateful. But the effective
treatment consists in scraping out the yellow infiltration with a
spud after the full effect of cocaine has been procured and then oc-
cluding the eye with a pressure bandage. (Sometimes the yellow.
oxide of mercury ointment may be immediately rubbed in.) Keep
this on so long as it feels comfortable, say for six or twelve hours,
and then foment for half an hour with a warm 3$ solution of boric
acid or of corrosive sublimate, 1 to 5,000, or with warm water.
The warm applications may be renewed for one-half hour every
two hours. The loss of substance soon shows signs of repair and
recovery sets in. A similar effect is obtained, though less easily
and perfectly, by touching the yellow spot very carefully with a
fine brush dipped in solution of nitrate of silver 6$ (gr. xxx. ad 1 i.)
or of carbolic acid 20$, or of corrosive sublimate 1 to 2,000. By
these chemical methods, the germs are destroyed, but a risk of in-
juring healthy cornea is incurred which does not attend mechanical
remoA7al of the yellow deposit. To promote healing of the ulcer
the yellow oxide ointment, gr. ij.-vi. ad 3 i., may be rubbed in once
daily, after the irritation has subsided.
THE CORNEA.
397
2. Hypopyum Keratitis. Ulcus Corneal Serpens.
The condition thus denoted is more serious than the preceding.
Besides purulent infiltration, ulceration attends it, and in most cases
(i.e., 80$) there will be pus in the anterior chamber (hypopyum).
While it has various grades of intensity, it is always a grave dis-
ease. Vision is rarely perfectly recovered, and the proportion of
cases in which the eye is entirely lost may be as high as 19.2$.* In
Arlt's2 clinic, the total loss was 9.5$ ; in Horner's,3 it was 14.2$.
Young persons may have it, but a large proportion of its subjects
are above middle life; it chiefly appears among the old and feeble.
It may have an acute and sthenic character, but it commonly is
torpid in its behavior, although none the less destructive.
Etiology.—In many instances there has been an injury, espe-
cially by chips of stone or metal flying from a workman's hammer.
The traumatism gives oppor-
tunity for infection by micro-
scopic germs. If they are
not introduced by external
agencies, they are in many
cases furnished by the secre-
tions of dacryo-cystitis. The
striking fact has been proven
that from 20$ to 32$ of the
cases have this complica-
tion. Still more direct proof
of infectious origin is found
in the fact that the disease
in all its phases can be artificially produced in rabbits by inocula-
tion of pure cultures of the coccus pyogenicus aureus, and other
germs. Stromeyer4 established the same fact in 1873 by the appli-
cation of putrescent material and showed that the reaction after the
infection was much more intense, although the wounds were trifling,
than when the wounds were clean, even though they were extensive.
The presence of pus in the anterior chamber as well as in the
cornea is explicable in several ways. It may find its way through
the posterior surface of the cornea, or may creep down between the
membrane of Descemet and the corneal substance, and filter
'Lucanus, "Ulcus cornese serpens," inaug. dissert., Marburg, 1882, p. 31,
Clinic of Prof. Schmidt-Rimpler.
2 Bergmeister, Klinische Monatsblatter, 1874, pp. 78-87.
sBokowa, Inaug. dissert., Zurich, 1871.
4 Graefe's Archiv, xix., 2, S. 26.
398
DISEASES OF THE EYE.
through the ligamentum pectinatum. In addition, the exudation
comes from the iris, because inflammation of the iris and ciliary
body is the usual accompaniment of the disease. Sometimes a dis-
tinct streak of pus can be traced from the ulcer to the bottom of
the cornea. The quantity of hypopium is in a measure proportion-
ate to the severity of the attack. A considerable collection of pus
between the corneal layers (onyx) is not so frequent as hypopium,
and may be distinguished from the latter by focal illumination and
by the circumstance that it will not alter its form or situation
when the patient lies upon his side or hangs the head well forward.
The course and symptoms are as follows: In the sthenic cases
the attack is sharp, there is acute, sometimes agonizing pain and
photophobia. The hyperaemia is not extreme; it is confined to the
vicinity of the cornea; there is no chemosis or swelling of the lids;
secretion is chiefly watery, but as the disease advances becomes
puriform. Very often the middle of the cornea is the spot attacked
and the chief lesion will be upon the lower half. At the outset
there will be a small bright yellow spot with ulceration, surrounded
by a hazy areola. The aqueous humor will be a little turbid and a
slight yellow precipitate soon shows itself at the bottom of the
anterior chamber. If not already visible it may be brought to
view by letting a drop of water brim over the edge of the lower
lid. Rapidly the ulceration and infiltration spread, and pain shoots
into the brow and temple, besides attacking the eye. The pupil will
be small and the iris discolored. The illustration, Fig. 131, shows
both extensive purulent infiltration of the cornea and hypopyum.
Under asthenic conditions the onset is not so distressing; the
hyperaemia quite moderate, the tendency to ulceration is more de-
cided than to infiltration, although the bottom of the ulcer is a
yellowish gray. A patient may permit the process to go on until
a considerable part of the lower half of the cornea is involved be-
fore applying for aid. Such an occurrence is, in fact, frequent, and
during the one to three weeks which have elapsed, the suffering
has not been intense. Meanwhile pus has been accumulating in
the anterior chamber, and it may be half full. The material is
fibrinous and plastic as well as puriform. If the process be un-
checked, perforation occurs with prolapse of the iris and general
suppuration of the eye. If this be not the turn of affairs, the cor-
nea bulges as the iris protrudes in a black bead, pus finds vent,
perhaps the lens escapes; a staphyloma corneae is formed. It is
not necessary to try to depict the various phases of the destructive
stage of the disease. We see it often in old people under the guise
just sketched.
Treatment—If seen early, when the ulcer is small and the hy-
popyum slight, great relief will be afforded by fomentations with
THE CORNEA.
399
hot water and by solution of sulph. atropiae, gr. ij. ad § i., once in
three hours. Hot water may be used for half an hour, and the eye
closed for half an hour. If pain be severe, give an anodyne, perhaps
hypodermatically. The ulcer, if small, may be scraped with a sharp
spud or curette. If it be more than two millimetres across and the
purulent infiltration extend for a considerable space around, the
best remedy is the actual cautery. A special electrode has been
made for this purpose, and the thermo-cautery of Paquelin has
been reduced to such fineness as to be eminently suitable. If
these implements are not at hand, a knitting needle heated by
an alcohol lamp may serve passably well. The cautery, if limited
accurately to the infiltration gives no pain. It will, however, be
proper to use a 4$ solution of cocaine, or with children an anaes-
thetic. With the galvano-cautery great care must be used not to
let the point go too far, although all the infiltrated tissue must be
burned.
In case a suitable cautery is not available, I can fully coincide
in the suggestion of the late Dr. E. Williams,1 who recommends
pure carbolic acid to be applied " by means of the bulbous end of a
very small probe and confined rigidly to the ulcerated surface."
Dipping it very lightly in the acid, all parts of the ulcer are touched
until they turn white. Minute adjacent abscesses are also touched
or rather bored out by the probe. Such an application may be
made once a day until the ulcer is checked. The cautery will not
need repetition so often. Some prudence is necessary in the choice
of cases, because an additional amount of opacity may be produced
by the cautery. It is in the early and progressive stage that it
may most confidently be employed.
All the measures suggested for treating the ulcer, viz., scraping
it, the actual cautery, and the carbolic acid, are similar in intent
because they destroy pernicious germs, and thus check the spread
of the disease.
In moderate cases hypopyum will spontaneously disappear.
Often paracentesis will give great relief from pain, and it may be
done at the lower edge of the cornea or better through the ulcer.
If the ulcer be deep, and nearly central, reaching almost to the
membrane of Descemet, but is not more than three millimetres
across, a very fine cautery may be pushed through its bottom and
will evacuate not only the aqueous humor, but bring the hypopyum
to the opening, where it may be pulled out by forceps. It is usually
tenacious from the admixture of fibrin. One will not always suc-
ceed in this manoeuvre, and it is better, when there is considerable
pus, to make a broad incision to be sure to bring out the exudation.
1 Report of the Fourth International Ophthal. Congress, p. 102, London,
1873.
400
DISEASES OF THE EYE.
The treatment of ulcus corneae serpens by a free incision across the
whole ulcer, as formulated by Saemisch, was a great advance upon
previous modes of treatment. But if seen early, the necessity of
the extensive incision across the whole cornea may be avoided by
the cautery.
Should the ulcer be large, say six millimetres, and the pus copi-
ous, we have the choice of two methods. The one is to cut across
the cornea with a Graefe's knife through the middle of the ulcer
and evacuate the pus (Saemisch). The wound will usually not need
to be opened again; the secretions flow away continuously. The
eye must be washed out with antiseptic fluids, preferably with
diluted Labarraque's solution of chlorinated soda, 1 to 5, or with cor-
rosive sublimate, 1 to 3,000, as often as the formation of secretion
requires. Similar antiseptic lotions are suitable in the milder cases
just mentioned. Hot fomentations will be alternated with a pres-
sure bandage. The details of management will be controlled by the
facility with which the eye can be kept clean. Immediately the
necrotic tissue is eliminated and healing begins, although some-
times the ulcer will for a time extend. Usually, but not always,
the iris is caught in the wound.
The case will require several weeks for complete healing. There
may be sufficient clear cornea left to permit an iridectomy. On
the other hand, a staphyloma may be formed which may need oc-
casional puncture to restrain its growth, and it may subsequently
require iridectomy or other operation. Still another termination is
that the whole cornea may be ravaged, may leave no opportunity for
improvement of sight, and may flatten down into an opaque cicatrix.
The alternative to the treatment by Saemisch's incision is to
perform an iridectomy, as advised by Graefe, and at the same time
take out the hypopyum. The wound must be large, and preferably
it will be above; yet one may be obliged to choose another locality.
If the secretion be cohesive it can be readily removed. But some-
times it has no tenacity and must then be washed out of the an-
terior chamber. This may be done by a rubber bulb syringe, using
a 3$ solution of boric acid, or solution of biniode of mercury 1 to
20,000, or of corrosive sublimate 1 to 5,000. The last two fluids
have the great advantage of coagulating the secretion, but may
possibly cause opacity of the endothelium. Forcible irrigation of
the wound will carry the fluid into the anterior chamber. Panas
has a special piston syringe whose point is carried through the
wound and of course acts more directly, but demands great care.
Ordinarily one must also resort to forceps.
For the most severe cases the complete division of the ulcer
will be preferred. If the quantity of pus is moderate, the ulcer
deep but limited, and a considerable part, perhaps one-half of the
THE CORNEA.
401
cornea, clear, a broad iridectomy with irrigation of the anterior
chamber will both check the disease and provide an artificial pupil.
Since the value of the actual cautery has become known, it will
not be so often needful to resort to an operation, or a less extensive
paracentesis will suffice. An important practical point is to know
when the cauterization should be repeated. When the cornea has
grow7n very thin and new spots of infiltration appear, one may fear
to employ so potent a remedy; nevertheless, several applications
may be needed, each being done with caution. The reaction is slight.
It is not safe to use it upon a prolapsed iris projecting in the middle
of an ulcer. Extension of suppuration into the interior of the globe
has thus been produced (Knapp1). So long as the edges of the ulcer
are infiltrated with pus they may be burned until the process ceases.
But upon the surface of a deep ulcer after one good cauterization,
one may be content to apply sol. corrosive sublimate, 1 to 2,500, or
1 to 2,000, or the aqua chlori or liq. sodae chlorinatae diluted, 1 to 7.
Such topical dressings, limited to the ulcer by a small swab, may
be repeated two or four times daily and weaker solutions with
much greater frequency. Iodoform has undoubted value. It must
be in extremely fine powder and dusted upon the cornea twice daily.
It may be mixed with vaseline. Aristol will serve the same pur-
pose and is void of objectionable smell. Pyoktanin has failed to
justify the warm encomiums of Stilling. Known as methyl blue
and methyl violet it has been extensively tried, and while by some
it is much praised in treating purulent conjunctivitis, phlyctenula,
and corneal ulcers, the weight of opinion is that it is not better
than remedies already in use. It is not poisonous, is a true anti-
septic, and is used in sol. 1 to 1,000 (see Cent ralblatt fiir Augenheil,
Supplement, 1892, pp. 504, 505, etc.). Tincture of iodine to the ulcer
is much praised by Chibret.
Application of pure carbolic acid by a probe is likewise, as I can
testify, effective. It must be accurately used to the infiltrated
puriform edges and spots, and the surface drenched with sol. boric
acid, etc. (Dr. E. Williams).
In spite of treatment, sometimes because of extreme debility, or
because of neglect to apply seasonably for aid, suppuration may
extend to the interior of the globe. It may take on a chronic com-
paratively painless course ending in shrinking, or there may be
great swelling and chemosis, orbital cellulitis, exophthalmus, violent
pain and constitutional reaction. Under the latter circumstances,
the eyeball should be freely opened, the lens evacuated and the
vitreous allowed to escape. Many times have I wiped out the con-
tents of the sclera with a sponge (evisceratio bulbi) as I stated in
1 Transactions American Ophthal. Society, 1885, p. 44.
26
402
' DISEASES OF THE EYE.
1872.1 This will not always relieve the orbital infiltration, and it
may be needful to divide the deep part of the sclera to give vent to
the fluids. Enucleation gives more complete relief, but it is attended
with much bleeding, is a severe and unwelcome operation and is
not without risk to life (see on Enucleation of Eye). The age and
condition of the patient must be well considered before resorting
to it. Evisceration will in the end afford a better stump for a
glass eye, than will be left after enucleation. In hospital practice
enucleation will be more frequent, because the patients are more
quickly enabled to return to work. Still another mode of dealing
with suppurative panophthalmitis is proposed by Chibret,2 viz., to
make a large incision in the cornea, to open the capsule and let out
the lens—the fluid pus escapes; to pick out with iris forceps all
thick and cohesive purulent material; to inject a solution of corro-
sive sublimate, 1 to 2,000, repeatedly, until the fluid returns perfectly
clear and until by picking and washing all pus is removed. Then to
stuff the eye with absorbent cotton smeared with iodoform oint-
ment and cocaine; over all a compressive bandage (a sterilized
sponge would be better because less likely to need removal). The
bandage to be changed once in twenty-four hours, and for two or
three days the injections to be repeated. Chibret declares this pro-
ceeding much superior to those above described, and it deserves a
trial. If the suppurating eye is left to itself, the case goes on slowly
and painfully; the sclera perforates in several places, the discharge
is copious and offensive, the health suffers, and after weeks or
months the eye shrinks to a stump,
Neuro-Paralytic Keratitis.
By paralysis of the fifth nerve or of its ophthalmic branch both
the cornea and conjunctiva lose sensibility. In consequence ulcer-
ation and suppuration of the cornea occur. It has been proven both
experimentally and clinically that the immediate cause lies in the
diminution of the impulse to winking, and the consequent drying of
the surface of the cornea, the shedding of its epithelium and the
penetration of foreign particles and germs. If the cornea alone
have become anaesthetic, as happens after optico-ciliary neurectomy,
the sensibility of the conjunctiva and the function of the lids re-
main and no harm ensues. If total anaesthesia of the front of the
eye exist, and the lids be kept closed, the cornea remains intact.
Formerly the cause of the lesion was thought to be due to im-
1 Report of Fourth International Cong., London, p. 27, 1873
2 Archives d'Ophthalmologie, quoted in The American Journal of Ophth.
(Alt), Feb., 1887, p. 55. H
THE CORNEA.
40:;
pairment of trophic fibres which preside over the nutrition of the
cornea and which accompany the sensitive fibres of the nerve.
Even now we cannot utterly refuse to admit such an influence, be-
cause in some cases not only has the cornea become inflamed, but
ulceration of the skin of the forehead has occurred (Higgins).1 The
process is very slow and naturally painless. Ocular tension is
usually slightly reduced. The striking feature is that severe con-
junctival and corneal disease displays itself, with perhaps copious
purulent secretion and inflltration, without pain or photophobia.
The disease is happily not frequent, but if not understood is very
destructive.
There may have been severe neuralgia preceding the anaesthe-
sia. The cause will be some lesion of the fifth nerve or of its first
branch either below or above the ganglion of Gasser: by an injury,
a tumor, syphilis, tubercle, etc., etc. See E. von Hippel, Graefe's
Archiv, XXXV., 2, S. 217, who attributes the lesion to the loss of
the stimulus which the fifth nerve supplies, to production of moisture
from conjunctiva and lachrymal gland; also, de Schweinitz, Archiv
fiir Augenheilkunde, XXV., 1, 2, 152. Gaule (Centralblattfiir
Physiologie, V., 15, 409, and 22, 689) saw lesions of the epithelium
and of the corneal corpuscles under the microscope in fifteen min-
utes after section of the ganglion of Gasser; of course the want of
moisture could be contributory to such lesions, but not essential
and primary. His experiments are very suggestive and leave us
still uncertain as to the true meaning of the process (see p. 161,
where a lesion almost certainly in the pons caused mild neuro-par-
alytic keratitis). See Knies, "Die Beziehungen des Sehorgans,"
etc., p. 214, Wiesbaden, 1892.
Treatment consists primarily in protection of the eye. To this
will be added warm fomentations, antiseptics, paracentesis, iridec-
tomy, and other means already mentioned. I have not used the
actual cautery in such a case. The use of vaseline on the cornea
will be sufficient in mild cases, and is always worth trying. A
striking illustration of the importance of protection was as follows:
A man, aged 66, had a tumor behind the angle of the lower jaw
which had been growing for more than 50 years. It caused total
paralysis of both the seventh and the fifth nerves of the right side.
He was unable to shut the eyelids, and the insensibility of the surface
affected not only the eye, but the cheek, the lids, the forehead, the
cavity of the mouth and the nostril of the right side. There was
conjunctivitis, ulceration of the cornea, and hypopyum. Closure of
the lids by india-rubber plaster brought some improvement, but to
make the protection effective the outer halves of the upper and
lower lids were pared just within the lashes and united by sutures.
1 Medical Times and Gazette, ii., p. 856.
404
DISEASES OF THE EYE.
After they grew together the destructive process in the cornea was
subdued. The patient died not long after from erysipelas. It is
worthy of remark that in spite of the trigeminal anaesthesia there
was at times severe pain in the forehead and temple of the same
side.
Besides protection of the eye, electricity has in some cases seemed
helpful, that is, by the constant current: Nieden1 reports benefit
from hypodermic use of strychnia in two cases which were due to
injury of the head.
When an acute attack has passed and left an opacity of the cor-
nea it may become necessary to perform iridectomy for restoration
of vision, and by proper precautions any further trouble may be
prevented. For a patient who had paralysis of the fifth and seventh
and other nerves of the right eye and atrophy of the optic nerve of
the left, and in whom acute keratitis occurred, vision was partially
restored by iridectomy, and was secured by wearing a bandage at
night and using vaseline upon the conjunctiva and cornea several
times daily. In another case for whom I performed iridectomy to
arrest the active process, the patient aided me in the operation and
felt no pain until section of the iris, which exhibited its usual sensi-
bility.
Kerato-malacia.
This term describes a rapid and destructive suppuration of the
cornea, not preceded by paralysis of the fifth nerve, but ensuing
after very debilitating diseases. It occurs chiefly in children after
scarlet fever, or exhausting diarrhoea, or typhoid fever; in the
marasmic; it has been seen in hereditary syphilis; it has been de-
scribed as occurring after encephalitis. A state of torpor and in-
sensibility with imperfect closure of the lids invites the entrance of
foreign bodies and of infectious germs, and thus practically puts
these cases under the same category with those described in the
preceding section. Adults may also be affected, as in cholera or
scurvy, or low fevers, etc., etc. Naturally there wrill be little or no
pain, and the conjunctiva will show at the outset but little vascu-
larity. The conspicuous symptom is the infiltration of the cornea.
This may at first be circumscribed., and be confined to the inter-pal-
pebral opening and gradually enlarge both in surface and depth—
or in some cases the whole cornea may be rapidly involved and
within two or three days become a yellow slough. One eye alone
or both may be involved. Such a condition has been observed in
connection with that form of mycotic conjunctivitis described by
Leber as conj. xerotica, which is characterised by small, scaly, glis-
1 Arch. f. Augenheilkunde, xiv., ii. and iii., S. 249 (German ed.).
THE CORNEA.
405
tening white patches upon its exposed ocular surface, and which
are colonies of bacilli.
A similar purulent destruction of the cornea sometimes follows
operative wounds, as after cataract extraction. Whether infection
from without will explain all of these cases is doubtful; they occur
in spite of antiseptic precautions, and among the very feeble and
the very fat.
Prognosis is bad, and often death ensues. All that is to be said
about treatment has been previously stated.
Ulcers of the Cornea.
Reference has already been made to ulceration of the cornea
as an accompaniment of acute inflammations, but we meet with it
also as a primary affection, and even if it is secondary to some other
disease, it often is the subject of anxiety and attention. We may
deal with ulcers as progressive or stationary or regressive. Their
position is of importance; if within the pupillary region they inevi-
table implicate vision; if near the limbus, they are less liable to
damage sight, but if they go deep they may give rise to dangerous
cyclitic inflammation. Severe ulcers are apt to be attended with
iritis.
A progressive ulcer usually has a grayish bottom and steep or
undermined edges; it will cause pain: when stationary it is apt to
be shallow, semi-transparent and with sloping edges and not pain-
ful; when healing it is apt to be vascular and there will be vessels
l'unning into it from the adjacent parts; it may not be clear, but
color will be a bluish white instead of a dirty gray. There may be
many minute ulcers. Sometimes a torpid facet remains for months
with very little change. The healing process always requires weeks
or even months.
A progressive ulcer may advance either in depth or extent or
both. If it go deep the posterior elastic lamina always offers the
most resistance and will sometimes bulge as a vesicle before it gives
way. Perforation may occur at one point, or, rarely, at several. As
the aqueous humor escapes, the iris presents in and adheres to the
opening, forming anterior synechia. This attachment is generally
permanent, and if the ulceration is large, when healing occurs, there
will be an opacity, a change of curve in the cornea and distortion of
the pupil, leucoma adhcerens. When perforation is exactly central
and small, the lens falls against the cornea, proliferation of the
capsular epithelium takes place, adhesion continues for a time and
as the normal state returns the lens recedes, leaving a white mark
at its anterior pole and sometimes a delicate thread leading to the
406
DISEASES OF THE EYE.
scar on the cornea to denote the exudation which has been thus
stretched out, and perhaps has been broken.
In certain, not frequent, cases where the pupillary edge has been
caught in a perforation, the hole is not fully occluded by exudation,
its sides become lined by epithelium, and a permanent aperture re-
mains, a fistula corneal, through which the aqueous constantly leaks.
After large perforations we may have escape of the lens, or pet-
haps of the vitreous, and the end may be either phthisis bulbi, or
staphyloma. In most cases, whether the lens do or do not escape,
the iris prolapses, and partial or total staphyloma ensues.
An ulcer in some cases increases in extent and not in depth; an
instance is the so-called ulcus rodens (Mooren) which begins at the
margin and creeps centripetally over the whole cornea. It has a
grayish bottom and undercut edges; blood-vessels quickly come
into it. It makes steady and rather slow progress and it obsti-
nately resists treatment. Moorenx describes four cases, in all of
which the sight was lost. Sattler has used the galvano-cautery to
advantage; Schmidt-Rimpler brought one case through, by anti-
septics, warm fomentations, and scarification of the vessels. One
case under my care was vainly treated by occlusion and other means
and finally yielded to Saemisch's incision. For such cases (see
Jany2) scraping the bottom and edges, the actual cautery, anti-
septic lotions, occlusion, and perhaps paracentesis, will offer the
best chances of success; these being aided by other means to be
presently mentioned. Another kind of progressive ulcer begins at
the edge of the cornea and ploughs a furrow around its periphery;
often strangely transparent. It is called the ring ulcer. It occurs
in the feeble, especially the old, and it can make rapid havoc of the
eye. There will be little pain and not much hyperaemia, but pro-
lapse of the iris and loss of the eye too quickly come to pass. The
feebleness of the subjects makes treatment of little avail against a
process which probably is of infectious origin, although no investi-
gations on this point have yet come under my notice. Fortunately
such cases are not common, and to them the same rules of treat-
ment will be proper as to the preceding. Sometimes the disease is
more slow and then gives a chance for treatment.
In general it may be said that to check a progressive ulcer we
must resort to antiseptics: sol. sodae chlorinatae, 1 to 7, or corrosive
sublimate, 1 to 1,000, or iodoform in powder or ointment 10$, or sol.
nit. arg. 2$, with a small fine brush'to the bottom of the ulcer. The
edges may be scraped or touched with the actual cautery; all dead
1 Ophthalmiatrische Beobachtungen, 1867, p. 107.
8 Centralblatt f. Augenheilkunde, June, 1885, p. 162.
THE CORNEA.
407
tissue is to be removed. We may use pure carbolic acid on the tip
of a probe with a steady aim and light touch. We may do paracen-
tesis more or less free; iridectomy may be suitable if iritis is already
established, but the simpler operation of paracentesis, as a rule, is
better at first. If the ulcer be near the middle of the cornea atro-
pia should be used; if it be marginal, sulphate of eserine, gr. ss. or
gr. i. ad 5 i., is to be preferred. The latter remedy sometimes pro-
vokes iritic adhesions and should not be used in strong solutions.
One may even use sulph. atropia once daily and sulph. eserine twice
or three times daily in certain cases. Sol. cocaine 4$ and eserine,
gr. i. ad § i., are to be highly commended. To the above we may
add warm fomentations for periods of an hour several times daily
and a bandage.
It is not intended that all the above category of proceedings
should be applied to every case. Good judgment must select what
is suitable. When a prolapse of the iris becomes prominent para-
centesis may be done. Pinto (Ann. d'Oculist, April, 1892, p. 246)
makes the happy suggestion to treat the exposed boss of prolapsed
iris by first loosening it from its attachment if needful, using a
fine probe under the corneal edge, excising it, and then cover the
wound with a little graft of the ocular conjunctiva. Bandage both
eyes for three days. A firm white scar eventually ensues which
excludes infection. He does this during the early period of pro-
lapse and secures a more speedy healing than when the ulcer is
left to itself.
The treatment of torpid and stationary ulcers is by stimulation.
An old remedy is tr. opii diluted with water 1 to 5, but the yellow
oxide of mercury in vaseline, gr. ij. or gr. x. ad 3 i., well rubbed in
once daily or every second day, is to be commended. Sometimes
touching the congested palpebral conjunctiva with nitrate of sil-
ver solution, gr. v. ad 3 i., or a solution of tannin in glycerin, gr. x.
ad 1 i., is to be preferred. For these cases when very chronic one
may have to employ a variety of collyria, while the most important
factor is time (tinctura temporis!).
Keratitis Postica.—Uveitis Anterior.
Inflammation of the deep layer of the cornea (sometimes called
the uveal layer) is produced by inflammation of the choroid, ciliary
body, and iris. Two forms may be referred to. One which is
common exhibits numerous minute dots on the posterior surface
and chiefly on the lower half of the cornea. These dots are in part
precipitations from the aqueous and in part proliferations of the
endothelium. The disease is primarily a serous iritis or cyclitis
and was formerly called by the inelegant name of Descemitis.
408
DISEASES OF THE EYE.
There will be faint circum-corneal injection, discoloration of the
iris, probably adhesions of the pupil and slight turbidity of the
aqueous fluid (see Fig. 145).
Another lesion of the cornea appears during or after cyclitis as
a brownish spot on its centre and in its substance, which under a
low magnifying power is resolved into a cluster of molecules. It
stays for some weeks or months and may not wholly disappear.
The treatment will be included in that of the original disease, with
the addition of hot fomentations. The conditions now alluded to
will be again mentioned under the head of serous iritis. We have,
in fact, a disease in which the corneal manifestation is subsidiary to
a larger process. But of these post-corneal opacities, which have al-
ways excited much curiosity, we have late investigations by Fontan,
Fig. 145.
who examined two eyeballs thus affected. The more valuable case had
traumatic irido-cyclitis. The membrane of Descemet was normal—
its epithelium was not proliferated, but the new deposits were heaped
upon them in little hillocks, and consisted of cells, some of which were
3roung and active, others crenated and deformed. They were
regarded as migrated and altered lymph corpuscles, and between
them were granular particles supposed to be cement. Similar cells
existed on the iris and on the anterior capsule, and in the anterior
chamber was coagulated fibrin, which near the iris became fibril-
lated. In the iris and the canal of Petitx was fibrinous exudation,
and the ciliary body was similarly infiltrated and was atrophic.
The deep part of the choroid as well as the retina and optic nerve
had suffered a degree of atrophy, and the arteria centralis was
blocked by a clot. The corneal lesion evidently was only an
outlying indication of a deeper lesion and not a disease per se.
Whether all cases of keratitis punctata may be so regarded remains
to be learned. Further discussion will be found in pp. 442, 466.
To the pathological conditions of the cornea already described
various phases of disease might be added which are rare, or which
'Recueild'Ophthalmol., Nov., 1888.
THE CORNEA.
409
do not fit into ordinary classifications. In fact, the pathology of
the cornea is a large field of study and is not compressible into
any ordinary treatise. The following may be referred to:
A. A chronic hypertrophy of the epithelium at the limbus which
in time encircles the entire cornea with a dense whitish band, which
encroaches for a width of two to four millimetres upon the trans-
parent structure. Its free border is a little elevated and abrupt.
There is no ulceration and no vascularity. Sometimes attacks of
more or less catarrhal conjunctivitis occur and are apt to last several
weeks. During the spring they are said to be more frequent, and
from this cause the disease has been styled vernal catarrh. It is
described at length by Saemisch1 under conjunctival affections, and
is perhaps less frequent in this country than in Europe. I have
seen it occasionally, and it has been previously mentioned (p. 362).
B. Keratitis dendritica exulcerans mycotica. By this title
Emmert2 describes cases which begin with a small superficial ulcer
and extend in a dendritic or branching form close under the surface
Fig. 146.
making a figure resembling a twig of a tree. The lines are white,
and the epithelium is soon shed, leaving open furrows. Subjective
symptoms are severe and if the case when first seen is well advanced,
it will be obstinate; recovery occupying six or more weeks. Inves-
tigation found bacilli in the infiltration.
The only successful treatment was washing the eye with corro-
sive sublimate, 1 to 1,000 (how often is not stated), and repeated use
of eserine-vaseline ointment, 20$. There is no tendency to iritis.
For months after recovery the white streaks could be discerned.
Hansen-Grut3 describes cases which in many respects are similar,
but in which the subjective symptoms were not so severe and ulcer-
1 Graefe and Saemisch, " Handbuch," Bd. iv., S. 25.
8Centralblatt f. Augenheil., Oct., 1885, S. 302.
3 Congres p^riodique internationale, Copenhagen, 1886, B. hi., p. 38.
410
DISEASES OF THE EYE.
ation did not take place over the lines of inflltration. He believed
them to be mycotic, but was not able to prove it. See case quoted
on page 358 (see Fig. 133).
C. In old cases of glaucoma and similar forms of degenerative
disease of the globe, a chronic inflammation attacks the cornea along
the line of the lid opening. Opacity begins at each side within a
millimetre of the limbus, as minute specks which multiply and
finally after long duration compose a band or zone across the struc-
ture from two to four millimetres wide. It sometimes happens
that, besides minute yellow dots which are common, calcareous
specks and scales appear within the stripe and may even compose
a continuous film. Of this quality are certain cases described by
Nettleship x in which the eye had not lost vision and for which iri-
dectomy was available after removing the scales. Such stripes
running horizontally across the cornea were described by Graefe
as accompaniments of glaucoma and irido-choroiditis, but there are
evidently varieties in their quality and relationships. I have seen
this band well marked in an eye lost by sympathetic ophthalmia.
I did iridectomy and the whole vitreous was found fluid and escaped.
The cases rarely admit of benefit by any method.
D. Sclerosis of the cornea is a dense yellowish opacity of its
substance which follows upon some chronic affections and looks like
atheromatous patches of the arteries. It ensues on prolonged and
profound disease of the inner structures and may involve its en-
tirety. We also meet a gray opacity coming down from the lim-
bus affecting both the epithelium and parenchyma, and showing a
few vessels in its midst; unattended by injection of the ciliary
vessels or photophobia or pain—very slowly progressive and re-
bellious to remedies. It behaves like a sclerosis, but doubtless is a
kind of inflammation, in default of any accurate knowledge of its
true nature.
E. Arcus senilis, or gerontoxon, appears as a grayish-white line
at about one millimetre from the limbus; begins usually above; it
may encircle the whole cornea; it occurs chiefly and frequently after
middle life, but sometimes appears before this period. It consists
of fatty degeneration of the cellules and fibrillae, and has no special
importance. Its presence does not contra-indicate operations.
In addition to the above-mentioned conditions reference may be
made to p. 366, where several forms of herpetiform disease are
alluded to, and besides these we have other lesions. One of them
called by Stellwag nummular keratitis, is referred to in Central-
blatt fiir Augenheilkunde, Dec, 1890, p. 362. I have seen an
instance of it, and it has affinities with the interstitial forms of
keratitis.
1 Archives of Ophthal. (Am. ed.), vol. viii., 3, 293.
THE CORNEA.
411
SEQUELS OF KERATITIS.
We shall consider opacity, fistula, and staphyloma of the
cornea.
Opacity.
The attempt has been made to classify opacities according to
their density, by the names nubecula, macula, leucoma, albugo, etc.,
but the terms have no exact value. It is of great importance to
recognize how great is the disturbance of vision which almost invis-
ible opacity wiU cause, if it overspread much of the pupil. This is
often brought to view among patients with strabismus. To discover
a very faint opacity one must use oblique illumination in a dark
room or examine with the ophthalmoscope and feeble light. A
plane mirror having behind it a convex glass of three inches focus
will do the best service. One may also realize the injury to vision
on attempting to see the fundus by the upright image through a
small pupil. Distant vision is always more disturbed by faint
opacities than near vision. An object will be held at short range
and no complaint may be made, but no glass will greatly improve
distant sight. Sometimes a cylinder may be helpful, because of
astigmatism, and it frequently is of a mixed variety, but much im-
provement is not often obtained. The use of a stenopaic slit (Don-
ders) is seldom accepted, because the field of vision is so greatly
reduced. A well defined opacity partially covering the pupil is
much less damaging to vision even if dense, than a faint haze with
filmy edges.
The cure of opacities depends on their density and their extent.
The more recent, the more likely are they to improve. So long as
blood-vessels remain in their vicinity the improvement will continue.
The restorative action will go on for months, and naturally with
more energy in young than in old subjects. Treatment consists in
stimulating applications, or, in popular speech, in using " something
to cut the film." The most serviceable are, very finely powdered
calomel dusted daily into the eye and which is especially suited to
children. Ointment of the yellow oxide of mercury, gr. ij. vel x. ad
1 i., to be used every night, or once in two or three nights, according
to susceptibility; astringent drops in various strengths, viz., sul-
phate of zinc, alum, sulphate of cadmium (not nitrate of silver be-
cause prolonged use stains the conjunctiva); tannin in glycerin,
3 ss. ad 1 i.; tr. opii. diluted, 1 to 10; sol. iodide potass., 1 to 3 or
1 to 2; common salt, 1 to 5 or 20; hot fomentations; powdered
sugar, molasses, etc. None have any specific value, and one may
choose and vary according to the susceptibility of the case. There
412
DISEASES OF THE EYE.
will be extreme variety in this regard. The object is to irritate and
cause hyperaemia, not to last longer than a patient can tolerate.
It may be assumed that opacity will grow fainter for at least a
year and sometimes longer. In some extreme cases of pannus
with very torpid cornea the crystal of sulphate of copper may be
applied to its surface.
When no further absorption is possible and a dense opacity ex-
ists, two proceedings remain, and they are often combined, viz., iri-
dectomy and tattooing. An artificial pupil should, as a rule, never
be made during the recent stage of an opacity unless very dense
and extensive. Its effect is to promote transparency of the cornea,
and the vision is damaged by the enlargement of the pupil. When,
however, the pupil is covered or the iris is prolapsed and a margi-
nal part of the cornea is more or less clear, an iridectomy will be
in place. Sometimes a large piece of iris should be excised, as
when considerable cloudiness covers the part of the cornea where
the opening in the iris must be made; at other times a small pupil
must be made in case the cornea at the site to be chosen is rela-
tively or really clear. It is often difficult to do the operation when
there is anterior synechia or partial staphyloma. With a shallow
anterior chamber a narrow Graefe's knife should be chosen and
cut at a tangent to the edge of the cornea. A narrow iris knife
will serve the purpose in most cases. In some instances of central
opacity with free pupil, incision of the sphincter iridis by Wecker's
scissors (pince ciseaux), or by a fine scissors invented by Mr. Carter,
may be possible and sufficient. To make a small pupil the corneal
wound must be small, it must not be very oblique, else the internal
wound will not lie at the desired point, and a Tyrrell's blunt hook is
better suited to the removal of a small piece of iris than forceps.
The shank of the hook must be soft to permit bending at any angle.
Good judgment and considerable technical skill are often required
in dealing with the cases we are now considering. The spot at
which a pupil is to be placed is frequently not a matter of discre-
tion, because there may be only one clear region If, however, a
choice is possible, the lower segment of the cornea is to be preferred
to the upper; downward and inward, or downward and outward
are the best localities. If there be only one available eye and the
upper part of the cornea must be chosen for the pupil, a free divi-
sion of the superior rectus muscle will both turn the globe some
degrees down and help the lifting of the upper lid by co-ordinate
action so as to bring the pupil to bear for vision. This must not
of course be done, if the other eye have good vision or if there be
any attempt at binocular vision.
The degree of sight gained by an artificial pupil is rarely good.
The curve of the corneal margin is always irregular, it is usually
THE CORNEA. 413
made more so by the attending inflammation—we often must deal
with a hazy structure, and it happens sometimes that the lens is
partially or perhaps wholly opaque. It is not often that a patient
is enabled to read except at a very short distance. But the ability
to go about alone, to see large objects, and emancipation from ab-
solute blindness or helplessness is a great boon. The enlargement
of the field on the side which otherwise would be blind is ample
justification of the operation. It may be remarked that because in
one eye the pupil is central and in the other it may be at the mar-
gin of the cornea, this condition does not involve diplopia. Patients
will seldom use two such eyes in combination, because they differ
greatly in acuity, but if diplopia occur it is due to want of
proper direction of the eyes, not to the optical disagree-
ment.
Very dense opacities which are a blemish both to sight
and to personal appearance may be tattooed with India
ink, as was suggested by Wecker. Some improvement of
vision is gained by doing it where an artificial pupil has
been made, because the quantity of diffused light entering
the eyes is reduced. The most favorable conditions are
when the cornea is normally or abnormally thick.
The ink will not lodge in a thin and staphylomatous cor-
nea, and in such cases the operation sometimes sets up dis-
agreeable reaction. Recent cases must, of course, be
avoided. Sometimes the pigment is introduced into the trans-
parent cornea to diminish the injurious influence of a large
iridectomy upon vision; it acts as a kind of diaphragm.
The fine (and expensive) quality of ink is to be used. The end
of the stick should be soaked for an hour in water to make it pasty,
and a bit of the paste equal to the size of the spot to be colored
placed upon the cocainized cornea. Perfect anaesthesia can be pro-
cured by using a 4$ solution of muriate of cocaine, two or three
times. Fixation of the globe by forceps which have no teeth, or
whose tips are made of tortoise shell, avoids wounding the conjunc-
tiva (although not the possibility of tearing it), and the consequent
chance of staining it by overflow of the pigment. Numerous and
rather forcible pricks with a bundle of needles driven obliquely in
various directions will force the ink under the epithelium, and if it
be thick enough one sitting may suffice (see Fig. 147). If needful
the proceeding may be repeated. In some cases, especially if the
cornea is thin and vascular, the pigment is absorbed and disap-
pears in a year or a few months. It has been found in remote
parts of the cornea, whither it had been carried by the wandering
cells. On the other hand, I have recently seen a patient in whose
eye the blackened spot remains eight years since I made it. These
414
DISEASES OF THE EYE.
deposits have not been known to cause any irritation, although, as
stated, the operation sometimes causes reaction. A series of needles
arranged side by side is preferable to a single grooved needle. It
occurs to me that a case of sympathetic ophthalmia after tattooing
has been reported.
A calcareous deposit sometimes forms upon the exposed part
of the cornea in eyes degenerated after irido choroiditis, after inju-
ries, as well as upon staphylomatous corneae; the tissue may other-
wise be clear. The deposit occurs beneath the epithelium in irregu-
lar specks and* lines and slowly increases during years. It causes
no irritation until it attains considerable size and causes erosion of
the epithelium; then it acts as a foreign body and should be scraped
away under the influence of cocaine.
Fistula of the Cornea.
If perforation take place so that the pupillary edge of the iris is
caught in the hole, it sometimes happens that occlusion does not
occur, but the canal becomes lined by epithelium and remains per-
manent. A leakage of aqueous goes on, the eyeball becomes soft
and irritable. Usually this happens after severe and extensive
ulcerations. The point of perforation is denoted by a black spot, at
which a little of the iris may be seen and the cornea will be very
thin. The accompanying conditions may be various; I have known
an instance in which a fistula remained for seven and a half years
and resisted all treatment. The w7hole cornea was opaque and
iridectomy was impossible. Several attacks of inflammation oc-
curred and I performed enucleation.
Treatment.—The attempt should be made in recent cases, to
cause adhesive inflammation by touching the fistula with the
actual cautery, using a fine point, or with a sharp point of nitrate
of silver. The latter will cause more reaction than the former. If,
after fair trial this method fail, an iridectomy will generally suc-
ceed. Besides the surgical measures, rigorous occlusion by a band-
age should be enforced and sometimes this alone will suffice. Pinto,
Ann. d'Oculist., April, 1892, p. 249, suggests transplantation of a
conjunctival graft after loosening the iris from the cornea (see p.
407). In very few instances will removal of the eye be demanded.
It should not be practised if any useful sight can be secured.
Staphyloma Corner.
This deformity is a usual outcome of extensive ulceration, and is
explained by the pressure of the intraocular contents upon the
fresh reparative material before it has gained strength for proper
resistance. For a period lasting sometimes for w7eeks, while the
neoplastic structures are young, they have a certain translucency
THE CORNEA.
415
which deludes the patient into supposing that useful sight will re-
main, and the inexperienced physician may share the same belief;
but gradually, as the tissues gain thickness, discernment of objects
becomes more vague, and finally
nothing but quantitative percep-
tion remains. The new mem-
brane, which takes the place of
the destroyed cornea, as it bulges
forward acquires more resisting
power, and finally an irregular
conical or rounded prominence of
more or less opaque hue remains,
which constitutes a corneal sta-
phyloma. In some cases, where
the whole cornea melts away, the
transparent lens with undamaged
capsule presents itself, and per-
suades the patient that the sad
prognosis which had been given cannot be true; but a veil of
opaque tissue slowly forms which shuts off sight.
The term staphyloma simply signifies a bulging, and we may
have a great variety of forms. The shape may be conical with its
apex central or lateral; it may be globular, it may have a lobu-
lated surface (likened to a bunch of grapes); it may be limited to
the cornea or involve the ciliary region, or be accompanied by dis-
tention of the whole globe (see Figs. 148, 149). It may be densely
white or bluish, and traversed by large vessels: it may be thin and
dark colored. The iris is commonly adherent to and incorporated
Fig. 148.
Fig. 149.
Fig. 150.
with the cicatrix. The lens is generally opaque or may have
escaped, or have been wholly or partially absorbed. The deeper
structures may have undergone morbid changes; but often per-
ception of light remains.
The tension is usually in excess, but the yielding of the tissues
commonly averts serious glaucomatous symptoms. But in Fig.
150 the optic nerve is deeply cupped and the retina detached.
416
DISEASES OF THE EYE.
Complications, however, arise, in case, as is not infrequent, acute
inflammation occurs. Then there will be intense injection and
extreme pain, and serous effusion in the lids and other tissues.
Suppuration and ulceration may attack a corneal staphyloma,
The outgrowth may be so great as to push the lids far forward,
and it may become impossible to close them during sleep. It is
noteworthy how the upper lid adapts itself to an advancing
staphyloma and succeeds in protecting it. On the summit of a
staphyloma calcareous deposits sometimes form and cause extreme
irritation. We meet the deformity oftenest as the result of oph-
thalmia neonatorum, or small-pox or gonorrhoeal conjunctivitis.
Treatment—With a large perforation, if staphyloma threatens
and considerable clear cornea remains, we may retard or reduce its
growth by a broad iridectomy. This will be feasible when there is
no considerable conjunctival secretion. If during the acute stage
of an inflammation the whole cornea has been swept away and the
lens lies exposed to view, it will be well to open the capsule and
permit its escape, thereby promoting a tendency to collapse of the
eye, rather than to the formation of staphyloma.
During the formative stage of a staphyloma repeated puncture,
especially while the cicatrix is thin, will check or prevent its growth.
When a staphyloma has been fully established, it is often need-
less to do anything with it, especially if the subject be very young
and if it be practically stationary in size, and not large. When be-
cause of its size or for any reason improvement is desired, we may in
some cases perform iridectomy provided the cornea is alone involved
and is in part transparent (see Fig. 148). Care must be taken not
to confound with these cases those in which the projection may be
semi-transparent and large, including the whole anterior part of
the globe, and which are sometimes called by the name of megal-
ophthalmus. They are often instances of intra-uterine iridocycli-
tis and therefore congenital. The same pathological process may
be added in the ordinary development of staphyloma and issue in a
similar enlargement. For such conditions a succession of iridec-
tomies may accomplish reduction. Sclerotomy is sometimes prac-
tised, and in one or two instances I have effected the desired result
by entire avulsion of the iris. Reference will be made to this mat-
ter in the chapter on iritis.
When complete and obtrusive, the purpose of treatment is to re-
duce its size, and make possible the wearing an artificial eye. In
some instances, where no visual improvement is expected, it is bet-
ter to attempt iridectomy rather than to remove any portion of
the cicatrix. There may be only a bluish semi-transparent corneal
margin, through which the atrophied iris is visible. A narrow
Graefe's knife, supplemented by sharp-pointed scissors curved on
THE CORNEA.
417
the flat, will make an adequate marginal wound and shreds of iris
may be torn out. There is danger of loss of vitreous, but by great
care a result may be obtained which will avoid the risks of an ex-
cision operation. The proceeding amounts simply to sclerotomy,
and the abatement in size is gained by the procurement of a thin
filtration cicatrix. Such a proceeding admits of repetition.
For many cases we are obliged to choose between excision of
the staphyloma and removal of the globe. In favor of the former
is the much better stump on which to rest a glass eye; and a care-
ful regard to personal appearance is extremely important. It often
has great influence in the procurement of employment, especially
among clerks, seamstresses, etc. In children enucleation must, if
possible, be avoided, because the soft tissues of the orbit may not
keep up a proper development during the child's growth. When all
proper allowance is made for these considerations, in a certain
number of cases enucleation will be practised. This will be done
when there is a tendency to attacks of inflammation, when there may
be irritation of the other eye more or less sympathetic in character,
and this may be superficial, i.e., conjunctival, or more deeply seated.
I have known myopic persons for whom enucleation of a staphylo-
matous globe was the only suitable proceeding, because no risks
could be taken of the integrity of the remaining eye. This prin-
ciple applies of course to all persons, and if there be reason to ap-
prehend sympathetic trouble of a good eye, the diseased eye must
be removed in toto. It also happens that patients cannot afford
the longer time and greater attention required for the healing pro-
cess after partial removal. To them a better appearance may be
less important than quick recovery and absolute immunity from
future risk or annoyance.
Partial removal is done in various ways according to the shape
and size of the staphyloma, If very thick and conical the apex
may be sliced off, the lens evacuated, the parts thoroughly cleansed
with sol. corrosive sublimate, 1 to 5,000, and the eye closed with a
bandage and absorbent cotton. Keep the latter moist with sol.
corrosive sublimate, 1 to 3,000. The wound will slowly cicatrize and
there may be no unpleasant reaction.
If the mass is more globular a sector may be excised like a
quarter of an orange, by transfixing with a Graefe's knife and
completing the removal with scissors; then draw the edges to-
gether with from one to three fine black-silk stitches. When the in-
cisions and punctures do not go beyond the limbus this proceeding
is entirely safe. For greater security the base of the staphyloma
may be transfixed by one or two common discission needles which
serve to prevent loss of vitreous and to handle the globe. The
incisions in the cornea may be A7ertical (see Fig. 151).
418
DISEASES OF THE EYE.
When we have a larger distention to deal with, including the
ciliary region, a more serious method has to be used. A large
wound opening the eyeball is liable to be followed by intraocular
hemorrhage, because all the tissues have degenerated and grave
inflammation may follow; free loss of vitreous is inevitable. To pre-
vent it Mr. Critchett proposed to pass three needles vertically across
the globe behind the point where abscision is to be practised, and
then remove the staphyloma. The threads of the needles should
then be drawn through, and the eye may be shut without loss of its
contents or much risk of bleeding. The sutures pass through the
ciliary region, which is objectionable both on account of liability to
immediate inflammation and also to possible sympathetic trouble
in the future. To avoid this risk Dr. Knapp dissects the conjunc-
tiva away from the base of the staphyloma, passes through it
the threads, abscises the
cornea and draws the con-
junctiva over the opening.
In either case one may re-
move the lens if it be pres-
ent. I have seen serious
reaction follow both the
above methods of operat-
ing, and seen it extend to
severe orbital cellulitis.
This experience took place when antiseptic methods were not in
use, and I can well believe that at the present time very different
results may be the rule. I should rigidly keep the eye occluded
from contamination with germs, by irrigatio.n with sol. corrosive
sublimate both during the operation and for all the period of heal-
ing, which would be ten to fifteen days.
Retention of the vitreous is aimed at in the above operations,
as a means of avoiding intra-ocular hemorrhage. But the walls of
the globe may be so thin in cases of very great enlargement that
one may better excise the anterior half of the globe, wipe out its
contents, and waiting until all bleeding ceases, may put in stitches
to draw together the edges of the selera; then apply cotton and
bandage and soak continuously with sol. corrosive sublimate. In
all the above operations, if the eye is extensively opened it will be
prudent to use ether or chloroform rather than cocaine to insure
quietness of the patient.
Mr. Mules proposes to put a hollow glass sphere, which he calls
an artificial vitreous, inside the sclera after all its contents are re-
moved, and let this remain. He claims advantages for wearing a
glass eye. The healing requires some weeks and the method will
be described in greater detail hereafter (see p. 774).
Fig. 151.
THE CORNEA.
419
Cornea Conica, Staphyloma Pellucidum.
The cornea sometimes changes its curve to such a degree as to
become visibly deformed yet transparent. This process is not in-
flammatory, but seems to depend upon atrophy of the substance.
Because the conspicuously conical condition was the first to be no-
ticed the disease is known by this name. The shape may be more
or less globular or of some irregular type, and if conical the apex
may be central or is more usually below the centre of the cornea
because influenced by the pressure of the border of the upper lid.
The disease belongs chiefly to the juvenile period of life, i.e., be-
tween ten and twenty-five, although it may be set up much later.
It progresses slowly during several years, seldom rapidly; it ordi-
narily comes to a stand, yet progressive cases are seen in which an
extreme elongation takes place. Such are figured by Von Ammon
and Dairymple and others. Rupture has been reported, but this is
excessively rare. At its beginning and during its progress there
■will be pain and marked symptoms of asthenopia. Distant vision
will grow bad, objects will have to be brought close to the eye, and
if the attempt is made to correct the near-sightedness by ordinary
glasses there will be little or no success. All such glasses cause
pain, and if they improve sight do not bring it up to standard.
Diagnosis.—For pronounced cases recognition is easy because
there is a peculiar brilliancy in the reflex from the cornea and the
form may be conspicuous whether viewed in face or in profile. Seen
from the side the top of the cone looks dark because of total reflec-
tion of light. As a fact if very thin, the summit of the cone is some-
times a little opaque. During the incipient stage we make the
diagnosis by the mirror of the ophthalmoscope, by Placido's disc,
and by the ophthalmometer of Javal and Schiotz. Illuminated by
a feeble light and viewed with the ophthalmoscope from twelve or
sixteen inches, as the light is made to play from side to side over
the cornea a shadow appears at the summit of the curve which
comes and goes, and may assume a vortex-like shape as it varies in
extent; if the vessels are seen they will be broken and twisted
and not of the same size as viewed through different localities.
Inspection of the fundus by the inverted image displays irregularity
in the refraction which becomes very noticeable if the objective lens
be moved from side to side. Inspection by the upright image is
unsatisfactory, because it is impossible to see a continuous field
with one correction and often no glass will afford a proper view.
The vessels appear distorted and broken, and one calls to mind
the effect caused by looking through bad window glass. The optic
nerve is irregularly elongated and its image shifts with the point
of view. If a patient faces the gas light and this is condensed by a
420
DISEASES OF THE EYE.
two-inch lens upon the axis of vision, an observer looking at the
eye from the side, i.e., at right angles to the line of illumination,
will in marked cases see a red reflection of light from the concave
surface of the apex of the cone. It will glitter as does the pupil
under the ordinary mode of illuminating the eye. This observation
I have made for the first time within two years. It had not been
previously noted.
Placido's disc, referred to on page 126, gives a simple and strik-
ing exhibition of the deformity.
If the cornea be examined by the ophthalmometer, the images
of the test objects will undergo various distortions, the sides be-
coming curved, and it will be impossible to give any accuracy to
the measurements. There will be the most irregular forms of astig-
matism, and it will attain very large degrees, often ten or more
dioptries. An interesting fact revealed in this examination is that
I have seen the images pulsate synchronously with the pulse, which
is evidence of great thinness of the cornea. It is also noticed that
the curve of the cornea constantly changes under the pressure of
the lids, especially of the upper.
In using trial glasses, while astigmatism may be readily made
out the symptoms are variable, the lines in Green's card seem to
be curved and the patient makes contradictory answers and cannot
be made to accept any combination which fully corrects the error.
Treatment.—During the early period we must find the glass
which shall be the best approximate correction. The stenopaic slit
placed in the meridians of greatest and least curvature does good
service. With it we use only spherical glasses. Better than this
is Dr. Thompson's way of employing Scheiner's experiment (see p.
82). For this a large pupil is required and one may have to use atro-
pia. The patient looks through two minute holes in a black metal
disc which are either three or four millimetres apart, at a small
gaslight, not more than one-half inch high, at twenty feet in a
darkened room. Over one of the holes a red glass is placed. By
such an arrangement a patient sees two lights when the holes
stand in a meridian of the eye whose refraction is ametropic. If
the meridian be myopic the red light will be seen on the side which
agrees to the position of the hole covered by the red glass. If the
meridian be hyperopic, the red light will be found upon the side
opposite to the position of the hole covered by the red glass. The
holes are to be placed in meridians which cause the widest and
least separations respectively of the images. The disc is inserted
in the trial frame, and by noting the behavior of the images as it is
turned, and the place occupied by the red light, one may with
spherical glasses bring the images together, in each principal me-
ridian. One will often find myopia in one meridian and hyper-
THE CORNEA.
421
opia in another. The principal meridians are not always exactly
at right angles to each other. The difference in optical value of
the meridians gives the proper cylinder, and vision may then be
tried by the test letters.
It is by no means rare that simple convex cylindrics are to be
preferred without spherical correction. In some cases concave
cylindrics placed at right angles to convex cylindrics give a flatter
field and better vision than spherico-cylindrics. In all cases the
extent of field is quite limited. The curve of the cornea is incon-
stant and a slight turn of the eye alters the refraction. We must
not finally depend on the result as found with atropine, but correct
it by future trials and let the experience of the patient control the
ultimate choice. One may test the patient as he faces the light to
get the result with a small pupil. Eserine and pilocarpine may be
tried with or without glasses, because with a small pupil the optical
error is less hurtful; the myotic may be used continuously if help-
ful. The glass which serves for distance will not always suffice for
near work. Much latitude must be allowed for personal peculiar-
ities and only persistent and intelligent trial can decide what will
serve best.
Raehlmann1 proposes hyperboloid lenses for correcting the error.
He makes two systems called respectively A and B. In system A
the axis of the hyperbola is one-third of a millimetre. In system
B the axis is two millimetres. In each system are eleven numbers.
Each glass is thirty millimetres in diameter. It has not been my
fortune to meet a case to which these glasses have been peculiarly
adapted, but Dr. Hay, of Boston, has found them. In theory such
glasses ought to be a complete correction for some cases, but there
is clinically no regularity in the type which they assume, hence, no
regular curve is applicable to all. The choice of the best hyperbo-
loid glass must be made experimentally, and its position must be
shifted until the most useful spot is discovered. When successful a
larger and flatter field as well as better vision are gained.
When a case has gone too far for correction by glasses, surgical
measures may be employed. Paracentesis, unless repeated a great
number of times, has no value. Iridectomy has been employed with
a view to diminish the deformity, but in this respect it has little
value; by displacing the pupil and supplementing it by tattooing
the apex of the cornea, it is said that useful results have been se-
cured, aided by suitable glasses. But the cornea is too thin to ad-
mit of being colored and without doing it the artificial pupil confers
very moderate benefit, or it may prove detrimental to sight. The
1 Klin. Monatsblatter, July, 1881, p. 303; Berliner Klin. Wochenschrift,
1880, No. 34.
422 DISEASES OF THE EYE.
operation of iridesis (Critchett), by which the pupil is drawn to>
one side of the cornea and made to adhere by tying a fine thread
around the little prolapse, has in some cases provoked cyclitis and
sympathetic trouble of the other eye. Its optical effect is good,
but its risks have caused it to be laid aside. Mr. Critchett insisted
that if the wound were placed not in the limbus but at an appreci-
able distance inside the margin of the cornea the injurious after-
effects would not occur. But other methods are available to which
such objections do not apply: they consist in removing portions of
the cornea either mechanically or by setting up ulceration. Excision
of the apex by a Graefe's knife and scissors and drawing the wound
together by fine sutures has been practised. It is a very delicate
proceeding, but entirely feasible, as my experience testifies. The
stitches must be of the finest filaments obtained by unravelling
twisted silk, and the curved needles both very small and sharp,
made expressly for the purpose. The whole corneal tissue is in-
cluded and the stitches must come out in two days. There need
be no prolapse of iris and the scar may be very small.
Mr. Bowman removed a disc from the summit of the cornea by
a trephine or sharp punch which cut its way through by rotation,
and the opening was left to close by granulation. Prolapse of the
iris ensued and generally iridectomy became necessary.
Graefe caused ulceration by shaving off the surface of the apex
and touching the spot with solid nitrate of silver for successive
days until an ulcer was caused with suppuration. This was treated
in the usual way, and after it had healed the cornea would be
flatter. Several months later iridectomy, tattooing, and glasses
completed the treatment. This method, it is evident, is attended
with some risk and requires considerable time for its accomplish-
ment.
A method less dangerous and more prompt (Hirschberg) is to
touch the apex with the actual cautery over a small surface which
can be easily defined and the burning may be repeated until a suit-
able effect is gained. Perforation of the cornea must occur and
prolapse of iris. When the cicatrization is complete, iridectomy,
tattooing, and glasses will be in order.
It has been proved that the effects of prolapse of the pupillary
portion of the iris are far less important than of its peripheral
parts. Moreover, as the prolapse takes place near the centre of
the cornea the dragon the iris is a minimum. Care must, however,
be taken to render the prolapse as small as possible, because it
large it may cause mischief. My own preference is for excision of
a small piece and uniting with sutures.
Transplantation of the cornea has been tried in cases of total
opacity, but not with any satisfactory results. Von Hippel has.
THE CORNEA.
423
devised a spring trephine for its accomplishment and reported
enough success to stimulate further trials. See also Strawbridge.
The insertion of a bit of glass shaped like a shirt-stud has also been
employed, but with little encouragement.
Fig. 152.
The cornea is the seat of morbid growths. We have congenital
fibroma (Graefe), also sarcoma, both white and pigmented, and the
tumor may be highly vascular. Fig. 152 shows a specimen of epi-
thelioma from the museum of the N. Y. Eye and Ear Infirmary.
Papilloma is reported by Dr. Ayers. Tubercular disease and
cancerous ulceration may attack it.
CHAPTEK YI.
THE SCLERA.
Anatomy.—The sclerotica (sclera), which constitutes the greater
part of the outer tunic of the globe, is characterized by toughness,
resistance, and a little elasticity. Its structure is very like that of
the cornea, except that it possesses blood-vessels, while its fibrillae
are coarser and less regularly arranged. They are gathered into
bundles, and cross each other in various ways, and are united by a
homogeneous cement. There are lymph channels and fixed cor-
puscles, and also wandering cells, and a little pigment which in the
African race becomes considerable. Its greatest thickness is be-
hind, viz., 1 mm., while at the equator it is re-enforced by the ten-
dons of the muscles, and just behind their insertions we find its
thinnest portion, viz., 0.4 mm. In front it is covered by the con-
junctiva, and beneath this, is a loose episcleral connective tissue.
Behind, fifteen degrees to the inner side of the macula lutea, and a
little (three degrees) above the horizon, it is perforated by the bun-
dles of fibres of the optic nerve. The place of entrance constitutes
a sieve-like perforation called the lamina cribrosa. The sheath of
the optic nerve joins the sclera. The inner surface of the sclera is
lined by endothelium, which has an imperfect layer of large polyg-
onal cells and pigment. On its outer surface the layer of connec-
tive tissue constitutes part of the capsule of Tenon (oculo-orbital
fascia). The posterior part of the sclera, for a space about ten to
twelve millimetres in diameter, of which the optic nerve is nearly
at the centre, is pierced by blood-vessels and nerves known as the
posterior or short ciliary. Immediately around the entrance of the
optic nerve a few vessels anastomose, and compose a circle which
forms the only medium of connection between the blood-supply
of the retina and that of the choroid. Exceptionally vessels known
as optico-ciliary are found. The ciliary vessels go to the choroid,
ciliary body, and iris (uvea). At the front the sclera is penetrated
by the terminals of the muscular twigs known as the anterior cili-
ary vessels. The nerves penetrate the sclera behind, in the same
region with the vessels. They are twigs from the ciliary ganglion,
whose roots of origin are the oculo-motor (3d), the ophthalmic (5th),
THE SCLERA.
425
and the sympathetic; it lies nearly at the orbital end of the optic
nerve to its outer side. Besides, the short ciliary nerves we have
the long ciliary nerves, two or three in number, given off from the
nasal branch of the ophthalmic, which mingle with the short ciliary
nerves and are distributed to the ciliary muscle and iris. The junc-
tion of the sclera with the cornea is by continuity of fibres, which
have no distinct line of demarcation.
Episcleritis, Scleritis.
Inflammation of the sclera is not frequent, is more apt to be
chronic than acute, is seldom extensive, but occurs in patches; these
have a long duration and are likely to come on in successive spots,
giving rise to the epithet migrans. They may be confined to the
sclera or be complicated by attacking the cornea or the ciliary
body and iris and deep structures of the eye. We have episcleri-
tis, and scleritis. The latter may be general, s. diffusa, or it may
be in spots, s. circumscripta.
Episcleritis
Is an inflammation of the subconjunctival connective tissue, which
occurs in patches, most generally at the region of the inner and
outer angles, and may be either acute or chronic.
Usually it presents an area of pink made up mostly of fine
vessels on some spot between the insertion of the recti and the
margin of the cornea; it is broadly elevated and may or may not
be painful on pressure.
In the beginning, the conjunctiva may be a little oedematous, but
there is no eruption upon it and no sticky secretion. The red spot
pei'sists with varying degrees of discomfort for weeks, and on dis-
appearance often leaves a gray or leaden colored surface. It may
have no successors, but unfortunately this is not the rule. Other
patches, either in the vicinity, or on other parts of the sclera, pre-
sent themselves, and go through a similar evolution. There may be
intervals of weeks or months or perhaps years. A persistent spot
of chronic episcleritis, at the outer and inner portions of the pal-
pebral opening, is common among those who are greatly exposed
to wind and weather, like seamen and laborers, and especially in
later life. There will be decided thickening both of the conjunctiva
and sub-conjunctival tissue. The above conditions are compara-
tively harmless although annoying.
Treatment of episcleritis varies according to the conditions pre-
sented. There are mild cases of a fugacious character in which
almost no subjective symptoms exist, and the person is aware of
his disease only by seeing a bright pink area of redness over the
426
LWSEASMS OF THE EYE.
insertion of one of the recti. This passes off in a day or so with-
out treatment. In the pertinacious varieties there may be in the
beginning some pain and heat, when cold applications will be
chosen; we generally are called on to prescribe after this stage has,
passed. If the sclera alone is involved we may use atropia to allay
uneasiness and be certain that the iris escapes, and warm applica-
tions will be grateful. If there be pain not thus controlled, a com-
pressive bandage may give relief, and scarifications have been prac-
tised, in case there is local oedema and sensitiveness on pressure.
In the torpid state mild stimulation is applicable, by dusting calo-
mel on the spot, by friction with ointment of the yellow oxide of
mercury, gr. ij. ad 3 i., and by simple massage.
Sometimes iodide of potassium is appropriate, and other gene-
ral remedies to be spoken of presently, because this condition shades
into scleritis circumscripta, which is liable to be a diathetic disease.
Pterygium is frequently developed upon episcleritis.
Scleritis Diffusa Acuta.—In its acute form, including all the
anterior portion of the globe, this is a rare disease, and must not be
confounded with conjunctivitis or iritis. So rare is it that extreme
care must be taken to exclude both the above maladies, viz., con-
junctivitis by the absence of secretion, iritis by the clearness of the
aqueous humor and want of pupillary adhesions. It is eminently
of rheumatic or gouty origin; it is extremely painful. There will
be diffused bright pink injection, with only watery secretion, and
intolerance of light and little reduction of sight. It may be less
severe in attack and pursue a mild course during several weeks.
On the other hand, I have seen a case which seemed to originate in
scleritis and which developed acute inflammatory glaucoma. The
patient had gouty kidney as subsequently shown by autopsy.
Treatment will consist in using atropia as an anodyne several
times daily, cold water if agreeable, more often dry warmth is
preferred, or hot fomentations. To this add proper constitutional
treatment according to the diathesis. It is always needful to ex-
amine the urine for indications. For the rheumatic cases, Rochelle
salts or salicylate of sodium will be chosen; for gouty cases iodide
of potassium, or colchicum, or both combined. The general habit
of the patient, whether plethoric or anaemic, must always be taken
into account. A combination of conjunctivitis and scleritis, catar-
rho-rheumatic ophthalmia (see pp. 292-296) sometimes arises in
which pain is a conspicuous feature. Antifebrin, gr. v. every four
hours, or phenacetine, gr. v.-x. every four hours, or antipyrin,
gr. x. pro re nata, are welcome substitutes for ordinary anodynes.
Scleritis circumscriptaresemb]es in some degree episcleritis and
may result from it, yet a correct appreciation of its occasional re-
sults and complications makes its separate description appropriate.
THE SCLERA.
427
It is usually a chronic affection, and if while confined to the sclera
it is unpleasant and obstinate, nothing need be added to what has
been said above to afford a suitable idea of the measures proper to
its management.
Where, however, it is not limited exclusively to the sclera, a
more serious situation arises. It may extend to the cornea or to
the subjacent structures, viz., the ciliary body and iris, and either
without or with implication of them, it may bring about staphy-
lomata; in some instances after a protracted course in which the
sclera has become thin and bluish, the anterior part of the globe
becomes elongated to a pear shape, while the margin of the cornea,
from its haziness, can hardly be defined from the sclera, and perhaps
the whole of it has been rendered cloudy. One or more staphylo-
mata may arise in the ciliary region.
Sclero-keratitis denotes the invasion of the limbus or of the
sclera very near to it. A dense opacity creeps into the cornea ac-
companied by vascular injection of the sclera and sometimes too of
the corneal substance. There may or may not be thickening of the
sclera; the disease may remain on one spot or spread around the
limbus. The corneal opacity may be thick and yellowish and in-
delible (sclerosis) or it may be of the usual type and become fainter
or disappear. Iritis or cyclitis may or may not co-exist.
The process is apt to engage the iris, because of the high vascu-
larity of this region and the vicinity of the canal of Schlemm, and
so it readily reaches the ciliary bod3T. If this occur, the globe be-
gins to lose tension, pupillary adhesions appear, the vitreous begins
to be hazy. With advancing progress the phenomena of irido-
cyclitis become pronounced, there may be intra-ocular hemorrhages
and marked reduction of the globe and of sight, or with occlusion
of the pupil the opposite conditions of secondary glaucoma and in-
creased tension may develop.
Staphyloma of the sclera sometimes occurs as has been re-
marked, and it is difficult in old cases to decide whether the morbid
process has begun from without or from within. It may begin
from the sclera, but more frequently it begins from the ciliary
body and iris. By either method the shape of the eyeball may be
badly distorted. When the lesion concerns the region of the limbus
chiefly, the pear-shaped elongation occurs; when it attacks the
ciliary region there may be one or more decided bulgings of dark
blue color with a few large vessels running to them and their walls
are extremely thin. In some instances a row of them encircles the
globe in front of the tendons of the recti (intercalary staphylomata)
and the form reminds one of the seed capsule of the poppy. When
a single protuberance occurs, there may be considerable sight or
none at all. When there are many, there will be occluded pupil,
428
DISEASES OF THE EYE.
the lens may be opaque or shrivelled, the iris bulged forward to the
cornea, or the anterior chamber may be very deep and the cornea
thin and distended, and no vision.
The morbid conditions above alluded to occupy years in their
progress and are associated with some faults of nutrition, which it
is not always easy to appreciate. The anaemic, the gouty and
rheumatic, the chlorotic and scrofulous are the chief victims. Both
children and adults may be affected. One severe acute case re-
quired large doses of quinine to control profound malarial infection,
and to this was added salycilate of sodium. Obstinacy and long
duration are signal features.
Treatment will be both local and constitutional.
If the case be complicated with deeper troubles, as acute kera-
titis, and do not yield to atropine and hot fomentations and suitable
constitutional means, I have in a few instances practised iridectomy
with benefit. The case must, however, be urgent to warrant it.
When iritis appears, we employ atropine to secure mydriasis.
If any tendency to staphyloma occur, a pressure bandage may be
of some avail. Its development is due more to local weakness of
structure than to increase of intraocular tension, hence, iridectomy
is not indicated. For a quiescent and fully formed staphyloma its
excision may be practised instead of enucleation, and the edges of
the wound united by fine sutures. When there are many staphy-
lomata and the deformity is conspicuous, enucleation may be the
only resource. If, however, the distention affects the ciliary region
uniformly without exhibiting particular prominences, sclerotomy
may be tried and repeated. The cases referred to merge into those
known as hydrophthalmus, whose beginning is by internal inflam-
mation of the eye, and for which a large iridectomy is sometimes
useful. I have also brought about the diminution of the eye by
total avulsion of the iris. To do this a wound about six millimetres
long is made at the edge of the anterior chamber, a slender pair of
straight forceps with blades of unusual length is passed to the op-
posite side of the chamber and seizes the iris at its periphery.
Dragging gently and with a to-and-fro motion, it may yield with-
out laceration and gradually it tears away from its insertion and
is drawn out of the eye. Only a small number of cases will lend
themselves to this proceeding.
The use of a seton to induce suppuration of the globe is not to
be advised. Preferable to this is evisceration of the eye after re-
moving the cornea, or so much of the anterior part as may be need-
ful. Strict antiseptic treatment may prevent suppuration. The
contents of the sclera may be wiped out with a sponge. Reaction
will be greater after this proceeding than after enucleation, because
there is always some orbital cellulitis, as I have had occasion to
THE SCLERA.
429
notice. It is, however, most desirable to avoid enucleation if pos-
sible, because the tissues of the orbit have been reduced by pressure
of the enlarged globe and a yawning chasm is left which an artificial
eye cannot suitably fill. For young persons and females this con-
sideration is most urgent.
Constitutional treatment in cases with active symptoms is of
value. Recognizing the uncertainties of the choice of a proper
remedy, inquiry must be made into the diathesis and regard paid
to the general condition. With feeble subjects, tonics and quinine
and arsenic do good service. I have often relied on the last, and
find that Horner speaks well of it as having been advised by Crit-
chett. In scrofulous persons, cod-liver oil and iron will be given.
To the gouty a' careful regulation of diet and habits, the use of
baths, of mineral waters, especially of the lithia waters, and best,
taking them, if the season suit, at the springs where they flow; the
careful trial of iodide of potassium in small doses, the cautious use
of colchicum, are serviceable suggestions. Salicylate of sodium, and
alkalies like Rochelle salts or citrate of potassa may be given to
the rheumatic. I have known a gouty subject of delicate health
much benefited in the later stages of a protracted and distressing
attack by hypodermic injections of muriate of pilocarpine about
one-eighth of a grain. In all cases great care must be taken to
protect the eye from irritation by light and wind and dust and
over-work. The general hygiene must be cared for intelligently
and in detail. Change of climate and trial of various resorts will
often have to be summoned to our assistance.
CHAPTER VII.
THE IRIS.
The structure which we meet next in order is the iris. We
have to take up its consideration separately, but this does violence
to its anatomical and pathological relationships. It is continuous
with the ciliary body and choroid, which unite with it to form
the vascular tissue, called as a whole the uvea. Together they are
supplied by the same nerves, and morbid action often, if not gener-
ally, affects the whole to a greater or less degree, beginning at one
part and going from behind forward or beginning at another and
going from before backward. Among children, especially, does
this involvement of the whole uveal tract in inflammations, prevail.
The nutrition of the aqueous humor, of the crystalline lens, and
the vitreous depends upon the uvea; while we often find the ciliary
body affected immediately, and the vitreous remotely, by its in-
flammations. For the sake of convenience we are nevertheless
obliged to parcel out the structures, and discuss the iris and its
diseases as a separate topic.
Anatomy and Physiology.—It is a highly organized structure,
composed of muscular fibres, pigment, epithelium, connective tis-
sue, blood-vessels, lymphatics, and nerves of every type, and gan-
glia. It is a curtain whose periphery is attached to the sclera at the
edge of the endothelium of the cornea by the fibres of the ligamen-
tum pectinatum (pillars of the iris). This locality is the angle of
the anterior chamber and sometimes is called the angle of the iris.
The membrane is perforated by a round opening, the pupil, which
appears to be in the centre, but is really a little to the nasal side.
It rests upon the anterior capsule of the crystalline, over a large
area. Between it and the lens a circular space is formed, called the
posterior chamber. In section this has the general form of a tri-
angle, into whose base the ciliary processes project. In front is
the iris, behind is the lens-capsule and the suspensory ligament or
zonula of Zinn. Between the iris and the cornea is the anterior
chamber. The anterior and posterior chambers compose the aque-
ous chamber, and in its fluid the iris floats, giving the most perfect
chance for its muscular fibres to exert their force. They are flat,
THE IRIS.
431
arranged in bundles, and are of the unstriped variety. Certain
fibres are arranged in curves about the pupil, constituting the
sphincter, which is rather nearer the back than the front surface,
and can be readily recognized, while other fibres run in radii and
are more deeply situated, and have been called the dilator pupillae;
the muscular character of the latter is denied (Grunhagen, Evers-
busch, Fuchs).1 In favor of a dilating muscle, see a later article
by Ewing.2 The sphincter fibres join each other near the pupil
in curves or arcades which are often conspicuous. The sphincter is
under the control of the third cerebral nerve, the motor oculi. The
stroma of the iris consists of layers of connective tissue and blood-
vessels, amid which are found spindle and wandering cells and
fibres. Its cells do not contain pigment, and have been confounded
with the muscular fibres. On the front of the iris is a layer of
endothelium, whose edges overlap, and on the back of the iris is a
much thicker layer of endothelium deeply charged with pigment,
and called in a restricted sense the uvea. This word should, how-
ever, be reserved to describe the whole internal pigmentary struc-
ture of the globe, viz., iris, ciliary body, and choroid. Schwalbe
calls this layer the retinal part of the iris and divides it into two
layers. For details see Fuchs, 1. c. The front of the iris is checked
by numerous threads amd pits, and is, therefore, quite rough. Be-
neath the anterior endothelium is a limiting membrane, and numer-
ous blind openings or crypts (Fuchs) are scattered about, not lined
by endothelium, and they communicate with clefts which consti-
tute a system of spaces which surround the middle layer of vessels.
The brown pigment is scattered upon it irregularly and the differ-
ing hues of its surface are to be explained by referring them to
interference phenomena. (See Rood, "Modern Chromatics," pp. 55
and 58.) The pigment is of the same quality and quantity, no
matter what may be the effect of its distribution in causing the
iris to seem blue, brown, hazel, pied, etc.
When the pupil contracts the iris broadens and grows thinner,
its tissue stretches in a radiating manner, which produces diminu-
tion of the pupillary openings of the lymph spaces and simultaneous
expansion of the peripheral lymph openings. When the pupil dilates
the iris grows both narrower and thicker, its tissue is relaxed, the
lymph clefts near the pupil are enlarged, while the ciliary or
marginal openings of the lymph vessels and the crypts become
smaller. The method of dilatation is not fully understood; ascribed
by some to the reduced calibre of the vessels, this is denied by
Fuchs and assigned to the anterior limiting membrane, but with
hesitation. In the movements of the iris the anterior layers slide
1 Graefe's Arch. f. Ophth., Bd. xxxi., Abth. hi., S. 69.
* tirade's Areh. f. Oph., Bd. xxxiv., Abth. hi., S. 1, 1888.
432
DISEASES OF THE EYE.
over the posterior, and this is facilitated by the existence in its
middle of a loose tissue containing large spaces. Hence its perme-
able character and capacity for absorption.
Most of the prominent lines upon the front of the iris are blood-
vessels. They are extremely plenty, and their walls are thick.
The sensibility of the iris is acute, and the sensibility of the cornea
may be totally lost through paralysis of the fifth nerve, while the
iris twigs are unaffected, as I have seen demonstrated by iridec-
tomy in a case of trigeminal paralysis.
The iris is under the control of the ciliary (lenticular) ganglion
which lies upon the outer side of the optic nerve about three-
quarters of an inch behind the globe, and has three roots—the sensi-
tive from the ophthalmic branch of the fifth nerve, the motor from
the third, and the sympathetic from the carotid plexus. The effer-
ent twigs are numerous and fine and surround the optic nerve as
they pass to the posterior part of the sclera, and are called the
short ciliary. There are other nerves called the long ciliary which
have no connection with the ciliary (lenticular) ganglion, but pro-
ceed from the naso-ciliary branch of the ophthalmic (first division
of the trigeminus) as it crosses the optic nerve very near the canal.
They are usually two, rarely three in number. They must receive
twigs of sympathetic fibres from the carotid plexus. They enter
the sclera in company with the short ciliary nerves and go for-
ward to the ciliary body upon the horizontal meridian. They
seem to control the consensual contraction of the pupil—i.e. its
concurrence with the behavior of its fellow—also the contraction
due to convergence; and they confer sensibility upon the cornea
(Querenghi, in Knapp's Archives, XX., No. 3, p. 428). These func-
tions seem to be transmitted directly from the brain, and without
intervention of the ciliary ganglion.
The iris acts as a diaphragm to cut off the marginal rays of
the cornea and lens which could not be correctly focussed, and
the size of the aperture varies with the quantity of light. The
pupil serves to sharpen the image upon the retina, and it regulates
the quantity of light received. Its action is reflex, the afferent
nerve being the optic and the efferent nerve the motor oculi. The
cerebral centre is the corpora quadrigemina, and the third nerve,
just behind the centre for the ciliary muscle has a special nucleus for
the sphincter iridis (see p. 152, Fig. 72). See for an account of the
action of the pupils, " Die Entstehung der reflectorischen Pupillen-
Bewegung," Magnus, Breslau, 1889. Contraction of the pupil is an
active force, while dilatation occurs less promptly. The diameter,
with repose of accommodation, varies from 2.5 mm. to 5.8 (Woinow),
but the differences among individuals are very great. The pupil is
larger in children than in adults and in old age it becomes small.
THE IRIS.
433
The pupils of the two eyes act consensually. If one eye be shaded
and the other exposed to bright light, the pupil of the covered eye
acts in harmony with that of the other. Contraction of the pupil
occurs from stimulus of light, from convergence of the visual lines
and from efforts of accommodation.
The pupils, the accommodation, and the convergence of the
visual lines possess activities both in co-operation and separately,
which need to be clearly understood. Consensual contraction will
occur in a blind eye when light rails upon its seeing fellow. If both
eyes are blind and do not react to light, they will both contract
upon convergence of the visual lines. Efforts of accommodation
cause contraction in eyes insensitive to light; i.e. blind. Pupils
may contract sluggishly to light and actively to convergence. If
there be homonymous lateral hemianopsia, the pupils will contract if
light be thrown exclusively on the blind halves of the fields if the
lesion be behind the corpora quadrigemina, and will not contract
if the lesion be in front of or at these optic lobes (see p. 708).
The explanation of these facts is found in the anatomical ar-
rangements. The pupillary centres are linked with the centres for
convergence (see p. 154), and their influence comes down the oculo-
motor nerves; so with the accommodative centres. The light re-
flex goes up the optic nerve and by the anterior quadrigeminal
bodies is transferred to the oculo-motores. Still further1 it seems
established that the short ciliary nerves from the lenticular gan-
glion in the orbit control the accommodation, the pupillary con-
traction upon illumination, and the sensitiveness of the cornea to
pain, while the long ciliary nerves control the contraction of
the pupil to consensual action and upon convergence and give
the tactile sense to the cornea. Dilatation of the pupil, apart
from the cessation of contraction on suspension of the producing
cause, occurs from many causes, viz., irritations of the surface by
galvanism or tickling, psychical emotions, such as fright, with
deep inspirations and expirations, etc. Myopes often have large
pupils. On the other hand, severe irritation of the cornea, as by a
foreign body or a blow on the eye, causes strong and spasmodic
contraction which will often resist repeated use of atropia. The
passive state of the pupil is moderate contraction, and this we find
in sleep. In nervous and excitable persons the pupil is large.
The separation of the pupils from each other varies with age
and sex, and with the form of the face. In adults it has a average
of 58 mm. The interpupillary distance used by Nagel2 in his
tables, respecting the metric angle of convergence, varies from 50
to 75 mm., and he takes as a general basis of his calculations 63 mm.
1 See Querenghi quoted in Knapp's Archives for Ophth., July, 1891, p. 428.
*See G. and S., Bd. vi., 8. 481.
434
DISEASES OF THE EYE.
Functional Diseases of the Iris.
We have variations in size and activity of the pupil due to irri-
tating and paralytic causes and these may be either local or remote.
There may be permanent or temporary enlargement (mydriasis)
or diminution (myosis), or the conditions may alternate (hippus).
A strictly ocular affection will usually concern but one eye, while
it also happens that monocular mydriasis of moderate degree may
depend on incipient brain disease, which may be the precursor of
insanity—such is sometimes the fact in general paresis.
1. Mydriasis, dilatation of one pupil of slight amount with preser-
vation of its activity, is sometimes seen as an unimportant affec-
tion which may continue for years. Its cause is unknown, but is
probably local to the iris. 2. Large and permanent dilatation is
caused by sudden increase of intraocular pressure as in acute glau-
coma, and a similar result belongs to the advanced stage of chronic
glaucoma. 3. When the retina or optic nerve are so far diseased
as to greatly reduce the light sense, dilatation becomes permanent
and extreme. In glaucoma the mydriasis is due to impairment of
the motor fibres and reduction of the reflex sensibility; in amauro-
sis it is due to interruption of the reflex action. 4. Mydriasis is
sometimes the result of a blow on the globe. 5. Mydriasis ensues
sometimes after diphtheria and is a local paralysis, while more fre-
quently paralysis of the ciliary muscle and loss of accommodation
without dilatation of the pupil is the sequence. This will get well in
a few weeks and is benefited by electricity. 6. Paralysis of the
third nerve causes mydriasis which can be increased by atropia;
the cause may be orbital or intra-cranial. The anterior pair of
the corpora quadrigemina preside over the contraction of the
sphincter pupillae and between them and the origin of the third
nerve communicating fibres (Meynert's) pass, and here we have the
centres for pupillary contraction, for convergence, and for accom-
modation, close togther, but distinct (see p. 147 et seq.).
Mydriasis from irritation of the pupil-dilating centre, is moder-
ate in degree, is consistent with mobility of the pupil and occurs:
" a, in hyperaemia of the cervical portion of the spinal cord, and in
spinal meningitis; b, in the early stages of new growths in the
cervical portion of the cord; c, in cases of intra-cranial tumor and
other diseases causing high intra-cranial pressure, according to
Raehlmann, although Leeser points out that these may also give
rise to paralytic mydriasis; d, in the spinal irritation of chlorotic
or anaemic people, after severe illness, etc.; e, as a premonitory
sign of tabes dorsalis; /, in cases of intestinal worms and some-
THE IRIS.
435
times in other forms of intestinal irritation; g, in psychical excite-
ment, e.g., acute mania, melancholia, progressive paralysis of the
insane (often then unilateral with myosis in the other eye)"
(Swanzy). Irritation of the side of the neck will dilate the pupil.
2. Myosis.—Contraction of the pupil, as said already, may be
caused by a blow or by a foreign body on the cornea. When the
aqueous humor is evacuated the pupil contracts. Hyperaemia of
the iris causes contraction of the pupil. Myosis from irritation of
the cerebral centre is found in, " a, the early stages at least, of all
inflammatory affections of the brain and its meninges, in simple
tubercular and cerebro-spinal meningitis. When in these diseases
the medium myosis gives place to mydriasis, the change is a seri-
ous prognostic sign, indicating the stage of depression with paraly-
sis of the third nerve; b, in cerebral apoplexy the pupil is at first
contracted, according to Berthold, who points out that this con-
traction is a diagnostic sign between apoplexy and embolism, in
which latter the pupil is unaltered, c, in the early stages of intra-
cranial tumors situated at the origin of the third nerve or in its
course; d, at the beginning of an hysterical or of an epileptic at-
tack; e, in tobacco amblyopia, from stimulation, probably, of the
pupil-contracting centre by the nicotine; /, in persons following
certain trades as the result of long-maintained effort of accommo-
dation (watchmakers, jewellers, etc.) the pupil-contracting centre
being subject to an almost constant stimulus; g, as a reflex action
in ciliary neurosis; consequently in many diseased conditions of
those parts of the eye supplied by the fifth nerve " (Swanzy).
Paralytic myosis occurs in diseases of the spinal cord, i.e., its
cervical portion (cilio-spinal centre): e.g., injuries, and inflamma-
tions especially of the chronic form. Spinal* myosis appears under
two forms, one of which is simple, and the other is known as the
Argyll-Robertson pupil who first called attention to it, 1869. In
the simple form there is medium contraction, and the pupil reacts
both to light and to visual convergence. We find this in gray
degeneration of the posterior columns of the cord. The very
minute pupil sometimes seen in tabes dorsalis is probably due
to secondary contraction of the sphincter pupillae. The Robert-
son pupil is one which is contracted and responds very feebly
or not all to light; but actively responds to convergence. Put
the patient before a window and let him fix on a distant object,
like a tree or house. Shade his eyes with the hand. If the pupils
react little or not at all, bid him fix the finger at 10", and the result-
ing prompt contraction, whether the eyes be shaded or exposed,
1 " Eye Disturbances in Tabes Dorsalis," Schcueichler, Arch, fiir Ophthal.,
vol. xii., p. 333, 1883; Am. Ed.
436 DISEASES OF THE EYE.
indicates the symptom. That convergence and not accommodation
causes the myosis can be proved by putting on glasses, — 4. D if the
patient be emmetropic, and observing the pupillary effect as the
eyes fix with parallel axes and again fix at 10". The centre for con-
vergence is anterior to that for accommodation. (See Magnus,
"Anleitung zur Diagnosis von Storungen d. Central. Ophth. App.,
Breslau, 1892).
Sluggish reaction to light, with prompt response to convergence,
is due to extension of spinal disease to the communicating fibres
of Meynert from the tuoercula quadrigemina to the pupillary
nuclei of the third nerve, or to disease of fibres from the optic tract
to the pons Varolii. It is a symptom of locomotor ataxia and of
multiple sclerosis.
Lesions due to Disease of the Cervical Sympathetic (reference
has been made to this condition on p. 432).—Disease of the cervical
sympathetic is either irritative or destructive, and as its functions
are dependent on integrity of. the spinal cord, disease of the latter1
or of the vertebrae between the fourth cervical and the second dor-
sal (cilio-spinal centre of Budge) may cause symptoms closely re-
sembling those primary to the sympathetic. Reflex symptoms
from the brain of an emotional character simulate some of the
symptoms to be referred to, and we find them likewise to some
degree in progressive muscular atrophy.
The symptoms of irritation of the cervical sympathetic which
were conspicuously pointed out by Horner 2 (1873) are, dilation of
the pupil, widening of the palpebral fissure, slight protrusion of the
globe, pallor of the side of the face and head, with slight fall of
local temperature, sometimes increase of perspiration, and acceler-
ation of the heart's action. Usually the only symptoms are en-
larged pupil, pallor of face, and rapid pulse—the exophthalmus and
wider palpebral fissure are, however, added to the picture in most
of the cases noted by the ophthalmologist. Destructive disease
of the sympathetic occasions symptoms the converse of the above,
viz., marked contraction of the pupil, which irresponsive to
change of light or to irritation of the side of the neck, slightly in-
creased by accommodation, and resists the action of mydriatics.
The vessels of the iris, choroid, and retina disposed to be dilated,
the upper lid droops and the lower lid is slightly lifted, owing to
paresis of Miiller's muscle. Retraction of the eyeball is a late
symptom and not often noticed. The vessels of the face, conjunc-
tiva, nostril, ear, and scalp are dilated, giving a flush to half the
face and head, with sense of heat and rise of temperature by 1.5° F.
measured in the nasal or auditory meatus. This congestion has
1 Ross : " Diseases of the Nervous System," 2d ed., 1, 686-688.
2 Klin. Monatsblatter.
THE IRIS. 437
been known to last three years, in other cases for nine months,
followed by a return to normal conditions more or less perfect.
The contrast of the states above described has led to the division
of the disease into two stages, but in the condition of recovery the
half face may remain paler than the other side and be less sensitive
to changes of external temperature.
Increase of tears and perspiration is an inconstant symptom,
and the opposite states have been observed. Palpitation of the
heart is often an annoying symptom and may be associated with
a slow pulse, viz., to 74 or 66. At a later stage the pulse may rise,
but never higher than 88 (see Mobius' x case). Slight atrophy of
the face is a late occurrence, and glycosuria2 has been observed.
Causes are various, viz., tumors pressing on the sympathetic
cord or ganglia, such as enlarged glands, aneurisms, or abscess;
cicatrices of old wounds; extension of inflammation from the apex
of the pleura in phthisis or chronic pleurisy; injuries and wounds
in the neck,3 and, as above said, any lesion of this region of the
spinal cord or vertebrae calculated to provoke irritation of the
sympathetic.
Pathology.—In the very few cases where an autopsy has been
made there has been found (1) parenchymatous inflammation of the
cells of the ganglia, attended by swelling, loss of nuclei, granular
and fatty degeneration and by atrophy, together with degenera-
tion of the fibres issuing from the cells. (2) A sclerotic process in
the connective tissue in and about the ganglia and in the nerves,
resulting in such an increase in the interstitial tissue as to com-
press and injure the cells and axis cylinders. These may be ob-
served in the later stages of the diseases. (3) In a number of cases
the vessels within and about the ganglia have been found dilated,
tortuous, and varicose, and hemorrhages from them are not
rare.
Prognosis depends on the cause and upon the estimated
amount of injury to the nerve or its ganglia.
Treatment—Removal of any curable causes, as tumors, etc.,
attention to general health; sources of reflex irritation are to be
eliminated. If the nerve has been severed its ends may be united
by a stitch, although there are no precedents. Electricity and
galvanism have been tried and abandoned as useless by all good
authorities.
The above account has been abridged from an article by Dr.
1 Berlin. Klin. Wochen., 1884, No. 16.
* Gerhardt: Volkmann's Sammlung. Klin. Vortrage, No. 209, " Ueber
Angioneurosen," p. 11.
3 Hutchinson : Ophth. Hosp. Reports, v., 138, 1865.
438
DISEASES OF THE EYE.
M. A. Starr, on vaso-motor and trophic neuroses, and his refer-
ences are as follows:
Ogle, Med. Chirurg. Trans., xli., 397-440, 1858, 27 cases; Porteau, " Le
Nerf Sympathetique," These de Paris, 1869,19 cases; Eulenbergand Gutmann
"Die Pathologie der Sympathies," 1873; Nicati, "Le Paralysie du Nerf
Svmpathique Cervicale," 1873, 25 cases; Seeligmuller, Inaug. Dissert., 18-J;
Mitchell, "Injuries of Nerves;" MObius, "Pathology der Sympathicus,
Berlin. Klin. Wochen., 1884, Nos. 15-19.
Paralytic myosis is found in general paralysis of the insane.
Myosis also occurs from paralysis of the cervical sympathetic. If
it be divided (Claude-Bernard), the pupil contracts, the eyeball is
retracted, the palpebral opening becomes smaller, the temporal
artery is dilated, the corresponding half of the face becomes con-
gested, warm, and moist with perspiration. Similar results have
followed cases of injury.1 Horner has called attention to this con-
dition occurring spontaneously. Willbrand published a case where
such symptoms were caused by the pressure of a lymphatic gland
in the neck.2 The pupillary fibres of the sympathetic leave the
cord at the upper dorsal and lower cervical vertebrae and going
through the superior cervical ganglion enter the carotid plexus;
they then pass through the ciliary ganglion in the orbit; but not
the whole of the fibres take this course to the eye, because it is
found that if the ciliary ganglion is extirpated, irritation of the
trunk of the sympathetic will dilate the pupil. The ophthalmic
branch of the fifth nerve, and probably other nerves also furnish
channels of access for sympathetic fibres (Hensen and Volckers).
3. Hippus.—Alternate contraction and dilatation of the pupil
occurs sometimes in cases of nystagmus, but as a symptom of dis-
ease of the nervous system it has been seen in multiple sclerosis,
after epileptic attacks, in hysterical spasms and in nervous persons,
and the oscillations persist under exposure to bright light. During
the phenomena of the Cheyne-Stokes respiration something similar,
but less intense, is observed; at the beginning of the cessation of
breathing the pupil contracts and ceases to respond to light, at the
first movement of respiration or a little before, the pupil will dilate.
Rhythmical contractions have been seen in typhoid fever during
the stage of brain trouble.
Treatment of the above conditions requires but few words. Sel-
dom will any measures be required, because the constitutional mal-
ady will be the object of attack. For mydriasis of a purely local
character, which is very rare, weak eserine solutions may be given,
1 See Archives G6n6rale de Medecine, tome xiv., p. 286,1869, also Hutchin-
son, Ophth. Hospital Reports, vol. v., p. 138, 1865.
* Graefe's Archiv f. Ophth., Bd. 1., S. 319, 1854.
THE IRIS.
439
but what is to be said at this point will be deferred to the section
on paralysis of accommodation.
Irido-donesis or Tremulous Iris.—A partial or total dislocation
or absence of the lens, will permit the iris to oscillate in sudden
waves. It is sometimes seen in hydrophthalmus. It depends upon
want of support, and may or may not be accompanied by fluidity
of the vitreous. Nothing is to be done for it.
Regarding the effect of medicines on the iris, see pp. 218, 221,
Congenital Defects of the Iris.
1. The want of pigment which characterizes albinoes gives to
the iris a silvery whiteness seen in white rabbits or a tinge of red
or a reddish-brown color when there is not total lack of pigment.
2. The iris is sometimes entirely absent, a very rare defect,
irideremia.
3. Coloboma, partial or complete and affecting either one or
both eyes, is not very uncommon. It is almost always vertically
downward, in rare cases it is lateral. The iris alone may be divided
Fig. 153. Fig. 154.
or the fissure may also run through to the choroid. In other re-
spects the eye is apt to be imperfectly developed. It may be too
small (mi cr ophthal mus), there may be partial cataract, generally
vision is amblyopic. The pupil remains contractile.
4. Membrana pupillaris perseverans describes what usually
consists in the presence of one or more threads running across the
pupil, attached not precisely on its edge, but a little outside of it,
and which is the remnant of the membrane which during foetal
development is differentiated into the choroid behind and the mem-
brana pupillaris in front. Various kinds of this defect may occur,
and they have been well indicated by Dr. Collinsx in diagrammatic
sketches. In certain types there is adhesion to the lens capsule as I
1 Royal London Ophthalmic Hospital Reports, vol. xii., July, 1885, p. 195.
440 DISEASES OF THE EYE.
have seen and sometimes this might be mistaken for inflammatory
adhesion. See case by Seggel, Klin. Monatsblatter, Aug., 1890.
Polycoria is a rare anomaly indicating a multiplicity of pupils.
The supernumerary pupil may be close to the normal pupil or at the
periphery. The congenital defect must not be confounded with
perforations due to atrophy. See Schweinitz, Trans. Am. Oph.
Soc, 1891, p. 59.
Inflammation of the Iris.
The signs of iritis are objective and subjective. The former are
found in change of color and texture of the membrane, and in the
abnormal behavior of the pupil, and in the usual tokens of injected
blood-vessels and external effusions. Change of color of the iris
in the early stage will consist in loss of the brilliancy of its surface,
the tracery and pattern are blurred, the tone becomes darker,
and with increase in the inflammation a marked difference ap-
pears between the two eyes, if one remains unaffected. If both
are implicated a blue iris will change to a dull gray, a hazel to a
dirty brown, and in advanced cases a greenish hue is often seen.
The tissue is swollen and infiltrated, under a lens or by the naked
eve hemorrhages may be seen, and spots or even masses of exuda-
tion. The aqueous humor is turbid and this is best seen by noting
that the pupil is smoky instead of a clear jet black; condensed light
will bring out this fact. The pupil will be small, it will not respond
to variations of light. Adhesions or deposits of pigment may be
seen at its edge, and their formation (posterior synechia) takes
place at a very early period.
If not readily discernible, adhesions will become apparent upon
dropping a solution of atropia into the eye. They are the decisive
and most constant indication of iritis. The smallness of the pupil
results from the swelling which pushes the membrane into the
space which is free for it to occupy, and hence both the earliness
and the constancy of adhesions to the lens.
The cornea may be clear, and its reflex bright, but in bad cases
it becomes dull, its surface steamy, and upon its deep layer pig-
ment deposits will be seen. The turbidity of the aqueous humor
sometimes gives rise to yellow precipitate in the anterior chamber
(hypopyum). The globe is painful to the touch, its tension may be
normal or sometimes increased; a state of minus tension indicates
the complication with cyclitis and belongs especially to chronic
cases.
We have also hyperaemia of the ciliary vessels; in the mild cases
or as the disease abates, there will be a corona, limited to the vicin-
ity of the cornea, composed of pink, fine, nearly straight vessels
THE IRIS.
441
radiating outward, and forming a zone about six millimetres wide,
with prolongations running out toward the recti muscles (see Fig.
155). This is sometimes called the iritic zone. Let the disease be
severe, and the conjunctival as well as deeper vessels will be en-
gorged, the whole front of the eye will
be intensely red, there may even be a
little sub-conjunctival effusion. The
secretion will be lachrymal, not mucoi-
dal nor purulent, the eyelashes will not
be stuck together, the lids will be im-
perfectly opened.
Subjective symptoms will be, im-
pairment of sight, which occurs very
early, and often becomes extreme, in-
tolerance of light, and pain. The pain
is a conspicuous feature from the outset. Situated first in the globe,
it soon radiates along the branches of the fifth nerve, chiefly the
supra-orbital and malar. Tender points will be at the supra-or-
bital notch and at the vertex. It spreads sometimes to the side of
the nose and the inner side of the orbit, but the usual complaint
is of the forehead and top of the head and the temple; the pain is
most severe toward night, or early morning, and often robs the
patient of sleep.
Adhesion of the pupil has been said to occur at an early period,
and the reason is that the aqueous quickly is saturated with plastic
material, and readily glues together surfaces which are already in
contact (Arlt). If atropia is used very early, these adhesions, being
soft, will give way in part or wholly. If the pupil should become
Fig. 155.
Fig. 156.
round and fully expanded, it may again acquire adhesions in its en-
larged condition. If the adhesions only yield in part, the pupil
acquires most irregular shapes according to the number and
breadth of the attachments. It will be festooned in various ways
(see Fig. 156). The plastic exudation becomes firmer and better
organized as the disease advances. If treatment is inefficient, the
442
DISEASES OF THE EYE.
pupillary space becomes choked (occlusion of pupil), the edge be-
comes wholly adherent and communication between the anterior
and posterior chambers will be cut off (exclusion of the pupil).
Iritis may attack one eye, or both, and either simultaneously or
successively. It may be so slight as to pass in a few days, it gen-
erally lasts two to six weeks, if neglected it may continue for
months, and the end be atrophy of the globe and hopeless loss of
sight. It is not a disease to be treated expectantly, its tendencies
are toward mischief.
Pathologically we have several varieties, viz., serous, spongy,
plastic or gummy, and suppurative iritis.
Iritis serosa was formerly called Descemitis, aquo-capsulitis,
etc. It is often inconspicuous in symptoms; there will be slight
circum-corneal hyperaemia, vision will be reduced but perhaps not
seriously, the eye will feel uncomfortable. By close inspection the
posterior surface of the cornea is found dotted with specks of pig-
ment, especially upon its lower third, the aqueous is cloudy and
may be in large quantity, the iris is pushed backward and dull, the
pupil acts sluggishly and may have some synechiae. Iritis with
abundant serous effusion, as shown by the fulness of the anterior
chamber, may be an acute and painful disease, while the condition
now mentioned is a chronic affection, with inconspicuous symp-
toms, which may last for we^ks. Dr. Knies in 18T9 reported an
autopsy of such a case and found that the whole uveal tract was in-
volved, as well as the sheath of the optic nerve up to the chiasm.
The optic nerve was inflamed and also the retina, for a small area
around the papilla. The deep part of the vitreous was liquefied
and detached; its anterior part permeated by granular cells and
membranes. The disease, therefore, must be generalized, as much
more than iritis, and might perhaps be called uveitis serosa or
uveitis anterior (see p. 407), cyclitis serosa.
The changes which iritis produces in the aqueous humor have
been referred to, especially that it becomes highly albuminous or
fibrinous. It seems likely that its chemical alteration has some
effect in dissolving the pigment of the iris and thus favors its de-
posit upon the back of the cornea. The turbidity of the aqueous
arises from the presence of lymphoid cells, pus, and red blood-cor-
puscles. They become disorganized into molecules, and may pre-
cipitate as hypopyum. They form groups and masses deposited on
the cornea and they will be felted in a wel> of fine fibrillae.
The most marked illustration of turbidity of the aqueous ap-
pears in a condition which has been called spongy iritis (Knapp).
I have seen it a few times. The iris is pushed far back, is very
much obscured by the muddy aqueous. As the fluid grows clear,
beginning at the edge of the anterior chamber, the material seems
THE IRIS.
443
to have a semi-solid form and has been mistaken for a dislocated
lens. It may seem to come from the iris like a cyst; usually it is
absorbed completely, shrinking to a thin membrane covering the
pupil and finally disappears without synechia. Arlt has examined
the material and describes it as sero-fibrinous and hemorrhagic, in
which the fluid and cell elements tend to separate, and the fluid
also separates into a network of very fine fibres like the exudation
of croupous pneumonia, and a formless gelatinous mass.
While plastic exudation is the rule in iritis, we have a type in
which yellow nodules and masses, one or many, project from the
surface of the iris, and are called gummata or condylomata, a name
given because of their very generally syphilitic origin. They are
a brownish-yellow, or streaked with blood, or may form a deposit
at the bottom of the anterior chamber. When absorbed they
leave a grayish discolored spot where the iris will be adherent and
atrophied; sometimes such changes are very extensive. The same
deposits occur in the ciliary body and choroid and a large mass
may soften the sclera and project in a considerable tumor. Similar
nodules are occasionally seen in rheumatic forms, and the resem-
blance to tubercular deposits must not be forgotten (see below).
Suppurative iritis is generally the effect of wounds or of opera-
tions, or of infectious diseases. It is usually only a part of general
suppuration and need not be specially described.
A most insidious and rather rare form of iritis occurs with
almost no signs, either subjective or objective, save dimness of
sight and posterior synechias revealed by atropia. I have seen it,
and Mr. Hutchinson describes cases, Trans. Oph. Soc. United King.,
Vol. VIII., pp. 95, 117, under the name of quiet iritis.
Complications.—Supposing the disease to have apparently
started in the iris, we have already referred to the possibility of
faint haziness of the cornea and to the deposits upon its posterior
surface. It is proper to emphasize the intimate connection which
subsists between all parts of the uveal tract, and that the iris is
often only the middle factor in a morbid process which may reach
in both directions, forward or backward. The iris is acted upon by
inflammations of the cornea and sclera, as well as by inflamma-
tions of the choroid and ciliary body. On the other hand, one must
be vigilant to note when iritis passes into irido-cyclitis or irido-
choroiditis. There will always be hyperaemia and some tissue
changes of the deeper uveal structures, but lesions which demand
attention often appear and will be referred to later.
It has been asserted that during iritis the retina and optic nerve
may be inflamed without concomitance of the ciliary body and cho-
roid (Schnabel), that a gray or yellowish infiltration may appear in
the retina near the optic disc, that the latter may be hyperaemic and
444 DISEASES OF THE EYE.
swollen, and that this condition may last longer than the attack of
iritis, and account for persistent dimness of sight. It is difficult to
decide the existence of such lesions while the media are hazy, and
it is true that the disc if discernible will sometimes be red, and at
other times free from hyperaemia; careful observation has enabled
me to note this difference. When, however, the media are clear
enough to permit accurate inspection by the ophthalmoscope, one
may sometimes find co-existent retinitis and neuritis. Such a case
with plastic exudation into the papilla I clearly remember.
The haziness of the vitreous is occasioned by the presence of
minute particles and threads and perhaps membranes. It may be
partially or even wholly liquefied. In chronic cases it undergoes
serious disorganization, which will be hereafter referred to and
which remains. Much of the alteration caused by acute attacks
will pass away.
Sequelae of iritis are: 1st. Adhesions of the pupil to the capsule
of the lens, and if they have been torn oy atropia, pigmentary spots
remain. 2d. The exudation may become organized into a membrane
filling the whole pupil, and it is sometimes vascular. If it be very
dense it will be indistinguishable from cataract and has been called
spurious cataract (see Fig. 157). The pupil is always small in such
circumstances. The complete adhesion of the pupillary border
Fig. 157. Fig. 158.
forming an annular synechia is called exclusion of the pupil. If, in
addition, the pupillary area is occupied by a membrane we have
occlusion of the pupil. 3d. In consequence of the conditions just
mentioned, the posterior chamber sometimes becomes distended
with fluid and the iris bulges forward, while the pupillary edge is
drawn deeply backward, and the periphery pushed against the cor-
nea (see Fig. 158). If at certain parts the iris is more adherent or
atrophied, its surface will project in cyst-like forms. It may even'
be pushed forward so far as to be in almost complete contact with
the cornea. The eyeball will be hard and we shall have the con-
ditions known as secondary glaucoma. 4th. The posterior surface
of the iris may be more or less completely glued to the lens, taking
the contour of its surface, and its periphery may be deeply retracted
THE IRIS.
445
forming a circular furrow, which indicates that it has acquired
adhesions to the ciliary processes. The tension is apt to be reduced.
The evil effects of pupillary adhesions are proportionate to their
extent, apart from obstruction or opacity. Broad adhesions which
may not be complete, sometimes occasion relapses of inflammation,
but this tendency is not so great as was at one time supposed.
They render the eye irritable and may require an operation, iridec-
tomy, but not until the necessity for it has by experience been
demonstrated. A few small adhesions will not occasion trouble in
most instances. Much depends on idiosyncrasy. If, however, the
pupil is extensively or wholly adherent, the above-named results
become very threatening, if not destructive to sight, and demand
interference.
5th. The capsule of the lens may become thickened by prolifera-
tion of its epithelium, for a space corresponding to the pupil.
Deeper and more serious lesions of the lens occur in chronic and
complicated cases, causing so-called inflammatory cataract. The
lens is densely white or yellowish, may be chalky, is small, the cap-
sule shrivelled and very thick and the iris will be atrophied. Atro-
phy of its tissue occurs in many chronic cases, and is recognized by
extreme thinness of its structure, often in patches, which will seem
to be frayed out and gray and permit light to pass through when
viewed by the ophthalmoscope or under oblique illumination.
Causes of iritis are local and constitutional. It may come from
functional strain, from injury, from operations, from penetration of
foreign bodies, by extension from adjacent structures,viz., the cornea,
the choroid and ciliary body, from a swollen lens, detachment of the
retina, etc., and we have it also by communication from the oppo-
site eye as sympathetic ophthalmia. More frequently it is due to
constitutional causes, especially syphilis, both secondary, tertiary,
and hereditary, and sometimes intra-uterine. Rheumatism and
gout are next in potency as causes, and the attacks which they
cause are obstinate, painful, and recurrent. Syphilitic iritis is of the
plastic tendency, rheumatic and gouty are more serous. Gonor-
rhoea occasionally causes iritis. The arthritis of gonorrhoea is due
to the presence of the gonococci (diplococci of Neisser) in the joints,
and the same explanation is probable for iritis. Other causes are,
malaria, febris recurrens, variola, scrofula, tuberculosis, diabetes
mellitus, and conditions which are unknown. Syphilis stands for
the cause in about 60$ of all cases (Alexander).
It is impossible by the quality of the inflammation to declare
what may be the constitutional cause, except in a restricted sense,
as for example, that gummy exudation is almost certain to be
syphilitic; etiological deductions must come from inquiry into gen-
eral symptoms.
446
DISEASES OF THE EYE.
Prognosis.—The duration will depend not only on the severity
and cause of the attack and upon its complications, but very largely
upon the nature of the treatment. If the pupil is freed from adhe-
sions at an early time or these are few, this is a most favorable
circumstance, if not, the duration will be long and the result more
or less damaging to sight. The rheumatic and gout}- types, while
less likely than some others to inflict harm to vision, are apt to be
tedious and painful. The duration and injury belonging to syph-
ilitic forms, are in proportion to the quantity of exudation and to
the complications with deeper parts. Seldom will an attack yield
in less than two weeks and it may go to eight weeks or in case of
complications to many months. In simple cases vision will be per-
fectly restored.
Treatment—A patient must be kept in more or less seclusion
in a room moderately dark and avoid use of his eyes and all sources
of irritation. In some severe cases he will be confined to bed. It
is true that patients, especially in dispensary practice, go out of
doors and sometimes do not interrupt their avocations, but the
effect is unfavorable, even though the sore eye be bandaged or
shaded.
The essential and master remedy in iritis is sol. atropiae sul-
phatis, from the beginning onward to the end. The prevailing fault
is to use it with too much caution. Its potency when the iris is in-
flamed is far less than when the eye is normal; the reasons are as
follows: The activity of endosmosis through the cornea is impaired
because its tissue is surcharged with fluid, and the tension of the
globe is increased. The swollen condition of the iris, the inaptitude
of its muscular fibres to contract, the hyperaemia and the adhesions
combine to oppose its effect, even when the solution has entered the
aqueous chamber. For these reasons, a solution, gr. iv. ad ? i.,
must be used in such frequency as will effect the purpose. This
will vary in different cases. It will be dropped in four to six times
daily, or once in two hours; or it may be put in six times an hour
three times daily, or four times an hour three times daily. For
iritis after extraction of cataract, I use a solution, sixteen grains to
the ounce—the condition not permitting frequent instillations, and
for this reason the strength is increased. So long as certain per-
nicious effects presently to be described do not occur, the effort to
dilate the pupil is to be perseveringly pushed until it is actually
accomplished. But there are certain possibilities of harm in atro-
pia, not to be overlooked: viz., its poisonous constitutional effects.
Patients may quickly complain of dryness of the throat, and it will
be seen to be red and the saliva scanty—this is not to be heeded as
dangerous; but when a flushed face, a quick and feeble pulse,
nausea, prostration, and fainting appear, and when, as sometimes
THE IRIS.
447
occurs, delirium, at first talkative, afterward with delusions and
violence, shows itself, the situation is sufficiently alarming. Some
persons are specially susceptible, and are disturbed by small quan-
tities. When such signs arise, the atropia must be stopped, alco-
holic stimulants given, and, if violent delirium exist, hypodermic
injections of sulphate of morphia, gr. 4; to \, repeated as needful.
To prevent poisonous symptoms, care should be taken to drop the
solution into the outer rather than into the inner angle, and hold
the head so that the fluid does not readily flow toward the puncta;
pressing with the finger over the puncta and sac is of service to
hinder passage of the solution into the throat. Another, but less
frequent and less serious effect of prolonged use of atropia, is that
it causes a form of granular conjunctivitis. It is now believed
that the presence of germs in the solution accounts for this
effect. Cohn, Hirschberg, Franke, and Ruppell all concur on
this point. It is better to sterilize the solution by boiling, and to
change frequently. Atropia may be mixed with vaseline and
so employed. If atropia must be abandoned, we have a sub-
stitute in duboisia, to be given in the same strength of solu-
tion, or hyoscyamine; but both these are in a degree liable to
cause like constitutional effects. Usually all mydriatics must be
renounced until the toxic symptoms subside, and then resumed in
such degree as may be tolerated. One need not expect the full
effect on the pupil at the beginning, and if there be great hyper-
aemia, the use of leeches to the temple will promote its absorption; if
the anterior chamber be deep and the eye tense, paracentesis will
greatly aid its effect. It is a common experience that as soon as
the pupil enlarges to a considerable degree, say to about six or
eight millimetres, the symptoms speedily give way and recovery
sets in. This will take place even though some adhesions remain.
Yet in rheumatic iritis this happy sequence does not always ap-
pear. The aqueous remains turbid and in large quantity, and pain
continues. It will be well to apply two leeches to the temple,
and paracentesis may be admissible. It is also efficient to give a
hypodermic injection of morphia. Another remedy to aid in bring-
ing about dilation is the hypodermic injection of muriate of pilo-
carpine, gr. \ or -fa (Schweigger). Iritis may occur in persons
of gouty diathesis as the first token of their constitutional
tendency. If it appear when there is great depreciation of health,
the disease may not be violent in intensity, but is likely to be most
pertinacious in duration, and aggravating in its ups and downs.
Nothing but general hygienic measures will in some cases be of any
value—except, always, atropine.
An additional application is warm water, and the temperature
such as the patient prefers. Sometimes for suppurative iritis, es-
44. Whether
the cosmetic results thus obtained were satisfactory or not is ques-
tionable, but this seems to have been the case with twelve of the
patients. Four other cases were not followed up, and it is not
known whether enucleation was necessary at a later period. In 16
cases subsequent enucleation was inevitable = lOf 0r mild inunction, subordinating the specific to the generally
invigorating methods.
Similar remarks apply to the forms of general choroidal atrophy.
We can afford little aid and must simply caution the patients
584
DISEASES OF THE EYE.
against excessive eye work, and do everything to improve the
health. Iron and cod-liver oil, extract of malt, good food, etc., are
our reliance in the simple wasting forms, senile and otherwise.
Sometimes a sort of marasmus coexists and several times I have
known such patients to become insane, as if the wasting of the
choroid was but the precursor of similar impoverishment of the brain.
The choroidal and other changes which attend upon some cases
of high myopia, likewise require hygienic rather than therapeutic
measures. To these, attention has already been directed.
Choroiditis Metastatic a.
Under this title we have a disease affecting retina, choroid, iris,
etc., consecutive to some other and generally distant malady, such
as cerebro-spinal meningitis, puerperal fever, febris recurrens, ty-
phoid fever, severe scarlatina, erysipelas, mumps, caries of cranial
bones, surgical operations, septic or ulcerative endocarditis, etc.
It usually involves the iris, and sometimes shows hyperemia of
the ciliary region. Frequently no outward sign is seen, and at-
tention is attracted either by accidental discoverv of blindness or
by the white or yellow reflex perceived through the pupil. The
general state of the patient is often so critical that this occur-
rence is overlooked. The eye is apt to be a little soft, not tender,
and care must be taken not to confound the disease with glioma.
Sometimes the whitish mass covers the fundus in a thick layer,
sometimes it fills almost the whole vitreous. It may have blood-
vessels upon its surface. A precisely similar condition may spring
from injury of the floor of the orbit as I have once seen.
In all cases the disease is of embolic origin and in many in-
stances the vessels have been found plugged with micro-organisms.
A more severe type may arise in which putrid infarctions, that is
germs, may excite violent inflammatory reaction, amounting to
general suppuration of the globe, with attending inflammation
in the orbit. Left to itself pus may find its way out of the eye, as
in any case of suppuration, and shrinking of the globe ensue.
Pathological investigation of these cases has settled unques-
tionably their infectious nature by the presence of germs in the
vessels, and been a strong argument in proving the doctrine now-
held on this subject. We need not go into details.
Suppurative choroiditis comes more frequently from local
causes than from constitutional, viz., from wounds, from foreign
bodies, after operations, etc. As a matter of fact we know that
infection is in most cases added to traumatism and we need not
dwell upon the clinical features in detail. We generallv have
panophthalmitis with less or more implication of the orbital tissues
CHOROIDEA.
585
Diagnosis.—Leaving out of view the severely acute cases which
eventuate in general suppuration and finally in phthisis bulbi, it is
proper to call attention to the milder and so-called benign cases,
where there is no great reaction and in which the appearance of
the eye is little changed. The discrimination between these cases
and tumors, especially glioma, is very important. So far as the
appearance of the intraocular mass goes, it is often absolutely im-
possible to rely upon any points of distinction. Color, tension,
presence of vessels, situation, extent afford no certain criteria.
The main reliance is the previous history; whether there has
been a fever, or cerebro-spinal meningitis, rheumatism, or any of
the conditions which can produce metastatic effects. On this point
the inquiry must be searching and minute. Some light is thrown
on the diagnosis of metastatic choroiditis by the presence of iritic
adhesions, which are more likely to exist in pseudo-glioma than in
true glioma; there will be perhaps discoloration, or, perhaps, atro-
phy of the iris; the tension is likely to be minus: there may be
tenderness on pressure. A careful estimate of all the symptoms
and of the history is the best guide. (See p. 516.)
Treatment—Considering now onh7 the milder cases, it maybe
said that only palliative treatment is to be adopted, because as a
rule, when examined the case has reached an incurable condition and
the eye is innocuous. Under some circumstances we are compelled to
decide upon the necessity for enucleation; first, because the history
and the diagnosis may be so obscure that serious doubt remains as
to the existence of glioma or other tumor—then, of course, one
would not hesitate to remove a useless eye rather than run the risk
of extension of the disease and death: secondly, the eye maybe
irritable, be tender on pressure, disposed to recurrent inflamma-
tions and to atroplry; in such conditions we would enucleate it.
Panophthalmitis Suppurativa.
It will be convenient at this point to consider this condition,
notwithstanding it originates from many other causes than from
primary choroiditis. We need give little heed to the multiform
causes and may confine our attention to symptoms, diagnosis, and
treatment.
Symptoms.—If the starting-point be from without, as after
lesion of the cornea, this tissue will present suppuration and ulcera-
tion ; there will be chemosis, swelling of the lids, and as the process
advances, exophthalmus, and great tension of the globe and of the
circumocular tissues. On the other hand, if the lesion starts from
within, as because of a penetrating foreign body, the cornea may
for some time remain clear and the pupil movable. The iris and
586
DISEASES OF THE EYE.
aqueous humor will be muddy, some chemosis will soon show itself,
and presently a yellowish reflex will be caught deep in the eye.
Sometimes the eyeball is soft instead of hard even during the early
stages of the malady; a fact explained by the special implication
of the ciliary body. Usually with its progress infiltration of the
cornea ensues and the complete picture of suppurative panophthal-
mitis is seen. Sight is lost at a very early date, and pain from the
beginning is distressing.
Diagnosis is almost self-evident. We have great reaction and
swelling in gonorrheal conjunctivitis, in acute glaucoma, and in
the very rare cases of thrombosis of the cavernous sinus. In
the first disease the abundant purulent secretion and the mode
of onset declare its character, which may in the end develop into
panophthalmitis. In the second disease we have what may be
called acute serous panophthalmitis, and the watery effusions with
moderate or no secretion, except tears, are sufficient distinction.
The last disease is rare and the eyeball is not primarily implicated,
although there may be exophthalmus and great oedema, and it may
be left out of view. The origin of the attack, whether from without
or from within, whether spontaneous or traumatic, the formation of
pus in the interior, or on the cornea, and the evident invasion of the
deep structures in an acute process, adequately declare the true
nature of the disease.
Treatment.—Efforts to save the sight never succeed; we may
check the severity of the attack, yet to do this operative interfer-
ence may be required. The severe pain calls for anodynes and
constant application of cloths taken from a block of ice; a piece of
ice resting on the eye is not well borne. Leeches to the temple are
sometimes useful in robust subjects, but their value is limited to
the early stage. The main reliance is on persistent use of cold ap-
plications night and day during the very early and progressive
period and on anodynes, especially hypodermic injections of mor-
phia in the temple. The degree of relief and the instincts of the
patient will dictate the persistence of the application. But very
often hot applications are more grateful from the outset and soon
under any circumstances the preference for hot fomentations will
be very decided. Use absorbent cotton soaked in hot solution of
corrosive sublimate, 1-3,000. If tension is very great, we may
make an incision across the front of the globe and let out the lens
and infiltrated vitreous. Notwithstanding the great infiltration of
the orbit and lids and the exophthalmus, enucleation may be the
only effective resource. The operation is difficult, is attended with
much bleeding and may be severe in its effects. Some cases are on
record in which death has resulted. Prof. Graefe, of Halle and
Dr. Bunge have dwelt on the danger of a fatal issue, and have pro-
CHOROIDEA.
587
posed to substitute evisceration of the eye. In fact, they employ
this proceeding in many instances which are not suppurative. The
risk of death is not great, yet it is enough to give us pause when
we have a feeble and especially also an old subject. We may then
resort to the less radical method of emptying the sclera of its con-
tents. This may be done after opening the globe by wiping out the
cavity with absorbent cotton and forceps, and washing the interior
with corrosive sublimate solution, 1 to 3,000. (See page 544, where
the subject has been considered.)
Laceration of the Choroid.
Injuries, such as blows, which make no external mark and seem
not to have caused serious mischief, sometimes cause laceration of
the choroid. The place of its occurrence may be near the equator,
but is much oftener about the posterior pole. It may surround
the optic nerve in a crescentic form; it may be at the macula, and
be a straight rent. There may be two or more, concentric with each
other. Usually the retinal vessels are not torn, even if they tra-
verse the fissure. There is hemorrhage about the spot, and vision
is affected, of course most seriously when the rupture is near the
macula. When the blood begins to be absorbed the sclera appears,
and after a time an intense pigmentation occurs in and about the
tear. I have a drawing of a case of long standing in which the
deposit of pigment is extraordinary, and the rupture is not less than
ten discs in length.
There may, of course, be other injury to the eye, viz., blood in
the vitreous, luxation of the lens, etc. With laceration near the
macula, metamorphopsia may be produced. Sometimes vision im-
proves and healing of the rupture has been ophthalmoscopically
observed. On the contrary, permanent impairment of sight is the
usual event, and there may ensue detachment of the retina.
The mechanism of choroidal rupture has been elaborately dis-
cussed by Hughes l who also gives a full bibliography. There is no
treatment for these cases except rest and protection of the eye, and
abstinence from use until signs of irritation have disappeared.
Hemorrhage into the choroid is either spontaneous or trauma-
tic and differsifrom bloody effusions into the retina, in the appear-
ance of the spots. They are in irregular and broad patches, rather
than in streaks and elongated forms. The blood may burst through
the retina into the vitreous, or be diffused in the suprachoroidea
and lift the retina. If the quantity be large the smoothness of the
retina, the regular course of its vessels, and the deep color will
1 Graefe's Arch. f. Ophth., Bd. xxxiii., Abth. iii., 21.
588
DISEASES OF THE EYE.
suggest the lesion. It may also happen that a real detachment of
the retina from the choroid will co-exist. We see spontaneous
hemorrhage in acute choroiditis and in chronic forms, especially
complicating high myopia; we also find it with disease of the heart,
sclerosis of the vessels, pertussis, in blood diseases, etc.
Detachment of the choroid, by serous effusion is very rare and
is analogous to the condition so much more frequently affecting the
retina. It has once or twice come under my notice in the living
eye. It is common in museum specimens. Detachment arises
from tumors, from hemorrhage, and from chronic retino-choroiditis.1
The globe is apt to become soft.
Choroidal hemorrhage may be the precursor of tumors, it can
occur with chronic disease of the choroidal vessels in which there
are no large patches of atrophy, and it may give rise to acute glau-
coma.
Treatment depends upon general indications, and consists largely
in hygienic care both of the eyes and health. Absorption goes on
slowly during weeks or months and the blood passes from dark to
lighter shades of color, ultimately, as a rule, leaving a spot of ex-
posed sclera, speckled and bordered with pigment.
Tubercles of the Choroid.
Such deposits are found chiefly in cases of acute miliary tuber-
culosis, and especially of tubercular meningitis. In the latter dis-
ease it is not, however, more frequent than in 35$ to 40$ of the cases
(Michel). Children are naturally the most frequent subjects, and
it is of course difficult many times, because of the serious nature of
the general illness, to investigate the eyes. We find most com-
monly small specks in the choroid grouped chiefly about the central
region, sometimes near the periphery; more rarely there will be
a single one bigger than the optic papilla not far from the macula,
and occasionally there is a copious deposit of tubercle in the cho-
roid, with cheesy degeneration, invasion of the vitreous and the gen-
eral aspect of metastatic inflammation. The small isolated specks
are generally few, but may number thirty or forty and push their
way through the hexagonal epithelium, are dimly white, with a
pink areola, as the adjacent choroid loses its color. Sometimes at
a very early period hemorrhages co-exist. They have no sharp-
ness of outline, no pigment deposit and lack the brightness of spots
of choroiditis. Sometimes the optic nerve is inflamed. Thev may
appear with rapidity, pari passu with the general disease, but one
case is on record where a local deposit preceded by six months the
1 See Elsching, " Choroidal ablOsung," Archiv f. Augenheil. B.
CHOROIDEA.
589
symptoms of general tuberculosis (Frankel). They do not impair
sight, except when accompanied by neuritis or when, as rarely
happens, they set up choroiditis, as will be known by pigment ac-
cumulations; generally both eyes are affected. Such cases natu-
rally fall under the eye of the physician oftener than of the ophthal-
mologist. (See colored plate Fig. 15, taken from case of tubercular
meningitis. There is optic neuritis.)
The diagnosis is not simple because the elevation may not be easy
to recognize, yet when large enough this will form the principal fea-
ture, together with the variations in size, the indistinct border, ab-
sence of pigment, and lack of the lustre which characterizes colloid
excrescences. To this will be added the youth and general symp-
toms of the subject. They affect by preference the larger choroidal
vessels, and their adventitia. Much study has been given to this
subject by pathologists, Cohnheim, Weiss,1 Haab,2 etc., and inocula-
tion of Guinea-pigs has demonstrated this lesion as part of general
tuberculosis. For a careful account see Michel, " Lehrbuch," 1890,
pp. 403-406. There is no treatment. Prognosis is bad. We also
find large masses of granulation tumors (tubercles) in the vitreous,
the sclera, and the choroid.
Tumors of the Choroid.
Although of rare occurrence, viz., about once in 1,500 eye cases,3
choroidal tumors challenge attention because of their serious effects
both local and constitutional. The most frequent of them, 85$, are
sarcomata, and of these the pigmented are largely in excess of the
w7hite. In a small number of cases the origin of the growth is
associated with an injury—as a rule we know nothing of the cause
—and the eye is the primary seat of the disease. Usually we do not
meet a case until the tumor has attained an important size, but if
its site, as sometimes happens, is at or near the macula lutea, the
loss of vision will bring the patient for examination while the
growth may be very small (cases by Becker and Knapp). In one
instance under my observation a severe retinal hemorrhage across
the middle of the fundus was the precursor of a tumor which de-
veloped in the periphery and of which the earliest indication was
a mesh of vessels which could be very imperfectly examined be-
cause of their equatorial locality. The symptoms will vary with
the site and development of the growth. Four stages are recog-
1 Graefe's Archiv, v. xxiii. 2 Ziegler, " Path. Anat.," p. 768, 1884.
3 Fuchs, "Das Sarcom des Uvealtractus,'' Wien, 1882. Berry, "Diseases
of the Eye," p. 348, 1889, gives the frequency in Great Britain as 1 to 2,218.
590
DISEASES OF THE EYE.
nized (Knapp) -11st, the period of early growth while no irritation
is occasioned; 2d, the period when inflammation is excited, which is
generally glaucomatous and exceptionally takes the form of irido-
cyclitis with possible suppuration of the cornea and atrophy of the
globe; 3d, where the tumor makes its way to the exterior of the
eye; 4th, the period of metastasis to some remote part.
In the early stage the inception is the formation of a mesh of
dark and convoluted vessels at some spot in the retina, and a bluish-
gray opacity of the membrane (Michel). If within the field of obser-
vation we may see the elevation of the tumor, but it is very common
for the retina to be lifted from it by serous effusion, and while dis-
cerning this we may fail to recognize the tumor underneath. If
the retina be not too opaque, we may see the brown surface of the
tumor to which the pigment epithelium always adheres, and perhaps
irregular and broad vessels coursing over it. Attention will be at-
tracted to the comparatively smooth surface of the retina, that it is
not wrinkled, nor the vessels knicked, and that it flutters little or
not at all. It is important to use very strong light and to have
a dilated pupil. A large mirror of short focus is useful, and
intense focal illumination may reveal the vascularity of the
tumor. The tension may be normal, but if increased there will be
strong suspicion of a tumor. The history may give some aid to
the diagnosis. If it appear that a slowly increasing loss of sight
has at any time been succeeded by a sudden and notable aggra-
vation and without pain, we suspect tumor. The first effect will
he due to the tumor, the second to the detachment of the retina.
Under any circumstances the diagnosis is difficult and it may be
rendered impossible by hemorrhage into the vitreous. There will
be impairment of vision, either central or peripheral, but other sub-
jective symptoms are variable and may be wanting. Sometimes
the lens becomes hazy at this stage. In outward appearance the
eye will be healthy. The duration of the first stage is from six
months to four years (Fuchs), the average of 67 cases being 21
months. At the second stage, when inflammation is set up, there
is already increased tension, b}7 the interference of the tumor with
the vortex veins and with excretion at the canal of Schlemm, as
well as by the bulk which it adds to the ocular contents. If the
vision has not already been annulled by total detachment of the
retina, it quickly disappears with the symptoms of acute glaucoma.
A grayish-green reflex may be possible from the fundus, but no
details will be visible. The lens may perhaps be pushed out of
place. The acute attack may pass and the eye go into the state
of chronic glaucoma. It may yield to the pressure and develop
1 "On Intra-ocular Tumors," 1869.
CHOROIDEA.
591
scleral staphylomata, In a small proportion of cases considerable
exudation occurs from irido-cyclitis and the globe afterward
shrinks. Such an atrophied eye has been known to cause sympa-
thetic ophthalmia (Fuchs, 1. c, p. 253). After a longer period, which
may be more than a year, the growth will show itself on the out-
side of the eye, either in front or behind, and the rate of progress
will then be rapid. There will be cessation of pain, but the de-
formity will be likely to attract notice. A mass of some size may
exist posteriorly without being recognizable. No limit can be as-
signed to the development of the growth at this stage. It may
extend along the optic nerve or make its way out of the sclera by
way of the vessels and nerves. There ma}' be separate tumors in
the orbit, it may encroach upon the bones and surrounding cavities.
In former times very large tumors were sometimes met with, the
so-called melanotic cancer of the eye. The disease may penetrate
to the brain and provoke cerebral symptoms. During the fourth
stage we have signs of cachexia and the local indications of disease
of the liver or stomach, or lungs, as the case may be. The liver is
the organ usually attacked.
Diagnosis.—If the retina adhere to the tumor there will be no
difficulty in its recognition, and by the upright image its size can
be approximated. It can happen that a subretinal effusion near
the equator presents a smooth, uniform, rounded contour without
flattening of its surface or twists in the vessels. Such a condition
I have seen and have erred in diagnosis, believing the lesion to be
a tumor. Enucleation proved the contrary. In case of sub-
retinal effusion there will be obscurity, but hints have already
been given which are important, and besides those which relate
to the appearance of the retina the tension of the eye is to be noted.
If clearly above normal, the inference in favor of tumor, compli-
cated with sub-retinal effusion, is very strong. During the glauco-
matous stage it may not be possible to escape error, in spite of
careful inquiry into the history. If, however, iridectomy result in
only temporary or negative relief, enucleation will be likely to be
obligatory. It has been said that strong light and full magnifying
power should be used in ophthalmoscopy, aided by dilated pupil.
The employment of a convex lens 3" to 4" focus held near the eye
and the mirror at about six or eight inches from it (Becker) in-
creases both the illumination and the enlargement, and meets the
special requirements of these cases.
Treatment.—Enucleation at the earliest period is imperative.
The optic nerve should be severed as near the apex of the orbit as
possible; it is not difficult to excise ten to fifteen millimetres if the
globe be drawn well forward by putting the finger behind it, or
slipping a strabismus hook over the nerve to make traction, and
592
DISEASES OF THE EYE.
crowding the scissors well backward to make the cut of the nerve.
Hemorrhage is with this proceeding copious, and is to be checked
by pressure of the finger or thumb in the orbit. It is far better to
take the time needful for the purpose, than to pack a sponge into
the orbit and apply a compressive bandage. Getting out such a
sponge will give sharp pain and perhaps reproduce hemorrhage.
Perchloride of iron is objectionable. When active bleeding ceases
a firm flannel bandage may be applied upon a sponge and absorbent
cotton laid on the outside of the lids. The dressing may be re-
moved in six hours.
If the growth has invaded the orbit we may be obliged to empty
the cavity, and the operation may be formidable. We may have
to split the outer canthus to get sufficient access to the growth.
The best instrument is a large pair of scissors curved on the flat
and with blunt points; with this and a strong pair of forceps the
mass can be turned out of the orbit with the least bleeding. The
shut scissors serve admirably for scraping or tearing away the
periosteum and growth from the bony walls, and very little cutting
will be required, except at the beginning and finish of the opera-
tion. If the bone has been attacked, we may remove with a sharp
spoon as much as may be feasible, recognizing that we may pene-
trate adjacent cavities. Invasion of the cranial cavity is, of course,
highly dangerous. Chloride of zinc paste is sometimes applied to
the bony wall to cause exfoliation of diseased bone; doing this is
not without risk. Great care will be taken to adopt antiseptic
precautions, and if bleeding is not controlled by presure or by tor-
sion, a small thermo-cautery (Paquelin) may be used, but with ex-
treme caution if the vessel be at the apex of the orbit. One in-
stance in which I used a cautery proved fatal by meningitis. It
appears from statistics by Fuchs (1. c.) that of forty-five cases of
removal of the contents of the orbit only two resulted in death.
Prognosis.—Local return of the disease is rare and occurs
chiefly when it has advanced outside of the globe. The possibility
of an isolated tumor in the orbit is not to be forgotten. On the
other hand, reproduction of the disease in a distant locality, espe-
cially in the liver, is highly probable. Many times the ultimate
history remains unknown, and it is impossible to get accurate sta-
tistics on a large scale. Of twenty-two cases, Fuchs had knowl-
edge of the result in seventeen; respecting them thirteen had died;
of these, eleven by metastasis, and he was satisfied of immunity in
one case only (1. c, p. 276). Metastasis occurs within periods vary-
ing from a few months to five years; the average is two years.
The tumors most likely to show malignity are the pigmented round-
and small-celled sarcomata; the spindle-cell, colorless, and slightly
vascular tumors show less disposition to metastasis. The sarcoma
CHOROIDEA.
593
cells have been found floating in the blood-vessels of the choroid,
showing that they actually enter the general circulation. While
the liver is the most frequent place of deposit, the stomach, the
lungs and other tissues may be involved. No form of tumor is so
liable to be reproduced by metastasis as sarcoma. Griffith (Ophth.
Review, Dec, 1891, "Prognosis of Choroidal Sarcoma") tabulates
23 cases whose ultimate history was known. Of these 14 were
alive after periods varying from three to ten and a half years, and
the 9 who died reached their end by local or remote effects of the
disease after periods varying from one year and four months to
six years and eight months. Prognosis is better for the younger
subjects. See other references in Griffith's paper.
Of the other kinds of tumor which may be found in the choroid
we have angiomata, fibromata, chondromata, tuberculosis, and in-
flammatory swellings. Choroidal tumor has been known in a very
few instances to cause sympathetic ophthalmia.
Ossification of the Choroid.
In eyes which have long been shrunken by irido-choroiditis we
not infrequently meet with ossific formation in the inflammatory
tissue. There may be either plates and spicuke or there may be a
mass or a thick shell of bone. Very perfect specimens have some-
times been found. The stump is usually small, veiy flattened, and
if grasped between thumb and finger, the bony mass will be recog-
nized by its hardness and sometimes by its rough projections.
Sympathetic irritation of the fellow-eye is sometimes caused by
these stumps, and they should then be extirpated. A sharp hook
should be used to seize the globe and give control over it, else the
operation will prove troublesome.
38
OHAPTEE XVII.
THE RETINA.
An account of the appearance of the retina when viewed by the
ophthalmoscope is given on pages 34 et seq. We shall learn most
about it by the direct method, which, to be exact, presupposes
transparency of the media, and proper correction of optical errors
both in the observer and the patient. If these conditions cannot be
fulfilled, the indirect method will yield much information, and in the
case of patients confined to bed is often the only one available.
The blood-vessels are a principal object of study, and they help us
to form a judgment as to the state of the retinal tissue. Allow-
ance must be made for considerable variation in transparency of
the retina, especially near the optic nerve, and in direct ratio to the
depth of pigmentation of the fundus. On the nasal side of the
nerve the retina is always thicker than on the temporal and is less
clear. It is not necessary to repeat what has been said, p. 47, and
an attempt to relate minutely the physiological variations in the
look of the retina would be of little profit. Each observer must
learn them for himself, and must govern his judgment of objective
signs by examination of visual acuity, and of the visual field, and
of the color sense, both central and general. In most cases of de-
fective function, we can see tissue changes which, if not strongly
pronounced, are evident enough; but when we bear in mind how
intricate is the structure of the retina it will not be surprising that
functional impairment may exist which will not betray its physi-
cal cause to the moderate magnifying power at our command.1
Minute Anatomy of the Retina.—It consists of ten layers which,
beginning from the inner surface, are named as follows: 1, mem-
brana limitans interna; 2, optic-nerve fibres; 3, ganglion cells- 4
internal reticular or molecular or granular layer; 5, internal gran-
ule layer; 6, external reticular layer; 7, external granule layer; 8
membrana limitans externa; 9, bacillary layer or rods and cones-
10, hexagonal pigment epithelium. Besides, the retina is traversed
by numerous fibres of connective tissue which run perpendicularlv
through it and are known as the fibres of Miiller. In Fig. 226 the
relation of these parts is schematically represented. The optic nerve
1 As to the appearances of the macula see G. Lindsay Johnson, Archives
of Ophth., Jan., 1892, with colored plates.
THE RETINA.
595
fibres, the ganglion cells, the outer and inner granule layers and the
bacilli are the nerve structures; all other parts are considered as
modifications of connective tissue. The membrana limitans interna
serves to separate the retina from the vitreous. The succeeding
five layers, from the optic nerve fibres to and including the external
reticular, also called intergranular layers, are grouped together as
the cerebral layers of the retina; the outer granules, the membrana
limitans externa, and the rods
and cones are called the epithe-
lial layer of the retina. This dis-
tinction fixes upon the latter as
the percipient elements, and re-
gards the former as vehicles for
conve3ing impressions. To the
former must be added the hexag-
onal pigment cells, in which we
find the visual purple, and which
penetrates among the bacilli to
a greater or less degree accord-
ing to the stimulus of light.
Very delicate processes run in
from the pigment cells among
the bacilli, and along these (not
the cones) the molecules push
themselves in greater quantity
as the light is more intense.
In various parts of the retina
the layers are in different pro-
portions, for example, near the
papilla the optic fibres are in
excess. At the region of the
macula, important changes oc-
cur, while the fovea centralis
has a character wholly unlike
the rest of the retina. The fovea
has a slightly oval shape, being
horizontally 0.2 mm. and vertically, 0.15
measures in the visual field 1|° (Bunge)
Fig. 226.
mm. in diameter and
It does not exist in the
embryo and is not yet visible at birth (Kolliker). The optic nerve
layer disappears, the ganglion cells instead of being in a single layer
as elsewhere, increase to seven or eight layers, the external gran-
ules are found, and the cones are enormously elongated and multi-
plied; while the rods do not exist. All other layers are wanting.
The number of cones at the fovea is estimated at 7,000 (Kuhnt).
We can thus understand the high sensibility of this spot where
596
DISEASES OF THE EYE.
nothing but the epithelial structures are found. At its margin-
(ora serrata) the retina becomes very thin, the optic nerve fibres
and the ganglion cells almost cease, while the reticular layers dis-
appear and the connective tissue becomes more abundant. The
retina as it appears in section is well shown in Fig. 227, from
Poncet. Its structure is nearly normal.
The optic fibres as they enter the retina, lose their neuroglia
and become simple axis cylinders; they unite at acute angles in a
Fig. 227 — Section of Normal Retina. X350.—Eye removed for Sarcoma, Retina detached but
almost normal.—1. Vitreous; 2, hypertrophied cells of vitreous; 3, membrana limitans interna; 4,
fibres of Mutter (they are slightly hypertrophied)—they are part of the connective-tissue frame-
work; 5, layer of optic nerve fibres, nuclei more numerous than usual; 6, layer of ganglion cells;
some of them have undergone colloid degeneration; 7, internal molecular or reticular layer; 8,
layer of inner granules; 9, external molecular or reticular layer—in this as in the internal molec-
ular layer the fibres of Miiller are abnormally distinct; 10, layer of outer granules; 11 and 12, layer
of rods and cones, in which a distinction is made between the body of each element, 11, and the pro-
cess, 12, which is its continuation. The line which separates 10 from 11 is called membrana limitans
externa, it is not marked by a figure; 13, layer of epithelial pigment in polygonal cells. Fibres of
Miiller are also called radiating fibres.
plexiform manner, and as has been said can often be seen coursing
upward and downward from the disc and sloping off along the prin-
cipal vessels. Such of them as go transversely outward toward
the macula are much finer than the above named, and we find that
to the macula a large quota of fibres is directed, amounting, as
Bunge found, to about one-fourth of all the fibres contained in the
optic nerve. For the ophthalmoscopic appearance of the normal
fundus see Plate III., Fig. 1, and colored plate Fig. 1.
THE RETINA.
597
Opaque nerve-fibres constitute a congenital peculiarity which
is not very infrequent. By this is meant that certain fibres coming
from the papilla retain their neurilemma for a distance after they
enter the retina. They are grouped into a brush or cluster, which
is usually either above or below, and exhibits considerable variety
of form. It may be short and clumpy, or sweep in a long, wispy
plume, and usually along the principal vessels. I have seen a case
in which most of the centre of the fundus was whitened by such a
condition. Such a case is figured in Liebreich's Atlas. To recognize
the nature of this peculiarity, observe that the white or yellowish
patch conceals the edge of the disc, has a glistening, striated surface,
and the markings are in parallel lines; the edge is marked by a
hair-like fringe. The surface is intensely white or yellowish, the
vessels are partially concealed, and fine glistening markings
appear. Surrounding parts of the fundus will be normal; some-
times the optic nerve looks very red, but it will not be swollen.
In slight cases there will be no impairment of sight, in extreme
cases there will be amblyopia. Sometimes spots of opaque nerve-
fibres crop out, remote from the principal locality, but very rarely
does the abnormality reach beyond the macula or its vicinity.
(See Plate III., Fig. 2.)
Coloboma of the macula is an extremely rare congenital anom-
aly which resembles a patch of choroidal atrophy situated at the
macula. It is a white or pinkish-white spot, usually without any
pigment-deposit such as characterizes choroiditis, although in the
colored plate given by Loringx some pigment granules are shown.
Vision may or may not be reduced. This lesion has by many been
disputed. I have not seen it. Testimony and argument in behalf
of its nature and existence are found in several papers condensed by
Manz, in Jahresbericht der Ophthalmologic, 1885, p. 279. See Plate
I., Fig. 11, opposite page 114; illustrations taken from Fuchs.
Silex reports two cases under this title, but thinks them to be
choroidal atrophy and not coloboma.2
Diseases of the Retina.
Hyperaemia, apart from actual inflammation, is not easy to
make out. The blood-vessels are often tortuous, are in some cases
very abundant, they vary in size in different persons, and there are
so many varieties possible within physiological limits that it is
difficult to be sure that a particular case is abnormal. We form
a judgment chiefly upon the appearance of the optic nerve. It
1 " Text-Book of Ophthalmoscopy," Plate III., Fig. 4; description p. 94,1886.
3 Archives of Ophth., March, 1889, p. 8(i.
598 DISEASES OF THE EYE.
may be very red, but this often arises from refractive error like
hypermetropia or astigmatism, from prolonged use or excessive
weeping, from irritation of a foreign body in the cornea, from sym-
pathy with some other morbid process, like iritis, etc. In short
hyperaemia is rarely idiopathic, whether in the nerve or retina, but
symptomatic.
Nevertheless, there are cases of enlargement of the retinal ves-
sels from orbital tumors, from obstruction to the cerebral vessels
near the sphenoidal fissure, from emphysema of the lungs, from an
open foramen ovale, from aneurism, etc. A condition of teleangi-
ectasy is figured by Liebreich, and also a case where the veins were
converted into a series of bead-like expansions.
The intraocular circulation is in great degree independent of
fluctuations in the general system, and though an off-shoot from
the blood-vessels of the brain, it participates little in its circulatory
changes. A sharp distinction is to be made between idiopathic
hyperaemia, and hyperaemia attendant on inflammation. With men-
ingitis it is common, with brain disease its occurrence is variable.1
Pulsation of the retinal arteries, as a result of resistance to
the influx of blood from increased intraocular tension, as in glau-
coma, has been referred to; the same effect can arise from reduced
impulse of the heart. We see this in insufficiency of the semi-lunar
valves of the aorta, and less markedly with aneurisms of the arch
and of the ascending aorta. The combination of aortic insufficiency
with insufficiency of the mitral valves diminishes the tendency to
pulsation of the retinal vessels (Michel). A different kind of pulsa-
tion, viz., one which is less abrupt and reaches over a longer stretch
of the arteries occurs in exophthalmic goitre.
Venous pulsation as a physiological condition has been referred
to (page 51), it also occurs with aortic insufficiency, with hyper-
trophy of the heart, with increase of arterial tension. During syn-
cope the arteries will be small and the veins full.
Ancemia of the retina, in the sense of diminished flow of blood
independent of obstructive causes which will be referred to, scarcely
presents itself. The retinal circulation may for a time be arrested,
as in migraine and syncope, and in anaemic persons sometimes the
color of the blood becomes pallid, and its impoverished state pro-
motes hemorrhages, as in pernicious anaemia, chlorosis, etc. and
inflammatory changes may be added both in the nerve and retina.
Spasm of the Retinal Arteries, Megrims, Epilepsy of the
Retina.—In consequence of vaso-motor irritation the retinal ar-
teries may be firmly contracted and completely arrest the circula-
tion. Irritation of the S37mpathetic in tlie neck causes their con-
'See Gowers, "Diseases of the Brain," p. 173, 1887.
THE RETINA.
599
traction, while extirpation of the superior cervical ganglion induces
enlargement of the vessels in much slighter proportion (Becker).
Suspension of the circulation causes partial or total blindness
in one or both eyes. A so-called blind headache is a mild form
of the disorder, and the vessels of the brain as well as of the eye
may be affected.
The visual disturbance may be the most conspicuous phenome-
non, and should not be regarded as of serious moment. An illus-
tration is the following:
Mrs. W., widow, forty-three years of age, was brought to me by Dr.
Burchard. She seems well nourished, but is said to have lately lost flesh.
Has an anxious expression, been greatly worried by business cares, and been
under much excitement. Three months ago she began to have partial ob-
scurations of sight in the right eye. In a little time the sight would " go
out," as she said, every twenty-four hours, and everything be dark for fifteen
to thirty minutes. This condition affected the right eye only, until a few
evenings since. Then, while at the opera, she became totally blind for about
one-half hour, and, as she says, was in total darkness. Since then the same
thing has happened every evening, and lasted from a few moments to half
an hour. At my interview with her in my office she exclaimed that sight
was leaving the left eye. Her vision had been {previously examined and
found to be %% in each eye. While the dimness existed I inspected the left eye
with the ophthalmoscope. The arteries were reduced in size, the veins were
normal, no other notable appearances. The attack passed in a few minutes,
and then the arteries grew larger and were like those of the other eye. There
were no signs of effusion or inflammation. My advice was to use bromide
of ammonium, to take nourishing food, and try to secure exemption from
anxiety and care.
In another instance in a man, the blindness was complete in both eyes for
sixteen hours and then perception of light began to return. I saw the man
twenty-four hours after the beginning of the attack and by the ophthalmo-
scope recognized great pallor of the optic nerve, tenuity of the arteries, the
veins about normal. Xo effusion or swelling of the nerve or retina; tension
normal; pupils active. Could count figures at six feet. By inhalation of
nitrite of amyl, vision was restored in twenty minutes to the normal degree
and the intra-ocular circulation was resumed.
The patients may be subject to migraine, and besides the head-
ache and nausea have that kind of scotoma which is marked b}7
a bright and angular outline constituting the so-called fortifica-
tion line, because it resembles a fort with bastions. The attacks
are usually of short duration, so far as vision is concerned. The}'
sometimes take on the form of hemianopsia, which implies that
the disturbance is cerebral and not ocular. This will be again re-
ferred to. In case of prolonged or severe attacks, inhalation of
nitrite of amy] from three to five drops at a time will bring speedy re-
lief. Prognosis is good. General treatment would consist of tonics
and sedatives of the milder types, and attention to derangements of
the stomach, the uterus, or any debilitating causes. One must be
600
DISEASES OF THE EYE.
careful not to confound these cases with the temporary obscura-
tions which occur in glaucoma, and attention must be given to the
state of ocular tension and to the possible existence of excavation
of the nerve, etc. These conditions being absent, heart tonics are
indicated, viz., digitalis, nitro-glycerine, etc.
Ischcemia of the Retina.—The condition to which this name is
applied has been recorded a few times and it was first mentioned
by Alfred Graefe.1 The question will naturally arise as to how it
may differ in essence from the spasmodic suspension of the circula-
tion just now referred to. The subjects have always been per-
sons who have been much reduced by illness. In a case which I saw
in consultation with Dr. Knapp2 the child, three years of age, had
been much prostrated by whooping cough and when convalescing
suddenly became totally blind, losing perception of light in both
eyes. The optic nerves were white; in the left eye some arteries as
fine as threads could be discerned, in the right none could be found.
In both eyes the veins were of extreme tenuity. There were no
other lesions. We have, therefore, seriously diminished heart's
action and an impoverished state of the blood. That this situation
alone may not be an adequate explanation, is probable from the
observations of von Graefe upon cholera patients. He examined
the retinal circulation, when they were in the last stages of prostra-
tion and never failed to find blood in the vessels, and they had cor-
rect vision so far as could be ascertained. He, therefore, thought
that some obstructive cause was to be assumed in cases of ischaemia
and that it was likely to be found in the optic sheath. It seems likely
that we have a partial thrombosis behind the eyeball and that these
cases are to be referred to this head. Both eyes have been affected.
Treatment is directed to promote the action of the heart and
improve the health, and on the other hand to diminish resistance
to intraocular circulation. Rest in bed, good food, digitalis and
stimulants and tonics meet one indication, while paracentesis corneae
and iridectomy have been resorted to for the other. The former
may be tried for twenty-four or forty-eight hours before employing
the latter. Alfred Graefe did iridectomy on the tenth day of total
blindness in one eye, and on the thirteenth day of total blindness in
the other eye with restoration of perfect vision. Inhalation of ni-
trite of amyl will also suggest itself. As a rule the prognosis is
good, but atrophy of nerve may result.
Embolism and Thrombosis of the Retinal Vessels.—Each of
these conditions may occur separately, or in combination. There
may also be complete or partial suspension of the circulation. The
'Graefe, Archiv f. Ophth., viii., i., 143.
2Archives Oph. and Otology, vol. iv., p. 448, 1875. See also Poolev
Trans. Med. Soc. State New York, 1878, p. 153.
THE RETINA.
601
obstruction may be in the central arterial or venous trunk, and
may also take place in one of the retinal branches1 (Saemisch).
The first case of embolism of the arteria centralis retinae was seen
by von Graefe2 in 1858, and he founded his diagnosis upon the in-
traocular appearances and upon the presence of endocarditis which
was due probably to a severe blow upon the chest. The correct-
ness of his judgment was verified b}- an autopsy eighteen months
later. The remnant of a plug was actually found in the retinal
artery just behind the eyeball.3 The symptoms of this case were
very striking and its pathology beautifully simple. Very few such
observations have been made since. Some cases have seemed to
point to partial arterial occlusion, because some blood was evi-
dently circulating in the retinal arteries and vision was not wholly
abolished. An emphatic feature of thrombosis is the presence of
hemorrhages with signs of obstruction to the venous circulation,
while vision is not so entirely destroyed as in cases of complete em-
bolism. Still another lesion which may happen is hemorrhage into
the sheath of the nerve (Magnus4), and a case in which this was
assumed was reported by Sands5 in 1866. A further complication
giving rise to a different ophthalmoscopic picture is related by
Angelucci.6 He found at the autopsy the central vein completely
occluded by a plug, also surrounded by a mass of exudation and
clot which extended some distance up and down the nerve, and
through the compression of this material the artery where it lay
in contact with the venous plug, was completely shut up; behind
this point it contained blood. In another case he found similar
conditions without an}7 hemorrhage in the nerve (1. c.)
An admirable discussion of this subject is given by the late Dr.
E. G. Loring and illustrated by many plates in his "Text-book of
Ophthalmoscopy," vol. 2, p. 59, 1891. See also " Embolie der
Arteria Centralis Retinae,'' Fischer, Leipzig, 1891, a treatise
founded on 148 cases.
Symptoms.—Always there is sudden loss of sight without pain
or external symptoms. In Graefe's case, which was the type of
simple and complete embolism of the central artery the blindness
was total. The optic nerve at the outset was extremely pale, the
arteries reduced to a minimum, their finer twigs invisible, the veins
smaller than usual, but fuller toward the equator than at the nerve;
1 Monatsblatter f. Augenh., 4, 32, 1866, with plate.
2 Graefe's Archiv fur Ophth., Bd. v., Abth. i., 5, 13G, 1859.
3 Schweigger, " Vorlesungen tiber den Gebrauch des Aut?enspiegels," Taf.
iii., Fig. 10, 1864. See also his "Handbuch der Augenheilkunde."
4"Die Sehnervenblutungen," Leipzig, 1874.
5 Trans. Amer. Oph, Soc, 1866, p. 2.
* Monatsblatt. f. Augenheilk., xvi., 444, 1878.
602
DISEASES OF THE EYE.
there were no hemorrhages and no opacity of the retina. Neither
phosphenes nor arterial pulsation could be excited by pressure; the
tension was slightly minus. In ten days a slight to-and-fro move-
ment appeared in the veins, and soon a little perception of light was
gained in the inner and upper part of the field. Within a week a
dense white opacity appeared over a considerable region around
the macula and at its centre the fovea shone as an intense cherry-
red spot. Its brightness was ascribed to the effect of contrast,
and the color to depend on the shining of the choroid through the
extremely thin membrane. Subsequently the appearances became
those of complete atrophy of the nerve. Appearances such as are
described., are depicted by Liebreich (Atlas, PI. VIIL, Pigs. 4 and 5).
In Fig. 228 from Magnus' Atlas the white exudation in the macula
contrasting with the bright foVea is shown, while the optic nerve is,
also infiltrated and the arteries filiform.
We find variations from the above symptoms, in that sometimes
the nerve becomes red again, and may be infiltrated. Autopsy
has shown that there was a mixture of embolism and thrombosis.
Again hemorrhages may occur. In Nettleship's1 case of arterial
embolism it appeared that the obstruction occurred at the branch-
ing of the artery in the papilla, that it happened twice and that
one branch was left pervious. Hemorrhages indicate either that
the arterial flow is not absolutely arrested, or they point to venous
thrombosis as the chief lesion. Embolism of a single arterial
branch on the retina was accompanied in Saemisch's2 case with dif-
1 Oph. Hosp. Reports, vol. viii., p. 1, 1874.
2 Monatsblatter f. Augenheilk., iv., 32, 1866.
THE RETINA.
603
fused opacity, but no hemorrhages, while in Knapp's 1 case there
were abundant infarctions.
The central retinal opacity in some cases begins immediately, it
ma}7 be delayed a week, after a time begin to disappear, and the
borders of the arteries may show a white streak or be converted
into white threads. The same kind of opacity occurs in the retina
after contusions of the optic nerve in the orbit, or after its section.
Experiments by Berlin on rabbits and frogs showed that when
caused by section of the nerve, the change began in the nerve fibres
and ganglion cells and extended to the remaining layers. Gowers
found the retina infiltrated with small round cells. While embolism
of the trunk cuts off the arterial supply to the inner layers of the
retina, the outer layers possess their proper supply in the chorio-
capillaris. Infiltration of the nerve may or may not take place.
Thrombosis of the vena centralis will present various symptoms
depending on whether the calibre is entirely or partially closed.
If closure is complete, the nerve is nearly obliterated by hemor-
rhage and effusion, abundant hemorrhages cover the retina and
accompany the vessels, and mixed with the patches will be yellow-
ish exudation. The veins will be tortuous and enlarged, inter-
rupted by blood and exudative plaques, the arteries may be small
and straight, contrasting with the sausage-like (Michel) look of the
veins. The retina will be soaked and there may be a diffused opac-
ity about the macula.
Imperfect closure of the venous trunk will exhibit fewer hemor-
rhages and these chiefly in streaks about the nerve, the retina will
be less oedematous and opaque, but the contrast between the veins
and arteries will be conspicuous. Pressure may or may not cause
pulsation in the arteries and it may appear in the veins. Vision
is greatly reduced, sometimes destroyed, is sometimes eccentric.
Sometimes it will be partially restored. Tension is not affected.
The progress of the case will be slow, and there may be recur-
rences of hemorrhage.2
As an exception to the features thus related, the case examined
by Angelucci3 should be mentioned, where thrombosis of the veins
caused at the same time occlusion of the arteiy by pressure and
there were no hemorrhages either in the nerve or retina. The ar-
teries were in places empty and the papilla was infiltrated.
Primary retinal phlebitis, i.e., confined to the veins of the fundus,
is described by Mules4 in three cases, and of one a colored plate is
given. The affected veins are much swollen, dark for a part of
1 Archives of Ophthal., vol. iii., No. 1, p. 38.
2 Priestley Smith, Ophth. Review, iii., pp. 1, 33.
3Monatsblatterf. Augenheil., xvi., 450, 1878.
4 Trans. Ophth. Soc. United King., ix., 130, 1889.
604
DISEASES OF THE EYE.
their extent, and the rest pale and collapsed—in two cases hemor-
rhages; in one case the cherry-red macula with surrounding white
oedema; in two cases total central scotoma. The onset of the
thrombosis in two was slow, in one was sudden. Sight not re-
stored; in two cases all ophthalmoscopic traces ultimately
vanished. A case is also delineated by Loring, "Text-book of
Ophthalmoscopy," 1891, Plate I. The cause is attributed to an
overdose of quinine. See below, p. 700, remarks from De Schweinitz.
The isolation of the retinal circulation explains the peculiarities
produced by embolism, because it communicates so sparingly with
other vessels, viz., only with the choroid at the papilla. Sometimes
a small twig (Schwalbe) runs up parallel to the central artery to
the papilla and may account for some instances where the vascu-
larity of the papilla was subsequently restored (Schmidt).1
Embolism of cerebral arteries happens in conjunction with retinal
embolism. The ophthalmic artery may also be occluded and in one
case irido-cj'clitis ensued (Schmidt), while in another (Nettleship)
acute glaucoma demanded enucleation.
In all cases, in order to establish the diagnosis of embolism, some
source must be found whence the foreign body can be derived;
from vegetations in the heart or aorta, aneurisms or atheroma of
arteries, from thrombosis of the uterine veins or of those of the
lower extremities after pregnancy or abortion, etc.
Thrombosis occurs mostly in elderly persons, whose arteries
have degenerated and whose heart is likely to be fatty, or who may
have marked emphysema and whose circulation is therefore feeble.
It may come from a real phlebitis or periphlebitis.
Diagnosis.—A sudden loss of sight, attended by great reduc-
tion in the calibre of the retinal arteries and bleaching of the nerve;
followed soon by haziness of the central region of the retina, be-
speaks arterial embolism. When hemorrhages are abundant, the
probabilities are in favor of thrombosis or of a mixed condition in-
volving both arteries and veins (see Priestley Smith, Oph. Review,
Vol. III., 1884, p. 47). What has already been said need not be re-
peated. It will not always be possible to decide what the precise
lesion is, nor is it important apart from its scientific interest. Fig.
3, Plate III., presents some of the features described, but is spoken
of as simply apoplexy of the nerve and retina. The cases are not
frequent and
Prognosis in general is bad. A case seen by Mr. White (Oph.
Review, Vol. I., 49, 1882) recovered sight under the eye of the sur-
geon who was making pressure on the globe, and he saw the circu-
lation re-established. In a case seen by Mr. Eales (Oph. Review,
•Graefe's Archiv f. Ophth., xx., 2, 287, 1874; and see also Trans. Am.
Ophth. Soc, 1890, p. 672.
THE RETINA.
605-
Vol. I., 139, 1882) vision was restored in nine months, with the ex-
ception of a central scotoma. There is one case (Nettleship, Oph.
Hosp. Reports, Vol. XL, 111, 260, 1887) in which both eyes became
blind at successive periods, each retaining in the upper part of the
field a sector where light could be perceived. Hirschberg reports
a case where each eye was affected (Centralblatt f. Augenheil.,
Jan., 1884) and at the autopsy the obstruction was not found. Re-
covery after embolism must mean displacement or disintegration
of the plug. After thrombosis it would not seem so improbable,
provided absorption occurred before destructive changes had en-
sued in the nerve and retina. Yet very rarely has any useful vision
been restored.
Treatment can, in the nature of the case, accomplish little. Iri-
dectomy and paracentesis have been tried in vain, with a view to
lessen the opposition to the circulation within the eye. An em-
bolus if loosened must soon be again caught fast, and the only hope
would be in its becoming disintegrated. To this end as Mauthner
proposed Mules (Ophthalmic ^^SS^f^r?^s^^^&:f('f%
Review, 1888, p. 245) and
Gifford (Jour, of A in. Med.
Assoc, April 12th, 1890)
have, in cases where a retinal
branch was involved, re-
sorted promptly to massage
of the globe, and re-estab-
lishment Of the circulation Fig. 229—The specimen was taken from a case of
T-l hemorrhagic glaucoma. 1, Layer of optic fibres be-
haS been Witnessed. r Or tween which the internal granules are visible: 2, some
. ganglion cells: 3, varicose vessels, their walls have sep-
thrOtnboSlS there WOUld Only arated from the lymphatic sheath; 4, lymph sheath
■, . ,. . . .. deeply pigmented and much larger than the vessel.
be general indications oi
tonic and recuperative treatment and the hope of absorption.
Unhappily in nearly all the cases, atrophy of the nerve and retina
is the outcome.1
Hemorrhage into the retina presents itself under other condi-
tions besides those just mentioned, viz., from injuries, as a compli-
cation of choroidal and retinal inflammations, in cases of choroidal
atrophy in myopic eyes, and it also happens idiopathically. We
find it in young persons of delicate health, in such as have a hemor-
' The literature of this subject is large. See a paper by Loring, Am.
Journal Med. Sciences, lxvii., 313, 1874. See also Michel, Archiv f. Ophth.
(Grraefe), xxiv., 2, and Ophthalmic Review, vol. i., 52, 1882. A paper by
Priestley Smith, Oph. Review, Jan. and Feb., 1884, is of much interest, For
a verv complete and minute discussion of the whole subject, see "Text-book
of Ophthalmoscopy," Loring, part 2, pp. 59-97, 1891. Also "Embolie der
Arteria-centralis-retinaj," Fisher, Leipsig, 1891, p. 246.
606
DISEASES OF THE EYE.
rhagic diathesis, as an effect of menstrual disorders, or of pulmonary
emphysema, or of severe coughing or sneezing. It specially occurs
in pernicious anaemia under the form of small streaks clustered
about the nerve and scattered over the surface, while white patches
may appear and sometimes neuritis. Severe retinal hemorrhage
with atrophic changes in the choroid occurs in connection with
great losses of blood at the menopause. The gouty or rheumatic,
or syphilitic dyscrasas, are frequent factors.
In new-born children retinal hemorrhages occur with considera-
ble frequency (10$ to 32$), especially when the labor is difficult.
Permanent injury may not remain, but as the region of the macula
is the favorite situation, a grave amblyopia may ensue for which
no ophthalmoscopic lesion may afterward appear. This fact ac-
counts for a certain number of cases of congenital amblyopia
(see remarks on strabismus). CEdema of the retina and also of
the optic nerve are met with, although less frequently, in these
subjects.2
In all cases of spontaneous hemorrhage a weakened condition of
the vessels is to be presupposed or a perverted quality of the blood,
or both combined (splenic retinitis). The association of retinal
hemorrhage with cerebral or other visceral apoplexy is not infre-
quent. The capillary aneurisms found in the brain may also exist
in the retina and be the explanation of hemorrhage (see Fig. 203).
While these are too small for the naked eye to discern, aneurisms
have been noted which were readily visible and numerous (Hirsch-
berg).
It is, therefore, important to study the remote as well as the
immediate occasions for the occurrence, because not only will the
possibility of recovery be often bound up with them, but the wel-
fare, and even the life of the patient may be concerned. If the
hemorrhage be not at or near the macula, the injury to sight will
be moderate and be occasioned in part by slight turbidity of the
vitreous, while a larger bleeding will do mischief to other regions
of the retina by interfering with the conductivity of the nerve-fibres,
and possibly by setting up inflammation. When it occurs in the
fovea centralis, a dense central scotoma ensues, and sometimes the
patient speaks of seeing things of a red hue. Abundant hemor-
rhage may deeply cloud the vitreous, and it is often impossible to
trace the source of the effusion until the media have cleared up.
The most common starting point in such severe cases is the ciliary
body or choroid, while from the retina the bleeding is usually less
1 See case with autopsy by Elizabeth Sargent, Archives of Ophthal., xxi
1, 39, 1892.
2 Naumhoff: Graefe's Archiv f. Ophth., xxxvi., iii., 180-246, 1890.
THE RETINA.
607
copious, and, therefore, confined to its own tissue. Because the
retinal vessels are situated in its anterior layers, it follows that
recovery of sight is often possible, but if the blood should invade
its posterior layers, more or less dimness of vision must be expected
to remain. Allusion may be made to hemorrhagic glaucoma, and
to the possibility of a choroidal sarcoma being imminent (see Fig.
229), and to the bleedings which sometimes follow operations for
cataract or iridectomy.
The blood may be absorbed without leaving any visible altera-
tion of tissue, while, on the other hand, a pigment-deposit may
occur, or a whitish substance be left, which indicates inflammatory
exudation, followed by secondary changes. Sometimes extensive
cicatrizations and pigmentary deposits remain, especially if cho-
roidal disease co-exists. When bleeding takes place into the mac-
ula, it is rare for sight ever to be perfect, and even if much be
gained, metamorphopsia is liable to remain. A word may have
some of its letters displaced or distorted, and the letters on either
side of these be natural.
Retinitis apoplectica, or hemorrhagica as it is called, occurs
chiefly among elderly persons and presents very marked features.
Both the nerve and retina may be involved, there may be chronic
renal and perhaps cardiac disease, and the blood-vessels be affected
by atheroma or syphilitic degeneration. The affection is sometimes
in both eyes. The attack comes suddenly, with great loss of sight,
and without premonitory symptoms, except sometimes headache
and dizziness. The lesions in a well-marked case are as follows:
The nerve is hypersemic and swollen, its tissue oedematous, and
edges indistinct—in some cases the outline is obliterated. The
veins are full and tortuous, the arteries small, and some may be
thread-like. The retina has a wateiy look, and the fundus is spat-
tered with small hemorrhages, many of which are in short lines
running parallel to the vessels (see Fig. 3, colored plate). There
will also be larger and irregular patches of blood, and these usually
by the side of a vessel. It will be noticed that one or more arteries
are exceedingly reduced, or may be absolutely empty, and they will
be close to or lost in one of the blood-patches. Near the hemor-
rhages, spots of yellowish exudation will be found, which may be
extensive and numerous. If hemorrhage has been copious, the
vitreous will be a little hazy, and further obscure the retina.
The features of various cases differ greatly. Sometimes the
stress of the lesion is on the optic nerve, while only a limited space
in the retina is affected (see Fig. 3, Plate III.). Sometimes the retinal
lesion is at the macula and its vicinity. Sometimes the ecchymoses
run along one vessel, partly covering it up, and spreading around it,
608
DISEASES OF THE EYE.
attended with blotches of yellow exudation, and not far from them
will be found some empty and thread-like vessels. It was the early
opinion that the starting-point of this affection was an inflamma-
tion of the retina, and that the hemorrhages were sequent to the
inflammation; but these cases are really instances of obstruction
and thrombosis of one or more arteries, and the inflammatory
oedema, the hemorrhages, and the exudation, follow upon the oblit-
eration of small or larger vessels. Such effects are possible in the
retina, because its vessels anastomose sparingly with others, and if
obstructed cannot obtain any aid. In fact, the branches which go
to the macula and to the periphery, and also to other portions of
the retina, constitute regions of so-called terminal vessels in the
sense which Cohnheim has emphasized, and these localities, there-
fore, are liable to the peculiar changes he has described in treating
of embolism. On this view the retinal process is an infarctus, with
the same alterations which are found in the lungs, liver, and joints
under similar conditions. Taking into account the modifying influ-
ence of the anatomical character of the retinal circulation, we have
in these cases illustrations of the effect of chronic endarteritis or so-
called arterio-capillary fibrosis as a purely local condition. The
changes in the walls of the vessels have preceded the outbreak and
their character has been of late carefully investigated. See article
by Meigs: The Medical Record, Aug. 24th, 1889. Under the mi-
croscope rupture of the vessels is rarely found, and the hemorrhage
is therefore chiefly by diapedesis. Phlebitis, with thrombosis, may
be confounded with the above affection; but close attention will
reveal the differences (see p. 551). See also Loring (I. c, p. 88,
part 2).
In all that has been said about hemorrhages into the retina the
distinction must be borne in mind between the cases dependent on
changes in the composition of the blood such as pernicious anaemia,
malaria, menstrual disturbances, etc., and those in which vascular
changes, whether arterial or venous, are primarily at fault. In
these two classes prognosis will be different, local features will
vary, and treatment be dissimilar. I have refrained from portray-
ing in detail these several classes, but call attention to the distinc-
tion to be made among them.
Another class of cases deserves mention, viz., those among
new-born children. Naumhoff (Graefe's Archiv, XXXVI., 3, p.
180) examined the eyes of 47 new-born children who had died at
or soon after birth, and found under the microscope choked disc
retinal oedema, and hemorrhages in the retina and choroid. All
these lesions, and especially the last, are due to the compression
produced by protracted labor and a narrow pelvis. The hemor-
rhages were the result both of rupture and diapedesis. They were:
THE RETINA.
609
often absorbed without leaving any traces visible to the ophthal-
moscope. Congenital amblyopia and congenital atrophy of the
optic nerve may sometimes be explained by the lesions referred to.
Prognosis depends much on the general condition of the patient
and on the extent of the bleeding as well as on its situation. A
breach in the macula will seldom permit of perfect restoration.
There will either be metamorphopsia or scotoma. With young
and vigorous persons entire absorption may occur, while glistening
or opaque white spots and black pigmentations often remain. In
elderly subjects there may be partial recovery of sight, but the
future as to cerebral hemorrhage is often ominous. Atrophy of
the optic nerve may ensue, and the spots of hemorrhage or exuda-
tion be subsequently designated by whitish tissue; or, if the blood
be fully absorbed, insterstitial changes in the retina may preclude
useful function. The obliterated vessels are usually not restored,
and those which at first were pervious may be slowly thickened
and diminished, and bordered with white lines.
Treatment—The local measures are simple, viz., rest, avoidance
of light, abstention from use. Dry cupping and in very plethoric
subjects leeches to the temple might be indicated. Exceptionally
it may be proper to use atropia. The object is to promote absorp-
tion and to prevent recurrences. The course to be adopted will be
determined by the general condition, the remote cause. With young
subjects invigorating treatment, fresh air, generous diet, proper
rest will be indicated. Attention must be given to menstrual dis-
orders. In one of my patients removal of the ovaries was under-
taken. In cases of pernicious anaemia the restoration of the func-
tion of the stomach and food assimilation, coupled with blood and
nerve tonics, iron, strychnia, etc., will be regarded. To coun-
teract the hemorrhagic diathesis, a visit to chalybeate springs, and
the persistent use of iron and tonics will suggest themselves.
Everything that will help digestion and assimilation, will be laid
under contribution; the hypophosphites are often useful.
In elderly subjects, where the heart and arteries and perhaps
the kidneys are at fault, the indications are different. There has
often been great mental strain, and there is apt to be much nervous
excitement. Suspension of business cares or literary labor, avoid-
ance of worry, procurement of sleep, proper action of the bowels
will naturally suggest themselves. Mild cathartics, moderate
use of bromides, sometimes diuretics, will be proper for certain
cases. For persons with a small and especiall}7 a hard pulse in-
dicating high arterial tension, nitro-glycerin in doses of gr. yfg has
become an established remedy. Change of scene and quiet and
cheerful surroundings are often important. Small doses of iodide
of potassium ma}7 be useful. For gout and rheumatism, the special
610
DISEASES OF THE EYE.
remedies will be chosen. In short the treatment must be based
upon a proper appreciation of the general condition of the patient
and varied according to circumstances. Especial regard must be
had to the dangers of cerebral hemorrhage, of. which a retinal
bleeding may be the signal. From one to twelve months and
perhaps a longer time will be required for the completion of the
recuperative process.
Miliary aneurisms have been seen in the retina similar to
those of the cerebral arteries, and they may occasion hemorrhages
Retinitis.
Symptoms.—In describing inflammation of the retina, we are
obliged to take account of the optic nerve, because in most cases
both are concerned; yet it happens that each may be inflamed
without apparent participation of the other. The subjective symp-
toms are dimness of sight in every possible degree, sometimes lim-
itations of the field, and micropsia, megalopsia, or metamorphopsia,
and frequently irritations of the retina in phosphenes, scintillations,
or glimmerings:—there will be no pain. Objectively the eye shows
no external alterations, either in its blood-vessels, in the action
of the pupil, or in any way, unless retinitis be complicated by
other diseases. By the ophthalmoscope we find a variety of ap-
pearances, consisting of exudations and alterations of tissue and of
the blood-vessels. Before describing them it is well to call atten-
tion to the variations in the look of the retina, which are not mor-
bid in the sense now intended. Such, for example, are the white and
often abundant nerve-fibre striation running out of the disc, the
turgid vessels, the red or leaden-hued nerve, with indistinct edges,
to be found in some cases of marked hyperopia: to these add the
flashing glitter seen mostly in the eyes of young persons, and
sometimes called the shot-silk appearance, and the variable look
of the macula in different persons and at different ages (see p. 50).
Moreover, the blur which results from astigmatism or from hazi-
ness of the media must be carefully discriminated. Always must
the observer be careful to determine the refractive conditions be-
fore he pronounces upon the status of the retina. To this end the
direct ophthalmoscopic method is far to be preferred to the indirect.
The following pictures present themselves as types of retinal in-
flammation apart from its etiology
a. Capillary congestion of the optic nerve without swelling, its
edges partly or wholly blurred; the fibres which pass into the ret-
ina are uncommonly distinct above and below as fine parallel lines
they fade gradually and suggest a slight degree of infiltration. The
THE RETINA.
611
veins are turgid and wavy, the arteries of normal size. There may
be a whitish line along the large vessels. No other textural change,
except possibly one or two yellowish-white dots in the retina near
the macula. This may be a low grade of inflammation, and is of
doubtful significance.
b. Deep redness of the optic disc, the edge almost or wholly
obliterated by the striation radiating into the retina about its en-
tire circumference, the veins full and dark, the arteries large. Both
are tortuous, and the light-streak is scarcely to be noticed—the
tissue of the retina soaked with transparent fluid, and evidently
swollen—the pigment-epithelium not to be recognized (i.e., the nor-
mal granular look is wanting) and the reflex from the fundus is
reduced. This is a case of moderate serous effusion.
c. Slight, perhaps no hyperaemia of the nerve; edges not well
■defined; vessels not noticeably altered; the retinal tissue pervaded
by a gray, misty infiltration, seen best at the edge of the illuminated
space, or by reduced light, and this cloud occupies the middle por-
tion of the fundus; the fovea too dark, but impossible to be defined;
the small vessels of the macular region conspicuous by their number
and size; and along the large vessels a border of more positive
gray; slight plastic exudations, such as are often found in syphilis.
This is sometimes called retinitis centralis,
d. A rare condition is a patch of exudation in or near the centre
of the fundus, partly concealing the vessels, of considerable extent,
with soft edges, and no other changes, except bright hyperaemia of
the nerve. A local plastic exudation.
e. At the macula a group of bright lustrous dots, few or
many and apt to be arranged in radiating lines, while the nerve
ma^7 be a little red: likely to be a mild albuminuric lesion. In
the highly developed types of this condition, we have the nerve
swollen and hyperasmic, its edges blurred, its tissue infiltrated,
the adjacent retina swollen; a little distance from the nerve
there will be white patches of irregular form; near or at the mac-
ula lutea are similar patches, which may be rounded or arranged
in radii, like an imperfect star; extravasations along the vessels and
in spaces where no vessels are visible. The white plaques or dots may
be of a dull hue, like greased paper, or be intensely white and glis-
tening. Sometimes the hemorrhages are more numerous than the
white deposits; sometimes they are few, and large surfaces of white
are seen. I have a picture of a case in which the fundus reminds
one of a " mackerel sky " in full sunlight, so numerous and fleecy
and bright are the clouds. In some cases pigment-deposits and
choroidal lesions can be seen. The remarkable white infiltration of
the retina found after embolism and thrombosis is not included in
the present description.
612
DISEASES OF THE EYE.
f. There are cases in which slight infiltration of the retina, hazi-
ness and redness of the nerve, and fulness of the vessels, are associ-
ated with turbidity and floating bodies in the vitreous, perhaps
with hemorrhages or even with new vessels in the vitreous. This
is the retinitis attendant on some cases of iritis and choroiditis, and
is sometimes styled retinitis proliferans.
g. In panophthalmitis or suppurative choroiditis, the retina is
infiltrated with pus, but the condition belongs to pathological
anatomy rather than to clinical study.
h. Still another condition occurs after a blow upon the globe*
It is a faint grayish-white patch, perhaps attended by hemorrhage,
sometimes at the equator or at the central region, which comes
almost immediately and will generally disappear in a day or two.
From the above it is seen that the features which command at-
tention are changes in the color, texture, and outline and level of
the nerve; opacities and infiltrations of the retina; the course and
size of the vessels, the clearness of their light-streak, infiltration
and thickening of their walls (perivasculitis); hemorrhages; we
may also have sub-retinal effusion, pigment-deposits, and choroidal
disease; complications on the part of the vitreous, lens, iris, and
other structures may co-exist.
Etiology.—It is rare to find retinitis as a simple or idiopathic
affection. It is rather the effect of constitutional dyscrasia, of blood
disease, of disease of the brain, of lesions of other parts of the eye, or
of injury. Exposure to extreme light will cause the milder types, as
seen in Arctic explorers and among travellers in hot climates. But
the chief causes are kidney disease, diabetes, syphilis; from men-
strual disturbances, leucocythaemia, poisoning by lead and by phos-
phorus, etc. The most common is albuminuria, from whatever cause;
next may be syphilis, while association with brain disease is a fre-
quent occurrence.
Acute Traumatic Retinitis.—Mention should here be made
of the appearance on the retina, after a blow upon the eye, of a
circumscribed grayish-white patch, which lies below the vessels and
may occupy either the middle or peripheral parts of the fundus.
It was first described carefully by Berlin (Klin. Monatsbldtter,
1872, p. 46), and a case is given by Jackson, with colored plates, in
Trans. Am. Oph. Soc, 1888, p. 68. It comes soon after the injury
and may entirely disappear, unless there have been hemorrhages,
within twenty-four hours or three days. There may be temporary
astigmatism from commotion of the lens. Marked amblyopia is
caused, and this may not disappear until long after the visible
changes have vanished. Sometimes there is from the beginning
amblyopia without any exudation. The condition is liable to be
THE RETINA.
613
overlooked, because the pupil is often rigidly contracted (see p. 478;
see also Loring, "Text-book," Vol. II., p. 174).
Retinitis Simplex.—Such a name is justified by the fact that
we sometimes are unable to assign any special cause for the lesion,
while the appearances may be the same as in some cases of syphi-
litic retinitis, or even when some remote disease, like menstrual
disorders, is considered to be the provoking affection. The* purpose
is to call attention to the lesion as shown in Fig. 17, colored plate.
We have hyperaemia of the nerve, its edges not specially ill-defined,
but concealed by a faint infiltration which resides in its substance
and extends over a large area of the adjacent retina. The
bluish haze is thin and delicate without specks or striations or
plaques, but is evidently chiefly a serous effusion into retina and
nerve. One artery going upward is a little enlarged and upon an-
other directed to the macular region, is a minute hemorrhage. The
real lesion of the case, as described by Jaeger, was probably in the
nerve, and will be again referred to, but the retinal appearances are
those of simple or serous retinitis. This infiltration passed entirely
away as usually happens, and if there be no deeper lesion sight
may be fully recovered.
Retinitis Albuminurica.—This condition occurs chiefly in the
chronic forms of kidney disease, but also arises during the albu-
minuria of pregnancy and after scarlet fever, diphtheria, and rarely
after measles. It may develop with any of the forms of kidney
lesion, the fibrous, the fatty, the amyloid, etc. While hypertrophy
of the heart often co-exists, it is not necessary to the retinal com-
plication. The retinal affection appears when renal symptoms have
already been fully declared, and it is also the first signal to attract
attention in a notable number of instances. Uraemic symptoms,
such as headache, morning nausea or vomiting and palpitations, with
frequent micturition, especially at night, may have existed, and some-
times not even these signs have betokened the trouble. The severity
of the local changes varies greatly, while in a very few cases there will
beurasmic blindness or amblyopia without ophthalmoscopic appear-
ances. The central portion of the retina is usually the part selected,
and perhaps to the exclusion of the optic nerve, and, on the contrary,
the nerve may be deeply infiltrated and swollen, with trifling implica-
tion of the retina. As a rule both nerve and retina are concerned.
Diffused opacity, white patches large or small, and dots, hemor-
rhages, and with less frequency pigment-deposits and detachment
of the retina, are what we find. It is highly exceptional to have
but one eye involved (such cases are recorded; see Ophth. Review,
Jan., 1885, p. 23; De Schweinitz, "Diseases of Eye," p. 431, 1892);
frequently one is much worse than the other.
614
DISEASES OF THE EYE.
It is declared that a faint and diffused opacity about the centre-
of the fundus and some redness of the nerve have been seen as the
earliest evidence of the disease (retinitis simplex), but few have
had opportunity to make this observation. Not infrequent is it,
as said above, to be asked to explain loss of sight in a person sup-
posed to be well, and find besides slight congestion of the nerve,
that the fovea centralis has a darker hue than usual without
distinct pigment granules, its border may be strongly accentuated,
and make a rather conspicuous dark ring; near to it will be
glistening white specks or streaks forming the rays of a star of
unequal length, generally wanting on one side, and often broken up
into dots (see Fig. 4, colored Plate). So brilliant are they and so
noteworthy is their stellate arrangement, however incomplete, that
on this appearance alone a diagnosis of kidney disease is often haz-
arded. Fatty degeneration of the fibrous tissue (radiating fibres of
Miiller) explains these cases. The tendency to stellate form may
remain when the white deposits are very large and conspicuous; we
may find hemorrhages and diffused opacity without plaques. Again
the stress of the lesion may fall on the optic nerve, which will be
swollen, red, permeated with newly-formed vessels and infiltration,
the veins turgid, tortuous, and pulsating, and the arteries small, the
border indistinct and encroaching on the retina, which also looks
gray and swollen—the " Medusa head " nerve. (See Fig. 9, colored
Plate.)
The more usual phenomena are a nerve somewhat or not at
all swollen, red, or it may be of a dark and slaty hue, or even
dead white, with small vessels; there will be white or greasy
or grayish-white patches, covering sometimes a large area, per-
haps so brilliant and extensive as to resemble a cloudy sky in
sunlight. Sometimes they coalesce into a large and shining sur-
face around the nerve, which can with difficulty be outlined in its
midst, and the vessels will in places be obscured. There will be
spots of hemorrhage, small or large, upon or away from the vessels,
seldom numerous. Sometimes the white plaques develop so thickly
about the nerve as to become continuous and then the vessels will
by their irregular course and swelling show the hindrance to the
circulation, while the border of the nerve may be indistinguishable.
There will naturally be great variety in the pictures presented.
Should the case continue long enough the stage of atrophy sets
in. Hemorrhages and patches disappear, perhaps completely,
although the stellate figure at the macula is the most persistent •
the nerve grows pale, the vessels become small and bordered by
white streaks; branching fibrous lines may appear in the retina, the
hexagonal pigment may show irregular proliferation, especially
about the macula and at the extreme periphery. It has even been.
THE RETINA.
615
observed (Leber) that all characteristic lesions fade away and only
the ordinary signs of optic-nerve atrophy with reduced retinal ves-
sels remain (Fig. 2, colored Plate); yet very commonly some traces
of exudation continue. The process requires months. Vision is
sometimes not at all impaired, and it may be abolished. Every
gradation is possible. With large hemorrhages there will be sco-
tomata. The field is not limited, color sense not affected.
Diagnosis often is obvious, and yet in many cases very critical
inspection is needed to see minute lesions. While in the large
proportion of cases no hesitation need arise, we may not neglect a
thorough examination of the urine chemically and microscopically.
It may be that no traces of albumin may at the time be found, not-
withstanding the presence of Bright's disease, and this will chiefly
be in cases of the fibrous kidney, but it has also been found that
disease of the brain has in a few instances been attended with all
the ophthalmoscopic signs supposed to be peculiar to albuminuric
retinitis, and that the kidneys were perfectly normal (Schmidt,
Graefe's Archiv). Similar ophthalmoscopic appearances also oc-
cur in diabetes mellitus, in leucocythaemia, and with less degree of
likeness in pernicious anaemia.
Cause and Development.—The cause and quality of the kidney
lesion determine to a certain extent the behavior of the ocular dis-
ease. We have to separate the chronic types of renal disease
from those connected with pregnancy and the exanthemata.
Considerable study has been given to ascertaining at what stage
in chronic Blight's disease the retinal complication occurs, and
it is probable from statistics collected by Earlesx that it exists
more frequently than is generally supposed, because he found
retinal changes in one-third the cases of Bright's disease, and
of these ten out of thirty-one had no visual defect. Both he and
other observers give the comparative frequenc}7 at from 20$ to 33$
of cases of albuminuria; and because defect of sight usually occa-
sions ocular inspection, it seems probable that the renal disease or
rather the general degeneration of capillaries and small vessels
has advanced to a high degree at the time when the eye lesion ap-
pears. On the other hand, routine ophthalmoscopic examination of
all cases of albuminuria, as was done by Earles, would probably
show that the retinal complication is not postponed so late nor is
it so infrequent as most writers have indicated. In pregnancy it
has been seen at the third month and may be postponed until the
seventh or eighth. It has been known to follow miscarriage. De-
tachment of retina in both eyes has been noted by Dickinson2 in a
scarlatinal case, and by Wadsworth3 in a case of pregnancy. In-
1 Birmingham Medical Review, Feb., 1880.
J "Diseases of Kidney," p. 230. 8 Trans. Am. Oph. Soc, 1887, p. 574.
616
DISEASES OF THE EYE.
flammation of the retina alone, occurs in about three-fourths of the
cases, of both retina and nerve in about one-fourth, and of the
nerve alone (pure papillitis) in a very small percentage (Bull1).
Hemorrhages appear in more than half the cases, and in certain
exceptional instances they are the conspicuous and almost exclu-
sive lesion. If at the macula, central scotoma and great damage
to vision follow. " Hypertrophy of the left side of the heart with-
out valvular lesion occurred in seventy-nine cases out of one hun-
dred and three reported. There was hypertrophy of the left side
of the heart with valvular disease in sixteen cases. In eight cases
there was no cardiac disease at the time of the first examination,
though hypertrophy of the left side subsequently developed in all
but three cases " (Bull, 1. c.)
Uraemic blindness may complicate the retinal lesion, or occur
without it; it has been especially noted in scarlatina cases, and
usually ends in recovery, but the blindness may be permanent.
The nerve is generally much swollen. I once examined a case
immediately after an attack which lasted only a few minutes;
there was oedema of the nerve and retina, the arteries very thin,
the veins tortuous. The patient had waxy disease of kidneys,
liver, spleen, and intestines, and was extremely blanched.
Prognosis is better respecting vision than as respects life in
the chronic cases. In pregnancy complete recovery is frequent,
while atrophy in various degrees may occur. Even detachment of
the retina has been known to disappear (Hirschberg, Wadsworth,
1. a). Hutchinson observed in one patient retinitis and recovery
in a series of pregnancies, but finally she became blind by atrophy
of the nerve. While improvement in vision is frequent, complete
restoration is infrequent. The duration of life in chronic renal cases
after eye symptoms arise, is given by Bull in 103 chronic cases, 1.
c; viz., more than 50$ died within the first year and the majority of
these within six months; 17$ died within the second year. Sudden
death by apoplexy is not rare. On the other hand, a patient sev-
enty-four years old whom I saw and who had liad impaired sight
for one year, survived ten years. Often the patient becomes indif-
ferent to the eye trouble as the general malady increases. If, as
happens, syphilis be the cause of the renal disease prognosis under
correct treatment will be good.
After scarlet fever complete recovery is common. I have seen
a case of papillitis in both eyes following a very mild type of scar-
let fever in which perfect vision was retained, and each optic disc
was the seat of nodular masses of colloid degeneration, piled to the
height of 3 D and remaining substantially unaltered for seven
years. (See Fig. 13, colored Plate.)
1 Trans. Am. Oph. Soc, 1886, and Medical Record, July 31st, 1886.
THE RETINA. 617
Pathology.—By the kindness of Dr. John E. Weeks the follow-
ing statement is presented. His views are also set forth in the
Archives of Ophthal., XVII., 3, 277, 1888:
" The diffuse white patches which occur in retinitis albuminurica
are due to an escape of the plasma of the blood into the nerve-
fibre layer, where it coagulates over larger or smaller areas. Blood-
vessels passing through these patches are more or less hidden by
the exudation. In addition to the exudation which gives the white
appearance wherever it occurs, the tissues are oedematous from the
presence of a serous fluid, very little opacity being apparent. The
well-defined white patches which appear grouped in the region of
the macula usually originate from the exudation of blood plasma
Fig. 230.—Changes in the Retina in Albuminuric Retinitis.—1, Fibrinous deposits in the nerve-
flbre layer; 2, fibrinous deposits in spaces between Miiller's fibres; 3, blood corpuscles; 4, gan-
glionic cells; a, membrana limitans interna; b, nerve-fibre layer; c, ganglion-ceU, layer; d, inner
granular layer; e, inner nuclear layer;/, outer granular layer; g, outer nuclear layer; h, mem-
brana limitans externa; i, rods and cones. Camera lucida, x300 diameters.
from the capillaries and venules that dip down into the deeper
layers of the retina. This exudation occupies spaces in the middle
layers of the retina, made by the separation or spreading apart of
Miiller's fibres, and breaks the continuity of the granular and
nuclear layers. Miiller's fibres are put on the stretch and the
retina is much thickened where these deposits occur. These
peculiar patches occur chiefly in the region of the macula and optic-
nerve entrance. This fact is explainable on the ground of the
richer network of capillaries that is present in these parts of the
retina. The nature of the exudation in the deeper layers of the
retina is the same as that in the superficial layer, but it occurs in
larger masses in the deeper layers and from its position cannot be
so readily absorbed. It is therefore subject to greater changes.
After the deposit has existed for some months it is found to consist
618
DISEASES OF THE EYE.
of fibrin, coagulated masses resembling coagulated albumin, which-
subsequently degenerate into masses of a colloid nature, a few-
small globules of fat here and there in old deposits, and an occa-
sional bunch of fat crystals. The latter are rare. Blood corpuscles
are not infrequently met with in these masses of exudation. It is
evident that exudation into the retina in retinitis accompanying
albuminuria takes place under two conditions. First, from impair-
ment of the walls of the vessels due to mal-nutrition (without
thickening or the presence of any cause of obstruction to the
blood current), which causes leakage of their fluid contents. This
condition obtains in retinitis accompanying acute nephritis, per-
nicious anaemia and scorbutus, and is dependent on a depraved
condition of the blood. The vessels having the thinnest walls, capil-
laries and veins suffer first, and it is from'these that the escape of
the elements of the blood first occurs. Very little true inflamma-
Fig. 231.—Albuminuric Retinitis. Rupture of Wall of Vein.—1, Wall of vein; 2 and 3, blood
corpuscles in the superficial and deeper layers of the retina. Camera lucida, x250 diameters.
tion accompanied by increase of nuclei and the aggregation of
leucocytes takes place ; however, in rare cases this does occur, and
great impairment of vision due to connective-tissue changes in the
optic nerve may result. Purulent conditions do not develop. Ex-
udation may take place from the ciliary processes and iris.
" The second condition that causes exudation into the retina is
obstruction to the blood current. This occurs in retinitis accom-
panying chronic interstitial nephritis and is due to an endo-
arteritis. The changes in the retinal arteries are indicative of a
general arterial disease which may be most advanced in the
retina, and not become manifest in the kidneys until a later date.
The changes in the walls of the vessels are found in all the tissues
of the globe, the walls of the arteries being most affected. Afibro-
hyaline degeneration of the tissue elements immediately beneath
the endothelium develops, involving the elastic lamina and in some
arteries the muscularis also, particularly the inner layers. This
THE RETINA.
619>
degeneration leads to thickening of the walls at the expense of the
lumen. It may occur irregularly or may affect a vessel through-
out its entire course. The lumen may be partly or entirely obliter-
ated; when entirely obliterated the endothelial cells are crowded
together, forming the axis of the cylinder. Karl Theodorx speaks.
Fig. 232.—Section of Iris in a case of Albuminuric Retinitis. Marked Fibro-Hyaline Changes in
the Walls of the Vessels. Camera lucida, X 250 diameters.
of having observed fatty degeneration of the endothelial cells. If
it occurs it is not common. The capillaries, particularly in the
chorio-capillaris of the choroid, are often completely occluded by
fibro-hyaline degeneration of their walls. The veins are affected,
but to a less degree than the arteries or capillaries. The nerve
Fig 233.—Choroiditis in unusual degree, cross section.—1. Hexagonal epithelium almost intact;-
2, minute capillaries; 3, cell infiltration most abundant about diseased vessels; 4, large vessels
which have undergone colloid degeneration; 5, lamina fusca with moderate pigment proliferation.
fibres apparently become slightly thickened and in old cases under-
go granular degenerative changes, disappearing entirely. It is
extremely doubtful whether the fusiform enlargements of the
nerve fibres pictured and described in nearly all the text-books on
ophthalmology ever occur. In medulla ted nerve fibres this change
1" Ein Beitrag zur Pathologischen Anatomie des Auges," Dr. Herzog Karl
in Bayern, 1887.
'620 DISEASES OF THE EYE.
does take place, but the axis cylinders alone do not seem capable
of assuming such form. The ganglion cells become much enlarged
by imbibition, pass into granular degeneration, and disappear.
Miiller's fibres swell from imbibition and pass into a hyaline de-
generation. Fatty changes may occur but are not usual. The
granular and nuclear layers undergo but little change except in
old cases, when general atrophy of the retinal tissue results.
Granular degeneration of the bacillary layers occurs in advanced
cases. Hyperplasia of the retinal pigment is sometimes observed.
In certain cases the nutrition of that portion of the optic nerve im-
mediately back of the globe is seriously interfered with by the de-
generation of the walls of the vessels. Atrophy of the nerve fibres
follows, the connective-tissue trabecular become thickened, and the
function of the nerve is partially or completely destroyed."
Michel and Herzog Karl Theodor (1. c.) have shown that in certain
types sclerosis and degeneration of the vessels, especially of the
finer ones, precede all other changes and are found in the earliest
phases of the disease. The granule layers suffer severely, because
they lie between the retinal and choroidal systems, and with de-
generation of both of them their nutrition is greatly damaged.
The degeneration of the optic fibres is similarly explained, being in
proportion to the vascular lesions. The vitiated state of the blood
due to the damaged kidneys necessarily has a large share in the
deteriorating processes, and because of the altered state of the
vessels effete products are more difficult of removal. The lesions
of the papilla seldom pass beyond the lamina cribrosa, but have
been occasionally found as far as the chiasm, and even atrophy has
been noticed. The distinction made by Dr. Weeks1 into the gen-
eral classes of cases is also set forth in his article (1. c.) and has prac-
tical value in both prognosis and treatment. Many times the prog-
nosis in the cases with vascular degeneration is comparatively good.
Sometimes we have a blood dyscrasia brought about by acute
renal trouble: the kidney disease preceding the changes in the
retina. These are the cases of pregnancy, scarlet fever, diphtheria,
measles, and all forms of acute nephritis. In them oedema and
white plaques are the first evidence in the retina; hemorrhages
more or less numerous often follow. Another class depends
upon general (sj'stemic) diseased condition of the arteries, capil-
laries, and to a less extent of the veins. The eye symptoms may
precede the kidney symptoms. A slight hemorrhage near the
macula and a few bright dots will be the first tokens in the eye,
while oedema and white patches develop later. The larger pro-
portion of cases belong in this class.
1 Archives of Ophthal., xvii., 3, 277, 1888.
THE RETINA.
621
Treatment—The principal indication in the chronic cases is te
protect the eyes from strain and irritation. Local blood-letting is
inappropriate, and nothing is to be done which will reduce the
health. Confinement in dark rooms is not to be thought of; colored
glasses may be used if the light be offensive. The whole treatment
is contained in what is most suitable for the renal affection. All
the rules of hygiene, climate, clothing, food, and exercise, as well
as the proper medication, iron, diuretics, sometimes mild mercurials,
etc., are to be adopted which would be enforced if the eyes were
not at fault. For the cases of vascular degeneration Meigs recom-
mends carbonate of ammonia and digitalis.
Loss of sight during pregnancy, whether from purely uragmic
symptoms or from visible retinitis, raises the question of the pro-
duction of premature delivery. Loring1 published a case in 1882,
in which, at his suggestion, premature labor was resorted to, to
save sight in a woman who at three successive pregnancies was
the subject of atrophy of the optic nerves or rather of low neuritis
optica. This was successfully done during the third month and
achieved the desired purpose. No albumin was found in the urine,
yet the lesion was attributed to the kidneys. Howe,2 Pooley5a
Moore4 have recorded cases wherein the retinal disease was clearly
pronounced and by removal of the foetus sight was restored. Ob-
stetric authorities agree that albuminuria raises this question
because of the danger to life of both mother and foetus. The
grave significance of loss of sight as denoting advanced degenera-
tion of the kidneys, lends added importance to the situation and
must be considered as arguing in favor of interference. The
uraemic state of the blood is dangerous to the life of the foetus
when it has already caused lesions in the optic nerve and retina of
the mother, and with the prospect of convulsions and peril to the
mother's life, resort to artificial labor may be abundantly justified.
This point in the management of labor must in the future be re-
garded with more attention than it has received, and because there
may be lesions of the nerve or retina without impaired sight, in-
spection with the ophthalmoscope is strongly recommended, for
the same prudential reasons which call for examination of the
urine even though there are no urgent symptoms. Vision may
also be impaired with little or no visible lesion. The following
conclusion by Howe seems judicious: "The induction of labor is
'Trans. Am. Oph. Soc, p. 423, 1882, also X. Y. Med. Journal, Jan. 20th,
1883.
2 Am. Journal of Ophthahn., vol. ii., 5, 6, 1885.
8 Medical Record, Jan. 28th, 1888.
*N. Y. Medical Journal, April 17th, 188G, also Thompson, Medical Record,
March 3d, 1888.
622
DISEASES OF THE EYE.
warrantable when the retinitis appears in the early stage of preg-
nancy and persists in spite of proper treatment, but is not war-
rantable in the last few weeks, in spite of the greater ease with
which it is accomplished, unless the inflammation is unusually
severe."
Retinitis glycosuria is much less frequent than retinitis albu-
minurica. The features of both are to a great degree identical.
There can be no doubt as to the competency of diabetes alone to
cause retinitis, while it happens that in some cases sugar and albu-
min may coexist in the urine. It would seem that hemorrhages
are more frequent and abundant than in albuminuric retinitis.
They break into the vitreous, and therefore opacities in this struc-
ture are common. A case which I described in 1869, was one of
diabetes without Bright's disease, and occurred in a lady sixty
years of age. When first seen, in 1867, the retinal lesions were
slight, but they soon increased, and hemorrhage was abundant.
Improvement occurred to a marked degree, but a little more than
two years afterward the lesions returned, and sight was worse than
before; the general health had also materially fallen off. This
patient had iritis before the retinal trouble. Galezowski reports a
case in which iritis serosa and afterward acute glaucoma followed
the retinitis; iridectomy proved of no value and enucleation was
done. The essential lesion seemed to be copious hemorrhage and
its transformations. The bright yellow patches are less liable to
affect the macula; perhaps they lie more frequently between the
disc and the macula. Their form is more nodular and irregular
than is usual in the albuminuric type. Weeks reports hemorrhages
and acute glaucoma in one of his cases of albuminuric retinitis, 1. c.
For the literature see Leber, in Graefe and Saemisch, Vol. V., p.
596. Hemorrhagic retinitis is said to have been found in diabetes
insipidus (Loring). Amblyopia without visible lesion also occurs
in glycosuria, and is characterized by central scotoma for red, like
alcohol amblyopia. To this reference will again be made. Noth-
ing need be said as to treatment, because it is included in that of
the general disorder.
Retinitis leucocythcemica, also called splenic or leukaemic
retinitis, is extremely rare, and its appearances are given in Lieb-
reich's Atlas, Tab. X., Fig. 3. A peculiar orange hue pervades the
fundus and there will be both exudations and hemorrhages. In
persons of dark complexion the yellowish tinge may not appear,
but the swollen veins will not be dark as with normal blood (Hirsch-
berg1). The ocular conditions are described at some length by
Gowers.2 It may be characterized by hemorrhages, and Poncet
1 Centralblatt fiir Augenheil., April, 1887, p. 97.
8 "Medical Ophthalmoscopy," p. 190, 1879.
THE RETINA.
623
gives a figure which shows how in this, as in other retinal hemor-
rhages, degeneration of the vessels has prepared the way for the
accident. Loringx lays stress upon the great tortuosity of the
veins and their increase in diameter, while the arteries are less en-
larged or even reduced. De Schweinitz2 signalizes white spots
with red borders, especially near the equator and the macula.
They vary in size and are sometimes elevated. The3T consist of
leucocytes, surrounded by red blood-corpuscles. Sometimes albu-
min is found in the urine. Examination of the blood will reveal
the nature of the malady. Changes may occur in the choroid and
its tisssue be infiltrated with leucocytes.
Retinitis Syphilitica.—While a very large proportion of cases
of retinitis are caused by S3rphilis, it is not possible to establish the
cause by the local appearances, any more certainly than with
iritis. Certain types of inflammation are more common, viz., of the
anterior layers, yet the deep layers are also the starting point of
the disease, because it so frequently is associated with choroiditis.
It occurs as a primary affection, and it also accompanies iritis and
cyclitis, and then haziness of the vitreous may obscure its presence.
It comes with the secondary lesions, more rarely with the tertiary,
and it is also hereditary. As neuro-retinitis it occurs with syphili-
tic brain disease. The signs which we most often find are conges-
tion of the optic nerve, without swelling, its edge blurred, the veins
enlarged, the arteries normal, or even reduced; the retina at the
nerve-border is faintly striated, sometimes is gray or dark, and
about the centre of the fundus it has a faint haze, most easily dis-
covered by weak illumination; along the larger vessels, the whitish
exudation is more intense; bright white or yellow spots are not
very uncommon (these are shown in several plates by Ole B. Bull:
"The Ophthalmoscope and Lues," Christiania, 1884), and a consid-
erable patch of yellow exudation can occur, but is more rare. If the
vitreous be hazy, the veins will, by their distortion, best indicate
the fact of retinitis; but the diagnosis of retinitis then requires
great caution. One must see exudation to make it certain. Hemor-
rhages are infrequent. Fig. 17, colored Plate, might be an instance
of syphilitic neuro-retinitis with slight opacity. There is a marked
predilection for the central portion of the fundus, where little more
than a delicate cloud will be seen, and there may be minute yellow
points in an irregular group, perhaps erosion of epithelium and cen-
tral scotoma (Graefe). Relapses are frequent and characteristic.
Still another type has been called retinitis proliferans (Manz:
Graefe's Archiv, XXIL, III., p. 229, 1876), in which membranes
1 "Text-book of Ophthalmoscopy," vol. ii., p. 154, 1891.
8 "Diseases of the Eye," p. 434.
624 DISEASES OF THE EYE.
and bands of connective tissue spring out from the retina into the
vitreous and are often traversed by newly formed vessels. They
may originate from the nerve, and be so thick as to obscure the
fundus, or present large openings through which one may look
(see Fig. 16, colored plate). Hemorrhages may occur in the retina,
the nerve will be red and spots of exudation will be seen, white
lines course along the vessels, and vitreous opacities frequently
follow them. In time pigment proliferation and choroidal erosions
appear, and both these lesions may be very extensive. The condi-
tion is at length identical with choroiditis disseminata. The dis-
ease will be very chronic.
Again, we may find the retina covered with bluish-gray streaks
and patches of connective tissue, which lie below the vessels and
may be most abundant toward the equator. They have a fluffy or
sometimes fibrous look, and intermingled with them are choroidal
erosions. In all these conditions the nerve participates and from a
state of congestion often passes into the pallor of atrophy.
The retina may become implicated in cases of iritis, as has
already been stated, although haziness of the media makes its de-
tection difficult or impossible. In the case of a boy seventeen years
old ten days after the onset of gummy iritis, neuritis with plastic
exudation, pallor and reduced vessels, made its appearance and
was followed in a month by oedema of the disc and diffused faint
infiltration of much of the retina. Meanwhile the iritis subsided
and the media became clear. The other eye took on iritis, and
through the moderately hazy media the optic disc was seen oede-
matous and slightly swollen, the veins turgid and capillaries con-
gested. It may also happen that only the macular region is
affected and the lesions be very faint, consisting only of a finely
speckled or granular appearance. It also happens in pronounced
cases that the vitreous becomes filled with large flocculi, and the
choroid as well as the retina shows extensive lesions.
Visual disturbance may be very serious or moderate. It will
often be in no proportion to the visible alterations. With little
tissue-change the vision may be very bad, and vice versa. Meta-
morphopsia takes place from exudation into the macula, by which
the cones are thrown into disorderly arrangement—either spread
out, or. packed together more closely. In some cases objects ap-
pear too small, because fewer cones receive the image (micropsia),
and the contrary may happen, at least theoretically, if too many
cones be clustered into a given area (megalopsia). This last con-
dition is seldom, if ever observed, because if the cones become
condensed they usually undergo atroplry. No kind of glasses,
either spherical or cjiindric, can relieve the condition. The patient
complains that words or letters do not stand in regular line but
THE RETINA.
625
are thrown above or below adjacent ones. Lines drawn parallel
will show a curve at some point either in or out. Other disturb-
ances are scotomata, which may be central or eccentric, and some-
times are ring-shaped, with central vision unimpaired, and the ring
of dull or absent vision may be complete or incomplete. Forster
has noted a distinction in these cases of positive and negative sco-
tomata. The former appears to the patient as a dark spot in the
visual field, and is most emphatic in feeble illumination. Conse-
quently, a patient so affected seeks a strong light, and then reads
fairly well. The lesion may be due to retinitis circumscripta (at
the macula), to choroidal hemorrhage, or other choroidal disease.
A negative scotoma is not recognized by the patient under any
degree of illumination, whether strong or feeble; it is a color-
scotoma, and chiefly for red. The negative scotoma occurs in dis-
ease of the optic nerve, and in the amblyopia from tobacco and
alcohol. Color-perception in syphilitic retinitis is good, except in
the event of atrophy. Scintillations and phosphenes are frequently
complained of—one eye alone, or both eyes, or each in succession,
may be affected.
Treatment is constitutional, and consists of mercurials and
iodide of potassium. Regard must be had to the patient's general
condition, and to the stage of the disease. If tertiary, the propor-
tion of iodide will be larger; but mercurials, to the degree of toler-
ation without ptyalism, are essential. As Ave must often anticipate
a long period of treatment, care must be taken to keep up the gen-
eral health, and frequently the form of the remedy is to be changed.
The activity of the process will suggest the kind of mercurial.
Iron and tonics are often to be invoked. Should the disease be
hereditary, the same remedies are to be used, while cod-liver oil
and means of promoting nutrition are to be especially regarded.
In many cases serious complications occur in other tissues of the
eye, or there may be tokens of the disease in the brain.
Prognosis depends on the extent to which the system has been
tainted, and its r«_ pcnsiveness to treatment. Affection of the deep
layers of the retina is more unfavorable than of the anterior lay-
ers; therefore, cases attended by pigment-changes, or by choroidal
exudation and atrophy, are unfavorable. Atrophy of the optic
nerve, and of the retina, sometimes ensues. See Fig. 2, colored
Plate, in which the white lines along the vessels and the dirty blue
color of the nerve are the typical features. Of the uncomplicated
cases, very many recover, with no injury to sight. While the dura-
tion of many cases is from six to eight weeks, others are more
obstinate and may continue for years. When the macula alone is
concerned, there is great reason for anxiety, because serious im-
pairment of sight is prone to remain, and we find choroidal changes,
or permanent exudative deposits, or development of nerve-atrophy.
626
DISEASES OF THE EYE.
There is said to be a form of retinitis dependent on oxaluria,
and Michel says it occurs also in phosphorus-poisoning. In both
cases we have fibrous degeneration of the small vessels, and the
consecutive changes already referred to. In view of the frequent
occurrence of similar pathological lesions from diverse etiological
causes, it is proper to utter the caution that the ophthalmoscopic
picture cannot ever alone be safely relied on to inform us of the
constitutional nature of the disease. This conviction becomes more
and more strong as one's experience widens.
Retinitis punctata cdbescens (Mooren) is an excessively rare
condition and indicates a peculiar degeneration, in which minute,
isolated, white specks appear in the retina, behind the vascular
layers, and scattered over the fundus. There is very slight change
in the nerve, perhaps a little gray discoloration. Vision is moder-
ately impaired and we have little knowledge of either the pathology
or treatment. I have seen two or three instances.
Pigment Degeneration or Sclerosis of the Retina.
Retinitis pigmentosa.—The affection thus designated is a chronic
disease usually congenital, consisting of proliferation of connective
tissue in all the layers of the retina, atrophy of the nervous ele-
ments, intrusion of pigment into its tissue, with a tendency to follow
the blood-vessels. The same features belong to choroido-retinal
affections already considered, but with the difference that, unlike
them, the alterations are not confined to the outer and middle ret-
inal layers, but concern them all. Such is the anatomical distinc-
tion which Leber points out. The chief feature in the disease is
the pigmentary deposit, which comes from the hexagonal epithe-
lium, and so far from showing any of the usual signs of inflamma-
tion in enlarged vessels, exudation, and hazy vitreous, we have
simply the tokens of atrophy. The walls of the blood-vessels are
thickened, and their calibre reduced; the pigment-epithelium is in
some places atrophied, and in other regions its cells are multiplied
and penetrate the whole thickness of the retina. These lesions ap-
pear without the occurrence of any other disease, and without any
outward sign. On the other hand, they also arise in a secondary
way after other maladies, such as corneal staphyloma, irido-cho-
roiditis with closed pupil, exudative choroiditis, etc.
In regard to the pathology of this disease, much has been writ-
ten, and the process is interesting, but we must pass by most of
the details. The periphery of the retina is the usual seat and be-
ginning of the disease, and it advances centripetally toward the
macula lutea. The rods and cones are destroyed at an early stage,
and sometimes the retina is in spots converted wholly into connec-
Noyes' " Diseases of the Eye.
Chart II
w
Right Eye. Left Eye.
Retinitis pigmentosa. Extreme reduction of fields concentrically. J. D., set. 26. An uncle has
similar affection. Sight noticed to fail twelve years ago. O.D. V = &% ; O.S. V = f#. Fundus
like that of albino in absence of hexagonal pigment and distinctness of chorio-capillaris.
Retinal vessels at periphery bordered by pigment. Middle of fundus doted with pigment as
if sprinkled from a pepper box. Optic nerves dull, opaque, have a greasy look; vessels small.
Left Eye. Right Eye.
Retinitis pigmentosa with slight peripheral limitation of fields and broad ring scotoma
which encroaches closely upon the region of the macula. Dr. B. E. H., tet. 46.
O.S. +4.50 V=H? O.D. +4D. V = |§?
THE RETINA.
627
five tissue. Where the pigment is thickest, the retina and cho-
roid adhere together. The vitreous layer of the choroid is studded
with masses of colloid deposit, and the optic nerve is atrophic,
even up to and beyond the chiasm. Sclerosis of the choroidal ves-
sels occurs to some degree. Atrophy of the nerve accompanies the
later stages and may be complete.
The subjective symptoms are peculiar: there is a loss of periph-
eral sight, which gradually advances toward the centre, and for
a long time the central vision may, in good light, remain correct.
But the peripheral blindness impairs the patient's freedom of
movement, because it compels him to constantly turn his head to
acquaint himself with surrounding objects. In addition, these
patients when in dull light experience a grievous reduction of cen-
tral vision. At night they become almost helpless, and their
malady has hence been called hemeralopia. When walking at
night, they keep their eyes on the sky to help guide their steps, and
grope in much uncertainty. After a time central vision, even with
good light, becomes affected, and in the end total blindness ensues.
The symptoms in most cases begin in early life, while in a few no
trouble was noted until fifteen or twenty years of age. The consum-
mation of the disease usually comes after twenty to thirty years of
age. It is found in families, and inter-marriage of kindred has
been considered greatly instrumental in its production. Leber finds
about twenty-five per cent of the cases within this category. It is
sometimes congenital; see chapter on blindness. Some cases after
attaining a certain stage remain stationary at any rate for years.
Special peculiarities sometimes appear in the symptoms. I have
notes of three cases in which the central vision was good, while ex-
terior to the visual axis was a zone of blindness, outside was another
zone of good vision, and at the periphery again was blindness (ring-
scotoma). Such cases prove that for a certain zone in the peri-
phery the rods and cones remain intact, while across the interior
blind zone, where the bacilli are destroyed, the optic fibres con-
tinue uninjured. Again, Leber, who has studied this subject with
care, puts under the same general head certain cases of congenital
night-blindness, or amblyopia, without concentric field-limitation,
and which exhibit no pigmentary changes. They may remain in
statu quo for a lifetime, and certain remarkable examples of hered-
ity are given, extending as far as through six generations (see G.
and S., Bd. v., p. 650). In the congenital cases nystagmus is fre-
quent. The pupils act slowly, but respond to light.
To the ophthalmoscope the appearance is striking. Dots and
network of pigment are scattered over the periphery of the fundus
(see Fig. 234). Often the choroidal vessels are strongly brought to
view by atrophy of the retinal epithelium. They may also show
628
DISEASES OF THE EYE.
yellowish or buff outlines, which indicate their sclerosis. The retinal
vessels, both arteries and veins, are small; their walls thickened, pos-
sibly bordered with a whitish line, and upon them pigment will lie
in greater or less quantity. The optic nerve I have, in certain in-
stances, seen to be red, although not swollen; in most cases it will
be gray, and ultimately a dirty white. In all cases it is opaque, by
interpolation of connective tissue. In the last stage the nerve loses
its capillary vessels, and the emergent arteries are reduced to
threads. Opacity at the posterior pole of the lens is frequent; it
finally may become complete cataract. The rate of progress is
extremely slow. Vitreous opacities are not common, but I have
Fig. 234
noted them and seen the vitreous so fluid that accompanying cat-
aract was easily extracted in its capsule. I have also seen irido-
choroiditis ensue. Some cases which strongly resemble retinitis
pigmentosa are syphilitic, either acquired or hereditary.
In them the disease may be confined to one eye, but in the typi-
cal cases both eyes are involved. A certain proportion of deaf-
mutes have this lesion, and among idiots, as Liebreich showed, it is
not rare.
Diagnosis.—Very little difficulty can arise in ordinary cases.
Choroiditis disseminata is either in isolated spots, or most exten-
sive, and presents exudations and atrophy, with white blotches and
pigment-deposit in greater quantities. Sometimes choroidal lesions
will strongly simulate the retinal lesions, but neither is the diffi-
culty frequent, nor would an error be important. Neuro-retinitis of
acute type and syphilitic, is sometimes although rarely attended
Noyes' " Diseases of the Eye.'
Chart III.
flight Eye
A
Oct. 18th, 1887.
ISO
Oct. 18th, 1887.
flUjKbTje
BO go
/OO >0
130
Nov. 4th, 1889. Nov. 4th, 1889.
Neuro-retinitis pigmentosa. Miss A. T. B., p. 629
THE RETINA.
629
by pigment-deposit at the periphery, in this respect simulating the
disease in question, but the activity of the process sufficiently dif-
ferentiates it.
Donders has shown that the essence of the disease is not the
pigment-deposit, but the atrophy of the retina. The insensitive
region of the retina he found to be in advance of the pigment-
district, by throwing upon it, with the mirror, a very small flame,
which was not perceived, although not resting upon the pigmentary
region.
Hemeralopia, without pigmentation, is less prone to increased
loss of sight, and more frequently comes to a stand.
Prognosis is unfavorable, although the rate of advance is slow.
Treatment is of little value. Alteratives, derivatives, strychnia,
etc., have no permanent yet sometimes an apparent temporary
effect. Attention should be given to proper care of what sight
remains, and its economical employment.
Some have thought (H. Derby *) that galvanism mildly applied
has influence in retarding progress; others who have tested it have
found no benefit and in some cases apparent harm. There are
sometimes long pauses in the progress of the disease and some-
times, when the field has been excessively contracted, central vision
may remain fairly good and so continue for years.
Neuro-retinitis Pigmentosa.—At this point may be introduced
a form of disease which bears in many respects the closest analogy
to the pigment degeneration of the retina above referred to.
Under that description the occasional occurrence of inflammatory
lesions in a mild form was referred to, but that affection is an atro-
phy, while the type now considered is chiefly and notably inflam-
matoiw. The analogies between the types are the peculiar and
prolific deposition of linear and irregular formations of pigment,
resembling bird tracks, or oriental letters, bone corpuscles, etc., and
especially disposed to run along the vessels. There is in the present
type peripheral limitation of vision. The differences are that the
former is chronic, requiring many years, is not amenable to treat-
ment, and the latter is comparatively acute, has opacity of vitreous,
infiltration and hyperaemia of the nerve, and does yield to treat-
ment. In the latter a scotoma may develop, and be the conspicu-
ous feature. This is very unlike the ring scotoma sometimes found
in retinal pigment degeneration.
The features of the disease can be best portrayed by giving the
following case, now under treatment:
Miss A. T. B., set. 13, came to me in October, 1887. She is the second of
1 Trans. Am. Oph. Soc, 1887, p. 217.
630
DISEASES OF THE EYE.
six living children. Her mother's first pregnancy resulted in miscarriage at
6J months. The patient was born three weeks before full time and weighed
nine pounds. She has always been " nervous," never had any serious illness.
About three years ago it was noticed that she did not see well in going from
a light to a dark room. She would stumble over the baby, did not notice a
man who was laying a carpet. The trouble has increased very much within six
months and she cannot use her eyes. O.D., V = 0.5; O.S., V = 0.5; glasses do
not improve. There is no hereditary lesion except that cataract has existed
in the father's family. By ophthalmoscope : O.S., nerve seen with + 3D,
and so is the macula and the region between them. The macula appears as
a small red spot closely resembling a small bleb, and the retinal tissue between
it and the nerve is stippled, has a whitish reflex and is elevated above sur-
rounding fundus as if by effusion. Surrounding parts of fundus best seen
with + 1D. At the periphery are on all sides numerous pigment deposits in
the retinae. In O.D. are similar appearances save that the region of infiltra-
tion in macula, nerve, and included space is less elevated, being seen with
+ 2 D. Visual fields shown in chart. Ordered " mixed treatment."
"Within two weeks vision improved to 0.7 O.U. Four months later a
whitish reflex noticed around the region of the macula in each eye. Some
fine vessels concealed; nerve diffusely swollen and surrounding retina striated;
opacities in anterior part of vitreous. Three months later the exudation
about the macula had disappeared. Infiltration and redness of nerves the
same. Besides mixed treatment has been taking iron: " Blaud's pills."
In November, 1889, V. O. U. = 0.7. Vitreous in each eye less hazy; swell-
ing of nerves abated; redness less intense; surrounding radiation more de-
fined but less abundant, now resembles opaque nerve fibres. Pigment depos-
its have approached nearer the central part of fundus.
Visual fields show a scotoma as indicated in charts, and the several charts
show the changes wliich have occurred. The case is still in progress.
Detachment of the Retina. Sublatio Retinae: Amotio
Retinae.
Displacement of the retina by fluid effusion between it and the
choroid, when very pronounced and the retina opaque, can be
seen by the naked eye, but ordinarily is to be recognized only by
the ophthalmoscope. It presents various appearances, according
to the nature and quantity of the fluid. It may be partial or total,
and may occur at any Dart of the fundus. Its more frequent seat
is near the equator. It often develops within a few hours, but
may require a period of one or two weeks. The subject is some-
times unconscious when it occurs, and usually there is no pain. In
some cases there are premonitory flashes of light or of color. The
visual field is mutilated, although a degree of light-perception is
often present over the damaged part. When the central part of
the retina is not visibly disturbed, it may have good sight, but fre-
quently this is not the case. Objects may appear crooked (meta-
morphopsia) when the macular region is only slightly disturbed.
It is very common, both before and after the occurrence, to hear
complaint of muscee, and examination reveals floating vitreous
THE RETINA.
631
opacities, sometimes very numerous. It is almost the rule to have
diminished tension. Examination by the inverted image gives an
idea of the extent and relations of the fluid, while the upright image
tells the depth of the effusion and the details. What arrests atten-
tion is, that the retina bulges over a greater or less extent toward
the middle of the eye. If the membrane itself is not clearly noticed,
its disturbance is seen by the position of the retinal vessels. They
suddenly bend toward the centre of the eye, become tortuous, trem-
ulous, and dark in color, with no light-streak. They may take an
abrupt change of course, follow long curves, or wriggle in short
bends (see Fig. 5, Plate III.). They will generally undulate. The
retina may be clearly visible, because of a drab, gray, speckled, or
glistening appearance; on the other hand, it may be transparent
and difficult to recognize. If the amount of fluid is great, little
trouble will be had in diagnosis, but if small and transparent it
may take close inspection to discover the disease. Nothing
but a rippled surface may be seen, and this will sometimes be
the condition upon a region where effusion has taken place and
afterward disappeared. If the illumination is thrown upon
the edge of the detached retina, the choroidal vessels will come
out with great distinctness, in case the layer of fluid is thin
and transparent. The light is refracted through it so as to
make them appear to start forward. In some cases the sac
may look like mahogany, and then the effusion is bloody. I
have seen zones of different color, in different parts, passing from
mahogany to yellow and then to transparency. It is ver}7 common
to find whitish spots on the retina, or bright streaks, and some-
times cholesterin. It does not generally happen that the region
of the yellow spot is involved, but when this does occur, and the
amount of fluid is moderate, the fovea appears bright red, and con-
trasts vividly Avith the adjacent retina. This hue is in consequence
of the thinness of the membrane at this point, which permits the
choroid to shine through it. This is essentially the same explana-
tion of a similar contrast in embolism. In a traumatic case—a cork
popping from a bottle (Trans. Am. Oph. Soc, 1871, p. 128)—I was
convinced that the fovea had remained upon the choroid and been
torn from the surrounding retina. Precisely the same look I have
once since met with, although I have seen clear instances of a de-
tached fovea, and I therefore regard a central laceration as possi-
ble. It is very common to find a rent in the retina at its peripheral
part (see Fig. 6, Plate III.); there ma}7 be as many as three, near
together and parallel. Sometimes a loose tongue is torn up, and
through the opening the choroidal vessels are visible. Liebreich's
Atlas pictures such a condition most truthfully.
The fluid beneath the retina is albuminous; even when it is not
632 DISEASES OF THE EYE.
bloody it contains blood- and lymph-corpuscles, fat-cells and crys-
tals, pigment, and epithelium, etc. In a large number of cases the
vitreous is liquefied, as is proved by the tremulousness of the sac.
After a considerable time, the rods and cones become macerated
and destroyed, or by interstitial inflammation fibrous tissue may
be developed, and atrophy of nerve-elements ensue. Sometimes
the separated retina remains practically normal. This happened
in a case of choroidal sarcoma (Poncet), see Fig. 201.
The above description belongs to the cases which may be called
simple or idiopathic in character. But we find separation of the
retina as an outcome of a large number of morbid conditions of
various origin, in which cyclitis or choroiditis bears a part. Al-
most every case of chronic irido-cyclo -choroiditis has detached
retina. It is a pathological condition very common in museum
preparations. It appears often as a total detachment, and in its
last result presents simply a cord running from the optic nerve to
the ora serrata, within which no recognizable traces of the vitre-
ous remains (see Figs. 161,162, p. 431). In condemned eyes with closed
pupil the total want of perception of light often warrants the diag-
nosis of the above condition. As a part of the suppurative process
in purulent choroiditis or retinitis, separation of the retina occurs.
I have seen in such a case flecks of blood scattered thickly over the
whole retina* with yellow serum between choroid and retina. Cys-
tic degeneration of the retina is described by Iwanoff and Leber,
both in the late and earlier stages of the retinal disease; it is only
seen by the microscope.
In very many cases the lens in time becomes opaque and pre-
sents the characteristics of soft cataract.
Etiology.—Statistics collected in Horner's clinic1 of 300 cases,
show that about one-half the cases (48$) occur in myopic eyes,
while 16$ were due to injury. One-half the cases were in persons
above fifty years of age. In twenty-seven cases both eyes were
affected and the interval between the successive attacks varied from
three weeks to nineteen years. Besides myopia, which in the great
majority was of high degree, inflammatory processes of the iris,
ciliary body, and choroid and retina accounted for many of the re-
mainder. The immediate and active cause could often be set down
to some congestion due to overwork, strain, cough, hard drinking,
emphysema, etc. There is often more or less night-blindness, and
there may be much complaint of the dimness of light impression
for the preserved portion of the field. Leber speaks of a tendency
to confuse blue and green (Berry, p. 289).
Many theories have been advanced to explain the pathogenesis
1" Klinische Studien fiber Netzhautabl5sungen," Walter, Zurich, 1884.
THE RETINA.
633
•of the affection, and that which seems to be most satisfactory is
given by Nordenson.1 The condition precedent is the change of the
vitreous from a fine to a coarse fibrillar structure and its consequent
shrinking. As this goes on fluid is effused between it and the
retina, and the progress is usually from behind forward. If the
vitreous acquires adhesion to the retina, which it may do over a
large or small area, it will as it shrinks pull it from the choroid,
and in so doing it often happens that the retina is torn. As the
membranes are most intimately coherent in front of the equator,
this will usually be the place of rupture. When this opening is made,
the fluid vitreous rushes through and lifts up the retina, and the
effusion is simply a change of place and not an addition of fluid. As
a fact Nordenson found among 119 cases, a laceration of the retina
in 49 (38.6$); certainly it might exist in many others and because
very peripheral, not be open to discovery. This theory accounts for
many sudden cases, w7hile in cases which progress slowly and re-
specting whose duration or beginning no accurate history can be
elicited, it is fair to assume that the vitreous had adhered to the
retina over a large surface and with its slow shrinkage the retina
is pulled off and fluid is effused beneath it ex vacuo. The cause of
the lesion in the vitreous is to be looked for in chronic disease of
the choroid and ciliary body; in this we have the explanation of the
frequent implication of myopic eyes. It is not necessary to find
visible opacities in the vitreous to warrant the above theory, al-
though they are extremely common. We need not attempt to
account for some special peculiarities which are met with; the
elaborate treatise of Nordensen, who wrought out the explanation
first announced by Leber, will answer many questions.
Diagnosis.—Besides impaired sight we have the notable defect
in the field, and the ophthalmoscopic appearances will in the great
number of cases be easily interpreted. The inverted image will
give a correct idea of the relation of parts, while with the mirror
alone an unexpectedly distinct view of the separated retina appears
at a distance from the eye, denoting a high degree of hyperopia.
If the retina flutters much, this indicates fluid on both sides of it.
If it be tense and smooth it will lie in contact with the vitreous.
Difficulty in diagnosis arises only with small and transparent effu-
sions, but they will be revealed on careful study with the upright
image. An important matter is to recognize intraocular tumor con-
cealed beneath a separated retina. The most valuable distinction
is that as a rule in simple retinal detachment ocular tension is
minus, while with tumor it is plus. Some rare exceptions have
been noted, but the rule is in general a good one. There will be a
1 " Die NetzhautablOsung." Wiesbaden, 1887, pp. 255, with 27 plates.
634
DISEASES OF THE EYE.
history of slow encroachment on the field; there may also be hem-
orrhage into the vitreous with tumor. (See also p. 590.) If the
retina be thickened, and have whitish deposits mingled with a
mesh of vessels upon its surface, and be at the same time tense and
rounded, there will be great liability to suppose it to be a tumor:
especially when located at the periphery. I have been compelled
to wait the course of events which alone could settle the diagnosis;
and I have fallen into a mistake in recommending enucleation
when it was needless, because I was deceived as to the existence of
tumor.
Prognosis is, as a rule, unfavorable. An advantage attaches
to the descent of the fluid to the lower part of the eye, which
commonly occurs at an early date, because blindness above the
horizon is less injurious than blindness below the horizon. The re-
placed retina generally recovers part of its function; it happens, too,
that some absorption is common, and it is of the greatest conse-
quence to have the macula escape. But, even when it does not seem
to be involved, we may find metamorphopsia. I have already men-
tioned the dulness of sight due to torpor of the retina, and degenera-
tive changes are quite probable; moreover, a detachment is prone in
time to become greater or even total. It is a frequent thing to
have cataract occur; I found it twelve times among fifty-eight
eyes. Spontaneous recovery takes place in a few cases; I have
observed it four times—all were myopic persons—two were not
treated at all; one was treated antiphlogistically, and he had irre-
coverably lost one eye already by the same lesion; the fourth was
operated on by scleral puncture, and with no good result for three
months, but after that time a great portion of the fluid disappeared.
Leber reports a case which recovered spontaneously from five suc-
cessive attacks.
Treatment.—This will be either medical or surgical or both. A
widely prevalent practice has been confinement to bed for four or
five weeks, a pressure bandage on both eyes, and use of infusion
of jaborandi, or hypodermic injection of the muriate of pilocarpine
gr. £ to i, to active diaphoresis and salivation. The last-mentioned
remedies are often not well borne, and many have lost faith in
them. Improvement has been witnessed in many cases, as the
result of confinement, but it is extremely irksome and to many
subjects intolerable.
Therefore, an entirely opposite plan sometimes commends itself,
viz., to use such means as will best and most rapidly promote the
general nutrition, withholding the eyes from all use. This means
abundant food, exercise, and recreation, and avoiding everything
likely to cause congestion of the head, and is not inconsistent with
light purgatives and such medication as circumstances may indi-
cate.
THE RETINA.
635
A choice between these opposing methods may be determined
by the patient's general condition. If the health should be such as
to contra-indicate the treatment by bandage and close confinement,
it would certainly be unwise to enforce it. If not injurious to
health, it is most suitable to resort to it at the outset. To compel
absorption Dr. Carl Koller (personal communication), has had one
success by injections of pilocarpine reinforced by insisting upon
total abstinence from all fluids for several days and confinement
to bed. Cases of recovery are reported by those measures, and by
other means, viz., iodid potas., salicylate of sodium, tonics, etc.
To bring about the replacement of the retina there must
either be absorption of the underlying fluid or its transfer to the
front of the retina. A most natural suggestion is to let out the
fluid. Von Graefe and Bowman punctured the sac to let its con-
tents diffuse into the vitreous. Some gratifying results ensued, but
rarely were they permanent. Some cases were rendered worse; I
have had two such unpleasant experiences. At present this treat-
ment has been abandoned. Wecker at one time introduced a gold-
wire suture or seton into the sclera. This has not found general
approval, and some cases of destructive irido-choroiditis have been
caused. Puncture of the sac through the sclera was done to evacu-
ate the fluid externally in 1859 by A. Sichel, and has been repeated
since by Arlt, Alfred Graefe, Hirschberg, and others. A paper by
Hirschberg, reporting ten cases of scleral puncture, authorizes the
conclusion that no harm is done, that in most cases improvement
is gained, but that relapses are to be expected. Higgins reported
double optic neuritis with subretinal effusion in both eyes, and in
one tapping was done once, and in the other eye was done three
times, and in neither case with any benefit. Detachment in both
eyes during pregnancy has been reported by Adamuck, and others,
resulting from albuminuria and sometimes precipitated by eclamp-
sia. Premature delivery in Adamiick's case restored both eyes.
The vitreous is always assumed to be in a degenerate state to ad-
mit of the detachment of the retina.
Wolfe, of Glasgow,1 makes an incision upon the line of a meri-
dian six to eight millimetres long, first laying off the conjunctiva
and keeping it retracted by hooks, and by the large wound insures
free drainage of fluid. He has reported several cases of good re-
sult. When the retina floats freely, this is all that is required. If,
however, the retinal sac is smooth and tense, denoting a vitreous of
normal consistence, it would seem proper to puncture the retina at
the same time that the sclera is incised. Brailey gives three cases
of decided improvement after incision. In his cases, as in most
1 Annales d'Oculistique, 91, 92, p. 149, 1884.
636
DISEASES OF THE EYE.
others, the patients are put to bed with bandaged eyes for a week
or so, and may also receive diaphoretic treatment. An iridectomy
lance or a Graefe's knife may be used, and cocaine will suffice usu-
ally as an anaesthetic. The reaction is generally moderate.
On the whole the results of treatment are not encouraging, and
puncture has proved uncertain; either of no benefit, or if helpful
often for only a short period—in a few cases permanent, but in the
great number, as reported by many observers, relapsing in four or
five weeks. It may be said to be permissible but not obligatory.
So long as perception of light remains it may be employed. Sut-
phen reports a cure maintained for three years to last observation
(Trans. Am. Ophth. Soc, 1888).
Some French surgeons, notably Galezowski and Dransart,1 to
cure the separation have practised a broad iridectomy. The former
is less enthusiastic about it than he was at first, but still resorts
to it, especially when both eyes are affected. The latter reports
forty operations and eighteen recoveries. The duration of the
cases was from two weeks to about a year. The amount of vision
regained was in all the alleged recoveries, save one, very imperfect.
Further evidence is required to form an opinion on the real value
of this suggestion. Coppin2 reports upon eighteen cases treated
by iridectomy, and by the subsequent confinement to bed, bandage,
and derivatives, with the result of two successes, two temporary
improvements, eleven without benefit, and three losses. Some cases
are complicated with irido-cyclitis and synechiae, and to them iridec-
tomy would be applicable. After Wolfe's operation with a large
incision of eight to twelve millimetres and followed by such treat-
ment as above outlined for a month, in fifteen eyes he had eight
cases of improved vision lasting from five months to a year; in
one of these vision was £, while the remainder had £ to -gV; in five
no benefit was gained and two eyes were lost.
Evidently iridectomy does not offer much encouragement, and
the large sclerotic incision, even when reinforced by the derivative
treatment, gives a valuable success in very few cases. Injection
of tincture of iodine, two or four drops, into the vitreous has been
brought to notice by Schoeler, who reports five cases; three of these
continued to have detachment, but vision was much improved.
Abadie commends the treatment; Grosz and others find no bene-
fit from it; Schiess, 1890, in two cases obtained some absorption in
one case, and the patient had to be discharged prematurely because
of homesickness. In the other case the retina became replaced but
cataract ensued. This complication is reported by others. In his
1 Bulletins et Memoires de la Soci6te" Francaise d'Ophthalmologie, p. 178,
1886.
2 Ibid., p. 100, 1887.
THE RETINA. 637
original paper Schoeler (" Zur operatiren Behandlung und Heilung
der Netzhaut ablosung," Berlin, 1889) gives five chromolithographs
and other diagrams of the five cases on which he based his report.
He observed, in all cases but one, considerable reaction and vitreous
opacities. Treatment lasted for from four to eight weeks. He
emplo37ed hypodermic injections of biniodide of mercury (hyd. biniod.
0.25, kalium iodat 2.5, aq. distil. 25.0, of this one gram) once daily
into the muscles of the back while the reaction continued; also
atropine, etc. The cases on recovery presented reattachment of
the retina and spots of choroido-retinitis with pigment, evidently
indicating adhesions. Bull reports five cases with unsatisfactory
results, and in some great reaction (Trans. Am. Ophth. Soc, 1891,
p. 30).
Sometimes we are called upon to remove cataract, consecutive
to detached retina. By needling very little absorption is to be
procured. Only extraction is to be relied upon. The object is
simply in most cases to remove a blemish. It might be expected
that a partial detachment would afterward become more extensive
or total. This does follow, but does not necessarily occur. The
hope of improving sight by removing the cataract is in most cases
delusive.
The great frequency of this disease as a cause of blindness is
referred to in the chapter on Blindness.
Glioma Retinae.
Fungus Hcematodes of the Eye.—The subjects of the above dis-
ease are often infants, and none have yet been recorded whose age
was above sixteen years—the great majority are below ten. Very
rarely it is congenital. The cases are usually discovered by a bright
reflex from the interior of the eye, which arrests attention. Usu-
ally only one eye is involved, but both eyes may be implicated
simultaneously, or in succession. There will have been no prelim-
inary symptoms, and the anterior part of the eye may be normal.
The appearance varies with the extent of the growth. When
observed early, the ophthalmoscope or oblique illumination will de-
tect a white or yellow, or reddish-yellow surface, with blood-vessels
whose arrangement is more or less plexiform and unlike the retinal
circulation, and they will be minute, or possibly very scanty; excep-
tionally no vessels may be visible. The surface may be smooth or
nodulated. Inspection is easy, because the mass approaches the
nodal point. The retina may be in position or, more usually, it is
detached by effusion. The vitreous and lens are clear. As the
tumor grows, the lens is pushed forward; the pupil becomes slug-
gish and dilated—may become adherent; the anterior chamber is.
638
DISEASES OF THE EYE.
shallow; the anterior ciliary vessels are distended. If long duration
and sufficient growth be permitted, the coats of the eye give way
and bulgings appear. After a time the mass crops out, becomes
ulcerated, bleeds, gives forth sanious and fetid discharge, and a
projecting, hideous tumor may be formed. Pain begins with disten-
tion and external development, and cachectic symptoms soon arise.
Surrounding parts, such as the bones of the head or the brain, be-
come involved, and metastasis may take place to other parts of
the body. Death, either by exhaustion or by brain-disease, is the
final event.
Prognosis.—While fatal if left to itself, or if not cut short at an
early period, experience has shown that a very early removal of
the eye may save life, because in the beginning the disease is local.
We have authentic cases to verify this statement. Vetsch1 gives
thirteen cases, Hirschberg out of seventy-seven cases reports five
recoveries, i.e., 6.5$. He reports a congenital case (Centralblatt
fiir Augenheilkunde, April, May, 1884). I have had two permanent
recoveries or rescues in my own experience. When relapse occurs,
it is within a few weeks, perhaps within less than a month, or within
three months. Several children in the same family have been known
to be affected. Wilson reports eight children attacked, of whom
three died by this affection; the others survived ("Oph. Hosp.
Rep."). The most usual mode of death is by extension of the dis-
ease through the optic nerve to the brain. Should a relapse occur,
a fatal issue is almost certain. A single case is given in which the
patient survived after removal of a secondary tumor.
The Pathology of the growth has been carefully studied by
Xnapp, Hirschberg, Delafield, and others. It consists of small
round cells, similar to, but not, as once supposed, identical with the
granular layers of the retina, besides a scanty amount of fibrous
tissue and numerous blood-vessels. The starting-point is most
frequently from the external granular layer, but it can begin from
any other layers. Delafield remarks that the disease is anatomi-
cally identical with small round-cell sarcoma, and might be so
called. Hirschberg and Bull concur in this view. It may grow
outwardly or inwardly, more frequently the latter. Its prevalent
mode of extension is along the optic nerve.
Diagnosis.—There are several possibilities of error. 1. Glioma
should not be mistaken for choroiditis metastatica. In tne latter
case there will be a history of a preceding sickness, either of the
brain or of the spinal cord, or of both as cerebro-spinal meningitis,
and redness of the eye may have been noticed. There will usually
be a shallow anterior chamber, the pupil adherent and irregular
1 Arch, of Ophthalmology., Am. Ed., xii., 43, 1883.
THE RETINA.
639
the periphery of the iris retracted, the lens pressing against it, the
globe a little reduced, tension minus; the yellow mass may be vas-
cular, and the retina may or may not be detached. The symptoms
and history of an inflammation are the clue. It is conceivable that
an eye having glioma may be seen during the period of glaucoma-
tous outbreak and the existence of deep disease never have been
recognized, and at the time be masked by turbidity of the media.
Wadsworth reports one such case, and the cornea was suppurat-
ing. An incision to relieve the suppuration discovered the tumor.
2. Raab (Arch. f. Oph., XXIV., iii., 163) gives the anatomical de-
scription of three eyes enucleated as gliomatous, which had a pecu-
liar deposit of fibrous tissue behind the lens, resulting from cyclitis
or from choroiditis. In one case the lens and iris were pushed for-
ward, the pupil dilated to an extreme degree; there was a greenish-
white reflex, upon which red streaks could be seen; tension a little
plus; slight punctate opacity of the cornea; the peri-corneal and
ciliary vessels were injected. In the other two cases the facts were
somewhat different, but need not be repeated. In all, there was
increased tension, absence of inflammatory tokens in the anterior
part of the globe, and the presence of a light-colored mass in the
depth of the eye. As a rule in plastic cyclitis or choroiditis, there
will be diminished tension, tenderness on pressure, discoloration of
iris or ciliary hyperaemia. Yet inflammatory growths have been
mistaken for true glioma by the best observers.1 It must be ad-
mitted that a mistake in diagnosis would, under such circum-
stances, be pardonable.2
3. I have seen two cases, in young subjects, in which there were
intraocular deposits, resembling glioma in all microscopic appear-
ances, which ultimately terminated in the disappearance of the
mass. There were no signs of inflammation, and, for want of bet-
ter knowledge, they were called cases of " strumous " deposit.
4. Schiess (Basel, 1891, p. 48) reports a case in a child of three
years, of total detachment of the retina, presenting two nodular
white masses which, on examination, proved to be merely thickened
retina; yet the diagnosis was glioma. There was no history of
injury.
5. White sarcoma of the choroid is not found usually at the
early age when glioma occurs, yet I have one such case recorded in
a child eight years of age.
Treatment.—Medication is useless, and the only safety lies in
the earliest possible removal of the globe, with as much of the
1 " Untersuchungen fiber intraoculare Tumoren. Netzhaut Glioine," Dr.
J. R. Da Oama Pinto, 1886.
2 " Pseudo-glioma from Traumatism," Trans. Am. Oph. Soc, 1887, p. 485,
H. D. Noyes; also, Trans. Am. Oph. Soc, 1869.
640 DISEASES OF THE EYE.
optic nerve as can be excised. There is encouragement that life
can be saved if the disease be extirpated at an early date—the
operation would be proper at a later period to relieve pain, but at
the stage of fungus growth the undertaking may be too formida-
ble. The surgeon will act according to his discretion, in view of
all the circumstances. Should the painful condition present itself
of glioma in each eye, I should not hesitate to enucleate both, if,,
by so doing, a fair prospect of life could be secured. Carmaltl
reports removal of both eyes of a child two and a half years old,
for a glio-sarcoma in each, and that after three years the child was
living and health}7.
To remove an eye which is the seat of some disease that resem-
bles, but is not glioma, is a needless mutilation; but it may be
justly argued that it is better to err on this side than to fail to
remove an eye which is truly attacked by this formidable malady.
Moreover, the cases of pseudo-glioma may demand extirpation be-
cause of danger of sympathetic trouble to the fellow-eye.
There is one case on record of preservation of life when the dis-
ease reappeared in the orbit and was again removed. All its con-
tents must be taken away, and the walls of the orbit treated with
chloride of zinc. But the probability is against recovery.
1 Trans. Am. Surg. Assoc, June, 1892.
CHAPTER XVIII.
THE OPTIC NERVE.
Anatomy and Physiology.—Within recent years our knowledge
of this topic has gained so much in precision and richness that it
will be proper to give an account of it which shall clearly yet suc-
cinctly, set forth the recognized facts. Their bearing upon patho-
logy and upon clinical experience justifies or perhaps compels such
a rehearsal.
The optic nerve may be considered in three separate regions: its
cranial, its orbital, and its intra-ocular portions.
Within the skull the nerves are short and flattened, and they
diverge from the optic chiasm to reach the canalis opticus on either
side of the median line under the tip of the lesser wing of the
sphenoid bone. The chiasm is a flattened mass elongated trans-
versely and resting on the olivary process or body of the sphenoid.
The anatomy of the parts in this region is familiar; and it is only
needful to refer to the order of the organs arranged on the median
642
DISEASES OF THE EYE.
line beginning with the anterior perforated space and lamina ter-
minalis cinerea; behind, is the optic commissure or chiasm, from
whose posterior angles diverge the tractus optici, to wind around
the crura cerebri (peduncles). Abutting upon the posterior border
of the chiasm is the tuber cinereum, which narrows to the tube-like
infundibulum and terminates in the posterior lobe of the pituitary
body; and this is lodged in the sella turcica. Behind the tuber lie
the corpora mammillaria (albicantia, Candida); on either side of
Fig. 236.
and close to the median line and behind them is the posterior per-
forated space.
In Fig. 236 in which the parts are schematically portrayed,
the tractus opticus is seen passing around the crus cerebri
(German, fuss) to the corpus geniculatum laterale or externum,
in close relation with the corpus geniculatum mediale or internum
and reaches the posterior extremity of the thalamus opticus which
is called the pulvinar. Deeper connections are omitted. In Fig.
237 which is also schematic, the same relations are displayed
upon a nearly horizontal section. By this figure the following
facts are exhibited: Each optic nerve contains fibres coming from
the tractus of its own side (direct fibres, fasciculus non-cruciatus)
THE OPTIC NERVE.
643
and also fibres coming from the opposite tractus (crossing fibres,
fasciculus cruciatus). There is also seen a loop passing across
between the tractus which is the posterior commissural band of
Gudden: sometimes called inferior or arcuate commissure. Be-
neath this, but not exhibited, lies still another and much smaller
commissural band, which is Meynert's. See article on Chiasm, by
Stefan Bernheimer (Archives f. Ophthal, Vol. XX., No. 2, p. 163,
with illustrations, 1892), which fully confirms the above statements.
Fig. 237.—1, Corpora mamillaria lying upon the posterior perforated space; 2, Corpus geni-
culatum externum or laterale; 3,corpus geniculatum internum or mediale; 4,corpora quadrige-
mina to which proceed the brachia antica and postica.
Bindearm — Braehium. Schleife = Fillet or Lemniscus.
Zur Briicke = To the pons. Haube = Tegmentum.
Fuss = Pes. Hirnschenkel = Pedunculus.
Zum Thai. = To the thalamus. Strat. Zon.= Stratum zonulare.
The fibres of the chiasm intricately interlace, and the crossing
set are upon the ventral and inner (medial) region; the direct
fibres are upon the dorsal and lateral surfaces. The crossing fibres
are the more numerous as three to two. The question of the
arrangement of the fibres in the chiasm, as between partial and
total decussation, has been hotly contested. The proof that it is
partial is now conclusive. In fishes the opposite arrangement ob-
tains. The result is that the tractus opticus of the right side sup-
plies the right half of each retina; the tractus of the left side the
644
DISEASES OF THE EYE.
left half of each retina, because each optic nerve is made up of
fibres from both tractus. The separation between the temporal
and nasal halves of the retinas is by a line nearly vertical, as shown
by cases of homonymous hemianopsia, yet not always passing ex-
actly upon the vertical meridian—a circumstance to be dwelt upon
hereafter. The division is, however, sufficiently vertical and mesial
in each eye to enable us to locate the position of a lesion, either in
front of or behind the chiasm.
It is found that still other fibres enter into the chiasm, viz.,
some from the tuber cinereum, and some from the substantia per-
forata anterior and from the lamina cinerea terminalis which lies
just behind it in the recessus opticus. The fibres last mentioned
come down to the chiasm in part from the gray matter of the wall
of the third ventricle. The vicinity of the optic commissure to this
cavity makes its morbid conditions of great importance to sight;
for example, in hydrocephalus internus, etc. Among fibres derived
from the tuber cinereum are large ganglion cells (Wagner), which
are called by Meynert the basal ganglion. The fibres composing
the commissure of Meynert soon quit the tractus, penetrate in curved
lines the crura cerebri, and appear to terminate in the corpus sub-
thalamicum or ganglion of Luys which lies above and outside of
the corpora mammillaria. These commissural fibres of Meynert
and of Gudden serve to connect corresponding parts on opposite
sides of the brain and have no direct visual function.
Let us next trace the tractus to their sources. As they course
around the peduncles they divide into two roots, an outer or
lateral, an inner or medial. The outer root in part enters the cor-
pus geniculatum laterale (Fig. 237), and a portion goes beneath
it to the thalamus opticus at the pulvinar; still other fibres
crossing over the outer surface of the corpus geniculatum laterale
reach the more forward part of the thalamus, and constitute a
part of its radiating superficial striation (tectum thalami), stratum
zonulare in Fig. 237. Another set of the fibres of this group go tO'
the anterior corpora quadrigemina by the braehium anticum. See
Fig. 237 and Fig. 70, p. 150. This root of the tractus therefore
goes to the corpus geniculatum laterale, to the thalamus opticus,
and to the anterior corpus quadrigeminum (natis). From these
ganglia go forth the radiating visual fibres of Gratiolet, which in
the posterior third of the internal capsule finallj7 attain the gray
matter of the occipital lobe, indicated in Fig. 236, p. 642, as Stab-
kranz zu den optic. Centr. In their course they pass outside the
posterior horn of the lateral ventricle. It follows from this ar-
rangement that the same kind of hemianopsia will ensue from lesion
of the posterior part of the internal capsule and the designated
occipital region, as will be caused by lesion of the corresponding
tractus.
THE OPTIC NERVE.
645
The inner or medial root of the tractus goes to the corpus geni-
culatum mediale, Fig. 237, and by the braehium posticum reaches
the posterior corpus quadrigeminum (testis); a few fibres also go
there directly and another small portion go to the anterior corpus
quadrigeminum. Yet another bundle of fibres leaves the inner
root of the tractus before reaching the corpora geniculata to pass
through the outer part of the pedunculus (crus) cerebri and part
of it goes direct to the cortex of the occipital lobe. See left side
of Fig. 237. In this same bundle it is thought that fibres exist
which gain the cortex without traversing the primary ganglia;
but it is doubtful. If they do exist, they lend no support to the
theory of crossed amblyopia, which Charcot advocated and which
Gowers also is inclined to sustain. Stilling and Edinger emphati-
cally assert that yet another bundle of this group makes its way
through the crus to the pons Varolii, goes down alongside one of
the roots of the trigeminus to the posterior (sensitive) columns of
the cord. He calls this the radix descendens. By this bundle
are we enabled to comprehend the implication of vision which occurs
in locomotor ataxy. These fibres are indicated in Fig. 236 and in
addition other fibres reach the nuclei of the third nerve to establish
the recognized pupillary reactions. Out of the same internal root
of the tractus are developed fibres which terminate in the corpus
dentatum of the cerebellum. The inner root of the tractus is thus
perceived to be the principal medium of relation with the corpus
geniculatum internum and with remote parts; while the outer
root is destined chiefly to the thalamus, the anterior corpus quad-
rigeminum and the corpus geniculatum externum. These last-
named three bodies are the important endings of the optic nerve and
are known as the " primary visual centres." In the mass of gan-
glion cells and fibres (neuro-spongium) of which they consist, the
optic impressions are transformed in some unknown way and
conveyed by a new set of fibres, " the visual radiations," in the
posterior part of the corpus callosum, to the cortex of the occipital
lobe.
The internal or medial geniculate bodies and the posterior cor-
pus quadrigeminum (testes) take no part in vision. They are be-
lieved to be concerned in hearing. See M. Foster, " Text-book of
Physiology," Part III., p. 1089, London, 1890.
If both optic nerves are destroyed, atrophy ensues in a large
part of both tractus, the corpora geniculata lateralia, the anterior
corpora quadrigemina, and in the posterior part of the optic
thalami; but a part of the tractus is preserved, viz., the commis-
sures of Gudden and Meynert, which is ground for believing that
they have no direct function in vision. The corpora geniculata
interna and the posterior corpora quadrigemina are likewise not
visibly injured.
646
DISEASES OF THE EYE.
Striking testimony confirming the experiments of Gudden is afforded by
cases of congenital anophthalmus. See Haab (" Beitrage zur Ophthalmol-
gisches Festgabe, Friedrich Horner," 1881, p. 131), who examined the brain of
an idiot girl, 27 years old, in whom this condition existed. The body was small
and the head in relative proportion. The eyeballs were simply rudimentary,
their muscles well developed; the optic nerves and the chiasm were wanting;
the tractus simply small cords and composed of the commissural fibres of
Gudden and Meynert as prov-
ed by their destination. The
corpus geniculatum externum
(laterale) was entirely absent;
the pulvinar much reduced
in size. The anterior corpus
quadrigeminum not much re-
duced visibly in bulk, but
under the microscope it
showed manifest impairment.
The internal corpus genicula-
tum and the posterior cor-
pora quadrigemina entirely
normal in size and structure.
A few words on the
functions of the anterior
corpora quadrigemina
(bigemina). They are in
connection (1) with the
tractus opticus directly
by the brachia anteriora;
(2) with the corpora gen-
iculata lateralia and so
indirectly with the trac-
tus opticus; (3) with the
cortex of the occipital
lobe by the brachia an-
teriora and by the radia-
tion of the internal cap-
sule (Gratiolet); (4) with
the spinal cord by the
middle fillet (radix de-
scendens); (5) with the
nuclei of all the ocular muscles, and their relations to the pupils by
the so-called bundle of Meynert is especially important.
The connections of the posterior corpora quadrigemina are
much more obscurely known: (1) with the inner root of the tractus
by the corpus geniculatum mediale (perhaps also directly); (2) with
the cortex of the cerebrum and in both cases by way of the brachia
posteriora; (3) with parts belonging to the spinal cord (especially
the region of the acusticus) by the lateral fillet. That they have
i.—R,
Fig. 238.—Scheme of the Central Visual Apparatus.- ...
Retina, shaded where it is innervated by the left, clear where
innervated by the right hemisphere; No, nervus opticus;
Ch, chiasma; Tro, tractus opticus; CM, Meynert's com-
missure; CG, Gudden'sj;ommissure; I, lateral tract root;
_., sagittal medullary layer
of occipital lobe; Co, cortex (chiefly of the cuneus); Lm
median tract (Schleife).
THE OPTIC NERVE.
647
important relations to the auditory centres, and that the commis-
sure of Gudden subserves this function, is becoming a prevalent
opinion.
Fig. 238 condenses many of the above statements in pictorial
form, and its study will explain variations in visual disturbances
according to the different localities involved. For instance, disease
at No impairs only the left eye and all of its field; at Ch affects
the inner half of each retina and causes bitemporal hemianopsia;
at Tro causes right homonymous hemianopsia, and the same effect
ensues from lesions more caudad as far as the cortex.
After leaving the primary ganglia the visual fibres pass caudad,
as has been said, under the name of the optic radiation. The gen-
eral opinion has been that the whole region of the occipital lobe
and especially the cuneus is to be regarded as the visual centre.
Investigations by Henschen,1 whose elaborate and rigid analysis of
a large number of cases in great detail cannot fail to carry convic-
tion to the mind of the reader, establish a more exact and limited
region as the vital seat of vision. He shows that at a distance of
six or seven centimetres from the occipital apex the fibres are
gathered into a compact bundle which is about four mm. in diam-
eter. It lies at the level of the second temporal convolution. In it
the upper fibres correspond to the upper part of the retina and
the lower fibres to the lower part of the retina. The fibres to the
macula are probably more medial. The crossing and non-crossing
fibres lie in immediate proximity to each other and are inter-
mingled.
An extremely important and interesting discovery from
Henschen's analysis is that the really essential part of the
visual area is the calcarine fissure marked in his plates oc. Un-
less this is touched hemianopsia does not occur; when it is im-
plicated, hemianopsia is complete. This is a new fact, and for
proof one must study the cases and diagrams which Henschen pre-
sents. The subjoined figures represent the most conclusive cases
yet on record. In Fig. 239, case A (Henschen, Th. II., p. 324, Karte
G.), b37 Reinbardt, shows the lesion described, and there was no
other to account for the hemianopsia, although in the second and
third occipital convolutions were areas of softening, but which
could not, as will later be shown, cause this result. Case B (1. c,
II., pp. 386-394) was a hard drinker, who had no other nervous
symptoms than hemianopsia, and at the autopsy nothing was found
except what is shown in B and B'; the latter being a section of the
occipital lobe about five cm. from the apex. Some 500 microscopic
preparations of the brain were examined. The visual fields were
1 Klinische und Anatomische Beitrage zur Pathologie des Gehirns,
Upsala, 1892, Erster und Zweiter Theilen.
648
DISEASES OF THE EYE.
unequally divided, the line of demarcation running irregularly and
encroaching much on the blind halves. In case C, by Dr. Hun, of
Albany, N. Y., which is well known, the loss of sight was at the
upper quadrant only of each eye, on the left side, and the lesion
being at the lower part of the cuneus, the inference was that the
upper part of the cuneus answers to the lower quadrant of the
A B
Fig. 239.
retina. But the explanation must be that the upper lip 01 the cal-
carine fissure answers to the upper side of the retina and the lower
lip to the lower side of the retina. It must be added in further
proof of the above conclusions that Henschen depicts and quotes
many cases where the occipital lobes on the ventral, the posterior
and the external surfaces were affected, and some in which spots
THE OPTIC NERVE.
649
of the cuneus displayed lesions not touching the calcarine fissure,
and in none of them did hemianopsia appear. This point will be
again referred to.
It is, however, admitted that we find in the outer surface of the
occipital lobes, in regions not yet precisely defined, but lying near
the angular and supra-marginal gyri, areas connected with visual
acts in the sense of visual memories, and lesions in them give rise to
the singular symptom pointed out by Munk and called by him
psychic blindness, now spoken of as a form of apraxia. It has been
shown by many examinations, of which Henschen quotes several
(see Taf. VIII.), and one of the most carefully studied of which is by
Professor Donaldson,1 of Clark University, Mass., that in cases of
early or long-continued blindness the occipital lobes undergo
atrophy. Professor Donaldson says the atrophic area includes
the cuneus and angular gyrus, but does not pass over to the ven-
tral surface. The thinning which the cortex suffers is not the same
throughout the visual area, but is small in the cuneus and occipital
pole, large in the areas immediately surrounding it and finally small
again in the most outlying portions. Professor Donaldson thinks
the reason for these differences lies in the fact that the parts
most essential to vision are earlier and more highly developed
and more resistant to disturbing influences.
We may now turn back to a more detailed study of the optic
nerve.
As to the orbital part of the nerve: each optic nerve reaches
the orbit by the foramen opticum, or canalis opticus, and adheres
very closely to the wall of the canal by its external sheath. The
canal is five to six millimetres long. Important vessels here enter
the nerve—one is called the arteria centralis posterior. (It may be
well to call attention to the close proximity of the canalis opticus
and of the cavernous sinus which receives the ophthalmic vein, and
of the carotid artery, which sends off the ophthalmic branch to ac-
company the nerve in its passage through the foramen. The oph-
thalmic vein passes through the sphenoidal fissure.) From the
chiasm to the foramen, the nerve is usually about 10 mm. long,
while its orbital portion is 28 to 29 mm. long. It is round, and about
4 mm. in diameter. It touches the globe about 4 m. to the nasal
side of the optic axis and a little below it. It passes in the midst
of the ocular muscles, and is surrounded by fat and connective
tissue. It is somewhat sinuous in its course, and long enough to
permit free movement of the globe. As it proceeds forward it is
slightly twisted, so that the lower surface turns to the temporal
side. Within the skull the nerve has only a pial sheath; in the
1 "The Extent of the Visual Cortex in Man, as deduced from the study
of the Brain of Laura Bridgman," Am. Journal of Psychol., August, 1892.
650
DISEASES OF THE EYE.
canalis opticus we find an external or dural sheath, which also-
serves as periosteum, and within it is the internal sheath, more
delicate and closely attached to the trunk and also called the pial
sheath. Between the sheaths is a space occupied by delicate trabe-
cular of connective tissue and by lymph. This is continuous with
the arachnoid cavity of the brain, and may be injected from it, and
the cavities are lined by endothelium. Still another lymph space,
normally microscopic, lies beneath the pial sheath, but pathologi-
cally it is sometimes conspicuous. At a point varying from fifteen
to twenty millimetres distant from the globe, the nerve is pierced
by the arteria and vena centralis retinas, which enter it obliquely
and pass into the eye.
As the nerve gains the eye, its external sheath mingles with the
outer part of the sclera, the internal sheath passes inward a greater
distance, and mingles with the inner layers of the sclera. It also
sends into the nerve septa of connective tissue, by which it is sub-
divided into numerous fasciculi, about 800 (Schwalbe), but exhibits
a more compact mass when seen in cross-section than do most
nerves. On passing within the globe, not only are the enveloping
sheaths left behind, but the septa which isolate the fasciculi .also
turn aside and become attached to the adjacent sclera. The nerve
fibres now lose their neuroglia, and are reduced to naked axis-
cylinders. Thus liberated from all its accompanying connective
tissue, the nerve becomes transparent and of less diameter, viz.,
one and a half millimetres, and terminates as the optic papilla.
There is c mesh-work of fibrous tissue interwoven among the nerve-
fibres at the level of the sclerotic opening, which is called the
lamina cribrosa. It is made up of the connective-tissue sheaths and
septa above mentioned. This structure is the limit beyond which
inspection by the ophthalmoscope is impossible, and it is more or
less visible according to circumstances. The inter-vaginal space
passes into the sclera for a slight distance, viz., as far as the lamina
cribrosa. The optic-nerve fibres before they gain the retina must
not only pierce the sclera, but the choroid likewise; this they do
through a circular opening. The edge of the opening is sometimes
in close contact with the nerve, and sometimes a small space is left
through which the sclera can be seen from within as a ring.
Some facts as to the appearance of the optic disc, and the dis-
tribution of its vessels, have been stated on page 47 et sea. A
circlet of vessels (circulus Halleri) surrounds the nerve-head which
bring it into communication with both the choroid, the sclera and
the optic sheath, as well as with the retina. A much greater vas-
cularity is furnished at this spot than at any other outside point
of the globe. Stilling states that by the lymph-vessels of the
papilla some of the fluid of the vitreous humor escapes.
Noyes' " Diseases of the Eye."
Plate A.
6
THE OPTIC NERVE.
651
The number of fibres in the nerve has been estimated to be 438,-
000 (Salzer), and by Krause 1,000,000; one-fourth of them (Bunge1),
or one-third of them (Uhthoff2), are considered as belonging to the
macular region. Considerable interest attaches to the determina-
tion of how the various groups of fibres making up the optic nerve
are arranged. We have three sets for visual purposes, viz., the
macular, the direct (uncrossing), and the crossing fibres; besides,,
there are fibres for the pupillary reactions, and of these last we
know nothing. But of the visual fibres our knowledge as to their
arrangement is remarkably precise. Samuelsohri (1882), Vossius
(1882), Nettleship, Bunge (1884), and Uhthoff (1886), had singular
opportunities for defining the position of the macular group, and
the relations of the crossing and direct fibres have been ascertained
by several investigators through the study of cases of monocular
optic atrophy. Schmidt Rimpler's3 case is one of the most valuable.
But the subject has been handled by Henschen4 in his splendid
monograph in the most masterly way. Collating the work of other
persons and adding cases of his own, he has constructed a diagram
which is the representation of our present knowledge. It is, how-
ever, understood that scope must be allowed for individual varia-
tions. He traces the several groups through the orbit to the
chiasm and up the tractus. In Plate III. we have his conclusions.
Fig. 1 represents the retina of the right eye with the macular area in
the centre, denoted by the circle, and the papilla to the left. The retina is
divided by a vertical line into halves, of which the lateral (temporal) side is
supplied by the direct fibres and the medial (nasal) side is supplied by the
crossing fibres. The parts of the optic disc from which these fibres respec-
tively arise are denoted by the peculiar areas laid out upon its surface. Those
parts included within black lines go to the temporal side of the retina and
represent the direct fibres. The remaining portion composing an irregular
figure in red, contains crossing fibres, and they are destined to the nasal side of
the retina. But close inspection shows certain lines from parts of each of
these areas which lead to the macular region, and in a significant manner.
Their purpose will be understood by looking at Fig. 2, which is an enlarged
representation of the papilla. In it again the spaces marked out by black
lines indicate the direct fibres, and we find them in two bundles, one above,
another below, and the chief portions lie on the temporal side. From each
of these a small sector is marked off from which spring the fibres to the tem-
poral side of the macular region. These special subdivisions are seen also in
the papilla of Fig. 3, and from them go lines to the upper and lower quad-
rants of the temporal region of the macula. Again in Fig. 4 the intervening
area of the disc is marked on the nasal side with crosses, and on the narrower
1 " Ueber Gesichtsfeld und Faserverlauf im Optischendeitungs Apparat,"'
1884.
2 Bericht Ophth. Congress zu Heidelberg, 1884.
3 Archiv f. Augenheilkunde, xix., 1888.
4 Klinische und Anatomische Beitrage zur Pathologie der Gehirns, Upsala,,
1890, 1892.
652
DISEASES OF THE EYE.
temporal side with dots, and the latter space denotes the region from which
spring the crossing fibres to the macula. It is seen that the latter region
thrusts itself between the two bundles of the direct macular fibres like a
wedge. The whole macular area is represented by a sector of the papilla,
embracing about 90° on its infero-temporal side; a fact established by nu-
merous and conclusive examinations. This important point must always be
borne in mind, and the area is often marked by a physiological excavation.
It is also seen that the crossing fibres, which are usually heaped into an ele-
vation on the nasal side, cover the conspicuous part of the disc. This fact
explains certain appearances which occur in old cases of hemianopsia.
As we leave the eyeball, a section of the nerve just behind the entrance of
the blood-vessels shows a change in the relations of the two bundles of direct
fibres: they become narrower, more ribbon-shaped, and their temporal borders
approach each other, and this change continues until on a section near the
apex of the orbit we find an arrangement represented in Fig. 3. Now
the direct fibres form a crescent on the ventro-lateral side of the nerve, and
the crossing fibres fit into the figure in a gibbous area. The macular bundle
marked by dots is now ovoid and nearly central. At the optic canal, Fig. 4,
the nerve is flattened and the general relations of its constituent bundles are
not otherwise greatly changed. Just in front of the chiasm, Fig. 5, while
slight modifications are seen in the shape and position of the bundles, another
feature is more noteworthy, indicated by a few lines at the bottom of the
diagram running nearly horizontally across the bundle of direct fibres. They
are meant to mark the fact which Henschen is the first to clearly state, that
some strands of crossing fibres run across among the strands of direct fibres
and loop back again to their own side of the nerve. This interlacement was
seen by Michel and is thought by Henschen to explain in part his assertions
of the complete decussation of the tractus in the chiasm.
Fig. 6 displays a section of the front of the chiasm. In its middle are the
macular fibres for each eye, arranged in form of a baton, the direct fibres at
each end, the crossing at the middle. While the direct fibres to the periph-
ery of the retina are at each lateral edge of the chiasm, Henschen indicates
that at the extreme edges a few crossing fibres intermingle with the non-
crossing fibres.
Other diagrams are given to show the tractus at more caudal levels, and
they represent the crossing fibres as retiring to the ventral and mesial side
while the direct fibres appear upon the dorsal and lateral parts. The com-
missures of Gudden and Meynert form a considerable part of the tractus sec-
tion. Gudden's commissure unites the two corpora geniculata interna and
the posterior corpora quadrigemina, and it also sends a direct strand into the
optic radiation of each occipital lobe. The posterior corpora quadrigemina
are closely united by crossing fibres with one another, and a bundle also
passes into the lemniscus (fillet); Gudden's commissure seems by its termina-
tions to stand in intimate relations with hearing and perhaps even to be an
auditory chiasm. It remains intact when both eyes are destroyed, and
further confirmation of this view is found in the preservation of the internal
corpora geniculata when the cortical auditory centres in the temporal lobes
are extirpated (Knies, 1. c).
We have little knowledge of the function of MeynerVs commissure- it is
lost in the crus and seems also to pass into the ganglion of Luys beneath
the thalamus. '
THE OPTIC NERVE.
653-
DlSEASES OF THE OPTIC NERVE.
We are compelled to take account, not only of the head of the
nerve, as we see it in the eye, but of its intra-orbital and intra-
cranial portions and of its relations with the brain and the spinal
cord. It has come to be recognized that a proper investigation of
cerebral disease includes examination of the optic disc, and experi-
ence has shown that there may be in the latter moderate or very
marked or no alterations, and that visual disturbances are some-
times in a notable degree independent of the existence of demon-
strable lesions. That is to say, with notable physical alterations
there may be great or no disturbances of sight, while with a nerve
of normal appearance vision may be good
or otherwise.
We, therefore, are obliged to test all
the visual functions and also inspect the
optic disc in cases of cerebral or spinal-
cord lesion. As to visual functions, we
have to examine direct vision, if possible
by test types, and aided, if need be, b}7
suitable glasses: also the visual field (a
small perimeter which can be carried in
the hand has just been introduced by
Schweigger, which may be very useful in
bedside investigations either in hospital
or private practice (see Arch, of Ophth.,
June 1889, p. 187), the color sense, and look
for scotomata. With the ophthalmoscope
we are to view the optic disc both by the
direct and indirect methods, if possible.
The normal look of the optic nerve
and retina as seen by the ophthalmoscpe
is shown in Fig. 1, PL III. and in Fig. fig. 34o.-schweigger's Hand
1 colored Plate. The great variety of the
physiological appearances of the optic nerve has been referred to
on pages 46 and 114; much more might be said, but completeness
would prove both exhausting and unpractical.
Before entering upon the consideration of diseases of the optic nerve, it
seems necessary to refer briefly to selected portions of the anatomy and
physiology of the brain, in view of the fact that questions of cerebral lesion
and localization are often thrust upon us What was deemed necessary
in the case of the motor nerves and their cerebral origins and relations
seems equally proper when we enter the brain by way of the optic nerve.
The task will be made more easy by relying largely upon illustrations.
In Fig. 241 we have the cavity of the skull and the arrangement of the
€54 DISEASES OF THE EYE.
■dura mater, and also the base of the brain with the arterial distribution.
See also the base with the cranial nerves, p. 146.
Fig. 236, p. 642, gives the chiasm and tractus and primary ganglia. The
cerebral convolutions are shown in Figs. 242 and 243.
The " localizing areas " are shown in Fig. 244, for which I am indebted to
the kindness of Dr. M. A. Starr. The great motor region occupies the middle
portion of the hemisphere and runs over at the upper edge upon its medial
surface. The regions concerned with speech and hearing are below, for
taste and smell also below and on the mesial surface and for vision pos-
teriorly. The motor region responds likewise to nerves of sensation, and
the two functions are more or less intermingled. The frontal region seems
set apart for the higher mental functions. While individual areas can
be mapped out with a degree of accuracy, they shade somewhat into neigh-
Fig. 241.— 1, Sphenoid sinuses; 2, internal carotid artery; 3, inferior maxillary nerve- 4 ten-
torium cerebelli; 5, falx cerebri.
boring parts, and the intercommunication among separated and even dis-
tant regions is maintained in a most intimate manner by transcortical and
subcortical fibres whose arrangements we know very little about. Be-
tween the frontal and parietal region at the upper part of the hemisphere is
a space allotted to conjugate deviation of the eyes. This accords with both
clinical and physiological experiences. Some have set apart another region
about the angular gyrus for the same function, and when stimulated this
area will cause such movements. But this effect is simply in accord with
the fact enunciated by Knies that motor impulses to the eyes necessarily
emanate from the region to which visual impressions are conveyed and
these impressions are always binocular. The visual region becomes thereby
a centre for oculo-motor impulses and so-called visual memories and
hence impairment of this locality gives rise to so-called psychic blind-
ness. That motility of tlie eyes is not impaired by a one-sided lesion may
be explained by the continuous energy of the opposite hemisphere. It
THE OPTIC NERVE.
655
is, however, to be admitted that psychic blindness is a symptom which be-
longs peculiarly to lesions of the left angular and supra-marginal gyri and
not to the right. It varies as the subject may be right or left handed, as
does the motor centre for speech. Hence this region ought not to have a
special designation to this effect. It is known, moreover, that conjugate
Fig. 242.
•deviation from cortical lesion is not a localizing symptom (seep. 165) because
it ensues after lesions in most varied situations. tSo far as it has localizing
value it is to be traced to the anterior and upper part of the second frontal
convolution, to the corresponding point in the internal capsule, or midway
between them along the radiation, or to the pons, as set forth on p. 165. It
is also said that stimulation of the above-named anterior cortical region
causes opening of the lids, and here we find a special area for the levator
Fig. 243.
palpebrae controlling the opposite side. The reflexes which act upon the
movements of the eyes come, as common experience tells us, from almost all
parts of the cortex, communicate with the visual centres, and stand in most
immediate relation to the nuclei of the ocular muscles. The crossing of the
rootlets of the trochlearis and of the obliquus inferior gives rise to paralysis
of these muscles on the side opposed to the hemispheric lesion, while all
65G
DISEASES OF THE EYE.
other ocular muscles are affected on the same side. Another point to be
noted is that at the lower part of the motor area is a separation between the
regions for the lower face and for the upper face. The latter includes the
orbicularis and frontal muscles, and we know clinically that one set of
muscles may be paralyzed or be affected by spasm separately from the
Fig. 244.
other. Again we have had to refer on a previous page (see p. 155) to the as-
sociation between the facial, the abducens nucleus, and the hypoglossus
in the pons. We see again how closely the lower facial and the tongue
areas lie together in the cortex.
The motor speech area (Brocas) has long been recognized, but we now
yRoiissel 4.
Fig. 245.—I, Motor aphasia (type of Bouillaud-Beoca); II, centre for the face; in, agraphia (?);
IV, arm centre; V, leg centre: VI, verbal deafness; VII, verbal (psychic) blindness; VIII, move-
ment of the eyes; IX, Ferrier's and Walton's optic centre.
discriminate two principal kinds of aphasia, viz., motor and sensory. The
former includes inability to speak, aphemia; inability to write, agraphia;
loss of gesture language, amimia. The varieties of sensory aphasia are
mind blindness, viz., alexia and apraxia; mind deafness, viz., word deaf-
ness, music deafness, and paraphasia. The affections thus designated can-
THE OPTIC NERVE.
657
not be sharply localized because the disorders arise from disturbances both of
commissural fibres and of originating centres, and the intercommunications
are too complex to be fully unravelled. The lesion in aphemia (motor
aphasia, as generally understood) is in the posterior part of the third left
frontal convolution, or Broca's convolution; for agraphia, sometimes in the
base of the second frontal convolution, Exner; sometimes in the angular
gyrus, D6jerine Soc. de Biologie, Feb. 27, 1892. For gesture language
there is no known centre. The lesion in word blindness (psychic blind-
ness) is in the angular gyrus, extending forward into the supra-marginal,
and backward into the occipital. The lesion in word deafness is in the
posterior part of the first temporal and upper part of the second temporal
convolutions. In right-handed people the lesions are always on the left
side, in left-handed people on the right side. See Starr, 1. c, pp. 65-73: " The
various speech areas are all supplied by the middle cerebral artery, and the
frequency with which an embolus lodges in this artery, instead of plugging
a small branch of it, explains the frequency of mixed aphasia and the rarity
of pure forms, which are due to small areas of softening."
The Corpus Striatum.—This ganglion is in close relation with the cere-
bellum and with nuclei in the pons. It is also in connection with fibres that
come up from the muscle-sense tract in the spinal cord. Its functions are
therefore probably connected with securing co-ordinate and purposeful
movements. Destruction of this ganglion in the human brain, however,
produces no definite symptoms and lesions in it cannot be diagnosticated.
It is therefore called clinically a latent region.
The Thalamus Opticus.—The thalamus is in relation by its projection
fibres with the frontal, parietal, occipital, and temporal cortex (see Fig. 71,
p. 151). The fibres that go to the occipital cortex come from the pulvinar
and have to do with vision. We are for the most part ignorant of its func-
tions, although it has numerous masses of gray matter. A summary of
various theories may be found in Berger,1 pp. 17-19. Lesions of the posterior
part of the thalamus (the pulvinar) will produce partial blindness (hemia-
nopsia). Other than this, lesions of the optic thalamus produce no definite
symptoms which enable us to make a local diagnosis.
The internal capsule has already been referred to (see p. 157, Fig. 76), and
will again be mentioned under hemianopsia.
The Tubercula Quadrigemina.—Of these only the anterior are concerned
with vision, and the injury of one causes hemianopsia. They are so close to
the nuclei of the third nerve that when diseased some form of ocular paraly-
sis is probable, while general ataxy is likely to co-exist. If to these signs
hemianesthesia or hemiplegia be added the peduncles are probably involved.
The posterior quadrigemina are probably concerned with hearing and are
connected by the commissure of Meynert (the optic chiasm).
The Corpora Geniculata Externa and Interna.—Only the former have re-
lations to vision, and it is naturally very rare to find them exclusively
diseased. Henschen reports two cases (1. c, p. 65). In one the external
ganglion was entirely destroyed—there was homonymous hemianopsia.
Secondary degeneration had reached up to the occipital lobe, and gone
downward by the optic tract to both optic nerves. In both the cases the
tubercula quadrigemina and the lemniscus were involved (see p. 150, Fig. 70).
The corpora geniculata interna are probably concerned with hearing,
being affiliated with the posterior corpora quadrigemina. Lesions in the
1 " Les Maladies du Yeux, dans leurs rapports avec la pathologie generale,"
1892.
42
658 DISEASES OF THE EYE.
vicinity of the parts now referred to, bordering on the fourth and third
ventricles, give rise to other symptoms such as somnolence, forced move-
ments, loss of equilibrium, sometimes to polyuria or to glycosuria. It is
common clinically to have a complexity of symptoms whose analysis may be
far from easy.
" The red nuclei are connected with the anterior cerebellar peduncles on
the one hand, and with the lenticular nucleus and optic thalamus on the
other, and are concerned in securing equilibrium and the adjustment of the
body in space." See Fig. 70, which shows the relations of the primary visual
ganglia in the anterior part of the pons, as has already been described on p.
150, to the red nucleus and the nuclei of the third nerve, etc.
The Cerebellum.—" The cerebellum is connected with the pons, the cere-
brum, and spinal cord. The vermis, or median lobe, is the part which in
man is most important. Lesions of the lateral lobes or hemispheres pro-
duce few direct symptoms, and they are called latent regions. Injuries of
the median lobe, however, produce disturbances in equilibrium, forced
movements, and a peculiar form of inco-ordination in gait, which is known
as cerebellar ataxia. Lesions of the middle peduncles produce forced move-
ments also, the forced movements being either toward or away from the side
of the lesion, according as it is an irritating one or a destructive one."1
Tumors in the cerebellum are almost certain to be attended by optic neuri-
tis, and the general symptoms are usually conspicuous, as will be mentioned
hereafter.
Coloboma of the Sheath.—This at first sight would be thought
to be only a variety of that retraction of the choroid seen most fre-
quently in myopia, occasionally in hyperopia, and differing from it
in that it occurs on the lower edge of the disc. The apparent elon-
gation is always of moderate extent; it is not marked by pigment;
the border of the nerve runs into the exposed sclera or sheath im-
perceptibly and has often a bluish color and seems concave. While
the direction is commonly downward it mav also have a trend in-
ward. The hollow look and want of distinction between the nerve
edge and the crescent, and the unusual position will indicate the
diagnosis (see Figs, on Plate I., opposite p. 114).
Congenital defects of structure are sometimes observed, and
without referring to all the abnormal appearances, attention may
be called to signs of apparent atrophy or degeneration in loss of
brightness and translucency, in a bluish look and lack of clearness
of outline, possibly with or without any changes in the vessels.
Sometimes with these conditions function is normal, but true atro-
phy is sometimes congenital and due to intra-uterine inflammation.
Small cysts have been seen. Eversbursch and others have described
singular malformations.
Opaque nerve fibres (see Plate III., Fig. 2, and p. 545).
Sometimes a pigment spot is seen on the face of the disc, and
may be congenital. It is, however, very rare. It is extremely
'Dana, "Diseases of the Nervous System," 1893.
THE OPTIC NERVE.
659
common to have pigment deposit on the edge of the choroid, next
the papilla, and this is not ordinarily counted pathological.
Occasionally the disc is anatomically not circular, but oblong,
although this appearance is commonly the optical effect of astig-
matism (see page 114).
The presence of a mesh of connective tissue on the papilla or
sometimes running along the vessels, is not rare (see Fig. 246). It
is white or gray, semi-transparent, envelopes the vessels at their
emergence, and presents a great variety of appearances. It may
Fig. 246.
cover a large part or even the whole of the disc and may be very
dense. It is distinguished from opaque nerve-fibres by the irregu-
larity of the structure. The nerve with this exception looks nor-
mal, and no importance attaches to this peculiarity (see also Purt-
scher, Graefe's Arch., Bd. XL.).
Sometimes the vessels emerging from the disc turn to the nasal
instead of to the temporal side, and curve back to their destina-
tions.
Hypercemia.—Apart from inflammations, turgescence of the
capillaries and of the larger vessels of the nerve is found under a
great variety of conditions, and the greatest care is required to
distinguish from each other the symptomatic and the idiopathic.
By far the larger number of cases are symptomatic. Of these,
660
DISEASES OF THE EYE.
the majority are the effect of fatigue of the accommodation, as
found in refractive errors, spasm of the ciliary muscle, or other
causes of asthenopia. Another cause is cerebral hyperaemia, more
particularly of the meninges. It is not to be inferred that every
case of cerebral hyperaemia will reveal itself in congestion of the
optic nerve, but the concomitance occurs. In apoplexj7, the nerve
may be deeply red, and it may be pallid; no uniform sequence can
be asserted, as was once supposed. In cases of fracture of the
skull (or other injuries) hyperaemia is often seen, and, in general,
morbid processes at the base of the skull are apt to cause optic
congestion, if not inflammation. It is not safe to argue from this
symptom alone, but in a given case it will take its place among
other phenomena, and often have decided value in diagnosis. With
patency of the foramen ovale of the heart and general cyanosis,
venous stasis in the optic nerve is also seen. In those w7ho use
alcohol to excess the nerves are red. In plethoric persons there is
always fulness of the optic circulation, and the greatest scope must
be allowed for anatomical and physiological varieties. Hyperaemia
of the optic nerve is, therefore, of very uncertain value as a patho-
logical symptom.
In high degrees of hypermetropia it is always present, and the
nerve texture is generally streaked and marked with connective
tissue. We must always distinguish between hyperaemia with and
without opacity of structure. The latter may be recent or chronic,
and we must also look carefully for swelling as indicative of infil-
tration.
Anaemia of the optic nerve may show itself as a part of general
feebleness of circulation. A temporary arrest of circulation occurs
in some cases at the beginning of an epileptic attack, but the con-
trary has also been seen. Upon an attack of syncope the nerves
become pallid, as I have witnessed. In cholera, Graefe found the
current of blood still in motion through the vessels, during the last
stages of the disease, and remarks that the flow in the veins was
intermittent or jerky. Both arteries and veins were all extremely
small. Enough has been said on this point in the chapter on the
retina.
Hemorrhage may take place in the head of the nerve or within
its sheath. The first is easily seen and will be of variable amount.
It occurs after contusions, sometimes in new-born infants after
difficult or instrumental delivery, and in adults is the token of vas-
cular degeneration in various diseases, such as albuminuria, glyco-
suria, pernicious anaemia, etc. It may attend papillitis in its vari-
ous types, and thrombosis of the vena centralis, and embolism
of the arteria centralis. If it implicate the fibres destined to the
THE OPTIC NERVE.
661
macula, serious damage to sight occurs; otherwise vision may
suffer little.
Hemorrhage into the orbital part of the optic nerve or into
its sheath has been referred to on page 549. It comes after in-
juries, fracture of the base of the skull, of the orbit, from
diseased vessels, from pachymeningitis hemorrhagica. The oph-
thalmoscopic signs have been already dwelt upon I. c. More may
be learned about it by referring to Magnus, " Die Sehnerven Blu-
tungen," Leipzig, 1874, in which are related three cases with
two colored ophthalmoscopic plates. The symptoms have the
characters both of embolism and of perineuritis. The result in
these cases depends on the extent of the bleeding and on the com-
plications. Recovery may take place, or atrophy destroy the sight.
In Magnus's case, see p. 550, Fig. 202, the signs include gray exuda-
tion at the macula reaching to the optic disc and surrounding its
whole margin, though more pronounced on the macular side—the red
spot at the fovea centralis, circulation in the arteries reduced but
not abolished, the nerve swollen and red, not exsanguinated, the
veins small. At first peripheral vision was retained, but in five
days perception of light was destroyed and the nerve finally passed
into atrophy. The picture was taken six hours after the occur-
rence.
662
DISEASES OF THE EYE.
Priestley Smith* gives a diagram of the optic nerve Fig. 247 with
its sheath distended by a copious hemorrhage in the subdural space.
The bleeding took place into the sheath of each nerve. The man
had a fall, followed ten days after by a fit, and he died on the 13th
day in another fit. There was no optic neuritis, and at the autopsj7
it was found that hemorrhage had occurred in the left frontal lobe
and blood had escaped from its inferior convolution and extended
on the left side back to the cerebellum, both on the convexity and
the base. There was ampullar enlargement of each sheath behind
the eyeballs. In the diagram the blood lies external to the arach-
noid sheath, while nevertheless the sub-arachnoid space is filled
with clear fluid, doubtless the cerebro-spinal fluid which had been
forced into it. "The examination of the nerve discs was made
eighteen hours before death, and it is by no means certain that the
blood had at this time forced its way into the optic nerves/' The
case affords a perfect demonstration of the several sheaths and
the two inter-vaginal spaces.
Neuritis Optica.
Clinically we distinguish (1) papillitis affecting the head of the
nerve, (2) neuro-retinitis or neuritis descendens, (3) retinitis cir-
cumpapillaris or perineuritis, (4) neuritis retro-bulbaris. The
first two forms, including perineuritis, have well-marked and visible
features; the last often exhibits nothing to the ophthalmoscope.
It is sometimes a partial neuritis, i.e., affecting the axial fibres
alone, and is identical in chronic forms with toxic amblyopia.
1. Papillitis, in its purest and simplest form, is a serous infiltra-
tion with distention of the veins over all the retina, and while the
nerve is red and extremely swollen even to many dioptries, its tissue
is transparent and its diameter but little increased. There is nothing
more than venous and capillary stasis with their effects. But upon
such a condition other inflammatory lesions may be grafted, show-
ing gray exudations, infiltration of surrounding retina, with hem-
orrhages perhaps, etc. In pictures 7, 8 and 9, colored plates, are
seen gradations and varieties of the inflammation, from serous
oedema to deep infiltration and exudation belonging to neuro-
retinitis. See also Fig. 4, PI. III. Accepting the possibility in a
certain degree of distinguishing papillitis from neuro-retinitis we
cannot always make sharp distinctions and shall therefore describe
the various pictures which may be observed. When both nerves
are affected one usually follows the other, and the process is more
intense in one than in the other.
The most simple form presents a slight swelling of the disc with
'Trans. Oph. Soc. United Kingdom, vol. iv., p. 273, 1884.
THE OPTIC NERVE.
663
no change of color, no alteration of vessels, and the only recogniz-
able features are the low swelling and the want of transparency in
the tissue. It looks pasty and the appearance may be confined to
one half only. Such is the fact in a case now under notice in whom
only one nerve is affected, and the cause is albuminuria from syph-
ilis of remote origin. Here is oedema and true tissue change in a
very limited region. Yet this case may possibly be a true neuritis
reaching farther back.
In papillitis with dominant serous infiltration (choked disc), we
have swelling measured by two, three, or even six dioptries; the
mass is almost globular, and shows an extreme degree of parallax
when the observer shifts his point of view from side to side, Fig. 248.
The substance is bright red, the outlines are almost or quite obliter-
ated, and fine lines radiate into the retina; the arteries seem small,
and the veins are strangulated. Pulsation may exist in the veins,
and possibly in the arteries. In some
cases the surrounding retina is much
infiltrated, and again it is not. If it be
not, the border of the swollen nerve
will be well marked, and its increase in
size greatly emphasized.
In severe cases small vessels are ex-
tremely abundant, while the circula-
tion is evidently impeded, because they
wriggle in and out in a manner which
suggests the head of Medusa (see Fig.
9, Colored Plate, and Fig. 3, PL III.). Sometimes minute, or even
considerable hemorrhages appear. Such cases have pronounced
plastic inflltration often mingled with hemorrhages of the linear
type. The apparent diameter of the disc is much enlarged and it is
opaque, while translucency characterizes the simple choked disc.
Both varieties present great prominence of the papilla, and as a
rough measure of the degree it may be assumed that 1 D = 0.3 mm.
There may be acute papillitis with great exudation, and a
patch of yellow exudation at the macular region, resembling what
is seen in embolism, even to the bright red fovea and hemor-
rhages. There may also be opacities in the vitreous. I have seen
such a case which was monocular. This type approximates neu-
ritis descendens. While the intraocular end of the nerve is the
focus of lesion, a moderate amount is found in the orbital portion,
and if the condition be critically studied the deep-lying parts of the
nerve will not be found to have escaped as much as was formerly
thought. This will be referred to under pathogenesis. In the
great majority of instances both optic nerves are inflamed, and
this is almost the rule where intracranial affections are the cause.
664
DISEASES OF THE EYE.
But cases of one-sided neuritis occur in which the symptoms clearly
indicate intracranial lesions. Such cases are noted by Magnus,
Pagenstecher, Fieuzal, etc. I have had such cases under observa-
tion. We also have cases of cerebral tumor recorded, in which only
one optic nerve was affected, viz., one by Reich1 (details not given),
two by Hughlings Jackson,2 one by Pooley,3 one by Parinaud,4 and
one by Bouchut,5 also by Gowers and by a few others. The tumor
is usually on the side opposite to the papillitis. But for monocular
neuritis optica we are usually to seek the cause in the orbit, or at
least below the optic chiasm. It is common in such instances to have
other nerves involved. I have seen the third and fourth combined
with the optic, and the lesion was doubtless at the sphenoidal fissure.
I have also seen neuritis optica associated with paresis of the rec-
tus internus muscle, and the attendant exophthalmus and pain
indicated the situation of the disease to be in the orbit. On mon-
ocular papillitis, see Burnett and Oliver, Am. Journal Med. Sci.,
Jan., 1884, p. 138. In acute otitis media there may be monocular
papillitis and on the same side. In severe ear cases this lesion is
not very rare, especially with meningitis, as is set forth in the above
paper. Both abducentes may be involved, which implies a lesion
near the pons, and there may also be cerebral hemianopsia.
2. Neuro-retinitis, or neuritis descendens. The ophthalmo-
scopic appearance is usually less striking than in pure papillitis, yet
it resembles or merges into it in many cases. We have, however, less
swelling of the nerve, its borders are hazy or obliterated, the veins
tortuous and large, the arteries perhaps small. Some cases show
little swelling (Fig. 7, Colored Plate), the nerve will be deeply red,
while its texture will no longer be transparent, its edges will have a
corona of fine lines, and the arteries and veins be turgid. There may
in this and other cases be infiltration along the retinal vessels. If
the nerves have a plrysiological excavation, this may be recognized;
but it is not likely that the lamina cribrosa can be seen, because of
the infiltration. (For the opaque lines along the vessels, see Colored
Plate, Figs. 9 and 2.)
Again, more extensive changes can arise in which both optic
nerves and retinae participate, and are swollen, infiltrated, and
hyperaemic, while white or buff-colored specks or patches appear
in the retina, both in the neighborhood of the optic nerve and of the
yellow spot. They may be clustered in radiating streaks, in the
latter locality, precisely as are found in albuminuric retinitis.
1 Klin. Monatsblatter (Zehender), vol. xii.
2 Ophth. Hosp. Reports, vii.
3 Arch, fiir Oph. und Otol., Bd. vi., p. 27.
4 Annales d'Oculistique, Ixxxii., 19, 1879.
6Bouchut: "Ophthalmoscopic M^dicale," p. 144.
EXPLANATION OF PLATE III.
Fig. 1.—Normal fundus.
Fig. 2.—Opaque optic nerve fibres.
Fig. 3.—Neuritis optica with apoplexies of the nerve and retina.
Fig. 4.—Choked disc. Neuro-retinitis.
Fig. 5.—Sub-retinal effusion.
Fig. 6.—Sub-retinal effusion with partial absorption of fluid and laceration
of the retina, vide p. 630.
Noyes' " Diseases of the Eye.
Plate
Fig. 1.
Fig. 2.
^^^ ^H ™
JQ^r j
Wkf \ S¥fA V^l
^w '< % ^.'■'n'lj'J^B
<-^Ts. J;;A
1 ' f^A^k-
\..^j
/ .
^^*< f c 3&fiff< < 1 A^^JH E
Fig. 4.
Fig. 5.
Fig. 6.
THE OPTIC NERVE.
665
This is not a frequent occurrence, but one notable case of such
neuro-retinitis with brain tumor,simulating in the closest way the
retinitis of albuminuria is on record by Schmidt, but it is very
rare (Archiv fiir Ophth., XV., iii., 253). Some cases have fallen
under my notice.
Neuritis hemorrhagica or apoplectica exhibits features identi-
cal with those found in the similar affection of the retina. Such a
case which is monocular is now under observation. The loss of
sight, which was rather sudden in its beginning, has existed three
weeks; V in right f°- in left ff. In the latter, one is struck by the
extreme redness and swelling of the nerve. The papilla measures
between 3 and 4 D, is bloody red with hemorrhage and hyperaemia
and oedema; the arteries are reduced, the veins very large and tur-
gid and tortuous. The diameter of the disc is enlarged and its
border rather sharply marked. In the retina very near the nerve
on the nasal side are white exudation patches and hemorrhages.
The region of the macula and the rest of the fundus are normal.
Manifestly a phlebitis with thrombosis has happened in the nerve
near the globe and the papilla shows the natural effects in the ob-
structed circulation and oedema. The lesion is extending to the
retina and presents features resembling Fig. 3, Colored Plate.
3. Peri-neuritis was first named by Galezowski and has since
been depicted by Iwanoff and Wernicke (retinitis circumpapillaris).
It presents a moderate swelling of the periphery of the disc with a
depressed centre, the redness may be either marginal or general,
•and the surrounding retina exhibit striation and perhaps grayish in-
filtration. It is only a variety of the affection already described
and has been shown to be propagated by meningitis. Vossius has
seen it after traumatic orbital phlegmon. Alt observed it after
meningitis, and the cavity of the nerve sheath was obliterated by ex-
udation of fibrin and round cells. It is only needful to state its
-clinical features and cause. It has occasionally come under my
notice. We shall refer to retro-bulbar neuritis in a subsequent
section.
When the acute process abates, other features come out. Some
portion of the nerve may have a gray opacity, from formation of
connective tissue, and another part be red. The cases of most
acute infiltration may by slow gradations pass into gray or buff,
into a bluish or white color, and the result be connective-tissue de-
velopment and atrophy of nerve structure. On the other hand, the
nerve disc may regain transparency and its normal hue.
The length of time required for the culmination of acute neuritis
and for its entire retrogression, is impossible to be determined. It
has been seen to come on within a few days, and even in a single
night (Gruening), while its disappearance is always slow. Months
666
DISEASES OF THE EYE.
may usually be counted on, and Matthewson (Trans. Fifth Inter-
nal Oph. Congress, p. 63, 1876) reports a case of choked discs in
which the optic nerves remained in stcdu quo for three years. As
above stated, the termination may occasionally be in complete re-
covery, or in partial or total atrophy of the nerve-fibres. A state of
swelling, with a white and apparently flocculent texture, sometimes
remains a long time, and this has suggested the term " woolly "
(Hulke) (Fig. 8, Colored Plate). Gradually the swelling subsides,
and may eventuate in concavity. The borders for some time remain
fuzzy and obscure, but at length the choroidal margin comes out
black and distinct. White lines bordering the vessels continue for a
period, and at length may disappear. If a case be noted for a
sufficient time, the atrophic appearances ultimately succeeding se-
vere optic neuritis will in no wise differ from those which are seen
in cases of primary atrophy of the nerve. It has been thought that
a distinction could be made in this regard, but if sufficient time be
allowed, both processes will bring about the same ophthalmoscopic
picture. During a considerable period of time we may with some
confidence say that the atrophy in progress has been preceded by
inflammatory exudation.
A singular and rare accompaniment of optic neuritis has been
noted by Nettleship, Leber, Priestley Smith,1 and others, viz., a
persistent dropping of fluid from the nostrils. The subject is dis-
cussed by Berger and Tyrman,2 and in some of the cases quoted
there were polypi in the nostrils, in some there was anosmia, and
in some evident brain complications. The origin of the fluid has
been ascribed to hydrocephalus internus, and also to escape from
lymph vessels coming out through the lamina cribrosa. One case
of this type was verbally reported by Dr. C. S. Bull at the New
York Ophthalmological Society.
Subjective Symptoms of Neuritis.—There are no external signs,
usually not even in the pupil. There is no pain, except in case of
orbital affections—when there may be tenderness on pressure and
possibly swelling of the lids or proptosis. The central vision, as
has been said, may be normal in oedematous papillitis and' so
remain for months, but it at length fails, while in other and
acute cases it is reduced from the outset and continues to fail.
Should it be lost, the pupil will be enlarged and fixed. The more
chronic the course, the less will be the degree of visual change and
slower its decline. The visual field may undergo manifold modifi-
cations. Sometimes it will be normal, again there will be moder-
1 Ophthalmic Review, 1884, Jan.
8 "Die Krankheiten der Keilbein-Hohle und des Siebbein-LabarvntbP* "
Wiesbaden, 1886, p. 41. '
THE OPTIC NERVE.
667
ate peripheral contraction: there may be sector-like defects in great
variety or even hemianopsia either vertical or horizontal. Color
perception may be intact, but will suffer in the ratio of central am-
blyopia, and is often a valuable index of the progress of the lesion
and of the prognosis. There may be a central scotoma for color,
and peripheral perception be good. Color sense may be wholly
wanting or the failure may occur in the usual order of first green
then red and then blue. If the visual field steadily contract and
the color sense correspondingly fade, we may look for atrophy and
loss of sight; on the other hand, normal peripheral color sense in
the return of the red, then green, and finally disappearance of cen-
tral color scotoma, may be the course of the recovery, and mean-
while the visual field enlarges.
Ulrich asserts that papillitis does not greatly threaten vision in
itself, but that the danger arises
from consecutive atrophy, or from
concomitant pathological pro-
cesses in deeper parts of the nerve,
because the lesion sometimes
mounts upward. If an atrophic
state ensues, vision of course suf-
fers. Syphilitic processes not in-
frequently end in atrophy of the
nerve because of degenerative
changes in the blood-vessels which damage its nutrition, and the
same remark applies to syphilitic cerebral lesions.
The anatomical characteristics of neuritis optica are serous
and plastic infiltration, new vessels, hemorrhages, multiplication of
nuclei, thickening of the vessels, and especially swelling and vari-
cosity of the nerve fibres, sometimes deposits of granular masses
in the granule layers of the retina. At a later time we have con-
nective-tissue formation in the nerve and along the vessels with
disappearance of optic nerve-fibre structure. A notable circum-
stance which has excited great attention is distention of the optic
sheath in an ampulla close up to the globe. This has been much
dwelt upon in attempting to account for the occurrence of papil-
litis. See Fig. 249, taken from Pagenstecher and Genth, "Atlas
der pathologischen Anatomie des Augapfels," Plate XXXI., Fig.
1, and Fig. 133, De Schweinitz, " Diseases of the Eye," 1892.
But there is little doubt that this does not result from fluid flow-
ing down the dural space, but is rather the consequence of the
papillitis in loco. Gowers says, 1. c, p. 172, " The cause of the stran-
gulation is the compression of the veins by the inflammatory pro-
ducts Avithin the swollen papilla, and not, as once thought, their
compression within or behind it by distention of the sheath." This
668
DISEASES OF THE EYE.
distention is l>y no means alwaj^s present, at least in noteworthy
degree, 37et in certain cases, as in Priestley Smith's case of hemor-
rhage, see page 661, the fluid does travel down the dural or arach-
noid space, but there is not such uniformity in the event as to give
certainty to the " transport theory."
Besides the above-mentioned pathological changes, the head of
the neiwe may contain colloid bodies, see Fig. 250 (Pagenstecher and
Genth, Plate XXXII, Fig. 8), and these are displayed in a great
Fig. 250.
mass m the Colored Plate, Fig. 13, as seen by the ophthalmoscope
Interstitial neuritis, which is by far the most frequent, and may
be primary, or succeed the oedematous condition just described ex
hibits enormous cell-infiltration and increase of connective tissue
affecting both the neuroglia of the nerve-fibres and their interven
mg trabecular Round cells surround the fibres and the blood-ves
sels, and the latter are often greatly increased in numbers and
cause extreme swelling of the papilla. As the round cells subse
quently develop into connective tissue, which undergoes shrinking
the blood-vessels and nerve fibres in turn diminish or disappear
THE OPTIC NERVE. 669>
and atrophy ensues. According to Alt, two kinds of atrophy occur.
In one of them the nerve fibres become simply thinner, and we find
lying between them fatty cells, probably neuroglia-cells undergo-
ing regressive metamorphosis. In the other form, the nervous
element is represented bjr a grumous substance, formed of molecu-
lar fat-drops, that is detritus. This change may involve much or
little of the nerve structure, and it may at' any stage become sta-
tionary, or be continuously progressive.
The optic layer in the retina also becomes thin, while perivascu-
litis and interstitial retinitis are observed. The sheaths of the
nerve may be inflamed and hypertrophied.
The changes which occur in serous infiltration of the nerve are
well shown in Fig. 250, in which, besides the oedema, the retina
is seen to be crowded away from the papilla, and as a rather rare
condition colloid bodies* are scattered through the papilla. Proba-
bly this condition is not so rare as has been thought (see Colored
Plate, Fig. 13).
Again in Fig. 251 the swelling of the nerve by infiltration and
the proliferation of nuclei is well displayed. In Fig. 252 the separa-
tion of the sheaths from each other is Avell shown and the nuclei
are seen most thickly clustered together at the periphery and about
the central vessels. The inter-fascicular septa are strongly accen-
tuated.
«70
DISEASES OF THE EYE.
Etiology of Papillitis and of Neuritis Optica.—The attempt to
sever these affections from each other in their etiological relations
has little more success than the attempt to divide them in their
essential pathological significance.
It is probably true that papillitis more frequently signifies in-
tracranial or cerebral disease than does neuro-retinitis—yet each
may result from similar causes. If the theory of increased cranial
pressure be given up, as for the most part it is as the explanation
Fig. 252.
of papillitis, we only have remaining the difference of intensity in
the inflammatory action, to mark the distinction between the two
affections. Each comes from local and constitutional causes. If
monolateral, papillitis has usually a local etiology in the orbit or
erysipelas, etc. If bilateral, in the great majority of cases Ave have
disease of the brain as the cause, and most frequently tumors, per-
haps next in frequency will be injuries of the skull, then inflamma-
tion of the meninges, either simple or tubercular. Among tumors
are included neoplasms and cysts and tubercular deposits, etc.
Sometimes clots have this effect, but papillitis is seldom seen'with
apoplexy unless a hemorrhage breaks through to the base of the
THE OPTIC NERVE. 671
skull. It is also infrequent with hydrocephalus, either external or
internal (atrophy is more common). It occurs with acute otitis
media, as has been said, and is then apt to be monocular and to
imply meningeal complication. As respects tumors, neither their
character nor location nor size seem to be important. Yet their
location in the cerebellum is of all places most likely to produce
the lesion, and the tumor need not be bigger than a cherry. An-
nuske,1 Reich,2 and Edmunds and Lawford3 in long tables give
abundant proof of this statement.4 Papillitis is not a localizing
symptom in brain disease.
Papillitis often comes late in the progress of cerebral tumors.
Jackson gives an autopsy where symptoms of tumor existed nine
years, while papillitis appeared only six Aveeks before death. It
may arise very suddenly. It may appear and complete its course
before death occurs. On the other hand, it has been known to last
three years (Mathewson).
Reich, in forty-five cases of tumor with autopsy, found only three
where no papillitis occurred. The statistics of Bernhardt5 are mis-
leading in taking 22$ as the known frequency of choked discs in
cerebral tumors; because, as he says, in 47$ of the cases he discusses
nothing was said in the histories about the matter. He does, how-
ever, show by his tables that in 45$ of cases of choked disc vision
remained intact. The real proportion of choked disc in brain
tumors is undoubtedly approximated in the tables of Edmunds and
Lawford, 1. c, who, out of 107 autopsies, give a record of choked disc
in 77 cases, or 66$. Jackson has seen it in atrophy of the brain. In
a case of general hardening of the brain with large distention of
the ventricles by fluid, which I saw from the beginning, there were
only signs of atrophy of the nerve. Abscess of the brain may cause
papillitis, while abscess of the cerebellum seldom does. Tubercular
meningitis causes optic nerve lesion in 80$ of the cases, including
hyperaamia and effusion, but positive swelling occurs much less
frequently—in twenty-four autopsies it was found fourteen times
(Garlick6). See " Papillitis," by C. S. Bull, N. Y. Med. Jour., Feb.
21, 1891; and Morrow, Trans. Oph. Section Am. Med. Assoc, 1893.
The attempt to set apart the etiology of neuro-retinitis or
retinitis descendens, from that of papillitis can have only partial
success. The former may occur with tumors, but with less fre-
quencj7 than the latter. It comes from causes local in the orbit,
as erysipelas, injuries, periostitis; sometimes from orbital tumors,
etc. Also from empyema of the frontal sinus and from caries of
1 Graefe's Archiv, v. xix., iii., 165-300. 2 Klin. Monatsblatter, v. xii.
8 Trans. Oph. Soc. Unit. Kingdom, v. iv., p. 172, 1884.
4 See case of tumor of thalamus without neuritis, by Edes, with litera-
ture, ?sT. Y. Med. Record, May 24, 1890.
6 '• Hirngeschwulste," 1881, p. 23. 6 Med. Chirurg. Trans., lxii., 447, 1879.
672
DISEASES OF THE EYE.
the teeth (Vossius). Very often it comes from meningitis, simple
and tubercular; from congenital malformations of the skull; from
tumors in the substance or at the base of the brain.
We likewise have it not seldom as a purely local and limited
affection, an idiopathic neuritis which has no remote connections,
just as any other nerve may be thus impaired. In theoretical
statements too little stress has been laid upon this kind of idio-
pathic affection. See Hirschberg: Centralblatt fiir Prak. Augen-
heil., Nov., 1887, Ophth. Review, Jan., 1888.
As to constitutional causes, we have syphilis of the brain (gum-
mata) or of the meninges or of the nerve, and cerebro-spinal men-
ingitis. It comes from toxic agents, such as lead, albuminuria, gly-
cosuria. It follows typhus and typhoid and intermittent fevers,
pneumonia, measles, scarlatina, acute otitis media. About 6$ of
cases of multiple sclerosis exhibit it (Herter). It is not rare,
especially among youths of both sexes, from anaemia; in females.
from chlorosis, menstrual disorders and uterine disease (Mooren).
It is a common consequence of severe hemorrhages. It happens
at all ages; it may be congenital, and hereditary. Sunstroke has
been followed by neuro-retinitis (Holtz), and its occurrence in sym-
pathetic ophthalmia has been dAvelt upon (see p. 491).
Acute myelitis has, within a few years, been found to be accom-
panied by optic neuritis. The first case was published by Steffan
and Erb, another by Dr. Seguin, of this city, and I have joined Dr.
Seguin in contributing another.1 Dr. Chisholm has another. The
symptoms are those of acute, but moderate, neuritis optica, with
remarkable impairment of the visual field and of central vision.
There may be entire loss of direct sight; there may be any kind of
irregularity in the fields, including total abolition on both sides or
affection of one only; there may be repeated recoveries of sight
and relapses. The singular peculiarity of the cases has been that
vision, both direct and indirect, should undergo such great and un-
expected variations. The lesion of the cord was in its lower and
middle portions, as was fully manifested by symptoms of the blad-
der and the loAver limbs. No explanation of the optic neuritis has
been offered, although we may bear in mind that a root of the
tractus has been traced by Stilling through the crus cerebri along
the fillet to the posterior columns of the cord (see p. 591). The
agency of the sympathetic nerve has been invoked to explain the
optic neuritis, but this is nothing better than surmise. All the
cases have gotten well, both in respect to sight and to the func-
tions of the cord. In my oAvn case, large doses of iodide of potas-
sium were employed, gradually reaching three hundred grains daily,
1 Am. Journ. Med. Sci., July, 1879, 105.
THE OPTIC NERVE. 673
and Avere well borne. The case occupied about four months in its
evolution.
Note.—A case of double optic neuritis following myelitis is reported in
Annales d'Oculistiqu.e, tome eii., Jul., AoM, Sept., 1889, p. 123, Proceedings
of Soc. Fran. d'Ophth. «A man aged 30 is seized with headache and double
amblyopia. He has double optic neuritis. Very soon general neuralgia,
various paralyses, all the symptoms of myelitis ensue, and death occurs in
three months. At the autopsy are found plaques of sclerosis in the cord; the
optic chiasm reduced in size. Microscopically an interstitial inflammation
was found in the left tractus opticus, reaching the chiasm and propagated
along both optic nerves. Very remarkably, only the axial fibres of each nerve
were affected, leaving intact the peripheral fibres and the sheath, and the
whole process came to a stop at the middle of the orbital portion of the
nerves. Then another focus of inflammation appeared immediately behind
the lamina cribrosa, and produced the ophthalmoscopic appearances. There
were signs of perivasculitis abundantly. The disease was considered to be
syphilitic. The limitation to the axial fibres and the interrupted course is
very noteworthy, and the association Avith myelitis was demonstrated.
Pathogenesis.—When produced by orbital disease it is easy to
understand hoAV inflammation of the trunk of the nerve causes
SAvelling of the disc, because at this point the Aascularity is great-
est, expansion can occur only inward, because the lamina cribrosa
behind and the sclera on each side may be said to imprison it;
moreover, the absence of neurilemma predisposes the fibres to ex-
pansion by imbibition. The peculiar bulging of the sheath just
behind the globe was noticed by Stellwagin 1856. When Schwalbe
(1869) showed that the cavity of the sheath was a prolongation of
the arachnoid cavity of the brain, Schmidt-Rimpler, Manz and
others, assumed that the fluid came doAvn from the brain and caused
the papillitis. Leber, Trans. London Ophthalmolog. Congress,
held this vieAv in a modified form in 1881. To the naked eye the
optic nerve sIioavs no sign of inflammation behind the lamina crib-
rosa. Kuhnt* seemed to give evidence that the effusion in the
sheath caused degeneration of the fibres of the papilla. The ques-
tion arose Avhether the papillitis causes the effusion in the sheath,
or 1 he effusion causes the papillitis. Alt gi\es a case of peri-neuritis
following meningitis, Avith obliteration of the cavity of the sheath.
Oeller2 also gives a case of papillitis and other lesions following
cerebro-spinal meningitis without any changes in the sheath.
But other theories have been advocated and much discussion
arisen on this subject; the A~aso-motor neiwes (Benedict, Hughlings-
Jackson), and the trigeminal nerve (Loring); oedema of the brain
extending through the nerve (Parinaud); meningitis at the base of
the brain (Edmunds), etc., have been invoked. Careful study of the
trunk of the nerve by the microscope has shown that notAvith-
1 Graefe's Archiv, xxv., iii., 250. 2 Jahresbericht fur 1879, p. 443.
43
674
DISEASES OF THE EYE.
standing its frequently apparent normal look it is the seat of a low
grade of inflammation, which penetrates it by the pial sheath and
the entering trabecule, and thus reaches the interstitial connective
tissue, and also folloAvs doAvn the nutrient blood-vessels. The in-
terspaces are expanded by serum (Edmunds, Poncet), and nuclei
are abundant. Edmunds found at the periphery of the nerve the
most abundant infiltration (see Fig. 223). The name choked disc
comes from the theory of mechanical engorgement first propounded
by Graefe, which attributed the cause to interference with return
circulation at the cavernous sinus; still another theory of a similar
kind regards increase of pressure within the skull as the causatiA-e
agent. This doubtless has a degree of value, but not great, as
proved by the cases of hydrocephalus internus. It seems probable
that more than one factor enters into the result, but the most
potent cause is now held to be a low grade of neuritis proceeding
down the nerve, or following along the sheath. Mechanical hind-
rance at the ocular end of nerve, by compression of the retinal ves-
sels, aggravates the swelling of the papilla. To quote Gowers,
" distention of the sheath of the nerA7e alone is probably insufficient
to cause papillitis, but may, perhaps, intensify the process other-
wise set up, leading to retention or augmentation of fluid in the
lymphatic spaces in the nerA-e-fluid w7hich may in some cases pos-
sess an irritative quality."
It was Leber Avho in 1881 originated the idea that fluid com-
ing down the sheath by its chemically irritating properties pro-
vokes papillitis. Deutschmann, pursuing the suggestion (1887),
experimented with fluids charged with microbes (bacillus tubercu-
losis) which he injected into the cranial cavity of rabbits, and
produced papillitis and vaginal distention. By simple distention of
the sheath with unirritating fluid no result folloAved. He therefore
believes irritating properties to be essential to the material Avhich
is conveyed to the papilla. He expressly disclaims any difference
between papillitis and neuritis descendens, except in degree, and de-
clares that intracranial pressure is inoperative, except as it favors
the penetration of pathogenic material into the sheath. Dropsy of
the sheath is not necessary to papillitis, but may be wholly absent.
Parinaud, Annales d'Oculistique, p. 26,1879, assumed that there
is oedema of the brain and hence of the nerve and its lymphatics.
He recognized the low grade of optic neuritis present with papillitis
which other observers failed to see. His view may be too sweeping
as to oedema of the brain, and we perhaps cannot fully account for
the process by the method of Leber, nor does the suggestion of
Parinaud tell us how the inflammation of the nerve arises, but the
facts which he was the first to detect in the nerve are confirmed by
THE OPTIC NERVE.
675
Picque,1 and Avith these Ave may at present rest, having at least
gotten rid of some untenable and disproven theories. He finds
that in the great majority of cases the inflammation is propagated
by continuity of tissue along the meninges to the papilla; it enters
the nerve, and may be very slight or more pronounced, whatever
may be the originating cause of the process. He thinks the papil-
lary oedema or stasis results from the concomitant meningitis and
neuritis. Accepting in some degree the views of Deutschmann and
Leber, he finds the intervention of germs unnecessary.
Diagnosis.—What has been said is enough to establish the ex-
istence of papillitis, but we are also called upon to make the diag-
nosis of the cause and relations of the morbid condition of the
nerve. We may in some instances assume a purely local lesion
with no remote causation, but Ave often have to wrestle with the
problem of the originating lesion of the brain, its nature, and its
seat. We have first general symptoms of brain disease, and we also
have focal symptoms. The former are called distant, the latter
localizing symptoms. The distant or general symptoms are due
either to brain irritation or to brain compression, or to both com-
bined. They cannot always be separated, but those of irritation
are usually associated with hyperaemia and consist of headache,
vertigo, vomiting, photophobia, mental irritabilitj7, insomnia, pe-
culiar feelings of fulness and pressure about the head, noises in
the ears or in the head, tenderness about the scalp, and, in severe
cases, convulsiA7e symptoms and delirium.
The symptoms of brain compression are headache, vomiting,
mental hebetude, or dulness, perhaps some form of paralysis, con-
tracted pupils, and eventually coma. With these there is often con-
stipation and retracted abdomen. Pressure symptoms may be
associated Avith anasmia or oedema, and often in states of malnutri-
tion Avhere the brain is impoverished the symptoms much resemble
those of compression (Dana). For local diagnosis we are guided
by the focal symptoms, A'iz., those characteristic disturbances,
either paralysis, convulsions, or perversions of sensation belonging
to the portion of the brain affected. It is in this inquiry that ade-
quate knowledge of cerebral anatomy and function is essential.
At the same time our knowledge is incomplete, and we cannot
always reach a correct conclusion. For example, it is held that
headache, vertigo, vomiting, staggering gait, and optic neuritis
are positive symptoms of tumor of the cerebellum, the staggering
being especially significant of disease of its middle lobe. In
Henschen (1. c, Taf. XIX.) is a plate of a tumor of the tuber cinere-
um beneath the optic chiasm, and another from the mid-cerebellum
1 Archiv d'Ophthalmol., Sept., 1888.
676 DISEASES OF THE EYE.
resting upon the fourth ventricle which were not diagnosticated
during life other than as a tumor cerebri. Symptoms were not
sufficiently marked to make localization certain, although the
patient was in hospital twelve days before death and capable of
answers for ten days. There was optic neuritis.
Another item in diagnosis is as to the nature of the pathological
process, whether hemorrhage, tumor, abscess, meningitis, soften-
tening, embolism, etc. On all these points reference must be made
to treatises on diseases of the brain, such as Starr, Dana, Gowers,
etc.
Prognosis Avill be extremely various. It will depend mainly on
the cause and the severity of the lesion. The first consideration
is to determine if possible Avhether some grave disorder, general or
cerebral, is to be dealt with. If no malady of an incurable nature
has found lodgment, the eye trouble may disappear.
With a duration of amblyopia for months a partial or complete
restoration is possible, and this may sometimes be true with total
amaurosis. The underlying cause is the great factor. Better than
by objective appearances Ave must be guided by the visual and
color sense as to the true progress of the neuritis.
Reference to the special causes of the disease may more satis-
factorily set forth the probabilities of a case in hand. Meningitis
may cause neuritis of one or both sides. In acute tubercular men-
ingitis, visual damage will depend on the acuteness of the process
(see Fig. 15, Colored Plate), and, as depicted, it may be attended
Avith choroidal tubercles. There may be great obtundity of intel-
lect and attendant visual depreciation. Cerebro-spinal meningitis
may cause optic neuritis and useful vision may, as I have seen,
remain in one eye despite obvious atrophy. With chronic menin-
gitis it is more usual for blindness to ensue, yet restoration of sight
is possible both in children and in adults. E\7en if notable atrophy
occur in the neiwes, A'ision may remain good enough for ordinary
purposes and endure, as I haAre seen, for fifteen years.
Neuritis after orbital affections, including the cellulitis from
erysipelas, may have a rapid onset, with great or total loss of sight,
and end in either recoA7ery or blindness, the latter by atropliy, and
be either partial or total.
Syphilitic neuritis may proceed from the nerve trunk, the men-
inges, or the brain, and in the last case by formation of gummata.
One or both eyes may suffer and either together or in succession.
Thickening of the Avails of the vessels is often seen. Very rarely
the signs presented are the same as in albuminuric retinitis, even
to perfect representation of the stellate bright deposits at the
macula and hemorrhages. Hemorrhages about the veins are not
rare. Brain syphilis often gives rise to the " woolly disc," with
THE OPTIC NERVE.
677
swelling and plastic infiltration without much redness, although
the contrary and usual features are seen.
Neuritis from anaemia, chlorosis, loss of blood, or menstrual
disorders is usually of a Ioav grade with little exudation, but I have
seen choroidal lesions also about the macula. The course of the
malady is prolonged. Recovery may not come for a year. Progno-
sis is relati\Tely good.
As to albuminuric and glycosuric neuritis enough has been said
in a previous chapter.
Treatment—We are guided b}7 the nature of the malady and
by the cause. If there be a tumor or acute tubercular meningitis
Ave can do little. Local measures are most effective Avhen the
cause is basal or orbital, then dry or Avet cups to the neck or tem-
ple, sometimes the seton, the milder irritants, as mustard and tur-
pentine stupes, hot foot baths, occasional use of leeches if strength
permit, and moderate purgatiA7es Avill suggest themselves. These
measures will be especially chosen if there be local pain, or tender-
ness on pressure or on percussion. For rheumatic or gouty cases,
salicjdates, alkalies, lithia Avaters and the recognized medicaments
Avill have their use. Pilocarpine by injection will not be left out of
A-icAv as a possible resource. In anaemic neuritis, or that Avhich fol-
lows hemorrhage, iron will take the chief place. A long course is
to be expected and Avith encouraging results. Blaud's pills,1 or in
scrofulous subjects Blancard's iodide of iron pills, while in anaunic
subjects Flint's tablets2 are noAv carrying off therapeutic honors.
Among such subjects a change of formula, while adhering to the
iron, is often required. Quinine, strychnine, and various tonic mix-
tures, not omitting oleum morrhuae and extract of malt, Avith gen-
erous and especially meat and milk diet, form an essential feature
of the programme.
In no cases are the results of sagacious treatment more brilliant
than among the syphilitic, especially in the tertiary or gummy
varieties. Hen1 iodide of potassium, in full doses, Avith moderate
1 Ferri sulph., potass, carb., aa gr. iiss. Dose 1 to « J pills.
8 B Sodii chloridi,....... 3 iij-
Potass, chloridi, . gr. ix.
Potass, sulph., gr. vi.
Potass, carb., gr. iij.
Magnes. carb., gr. iij.
Sodii carb., . • . gr. xxxvi.
Calc. phos. praecip., 3ss.
Calc. carb., . gr. iij.
Ferri redacti, gr. xxvii.
Ferri carb., . gr. iij.
M. In capsules No. CO.
Sig. Two capsules three ;ime; 3 dai yafi ;er ej iting.
678
DISEASES OF THE EYE.
amount of mercurials, is the trusty weapon. The dose of iodide will
vary from gr. xxx. to 3 i. daily. The amount must be ascertained
by the exigencies of the case and the toleration of the patient.
Iron AA'ill go Avith it in women and weakly subjects, i.e., the iodide
of iron often, or possibly other forms. Mercurials may be given by
inunction, by the vapor bath, as the biniodide, gr. £ to ^o, or bichlor-
ide, gr. 1 to 3V A good formula for feeble subjects is Mass. hy-
drarg., gr. iij.; Ferri sulph. exsiccat., gr. i. Fiat pil. unam, ter in
die.
In many doubtful cases we give "mixed treatment" and often •
times hit; in fact, as in whist a player in doubt leads trumps, so in
neuritis the remedies we keep in readiness for a dubious case are
potassium iodide and corrosive sublimate.
As the stage of atrophy ensues we employ strychnine, phos-
phorus, arsenic, etc., in all cases whatever the etiology, and perhaps
without letting go the remedy previously employed.
Confinement in close rooms or in bed, or the exclusion of light
are not called for in most cases; on the contrary, if the situation
permit, fresh air, exercise, good food, and good hygiene are poAver-
ful aids to recovery.
In cases of tumor, should there be aggravation of symptoms,
increased doses of iodide may be required, and perhaps leeches ta
meet the presumable vascular afflux. We have to combat in these
patients most severe and obstinate headache. A prescription much
commended is tr. gelseminum and tr. veratrum viride, equal parts,
in doses of ten drops as required. See Taylor, Trans. Am. Ophth.
Soc, 1891, p. 189. We may haA7e to go through the Avhole list of
anodynes, but naturally avoid morphia as long as possible.
4. Retrobulbar Neuritis.—Cases of loss of sight occur which
exhibit very moderate lesions of the disc, but have features which
lead to the diagnosis of inflammation of the nerve between the
globe and the chiasm. It may be that only certain groups of fibres
are implicated and these may be either the central (axial) or the
peripheral. It folloAvs that we have either partial or total defect of
sight. In other words we can have a central scotoma either for
color or absolute, or a peripheral scotoma or a limitation of field
Avhich may be either for colored or for white light.
In considering the lesions of the various portions of the nerve
we have two groups of cases, one inflammatory and one toxic.
The former may be chronic, subacute, or acute. The lattter is.
always chronic and is due in the great majority to alcohol or to-
bacco or both. The correlation of alcoholic or tobacco amblyopia,
with the inflammatory retro-bulbar neuritis, has been justified'
within a few years by the investigations of Samelsohn, Vossius,
THE OPTIC NERVE.
679
and Uhthoff, who found that in all these cases the axial fibres
especially are the seat of the lesion. Graefe first suggested the
explanation of the simple inflammatory type, and Leber describes
it in Graefe and Saemisch, " Handbuch," vol. V., 829, grouping all
the kinds together.
In the chronic inflammatory cases there are scarcely any other
symptoms than the central, or possibly and more rarely a periph-
eral scotoma according to the part of the nerve attacked, with or
without reduction of visual acuity according to the severity of the
lesion. The disc may be red and a little hazy and the veins en-
larged, or it may be normal. There will be little or no pain and it
will be difficult to date the beginning of the affection. Only one eye
may be affected; to find the ailment double is rare.
In acute cases other symptoms appear, and the loss of sight
is quickly developed and may be very marked. There is pain
which may be severe, usually circumorbital, perhaps reaching
to the occiput; movement of the eye is uncomfortable; pressure
upon the closed lid pushing the globe backAvard causes pain, and
one eye must be compared with the other. The papilla may look
quite healthy for weeks even in cases which terminate in blindness.
On the other hand, it may in mild cases sIioav slight changes and
sometimes the region of the macula will be hazy. When only the
central fibres of the nerve are concerned the visual defect will be at
the middle of the field, and will be, as already said, either a color
scotoma, viz., green appears gray, or red fails to yield its proper
shade as compared with an eccentric part of the field, or an abso-
lute scotoma.
In testing these cases the bit of colored paper must be not more
than three millimetres in diameter, and while one is held in the line of
direct sight, another is simultaneously held alongside of it, making
an angle of 5° or 10° with the visual line. The colored paper should
be on a black ground, and in good light, while a quick answer
should be demanded. Visual acuity may be normal in good light,
but by reduced light, or with the color types of Ole Bull, or Stilling,
or by the light-sense types of Seggel, marked reduction may ap-
pear. Moreover, with common type, black on Avhite, vision may
be bad.
There will be great varieties in respect to the visual field and there
may eA-en be no perception of light. Hock 1 draAvs attention to cases
in which he thinks the periphery of the nerve, and probably the
sheath, Avas affected and bases his opinion upon the correspondence
which he found betAveen the position of invasion of the field, and the
direction in Avhich movement of the globe caused the greatest pain.
1 Centralblatt fur Augenheilkunde, April, Mai, 1884, p. 107.
680
DISEASES OF THE EYE.
For example, with great pain in looking upAvard, the upper part of
the field Avould show defect or reduction of color sense. He explains
this by the stretching of the sheath of the nerve under the move-
ment. In his cases the upper part of the field was first attacked,
central vision Avas gradually diminished, the lower part of the field
was for long exempt and recovery took place in the reA7erse order.
Nettleship * describes a series of cases of mixed kind.
The following, from my OAvn note-book, has typical features:
Mrs. M., thirty-four years of age, wife of a Methodist clergyman in New
Jersey, came to me on December 12th, 1878. Has been married thirteen years,
has one child, is in good health, except slight indigestion and occasional
rheumatic pains. The last menstruation was four days too soon. In right
eye V = f g; in the left merely sees movement of the hands on the outer side
of the field. Six days ago, on awaking in the morning, she found that the
left eye had only ability to discern the situation of the window. By noon
this had been lost for the central region, and remained only in the extreme
temporal part of the field, as found on my first examination. The pupil
normal, no headache and no head symptoms, but had " a feeling of deadness
about the brow and the opening of the orbit." Menstruates about every three
weeks, and flows copiously. By ophthalmoscope find the optic nerve injected,
and a little swollen and indistinct on all sides, except on the outer part, the
A-eins a little enlarged, the arteries rather small. All the retina rather hazy.
Ophthalmoscopic lesions greatly out of proportion to the loss of visual func-
tion ; patient put upon iodide potass. After a week she began to gain a little
better perception—sloAvly, the improvement continued. She took the iodide
for about four weeks, but no notes were taken until six months passed, when
in right eye V = f£, left eye V = T2ffV The nerve was decidedly pale, veins
large, arteries unchanged, and near the macula Avere many whitish dots.
color-perception very deficient; leaves on the trees appear black, and the
only color which she readily recognizes is yelloAv. In November, 1879, viz.,
eleven months from the beginning, find O. D. with + 48c. 90°, V = §§; O. S.
+G0c. 90% V = f£; visual field, O. S., normal, color-perception bad, nerve pale,
the outer half most decidedly.
In the case above cited, mention is made of pallor of the disc
at its outer side; this feature is sometimes but not always con-
spicuous in axial neuritis, Avhether inflammatory or toxic. It must
be carefully discriminated from a physiological excavation inclining
to the temporal edge. This Avill be easy if a normal-looking border
remains at the very edge of the disc, but even this feature may be
wanting without indicating pathological lesions.
It has been remarked that the field of vision shows a variety of
encroachments in different classes of cases. A case is presented
in which there seemed reason for the diagnosis of orbital neuritis
and which presented the unusual phenomenon of ring scotoma.
Miss Mary B. B., jet. 17, living in New Jersey, came to me in April, 1886.
1 Trans. Oph. Soc. United Kingdom, v..iv., p. 186, 1884.
THE OPTIC NERVE.
681
She had previously had asthenopic trouble, for which had been prescribed
both prismatic and spherical glasses, and tonic treatment. Her health was
delicate, had had pneumonia, Avas thought to be disposed to tuberculosis, was
very impressionable and apt to think herself seriously sick. Had been in
Florida for her health and for some time prior to the above date had been
quite Avell; had had otitis media and Avas slightly deaf: in both ears there is
perforation of the membrana tympani, and from the right there is discharge.
During the summer of 188.") both eyes and health had been good; during the
winter she began to run down, and often had dizziness. Her brother, who
is a physician, said that she had rheumatism of the abdominal muscles during
the winter, for which she took salicylic acid. In February, 1886, sight both
for distance and near began to fail. At my examination, April 13th, the ocular
condition was 0.D.+ ^ + ^ c. 90° V=i§. O.S.-HVs.+^c. 90: V=|$. Oph-
thalmometer shows astigmatism in each eye: 1 D axis 90°—180°. In each eye
has central scotoma for red, which is more decided in the right. The peri-
meter shoAvs no impairment of field in the left, but in the right there is ring
scotoma as shown in the chart (see Chart No. 1, p. 550), while there is no per-
ipheral limitation. In reading the test types at six metres with her glasses,
she linds with the right that alternate letters stand higher than the others,
with the left, each is divided from the next by a faint line. For three weeks
has had constant and severe headache—frontal, temporal and occipital—much
pain in the globes. Says she sometimes gets perfect \7ision for a few seconds
and then a cloud appears. She often sees a Avhite line running diagonally
across the page and is compelled to move the book to see the whole of a line.
The line is sometimes zigzag. Sees colored spots, blue, green, and red. Sight is
more dim at night. When riding on the railway or in a carriage often sees a
green stripe about two feet Avide by the side of the road, and it blots out of
A'iew everything Avhich it covers.
These symptoms suggest hysterical amblyopia, but the ring scotoma Avas
unmistakably demonstrated on several occasions. Outside of it, the test object
did not Avholly disappear but was very faintly seen. By the ophthalmoscope
O.D., deep central excavation of the nerve, Avhich slopes to the temporal
side, its tissue clear, edge well defined, no lesion of fundus. In O. S. similar
excavation and fundus normal to the periphery. Ordered potass, iodid. in
small doses, but it could not be borne, and she Avas given Blancard's pills of
ferri iodid. She Avent away from home on a ATisit. She also took cod-liver
oil afterward. She Avas seen at interA7als of about a month and her health
improved. On June 8th, 1886, in O.D. V= If; —in O.S. V= £$. Ring scotoma
in O.D. disappeared, but central scotoma for red remains. Had an attack of
dimness lasting fifteen minutes, during which could not recognize faces across
the street. Both eyes, fundus normal. Vision equally good both with and
Avithout glasses. On Sept. 15th. 1.S80, eyes entirely normal, color scotoma
disappeared. She continued well for tA\7o years, but in Jan., 1889, had slight
return of asthenopic symptoms.
The case Avas undoubtedly in great measure hysterical, but
there can be little doubt of the presence of actual lesions, and the
scotoma for red points to the axial fibres of the nerves, while the
ring scotoma in the right eye may denote peri-axial neuritis.
Causes of the disease in the inflammatory types are exposure
to cold, rheumatisni, syphilis, and the canalis opticus is doubtless
in very many cases the site of the reaction. Should the sphenoidal
682
DISEASES OF THE EYE.
fissure be involved other nerves Avould suffer, but we do not now
take them into consideration. There would be diplopia of such sort
as would be proper to the affected nerves. The epidemic of 1891-92
known as la grippe has been held to be a cause by various writers,
and with great probability. See paper by Weeks, N. Y. Med.
Journal, Aug. 8, 1891.
Prognosis is uncertain, yet many recoveries occur—some may
be complete, others partial.
Treatment will be mildly antiphlogistic and be determined by
the vieAV taken of causation. Iod. potass, will be most apt to be
useful, yet other indications may call for saline diuretics, or sali-
cylate of sodium; the Turkish bath may be useful. In anaemic cases
iron, arsenic, quinine, cod-liver oil, etc., will find place. The eyes
must be out of use and the general health attended to. Sometimes
the artificial leech, or dry cupping, or a blister may be employed.
Toxic Retro-bulbar Neuritis, Amblyopia from Alcohol or
Tobacco, Amblyopia ex abusu.—The dimness of sight caused by
alcohol or tobacco has long been clinically recognized, although
not until recently accurately understood. The literature is very
copious and much of it is polemic. The main facts can now be
stated Avith much assurance, since the publication of an article
by UhthoffJ which leaves little more to be said. He examined
1,000 patients who were detained in hospital because of alcoholic
excess (alcoholismus) and out of these found 6$ affected with am-
blyopia; in 6.5$ more, he found the peculiar nerve lesion with-
out amblyopia, and in 5.3$, pathological conditions of the nerve
and the adjacent retina. Added to these were some other le-
sions affecting the pupil, the muscles and the retina, making a
total of eye diseases among 1,000 alcoholic patients of about 30$.
In another category he studied 100 cases of alcoholic amblyopia,
and in almost all of them the lesion had continued more than six
weeks. For how long time the indulgence had continued is not
stated. The ophthalmoscope found in 63$ of the amblyopic cases
atrophic pallor of the temporal side of the nerve, often extending
below, and this lesion occurred in all the protracted ones; in 8$
there Avas slight but distinct haziness of the nerve and the adjacent
retina; in 28$ there was no abnormal appearance. That the outer
half of the nerve is often brighter or paler than the nasal side is
perfectly true of normal eyes, but in the cases designated an opaque
and emphatic whiteness reaching to the margin is to be noted, and
the importance of carefully distinguishing normal from abnormal
appearances is signalized by the result of autopsies. Absolute
blindness very rarely takes place from alcohol alone.
1 (xraefe's Archiv, Bd. xxxii., Abth. iv., 95-188, 1886; Bd. xxxiii., Abth. i
257-318. 1887.
THE OPTIC NERVE. 683
The pathological lesion is atrophy succeeding to inflammation
of the axial fibres of the nerve, and the beginning may be at any
part below the chiasm, although by preference it affects the distal,
that is, ocular, portion of the nerve. With deep lesions some time is
required for their manifestation at the disc—hence, in many cases.
no visible sign is afforded.
Other poisons cause similar lesions, and out of 204 cases of retro-
bulbar neuritis the following table is instructive; 138 were from
toxic causes, and 66 from miscellaneous causes, some of them in-
flammatory :—
.... 64 ... ?
.... 45 " ... 7
.... 23 it .. fi
.... 3 «'
.... 1 t< 3
.... 2 c( .. 4
— (< loss of blood at abortion.. , ?,
138 u (1 , 1 1 , 32 66
It appears that in Germany amblyopia from abuse of tobacco is
considerably less frequent than from alcoholic poisoning. In Eng-
land, judging from Mr. Hutchinson's papers, the proportion is prob-
ably greater. Mr. H. speaks of the great strength of the tobacco
used by his patients. The apparent infrequency of the affection
among Orientals is explained by the mildness of their tobacco. It
is very notable that all the first group of cases are males, and of
the second 38 Avere men and 28 women. Smoking seems more hurt-
ful than chewing, but I have seen the effect from the latter habit.
Indulgence for many years is usually necessary to produce the re-
sult.
The symptoms are color scotoma, or absolute scotoma, vary-
ing in size and either central or paracentral. The shape of the
scotoma has no relation to the kind of poison, as Forster has
claimed. There may be no reduction of acuity by test types, or
vision may be extremely bad. The periphery is not affected.
Patients are sometimes conscious of their color defect, as Avas the
fact with an artist who never drank wine or liquor, but smoked
constantly and complained that he no longer found himself able
to get the brilliant effect with reds which he had been accustomed
to. There is sometimes a glimmering sensation; there is no pain
either spontaneous or on pressure. Commonly both eyes are af-
fected and the progress of the disease is slow, both in culmination
684
DISEASES OF THE EYE.
and in recovery. The blindness relates to both red and green, and
in very rare cases to blue, just at the centre. Mr. Nettleship re-
ports a case of ring scotoma added to the central scotoma. Total
blindness is not common. Other symptoms appear in enfeeble-
ment of the ocular muscles, and to some extent of accommodation.
The muscular asthenopia is sometimes very conspicuous and
presents debility in all the binocular movements, whether of ab-
duction or adduction. A " Aveak heart " may or may not be dis-
covered; and so is it with the frequent pulse. A neurotic condition
is sometimes very pronounced. Pathological anatomy need not be
enlarged upon, but inspection of Figs. 253 and 254 from Uhthoff
shoAvs the localized character of the lesion and the different sites it
may occupy in different parts of the nerve. While all parts pre-
sent multiplication of nuclei, the heavily shaded parts (stained by
carmine) give the regions most concerned.
Treatment demands entire abstinence, which will be freely
promised and not so often practised. Iodide of potassium seems to
be of use, and Minor has found cases of recovery in tobacco ambly-
opia under its use when abstinence was not practised. The en-
forcement of abstinence Avith alcohol amblyopia is imperati ve. Dry
cupping, hot foot baths, and the Turkish bath may be of value.
Concomitant symptoms of dyspepsia, insomnia, must be suitably
dealt with. Hypodermic injections of strychnia, gr. T\ or ^¥ daily,
will often yield excellent results, and the remedy may be given in
granules in increasing doses, beginning with TV daily and going to
\ or more in some cases. In mild cases no drug may be needed. In
some quinine as a tonic acts well; Filehne J found in himself that
during four Aveeks after quitting smoking, sight grew worse and
then progressed to recovery. Resuming the habit was not at
first attended by harm, but later, sight again failed. Other toxic
agents cause axial neuritis: Fuchs reports stramonium, the leaves
smoked for asthma; the man used them freely, and did not use to-
bacco or alcohol (" Lehrbuch/' p. 477); and sulphur used in caout-
chouc factories, as bisulphide of carbon. Lead causes the same
effects and is much more rebellious to treatment. We may have
peripheral neuritis as well as axial, and we shall be guided in de-
ciding the localization by the quality of the field. The scotoma of
diabetes and of some other cases might be called toxic. See Knapp,
" Orbital Optic Neuritis," Archives fur Ophth., Jan., 1891.
(Note.—The pathological demonstration of axial optic neuritis is one of
the most interesting discoveries of recent ophthalmology. The first clear
case with autopsy was published by Samelsohn in 1882, who confirmed what
has already been conjectured with some correctness, by Leber, Michel, Lieb-
'Graefe, Archivf. Ophth., xxxi., 11, 2.1 1885; also Groeno^^^fe~Ar^ln>
xxxviii., 1,1. '
THE OPTIC NERVE.
085
reich and others, and then other cases were reported by Vossius (1882), by
Nettleship, Bunge (1884), and Uhthoff (1886), making it perfectly clear that
Fig. 253.
the fibres Avhich supply the central region of the retina enter the eye as a
wedge-shaped cluster on the temporal side of the disc, occupying nearly
* mti
its inner and lower quadrant. A similar and still more interesting fact
is the autopsy of Uhthoff's case, Avhere this lesion arose in a patient with
o8G
DISEASES OF THE EYE.
typical locomotor ataxy. At the present time a case of strabismus comes
under my notice in a boy of ten years, whose right optic nerve has diffused
hyperemia, V = 0.7, H = 1.5 D; the left eye has V = 0.1, H = 2. D, with a
defined central scotoma for red, and the nerve shows a distinct sector of
triangular shape on the outer and lower side of opaque white tissue, while
all the rest of the disc is hypersemic. The boy was noticed during his early
infancy to have peculiar eyes and squinted when two years old. This seems
to indicate congenital axial neuritis. Leber, G. and S., a\ 832, assigns simple
color scotoma with normal vision to the optic nerve lesion, and while color
scotoma Avith reduced sight he calls a retinal scotoma, that the latter is to be
demonstrated by testing the light sense with Forster's apparatus. Patients
feel more comfortable by dim light, yet in reality their A7ision is worse.
Atrophy of the Optic Nerve.
We speak of primary atrophy not of that Avhich follows inflam-
mation of the neiwe.
Symptoms.—They are both subjective and objective. The former
relate to vision, which may be impaired in the most various degree
—both as to acuity, the boundaries of the field or interruptions in it,
and as to color sense and light sense. With diminished sight there
is sometimes shrinking from strong light, while with greatly re-
duced sight the contrary obtains. Rarely, patients complain of
glimmerings; the onset of the trouble is slow. With total loss of
sight there will usually be dilated pupils, but on this point impor-
tant differences occur, because inactive, unequal, and reduced pupils
are characteristic of the large class of cases which depend on spinal
lesions. It is related of one case as a great exception that the
pupils reacted when exposed to light, yet there was absolute blind-
ness. The eye commonly looks normal, and there is no pain.
The ophthalmoscope alone reveals the true situation. (See
Colored Plate, Figs. 10 and 11.) The appearances of the condi-
tion are somewhat various. The nerve-disc is always opaque, and
in the greater number of cases is white; but we also find it gray,
leaden, bluish, or " dirty." Very often the lamina cribrosa is con-
spicuous, appearing as a mixture of white and dark dots, or intersect-
ing fibres. The nerve is flat, or more often concave, and is especially
apt to be saucer-like; the degree and kind of concavity will be
modified by the original form of its surface, whether or not it may
have had a physiological excavation. The outline is always in ad-
vanced cases sharply defined, and is often deeply pigmented. There
may be a time, if atrophy follows inflammation, when the border is
ragged, or striated, or ill-defined. According to the nature of its
surface, both as to color and form, the nerve may be uncommonly
bright and luminous, or of a dull hue. There are cases of partial
atrophy where the temporal half is white and pallid, while the
nasal side is red. Care must be taken not to hastily pronounce on
THE OPTIC NERVE.
687
such a condition, because such an arrangement is often normal.
The vital point in diagnosis is Avant of transparency in atrophic
nerve-tissue, while healthy substance always is transparent. (The
upright image and the edge of the illuminated surface as it is
moved by slight rotation of the mirror will best reveal the condition.)
A want of capillary vessels, and the development of connective
nerve-tissue, while healthy substance always is transparent. A
want of capillary vessels, and the development of connectiA-e
tissue, is necessarily implied in the above description. As to the
larger A7essels, the arteries Avill be small, sometimes thready, and
the veins, although larger, will also be of reduced size. Some-
times the A7essels are not much changed in calibre, and in other
cases they are almost entirely Avanting. It is not rare to find the
vessels bordered with gray or whitish lines, so-called perivasculitis.
(See Colored Plate, Figs. 2 and 6.) Often there Avill be traces in
the retina of a concomitant or pre-existing lesion.
The subdivisions of nerve atrophy are made according to color
into white and gray (Leber), the latter being regarded as significant
of spinal lesions, but there are mixed forms and the distinction has
only an approximate \*alue. For example, many old cases of glau-
coma have a gray or bluish-gray color. It must also be remarked
that in old age the nerve often loses its clearness and may become
gray or leaden with no evidence of impaired function. For recogni-
tion of slight changes in color the light must ahvays be Aveak.
Cases of a simply pallid nerA~e occasionally occur Avhich so closely
resemble atrophy as to be very puzzling. This may happen both
in young and older persons and naturally will appear in the anaemic.
The point to be especially noted is the texture of the nerve. A
pallid and pellucid nerATe is not atrophic. If pale and opaque, or
dead looking and into Avhose texture one cannot penetrate, this
denotes atrophy.
Another division of atrophy is into primary and secondary, or
into medullary and interstitial; the former is a lesion of the neiwe
fibres, the latter concerns the connective tissue and may be pre-
ceded by inflammation. Atrophy affects the papilla primarily or
secondarily, and may come from disease in the retina and choroid
or with glaucoma; it may ensue after lesion of the trunk of the
nerve; it may be cerebral or spinal or from numerous general
causes. Men are att'ected in much greater proportion than women.
It occurs at all ages and may be congenital. There is a hereditary
tendency which has been noted by Leber, Nettleship and others.
Many members of the same family haA7e been known to be victims.
Causes.—A carefully studied collection of cases by Uhthoff,1
amounting to 183, gives the following results:_______________
1 (jiraeiV's Archiv f. Ophth., xxvi., Abth. 1, 1880, and "Beitrage zur
Pathologie des Sehnerven," etc., Berlin, 1884, p. 50.
688
DISEASES OF THE EYE.
Men. AA'omen.
55 4
23 18
16 (j
13 4
3 5
3 5
6 1
0 4
4 0
2 0
2 1
2 1
2 0
0 1
1 0
0 1
Spinal cord............................... 59
Brain..................................... 41
Simple progressiAre ....................... 22
After neuritis optica..................... 17
Sudden embolism of arteries.............. 8
Disease and accident in orbit............. 8
Dementia paralytica..................... 7
Loss of blood............................ 4
Alcoholism................................ 4
Lead poisoning .......................... 2
Hereditary............................... 3
Injury.................................. 3
Epilepsy ................................ 2
Nephritis................................ 1
Railway spine............................ 1
Congenital with hydrophthalmia......... 1
183 182 ^SS 51 = 2Src
It is seen that in the above table only causes outside of the eye
are mentioned and Ave naturally take only these cases into consid-
eration. Just 100 of the aboAre cases depend on lesions of the brain
and spinal cord, or more than 547A, Avhich is higher than the figures
given by Galezowski2 from 166 cases. Still other causes are alleged,
A'iz., feA'ers, menstrual disturbances, etc. The numerical frequency
of spinal cord lesions as a cause of atrophy has been noted by many
obseiwers and the longer the cases are kept under observation the
more decided becomes this preponderance. The three S37mptoms
to be looked for in spinal cases are neiwe atrophy; abnormities in
the action of the pupils, viz., small size, inequality, torpor, lack of
response to light while responding to convergence and accommoda-
tion (Argyll-Robertson), indisposition to dilate upon sharp irritation
of the skin as by a pin prick or the faradic brush; and the want of
knee reflex. The last symptom may at a giA-en period be wanting,
yet in course of time appear; so Avith pupil abnormities. Both eyes
will be affected and blindness occur in from one to three years; a
shorter period is rare. In the great majority of cases, about four-
fifths, the whole field is impaired, and its reduction takes place con-
centrically from the periphery,while visual acuity steadily declines.
Sometimes Avell-defined sectors are cut out and very irregular out-
lines are formed, and the remaining portion may continue relatively
good for a long period. Singular maps are sometimes thus produced
—and as special peculiarities the following1 may be noted; blindness
of the upper or lower halves of the field—and cases where the field
becomes excessively narrow and regular, and with very fair vision
1 Journal d'Ophthal., 1, pp. 45-50. 180-212, 1872.
THE OPTIC NERVE.
689
both for white and colors. The occurrence of central scotoma in
spinal lesion has been noted. Defect of color sense is the rule, the
ability to recognize green is usually first lost, then red, then yelloAV
and blue. The proper limits of recognition of the respective colors
will be reduced and finally their perception gradually fades entirely.
Sometimes the first color lost is red—while green may remain. In
Avhat direction impairment of the field first appears in spinal optic
nerve atrophy cannot be declared; different authors give different
results.
In these cases further symptoms due to the spinal lesion Avill
eventually appear, A7iz., trouble of the bladder, staggering gait,
constriction of the body, burning of the feet, mental impairment,
\rertigo, etc.; while the lightning pains Avill have existed both long
before, during, and after the nerve atrophy.
Atrophy from cerebral causes includes those Avhich haA7e been
preceded by papillitis, and of which the causes have been previously
mentioned, A'iz., tumors, etc. (see p. 602). Perhaps one-fourth of
the cases are of this type, but this is necessarily mere conjecture.
Among other causes are meningitis, hydrocephalus, mechanical
pressure of tumors on the nerve or on its deeper connections, dis-
tention of the third ventricle pressing on the chiasm. Narrowing
of the optic canal by periostitis is not an infrequent cause. Injuries
which cause fissure of the orbit, or of the canalis opticus (Holden),
or of the base of the skull, may eventually show their effect
by atrophy, while amaurosis may haA7e preceded it. Disseminated
sclerosis and general paralysis of the insane (paresis) cause atrophy.
The latter is preceded by distinct but moderate chronic inflamma-
tory signs. Embolism of the retinal artery and sometimes cerebral
hemorrhage, or embolism, are followed by atrophy. Severe hemor-
rhage from any source is followed sometimes by optic neuritis and
also atrophy, either primary or secondary. I have seen atrophy
folloAv softening of the brain, but the sequence is rare. There are
also cases of hereditary and infantile atrophy of various causation.
Sclerosis of cerebral vessels has, Michel thinks, important influence.
Atrophy occurs in some cases as a primary lesion of the nerve,
both in the papilla at the onset, or retro-bulbar and subsequently
exhibited in the papilla. The condition is analogous to primary
optic neuritis. General diseases, viz., facial erysipelas, diphtheria,
typhus and typhoid fevers, diabetes, scarlet fever, menstrual ir-
regularities, pregnancy (Loring, " Text-book/' Part II., p. 206), etc.,
are correctly catalogued as causes. Many times we are quite un-
able to assign a cause with any assurance.
Morbid Anatomy.—In the medullary or parenchymatous atro-
phy which Ave have with ataxy and other conditions, the medullary
part of the nerve fibres disappears, at first becoming pale and
44
69C
DISEASES OF THE EYE.
%
varicose and interspersed with cells of granular fat, which will be
especially abundant in the chiasm and tractus. Amyloid cor-
puscles appear and more numerously in the cerebral parts of the
tractus After a time the nerve fibres are reduced to au indif-
ferent structure and the whole nerve becomes smaller. The con-
nective tissue becomes somewhat increased, though not to a
marked degree; the walls of the blood-vessels become thickened
and their calibre reduced. Few fibres or many may be involved
and in sectors or portions or for the whole length of the nerve, and
likewise in its cerebral contin-
uation.
As a result of the shrink-
ing of the constituents of the
nerve its size becomes reduced.
and it opens up the sheath
cavity as displayed in Fig. 255
I from Jaeger, especially around
'the papilla. . From this con-
dition the formation of the
shallow excavation of the disc
in extreme atrophic states is
understood.
The textural changes are
understood by the figures from
Poncet. Fig. 256 shows a por-
tion of a normal nerve; Fig.
257 shows the nerve which has
passed through the period of
inflammation and arrived at
atrophy. The case was one of
locomotor ataxy and the pa-
tient had been blind for thirty
years.
The atrophic process will
beset up by destructive disease of the retina, by loss or extirpation
of the eye, and will ascend to and beyond the chiasm. Gudden's
experiments in removing the eyes of young animals have plainly
demonstrated this effect. On the other hand, lesions of the corti-
cal sight centre, of the tractus, or of the chiasm, will A'ery slowly
give rise to nerve atrophy. The pressure of tumors, foreign bodies,
and exudation, exhibits the same effect. The descending process is
much sloAver than is the ascending.
Interstitial atrophy found after inflammation or after sclerosis,
exhibits a much larger development of connective tissue and nuclei,
with choking of the nerve fibres. They lose their myelin and are
Fig. 255.
THE OPTIC NERVE.
691
changed into connective tissue. A greater or less portion maj7 be
involved. In the retina the layers Avhich suffer are the optic fibres
and ganglion cells, while the remaining layers, as a rule, are intact.
Under this head comes a very interesting class of cases which
present the white color of atrophy with but little loss or possibly
serious loss, and ultimate recovery, of sight. In them interstitial
neuritis causes proliferation of connective tissue, Avith consecutive
choking and damage of the medullary sheaths, without destruc-
tion of the axis cylinders. Such cases are found Avith disseminated
sclerosis or in primary lateral sclerosis. See Zimmerman, Arch. f.
Ophth., XX., 3, 329, 1891.
Fig. 256.—1, Internal sheath; 2, sheaths of con- Fig. 257.—1, Internal sheath, hypertrophied
nective tissue, separating the bundles; 3, optic and sclerosed; 2, epidermic globules common
nerve fibres. in old persons; 3, nerve substances wholly
disorganized; 4, connective tissue- very abun-
dant about the numerous fine vessels.
Prognosis.—Seldom is it otherwise than bad. Proper weight
must begiAren to surrounding and causative circumstances, because
the nerve-lesion is often concomitant, and its character largely de-
pendent on the chief affection. Certain cases retain remarkable
sight, and they are more likely to be the interstitial where the pri-
mary affection is in the connective tissue. See Zimmerman, 1. c.
If the originating disease has ceased its activity, as, for example,
meningitis, and some sight remain, this may even improve. I
have seen several such instances, and more especially in young
subjects. Nettleship l has published several " cases of recovery
from amaurosis in young children," doubtless interstitial atrophy.
692
DISEASES OF THE EYE.
It is right to emphasize the statement already made, that pallor
of nerve must not be mistaken for atrophy. Neither must a true
atrophy be always pronounced progressive. The state of the field
and the quality of the color-sense are to be duly considered. One
consolation is often possible, viz., that the rate of progress to the
bad will be slow.
It is affirmed that regular concentric limitation is more unfavor-
able than fields of irregular outline caused by deep entering angles
of darkness. It has, however, been shoAvn that good vision within
a \7ery narrow field is sometimes long preserved. Cases Avhich
show a similarity in the irregularities of the two fields are to be
traced to brain-lesion, and they may or may not be curable. Our
ignorance of the true pathology of many cases should restrain us
from dogmatism, and, while Ave utter only the truth so far as we
know it, Ave ought to hesitate to pronounce a doom, which many
regard as Avorse than death.
Treatment—The first indication is to correct any and every de-
parture from normal function which we can discover or control,
in nutrition, sexual organs, lungs, etc.; also to counteract the
syphilitic, rheumatic, or gouty diathesis, and scrofulous tendencies;
to discriminate, so far as may be possible, diseases of the brain and
spinal cord, and, even Avhen there may be no token of syphilis, iodide
of potassium in high doses is justly esteemed. Under these heads
are included many possibilities of treatment suited to the peculiari-
ties of each case. Minute doses of " gray powder " are much in use
among English physicians. Corrosive sublimate in watery solu-
tion is also a useful prescription for children, giving gr. -fa or gr. T^
for several months. It has the advantage of being tasteless.
The tendency to attribute locomotor ataxy to syphilis lends
force to the prescription of mercurials and iodide of potassium, or
iodide of sodium. Many times the nerve lesion simply follows the
general malady and too often we are wholly at a loss to know
what to suggest. As the optic lesion goes with the spinal disease,
reference may be made to an important article by L. C. Gray, N
Y. Medical Journal, Nov. 16th, 1889. In former times setons and
blisters and moxas were much employed; now the actual cautery
is applied to the scalp and to the skin over the vertebrae, when the
brain or the spinal cord are thought to be congested. The faradic
brush, rest in bed, etc., are employed. "When, however, these pro-
ceedings have been tried or do not seem to have any claims for
trial, we are reduced to the use of a feAv remedies. The most im-
portant is strychnia. Nagel has the credit of having taken it up
systematically and with energy, and claims special benefit from its
hypodermic administration, giving of the sulphate of strychnia ^
1 Trans. Oph. Soc. United Kingdom, v. iv., 1884.
THE OPTIC NERVE.
693
to -fa grain once daily in the temple. His method has been largely
adopted, but it is to many patients inapplicable, because of the
constant attendance on the physician which it necessitates. One
may therefore substitute its internal administration in granules,
Avhich may be -Jw or fa grain each, and give a quantity sufficient to
bring about manifest constitutional symptoms. There Avill be a ten-
dency to cramps in the legs and in the loAver jaw, sometimes colic,
and sometimes exalted nervous excitability. The amount requisite
to obtain this result will be different according to the subject, but ^
grain daily is not a large amount for an adult male, and I have given
| grain with impunity. This amount is to be gradually reached by
adding to the number of granules eA7ery third day, until the proper
symptoms appear. When the full effect of the drug has been
reached, the dose is to be kept up for three or four weeks, or so long
as continuous gain is observed, and this may go on during three
months. If, after three Aveeks of strychnia-symptoms, no improAe-
ment in sight is disco\7erable, the remedy may be considered un-
availing.
Another remedy, which is less positively effectiA7e, but is to some
extent useful,, is phosphorus. It is sometimes given Avith strychnia.
It is not adA'isable to push it to the production of constitutional
symptoms, and it is not usually carried higher than TV grain dairy.
A combination of the following kind is a tonic of high A'alue, both
for these cases and in many debilitated subjects. It has the virtues
of both remedies.
Ii Acid, phosphorici dil.,......§ iij.
Strychnise,........gr. i.
Take thirty drops in water three times daily.
The proportions may be \-aried as desired.
As to zinc and nitrate of silver, I can say nothing from experi-
ence. Cod-liver oil is often helpful, but not as a specific like strych-
nia. Quinia and iron are many times indicated. ElectricitA7 has
failed to vindicate its pretensions to any real value, although, by
its capacity for exciting phosphenes, it fosters the hopes of a credu-
lous incurable.
Stretching the optic nerve by a strabismus hook passed behind
the globe has been practised (Wecker), but it has not been found of
value.
CHAPTER XIX.
AMBLYOPIA AND AMAUROSIS.
These terms designate certain cases of partial or total loss of
sight which do not present any visible intraocular lesions. Among
them retro-bulbar neuritis in any of its forms, inflammatory or toxic,
is not included. Hence, Ave omit the so-called alcohol and tobacco
amblyopia. Neither do we put hemianopsia in this category. We
use the w7ords to designate conditions whose pathology is not
known or which may be functional in character, to use an expres-
sive, though vague, term. There may be limitation of the field, or
scotomata, and loss of color sense. Some cases are permanent,
some are transient. With the advance of our knoAvledge, we shall
make less frequent use of these terms. For example, we speak of
uraemic or diabetic amaurosis and amblyopia, or of that which
occurs in pregnancy, of reflex amblyopia, etc., and as colloquialisms
they are justified. But cerebral cases, for example those with
lesion of the occipital cortex, are excluded.
What we may include in our catalogue of amaurosis and am-
blyopia will be such conditions as the following: 1. Traumatic. 2.
By lightning. 3. Hemorrhage, local or general. 4. Toxic, from
which we have excluded alcohol and tobacco. 5. Uraemic. 6. Dia-
betic. 7. Hysterical. 8. Migraine. 9. Reflex.
Some cases will for a time be reckoned as amblyopia until a
later stage shall show the true nature of the disease. The patho-
logical lesion may lie in some special cases, 1st, in the retina over
limited or more extensive portions and for months escape ophthal-
moscopic detection. It may also exist, 2d, in the deeper parts of
the orbital portion of the optic nerve, or in the chiasm. 3d. In the
tractus and its continuation in the brain—but these cases will sooner
or later exhibit recognizable tokens of atrophy of the papilla nervi
optici. 4th. Again, we have cerebral blindness coming under some
of the heads above designated and some denoted as mental blind-
ness because all recollection of visual impressions as well as ability
to perceive them has been obliterated.
Traumatic amblyopia or amaurosis is either a provisional term
awaiting the development of the true morbid status, or it may
simply mean that Ave are unable to discover the lesion. The trau-
AMBLYOPIA AND A3IAUR0SIS. 695
matism may be direct or indirect. A direct blow on the eye may
sometimes cause amblyopia without A-isible tissue lesion except as
folloAvs: First, immediate rigid contraction of the pupil. This has
been noted by Berlin, and probably by others. I have seen it a
number of times. The contraction is to the smallest size, and re-
sists for hours the most vigorous use of atropia. So small have I
found the pupil that ophthalmoscopic examination has been impos-
sible. Berlin (Klin. Monatsbl., XL, p. 42, 1873), Avriting upon con-
cussion of the retina, divides these cases into two groups: First,
those in Avhich central vision is moderately impaired (say to fa5^ or
to fa>$), Avhile peripheral sight is intact. In a few days sight is
fully restored and he thinks irregular astigmatism of the lens is
the proper explanation. See p. 613;
Second, Berlin, 1. c, describes cases of direct injury to the globe,
in Avhich he has seen a spot of Avhitish opacity of the retina at a
point opposite the place of injury, and sometimes also on the site of
the blow, which begins to appear within a few hours, and vanishes
after two days, Avith restoration of vision. I have noted this lesion,
and it must generally be sought at the extreme limit of the oph-
thalmoscopic field. Knapp has reported cases. In experiments
upon rabbits he ahvays found a subchoroidal hemorrhage at the
situation of the retinal opacity. Aub (Archives of Oph. and
Otol., aoI. II., 173) reports a case of metamorphopsia after a bloAV,
which implies disturbance of the retinal elements. There may be
innumerable complications of such injuries in other lesions of the
globe, such as hemorrhages, irido-dialysis, distortion of the pupil,
etc., but they are not now under consideration. Continual pres-
sure on the eyeball will cause blindness. Testelin reports it in a
man avIio, when drunk, lay for many hours with his eye pressing
upon his hand. Graefe cured a case of severe blepharospasm, which
had lasted eleven months, by section of the supraorbital nerves,
and the child had become almost blind. In the course of a month
sight returned, simply, as Graefe believed, because the pressure
Avas remoA7ed.
Another class of cases are due to indirect injuries by which the
optic nerA7e, either in the orbit or in the brain, has suffered lesion.
The explanation is to be given of not a few cases, which was brought
forAvard by Berlin in the Heidelberg Ophthalmic Congress, 1878, to
the effect that fissure of the roof of the orbit is far more frequent
than is generally supposed, and it extends often through the optic
foramen or through the sphenoidal fissure. To discover it, the dura
mater must be stripped from the bone, which is rarely done at an
autopsy, and it was found by Dr. von Holder to occur in ninety
per cent of the cases of fracture of the base of the skull. Naturally
by hemorrhage into the optic sheath, or by laceration of the fibres
696 DISEASES OF THE EYE.
of the nerve, sight would be injured, and atrophy might ensue,
while for a long time no signs could be seen Avithin the eye.
Apart from these cases, to Avhich reference will be again made
when speaking of lesions of the orbit, the nerve can suffer serious
injury by contusion within the canalis opticus, and hemorrhages
are quite likely to take place here where numerous \7essels come in
to supply the nerve. Within the category noAv discussed will come
the cases which sometimes have been called reflex amaurosis by
injury of the supra-orbital nerve as it passes through the supra-orbi-
tal foramen. Not now deciding the point of possible reflex influence,
it is certain that some of these cases are fully explained by fissure
of the orbital roof and consequent injury, direct or indirect, to the
optic ner\re. Analogous to this lesion is the following case in my
own experience:
A very large man fell into a hole in the street, and struck the
outer edge of one orbit on the pavement. He lost sight in the eye
of the injured side immediately, and after a few days the opposite
eye became very amblyopic. In neither was any lesion to be seen
by the ophthalmoscope. A fissure of the orbit probably extended
to both sides. It is important to inquire for bleeding of the nose,
and to search for subconjunctiA-al ecchymosis, Avhich may come to
A7ieAv several days after the injury. Both these signs would be
strongly indicative of fissure, but Avould not be indispensable as
symptoms.
Penetrating wounds of the optic nerve are rare, but two un-
questionable instances have fallen under my notice, in one of which
proof was found in the immediate and total loss of sight and in the
ophthalmoscopic appearances of the optic nerA7e (see page 687.) Of
course such cases are not samples of amaurosis, Avhich Ave noAv are
discussing, but they may explain some other cases wherein total
loss of sight folloAved a trifling injury (Haas,1 by penetrating wound
with a table fork) in which no lesion could be seen.
Traumatic amblyopia, or amaurosis, may occur through a great
variety of injuries of the skull or brain, to which no clue can be
found should the patient survive, but in Avhich some disorganiza-
tion of tissue undoubtedly has occurred to structures concerned in
A'ision.
Concussions of the spinal cord may cause loss of sight. More
than twenty years ago I saw a man who, by a railway collision,
received a sudden and severe blow upon the lower end of the spine'
whose force was transmitted in the line of the vertebras directly up-
ward. He suffered extreme pain at the base of the skull, and alon<*
the spine, while his sight was, as I remember, about ^, and the
1 Klini&che Monatsblatter, 1884, p. 280.
AMBLYOPIA AND AMAUROSIS.
697
visual fields Avere contracted to a space Avhose angle was less than
thirty degrees. In both eyes there was extreme hyperaemia of the
optic discs and of the large and small A-essels. For a number of
Aveeks the condition remained unchanged, and I do not knoAv Iioav it
finally turned out. In this case a paralysis of the fibres of the
sympathetic might Avell be assumed as the cause of the Avascular
dilation. What caused the extreme limitation of the fields is purely
conjectural.
Loss of sight by concussion of the spinal cord is chvelt upon by
Erichsen in his treatise 1875, and 1883, and in his chapter, p. 233, on
A'isual affections is much that is irrelevant and unproA^en. Later
Avriters have contributed to this subject under the head of trau-
matic neuroses. Charcot, Oppenheim, Strumpel, Dana have written
upon the subject, and a paper by P. C. Knapp, in Boston Medical
and Surgical Journal, Nov. 1st, 1888, covers the subject intelli-
gently. A case reported by Boland l gives the \7isual symptoms
happening in such cases: viz., as an immediate effect—colored
vision (not ahvays present, and is transient), contraction of the
fields, and monocular diplopia. In Boland's case the limitation of
the field was in onl}7 one eye. The monocular diplopia passed
away, but the retinal affection remained. Concussion of the lens
Avould appear to be the only explanation of the diplopia, AA'hile the
contracted field may be accounted for, perhaps, by hemorrhage
pressing on the ner\7e. In some autopsies minute hemorrhages
have been found scattered in the brain. Cases of this kind haA7e
great importance in their medico-legal relations because they arise
often after railway accidents. Unhappily because the symptoms
are almost wholly subjective and the temptation to simulation
and exaggeration great, the physician is compelled to a rigid scien-
tific and someAvhat sceptical examination, to justify him in hazard-
ing an opinion about their genuineness. His opinion Avill be eagerly
solicited in aid of a claim for compensation for alleged visual and
other injuries. While A'isual lesions may be produced, he must not
forget the possibilities of malingering.
Prognosis in traumatic amblyopia and amaurosis is good for
the mild cases, while for severe injuries of the orbit, of the skull, of
the brain, or the spinal cord, it must be guarded.
Treatment at first will be such as the special conditions of in-
jury call for, and Avhen the primary symptoms have passed, resort
may be had to strychnia, either by injection or by the stomach.
The mild cases of traumatic amblyopia Avill get Avell spontaneously
within a few days (see case in Hirschberg, Centralblatt, April,
1881, p. 100; by Reich, neuro-retinitis partialis after injury of skull
—recovery; also see case in GoAvers, p. 318, fracture of orbit, etc.).
' Boston Med. Surg. Journal, Nov. 10th, 1887.
69 S
DISEASES OF THE EYE.
Loss of sight by stroke of lightning has been recorded in
many instances and the lesions are diversified, \7iz., burn of the skin
and hair, and of the cornea, ptosis, especially production of cataract,
whose maturation may be rapid and is apt to begin at the posterior
pole; sometimes iritis and other inflammatory conditions, and ab-
solute amaurosis not accounted for by other lesions and occurring
very early. In other cases neuro-retinitis and partial atrophy of
the nerve have been discovered, as well as mydriasis and paresis of
accommodation, and we therefore refer to such injuries simply to
call attention to the liability to implication of the nerve and retina
with or without conjunction of other lesions. For careful report,
see case by Laker (Archives of Ophthal., Am. ed., \7ol. XIV., p.
181, 1885, and Buller, Archives of Ophthal, XVIL, 2, 131, 1888,
and Leber Graefe's Archiv, XXIIL, 3, 255.)
Amblyopia or Amaurosis from General Hemorrhage.—This
subject has been elaborately presented by Fries in an inaugural
dissertation (Beilageheft zu Klinische Monatsblat, Zehender, 1876),
based upon 106 cases recorded from 1641 to 1876. A number of in-
stances have been published by other observers since Fries. Mandel-
stammer,two cases from nose-bleed, Centralblatt, 1879, p. 175. The
sum of the matter is that, after severe hemorrhage, loss of sight
sometimes takes place, and may be immediate, may be within a feAv
days, or be deferred as late as the eighteenth day. In a number of
cases signs of neuritis, or retinal hemorrhage, or of retinitis, or of
atrophy of the nerve, were found, while in other cases no visible
lesions appeared. The pathological connection between cause and
effect is not understood. The source of the bleeding may be most
various: most often it is from the stomach and intestines; next in
frequency, it comes from the uterus either in childbirth, from abor-
tion, or during menstruation; it may be from the lungs, the blad-
der, the urethra, or by venesection, or by injury. The last is the
least common cause. It is sometimes attended Avith peculiarities
in the visual field, such as irregular defects or scotomata, In 90
per cent (Fries) both eyes are affected; in 47 per cent the loss of
sight is permanent, the pupils being dilated and perception of light
Avanting; in 31 per cent there Avas improvement, and in some of
these cases this occurred in only one eye; in 21 per cent entire
recovery was obtained. The time when recovery set in was vari-
able, that is, from a few hours to three or four months, and in one
case to nine months. The pathological appearances would natu-
rally have great influence on prognosis, and in the bad cases
inflammatory signs are most pronounced.
A lady, 52 years of age, under my own observation, in whom
uterine hemorrhages were of the greatest severity and for which
ovariotomy was undertaken, had choroido-retinitis and retinal hem-
AMBLYOPIA AND AMAUROSIS.
699
orrhages, but the great factor in the loss of vision Avas proved to
be the general depreciation by the loss of blood, because during a
period of months Avhen the hemorrhages ceased, Aision notably
improved, and again receded Avhen the bleeding recurred—mean-
time no intraocular changes could be detected to account for the
variation in sight. Subsequent to writing the preceding lines ad-
ditional loss of sight ensued, as the effect of atrophy of the choroid
and retina at the region of the macula, This was evidently the
consequence of impoverished nutrition and signifled coarse lesions
succeeding the finer ones Avhich had for so long a time impaired
Aision.
In a case reported by Ziegler,1 at the autopsy there was found
fatty degeneration of the optic nerves. See also Hirschberg in
Centralblatt f. Augenh., Sept., 1892, p. 257, showing neuritis and
atrophy.
Treatment will be modified by the general condition of the pa
tient and b37 the source of the bleeding. The most efficient means
of aiding recovery are: first, the vigorous use of strychnia by injec-
tion or by the stomach; second, by galvanism; third, by dry-cup-
ping about the temple; and fourth, by general invigoration, to
improA-e the action of the heart and the quality of the blood. This
includes iron, digitalis, cod-liver oil, quinine, etc. That the sight
does not more frequently suffer by large loss of blood is in part due
to the comparatiA'e independence of the intraocular circulation as
compared Avith the systemic; but this, of course, is only true Avithin
certain limits. Gowers (" Medical Ophthalmoscopy ") has condensed
many of the obserA7ations on this subject (see pp. 1S4-1SS), 1879.
So called amblyopia ex anopsia in strabismus has adA7ocates
(Leber and Theobald), and Avithin A7ery narroAv limits may be
conceded, but it Avill be left out of our category. See p. 181. On
the other hand congenital amblyopia, central or general, is not
rare. The cause will lie in retro-bulbar neuritis, axial or general,
in transient meningitis, etc.
Toxic Amblyopia and Amaurosis includes many pernicious
causes. Alcohol and tobacco, which excite a peculiar partial neuri-
tis, have been discussed. We have, moreover, lead, to which refer-
ence has been made, and osmic acid,2 which besides severe irritation
of the conjunctiva causes great and sudden amblyopia. Nitro-
benzol containing aniline is another toxic agent (see case by Litter,
in Hirschberg's Centralblatt, p. 118, April, 1881); the patient was
in coma, and the surface of the body, as Avell as the eye-grounds,
were intensely blue. Silver and mercury are said to cause ambly-
opia; likewise sulphide of carbon, used in the manufacture of india-
1 Fortschritt de Medizin, 1887, Nov. 15th, p. 735.
2Noyes, Trans. Am. Ophth. Soc, 1866, p. 34.
700
DISEASES OF THE EYE.
rubber—in which lead is used (see "Report of Case on Bisulphide
of Carbon," Trans. Oph. Soc. United Kingdom, 1885-1886).
Quinine, in large doses, has had the same result, as reported
by Graefe, 1857, Voorhies, Trans. Airier. Med. Assoc, p. 411, 1879,
Roosa and Griming, and others. Griming concludes his article on
quinine amaurosis (see Arch, of Oph., p. 81, March, 1881) by this
statement: " The patient, after the ingestion of a single dose or of
repeated doses of quinine in various quantities, suddenly becomes
totally blind and deaf. While the deafness disappears Avithin
twenty-four hours, the blindness remains permanent as regards
peripheric vision, central vision gradually returning to the normal
after some clays, Aveeks, or months. The ophthalmoscope reA7eals
ischaemia of the retinal arteries and veins, without any inflam-
matory changes." By others it is stated that permanent diminu-
tion of the \7essels and even their obliteration occurs: the neiwe
being pallid, that is atrophic. Color blindness, sometimes complete,
has been noted by Griming, Knapp, and Roosa. In one case central
scotoma for white was noticed (Iodko, Brunner, 1. a). There may
be permanent limitation of the field, Avhile central \Tision is good.
In most cases complete restoration takes place. (See "Ueber
Chinin Amaurose "—Inaugural dissertation by Brunner—under the
auspices of Horner, Zurich, 1882.) An exceedingly complete sum-
mary of cases is given by Atkinson, Journal of Amer. Med. Assoc,
September 28th, 1889. He finds the first reported case in 1841.1
Salicylic acid is, by Riess, reported to have had the same effect.
Full doses of santonine do not impair sight, but make all objects look
yelloAv. I have no knowledge of the appearance of the fundus in
these cases. In regard to lead, we find inflammation of the nerve
and retina, atrophy, and also amblyopia Avithout visible lesion.
There are also cases of brain-lesion. For lead amblyopia, treat-
ment by iodide of potassium gives good results; for inflammation
and atrophy, the prospect is unpromising.
Urcemic amaurosis and amblyopia due to kidney disease has
been referred to: it accompanies and subsides Avith other symptoms
of blood poisoning, viz., pain in the head, epileptoid fits, etc.
Glycosuria sometimes presents a chronic form of amblyopia,
viz., central scotoma for red, central scotoma for white light more
or less intense, even to totality; there may also be irregular peri-
1 Ue Schweinitz, experimenting on dogs, explains the process as follows ■
"The original effect of quinine is upon the vaso-motor centres producing
constriction of the vessels; that finally changes in the vessels themselves are
set up, owing, perhaps, to endo-vasculitis; that thrombosis may occur and
that the result of all these is an extensive atrophy of the visual tract "—Trans
Am. Oph. Soc, 1891, p. 2:5, with micro-photographs. The study of the patho-
logical process is very complete.
AMBLYOPIA AND AMAUROSIS. 701
pherical limitations of the field; there may be hemianopsia and this
can subsequently invade the whole field. Both eyes are concerned,
yet often very unequally. The affection may disappear under suit-
able constitutional treatment, yet prognosis Avill hang upon the
duration and severity of the amblyopia, upon the control which may
be gained over the general malady, and upon Avhether signs of optic
nerve atrophy appear. Even with evident nerve atrophy valuable
vision may long be preserved. See Leber, Graefe and Saemisch,
vol. V., p. 894, and a resume of diabetic affections of the eye, by
Dr. Moore, N. Y. Med. Journal, March 31, 1888.
Amaurosis of pregnancy has been referred to, and that it may
be quite independent of uraemia so far as can be discovered. In
the cases described only slight lesions could be found in the eye-
grounds, but the optic nerves showed a tendency to atrophy. This
occurrence has been known to happen tAvice to the same person.
It is very graA'e, and has required the production of premature
delivery as the only means of preserving sight; and this it has
accomplished (Loring). See page 567. Sudden amaurosis from
suppression of menstruation is reported by Samelsohn. After
tj'phus and typhoid fever, sight is sometimes impaired or lost, and
there will usually be atrophy of the nerve.
Hysterical amblyopia, or, as it is sometimes called retinal
ancesthesia, is a recognized condition, and has been studied by
Charcot, Landolt, and others. It is temporary, irregular, and at-
tended by other hysterical symptoms. Hemi-anaesthesia is some-
times a characteristic of the cases. There may be only one eye
affected, and but one-half of the field. There maA7 be only central
scotoma.
In epileptics temporary loss of vision sometimes occurs, aa hich
may be of various types, e.g., hemianopsia, perhaps preceded
by au aura of colored light (blue or red), or of smokiness, and
without vertigo or unconsciousness or spasm; or there may be loss
of sight in all the field, and the accompanying symptoms be various.
A central scotoma and irregular limitations of the field may occur.
But aside from these temporary perversions of vision is another and
more persistent affection of Avhich a mild form has been already re-
ferred to (see p. 197), to which Wilbrand called attention. The
fields undergo concentric limitation, and may be reduced to a very
narrow area, suggesting some organic lesion, yet nothing is visible
to the ophthalmoscope. The following case illustrates the subject,
and is especially pertinent because it was also studied carefully by
another and very capable observer, Dr. Starr:
Herbert B---, 26, single. Father nervous, subject to neuralgia; mother sub-
ject to sick headaches. Has had sick headaches occasionally as long as he can
702
DISEASES OF THE EYE.
remember; nosvphilis; no venereal; occasional muscular rheumatism. Always
has been nervous. Present attacks date back five years, formerly preceded
sick headache, noAv do not. He suddenly sees yellow sparks or flashes in
one half of the visual field of both eyes, either to right or to left, never in
front, and then in a " few seconds " the entire half of the visual field in which
he has seen the flashes becomes dark for a "few minutes." He feels dizzy
and bewildered at the time and as if about to faint, but he has never fainted
or fallen, or had a spasm or twitching of any kind and is quite sure that he
has never lost consciousness. The attacks are usually accompanied by palpi-
tation of the heart, and at times are followed by severe unilateral headache,
nausea, and vomiting. During the attack his face flushes, and his head feels
full. He can arrest the attack and diminish the after-effects by putting his
feet in very hot water at once. He knows of no cause for the attack. The
attacks occur about once a week, though the intervals vary in length. Di-
gestion good—no flatulence—bowels regular. Urine free from albumin and
sugar, and passed in normal amount, no excess of uric acid, no oxalates, no
cardiac or pulmonary disease. No disturbance of sensation, co-ordination or
motion. Reflexes normal and equal. No ataxia. Pupils equal, are widely
dilated, react promptly to light and in accommodation; Vf# in eacn eye.
Adduc. 9°, abduc. 6°, i° esophoria, no hyperphoria. Is a very tall, thin young
man, poorly nourished, " overgrown." Weighs 147 lbs. Visual fields normal
for light and white, colors not tested. Says he can always see well excepting
during attack. He fears insanity, has worried much, is a clerk at desk,
writing 10 to 11 hours daily. Says he is becoming irritable and forgetful.
Presents no mental symptoms during examination and memory of illness is
good. Examination, May 15th, 1888.
Diagnosis.—In a boy of neuropathic constitution hereditary migraine at-
tacks have developed of paralytic variety. The present attacks are of a mi-
graine nature—vaso-motor in character and not true epilepsy.
A case of hysterical blindness, or perhaps rather simulation,
lasting ten years, is given by Harlan, Trans. Am. Oph. Soc, 1889,
p. 327. See others by Moore, Trans. Am. Oph. Soc, 1888, p. 80.
Other eye diseases have been simulated in these cases, A7iz., glau-
coma, and for this iridectomy was once done by Cuignet. Prog-
nosis is good, and treatment must not be too serious. The main
question is diagnosis.
Under the name of amaurosis fugax cases of total loss of sight
without ophthalmoscopic findings and without hysteria have been
recorded and we can give very little account of their etiology. See
Snell, Ophthalmic Review, vol. i., p. 400, 1882.
Spasm of the retinal vessels or migraine, scotoma scintihans,
has been described elsewhere, page 546. In these cases the lesion
is in the retina, not in the brain as in the above case of Dr. Starr
and it can be observed by the ophthalmoscope.
One caution should be observed in these cases: not to confound
attacks of megrim with the temporary obscurations which occur in
glaucoma. For this reason, the tension of the globe, the state of
the optic nerve as to excavation, and the limits of the field, must be
exactly determined.
AMBLYOPIA AND AMAUROSIS. 703
Dyslexia, paralexia, alexia, are terms denoting certain hin-
drances to vision which do not consist in impairment of visual
acuity, but in incapacity to read at all or to read understandingly.
A person having dyslexia has normal acuity, and can read a few
acuity, but in Avant of capacity to read at all or to read correctly.
A person having dyslexia has normal acuity, and can read a feAV
Avords correctly, then suddenly puts doAvn the book and cannot be
induced to go on. There is no pain, no indistinctness, no fatigue,
but the poAver of attention and reading is exhausted. It makes no
difference avIiether he read aloud or to himself, he cannot proceed.
A careful examination excludes all refractiAre or other functional
error, or these may be perfectly corrected—hence the case differs
entirely from asthenopia Avith which it may be confounded. Berlin1
reports six cases and refers to others. In almost all there were
symptoms of cerebral disease, and in six autopsies lesions were
found in the left hemisphere not far from the third frontal convolu-
tion (Broca's region). In paralexia, the patient substitutes wrong
words for those he means to use. In alexia he sees but cannot read
the Avords. The last-named condition is part of the disease called
by Munk mental blindness, which consists in the loss of memory of
words or symbols describing objects, and depends on injury in the
cortex of the occipital lobe near the gyrus angularis. It may be
associated with agraphia, inability to Avrite, and other lesions. See
treatises on cerebral disease under forms of aphasia, etc. (e.g.,
Wernicke, " Gehirnkrankheiten," rid. 1, p. o'.sb; also Starr, " Familiar
Forms of Nervous Disease," and Herter, " Symptomatology of
Nervous Diseases," 1892, p. 200 et seq.
Mental or psychic blindness has chiefly been studied by experi-
ments on animals and it is not yet clear Ayhether it can be ascribed
to lesion of a particular part of the occipital region. According to
Michel it sometimes ensues after violence to other parts of the brain
and also after simple loss of the cerebro-spinal fluid. Discrimina-
tion betAveen true cortical blindness and psychic blindness is not
always easy, as is illustrated by a case reported bA7 Schmierling from
Archiv f. Psychiatrie in Zehender, October, 1889.
The cognate subjects of illusions and hallucinations, loss of
memory of faces, etc., concern the mental concepts and processes
which are associated with vision, and we shall briefly refer to them
under another head.
Hemeralopia, night-blindness, denotes unduly reduced vision
on the approach of night. The word nyctalopia which has been
used in the same sense is noAV discarded. This condition must not
be confounded with varieties of retinitis pigmentosa, in Avhich the
same symptom occurs, and each may be hereditary. Prolonged
exposure to bright light Avill occasion this torpor of the retina, as
1 " Eine besondcre Art des "Wortblindheit (Dyslexic)," 1'C7.
704 DISEASES OF THE EYE.
well as the opposite state of exalted sensibility, hyperalgesia.
Hemeralopia unconnected Avith recognizable lesion, such as choroi-
ditis, detached retina, etc., is sometimes epidemic, it happens to
soldiers on the march under a hot sun, to travellers in the tropics
and in the arctic zone, to glass blowers and others who work before
furnaces. Color sense and peripheral sight will be reduced in cer-
tain cases, but often not at all. It is noticed that insufficient or
poor food is a factor in the disease. The persons see well by day,
but when the light is reduced beloAV a certain degree, Avhether by
night-fall or artificially, their vision is far Avorse than it ought to
be. Sailors designate this condition as " moon-blindness," and at-
tribute it to lying on deck in the moonlight. It often attends
scurvy. See Leber in Graefe-Saemisch, vol. V., p. 994; Arch, of
Ophthal, vol. XII., p. 190, 1883.
Snow-blindness, as it is called, is a mixed condition, consisting of
intense photophobia, spasm of the eyelids, conjunctival and corneal
irritation, and sometimes chemosis. See case of Keratitis on p. 358
The bright glare and the severe cold combine to cause the condition
and the treatment consists in soothing applications to the lids,
Avarm Avater, with a little cocaine, 2^, protection by shades, goggles,
veils, etc., or, if possible, shelter indoors. Some of the cases exhibit
ancesthesia (torpor) of the retina, and some undue sensibility. For
the cases of ordinary idiopathic hemeralopia, prolonged exclusion
from light, Avith good diet, and in many cases antiscorbutics or cod-
liver oil, will almost surely afford relief.
The occurrence of asthenopic or reflex amblyopia, especially as
displayed in peripheral or "spiral" restrictions of the field, has
been referred to on page 197 (Wilbrand).
Amblyopia from irritations of the teeth, i.e., of branches of the
filth pair of nerves, has long found a place in text-books, but all
such eases demand rigid investigation to establish their verity. A
recent treatise1 brings together many alleged instances, and
credence may Avell be given to some of them. The teeth giving
rise to trouble are those in the upper jaw; sometimes only the
roots remain; they may or may not be painful. Frequently there
is periostitis, and the causal connection may be by communicating
inflammation to the orbit and the optic nerve. Again the relation-
ship seems to be purely reflex judging by the quickness of relief
after removal of the teeth. Galezowski reports iritis and infra-
orbital neuralgia relieved by extracting bad teeth (1. c, p. 97). Wid-
mark(l.c.,p. Ill) gives a casein The Lancet, July 10th, 1886, of blind-
ness cured in four days by removing carious molars. Neuralgia of
the eye may, as I have seen, be caused by irritation from a " wis-
1 " Maladies des Yeux et Maladies des Dents," par Courtaix, Paris, 1892.
AMBLYOPIA AND AMAUROSIS. ?05
dom " tooth, and various cases by Hutchinson and others are re-
ported by Courtaix, 1. c. Abscess of the antrum has led to abscess
of the orbit and even death; Avhile affections of the pupil,of accom-
modation, and of motor muscles are reported by trustworthy ob-
servers (Ely, Med. Record, 1882, p. 258). Blepharospasm and
photophobia are thus caused, and GalezoAvski gives a case where a
denture provoked these symptoms. The brochure of Courtaix is
Avorth study. Dunn gives a case where six months after an injury
sight began to fail in both eyes, and after trying other remedies,
section of the supra-orbital nerve gave relief to pain and sight
gradually returned. There may have been a neuritis with reflex
in optic nerve. See N. Y. Med. Journal, Aug. 9th, 1890; also, Des-
pagnet, Annales d'Oculistique, Aout, 1893.
The literature is collected by Leber, G. and S., V., p. 978.
Treatment of the above cases, when not already touched upon,
will vary with the nature of the cause so far as this can be dis-
covered. Some recover spontaneously and may be aided by mild
medication of the neiwine or antispasmodic or tonic character—en-
couraging prognostications are potent curatives. For toxic causes,
suitable antidotes and abstinence suggest themselves. The princi-
pal element in all these cases is to fix the diagnosis by absence of
lesions of the fundus, of errors of refraction, or disorders of accom-
modation, or disturbances in muscular functions, and the treatment
Avill folloAv the rational estimate of the case according to general
therapeutic laAvs. For emotional cases, a hypnotic to secure a good
night's sleep, a cup of hot, strong tea, a' mustard plaster to the
back of the neck, dry cups, brorno-caffeine, an injection of strychnia
and various devices suggested by salient symptoms are to be
brought into requisition. Under this head comes metallo-therapy,
which is soberly dwelt upon by some French writers—a scientific
form of " faith cure," and the galvanic battery is not to be forgotten:
especially the faradic current or the constant current Avith produc-
tion of phosphenes by frequent reversals of the current.
Hyperesthesia of the Retina.
This condition sometimes appears apart from any visible dis-
orders of other tissues of the eye, i.e., such as cause photophobia
and with which Ave are familiar in the preceding pages and apart
from sympathetic ophthalmia. It is sometimes the effect of ex-
posure to extreme light, and it follows prolonged seclusion in dark-
ness, Avhich Avill naturally be attended by impaired health.
Nyctalopia, in the sense of seeing better by dim than by a strong
light, may7 here be mentioned. As a mere symptom it belongs to
45
706
DISEASES OF THE EYE.
cases of mydriasis or albinism, to coloboma iridis, and to some
affections of the retina and optic nerve; it may appear with central
scotoma, and with small polar cataract; such persons get on by
night relatively better than by day.
We have, however, a more pronounced class of cases in whom
excessive sensibility to light is the conspicuous and distressing
symptom, it may be called hysterical hypercesthesia retinae.
It is found both in men and women. It is often associated with
some error of refraction, and is brought on generally during an
attack of illness. Light becomes unpleasant, and the patient desires
its partial exclusion, until at length nothing but absolute darkness
Avill be tolerated. One such instance is as follows: A man of actiA-e
mind and good health Avas submitted to an operation for varicocele.
During confinement to bed he amused himself bj7 studies in mathe-
matics. His eyes after a time gave trouble, and he caused the
windows to be shaded. Anxiety about the condition of his genital
organs made him morbidly sensitive, and this aggravated the
growing irritability of his eyes. After he was cured of the vari-
cocele he remained in a perfectly dark room for several weeks, and
Avas unable to bear the least light without great distress. When
he came to me he was encased in wrappings about his eyes, which
he Avas very loth to remove. By urgency and 'insistance, and as-
surances that his fears of blindness Avere needless, I finally suc-
ceeded in examining and testing his eyes, and found a high degree
of hypermetropia. Suitable glasses Avere given, and he soon
gathered courage to face the light and use his glasses, and was
perfectly restored. I relate this case from memory, and cannot
adequately convey the extremely distressing state to which a
highly educated and capable man had been brought.
An instance in a young woman, about twenty-three years of age,
Avas so intense in its character as to be absurd. To shut out all
light when going out-doors, she had constructed a visor of paste-
board, cotton wadding, and green cloth, which covered her head
and face to the end of the nose, like a huge mask, and was tightly
tied behind. Over this she wore a thick veil. These things were
removed in a dark room, and then no persuasion could induce her
to unclose her eyes. She finally consented to be chloroformed in a
dark room, and while she was unconscious the blinds were thrown
open, and, on waking, it was some time before she observed the in-
creased light, and thenceforward improvement took place. She
had muscular asthenopia, and there was a relapse afterward, but
the hyperesthesia ultimately disappeared.
In another case, of which I have full notes, the lady had been
for eight months in darkness. When she had laid aside the wraps
about her face, I succeeded, by the help of the atomizer playing- on
AMBLYOPIA AND AMAUROSIS.
707
the lids and by hopeful talk, in getting her to open her eyes, and in
tAvo hours she was able to bear the ordinary light of a room.
Afterward atropia was used. There Avas marked conjunctival irri-
tation and spasm of accommodation. Encouraging assurances to
the patient greatly aided her recovery, and by abductive prisms,
to correct muscular insufficiency, she Avas able to get moderate use
of her eyes.
These cases, as may be seen, belong strictly to the category of
asthenopia, but their predominant feature leads me to mention
them in the present connection and additional suggestions as to
their management are needless.
Hemianopsia—Hemianopia.
We have referred repeatedly in describing intra-ocular diseases
to limitation or invasion of the visual field as a frequent and im-
portant system. It occurs in glaucoma, in optic atrophy, in embol-
ism of a retinal vessel, in retinitis pigmentosa, etc. But it is
also a symptom of ocular affections which have an intra-cranial
origin, and to the discussion of these conditions we now attend.
What is to be said will be made intelligible in the light of the ex-
planations which have been given respecting the anatomy of the
visual path Avithin the brain (see p. 644). The title of the present
section describes the loss of one-half of the visual field of each eye,
because it is characteristic of intra-cranial affections now referred
to that the lesion is a double one and that it is symmetrical. The
usual condition is the loss of the right or left half of each field,
the boundary line being on the Arertical meridian: hemianopsia
homonymous and lateral. (The old word hemiopia is discarded,
because that signifies half-sight and our interest attaches to the
half-blindness.) We also have cause of bitemporal and of binasal
hemianopsia, the dividing line being central in each eye and ver-
tical. Cases are recorded in which the dividing line is horizontal—
as, for instance, in one eye the upper half, in the other the lower half
was Avanting,1 or both of the upper or both of the lower halves
ma}7 fail, or the defect may be confined to one eye. While cases of
these types may arise from intra-cranial lesions, they are more fre-
quently functional than organic, i.e., hysterical, or they depend on
causes within the eye such as partial embolism, detached retina,
etc.
Excluding the purely ocular cases, we divide hemianopia into two
broad classes, the peripheral and the central. Under the periph-
1 British Medical Journal, Nov. 22, 1890. Williams, Trans. Ophth. Soc.
United Kingdom, vol. xi., 190, 1891.
708 DISEASES OF THE EYE.
eral we place those arising from lesions of the chiasm and optic
nerves, and the central are such as depend on injury of the tractus,
the so-called primary ganglia, and the parts leading from them to
the cortex.
It is conceivable that a lesion at the canalis opticus may cause
hemianopia of one eye. Mauthner1 gives cases of monocular
hemianopia not due, as he thinks, to ocular but to nerve lesion-but
the diagnosis is not certain. Schmidt-Rimpler2 saw a case due to
aneurism of the internal carotid near the cavernous sinus. In
Mauthner's cases retro-bulbar neuritis is certainly not excluded,
because negative ophthalmoscopic appearances are frequent. It is
characteristic of peripheral cases that they shoAV a structural alter-
ation of the optic disc, either inflammation or atrophy. In rare in-
stances no such change appears, but if not an early, it is likely to
be an ultimate symptom. In central cases the contrary is most
frequent, i.e., the optic disc looks normal. To this rule exceptions
are more frequent than is the contrary in peripheral cases, because
we sometimes have to deal with cerebral tumors, and also if the
tractus be damaged inflammation or atrophy may reach the optic
disc. It has been noted by Mauthner3 and Wilbrand that in case
central hemianopia has existed for many years the eye whose tem-
poral field is Avanting and Avhich is supplied by the crossing fibres
will exhibit an atrophic disc and perhaps much reduced vision,
Avhile the other eye will be normal in appearance and function.
Peters4 relates such a case : A child nine months old received a
blow on the head which caused right hemianopsia—when seen at
the age of twenty-nine the right optic disc showed atrophy, V.=T2o°ir
—the left optic disc was normal, V. = fft. The explanation of this
fact will be seen by referring to the diagram from Henschen of the
arrangement of the crossing and the direct optic fibres in the disc
(see p. 651), by which the former overlie and partly conceal the
latter.
Within a few years attention has been called by Wernicke 5 to
a pupillary symptom Avhich may furnish a diagnostic sign between
peripheral and central hemianopsia. It is called the .hemiopic pu-
pillary inaction. The symptom has value when present, but to
utilize it the patient must be in a dark room and the illumination
carefully concentrated upon the extreme lateral half of the eye.
Furthermore we are told by very recent observers that it is not
always to be met Avith even in central disease. If the light be pre-
1 "Gehirn und Auge," 1881, p. 408.
2 " Augenheilkunde und Ophthalmoskopie," p. 137, 1885.
3L. c, p. 402.
4 Deutsche medizin. Wochenschrift, p. 1097, 1891.
6 Fortschritt der Medizin, i. No. 2, 1883. The same thing is discussed by
Wilbrand, " Ueber Hemianopsie," 1881, p. 89.
AMBLYOPIA AND AMAUROSIS. 709
cisely thrown upon the blind half of the eye, and the pupil do not
contract or contract sluggishly, Avhile if thrown upon the seeing
half it responds actively, the inference is that the lesion is at or in
front of the primary ganglia. If the pupil respond with nearly
equal readiness in case the light fall upon either the blind or seeing
half of the eye, the lesion will be located behind the primary
ganglia. The explanation is that Avhen the reflex arc for the pupil
is interrupted by disease of the primary ganglia (corpora geniculata
externa, pulvinar, corpora quadrigemina anteriora) or of the parts
peripheral to them, the same lesion which produces the hemiano-
pia extinguishes the pupillary reflex action and the hemianopic
pupil ensues. But a more central lesion which destroys the visual
function leaves intact the special fibres Avhich convey pupillary
impulses. Seguin 1 lays much stress on the value of this symptom.
A case is given by Oliver2 in Avhich the immobility of one pupil and
the sluggish reaction of the other aided in fixing the lesion as essen-
tially peripheral, although the inaction was not hemiopic. There
avis a tumor of the frontal lobe which sent down a prolongation
beneath the chiasm. Henschen reports some typical cases.
Bitemporal and binasal hemianopsia are explained by lesions
at the chiasm. The former implies a cause operating at the front
of the commissure or upon its ventral surface doing injury to the
crossing fibres of each optic ner\7e. It may be a tumor or an exu-
dation or deposit; it may spring from the sella turcica as a bony
growth. It may be caused by fracture at the base. Tumors may
form in this locality without disturbing the function of the chiasm;
see case by Bull3 in which concentric limitation of both fields and
late atrophy of the optic nerves took place, but not hemianopsia;
see also case figured by Henschen4 of one tumor beneath the chiasm
and another in the middle lobe of the cerebellum and whose posi-
tion could not be determined during the twelve days of observa-
tion before the patient's death. A case of Weir-Mitchell's5 Avas
diagnosticated correctly (a cyst as large as a lemon, caused by an
aneurism and attended by hydrocephalus externus—situation in
the sella turcica), and see Berry in Ophthalmic Review, 1S84, for
two cases and numerous references. See also Wiethe.6 Wilbrand,7
1 Journal of Nervous and Mental Diseases, xiv., Nov., Dec, 1887. Hed-
dreus claims priority in its discovery; see his treatise on the Pupil, 1880.
2 Trans. Amer. Opth. Soc, 1890, p. 479.
3 Trans. Am. Opth. Soc, 1892, p. 270.
4 " Klinische und Anatom. Beitrage zur Pathologie des Gehirns," Upsala,
1890, Theil 1., S. 98.
6 Journal of Nervous and Mental Diseases, 1889, xiv., p. 44, quoted by Wil-
brand.
6 Graefe's Archiv f. Opth., xiii., 3, 301, 1884.
1 " Ueber Hemianopsie und ihrer Verhaltness zur Topischen Diagnose der
Gehirnkrankheiten," 1881.
710
DISEASES OF THE EYE.
1881, collected sixty-four cases of lesions of the chiasm with au-
topsy. Very rarely is a true bitemporal hemianopsia present, be-
cause the tumor or other lesion is seldom small enough or so ex-
actly placed as to produce the supposed effect. On the contrary
we are apt to meet in one eye with total loss of sight or marked
amblyopia, and temporal hemianopia in the other. There will also
often be lesion of the third and fourth or other nerves, and various
" distant" symptoms. In rare cases there may be hemiplegia. At
some period, it may be late, there will be signs of optic neuritis or
atrophy seen by the ophthalmoscope. Convulsions are apt to oc-
cur and headache is usual and severe. In basal tumors numerous
symptoms arise as already said (see p. 158), and among them loss
of hearing, of smell, of taste, great neuralgia, and even exophthal-
mus can occur.—See case by Norris, Trans. Am. Ophth. Soc, 1890.
Binasal hemianopsia has been a few times recorded, but a
single case of Knapp's is as yet the only one with autopsy, and this
revealed an aberrant and sclerosed artery pressing on each side of
the chiasm. Similar conditions must be exceedingly uncommon.
Wilbrand J reproduces the visual fields of three cases of bitemporal
hemianopsia (Taf. VI.), one very precise and symmetrical due to
fracture at the base (no autopsy), and the other two with the re-
maining half-fields much encroached upon above, and there were
various cerebral symptoms (no autopsies). On the other hand he
quotes two cases of incomplete bitemporal hemianopsia, in one of
which the autopsy showed the cause to be a very large and widely
diffused tumor in the sella turcica whose precise origin could not
be traced and which had only partially injured the crossing fibres
of the chiasm. It is proper to add that Jatzow2 gives the visual
fields of ten patients in each of whom were found small insular bi-
temporal defects touching the vertical meridians and less than ten
degrees in area, which he explains by attributing them to neuritis
at the optic foramina—in both cases signs of optic neuritis were
present—and not to deeper lesions.
When Ave pass caudad of the chiasm to the tractus and deeper
visual paths we find hemianopsia to be always lateral and homony-
mous—that is, it is right-sided or left-sided. It may be complete
or incomplete, it may occupy one-half of each field, or one-quarter,
or a small sector, or an insular area. In some cases peripheral
parts of the half-fields are impaired, but the defects are ahvaj's
symmetrical in location and very nearly in outline. Sometimes a
very small test object must be used to discover them.
We have records of cases in which both halves of each field
have been invaded by successive attacks; for example, the half
1 "Die hemianopischen Gesichtsfeld formen," etc., Wiesbaden 1890.
2 Graefe's Archiv f. Ophthal, xxxi., 2, 264, 1885.
Noyes' " Diseases of the Eye."
Chart IV,
O.S. O.D.
Irregular homonymous hemianopia. Mrs. H., set. 27, Aug., 1879. While bathing and in
the water found sight suddenly impaired. Had some twitching of face and numbness of left
hand. No other symptoms. In October, 1888, heard that vision is the same; health good.
Cause probably a small hemorrhage—location doubtful,
180
O.S. V=A"
O.T>.Y = yh.
Mr. F., »t. 33 ; February, 1872. White atrophy of optic nerves. Syphilitic. Had brai
symptoms, but localization not practicable.
AMBLYOPIA AND AMAUROSIS. 711
of each field, and at a subsequent time a part of the remaining half,
leaving perhaps a quarter of the totality of each field intact. Sev-
eral such cases are quoted by Wilbrand, and he instances a number
Avhere total blindness Avas thus produced. But the most interesting
occurrences of this nature are described by Forster1 and Schweig-
ger.2 In Forster's case a man who became hemianopic on the right
side from apoplexy again became hemianopic, but on the other (the
left) side, by a second apoplexy five years later. At first inspection
the patient seemed completely blind, but there Avas a little activity
of the pupils to light and he could very sloAvly read S. H with each
eye—while V = fa. It was also found that he had a central field of
2|-0 to 3° in each eye below the horizontal meridian. There was no
perception of color. In Schweigger's case there was a similar cen-
tral field about 2° to 3° in diameter, V ^% and fa in the respective
eyes and color perception good. Tavo other well-observed cases
have been reported, one by Groenow,3 another by Berger.4 In the
former the fields were larger, oblique, and like the figure 8 in each
e3re—color sense good. There Avas loss of visual memories and of
localities, which implies a lesion in the lateral part of the occipital
lobe.
The variations in position and direction and character of the
boundary between the blind and seeing portions of the fields call
for some notice. Sometimes the field is cut into tAvo exact halves
upon the vertical meridian; again there is a semicircular notch at
the macula reaching over into the blind side; again the boundary
line is oblique or sinuous or it may be in part on the meridian and
in part not. In all cases of inequality of the two sides of the field
the blind portion is the smaller. The fact is very important to the
patient that a slight portion of the blinded side remains unhurt,
because it enables him in case the right half is gone,to read Avith
much greater ease than Avould othenvise be possible—because he
can anticipate the coming letters as Ave habitually do.
The explanation of this circumstance includes the explanation
of the preservation of the very small central fields noted in cases
of hemianopsia attacking each side of each eye in succession
(Forster, ScliAveigger). Forster assumed the existence of a special
blood supply from the pia mater at the cuneus for the region cor-
responding to each macula, but both Wilbrand and Henschen with
more probabilitj7 attribute the fact to the existence in each cuneus
of cortical structures representing each macula, or those parts in
each retina adjacent to the A7ertical meridian which giA7e Avhat
1 Graefe's Archiv f. Ophthal., xxxvi., 1, 1890.
2 Archiv f. Augenheilkunde, 2 and 3, S. 336, 1890.
3 ArchiA7 f. Psychiatrie, xxiii., 339.
4 Breslau Arztlich. Zeitschrift, 1885.
712
DISEASES OF THE EYE.
Wilbrand calls the overlapping area. This means in other words
that in all such cases each macula receives fibres and cortical im-
pulses from each cerebral centre.
A careful study of cases shows that the presence or absence of
an overlapping area has no relation to the portion of the visual
tract whose impairment causes the hemianopsia, i.e., as Gowers
says, no localizing inferences can be drawn from the varieties of the
boundary. Wilbrand (1. c, p. 47) remarks that the papillo-macular
bundle is of dissimilar size in different cases, and this evidently has
relation to the extent of an overlapping or common area in the
central region of the retina.
Before proceeding further it remains to be stated that we find
in addition to the hemiopic loss of light sense two other conditions,
viz.: first, cases in which there is half-blindness for color with pres-
ervation of light sense, i.e., hemi-achromatopsia; and second, cases
of visual hallucinations affecting only one-half of each field. The
former, A7iz., the hemiopic loss of color sense, has been found to be
confined in a few cases to one eye, Avhich proves that it is not always
due to a cortical or subcortical lesion. Siemerling (see Wilbrand,
1. c, p. 144) reports loss of light sense in one-half of one field and
only loss of color sense in the corresponding half of the other field.
Usually both eyes are affected, and the same is true of the hemiopic
hallucinations. Both these conditions may exist together, as shoAvn
by Wilbrand x in a case of a small clot in the subcortical visual
region near the posterior horn of the lateral ventricle. See also
case by Putzel.2 Swanzy3 enumerates eight cases, and in one
autopsy showed an old clot in the A7isual tract of the occipital lobe
below the posterior horn of the lateral ventricle.
We may now take up the consideration of hemianopsia caused
by lesions of the tractus and deeper parts. The optic tract for
about one-third of its course lies exposed at the base of the brain,
but then passes under the edge of the temporo-sphenoidal lobe
and winds up obliquel}7 around the crus. It is also crossed by the
third nerAre. Being thin and flat and narrow it is of course most
likely to be totally rather than partially damaged by a lesion.
Yet an instance of partial injury is given by Marchand 4 due to a
glioma in the right temporal lobe near the gyrus hippocampi, which
pressed upon its edge only, and caused loss of homonymous quad-
rants of the fields; there was slight double optic neuritis. See also
case by Norris, 1. c. It is naturally unlikely to be the seat of hem-
orrhage because of its small size, yet such cases are recorded
1 " Die hemianopischen Gesichtsfeld formen," etc., 1890, p. 56.
2 NeAv York Medical Journal, Aug., 1890, Jan., 1891.
3 Trans. Ophth. Soc. United Kingdom, ix., p. 23, 1889.
* Graefe's Archiv f. Oph., xxviii., 2, 64, 1882.
AMBLYOPIA AND AMAUROSIS.
713
(Wilbrand), and its vascular supply from the pia mater is common
to it and the chiasm and not associated with that of the crus. It
may be damaged by tumors proceeding from the base of the skull,
or from the adjacent crus or temporo-sphenoidal lobe. Norris1
gives a case of blindness of one-fourth of each field explained by a
glioma in the temporo-sphenoidal lobe, pressing on the tractus.
For other cases of tumor in this region, see Wilbrand, " Ueber He-
mianopsie," p. 93. It may also suffer from localized meningitis.
Lesions at the base may involve other nerves, especially the third,
the fourth, branches of the fifth and the sixth, while facial and audi-
tory and deeper nerves ha\7e also been noted to be affected. The
functions of the crus may be impaired, causing hemiplegia as the
result of a tumor in the temporo-sphenoidal lobe. There may be
hemianopsia with hemiplegia, hemianaesthesia, and impairment of
the lower facial and the hypoglossus, and the cause may be either
in the crus or in the internal capsule, more frequently in the latter
region. If the above symptoms be combined with oculo-motor
parah'sis of the opposite side the lesion Avill be in the crus.
Lesions of the tractus may ultimately be attended Avith signs
of optic nerve atrophy, or with neuritis when caused by tumors.
Some stress is laid upon the quality of the half-blindness. It is not
a gray or dark appearance, but is said to be an absolute absence
of all sense of light, which is not the fact in cortical half-blindness.
The hemiopic pupillary inaction Avill exist. What has been said of
the symptoms of tractus lesions applies in great measure to all the
primary ganglia, and Ave can almost never succeed in declaring
Avhich of them are involved. Henschen2 and Wilbrand give his-
tories and autopsies of cases where the several ganglia were dis-
eased, but no clearly significant symptoms can be elicited. What
most conspicuously presents itself for solution is the nature of the
lesion, and the general localization near the primary ganglia is
usually all that is possible. In Oliver's case a careful analysis of
all the symptoms of such a lesion is made, and the diagnosis
proved correct (Trans. Am. Oph. Soc, 1890, p. 479). The near
vicinity of the internal capsule excites various motor S37mptoms,
spasmodic or paralytic, as Avell as disturbances of the several
varieties of sensation. Lesions of the optic radiation and of the
cortex are now more readily recognized than formerly. Hemi-
anopsia may occur from direct interference Avith the visual path and
cortex or as a remote symptom. It is sometimes temporary at
the outset of an apoplexy in the cerebrum (Gowers). When the
course of the optic radiation and its precise form and size as de-
duced by Henschen, and the nearness of its approach to the pos-
1 Trans. Am. Ophth. Soc, 1890, p. 470.
2L. c, Zweiter theil, s. 207.
714
DISEASES OF THE EYE.
terior horn of the lateral ventricle, are considered, we can understand
how readily it may be encroached upon from all sides. We also
bear in mind that while the calcarine fissure is signalized as the
focus, so to speak, of the visual fibres, the whole of the cuneus and
the angular gyrus and adjacent occipital gyri are the seat of visual
memories, and that Avhen they are damaged hemianopsia may oc-
cur because the influence of the lesion may reach farther than
is apparent. In cortical hemianopsia we often have aphasia and
mind-blindness, especially if the lesion be on the left side. The
symptoms suggest lesions along the middle cerebral artery running
through the fissure of Sylvius (see Fig. 242). The aphasia is due
to the mind-blindness being amnesic, because Avords recall no
memories, and therefore correct speech is impossible. There may
of course be alexia and also agraphia. There may be monoplegias
of A7arious types, convulsions, athetosis, hemichorea, and various
distant symptoms, and they happen also in the lesions near the
primary ganglion. The diagnosis of cortical or subcortical hemi-
anopsia is not ahvays easy, but is founded upon the pupillary symp-
tom of Wernicke and upon the absence of hemiplegia and other
signs referred to above which appear usually in connection Avith
lesions of the pons, the crus, and the internal capsule.
It often happens that besides the hemianopia there is peripheral
limitation of the remaining half-fields. A marked illustration is
the case reported by Oliver (1. c, Trans. Am. Oph. Soc, 1890), where-
the remaining half-fields are extremely reduced. This is explained
by the general disturbance of the brain oppressing the A7isual
tracts. Analogous to this condition is the peripheral limitation
without hemianopsia, observed Avith tumors located in A\arious
positions not near the Aisual paths. There may be no optic neuri-
tis to account for it. Such a case is figured by Henschen, Taf.
XXXI., and others, and he remarks that these peripheral limita-
tions have no special significance. They are found with tumors.
and also with softening. Henschen giA7es the restricted fields in
several cases Avhere there was softening in the occipital lobes.
Wilbrand * gives the fields of a case of lesion of the internal cap-
sule in which the original conditions shoAved extreme limitation
besides hemianopsia, and after twelve months, recovery so far took
place as to leave only a quadrant of each field defective. He also
gives the fields of the same case taken by less acute and patient
observers before he made examination, and shoAvshow the extreme
difficulties belonging to such patients make errors very likely. He
justly suggests that imperfect examination, probably explains the
erroneous inferences of Charcot as to the occurrence of crossed
1 "Ueber SehstOrungen bei functionellen NerA7enleiden," 1892, pp. 47-54.
AMBLYOPIA AND AMAUROSIS.
715
amblyopia in lesions of this region. His assumption of a double
crossing of the visual paths has no anatomical foundation.
Diagnosis.—We have to consider the position of the lesion and
also its character. What has already been said gives hints in re-
gard to locating the lesion, but it is evident that hemianopsia in
Fig. 258. —Diagram (from Seguin) to illustrate Left Lateral Hemianopsia. LTF, left temporal
half-field; RNF, right nasal half-field; OS, oculus sinister; OD, oculus dexter; N. T., nasal and
temporal halves of the retinae; NOS, nervus opticus sinister; NOD, nervus opticus dexter; FCS,
fasciculus cruciatus sinister; FLD, fasciculus lateralis dexter (fasciculus non-cruciatus dexter); C,
chiasma or decussation of fasciculi cruciati—(commissures of Gudden and Meynert are omitted);
TOD, tractus opticus dexter; CGL, corpus geniculatum laterale (corpus geniculatum mediale and
the brachia are omitted); LO, lobi optici (corpora quadrigemina); POC, primary optic centres (in-
cluding corpora quadrigemina, corpora geniculata and pulvinar of thalamus opticus); FO, fascicu-
lus opticus, radiating visual fibres of Gratiolet in the internal capsule; CP, cornu posterius of lat-
eral ventricle; GA, region of gyrus angularis; LOS, lobus occipitalis sinister; LOD, lobus occipi-
talis dexter; Cm, cuneus and subjacent gyri constituting the cortical visual centre in man. The
heavy and shaded lines represeut the parts connected with the right halves of the retinae.
itself alone does not point to a definite locality. It must be inter-
preted in the light of concomitant symptoms. The nature of the
lesion must at the same time be considered. Apoplexy, embolism,
softening, tumor, gummy exudation, sclerosis, meningitis, tubercle,
716
DISEASES OF THE EYE.
aneurism, etc., these are to be passed in review. Moreover, Ave may
have multiple lesions, viz., several tumors or hemorrhages or spots
of softening, etc. We likewise have to look for remote disease,
albuminuria, diabetes, heart disease. Sometimes a retinal lesion
will give us light on this point, viz., spots of exudation or of hemor-
rhage. We have to take into account the history, rate of prog-
ress, and all the distant as well as focal symptoms.
A valuable epitome of symptoms is summed up in a series of
rules laid doAvn by Seguin,1 Avhich may be quoted.
1. Lateral hemianopia always indicates an intracranial lesion
on the opposite side from the dark fields. 2. Lateral hemianopia
with pupillary immobility, optic neuritis or atrophy, especially
if joined with symptoms of basal disease, is due to lesion of one
optic tract, or of the prima^ optic centres of one side, i.e., the
corpora quadrigemina and parts included within P.O.C.(see Fig. 258).
3. Homonymous sector-like defects of the same geometric order, Avith
hemianesthesia and choreiform or ataxic movements of one-half
of the body, without marked hemiplegia, are probably due to lesion
of the caudo-lateral part of the thalamus, or of the posterior (cau-
dal) portion of the internal capsule C. P. or F. O. 4. Lateral hemi-
anopia Avith complete hemiplegia (spastic after a few weeks) and
hemianesthesia, is probably caused by an extensive lesion of the
internal capsule in its knee and caudal part (pulvinar), i.e., farther
back and more profound than in supposition 3. 5. Lateral hemian-
opsia with typical hemiplegia (spastic after a few Aveeks): apha-
sia, if the right side be paralyzed and with little or no anesthesia,
is quite certainly due to occlusion of the middle and adjacent cere-
bral arteries Avith extensive superficial lesion, softening of the motor
zone and of the gyri lying at the extremity of the fissure of Sylvius,
viz., the inferior parietal lobule, the supra-marginal gyrus and the
gyrus angularis. There may also be alexia, word blindness. 6.
Lateral hemianopsia Avith moderate loss of poAver in one-half of the
body, especially if associated with impairment of the muscular
sense, would probably be due to a lesion of the inferior parietal
lobule and gyrus angularis with their subjacent white substance,
penetrating deeply enough to sever or compress the optic fasciculus
on its way posteriorly to the visual centre. If mental blindness
exists, the.lesion Avould lie in the more anterior central parts of the
occipital lobe. 7. Lateral hemianopia without motor or common
sensory or any accompanying symptom is due to lesion of the
cuneus only, or of it and the gray matter immediately surrounding
it, on the mesial surface of the occipital lobe in the hemisphere op-
posite the dark half-fields. The lesion may be partial or total.
'Journal of Nervous and Mental Diseases, xiii., Jan., 1886.
Noyes' " Diseases of the Eye."
Chart V.
Miss S., April, 1879, p. 644.
"* iao7
o.s. O.S.
Hemianopia. Miss S.. August, 1879, p. 717
AMBLYOPIA AND AMAUROSIS. 717
Most surgical cases come at once after convalescence within this
rule or within rule No. 6.
In all cases coming under rules 3 to 7, inclusive, the pupils re-
act normally; and rarely does the ophthalmoscope show any lesion
of the optic nerve, except of course in some tumor cases, where
neuro-retinitis may be expected.
Prognosis.—Cases of short duration Avith perfect recovery are
recorded. The defect at first complete,may after a time become
less extensive. This is chiefly true in apoplectic cases. The larger
number of cases remain unchanged. Prognosis depends chiefly
upon the nature of the lesion if this can be made out.
Chart V. gives the fields in a case seen by myself where partial
recovery occurred. The history is as follows:
Miss S---, aged thirty, came to me in April, 18T9. She was forewoman
in a large establishment, and, being extremely busy, had not slept well for
many months. Four weeks ago she had an attack of severe pain, running
from the left clavicle and shoulder to the head, Avhich drew it to one side,
and prevented sleep for two nights. Two Aveeks afterward she found herself
totally blind on the right side. There were no other symptoms. Examina-
tion found no disease of the kidneys, nor of the heart; she had not had rheu-
matism. Vision in each eye f §. By the ophthalmoscope: right disc not
SAvollen, vessels full; near it the retina a little oedematous, and has some dots,
while a bluish zone surrounds almost all the disc. Left eye about the same.
Visual fields have this peculiarity: that while there is symmetrical hemiano-
pia, perception remains on the extreme periphery of the blind sides. Treat-
ment was iod. potas., gr. x., ter in die. After a month her condition, which
had been one of great excitement, because of her impaired sight, Avas more
calm, and she slept well, yet felt very tired. Had some numbness of the left
leg and hand. Visual fields then shoAved a clearing up of the lower half of
the previously blind portion. See charts. In August following, the fields
were about the same, and in the retinae there Avere fewer specks than at first.
The right optic nerve Avas more hyperaemic than the left. Patient seen in
1882 and condition of fields the same: vision normal.
There is of course a serious question often as to life. For some
recoveries of sight see Lang 1 and Wilbrand.2 Michel3 says that
in 154 cases of hemiplegia,hemianopia occurred 59 Limes.
It may be said that patients are sometimes unaware that they
are hemianopic, because other paralytic symptoms absorb their
attention. In one instance I performed extraction of cataract for
an old lady and Avas not made aware of her hemianopia until the
time came for fitting glasses.
Treatment.—This must be in accordance with the indications of
the situation and cause of the mischief. For inflammatory cases
1 British Medical Journal, Nov. 23, 1889.
2 "Die Hemianopsie," 1881, p. 205.
s " Die Lehre der Augenheilkunde," p. 607.
718
DISEASES OF THE EYE.
the iodides, for man}7 cases bromides will be serviceable. We may
have to administer to severe pain by antipyrine or opium; Ave may
require derivatives, ice to the head, mustard to the lower limbs, dry
cups to the nape or temples. We must endeavor to decide upon
the nature and cause of the lesion to properly choose a line of
treatment. The eye symptoms are merged in the other features
of the case in most instances. In late periods strychnia injections
sometimes do good.
Literature on this subject is very extensive. Among papers
not referred to in the text are Starr, Amer. Journal of Medical
Sciences, Jan., 1884; Seguin, Journal of Nervous and Mental
Disease, 1886 and 1887; Hun, Amer. Jour, of Medical Sciences,
Jan., 1887 (includes his famous case of partial cuneus lesion);
Swanzy, Archives of Ophthalmology, 1891; ScliAveigger, Graefe's
Archiv f. Ophthal, XXIL, 3, 297, 1876. The seA7eral treatises of
Wilbrand, 1881, 1884, 1887, 1890, 1892, compose a library of infor-
mation. The contribution of Henschen, of which two parts are
out (Upsala 1890, 1892), is a magnificent example of exhaustive
work both in original research and compilation, Avith spendid illus-
trations. See also Magnus, " Anleitung zur Diagnostik der central
Storungen des optischen Apparates; " Tafel und Text, Breslau, 1892.
Amaurosis in Young Children.
It is not pretended under this head to give a nosological de-
scription of a particular disease. Many lesions of the optic nerve
and brain cause loss of sight in the early period of life. The condi-
tions may be congenital. What makes brief reference desirable is
the fact that while sometimes lesions of the optic nerve or retina
may be found, in certain cases none will be discovered. Moreover,
there will be sometimes unmistakable head symptoms, such as fits
and vomiting, headache, great excitability, or contrariwise stupor,
or unconsciousness; there may be motor disturbances in wasting
of the muscles of the face or limbs, paresis or paralysis, hydro-
cephalus, etc. Hereditary tendencies must be investigated, and
especial care given to search for syphilitic or tubercular, or men-
ingineal symptoms. Many cases are and remain blind, but on the
■* other hand partial and even complete recoveries are by no means
rare, and for useful observations on this subject see Vol. IV. of
Trans. Ophthalmological Society of United Kingdom, 1884, papers
by Mr. Nettleship. See also reference under head of retro-bulbar-
neuritis.
Undiscovered Monocular Blindness.
It is not very rare to haA7e a patient tell of sudden loss of sight
in one eye, discovered by accidentally closing the other. He nat-
AMBLYOPIA AND AMAUROSIS.
719
urally supposes the condition to have been suddenly produced;
examination may find complete optic nerve atrophy or choroidal
lesion, or extreme hyperopia, myopia or astigmatism, cataract, etc.,
none of Avhich can be recent. Such states of vision often exist with
strabismus.
On the other hand, total blindness may take place in one eye
without immediately attracting the patient's notice, especially if
there be some high optical error. An incident1 of this kind took
place in a young girl who Avas myopic 13 D, and in one eye sud-
denly found, on closing the other, that she had no perception of
light. Treatment by artificial leech and tonics Avas folloAved in tAvo
months by restoration to her previous condition. She Avas not
hysterical, had no cause for deception, and no explanation Avas
found by the ophthalmoscope. She had had severe headache for
six months; catamenia Avere irregular.
Simulated Blindness.
In countries where militar}7 service is compulsory, persons
sometimes seek to escape it by pretending to partial or total loss
of sight in one or both eyes. Such malingering is also practised
to secure pensions or to obtain compensation after the receipt of
injuries, and it sometimes presents itself as the expression of a
perverted imagination, among children and hysterical persons.
Pretence to total blindness is uncommon; to blindness more or less
complete in one eye (the right) is the usual claim. It is common
to find great exaggeration of an existing visual defect due perhaps
to opacity of the cornea, to myopia, to irregular astigmatism or
hyperopia or to congenital amblyopia, etc. The unexpected dis-
covery of the need of strong convex glasses is sometimes coincident
with an injury and will give plausibility to the claim that the con-
dition is entirely traumatic.
A striking instance came under my care in a man 45 years old with hyper-
opia 3 D, who had never used or needed glasses, and was wounded in the left
eye by a piece of glass in a railway collision. The wound was through the
ciliary region and severe. It Avas necessary to perform iridectomy, and the
patient suffered much. His real suffering was in my judgment far less than
he wished me to believe it to be. During a treatment extending through
several months, his pain and intolerance of light and grimaces of agony were
so disproportioned to the pathological conditions that I felt sure he was prac-
tising deceit. A suit for damages against the railroad company and an effec-
tive exhibition of his hypocritical symptoms before the jury, brought him
the handsome sum of $7,000. A month later he came to me with much candor
to show how remarkably well was his eye and free from trouble, while with
1 Dr. H. Derby, Boston Medical and Surg. Journ., Feb. 7th, 1884, p. 126.
720
DISEASES OF THE EYE.
proper correction by a spherico-cylindric glass vision was §-§• and it had been
the same before the trial of his suit in court. His vision continued equally
good for three years.
It is difficult to unmask the pretence of total blindness. One
must have opportunity to watch the person Avithout his knoAvledge.
By the ophthalmoscope nothing abnormal Avill be seen. The a etion
of the pupils will be noted, and one will be ready to suspect fraudu-
lent use of a mydriatic if dilated pupils fail to expand a little more
Avhen the eyes are in shadoAv, it being assumed that no lesion of the
fundus exists. The disposition of the pupils to contract upon con-
vergence of the visual lines will be remembered.
A person totally blind has a manner and carriage of the head
which are characteristic. If blind for a long time he has usually
acquired confidence in the guidance of another person and will Avalk
briskly when led by the arm. He will carry the head erect, often
thrown a little back or to one side. If told to look at his own hand
he will attempt to do it, and by strenuous insistence of voice and
placing his hand in front of the face he will look not far from its true
position by the help of general sensation. A malingerer will not be
likely to act thus. He will profess entire inability to look toward
his own hand, and will assert his entire helplessness. This stamps
his hypocrisy. One may flash the direct light of the sun for an
instant into the eye and mark the effect. It is not usual for skilful
malingerers to pretend to total inability to see light. They know
that this is unnecessary for their purpose. Hence the detection of
their fraud, if both eyes are claimed to be imperfect, must be made
by setting a watch upon their behavior and endeavoring to sur-
prise them when off their guard.
For detection of simulation of blindness in one eye Ave resort to
various devices. Let a prism be put before one eye with its base
vertical and oblige the person to walk some distance. He will be
obliged to shut one eye to escape the confusion of double vision if
stones and steps are in his way. Going downstairs will be a sharp
test when Avearing spectacles of this sort. Viewing a candle flame
at tAventy feet through prisms each say 7° with bases outward will
lead to convergence of the visual lines to unite the images, and the
action may be discovered by the observer.
A prism of 5° may be placed with its apex across the middle of
one pupil and thus cause monocular diplopia, using for the purpose
the good eye and screening the other. When the person is ac-
quainted with this phenomenon and admits its existence, a trifling
movement of the prism to cover the whole pupil and the simul-
taneous removal of the screen is likely to be folloAved by the ad-
mission that double images are still present, and the'fraud is
disclosed, because tAvo eyes are now being used (Alfred Graefe).
AMBLYOPIA AND AMAUROSIS. 721
Snellen arranged a series of letters to be hung in the AvindoAV
and alternate letters are made red or green by shifting behind them
a piece of red or green glass. The person examined vieAvs the
letters through spectacles of which one side is green and the other
red. As the letters are altered in color, the green Avill be invisible
to the eye Avearing the red glass, and the red invisible to the eye
Avearing the green glass. Stilling's colored letters may be used for
the same purpose. If the pretended blind eye can see green letters
through a red glass, the person lies. The red glass will be OA~er
the good eye and plane glass over the assumed bad one.
One may use strong com7ex and concaA-e glasses to view distant
print; putting a plane glass before one eye and by a reversible
frame make the person use unconsciously his bad eye.
The stereoscope can be utilized in A7arious ways. The person's
eyes must be exposed constantly to view and he must not see be-
forehand the cards put into the slide. On one side may be the
letter L and on the other F. Combined they make E, and if this be
admitted the case is proven. Other figures and marks may be used
Avhich differ on the two sides and by several trials success may be
achieved.
Harlan puts before the pretended blind eye a plane or weak
concave glass, — 25 D, and before the good eye a strong + glass,
say + 16 D. If the patient read the distant test type he convicts
himself.
With hysterical persons and children pretended blindness may
be the effect of self-deception. Occlusion of both eyes by plasters
or a bandage as a means of cure, is likely after a sufficient lapse of
time to haA7e this result. One may make a hypodermic injection
into the temple of a minute dose of strychnia, using say three drops
at a time, twice daily, of a solution gr. i. ad 3 i. The acupuncture
Avill be more effective than the strychnia. Some hysterical persons
have a deliberate purpose to deceive, and the claim of poor sight
must be judged in the light of other symptoms. I once cured a
young girl of excessive photophobia by subjecting her to chloro-
form and on recovery she was fully able to face the open windows.
46
CHAPTER XX.
THE ORBIT.
Anatomy.—The cavity which contains the eyeball is a quadran-
gular pyramid, in which Ave distinguish the base or opening, the
four Avails, and the apex. The angles of the base are rounded, and
the infero-temporal angle is at a lower level than the infero-nasal
angle. In other words, the upper and lower sides haA7e a slope
doAvnward toward the temple. The edge is somewhat sharp on
three sides, because it overhangs the interior surface. On the
fourth, viz., the internal side, the edge rounds off and slopes toward
the median line. The orbital margin is capable of great resistance,
because the bone is dense, and is buttressed by the zygomatic arch,
which expands into the malar bone, and by the arch of the frontal
bone. On the medial side, where strength is not needed, the bones
are very thin, but their celluloid arrangement gives great capacity
for dispersing the force of shocks. On the wall of the inner margin
Ave have the groove in which is lodged the lachrymal sac, in front
of Avhich is the insertion of the orbicularis, and behind which is the
insertion of the tensor tarsi. This edge is formed by the ascend-
ing process of the superior maxillary, which joins the nasal process
of the os frontis. The groove for the lachrymal sac is mostly chan-
nelled out of the lachrymal bone. At the supero-nasal angle the
supra-trochlear arteries and veins are found, and a little behind the
edge is the loop through which passes the tendon of the superior
oblique (trochlearis) muscle. At the distance of from six to ten
millimetres from the supero-nasal angle we have a foramen, or, it
may be, a notch Avhich gives passage to the supra-orbital nerve,
and to a small arterial tAvig. Beyond this the edge overhangs the
cavity more decidedly, until at the supero-temporal angle Ave find
behind it a decided fossa, in Avhich is lodged the lachr37mal gland.
Passing around the outer to the loAver border, we find, a little in-
side the middle of the latter, the region Avhere the infraorbital nerve
emerges. It comes out of the bone about eight millimetres below
the edge. The orbital margin is composed of the superior maxil-
lary, the frontal, and the malar bones. The prominence of the
frontal sinuses makes this the most elevated part of the region of
the base, Avhile at the temporal side the bone is thickest and most
THE ORBIT.
723
dense. The inner wall of the orbit is smooth and polished, pre-
senting the surface of the os planum of the ethmoid, and here we
have tAvo foramina in the suture betAveen the os frontis and the
orbital lamina of the ethmoid; the anterior foramen gi\-es passage
to the nasal branch of the ophthalmic nerve into the skull, and the
latter to an artery into the nose. The superior wall, or roof, of
the orbit is slightly conca\7e and smooth. Immediately next to it
is the levator palpebrae superioris; it is very thin, and at its an-
terior and inner part it separates into tAvo layers, between Avhich is
the frontal sinus. The surface of the outer wall is nearly flat, in-
clines outward from the median plane, and is composed of the
greater wing of the sphenoid, and of the malar bone. The inferior
Avail is thin, and furrowed by the groove for transmission of the
infraorbital nerve; below it lies the antrum. Between the outer
and the inferior walls, at their place of junction, is the spheno-
maxillary fissure, which sweeps from without and beloAv, imvard
and upward in an imperfect right-angled bend, bounding the pos-
terior part of the body of the superior maxillary bone, and opening
into the muscular mass and the vessels Avhich lie about the ptery-
goid process. It is through this fissure that a cut is made in ex-
cision of the upper jaw. The fissure is sometimes called the inferior
orbital. At the angle of junction between the superior and outer
Avail is another fissure, shorter than the preceding, which separates
the lesser and greater Avings of the sphenoid, and is called the
superior orbital, or sphenoidal fissure. It gives passage to all the
motor nerves of the eye, to the ophthalmic nerA'e, and to the oph-
thalmic ATein, while it has upon its cranial side the cavernous sinus,
and is occupied by dense connectiA-e tissue, AA7hich shuts up the
aperture firmly. At the apex of the orbit, above the inner end of
the sphenoid fissure, is the optic canal or foramen, Avhich perforates
the sphenoid at the junction of its wings Avith its body. The canal
is cylindrical, is rather larger in front than behind, i.e., funnel-
shaped, is from eight to nine millimetres long, and about six milli-
metres in diameter on the a Average. Its course is from below and
outward, upAvard and to the median line, and the canals of opposite
sides com7erge to each other. Posteriorly they open into the middle
cranial fossa. The canal contains the optic nerve and the ophthal-
mic artery. The sphenoidal fissure running outward, and the
spheno-maxillary fissure running doAvnward and outward, meet
and become continuous Avith each other just beloAv the optic 'canal.
These fissures are of variable length and breadth; they may differ
on the opposite sides of the same person, and usually grow larger
in later life. The spheno-maxillary fissure contains fat and connec-
tive tissue, and some vessels which communicate with the internal
maxillaiy.
724
DISEASES OF THE EYE.
The dura mater adheres to the sphenoidal fissure and to the
optic canal very firmly. It clothes the surface of the optic canal,
and, curving forAvard, is continuous Avith the outer sheath of the
optic nerve and with the periosteum of the orbit. There is also an
inner sheath of the optic nerve which is continuous with the arach-
noid, and which at the optic canal is firmly attached to the outer
sheath by meshes of connective tissue. In many cases this mesh-
Avork is sufficiently open to permit fluid to be injected from the
cavity of the skull into the space betAveen the two sheaths of the
nerve, as Avas proven by Schwalbe. But this is not invariably
possible, and in all cases the optic nerve is so closely attached to
the wall of the bony canal that it cannot be pulled aAvay from it.
Schwalbe says this adhesion is most intimate at the upper part of
the canal. The ophthalmic artery coming from the internal car-
otid and about two millimetres in diameter, lies in the canal beloAv
and to the outer side of the nerve. The recti muscles and the
superior oblique originate around the opening of the optic canal,
the rectus externus having tAvo roots, betAveen Avhich passes the
third nerve. The course of the muscles and the arrangement of
the oculo-orbital fascia have been described. It may be proper to
refer to the so-called orbital muscle of Miiller, which does not admit
of demonstration, but consists of numerous fibres of non-striped
muscle controlled by the sympathetic nerve in the neck. They
influence to a certain extent the degree of exophthalmus or its
opposite state of enophthalmus. They are found in the fissura
orbitalis, the inferior or spheno-maxillary. The ciliary ganglion
is referred to on p. 400.
The orbits stand to each other in such relation, that their axes
form an angle opening forAvard of about forty degrees, Avhose apex
would be at the middle of the anterior clinoid process of the sphe-
noid. The floor of the orbit slopes downward, forAvard, and out-
ward; the shape of the cavity thus inclines the eyeballs to assume
a position looking outward and doAvnward. The globe is placed so
as to lie nearer to the outer wall than to the inner, while the optic
canal is aboAre the leArel of the middle of the eye.
The remark must not be omitted, that owing to congenital
asymmetry of the skull it is quite frequent to find the floors of the
orbits not on the same plane. One will be higher than the other.
This appears in the position of the auricles as well as of the eyes.
Sometimes the action of the ocular muscles is for this cause
rendered painful—hyperphoria occurring—and if not painful their
unusual behavior under prism tests will be accounted for.
THE ORBIT.
725
Diseases of the Orbit.
We consider: 1st. Inflammatory conditions Avhich may take
place in the periosteum, in the connective tissue, in the capsule
of Tenon or oculo-orbital fascia. Inflammation may arise spon-
taneously or extend into the orbit from the face or adjacent
cavities. The bony Avails may be diseased, giving rise to hyper-
trophy or more frequently to caries. They maA7 also be perforated
or pushed inAvard by diseases in adjacent cavities. 2d. We have
spontaneous hemorrhage into the orbit. 3d. It is a favorite site
for the growth of tumors. 4th. We also have to consider traumatic
lesions. Because situated Avithin the orbit Ave might refer to in-
flammations and diseases of the lachrymal gland, but they have
already been discussed.
There are two important symptoms peculiar to orbital diseases;
one is displacement of the globe (exophthalmus), and the other is
its immobility, the latter, hoAvever, may occur through paralysis of
all its muscles and will then be differentiated by the co-existing
ptosis.
Periostitis Orbit^e.
This appears as an acute and a chronic condition. The causes
are traumatic, rheumatic, syphilitic, and so-called scrofulous. The
most common are syphilitic or scrofulous, and the disease is most
frequent in children. The favorite locality is the margin of the
orbit, while if the Avails are affected the symptoms are somewhat
different and more serious. If the disease is marginal and acute
there will be pain; oedema of the lids; some chemosis, beginning
equatorially; and the distinctive symptom is that the edge of the
orbit is very tender Avhen pressed by the finger and there may be
a tense SAvollen spot, highly sensitive and at a later time there may
be fluctuation. I have seen this symptom in exquisite degree in
cases of syphilitic acute periostitis, in Avhich the diagnosis was per-
fectly palpable (sit venia verbo!). The chronic form is diagnosti-
cated with more difficulty, except when nodular bony swellings
appear Avithin reach of the finger, and they may interfere Avith the
function of the nerves or muscles, or even with the position and
movements of the eye.
The disease when chronic is very tedious, lasting for months
and even for years. The course of the acute affection is various.
If an abscess form near the surface and is promptly opened, the
case may terminate soon and without retraction of skin or de-
formity. But if the case be neglected or the constitution unfavora-
726
DISEASES OF THE EYE.
ble, general phlegmonous inflammation may ensue, and this result
is the more likely the deeper the site of the trouble. In chronic
cases we often have caries of the bone, fistuheT retraction and ad-
hesion of the skin, deformity of the lids, either upper or lower, and
especially ectropium. Necessarily the eyeball suffers both in the
conjunctiva and possibly in the cornea. See chapter on the lids.
If the disease is parietal, its recognition becomes more difficult
the deeper its situation, and also more serious according to the im-
portance of adjacent regions, e.g., the roof. Besides pain, swelling
of lids, chemosis, stiffness and pain in moving the eye, there will be
pain on pushing the globe backward, it may or may not be laterally
displaced or perhaps pushed a little forward. There will be noc-
turnal exacerbations of pain and perhaps rise of temperature. A
deep digital exploration is of great importance to detect some spot
of marked tenderness. The smaller the surgeon's finger the greater
his advantage. In some cases it is alloAved to make a deep explora-
tory incision under antiseptic precautions with a narroAv blade or
exploring needle. Such a condition befell a friend of mine, a lady
about fifty years of age. No cause except a rheumatoid diathesis
could be given.
She had constant and troublesome pain over the forehead,
aggravated at night, and lasting for some weeks. It became
localized over the supra-orbital notch, and was attended by swelling
of the lid in that region. I ventured, after some delay, to pass in
a narrow knife, and on probing reached a spot of rough bone. Pus
in small quantity escaped, a fistula was established, and after many
months, and persevering treatment by injections, it was closed up.
It may happen, that when not evacuated in front, such an abscess,
breaking down the bony tissue, shall find its way into surrounding
parts, viz., into the ethmoid, the antrum, the frontal sinus, or into
the cavity of the skull and brain. The consequences of these seA-eral
events are readily understood. Again, caries or necrosis may take
place, and when the dead tissue reaches the surface, and comes out
through the formation of an abscess, a fistula ensues, and, when it
heals, deformity of the skin and lids will follow, viz., ectropion, or
exophthalmus, etc., of various degrees. A very notable case of this
kind came to me some years ago, and was published in the Trans.
Am. Oph. Soc, p. 129, 1870. The disease began at one year of
age, and when I saw the young lady she Avas sixteen. Pieces of
bone had been discharged. When I saAv her, there Avas protru-
sion of the globe from the orbit, the cornea was opaque and
turned downward; the upper lid adherent to the upper margin of
the orbit, and fully everted; the conjunctival surface covered by
thick and coarse granulations; the globe constantly exposed, and
requiring to be covered by a pad or the hand. In the orbital edge
THE ORBIT.
727
was a deep sulcus, from which bone had been exfoliated. The other
eye intolerant of light, and in a state of chronic irritation. The
treatment adopted, and which Avas the only resource, Avas enuclea-
tion of the globe, removal of all of the conjunctiATa, bringing down
the lids to proper position, and closing the orbit by flaps of skin so
as to cover it completely. The deep part of the orbit Avas filled by
growth of connective tissue, its cavity was narrowed by hyper-
ostosis of the walls, and there was no possibility of Avearing an
artificial eye.
Still another possibility is that periostitis near the apex of the
orbit not causing purulent effusion may cause inflammation of the
sheath and substance of the optic nerve. As Ave have seen on page
614, we may have from this cause sudden and total blindness, ambly-
opia, scotomata, etc, while ophthalmoscopic signs may be slight or
wanting.
Treatment.—During the acute period, usual antiphlogistic mea-
sures, especially hot fomentations and poultices, Avill be employed
locally, and in adults iodide of potassium will be used: in syphilitic
cases, gr. xx., ter in die, or perhaps in larger amount, Avhile in
rheumatic cases, gr. a7., ter in die, may suffice, but salicylate of
sodium may be preferable, and proper anodynes must be given,
such as antipyrine, gr. x., or bromide and chloral. Should an
abscess threaten, my judgment is in faAor of an early incision,
always using a long, narrow knife, or bistoury, for a sufficiently
deep puncture. If a fistula has formed, one must be very prudent
in its exploration by a probe. Often the deep parts are readily ex-
cited to inflammation. The proper Avay is to secure a full external
opening by dilating it, at first with laminaria probes, and later by
small sponge-tents, until access is gained to the deeper parts of the
sinus. Syringing with a fine tube should be practised daily, so
long as any secretion is pent up, and should the parts become
callous, or indisposed to heal, stimulating fluids may be introduced,
but always under strict limitations of prudence as to possible over-
effect. One must also know when to stop interference, lest healing
be pre\rented rather than promoted. An attack of acute cellulitis
is not hard to awaken, and is liable to be disastrous.
If rough or dead bone is felt by the probe, its situation deep and
extent limited, we may simply keep the outlet patent, and use
Avarm antiseptic injections of carbolic acid, 1 to 100, or of aqua
chlorinat., 1 to 20, or acid, boracici, 1 to 25. In A7ery old cases in-
jections of iodine tincture properly diluted or of sulphate of zinc,
gr. x., vel xx., ad $ i., etc., are permissible. But if the diseased
bone be easily accessible and of considerable extent it is to be
gouged or scraped or pulled aAvaA/. We may often be let into
neighboring cavities—the frontal or ethmoid cells, etc. The gen-
723 DISEASES OF THE EYE.
eral condition of the patient, especially in young subjects, is to
be property cared for in administration of good food, ol. morrhuae,
iron, especially syr. ferri iodidi; giving mild mercurials, viz., biniod.
hydrarg., gr. fa to -fa ter in die, or corrosive sublimate, gr. fa to
fa, Avith small doses of iod. pot., the object being to bring the nutri-
- tion up to the state in which healthy tissue shall replace the dead
bone. Hereditary syphilis will not be forgotten.
The possibilities of serious complications involving life are not
to be overlooked in case the roof is invoh7ed, because of the near
vicinity of the brain. An instructive paper on caries and necrosis
of the walls of the orbit is by Knapp, Trans. Am. Oph. Soc, 1889,
p. 325.
Inflammation of the Oculo-Orbital Fascia. Tenonitis.
It is not intended to refine needlessly upon the varieties of in-
flammation in the orbit, but we meet cases sometimes, whose dis-
tinctive features justify us in designating the above tissue as the
seat of the lesion. For example, a girl, fourteen years of age, pre-
sented herself, in whose left eye was to be seen a yellowish bleb
over the insertion of the rectus externus. The conjunctiva at the
equator was moderately injected; the globe was slightly prominent,
its movements were a little uncomfortable; pressure upon it caused
pain. Such an attack had occurred twice within six weeks, and
disappeared each time in less than tAvo weeks. The girl seemed in
good health, and no syphilitic or strumous taint was apparent.
Such a case would seem like episcleritis, but the prominence of the
eye, and its tenderness on being pressed into the orbit, located the
disease farther back, and pointed out its seat to be in the ocular
part of the capsule of Tenon.
It happens that the symptoms of the above case may become a
little more pronounced, so that instead of a local and well-defined
bleb, chemosis may begin at and surround the Avhole equator, and
reach the cornea attended by no distinct symptoms of scleral, con-
junctival, or other disease of the front of the eye, but attended by
swelling of the upper lid, slight proptosis, and slight restraint of
motion. Such is the picture of well-marked tenonitis. Its recogni-
tion is practicable only when the effusion is serous; its origin is
usually rheumatic Treatment should be mild, in soothing lotions
and choice of suitable rheumatic remedies, according to the indica-
tions; alkalies, iodide of potassium, salicylate of sodium, etc. Mild
cases will get w7ell in a feAv days. Dr. Bull has recorded cases of
the disease following operations for strabismus, and I have men-
tioned one of the same kind. Other operators have seen the same.
THE ORBIT.
729
Cellulitis Orbit^e.—Phlegmon of the Orbit.
This appears under the form of subacute inflammatory oedema
and of phlegmon. It may be occasioned by a A7ariety of lesions,
both traumatic and idiopathic. The mildest form of oedematous
cellulitis is as follows: a delicate boy, of pale skin, about nine years
old, complained of dull pain about the right eye; there Avere no
signs of ocular inflammation. After two or three days the globe
began to adAance from the orbit, and then I saw him. There was a
little SAvelling, but no redness of the lid, no chemosis nor conjunc-
tival redness; the eye stood forward several millimetres, and turned
outward. There was difficulty in movement, and occasional
diplopia. Pressure on the globe, when firm, Avas unpleasant; no
hardness or tumor could be felt on pushing the finger between the
globe and the orbital margin. Vision was perfect, pupil and fundus
natural. The symptoms continued the same for several days and
the eye finally returned to its place. Such a mild attack of cellulitis
is most likely to occur, as I have found, in young and not robust
children. It is not dangerous, and needs only mild external appli-
cations, such as warm infusions of opium or of chamomile flowers,
or the liquor ammonii acetatis, 1 part to 5 of water.
With increased severity there may bs considerable displacement
of the globe, perhaps diverging strabismus, and there may be
episcleritis with possible chemosis and the termination of the pro^
cess be in resolution.
Phlegmonous inflammation of the orbit is either idiopathic or
symptomatic. It may be ushered in with a chill and rise of tern
perature. There will be pain, SAvelling, and duskiness of the lids,
especially of the upper, and the eye will advance. Chemosis of a
yelloAvish-red color, with conjunctival vascularity, will appear; the
e37e Avill move Avith difficulty, and in the height of a severe attack it
will be absolutely rigid; pressure on the globe may or may not
make it recede, and Avill cause deep pain; exploration by the finger
in the circumocular sulcus will find the tissues firm, tense, solid, and
painful, some parts being more tumid and tender than others. This
last symptom of resistant and painful infiltration of the orbital
tissue is the important feature. In bad cases the eyeball becomes
im7oh7ed by infiltration and opacity of the cornea, and perhaps even
to general suppuration. The optic nerve, the sclera, and the interior
textures may in turn participate, and the end be panophthalmitis.
The cases vary in severity and general features, but the above facts
are the chief symptoms. Such a lesion may be metastatic from
puerperal feArer, remote phlebitis, septicaemia, carbuncles, or typhus
fever; it is more frequently coincident with facial erysipelas, and
730
DISEASES OF THE EYE.
then is usually double, with disease of the neighboring bony walls,
or with acute inflammation of the lachrymal gland. It has been
observed as a complication of purulent meningitis; but in such
cases there is strong reason for regarding thrombosis of the cav-
ernous sinus or neighboring veins as the middle factor of the process.
It has been caused by thrombo-phlebitis after extraction of a
carious back tooth (Vossius, Pagenstecher); it has attended foetid
nasal catarrh. It may result from caries of the parietes. In all
cases of suppuration of the eyeball there is more or less orbital cel-
lulitis.
Without external signs of eye disease, there may be danger to
sight from optic neuritis, and this is to be especially watched for
after facial erysipelas. In these cases there is thrombosis of the
orbital vessels.
The extension of erysipelatous inflammation into the orbit is a
frequent and grave occurrence. Not only may it destroy sight by
optic neuritis, as I have seen and has been reported by Knapp,
Spaulding, and others, but I have seen the cornea ulcerate and sup-
purate from its exposure during exophthalmus. If the cornea is
not so severely damaged, it is liable to be hazy and render ophthal-
moscopic inspection difficult. The nerve is apt to be SAvollen, pale,
infiltrated, and its vessels extremely attenuated, in the worst cases;
while in milder types congestion may exist with swelling, and the
more usual phenomena of neuritis. There is naturally great risk
of meningitis.
Prognosis is serious, and turns on the age, health, and habits
of the patient, and on the cause of the trouble, and on the efficiency
of treatment. The great majority recover.
Fatal cases through meningitis and abscess of the brain are on
record. Griffithx reports a case fatal on the seventh day because
of thrombosis of the ophthalmic vein extending to the sinuses of
the dura mater and causing death by thrombus of the pulmonary
artery by way of the jugular veins.
Treatment will vary according to the period when the case is
seen, and with its cause and complications. For a case seen early,
cold or warm applications, as the feelings of the patient dictate,
and free use of leeches on the brow and temple—six or eight of them
—will be judicious. When the swelling rises high, and if explor-
ation beneath the rim of the orbit detects deep infiltration and
resistance, and if the swelling of the lid be of a hard and brawny
type and there be much pain, an incision should be made at the
point of greatest tension and tenderness Avith a straight, narrow
bistoury, close and parallel to the wall of the orbit and generally
1 Ophthalmic Review, vol. iii., 1884, p. 147.
THE ORBIT.
731
aboA7e the eye. The knife should enter for one-half inch or perhaps
much deeper, and while the point may not cut Avidely, the opening
through the skin and fascia must be one-fourth to three-fourths of
an inch in length. By this incision the tension of the oculo-orbital
fascia is relieA-ed, the vessels are unloaded, serum finds A7ent, and
the tissues are relaxed. It is not necessary to find pus, but in case
such a focus has formed, the knife must aim for it and go to any
depth to reach it. My convictions are strong in favor of an early
incision with sufficient external opening, as a means of arresting
the phlegmonous inflammation and the formation of pus. I quite
disagree with a tendency to long tarrying until pus begins to sIioav
at the surface, because meanAvhile graA7e mischief can befall the optic
nerve and likewise the cornea, as the result of tension, pressure, and
contiguous inflammatory action. The wound is to be kept open by a
tent of borated lint, and washed freely with warm borated or carbo-
lated Avater or solution of corrosive sublimate to promote bleeding
and subsequent discharge, and lotions of warm water or acetate of
ammonia must be continued ; or hot poultices of ground slippery-elm
bark or of spongio-piline wrung out of hot Avater, should be kept up.
The patient's general condition will modify the local treatment, be-
cause if laboring under a grave general disease, fever, pyasmia, or
meningitis, there will be some hesitation about inflicting pain or
incurring much loss of blood. The principles of general surgery
must be our guide. When, hoAvever, orbital cellulitis and blepharitis
complicate erysipelas of the face or head, incisions are to be made
early, when the skin assumes the tense and dusky hue and hard
feel of phlegmonous infiltration. The circulation being strangu-
lated by the effusions, deep incisions offer the best chance of pre-
venting the sloughing of tissue and injury to the eye. In these
cases there is less danger from undue bleeding, because the vessels
are choked by the infiltration.
It need not be said that in many of the complicated cases, the
general disease demands the chief attention, by stimulants, sup-
porting food at short inteiwals, quinine, mineral acids, etc.
It may happen that the eyeball passes into general suppuration
or suppurative keratitis. For the latter, and for early stages of
the former, warm fomentations are to be sedulously used. Para-
centesis of the cornea, or its free division, may be required. For
suppuration of the globe, Avhere it has become tense, and is giving
great pain, an incision may be made into its anterior half to evacu-
ate the vitreous, at least in part. In these cases enucleation may
be done, as I do not hesitate to do when its suppuration is primary
and the affection of the orbit secondary. There are, in literature,
a number of cases of fatal results following abscess of the orbit,
but, discrimination must be made as to those which are associated
732
DISEASES OF THE EYE.
with other and grave disorders. For remarks on these condi-
tions see page 545 et seq. An extensive abscess of the orbit, when
purely local, threatens risk by extension backward to the brain,
and it may also, in some cases which terminate in recovery, cause
so much contraction of the connective tissue as to interfere with
the motility of the eye; this has been seen to take place, especially
between the levator palpebrae and rectus superior. Such mis-
haps are, however, uncommon, and recovery usually occurs with en-
tire restoration of function. Impairment of sight is unfortunately
not so rare, consisting in lesions of the optic nerve and retina, in-
flammatory and atrophic, also in intraocular hemorrhages and in
detachment of the retina. Some cases of amaurosis or amblyopia
do not show any ophthalmoscopic lesions, and in them the cause
may lie in the stretching of the nerve, or in inflammation of the
nerve behind the globe (retro-bulbar neuritis), or by exudation in
its sheath. It is said, too, that by pressure, the axis of the eyeball
may be temporarily or perhaps permanently altered, giving rise to
hyperopia or to myopia according to the direction in which its
force is chiefly exerted. But even from visual dangers, most cases
are ultimately safely delivered.
Thrombosis of the Orbital Veins and of the Cavernous
Sinus.
Thrombosis of the veins occurs necessarily in phlegmonous in-
flammation of the orbit, and does not call for special notice. The
process may, however, extend to the cavernous sinus and thence to
other sinuses, causing cerebral symptoms of a recognized charac-
ter, according to the parts involved. When the lesion extends.
to sinuses on both sides of the skull, we may have obstruction to
the venous circulation in both orbits simultaneously, producing
bilateral exophthalmus, oedema of the lids, with severe brain-symp-
toms.
In case, as often happens, the venous thrombosis is of a septic
quality, either by local or general infection, then we have small ab-
scesses in the skin and tissues of the lids. There may also be ab-
scesses in the eye muscles, and the patient is likely to succumb by
- purulent meningitis, abscess of the brain, or pyaemia.
We also have thrombosis of the cavernous sinus in conse-
quence of various intracranial lesions and associated Avith throm-
bosis of other cerebral sinuses. For example, Wreden1 elaborately
gives the history of cases, one of otitis media purulenta, and an-
other of sarcoma in the superior nasal fossa Avhich among other
severe symptoms developed thrombosis of the cavernous sinus.
There will be all the signs of phlegmonous inflammation of the
1 Archives of Ophthalmology and Otology, vol. iv., 65, 1875; vol. v., 82, 1876.
THE ORBIT.
I OO
orbit, and the swelling will perhaps reach the cheek and nose; there
Avill be exophthalmus, perhaps of both globes, immobility of the
eye, mydriasis, paralysis of the 6th and 3d nerves, and great pain
from irritation of the ophthalmic branch of the 5th extending over
all its distribution. There will be great turgidity of the retinal
veins, amblyopia, perhaps total loss of sight, or papillitis. The
very severe symptoms which attend this condition, gravely jeop-
ardize life, although one of Wreden's cases recovered. Berlin dis-
cusses the subject at length1 and instances a case in a horse in
which by unskilful phlebotomy in the jugular vein thrombosis of
one caA7ernous sinus and other lesions took place which proved
fatal. The affection is more apt to be double than single. Some
spot of caries, or local phlebitis, or purulent meningitis may be the
starting point. It can also originate in caries outside the cranial
cavity such as removal of an epulis (Landesberg)2 which proved
fatal by this occurrence.
Tumors of the Orbit.
There is an enormous literature on this subject, and the ob-
scurity which attends an exact diagnosis of the nature, extent, and
relations of the disease, makes every case an interesting study.
In an examination, Ave attend first to the objective symptoms,
viz., the exophthalmus, its degree, and the direction in which the
eye is pushed; the mobility of the eye, whether limited in any
special direction, in all directions, or not at all; the appearance of
the globe, Avhether unduly Avascular or itself the seat of a tumor or
deformity. We examine the tumor as to its resistance, solidity,
elasticity, fixity, or mobility, fluctuation, pulsation, its smoothness,
or lobular or nodular character, and Avhether it move Avith the eye-
ball. We press the globe oack into the orbit and note Avhether this
gives pain, Iioav far it Avill recede, and Avhether, in retiring, the
tumor also retire or be pressed forward. We listen upon the globe
and over the temple by a stethoscope, for murmur or pulsation.
We note Avhether neighboring vessels about the forehead or lids be
enlarged, whether the pre-auricular or the ceiwical lymphatic glands
are hypertrophied. We inspect neighboring cavities, viz., the nos-
trils, the vault of the pharynx, the frontal and maxillary sinuses,
so far as they are Avithin the means of examination. In some rare
cases Ave explore the tumor with a hypodermic syringe. The sensi-
tiveness of the cornea and the fundus oculi are also examined.
The subjective examination Avill embrace the age, sex, present
1 Graefe and Saemisch, " Handbook," vol. vi., p. 540,
2 Centralblatt fiir Augenheilkunde, 18S3, p. 332.
734
DISEASES OF THE EYE.
and previous health, constitutional diseases, especially syphilis, any
hereditary tendency to cancer, or its possible existence in other
parts of the body; the mode in which the disease appeared, and
exactly at what point, if this can be located; its rate of progress;
whether the onset was somewhat sudden or gradual; whether
there has been pain, or occasional attacks of inflammation; Avhether
a tumor was noted before proptosis appeared, or vice versa. We
may also examine for diplopia, and sometimes w7e may learn that
hypermetropia or even myopia has been developed since the growth
began.
The upper lid often undergoes remarkable elongation as the
globe advances, while in other cases the lids are stretched apart
and cannot properly cover the eye; in the latter case the cornea
may become inflamed and opaque. The examination of the orbit
may be made by thrusting the little finger betAveen the globe and
the bony margin on all sides as deeply into the cavity as possible,
not heeding the considerable displacement of the globe, as it yields
to the pressure. The object is to elicit information as to the seat
of the tumor; whether it spring from the walls of the orbit, be
located A\rithin the cone of the recti muscles or be outside of them;
whether it be attached to the globe or to the optic nerve; whether
it enter the orbit from an adjacent ca\Tity.
Many of these questions must be left unanswered. But we can
often tell by the fixedness and hardness of a tumor that it is at-
tached with some firmness to the bony Avails; then it will be out-
side of the muscles. If the tumor be mobile, it will to a great ex-
tent be free in the orbit, although, perhaps, partly attached. If
at the same time the globe is displaced in some oblique or lat-
eral direction, the tumor is probably outside the muscles. If the
globe comes straight forAvard, its motions are rather restricted,
although natural, and the tumor seem fitted closely into the apex
of the orbit, and vision has been destroyed at an early period, which
ordinarily is not the case, there is reason to locate it Avithin the
cone of the muscles and it may be an outgrowth in or upon the
optic nerve. A tumor of the optic nerve, of Avhich several are re-
corded, will not at first, of necessity, destroy sight, but at an early
stage blindness will follow.
It has also been noted that sometimes the nerve is not pushed
out straight, but has an S-shaped curvature. Deformity in the
contour of the o'rbit, Avhich is rare, and stoppage of the nostrils,
are indications of a growth in the antrum, and it may be discov-
ered by examining the gums and the mouth, and the cheek. The
use of the rhinoscopic mirror behind the velum palati will aid us
in discovering encroachments from neighboring cavities. One may
also explore the roof of the pharynx with the finger per orem. The
THE ORBIT.
735
nostrils must be inspected by mirror and speculum, and be exam-
ined by a long probe or by a cotton holder. It is not infrequent to
find polypi, especially in the upper fossae.
The next question is as to the nature of the tumor, and with
this and the previous inquiry are bound up both the prognosis and
treatment of the disease. We must often speak with caution about
the nature of an orbital tumor. The factors to be weighed are its
rate and rapidity of growth; the age of the subject, and his previ-
ous history; the hardness, smoothness, nodular character, mobility,
compressibility, fixedness, apparent vascularity; the state of the
eyeball; and the existence of murmur or pulsation; the presence of
distended or varicose vessels. We can speak with some confidence
respecting osseous groAvths by their physical characters, the slow-
ness of growth and painlessness, and the Avay the globe is displaced.
Tumors rapid in development, especially in young subjects, attended,
too, Avith large circumorbital or palpebral veins, and Avhich may or
may not pulsate or have a murmur, are likely to consist largely of
blood-vessels, and may also be malignant. Tumors not very rapid
in growth, either smooth or nodular, more or less mobile, and not
painful, offer a wide field of speculation as to their character, as
between fibromata, lipomata, sarcomata, myxomata, melanomata,
etc. Cysts are sometimes easily made out by obscure elasticity,
partial attachment, ovoid and smooth shape, but when deep
and of long duration they are only recognized by being opened.
Another class of tumors easily diagnosticated are degenerations
of the lachrymal gland.
There are also found echinococci, cysticerci, congenital serous
cysts, and bloody cysts.
Angiomata and erectile tumors are very likely to be associated
with similar anomalies of the skin, but this is not necessarily the
case—they are usually congenital. Cavernous tumors not congeni-
tal, as well as those Avhich are, can be generally made out by ob-
serving that they greatly increase in size by hanging the head
downward and forAvard, so as to cause venous congestion. They
are apt to be contained inside the cone of the muscles. They do
not have pulsation or murmur (Berlin). The distinction between
pure angiomata and highly vascular malignant growths, depends
on the greater rapidity of groAvth of the latter, their greater firm-
ness, and that the lymphatic glands are liable to be enlarged at an
early stage and the eyeball itself to be implicated. But there will
often be great uncertainty at the outset of the disease.
Among the rare ocular tumors are enchondromata and cylin-
dromata, while epithelial cancer sometimes reaches from the outer
parts into the orbital cavity. For description of tumors of lach-
736
DISEASES OF THE EYE.
rymal gland and of antrum, etc., see C. S. Bull, N. Y. Medical
Record, Aug. 24th, 1889.
Very seldom are both orbits invaded, yet this happens some-
times with ossific growths, and I have seen both orbits occupied by
lymphomata in a case of Hodgkin's disease; similar growths exist-
ing in the neck and elsewhere.
The above remarks include what may be stated respecting diag-
nosis and symptoms. As to course and prognosis, it may be said
that some tumors rapidly increase. These are the malignant forms,
which will embrace various forms of sarcomata, the so-called medul -
lary cancer, and some melanotic growths. In these cases the eye
may be involved, and the tumor may extend beyond the orbit, and
possibly grow to an enormous size. Pictures of such cases are
found in various books (Sichel, Dalrymple, Wells, etc.). When it
has reached the external surface, the tumor becomes fungous,
bleeds, emits offensiAe secretion and odor, causes hectic fever, ema-
ciation, exhaustion, and death. It may also produce absorption of
the adjacent bony Avails, and the fatal result may take place by
invasion of the brain.
Fibrous, fatty, cystic, enchondromatous, and less malignant
tumors grow less rapidly, and give trouble by the displacement of
the eye and the injury to sight. Angiomatous and cavernous tu-
mors have been known in a few instances to disappear spontane-
ously. Bony tumors are ATery slow in growth, but may attain
great magnitude, as I have witnessed. Mackenzie depicts a skull,
of which both orbits are filled by a dendritic mass of osteoid hyper-
trophy. Osseous growths are not painful except by pressure upon
and disturbance of adjacent sensitive parts. In almost all cases of
orbital tumors the exophthalmus is sufficient reason for attempting
relief. The injury to sight comes by neuritis (choked disc), detach-
ment of retina, intraocular hemorrhages, etc., but in many cases
the sight remains good for an indefinite time.*
Tumors of the optic nerve have within late years been men-
tioned considerably in literature; they are, however, rare, and none
have come under my notice. Wolfheim ("Ianaug. Dissert.," Konigs-
ber--, 1887) has recorded sixty-one cases of which thirty-six were
sarcomata. The references below 2 may be useful. In this country
'Andrews, Med. Rec, Sept. 3d, 1887; Pooley, Am. Oph. Soc, 1890, p. 611.
2 Leber in Graefe u. Saemisch, Bd. v., p. 910, with references, 1874. Wille-
mer, Graefe's Archiv, Bd. xxv., Abth. i., S. 161, cases up to 1878. Vossius,
ibid., Bd. xxviii., Abth. i., S. 1882. Johnston, Arch, of Ophth., vol. xiv., p!
151, 1885. Straub, Graefe's Archiv, Bd. xxxii., Abth. i., S. 205, 1886. Schiess-
Gemuseus, ibid., Bd. xxxiv., Abth. iii., S. 226,1888. Vossius, " Grundriss der
Augenheilk.," 1888, S. 371. Michel, " DieKrankh. des AugesimKindesalter,"
1889, p. 525; Zehender, xxix., June, p. 208, 1891, v. Gamier. Wolfheim gives
61 cases of which 36 were sarcomata. Inaug. Dissert. KOnigsberg 1887.
THE ORBIT. ^37
cases have been reported by Liddell, 1863, Strawbridge, Knapp,
Gruening, Holmes, and Johnson. The cases of the last three
authors are found in the Archives of Ophthalmology and are in-
cluded in the references below. The case by Dr. Liddell has not
been known to general literature and is reported in a pamphlet
in my possession. A number of text-books refer to the subject but
give no new cases. The best summary of the matter is given by
Michel and Vossius.
Michel refers to tubercular growths in the optic papilla, and in the
orbital portion of the nerve, and in his "Lehrbuch der Augen-
heilkunde/' S. 642, 1884, quotes a case which he had seen of a tumor
upon the intra-cranial portion of the nerve and the chiasm, in a
man Avho had elephantiasis of one lower limb.
In by far the largest number of cases the orbital part of the
nerve is the seat of the growth, which Araries from a hazelnut to a
goose egg in size. Sometimes the nerve goes through the tumor
and is spread out in it, at other times it is found upon one side of
it. The tumor grows either from the dural sheath or from the pial
sheath, and the stem of the nerve. From the dural sheath groAv
endotheliomata; from the stem, including the pial sheath and its
trabeculae, occur the greater number, which are various forms of
sarcomata, \\z., fibro- and myxo-sarcoma, etc. Cystoid degenera- ,
tion is frequent in portions of the tumors. These groAvths occur in
the early period of life, i.e., up to puberty in about four-fifths of
the cases, and nearly half of them before the age of seven years
(Willemer).
Symptoms \avy somewhat acccording to the size and form and
locality of the growth. As a rule the progress is slow (in one case
eighteen years) painless, and blindness comes very early. (The case
of Schiess was in the last respect very exceptional.) Papillitis or
atrophy of the nerve appears early. Exophthalmus takes place
either straight forward, or a little doAvnward and outward. Mo-
bility is little interfered with unless the growth be large. Often
the tumor may be grasped between the fingers and recognized to
lie within the cone of the recti muscles. It is sometimes elastic and
sometimes firm. Sometimes a diagnosis of the neural character of
the tumor is possible, but if large this cannot be expected.
Treatment consists in removal, which Avill usually include all
the contents of the orbit. Strawbridge, Knapp, Gruening, and
Schiess succeeded in preserving the globe while enucleating the
tumor. In the cases of Strawbridge and Knapp the eye afterward
suppurated.
Relapse of the groAvth has been noted only once, but the period
of observation has usually not extended beyond one or two years.
In five of the cases reported by Willemer death occurred as the
47
738
DISEASES OF THE EYE.
effect of the operation. In the above cases extension of intraocular
tumors, such as choroidal sarcomata and gliomata, along the optic
nerve are not included, neither are orbital tumors which may en-
Arelop the optic nerve.
A resume- of the neglected case of Liddell is as follows: The patient was a
young Avoman «t. 20, who after having had mumps had swelling of the eye-
lids of the left eye which lasted tAvo or three months. About Ave months later,
i.e., in the spring of 1851, sight in this eye grew dim, and six months later ex-
ophthalmus began and perception of light was soon lost. In twenty months
longer exophthalmus was so great that the lids did not cover the globe, pupil
widely dilated, and eye perfectly movable. In June, 1853, inflammation began,
ending in atrophy of the eyeball. In 1858, when seen by Dr. Liddell, the
tumor hung down almost to the level of the nostril, was tense and elastic.
Pronouncing the tumor benignant, extirpation was performed and in doing
it two cysts were ruptured: the orbit was wholly evacuated. The capsule of
the tumor was composed of the expanded and thickened sheath of the optic
nerve, Avhich entered at the posterior end and reappeared of unusual size a
quarter of an inch from the globe. The tumor was as large or larger than a
goose egg. On section it appeared Jo consist of a laminate tissue (fibrous)
Avhose color was of a reddish yellow. The ruptured cysts heretofore men-
tioned were lined by a smooth membrane. The patient was in good health
fiA7e years later, with no return of the growth. Two illustrations accompany
the paper.
Treatment—The only proper proceeding is operative removal.
Certain modifying considerations are to be kept in mind. Cysts
which extend too deeply into the orbit to be perfectly extirpated,
or Avhich communicate with adjacent cavities, must, after partial
removal, be treated by injections of stimulating fluids. For A7ascu-
lar or erectile tumors in very young subjects (infants), the use of
red-hot needles, or of electrolysis, to coagulate the blood, is expedi-
ent. The operation may be repeated once in tAvo or more weeks,
according to the degree of reaction and to the rapidity of growth.
Such tumors cannot be safely treated by irritating injections. In
adults they may be attacked b}7 excision, aided, if needful, by the
actual cautery in some convenient form, viz., hot-iron, electric cau-
tery or thermo-cautery. Often they are inclosed by a capsule of
fibrous tissue Avhich much facilitates the proceeding. In electroly-
sis it is better to have both needles of platinum, the positive pole to
Avhich oxygen goes must always be of platinum, it is in this that
the greater amount of albuminous coagulation takes place. For
small growths the negative pole may be applied by a sponge to the
temple, but when the tumor is large both needles must enter the
tumor, and consequently must be about two or three inches long.
It is best to coat the needles for a certain distance with collodion
where it is desired not to injure the skin. The positive needle being
first entered may remain in situ Avhile the negative needle after
being held in position a few minutes at a little distance, may be
THE ORBIT. 739
entered in succession at different points around the positive as a
centre, until a sufficient effect is attained. Reaction is sometimes
considerable and it is prudent to not attempt much at a first sitting.
An illustration of an unusually large angiomatous tumor is given
in Fig. 259. The child was a patient of Dr. C. S. Bull, by whose kind
permission the case is presented. Several applications of electroly-
sis Avere made, but the success was not complete. The tumor was
within as well as Avithout the orbit.
For osteoid growths the best means of removal is by the chisel
and mallet, attacking them at the base by very light and numerous
blows until they loosen (Knapp). But Berlin sums up his remarks
Fig. -.59.
on such tumors by some pregnant observations as to what is justi-
fiable according to the situation of the growth. He has collected
32 cases Avhich were operated on: of these, 9 had meningitis; 8
died; of the Avhole number, 16 had bonj7 growths in the roof of the
orbit, and of these 6 died, a fatality of thirty-eight per cent. This
sIioavs in a most startling Avay, how dangerous is interference in
this particular category of cases. • It certainly justifies absolute
refusal to operate, unless there be urgent indications and a full
presentation to the patient of the risks he incurs. The reasons for
operating can only be pain, the safety of the eye, and conspicuous
deformity. Osteoid tumors in other parts of the orbit may be re-
♦ moved with success, and if adjacent cavities are opened, no great
harm is done. A small gouge with a strong Avooden handle is a
good instrument, or one may prefer a chisel and mallet. If the
740 DISEASES OF THE EYE.
latter be used, the strokes must be gentle and the proceeding slow-.
Knapp reports a case of fatal ending, in a case of osteoma upon the
os planum whose removal was easy and recovery perfect as re-
garded the Avound. Meningitis took place without apparent cause,
but was attributed to irritation extending from the ethmoid cells
which were filled with polypoid masses.
For tumors whose relations, size, and probable character render
them fit for excision, the question arises: Can they be extirpated
without sacrificing the globe ? If unadherent to the eyeball, even
if they include the optic nerve, this is generally feasible. In 1866,
I excised a fibrous tumor of the orbit without removing the globe,
but sight was lost by suppuration of the cornea, consequent on ex-
trusion of the globe by inflammatory infiltration of the orbital
tissues and exposure of the cornea. The tumor was above the
globe, and my incision was made through the superior cul-de-sac of
the. conjunctiva, which resulted in ptosis, because the kevator pal-
pebrae had to be destroyed to reach the tumor. My purpose in
choosing this route was to spare the levator, but the seat of the
tumor defeated my design. The proper mode of approach to such
a tumor Avould be through the upper lid at the margin of the orbit-
In the rare cases of tumor of the optic nerve, the probability
of extension along the nerve into the brain makes early operation
important. Being situated within the space surrounded by the
recti muscles, it is manifestly proper to attack the tumor through
a Avound in the conjunctiva.
In removing a tumor, first decide in what way it will be most
accessible. If it be decided to go through the conjunctiA~a, choose
the side which offers the nearest approach to the mass, pass be-
tween the recti muscles by a Avound as large as can be made by
drawing them asunder, or detach a tendon, if needful, and tie to it
a thread, so that it may afterward be recognized. Use a pair of
narrow and strong scissors, curved on the flat, with rounded points,
to make the first opening, then with shut blades tear aAvay the
connective tissue down to the tumor, and push the tissues apart to
expose the mass. Attempt to bring it forward by a strabismus-
hook, or by catching it Avith a sharp hook if it be tough enough to
bear traction, and carefully cut away its surrounding connections
by small clips Avith the scissors. Progress must be slow, and tissues
must be torn rather than cut, as far as may be possible. If the
tumor be upon the optic nerve, push a strabismus-hook behind it to
the apex of the orbit, and when this has caught the nerve, run the
scissors alongside of it and cut the nerve beyond the hook; then
this hook, or a sharp hook planted into the tumor, will pull it round
to the front, reversing the globe and making its separation from
the eye very easy. In case entire or sufficient removal cannot be:
THE ORBIT.
741
accomplished within the space thus available, the globe may have
to be sacrificed. Before the operation, this possible contingency
must be stated to the patient, and his consent obtained. Small
pieces of ice pressed into the wound, or injections of water as hot as
the hand can bear, or pressure by the finger, will control the bleed-
ing, and the wound must not be closed until bleeding has stopped.
But the method above described is suitable for a small and ex-
ceptional number of cases. In the great majority the wound will
be made through the skin. It should be parallel to the margin of
the orbit, over the most prominent point of the tumor, and as large
as can be of any use. Rigorous antisepsis must be observed. After
going through the skin, the deeper dissection is to be done as al-
ready described. If possible the globe should be spared. In case
the tumor be found to penetrate adjacent cavities, it may be im-
possible to follow it and accomplish complete extirpation. The
surgeon must decide such questions according to his own judgment
or the requirements of the case. By such a method of proceeding,
it is surprising Iioav successfully a tumor may be dug out, both as
regards the loss of blood and immunity of healthy parts. All
bleeding must be arrested before the Avound is closed; it must be
cleaned with solution of corrosiA-e sublimate, 1 to 3,000, with a bulb
syringe. Close the wound by fine silk sutures, dress the surface
with a rag smeared with simple aseptic cerate, put over this a mass
of absorbent cotton soaked Avith sublimate solution, and retain all
by a flannel bandage which shall exert firm pressure. Generally an
anodyne will be needed. The wound will not be opened, if pain and
reaction be moderate, until after forty-eight hours.
Complete evacuation of the orbit (" exenteration ") is called for
when the eyeball is implicated in the growth, has already been de-
stroyed, or Avhen the tumor cannot otherwise be removed. It may
be that only an ordinary enucleation may be necessary, and this
be folloAAred by excision of the tumor and nothing more. But other
cases arise in which the orbit must be emptied of all its contents.
In doing this, the lids are split asunder at the outer angle; the
coverings of the mass are to be picked up and cleaned off until its
surface is fully in view; then, Avith a blunt instrument (the scissors
before mentioned, with shut blades, are my usual resort), insinuate
betAveen the tumor and the wall of the orbit at the most convenient
point, and tear and push away the parts, keeping in contact Avith
the bone until a way is made to the apex of the orbit. I strongly
deprecate the use of knives in such an operation. The principal
hemorrhage will occur at the apex of the orbit, and can be best
arrested by prolonged pressure Avith the tip of the finger. When
such an operation has been done for malignant disease, the walls
of the orbit are sometimes Avashed Avith solution of chloride of zinc,
;42
DISEASES OF THE EYE.
or smeared with a paste of this substance. If freely applied, a
scale of bone may, in consequence, be exfoliated, and serious risk is
incurred of meningitis; but, done not too vigorously, greater se-
curity against recurrence of the disease
is gained, and without dangerous risk.
The cavity should be copiously washed
out with sublimate solution, packed with
absorbent cotton similarly soaked, and the
whole kept wet under the flannel band-
age. The packing may be left for four
to seven days without removal, until in-
cipient suppuration requires it. Healing
will ensue in four to eight weeks.
Still more serious measures may some-
times be called for when not only must
the contents of the orbit be removed but
the eyelids also be sacrificed. Such a ne-
cessity has occurred to me several times,
and a recent instance is a patient who
furnished the adjoining illustration, Fig.
260. The picture there presented was his appearance after a
flap operation in 1880, rendered necessary by the removal of epi-
thelial cancer of the inner portion of both lids and of parts about
the caruncle. There was no return of the disease for five years,
when it reappeared in the flap which had been taken from the
forehead. It gradually extended until all the lower lid and some
of the adjacent cheek, the lower half of the upper lid and the inner
angle of the eye were implicated. The cornea was destroyed
and the globe adhered to the upper lid. In October, 1889, I re-
moved all the diseased parts, including the globe, found the dis-
ease had penetrated the ethmoid cells, which I had to open and
scrape as clean as was possible, and then was obliged to shut up
the orbit by a flap of skin brought up from the cheek which met
the remnant of the upper lid. The available tissue was scanty and
I was obliged to cut again upon the median line of the forehead,
dissect up the skin on its left side, detaching the brow from the
upper margin of the orbit, and by pushing down the integument, a
half inch was gained that sufficed to reach the flap from the cheek.
The incision shows clearly upon the picture, which was taken at the
end of the third week after the operation, and it was ten inches in
length, starting from the middle of the forehead and going to the
ear in a curved line. After doing the operation two long strips of
gauze soaked in sol. corros. sublim., 1 to 1,000, were packed into the
orbit and brought doAvn to the loAvest point of the wound in the
cheek. On the fourth day they were removed and very little sup-
Fig. 260.
THE ORBIT.
743
puration in the cavity took place. The patient is comfortable if
not comely (see Fig. 260). He remains well in October, 1803
Recurrent groAvths not infrequently demand attention, especially
after excision of sarcomata, and the globe will usually have already
been sacrificed. If these be not too large, say not bigger than a
moderate-sized hen's egg, the mode of removal by a blunt instru-
ment, scraping the Avails of the orbit and shelling out the mass, is
surprisingly easy and comparatively bloodless in many cases. It
may be needful to remove a tumor as many as four times.
I have ne\*er seen cases Avhich required resort to caustics to de-
stroy a groAvth, and such occasions must be rare.
It is not unusual after successful operations, for the globe to
become displaced forward, and so remain for some time. It may
also happen that the muscles undergo disturbance and cause diplo-
pia ; or ptosis may follow from greater or less injury to the levator.
Such disturbances will usually, in time, correct themseh'es. But a
more serious matter is the liability of the cornea to become inflamed,
both by possibh exposure as swelling comes on, or as it may he
bathed in secretions. Frequent Avashing Avith boracic-acid solution,
4 to 100, Avith sol. Labarraque, 1 to 10, the application of A-aseline,.
and the closure of lids by rubber plaster, are the best preventives.
Moreover, the sight is also endangered by the manipulations at the
back of the globe and about the optic nerve. It is, therefore, not
to be thought strange if the globe be saved, and sight be partially
or totally lost. At the same time preservation of the form of the
eye and its natural appearance is worthy of strenuous endeavor.
Prognosis as to recurrence and as to life are questions which
must be decided by the facts of each particular case. In simple
vascular groAvths, cysts, fibromata, osteoid growths and epithelial
tumors, the prognosis Avill be either absolutely or relative]}7 good.
Nettleship reports recurrence in loco of a tumor fourteen years
after its first removal. Sarcomata may not recur in loco, b^t in
the liver or some remote organ and within one or fiA7e years. T'-e
prognosis for glioma and for highly vascular cancerous turncri ■•-;
bad both as to recurrence and danger to life. The general sta'«-i
of the patient, the seA7erit}7 of the operation and the ascertained
" malignity " of the growth are the factors to be considered. After
exenteration it is impossible to Avear an artificial eye, the lids re-
tract and a screen is to be Avorn to hide the deformity.
Pulsating Exophthalmus.
Under this term are included affections of diverse nature, whose
common features are protrusion of the globe and pulsation, which
may be felt by pressure, and whose sound may be recognized by
744
DISEASES OF THE EYE.
the ear. The true pathological condition is various and may be
purely A-ascular or may consist in highly vascular tumors Avhich
may or may not be malignant. It is difficult in many cases, ac-
curately to distinguish between vascular malignant growths, or
angiomata, arterio-venous aneurisms, varicose dilatation of veins,
true arterial aneurisms, and thrombosis of the cavernous and ad-
jacent sinuses. The literature of the subject is large; exact knowl-
edge about it is small. In 1869 I tabulated the cases knoAvn to that
date, where the common carotid had been tied for pulsating exoph-
thalmus. Since then the subject has been summed up by Rivington,
Harlan, Nieden, Schlaefke, and most fully by Sattler in Graefe-
Saemisch, vol. VI., 744-948.
Schlaefke catalogued ninety-three cases of pulsating exophthal-
mus (Archiv fiir Oph., XXV., iv., pp. 112-162), and Sattler dis-
cusses 106. To these may be added 11 more collected since Sattler's
paper by Kohler, making the total 117 up to 1886. It is nevertheless
a very rare disease. The cases may be traumatic or spontaneous.
Out of Sattler's cases the traumatic were 59, idopathic, 32; Kohler's
11 cases were all traumatic, showing out of 102 cases about 70$ to be
traumatic. Of the spontaneous cases 6 occurred during pregnancy.
Following injuries of the skull we have a variety of symptoms. The
cases are usually rapid in development, vary in degree of protrusion,
are liable to attacks of transient inflammation of the conjunctiva,
and, in some cases show tokens of retarded circulation in chemosis or
in sAvollen \7essels. Hemorrhage from the nose is rather frequent
and maj7 be dangerous. It has also come from the conjunctiA7a.
Seldom is there pain; the patient is often conscious of a pulsating
bruit, and may have dizziness. Sometimes an enlarged vessel will
be found, projecting at the upper and inner or at the lower and
inner angle of the orbit, and it will pulsate; vision generally is un-
affected; motions of the eye are natural in extent and co-ordination;
there may be diplopia; in the fundus the vessels are enlarged, and
sometimes pulsate. In some cases there are dilated vessels of the
skin in the neighborhood of the lids. (Aneurismal dilatation of the
capillaries and small vessels of the adjacent skin, which may extend
into the orbit and cause protrusion of the eye and pulsation, are
excluded from consideration.) Pressure on the common carotid
stops the pulsation and also the bruit, both to the patient and
to the examiner, Avhile the globe retires a little into the orbit.
The eye can be pressed into the orbit a certain distance, and the
firmer the pressure the harder the pulsation. Stooping forward
increases the protrusion and the pulsation, and is unpleasant to the
patient, because of the sensation of weight. A few cases of spon-
taneous recoArery are recorded.
Formerly, all cases having most or some of the above symptoms,
THE ORBIT.
745
Avere styled aneurisms of the ophthalmic artery. Guthrie published
a case of double exophthalmus, in 1803, in Avhich, at the autopsy,
he declared that there Avas an aneurism of each ophthalmic artery.
Only one other similar has been seen, and that was in a cadaver
(Carron du Villard *): one ophthalmic artery having an aneurism
in the orbit.
On comparing the various catalogues of cases w7e find eighteen
autopsies, which have been reported since Sattler's paper. Of
these, two were cases of malignant tumor, in five cases there was
no aneurism and the arteries were not diseased. There remain
11 cases of aneurism of the following varieties: In 2 cases the
ophthalmic in the orbit; in 1 case the ophthalmic before entering
the orbit; in 4 traumatic cases there was aneurism of the carotid
in the cavernous sinus; in 3 cases carotid aneurism had ruptured
in the sinus, in 1 case there was spontaneous enlargement of the
carotid in the sinus. That is out of 11 cases of aneurism 8 were of
the carotid in the sinus. Taking the 5 cases which Avere not aneur-
ismal, in one it is thought that a small fissure of the carotid in the
sinus must have been overlooked, because the A7ena ophthalmica
was greatly enlarged and no other dilated vessels were present.
In another of this group was Bauman's in which there Avas nothing
but inflammation and distention of the cavernous, circular, and
transverse sinuses, and phlebitis of the ophthalmic vein. Somewhat
similar in character were the three remaining cases of the group.
Diagnosis.—The essential point in diagnosis is not so much to
distinguish where a possible aneurism may be located, whether in
the orbit or at the sinus, but whether the pulsating exophthalmus
depends on a tumor, benign or malignant, or on an aneurism, or on
inflammation of the sinuses. We may not attain certainty perhaps
in this attempt, but our treatment and prognosis will be controlled
in great measure by our belief in this regard.
As to vascular tumors, they are more resisting to pressure than
aneurismal swellings. Their deviation from the axis of the orbit,
in some lateral situation, Avill be important evidence of tumor; there
may be some other discoverable growth in the \7icinity: the de-
velopment has been rather slow with a tumor, and there has been
no serious injur}7; of course a true spontaneous aneurism of the
ophthalmic artery or of the carotid in the skull would have the
same features, but the further history would soon display different
behavior. A malignant tumor would show hemorrhages, rapid
growth, metastases, cachexia, and early death.
As to pulsating angiomata, they are less painful than malignant
1 Berlin, klinische Wochenschrift, xxiii., S. 550, referred to in Jahresbe-
richt ilber Ophthal., xvii. fiir 1886.
746 DISEASES OF THE EYE.
growths, their expansibility and the probable concurrence of simi-
lar groAvths in adjacent parts will afford helpful suggestions.
With aneurismal swellings, or such as for a therapeutic purpose
may be classed with them, the following points are to be noted:
Rupture of the carotid in the sinus cavernosus is the most common
lesion, and it is generally from injury. There is exophthalmus, pul-
sation, bruit, often audible to the patient. At the upper and inner
angle of the orbit is apt to be a small soft tumor, there may be
others about the opening of the orbit, the frontal veins may pulsate.
Pressure on the common carotid checks pulsation. Often a vari-
cose vessel with thick walls appears at the lower or upper angle of
the median side of the orbit and communicates with facial vessels.
Sometimes there is paralysis of ocular nerves, the sixth or the third,
this designates the interference in the sinus. In the eye one may
find distended retinal veins, atrophy of the nerve and Avith sudden
onset there may be severe neuralgia of the ophthalmic branch of
the fifth, and there is always pain. Vision may be unaffected, yet
various ocular conditions are possible.
For further information reference may be made to authorities
quoted, and especially, regarding the discrimination of varicose en-
largements of the ophthalmic vein and its branches, and of throm-
bosis of the cavernous and other cerebral sinuses, to Sattler, 1. c, p.
875 and p. 912.
An illustration of what was probably phlebitis of the ophthal-
mic veins coming from the cerebral sinuses is the following case,
which occurred in my observation:
M. M., aged twenty-two, native of Ireland, single, was attacked with an
illness four years ago, which kept her in bed for five weeks. She had fever,
great pain in the head, nausea and vomiting; constant noise in the left ear,
with some deafness, came on during the last two weeks. As she Avas recov-
ering, she found on waking one morning from sleep, that the left eye was
swollen, red, and protuberant. There was no pain nor loss of sight. The
exophthalmus soon attained its maximum and the eye seldom gave her
trouble. She had a few mild attacks of inflammation in it. It did not annoy
her, except that if she stooped it would come farther out and feel very heavy.
For nine months previous to the eye-trouble her menstruation had been very
scanty. When on her way from Ireland to this country she stopped at Lim-
erick, and there an attack of inflammation began, which continued until her
arriA7al. She came to the New York Eye and Ear Infirmary on May 9th, 1881.
The left eye projected half an inch beyond the other. There is some chemosis
and anterior ciliary injection. Media clear, pupil normal; nothing wrong
in the fundus, except that the veins on the papilla are enlarged; V = f£.
Below the eye, along the border of the orbit, is a pulsating swelling; the
angular artery is much enlarged, pressure on the eye makes it recede into
the orbit. By auscultation, no thrill is heard, but a low pulsating murmur.
Pressure on the common carotid stops pulsation in the vessel below the globe.
This vessel comes from the inner side of the orbit, and pushes out under the
skin of the lower lid like a large varicose trunk.
THE ORBIT.
T4T
On May 18th, patient etherized; the angular artery tied, and the protrud-
ing vessel exposed, and tied at the inner and lower side of the orbit; it was
then cautiously dissected up and traced into the cavity of the orbit, until it
reached the groove for the infra-orbital nerve, where it dipped down. A
ligature Avas put about this end and the A'essel excised. It proved to be a
vein, and was larger than a crow's quill. No severe reaction occurred. In
eighteen days both ligatures came away. In fifty days the patient was dis-
charged. Pulsation had ceased, and the eye had retired one-fourth of an
inch.
November 18th, 1881. The eye back to its proper place. The optic nerve
normal; A7essels of corect size; V = §§.
Prognosis.—Very few cases recoA7e** spontaneously. Sight may
or may not be preserved. A fatal issue in idiopathic cases comes
from lesion of other arteries, by apoplexy or other brain lesions;
in traumatic cases by profuse bleedings from the cavernous sinus,
most frequently. With tumors the prognosis is what their nature
indicates.
Treatment.—Sufficient time should be allowed for deA7elopment
of symptoms to enable one to form a fair judgment of the probable
nature of the case. For a vascular, and perhaps for a cancerous
tumor, if any operation were proper, it Avould be excision, while for
a lesion of blood-vessels an attempt at such a proceeding would be
most likely attended by dangerous hemorrhage and disastrous
results. For vascular tumors electrolysis is not to be forgotten.
Pressure on the globe is unavailable and ineffective; injections into
the orbit of astringent fluids have been practised successfully, but
doubtless these Avere cases of vascular tumor; injections of iodine
have been made, but with fatal results. Pressure on the carotid by
the fingers, or an instrument, has in some cases giATen happy results.
Sattler says, I.e., p. 926, that for idiopathic cases compression of
the artery for a short time every day may succeed if kept up for
Aveeks or months—for traumatic cases it must be unremittingly
continued for three to six hours daily until all pulsation ceases.
Out of 29 cases treated by compression, Sattler admits only 4 of
complete and permanent cure, and 5 were improved. Nieden,1
however, out of 12 cases thus treated reports 5 completely cured,
and 7 benefited.
Ligature of the common carotid in the neck is the remedy most
to be relied on. Sattler cites 63 cases, to these add 3 of Kohler's and
we have 66 operations upon 61 patients; and in 17 there was no good
effect or it Avas not permanent; in 8 death folloAved; in 41 the result
was successful. Some among these Avere cases of tumor which re-
duced the fair proportion of successes. The good result Avas secured
in from three to six Aveeks.
In 11 cases relapse folloAved, and for other particulars see Sattler.
' Archiv f. Augenheilk., viii., 127, 1879.
74S
DISEASES OF THE EYE.
Enophthalmus Traumaticus.
A rare but very interesting lesion is met with, for knowledge of
which I am indebted to Dr. Schapringer, who kindly permitted me
to see the case and published an account of it in the New Yorker
Medicinische Monatschrift, June, 1890. Only a few are recorded
in literature, and I quote the references from Dr. S.'s paper. The
case is briefly as folloAvs: A girl seA7en years old was struck by a
teacher in school and fell down, striking the right temple on an iron
grating. She was made dizzy but not unconscious—no bleeding
from nose or ear. Tavo hours later when she went home her mother
noticed the drooping of the right upper lid and sinking of the
globe. About six hours after the injury she was seen by Dr.
S., A\dio describes the folloAving symptoms: At the temporal ex-
tremity of the right broAV a black and blue spot the size of a half-
dollar; the upper lid which is not swollen falls so as to cover half
the pupil; in looking up the lid folloAvs the eyeball normally; the
globe has receded into the orbit about 2 mm. as nearly as could be
estimated—the curve of the cornea not altered—the pupil behaves
normally and is not contracted—tension of the globe decidedly
lessened—blood-vessels of the retina, both arteries and veins, some-
what enlarged. Movements of ej7e perfect in all directions; no
diplopia. No redness of skin of the right half of face or ear and no
sweating. Sensibility normal on both sides, no headache. No
tenderness Avhen globe is pressed into the orbit. Vision normal.
The case evidently is to be explained by a lesion of fibres of the
sympathetic nerve. But what fibres ? Evidently not those in the
neck, which cause contraction of the pupil and redness and sweat-
ing of the skin of the face on the same side (Horner's observations,
p. 436), added to ptosis. Neither is the twig of the oculo-motorius
to the levator palpebrae at fault, because the lid and the globe
would in that case not act consensually in looking upward. The
ptosis, the enophthalmus, and the hypotony are accounted for by
lesion of the fibres of the sympathetic which supply the orbital
muscle of Miiller and originate from the cavernous plexus; but the
escape of the twig to the dilator pupillae which forms one root of
the ciliary ganglion shows that the injury is local in the orbit,and
not severe enough to cause damage to it; while other twigs less
capable of resistance have been rendered paretic by intra-orbital
hemorrhage. After three days the symptoms disappeared. No
account is given of the late appearance of subconjunctival ecchy-
mosis which is apt to shoAV itself from intra-orbital hemorrhage.
Some undue secretion from the right nostril and swelling of its
turbinated bone were noticed.
Other cases where the amount of injury was much more severe
THE ORBIT. 749
are quoted ; but the enophthalmus Avas in them attributed to
cicatrization and atrophy of the orbital fat and connective tissue.
See references on p. 469 for lesions of cervical sympathetic.
See A. Nieden, Zehender, Klin. Monatsblatter, etc., 1881, p. 72.
C. Gessner, Archiv fur Augenheil. (Graefe), Bd. xv., iii.; Arch, of Ophthal-
mology, xviii., p. 269, 1889.
Distention of Adjacent Cavities.
We must allude to affections of adjacent cavities which cause
encroachment on the orbit, and chiefly the frontal sinuses, the
ethmoid cells and the antrum of Highmore.
Distention of the frontal sinus by mucus or pus (empyema)
is a chronic affection and can generally be easily recognized by the
Fig. 261
site oi the swelling, which is above and farther back than the lach-
rymal sac, and has resisting walls. The condition may be consecu-
tive to seA7ere nasal catarrh, syphilis, or periostitis within its cavity.
Polypus has been found in the caA7ity, and also small exostoses.
It is pertinent to call attention to the variable size and extent
of this cavity. In young subjects it has no existence, but becomes
750
DISEASES OF THE EYE.
of notable size after thirty years of age, and beyond that period of
life it may present the most remarkable variations in extent. Mac-
kenzie has written the best chapter on this whole subject (see "Dis-
eases of the Eye," pp. 93-121, Am. ed., 1855), in which he collects
cases from the older writers. I haA7e seen a few. The locality of
the swelling will generally suggest its nature, and the wall of the
sinus may be carefully opened by a strong knife. Afterward, long
treatment by antiseptic and astringent injections will be required.
It sometimes happens that a spontaneous opening takes place by
absorption of the wall and then the appearances are those of a cyst.
Fig. 261 shows a case recently under my care where the abscess
opened spontaneously and its outlet can be seen above the tendo
oculi of the right eye. Care must be taken not to mistake such a
condition for an abscess of the lachrymal sac (see page 295).
It is also possible to mistake such a retention tumor for an ex-
ostosis because it may sometimes have a thick bonj7 mass Avith
irregular surface on its anterior wall. The finger must be forced
deeply to disclose the error by reaching to the less resisting deep
part. Great exophthalmus is sometimes produced and consequent
diplopia. Sometimes there is profuse and perhaps puriform dis-
charge from the corresponding nostril.
I have once had to sacrifice the pulley of the trochlearis muscle
in operating on one of these tumors. The diplopia in a vertical
sense Avas at first very annoying, but gradually diminished.
After operating, the secretions of the sac for a time escaped in
front, but subsequently found their Avay backward through the
middle nasal fossa into the posterior nares.
This case resembles that quoted below from Dr. Knapp.
It is necessary in all these cases to examine the nasal cavities
and sometimes the cause will be found In hypertrophy of the middle
turbinated bone.
Distention of the ethmoid cells of a similar kind is described by
Dr. Knapp (see "Report of Fifth International Oph. Congress,"
1877, p. 55). The patient Avas a girl, fourteen years of age, who had
a tumor at the inner and upper corner of the orbit, resembling in all
respects an exostosis. The surface of the bone was exposed by a
^ free incision, and as a chisel was applied to its base for its removal
its Avails promptly gave way and disclosed a cavity filled with
stringy mucus. The opening was freely enlarged, the contents
were fully evacuated, and it was found that some of the fluid used
in syringing escaped from the nostril. In about a year the case
was cured.
Tumors in the antrum press on the floor of the orbit, perhaps
perforate it, and may displace the eye, and I have met with a case
THE ORBIT.
T51
in which a fibro-plastic tumor came up from the spheno-maxillary
fissure and pressed the globe forward.
There are cases on record of congenital malformation, chiefly in
the neighborhood of the lachrymal bone, and often in both orbits,
by which the brain comes into direct relation Avith the orbit; its
cerebro-spinal fluid pushes doAvn the dura mater as a cyst, through
an aperture in the bony Avails.
Hemorrhage into the Orbit.
With very few exceptions, this results from injury, either by
falls, blows, or penetration of a foreign body. The symptoms A7ary
according to the amount effused. If large, there Avill be propulsion
of the globe and ecchymosis of the lids and of the ocular conjunc-
tiva. If the quantity be small, the eyeball will not adA'ance, while
the lids and conjunctiva Avill be discolored. Finally, the distinctive
criterion of orbital hemorrhage of small quantity, is a tardy ap-
pearance of ecchymosis creeping down under the ocular conjunctiATa
and advancing toward the cornea. In some cases the lid alone is
the seat of discoloration. Spontaneous cases are so very feAv, and
their etiology so manifest, viz., scorbutus, Aiolent coughing, etc.,
that Ave may confine ourselves entirely to orbital hemorrhage from
injur}7. It has been pointed out by many distinguished surgeons,
and is classical in literature, that this symptom indicates fracture
of the orbit and most frequently of its roof. But Berlin (G. and S.,
VI., pp. 567-8) quotes six cases by ATon Holder, Avhere at the autopsy
orbital hemorrhage appeared without any fracture of any part of
the skull, as demonstrated by stripping off all the dura mater. But
in these six cases there had been severe falls or blows, and in some
of them there Avas intracranial hemorrhage. In some instances the
intracranial bleeding had reached into the orbit, in other cases the
orbital hemorrhage Avas idiopathic. As a proper offset to these
observations, von Holder furnished an account of 124 cases of frac-
ture of the skull, in 79 of which he found fracture of the roof of the
orbit, and of these 69 had hemorrhage into the cellular tissue of the
orbit, and in the remaining 10, blood was confined to the A'icinity of
the periosteum. It follows that in cases of severe injuries (either
fracture of the skull or commotion) Avith orbital hemorrhage, this
symptom, in ninety-tAvo per cent, indicates coincident fracture of
the orbit, Avhile in only eight per cent does it take place without
fracture of the orbit.
In some cases, severe hemorrhage may find its outlet through
the nose, and, perhaps, get into the stomach. This implies fracture
of the inner Avail and of the ethmoid cells.
752
DISEASES OF THE EYE.
In every case of orbital bleeding, the local conditions give us
anxiety, not specially on behalf of the eye and its surroundings, but
because grave injury has probably been inflicted upon the skull. It
may, however, happen that sight or other functions of the eye are
imperilled. The bleeding may cause atrophy of the optic nerve by
pressure, or laceration of the ophthalmic artery may cause false
aneurism, or cut off the supply to the retina. The muscles may,
one or more of them, be paralyzed. Such contingencies and others
are easily possible. (See pp. 695, 696.)
Treatment consists in cold or iced-water compresses, a pressure-
bandage, and rest. From three to four weeks will be necessary for
removal of the extravasation. To attempt to let out the blood
by an operation, when deeply situated, is useless, and likely to be
hurtful. This subject is really introductorj7 to another of which it
usually forms an incident, viz.:
Wounds and Injuries of the Orbit.
Dislocation of the globe may be produced by a push with a
cow's horn, by a man's thumb or finger in fighting, or by a blunt
arrow, etc; and by insane persons has been self-inflicted. I haA*e
seen it produced by a fall doAvn-stairs when the orbital region
struck against the top of the newel post. The globe was at the
same time ruptured. Considerable force and tact were required to
reduce it within the lids. Gouging, as it is popularly called, may
or may not be attended with rupture of the muscles. The eyeball
may seem unharmed, yet sight be wholly or partly destroyed by
injury to the optic nerve, or by laceration of the choroid. Treat-
ment will consist in replacement of the eye and cold-Avater dress-
ings, pressure-bandage, and subsequent proceedings as the symp-
toms indicate.
A spontaneous thrusting forward of the eyeball outside the
palpebral fissure has taken place in persons who have excessively
prominent globes. It is sometimes feared in exophthalmic goitre,
yet almost never realized. The upper lid can be easily slipped
again in place and if required the palpebral fissure can be shortened
at the outer angle by paring the edges of the lids and using a few
stitches.
Wounds of the soft parts at the margin of the orbit are often
caused by blows with the fist, especially when armed with brass-
knuckles or wearing a large ring. It is often remarkable how clean
cut and well defined the skin wound is, presenting to cursory in-
spection the appearance of an incised cut. It will be noted, how-
ever, that the deep parts of the skin are more extensively wounded
than the surface because cut by 'the bony edge, that there is consid-
THE ORBIT.
753
erable contusion and swelling, and the reaction is always greater
than after a simple incised wound. Suppuration often folloAvs.
To get rid of large clots of blood in the soft tissues, is sometimes
a matter of three or four Aveeks; and the process of absorption may
be somewhat hastened by pressure, or, if this be painful, by mas-
sage, while the surgery of the prize ring practises opening the
swollen skin Avith a lancet. With careful antisepsis and accurate
closure by the dressing of collodion and fibres of absorbent cotton
this is quite permissible.
Gun-shot wounds of the orbit are common both in civil and mili-
tary surgery. The most distressing cases are those in which the
ball enters at the side and goes through the orbital walls trans-
versely. It may lodge anywhere and may destroy one or both
globes, or may leave each seemingly intact. Usually, the sight of
one or of both eyes is destroyed, according to Avhether the missile
enters one or both orbits. The ball has been known to go into the
opposite upper jaw. Bleeding from the nose or mouth will indicate
to some degree its direction. Life may be spared, or may be de-
stroyed by inflammation extending to the caA7ity of the skull. I
haA'e seen two cases of this description. In one, the eye on the side
of entrance Avas sound to outward appearance, but sightless; the
other eye was atrophied. The explanation of the loss of sight is
easily understood.
When the bullet takes some other than a transverse direction,
the injury inflicted Avill depend greatly on its penetration, as Avell
as on its special direction, and Avill often be fatal. A case worth}7
of record I have reported in the Transaction of the American
Ophthalmological Society for 1881.
A circus proprietor, twenty-eight years of age, while in bed in a hotel in
Texas, was awakened from sleep by a man who presented a pistol to his face
and demanded his money, which was under his pillow. The assailant fired,
seized the money, and fled. The ball entered the left orbit close to the outer
canthus. For several Aveeks the patient was in bed, and was much of the
time unconscious. Four months afterward I saw him. There was no cica-
trix or irregularity which would indicate the place of entrance; the outer
orbital margin was regular and smooth; the eye was sightless, though per-
fectly capable of motion in all directions. The ophthalmoscope shoAved a
large laceration of the choroid on the outer side of the fundus, and atrophy
of the optic nerve. The left ear was totally deaf—not able to hear the
tuning-fork applied to the head. In the meatus auditorius was a swelling
of the upper wall close to the membrana tympani, which was covered with
tense skin, was tender to touch, hard, and about five millimetres across. It
was just such a protuberance as would be made by a small pistol-bullet
lodged in the bony meatus, and there I believed it to be. The patient did
not experience any unpleasant symptoms, and resumed his travels as a show-
man. His other eye, Avhich he had never depended on, had myopic astigma-
tism, and with —12c 180° he gained V = £§. In 1891 his condition remains the
same. . 0
754
DISEASES OF THE EYE.
I have seen the eye made sightless by the entrance at the outer
angle of the orbit, of a bullet from a toy pistol.
BIoavs upon the margin of the orbit sometimes implicate the
supra-orbital or the infra-orbital nerves, and to their injury has been
ascribed the loss of sight which sometimes has been known to
ensue. The true cause in the great majority of cases is to be sought
in fissure of the orbit reaching back to the optic foramen, as will
be referred to later. Amaurosis by injury of these branches of the
fifth nerve has long had a place in ophthalmic pathology, but it
stands on A7ery weak evidence.
Dislocation of the malar bone is an accident which can occur,
and I have recorded an instance (see Trans. Am. Oph. Soc, 1880).
It results generally from violent falls upon the face, whose force is
spent directly on the bone. It may Cause extensive orbital hemor-
rhage and possibly diplopia through interference with the inferior
oblique muscle. It Avill be recognized by a notch near the middle
of the inferior orbital margin, where the malar joins the superior
maxillary bone, and by another notch Avhere it joins the external
process of the frontal bone, and often the zygomatic arch is bent
or broken. Anaesthesia of the infra-orbital nerve, and pain in chew-
ing, because of pressure on the canine and adjacent teeth, are
symptoms which continue for some time. The symptoms vary a
little according to the direction in which the bone is displaced.
Fractures of the Avails of the orbit occur in a great variety of
Avays: by cuts, bloAvs, falls on the head, by crushing forces, etc.
Such an accident, Avith extrusion of the eyeballs from the sockets,
has been caused spontaneously in child-birth when there was de-
formed pelvis (see case reported by Berlin in G. and S., VI., p. 588),
and might result from injudicious handling of the forceps. If the
roof is implicated, there Avill be danger of inflammation of the brain,
yet out of 19 such cases collected by Berlin, 16 recovered. Frac-
tures of the inferior orbital Avails, besides opening the antrum,
damage the infra-orbital nerA7e, and are liable to be followed by
distressing neuralgia or by anaesthesia. Fracture here, and also
of the inner wall of the orbit, will be succeeded by emphysema of
the cellular tissue. In some cases this will be extensive, and a case
is reported by Knapp of exophthalmus produced in this Avay. Nose-
bleed will also occur.
Double vision may result from injury of the orbital walls, and
not always by implication of the nerves but by damage to the
muscles at their origin in the apex. Such a diagnosis can rest only
on probability, yet I think sufficient reasons can be adduced for this
opinion in the folloAving case:
Dr. T. J. H., set. 87, a physician of Massachusetts, consulted me in Janu-
ary, 1893. In October, 1892, he was thrown from a horse, striking on the left
THE ORBIT.
755
shoulder and the left side of the head. He was stunned but not unconscious,
no vomiting. He had to go to bed for a few days. Subconjunctival ecchymosis
appeared on the inner side of each eyeball soon after the injury. The fol-
loAving day he had diplopia, and ptosis of the right eye. At the present time
the falling of the right upper lid has practically disappeared. The diplopia
requires for correction a prism 10° base up, and a prism 15° base out over the
right eye, which means that movement upward and outward is restricted.
By the ophthalmoscope the optic disc was found a little gray and V = 0.8 (?).
In the other eye V = 1.0.
By studying Figs. 262 and 263 it will be seen that a frac-
ture at the apex involving the vicinity of the upper part of the
spheno-maxillary fissure will include the origins of the rectus ex-
ternus, the rectus superior, and of the levator palpebrae. Were
these muscles damaged by lesion of their respective nerves it would
Fig. 262 shows schemati-
cally the origins of the
ocular muscles. Rl, Rec-
tus externus (lateralis);
Rs, rectus superior; Lp,
levator palpebrae; Os, ob-
liquus superior; Rm. rec-
tus iuternus (medius); Ri,
rectus inferior; Fo, fora-
men of motor oculi (third)
nerve; Co, canalis opti-
cus.
be difficult to understand why other muscles supplied by the third
should escape injury; but supposing the mischief to consist in fis-
sure and some displacement of a fragment of bone at the apex (the
Aving of the sphenoid) the explanation seems easy. Ptosis as a re-
sult of fracture of the orbit is not very rare. In the above case my
belief is that the paresis of the levator palpebrae, of the rectus
superior and rectus externus, and the lesion of the optic nerve were
all due to fracture at the apex.
It will be noted that the rectus externus has tAvo heads and is
in close relation to the third nerve. . The same anatomical relations
are shown in Fig. 263, which displays the stumps of the divided
muscles in the right orbit, and part of the nasal cavity.
Fractures of the orbital Avails by penetrating wounds, as may
happen in fencing, or by a bayonet, arroAv, umbrella-ferrule, hook,
key, etc, are relatively more serious than those just mentioned.
Fig. 263.
756
DISEASES OF THE EYE.
This is true, most especially of the roof of the orbit. The external
wound may be trifling, the eye often escapes harm, but if the cavity
of the skull has been entered, the prognosis is very grave. In
twenty-five per cent of the cases (Berlin) the patient immediately
falls unconscious, but presently recovers. It is an important mat-
ter to know whether the wound has gone through the orbital
■ roof. The outward opening is often small, it partially closes, and
to find a way through it with a probe is very difficult, because the
eyeball is violently pulled around as the weapon enters, and after-
Avard returns to its place, thereby making the track sinuous. But
there is very grave doubt as to the propriety of venturing to use a
probe. The probability of the presence of a foreign body, or of the
displacement into the skull of fragments of bone, may justify prob-
ing when the wound is recent and the symptoms urgent, but the
surgeon's little finger is far safer as an exploring instrument, and,
on the whole, a prudent man would, in the great majority of cases,
refrain from meddling. Antiseptic precautions may render such
an exploration less dangerous than it would be AA7ithout them, but
a discreet surgeon will not permit his professional curiosity to im-
peril the patient's limited chances of recovery. Very seldom will
his probe or his finger be allowed to enter the orbit.
Cerebral symptoms, when they occur, may be due to intracranial
hemorrhage, or to inflammation. The latter class of symptoms
Avill be various, viz., pain, weakness, delirium, vertigo, paralysis,
coma, etc. But it is notable that head-symptoms may be tardy in
appearing, and be so long delayed as to make perforation of the roof
seem to be highly improbable, yet the dreaded tokens may in time
appear. In one case, forty days passed without any cerebral signs,
then the patient suddenly died after a foreign body was extracted
from the orbit. Berlin has gathered 52 cases of perforating wounds
of the orbital roof, of whom 11, i.e., twenty-one per cent, recovered;
but of these, three were hemiplegic, one had persistent headache'
and one became imbecile. The remaining 44, i.e., seventy-nine per
cent, died; of the deaths, one-half were from the immediate effects
of the wound, and the other half from the subsequent complications.
At the autopsy, the bony aperture Avas generally small, and
fragments had entered the cranial cavity. Wound of the brain
may be small, or in some cases very large. Of the causes of death
at a late period after the wound (18 cases), in 15 there was abscess
of the brain, with or without meningitis; in 2, thrombosis of the
longitudinal sinus; in 1, "pus at the base of the brain." In 6 of
them, bits of bone were found in the brain-substance.
An illustration of what may ensue from fracture of the orbit, is
the following:
THE ORBIT
757
A boy fifteen years of age, while dodging through a croAvd in a meat-
market, stooped to get on more easily, and ran against a large meat-hook.
Its point caught him in the right orbit, under its upper margin, tearing off
the upper lid from the inner angle, fracturing the edge and perforating its
roof. He was taken to the New York Hospital, and kept under treatment
for six weeks. At the end of that time he came to the New York Eye and
Ear Infirmary, and I found a scar running nearly the whole length of the
upper lid beneath the brow, the lid everted and immovable, its conjunctiA-al
surface converted into a florid mass of papillary granulations; the cornea
visible for its lower half, and in a state of fixed convergence. The globe
could not be moved, but the lid could be turned with the finger, and could
be slightly lifted by his efforts. The eye looked well, but Avas almost sight-
less. There was atrophy of the optic nerve, apparently the result of neuri-
tis. At the upper margin of the orbit was a deep notch, which evidently
went back into a deficiency in the roof. He was unable to say whether any
fragments of bone had come out. By a pressure-bandage on the readjusted
lid, the thickening of the conjunctiva, and the swelling of the lid so far
abated in eight months, that I ventured to try to bring the cornea to the mid-
dle of the palpebral opening. I divided the rectus internus, and dissected the
parts about the caruncle very freely, but could not turn the globe outward.
I then attempted to bring forward the externus, but could not rotate the
globe outward. Finally, I explored the orbit on its outer wall, behind the
globe, and found that this surface had been forced inward, and that the eye-
ball had become adherent to the periosteum at its posterior part. I tore
away this attachment, and then was able to rotate the eye to the middle of
the palpebral slit, where I placed it, and closed up the conjunctival wounds.
The reaction was not extreme, and the eye was permanently fixed in the
position where I left it. Some vision in the outer part of the field Avas ob-
tained, but the upper lid remains drooping over the upper half of the cornea,
of normal thickness and Avithout ectropion.
Fracture may go through the lamina cribrosa and the ethmoid,
with or without fracture at the base of the skull. A notable
symptom occasionally met Avith is the dropping of clear fluid
from the nose when the patient sits up. A case is quoted in
Berger and Tyrman (" Die Krankheiten der Keilbein-Hohle und der
Siebbein-Labyrinthes, etc.," Wiesbaden, 1886), p. 74, where the flow
took place from the nose Avhen the man sat up, and from the ear
when he la}7 upon the right side. Autopsy ten days after the in-
jury disclosed fracture across the sella turcica and of the lamina
cribrosa of the ethmoid.
Another class of cases of orbital fracture are those in which no
ordinary symptoms of this lesion appear, but in which, after an in-
jury to the head, loss of sight occurs in one or both eyes, and with
very slight symptoms in the fundus oculi. After a time the optic
nerve may show signs of inflammation or of atrophy. Again, there
are many cases of fracture running through the canalis opticus or
the roof of the orbit, simultaneously with fracture of the base, or
in some other region of the skull. The profound injury sustained
distracts attention from the state of sight, and we seldom know
758
DISEASES OF THE EYE.
that it has been impaired, nor would the patient, perhaps, be able
to tell us anything about it. A most interesting study of these
cases has been made by Dr. von Holder, who, in his capacity of
pathologist, examined 124 cases of fracture of the skull. Stripping
the dura mater from the base, he was enabled to detect injuries to
the bone and hemorrhages which would otherwise have escaped
notice. During forty years he made these observations and took
notes of what he found (Berlin, G. and S., VI., p. 604). Among the
124 cases, there were 86 of fracture at the base, and in 79 of them
the fracture extended into the orbital roof. Von Holder states that
out of 86 cases of fracture at the base, in 63 he found a fissure or
fracture running through the optic canal, and always through its
upper wall, and sometimes also through the inner wall; occasion-
Fig. 264.—Section of Skull through the Canalis Opticus, seen from behind, and parts in front as
well as behind the section taken away. The body of the sphenoid seen in the middle, with its un-
symmetrical sinuses. The letters n and n point to the walls separating the optic canals from the
sphenoidal sinuses. In this specimen they are excessively thin. In other cases one may be thin
and the fellow very thick or both may be very thick. The optic canals are seen in oblique section,
but their length and form are well displayed. This section is valuable to aid in understanding
various lesions at the canalis opticus, both traumatic and spontaneous.
ally on both sides. In 42 cases there was hemorrhage into the
sheath of the nerve, and he never found blood in the optic sheath,
unless its bony canal was fractured. The blood may be derived
from the cavity of the skull, or from the vessels of the sheath,
or from the torn central artery of the retina.
Other observers have seen the same symptom. If the quantity
were large, it might, as in a case reported by Knapp, be sufficient
to injure sight by direct pressure on the nerve, but smaller quanti-
ties might also destroy sight by interference Avith the central retinal
artery, causing ischemia retinae, and all the features, too, of em-
bolism. Prescott Hewitt gathered 68 cases of fracture of the base,
and found the orbit involved in 23.
Noyes' •' Diseases of the Eye
Plate B.
4.
\
THE ORBIT. 759
Possessed of these facts, it becomes intelligible Avhy, after a fall
on the head, total, or nearly total blindness may ensue, with, per-
haps, no ophthalmoscopic lesions.1 Ultimately, signs of inflamma-
tion or of atrophy or pigmentation of the disc may appear. Again,
venous hyperaemia, ischaemia of arteries, opacity of the nerve o:-
the retina by exudation, hemorrhage into the vitreous and into the
retina will suggest intravaginal hemorrhage. For example, Berlin
quotes 30 cases of blindness after injuries of the head, in which oph-
thalmoscopic examination was made. In 17 there was atrophy of
the nerve and in two there was pigment-deposit in the disc. A
case which I published was seen in the stage of neuritis. Another
case I have seen which presented nothing but slight fulness of the
veins. Other reported cases have exhibited hyperaemia of the disc,
ischaemia, hemorrhages, etc.2
The possibilities which may follow from injuries of the canalis
opticus and the ethmoid are emphysema of orbit and lids, injury
of optic nerve and damaged sight, dropping of fluid from the nos-
trils, lesion of the carotid artery and cavernous sinus with result-
ing pulsating exophthalmus, anaesthesia of the second and third
branches of the trigeminus nerve and lesion of other cerebral
nerves.
The loss of sight in fracture at the canalis opticus may come on
a few hours after receipt of the injury, as happened to a one-eyed
man who was struck on the brow by a heavy potato and the
result was extreme atrophy of the nerve. The precise lesion may
be a hemorrhage into the nerve or its sheath, or its laceration by
a splinter of bone. The quality of the process will vary accordingly
and the loss of sight be sudden or gradual. Seggel gives two cases
with charts of visual fields showing great and irregular limitation,
Archiv f. Augenheilkunde, XXIV., 4, 293.
Treatment of these injuries of the orbit is to be conducted on
general principles and according to the dominant symptoms. Of
course we have nothing to say on the general subject of fracture
of the skull. As to the orbit, loose and accessible bits of bone or
foreign bodies are to be removed, all excitement to be avoided,
antiphlogistic measures to be used, namely, cold applications, re-
moA'al of secretions, and maintenance of free escape of discharges.
In this connection it is important to consider what steps are to be
taken when symptoms of abscess, deep in the orbit and perhaps in
the brain, threaten. For orbital abscess there Avould be no hesita-
tion in promptly giving a free outlet. The employment of antisep-
tic methods in the operation, and in the subsequent dressing are of
course necessary. Should symptoms of brain trouble threaten and
1 See Callan, Trans. Am. Oph. Soc, 1891, p. 174.
'2 See also Berger and Tyrinan, 1. c, pp. 75, 96.
760
DISEASES OF THE EYE.
the escape of pus be so hindered as not to be otherwise insured, it
might be proper to enucleate the globe. It also becomes a ques-
tion as to the propriety of searching for pus in the substance of the
frontal lobe of the brain. The existence of a perforation of the or-
bital roof Avould invite further exploration, and modern surgery
moves in this direction. Some cases have been thus treated under
constant antisepsis, yet the difficulties of perfect drainage are very
great, and the tendency is to extension of the morbid process, and
no cheering results are yet recorded in these cases Avhen acute.
In cases of more chronic type the following may be cited from
my own experience.
In 1857, a boy, ten years of age, was brought into the New York Hospital
after having been run over by a street car. He had fracture of the occipital
and frontal bones. He remained about three months in the institution. He
recovered without paralysis or loss of any function, but was always subject
to headaches, and had a small fistulous opening at the upper and inner angle
of the right orbit, just under the broAV. In 1865 I saw him and noted the
fistula, and warned him that he was liable to have trouble from it. He lived
a wild life, and was sometimes drunk. In the latter part of 1865 I was
called to see him, and found he had serious brain symptoms. Consciousness
was not quite abolished; pulse slow, respiration stertorous. He had had
severe headache and been in bed for several days. By the ophthalmo-
vope I could only see hyperemia of both tiptvpr. Th^ usual discharge from
the fistula had recently ceased. I concluded that there must be an abscess
near this spot within the cranial caA7ity, and determined to trephine the skull
just above the fistula. A large crucial incision was made, and I trephined just
outside of the supra-orbital notch. The dura mater bulged into the wound.
I opened it and pus escaped. About half an ounce issued, and I put my
finger into the cavity over the roof of the orbit. The patient, who had sunk
into coma during the consultation over his case, recovered intelligence at
once, in half an hour was able to talk, and made a good recovery. He had
fungous granulations (hernia cerebri) from the wound, but at length by a
pad and pressure-bandage and excision this was controlled, and he has never
reported himself since.
h
Wound of the optic nerve is a rare injury, but of which I have
seen two cases and Aschmann (Inaugural thesis, Zurich, 1884) has
made a collection of twenty-one cases, of which two were in Horner's
clinic and the rest gathered from literature. One of my cases is
as follows:
In May, 1882, a boy aged 10 years, while playing soldier with a comrade and
fencing with sharpened sticks, was wounded in the left orbit. The end of
the stick broke off. He was not stunned nor did he feel much pain. He ran
up one flight of stairs; and got his grandfather to pull out the piece with
pliers. It measured 21 inches in length, tapered to a sharp point; at its base
it was about i inch thick and was slightly bent at the distance of a quarter
of an inch from the tip. Five hours after he was seen by Drs. Munn and
THE ORBIT.
761
Schoonover, Avho found the globe mobile, pupil dilated and fixed, no hemor-
rhage in conjunctiva or skin; no external lesion of eye, but no perception of
light. In tAventy-four hours I saw the patient and found the Avound at the
infero-temporal angle of the orbit, and almost no reaction, no exophthalmus.
Pushing the globe deep into the orbit caused pain, but movements of the eye
Avere painless and perfect. Pupil between 4 and 5 mm. in diameter, slightly
larger than the other and would neither dilate nor contract. No sensation
of light. No photophobia in other eye. By ophthalmoscope, the nerve is
intensely red, a little swollen, veins very turgid. The next day the color of
the nerve was normal and vessels of usual size. After ten days the temporal
side of the nerve was pallid. After four weeks the nerve became pale over
the whole disc, and the vessels remained normal in size.
No serious symptoms occurred at any period, although he became quite
excited from anxiety during the first few days of his confinement to bed. It
seems clear that the optic nerve was penetrated near the apex of the orbit,
perhaps only severely contused. The lesion was behind the entrance of the
arteria centralis, and no other organ save the optic nerve was damaged.
Probably the bony walls escaped injury, and it is likely that the nerve was
hurt A'ery near the optic canal.
Foreign Bodies in the Orbit.—Foreign bodies entering the
orbit and passing out of sight are extremely difficult to find unless
of considerable size. Even if they are large they may lodge in the
orbit without destroying the globe, as happened in an instance re-
ported by Mr. Carter, in Avhich a piece of an iron hat-peg, 3T3o inches
long, Avas buried in the cavity, and remained there for from ten to
tAventy days without the patient being aAvare of it. It Avas extracted
Avithout injury to the functions of the eye. It is not necessary to
say that foreign bodies Avhich can be seen or felt should be carefully
and immediately extracted. But the point of difficulty is to decide,
1st, whether a foreign body has entered the cavity; and, 2d, how to
find and remove it. A doubt arises as to the penetration of foreign
bodies in cases of wounds by bird-shot. The place of entrance is
very small, closes instantly, and heals promptly. It is often im-
possible to trace them, nor is it generally needful to meddle Avith
them. I have known a fragment of iron of considerable size, struck
off by a chisel, to enter and be completely hidden. The irregularity
of the piece, the yielding nature of the tissues, and the sinuosity of
the Avound, make exploration by a probe very unsatisfactory. As
above remarked, a reason for the difficulty is, that Avhen the foreign
body enters, it drags the eye around toward itself, and when it has
found a lodgment, the globe returns to its position and thus tAvists
the track of the wound.
A case, illustrating the difficulties of diagnosis and the proper
mode of treatment in obscure cases, will illustrate Avhat*needs be
said:
A man walking among bushes felt a twig strike his eye, and was convinced
that a piece of it had found entrance. Some bleeding occurred; he suffered
762
DISEASES OF THE EYE.
considerable pain; he found his sight uninjured, and for some time he did not
go to a physician. A chronic inflammation lingered about the lower part of
the eye, and he was annoyed by some pain and discomfort. The physician
looked at the inflamed part and everted the lower lid, but could see no sign
of wound or scar and prescribed for what he regarded as simple conjunctivi-
tis. The man's statement that a foreign body had entered or was present in
the orbit, he did not credit. For two weeks treatment by astringents was
kept up, when I was asked to see the case. I discovered in the inferior cul-
de-sac a small projecting granulation, as large as a No. 2 shot, and around
this the conjunctival and scleral hyperemia concentrated. I at once assumed
that there was a foreign body in the orbit and advised its removal. For two
weeks longer the same medical treatment was continued, and the patient
then was put into my care at the New York Eye and Ear Infirmary. No
THE ORBIT.
763
can be steadied by being pressed against the walls. The operation gave rise
to no serious trouble, and in ten days the man was discharged cured.
From the above case, I venture to advise the insertion of the
operator's finger into the orbit along the track of the foreign body,
and to use it both as an explorer and as a means of guiding the
search with forceps or other suitable instruments.
My experience in the following case brings up again the question
of how far interference with the brain and intracranial cavity
may be justifiable. The case was published in extenso in the Am.
Journal of the Medical Sciences, July, 1882.
Fig. 266.
A boy, aged 19, was injured by the explosion of his gun, and the butt of
the barrel, known as the breech pin, broke through the nose and went in out
of sight. It was not known that a foreign body had lodged, the wounds
healed except some sinuses and I saw him five months afterward. His ap-
pearance is shoAvn in Fig. 267. Explorations discovered the foreign body
depicted of natural size in Fig. 266. Extensive incisions and dissection Avere
required to trace its situation and its form was totally unknown. By using
large pliers, it was brought out from the nasal cavity and found to have pene-
trated the roof of the orbit and the frontal lobe of the brain—the position it
occupied is shown upon a prepared
skull in Fig. 2o5. Its presence in the
brain had not been indicated by any
symptoms Avhatever. On the four-
teenth day eA7idence of abscess in
the brain led to an operation for
evacuating pus by enlarging the
opening in the orbital roof. Pus
was found outside of the dura mater
and in the brain tissue. On the six-
teenth day beginning paralysis of
the opposite arm and leg showed
that full relief Avas not secured
against cerebral pressure. The
skull was trephined at the spot
shown in Fig. 267, an exploring needle introduced, and at the depth of If
inches pus was found. A drainage tube was passed from the trephine hole
to the orbital opening to give vent to pus.
From the beginning of treatment assiduous efforts were made to secure
free vent to secretions and these efforts Avere not relaxed up to the end of
Fig. 267.
764
DISEASES OF THE EYE.
the case. Death took place on the thirty-ninth day after removal of the
foreign body.
Exophthalmic Goitre, Graves' Disease, Basedow's Disease.
Under these names is described a condition presenting as symp-
toms, palpitation of the heart, hypertrophy of the thyroid gland,
and protrusion of the eyeballs. While the fully developed disease
includes these three items, any one of them may be wanting.
Moreover, while both eyeballs are usually extruded, and can be
pressed nearly back into their proper place by the fingers, one may
be more advanced than the other, and sometimes only one is af-
fected as is noted by Stellwag, and as I have once observed. Other
Fig 268.
symptoms are: extreme excitability of the patient; she is readily
startled, and has flashes of heat; pallor, and flushing of the face
quickly alternate; the action of the heart is very irregular and
thumping, its pulsations may be habitually one hundred, or mount
to one hundred and sixty; there may be some consecutive hyper-
trophy and systolic bruit, and also a bellows murmur over the large
vessels. The thyroid presents variable and sometimes unsymmetri-
cal enlargement. A choking in the throat (globus hystericus) is
common; the patient may be unable, for an instant, to catch the
breath or swallow. The eyes stand forward in a peculiar stare
and show the sclera above the cornea, and, as Graefe noted, this
iook of surprise or fear is aggravated by actual retraction of the
upper lid, which exposes the globe more than the pushing forward of
THE ORBIT.
765
the eye by a tumor is observed to do (see Fig. 268). Stellwag also
noted the infrequency and slowness of the action of the lid in Avink-
ing. By exposure the cornea and conjunctiva are irritated and con-
gested, and ulceration of the cornea has been observed. Sometimes
it is never fully covered by the lids, and is more exposed in sleep.
The pupil usually is natural in its action, although mydriasis has
been noted; movements of the eye are unimpaired, diplopia is rare
and transient, vision is not involved, the circulation in the fundus is
ordinarily not peculiar, yet Becker has noticed that the retinal
arteries pulsate, and not in the disc alone, but over a considerable
length; attacks of migraine can occur and with hemiplegia. The
disease is complicated with anaemia, and in Avomen often with
amenorrhcea and chlorosis, and the patients are hysterical. Melan-
cholia is common and mania may occur. We may also see diges-
tiA7e disturbances, nausea, vomiting, diarrhoea, bloody stools; also
cough, profuse sweating, and Bulkley has cited two cases with
urticaria. There may be nodules of inflammatory exudation, or
patches of transient redness, and increased heat of the skin, vari-
cose dilatations of vessels may take place on the nose or cheeks, and
there may be ephemeral tumors on the eyebroAvs and lids, attended
sometimes by dilated vessels. I have seen one case in which un-
doubted exophthalmic goitre was attended by firm tumors at the
lower border of one orbit, and Avhich, under the microscope, seemed
to be composed of enlarged lymphatic gland tissue; only one eye-
ball Avas protruded. A case of Heymann's had repeated paroxys-
mal attacks of conjunctivitis with membranous exudation.
The disease occurs with greater frequency among women, ac-
cording to Emmert (see Arch, fiir Oph., XVII., p. 30), in the ratio
of nine to one. The cause is not definitely ascertained; most of the
lesions are traceable to disturbances of the sympathetic nerve, but
where their origin may be is not determined. Some fix upon the
cervical sympathetic, and others upon the cervical portion of the
spinal cord. Sattler thinks the vaso-motor centre in the medulla to
be the primary seat of the lesion (Graefe and Saemisch, VI., pp.
941-1024). In all these structures autopsies have found lesions, but
not with uniformity. The heart is often a little, seldom greatly en-
larged, and there may be insufficiency of the mitral valves; the thy-
roid gland shows in old cases some increase of connective tissue
and colloid cystic degeneration, but at death it usually collapses; in
the orbit rarely is anything abnormal discovered, and the ocular
protrusion disappears. It is, therefore, justifiable to assign the cause
of exophthalmus to vascular enlargements, and Snellen has cor-
roborated this opinion by showing- that, with a stethoscone. a dis-
tinct vascular murmur can be beard durinsr life. Recklinghausen
has found fatty degeneration of the ocular muscles.
766
DISEASES OF THE EYE.
An autopsy by Dr. White* found the sympathetic in the neck
normal, likewise the spinal cord, save one or two insignificant hem-
orrhages. The lesions thought to be important were slight inflam-
matory signs at the lower end of the olivary bodies and from
here up to the restiform bodies were numerous hemorrhages
which could be further traced to the aquaeductus Sylvii. This au-
topsy corroborates the view which Sattler and others hold that we
are to look to the medulla oblongata for the site of the lesion,
whatever organ in it may be at fault.
FolloAving the publication of this autopsy is an article by
Manby2 in which the occurrence of exophthalmic goitre and of dia-
betes in different members of the same family is related. Three
such families are mentioned—and the coincidence points to the
vicinity of the fourth ventricle as perhaps the seat of trouble com-
mon to all these individuals.
The disease progresses slowly in most cases, while a few pa-
tients haAre the good fortune to reach an early recovery. There is
great emaciation and prostration, and in fatal cases the end is
brought about by asthenia or by phthisis.
For an extensive account of symptoms and characteristics of the
disease see Gowers on "Diseases of the Nervous System." Both
glycosuria and temporary albuminuria have been observed: some-
times extensive ophthalmoplegia externa—see paper by Fitzgerald,s
and by Story 4 who noted atrophy of the optic nerves in one case.
Treatment.— This will be determined by the character of the
leading symptoms. In many cases, and perhaps in the majority,
pronounced anaemia will call for iron in various forms and long
continued. Next in order Avill be remedies to improve the heart's
action, hence digitalis is much employed, and general tonics such
as quinine, strychnia, phosphoric acid will find place. The use of
ergot has been popular and has to me seemed beneficial. Galvan-
ism has been much tried and by Bartholow is said in three cases to
have done evident service, but many writers confess their disap-
pointment Avith it. Atropia or ext. belladonnae is strongly com-
mended by Gowers and must be given to constitutional effect.
JaneAvay, so late as May, 1889, in a paper before the N. Y. County
Med. Assoc, which graphically portrays the disease in its clinical
aspects, speaks favorably of the tincture of strophanthus, five min-
ims three times daily, gradually increased if necessary. He found
it succeed when digitalis had failed. He emphasizes the importance
1 British Med. Journal, March 30th, 1889.
2 British Med. Journal, May 11th, 1889.
3Quoted in Ophthalmic Review, vol. ii., 148, 1883.
4 Quoted in Ophthalmic Review, vol. ii., 161, 1883.
THE ORBIT.
767
of iron and of rest both physical and mental; menstrual irregular-
ities may either precede or succeed the disease.
Bromides and means of inducing sleep, especially sulphonal, will
have occasional application. It is advisable to avoid opium, al-
though Janeway, while giving this caution, says that exceptionally
it has done good. Its danger is consequent impaired nutrition.
A formula which I have many times found useful is: 1} Ferri
pyrophosphate, zinci bromidi, aa. 3i.; tr. digitalis, 3 v.; fl. extr.
ergotae, § iv. Dose a teaspoonful three times daily (Hammond).
One will need to vary remedies in the long progress of a case and
must test what will prove most effectual.
For attacks of dyspnoea and palpitation, hip baths, mustard to
the feet, cold to the thyroid gland are helpful. Sometimes ether
and chloroform are given, or a single dose of morphia for spasmodic
attacks.
In all cases the greatest importance attaches to hopeful assur-
ances of possible improvement and that the distressing symptoms
are not so dangerous as the patient thinks. Cheerful surroundings,
absence of care, avoidance of exitement, generous diet, sufficient sleep
and adaptation of remedies to occasional symptoms, codeia, chloral,
etc., besides steadfast perseverance in iron added to whatever other
remedy above suggested proves most fitting, is the line to be fol-
loAved. Should, from exposure, inflammation of the eye occur,
prompt measures must be instituted.
For the exophthalmus no particular treatment is to be adopted,
except, if the cornea and conjunctiva become dry through exposure,
a little purified (white) A-aseline or cosmoline may be put between
the lids tAvo or three times daily. A pressure-bandage is some-
times comforting, and pushing back the globes into the orbits gives
some relief; it may be necessary to hold the lids in approximation
by a strip of rubber plaster, or even to do tarsoraphy for partial
closure of the lids at the outer angle. Partial division of the leva-
tor, as suggested by Graefe, is not practised.
A recent discussion on the treatment of this disease is found in
N. Y. Medical Record, July 11th, 1891. Rest in bed, great atten-
tion to careful feeding, massage are much insisted on by Dr. W.
H. Draper, Avho also thinks iod. potass, beneficial in addition to
digitalis, etc. The general view was that the disease, being a neu-
rosis, is to be dealt with on general indications derived from symp-
toms, and that no special remedy can claim control. The experi-
ences of an electro-therapeutist are given by Rockwell, N. Y. Med.
Record, September 30th, 1893.
Recovery may take place Avithin one to five years. A fatal result
may take place by exhaustion, by intercurrent phthisis, and occa-
sionally the disease is complicated Avith organic heart lesion.
CHAPTEH XXL
USE OF ARTIFICIAL EYES.
Prothesis Oculi.—Great care must be taken to have, artificial
eyes fit easily and not be too large. They are of very little use
when both the globe and much of the contents of the orbit have
been removed; they serve best when an eye, only a little reduced
in size remains, and its surface is not sensitive. But generally they
are to be Avorn after the globe has been enucleated and the other
tissues are left. Under these circumstances a moderate degree of
mobility is possible, but varies in different persons. It is unavoid-
able that a deep furroAv should remain in most cases beneath the
broAv, because the draAving together of the conjunctiva in the cen-
tral cicatrix pulls down the superior cul-de-sac. When an eye fits
Avell, a patient is not conscious of its presence. Great pains must
be taken to preserve its polish. The enamel will begin to dissolve
aAvay in a year or more, according to the quality of the material
and of the ocular secretions. The eye should be Avashed carefully
with clean water or with dilute alcohol, but not kept in Avater for
hours, as during sleep. It should never be worn during sleep. If
much discharge from the conjunctiva is excited, the shell must be
very carefully examined for loss of smoothness on its edges or sur-
face, and the conjunctiA7a treated by mild astringents or boracic-
acid solutions. A little A-aseline will prevent the drying on the
shell, of secretion Avhich may be unavoidable. If, as happens after
long use or carelessness, the conjunctiva become granular, with
papillary hypertrophy, the shell must be laid aside and the parts
treated until the membrane recovers. Shrinkage may take place
by Avhich the conjunctiA7al space is much reduced, and only a small
eye can be Avorn. Sometimes the membrane becomes xeromatous
and no space may remain to hold a shell. Burns of the eye or
other injuries sometimes leave no cavity in which a shell can be
inserted. In several such instances I have enabled the patient to
wear an eye by cutting the tissues apart and introducing a piece of
conjunctiva from the rabbit. The transplantation is difficult and
tedious, and may need to be done two or three times. After the
healing has been completed greater space is gained, and this is
farther improved by Avearing shells of gradually increasing size
USE OF ARTIFICIAL EYES.
769
until room for a suitable one is secured. It may take six months
to attain this result. A young man has recently presented himself
for whom I did such a proceeding eleven years ago. He has con-
stantly Avorn a shell which is an excellent counterfeit, it gives no
trouble, and the conjunctival sac is healthy. Similar operations
are done to make room Avhen by trachoma, or by long use of a
shell, the sinuses are obliterated. Such cases are very difficult
to deal with, but sometimes success is gained and the shell can be
worn for a few years longer.
With children who lose an eye, or have one which is much atro-
phied, an artificial eye is of importance to prevent arrest of devel-
opment of the orbit and muscles. Such is the common belief, yet I
have a patient now seventeen years old from whom one eye was
removed when she was a little over a year old because of glioma,
and not only does she survive, but the
orbit and the region of the eye is
symmetrical with the other. The con-
junctiva has shrunken to some degree,
and it may be assumed that after many
years a greater diminution of the caA'ity
will ensue. Especially does this occur with those who have tra-
choma. It may be worn for a few hours daily to adapt the parts
to its presence. Constant Avear is undesirable, from risk of break-
age, and because irritation of the conjunctiva is to be avoided.
Unusual pains must be taken to keep the parts in a healthy state.
The shell may need to have notches cut in its edge, or require
some peculiarity of form to fit special irregularities. It is not very
rare to find an artificial eye irritate the parts so much as to cause
sympathetic disease of the other one. I haAre several times seen
this take place, and then its use must be absolutely forbidden. In
recent cases of enucleation the shell should not be worn until all
redness and swelling have disappeared—that is, in from tAvo to
three weeks. If the eyeball should be sunken because of an inflam-
mation, the stump may not permit the use of a shell for tAvo or
three months. An eye should not be Avorn upon a stump which is
knoAvn to contain a foreign body; enucleation should be practised.
Mr. Mules, of Manchester, England, advocates the use in certain
cases of an artificial vitreous which is a sphere of thin glass intro-
duced within the sclera after all its contents have been removed.
The scleral cavity must be thoroughly washed Avith sol. corrosive
sublimate, 1 to 3,000, all bleeding checked, and when the thin globe
is inserted it is held in place by silk sutures in the sclera. Such a
proceeding requires several Aveeks for healing to be complete, and
is applicable to cases where the form of the globe is not much
49
770
DISEASES OF THE EYE.
altered. There has not yet been much experience of surgeons with
it. While some unfavorable testimony has been given, Mr. R. B.
Carter1 reports 13 operations, of which 5 were failures, 7 immediate
successes, and 1 succeeded upon employing at a second trial a
smaller ball. He thinks the use of a horse-hair drain at the equator
and below important. He uses boroglyceride freely as an antiseptic
during the operation. Severe chemosis frequently occurs and de-
mands incisions. He speaks in much praise of the good appearance
and mobility of the eye in successful cases. One case of sympa-
thetic ophthalmia following this proceeding has been reported.
Hence the need of caution.
Lang, 1887, reports eigrht cases: speaks of the severe reaction and that
the average stay in hospital was twenty-four days. Cross mentions a case
where the artificial vitreous excited sympathetic ophthalmia, which disap-
peared after its removal.
Lang2 has in sixteen cases put a hollow glass or silver globe within the
capsule of Tenon after enucleation. Bleeding is fully checked by ice and ir-
rigation, the ball inserted, the upper and lower edges of the now whitened
capsule brought together by a silk stitch on the middle line, a horse-hair drain
inserted, and one other stitch put in on each side; the conjunctiva united by
stitches. Iced antiseptic dressing, " Alembroth wool," or absorbent cotton
Avith bichloride 1:3,000 constantly applied. In fiA7e days patients able to go
out. Surface stitches removed, deep ones left. The sphere is about two-
thirds the diameter of removed eye. Frost has done a similar operation.
Probably this proceeding will find favor; it is worth trying.
To insert an artificial eye, lift the upper lid with the fingers of
one hand, moisten the shell and slip its larger end vertically under
the upper lid. As it passes up, turn it into the horizontal position,
until it rides above the lower lid; with the other hand draw down
the edge of the latter and let it slip into place. To take out the
shell, push under its lower edge a small hook or the head of a large
pin to pull it forward, and at the same time depress the lower lid.
Raise it up gently and it will slide out by pressure of the lids.
Most persons soon learn to take out the shell with their fingers
and have no fear of dropping it.
Artificial eyes made of celluloid have been introduced within a
few years and are commended because they are inexpensive. But
they are less perfect in appearance than those of glass and they
have been found to corrode easily and to excite very soon severe
conjunctival irritation. An additional objection is that, when
they begin to corrode or decompose, they emit a peculiarly foul
odor.
'Medical Press and Circular, Aug. 17th, 1887.
8 Trans. Oph. Soc. United Kingdom, vol. vii., 286, 1887.
OHAPTEE XXII.
STATISTICS OF. EYE DISEASES.
The best statement Ave have on this topic is in an article by
Cohn in Eulenburg's " Real-Encyclopedia der Heilkunde," 1880, p.
602, based upon returns from 67 institutions between the years 1S69
to 1875 and embracing nearly 300,000 patients.
The relative frequency of the diseases is thus tabulated:
Diseases of: Per cent_
Conjunctiva...........................*....................30.
Cornea....................................................21.
Sclera.....................................................0.4
Iris................................ ....................g
Choroidea................................................. 1 #
Glaucoma................................................. \%
Retina, Optic Nerve, \
Amblyopia, v .................................. 5.
Amaurosis, )
Lens....................................................... 6.
Corpus Vitreum.......................................... 0.7
Globe...................................................... 2.
Refraction,
J-
Accommodation, ) "
Muscles.................................................3.
Fifth Nerve...............................................0.2
Lachrymal Apparatus...................................2.
Orbit...................................................... 0.2
Lids............................................,......... 9.
Various.................................................. 1.5
100.0
Besides the above statistics valuable suggestions maybe gained
by a study of statistics of blindness, and the following tables from
treatises by Magnus are reproduced Avith a feAv comments.
In his treatise on blindness among youth, 1886, Magnus confines
himself to those beloAv 20 years of age, and analyzes the statistics
of 64 European institutions for instruction of the blind. He gathers
3,204 cases of incurable blindness in both eyes, and subdiA7ides
them into, 1, congenital; 2, produced by idiopathic disease of the
eye; 3, produced by injuries; 4, produced by general diseases.
He gives the folloAving fables, pp. 12, 13, 1. c.
I t 4)
DISEASES OF THE EYE.
Amaurosis Congenita.
Anophthalmus....................
Microphthalnius..................
Buphthalmus.....................
Atrophia nervi optici.............
Retinitis pigmentosa.............
Atrophia retinae..................
Choroiditis and choroido-retinitis
Coloboma choroideae.............
Irido-choroiditis.................
Kerato-conus.....................
Keratitis..........................
Albinismus.......................
Glioma retinae....................
Cataracta com plicata congen.. .
Undetermined conditions1.......
Adhesion of lids to globe........
Myopia...........................
Total.
551 =
Per Cent.
17.19
16
81
38
113
73
17
21
3
14
3
1
4
1
118
43
1
4
=0.50
=2.53
= 1.19
=3.53
=2.28
=0.53
=0.66
=0.09
=0.44
=0.09
=0.03
=0.12
=0.03
=3.68
= 1.34
0.03
=0.12
Males.
327
6
43
26
62
40
12
12
3
11
3
1
2
1
78
25
0
2
Per Cent.
=16.32
=0.30
=2.14
= 1.29
=3.09
= 1.99
=0.59
=0.60
=0.15
=0.55
=0.15
=0.05
=0.10
= 0.05
= 3.88
= 1.24
=0.00
=0.10
Females.
Per Cent.
224=18.75
10=0.84
38=3.18
12 = 1.00
51=4.27
33=2.76
5=0.42
9=0.80
0=0 00
3=0.25
0=0.00
0=0.00
2=0.17
0=0.00
40=3.36
18=1.50
1=0.08
2=0.17
Blindness from Idiopathic Diseases of the Eye.
Blennorrhoea neonatorum.......
Blennorrhoea gonorrhoica........
Trachoma....... ...............
Conjunctivitis diphtheritica.....
Conjunctival diseases of undeter-
mined character...............
Keratitis.........................
Iritis...........................
Irido-choroiditis.................
Choroiditis......................
Sublatio retinae..................
Myopia.........................,
Glioma retinae...............
Neuro-retinitis hemorrhagica...,
Atrophia nervi optici...........
Glaucoma....................
Phthisis bulbi essentialis.......
Total.
Males.
Per Cent.
1060=33.08
753=23.50
15= 0.47
42= 1.31
14= 0.44
26= 0.81
15= 0.47
6=
61 =
14=
27=
4=
1 =
1 =
74=
6=
1=
0.19
1.90
0.44
0.84
0.12
0.03
0.03
2.31
0.19
0.03
Per Cent.
626 = 31.16
415 = 20.66
14= 0.70
27= 1.34
6= 0.25
20=
11 =
2=
41=
8=
18=
4=
1 =
1 =
54=
4=
0=
1.00
0.55
0.10
2.04
0.40
0.90
0.19
0.05
0.05
2.69
0.19
0.00
Females.
Per Cent.
434=36.32
338=28.28
1= 0.08
15= 1.26
8= 0.67
6=
4=
4=
20=
6=
9=
0=
0=
0=
20=
2=
1=
0.50
0.33
0.33
1.67
0.50
0.75
0.00
0.00
0.00
1.67
0.17
0.08
Blindness from Injuries.
Total. Males. Females.
Injuries of eve..................... Per Cent. 261 = 8.15 76=2.37 33=1.03 5=0.16 147=4.58 Per Cent. 202 = 10.06 63=3.13 25=1.24 4=0.19 110=5.47 Per Cent. 59=4.94 13—1 09
Operations............... .... 8=0.67 1—0 08
Ophthalmia sympathetica......... 37—3 10
]In the table of amaurosis congenita occurs an error of 10, viz., instead
of " undetermined conditions 53, of which males 35 "; the percentage shows
that the figures should be 43 and 25 respectively. The correction is made in
the text.
STATISTICS OF EYE DISEASES.
Blindness from Constitutional Diseases.
Scrofula ...........................
Syphilis...........................
Brain and its membranes..........
Atrophia nervi optici after hemor-
rhage...........................
Rubeola (Morbilli)..................
Scarlatina.........................
Variola............................
Exanthemata, unknown...........
Typhus...........................
Purpura hemorrhagica............
Orbital phlegmon..................
Pertussis...........................
Cholera........... ................
Febris intermittens................
Lead poisoning....................
Tobacco poisoning.................
Unknown...... ...................
Total. Males. Females.
Per Cent. Per Cent. Per Cent.
1063 = 33.17 686=14.15 377 = 31.54
243=7.58 142 = 7.07 101=8.45
32=1.00 23=1.14 9=0.75
262=8.18 200=9.96 62=5.19
2=0.06 0=0.00 2=0.17
114=3.56 73=3.63 41=3.43
97=3.03 60=2.98 37=3.10
240=7.49 141 = 7.02 99=8.28
14=0.44 9=0.45 5=0.42
32=1.00 20=1.00 12=1.00
1=0.03 1=0.05 0=0.00
1=0.03 1=0.05 0=0.00
4=0.12 1=0.05 3=0.25
1=0.03 1=0.05 0=0.00
1=0.03 1=0.05 0=0.00
2=0.06 1=0.05 1=0.08
1=0.03 1=0.05 0=0.00
16=0.50 11=0.55 5=0.42
Blindness from Unknoavn Causes.
Total. Males. Females.
Per Cent. 269 = 8.40 Per Cent. 168=8.36 Per Cent. 101=8.45
Summary.
Total.
Males.
Females.
Congenital..........
Idiopathic eye disease
Injuries...............
General diseases......
Unknown causes... .
Per Cent.
551= 17.19
1060= 33.08
261= 8.15
1063= 33.17
269= 8.40
3204=100$
Per Cent.
327=16.32
626=31.16
202=10.06
686=34.15
168= 8.36
Per Cent.
224=18.75
434=36.32
59= 4.94
377=31.54
101= 8.45
Again we have 2,528 cases of blindness of both eyes among both
adults and children observed by Schmidt-Rimpler, Stolte, Uhthoff,
Hirschberg, Landesberg, Bremer, Seidelmann, Katz, Magnus:
Congenital Blindness.
Anophthalmus and microphthalnius
Megalophthalmus..................
Cataracta complicata (accreta)......
Choroiditis..........................
Atrophia nervi optici................
Retinitis pigmentosa................
Atrophia retinae.....................
Anomalies of the cornea.............
Tumors..............................
Undetermined.......................
97
No. Per Cent.
27 1.068
11 0.435
3 0.119
4 0.158
19 0.751
19 0.751
2 0.079
5 0.198
1 0.039
6 0.237
DISEASES OF THE EYE.
Blindness from Idiopathic Eye Disease.
Blennorrhoea neonatorum.............
Trachoma and blennorrhoea in adults
Conjunctivitis diphtheritica.........
Cornea, diseases of....................
Irido-choroiditis, cyclitis, iritis.......
Choroiditis myopica.................
Choroiditis, choroido-retinitis.......
Retinitis pigmentosa acquisita.......
Retinitis apoplectica..................
Neuro-retinitis........................
Sublatio retinas.......................
Glaucoma............................
Atrophia nervi optici.................
Tumors of eye and vicinity...........
Undetermined........................
Blindness from Injuries.
Direct injury or wounds.
Unsuccessful operations.
Injuries of the head.....
Sympathetic ophthalmia
Blindness from Constitutional Diseases.
Syphilis..........................................
Blennorrhoea gonorrhoica1........................
Scrofula ........................................
Irido-choroiditis from meningitis................
Atrophia vel neuritis optica cerebralis............
Atrophia optica spinalis..........................
Atrophia vel neuritis optica from haemetemesis..
Atrophia optica from emesis.....................
Atrophia optica from hemorrhoidal hemorrhage
Atrophia optica from erysipelas faciei............
Atrophia optica with insanity....................
Atrophia optica with epilepsy....................
Atrophia optica after dysentery..................
Retinitis nephritica...............................
Typhus...........................................
Rubeola.........................................
Scarlatina........................................
Variola........................................
Unknown exanthemata...........................
Heart disease.....................................
Pregnancy and parturition.......................
Toxic amaurosis..................................
Orbital diseases.............................. ....
No.
12
23
1
36
176
59
10
2
1
2
1
4
2
5
24
16
13
56
6
1
11
1
1
463
Congenital diseases, 97; Idiopathic eye diseases, 1,696
wounds, 272 ; Constitutional diseases, 463. * Total, 2,528.
Injuries and
1 This item ought to come among idiopathic diseases.
STATISTICS OF EYE DISEASES.
775
Among cases of congenital blindness cataract takes the first
place, there being 20.510 of this class. The eye was in many
instances otherAvise defective, such as by absence of the iris, colo-
boma of the iris, etc. In all, the impossibility of securing useful
vision by curing the cataract, indicated accompanying defect in
the percipient structures. That this should be probable, is sug-
gested by the large number of cases belonging to other classes in
which these structures were involved, while the lens happened to
escape. Among the 118 cases of cataract 74 Avere operated on, and
in 44 no attempt was made.
It is understood that not only arrest of development but active
disease during intra-uterine life gi\res rise to cataract.
Microphthalmus was found in 81 cases. In three patients,
Avhile one eyeball was abnormally small, the other eye was not de-
\Teloped at all. In most cases other complications existed, A'iz., in
20 there was cataract, in 12 coloboma iridis, etc. The possibility
of perception of light is not always excluded. I have seen a case
at the N. Y. Ophth. Soc, by whom presented I regret to have for-
gotten, in which both eyes were microphthalmic and vision good.
Nystagmus is common, and such patients often have the trick of
digging their fingers into the orbits to excite phosphenes. The
theory has been advanced that in some instances the condition is
due, not to arrest of development, but to intra-uterine panophthal-
mitis and consequent phthisis bulbi. In some cases the eyes Avere
not larger than peas, and in respect to size there is great variation.
Megalophthalmus as a congenital condition occurs only one-
third as often as microphthalmus, and it has been variously inter-
preted. Regarded by some as the result of uveitis serosa, others
have spoken of it as being sometimes a glaucoma. The details of
the pathological anatomy may be found in a paper by Grahamer.1
Blennorrhoea neonatorum furnishes the largest number of cases
of blindness, both in the table of young persons and among adults;
in the former reaching 23.50 and in the latter 10.80. Among the
latter the number Avould naturally be feAver because they less fre-
quently live in asylums. That the future Avill show a great reduc-
tion in the number of blind persons from this cause, there can be no
doubt, since the early antiseptic proceedings recommended by Crede
and Haussmann both for the child and the mother are being adopted
in public and private practice. In addition to what has been said
already (see p. 333), statistics gathered by Dr. HoAve,2 of Buffalo,
N. Y., lend additional Aveight to the importance of the treatment.
He brings together tAvo groups of cases, in one group 8,798 cases in
1 Graefe^ Archiv f. Oph., xxx., 3, p. 265.
4Trans. Med. Soc. State of Xew York, p. 263, 1889.
776
DISEASES OF THE EYE.
which no precautions Avere taken, and another group of 8,574 cases
in which one drop of a 20 nitrate of silver solution was applied
to the eye. In the former the cases of blennorrhoea reached 8.660,
in the latter it was reduced to 0.6560.
Glaucoma contributes almost 100 of the cases of blindness, and
naturally it scarcely shows among the young subjects.
Sympathetic ophthalmia claims its victims among all ages—
viz., 4.580 among the young and almost the same among the older,
viz., 4.50. The number of blind persons through unsuccessful op-
eration upon adults, viz., 49, were almost all cases of cataract; and
the majority of them, viz., 30, belong to one operator, Katz, of Diis-
seldorf. No explanation is given of the cause or of the method of
operating. «
Further comment on the tables is needless. To such as choose
to study them many fruitful suggestions will occur. Papers which
maybe studied are "Die Blindheit," Magnus, Breslau, 1883; "Die
Jugend-Blindheit," Magnus, Wiesbaden, 1886; "Die Ursachen und
die Verhiitung der Blindheit," Fuchs, Wiesbaden, 1885; also " The
Blind of New York/' by H. S. Oppenheimer, Trans. Am. Oph. Soc,
1891, p. 156, a careful analysis of 572 cases; report of the com-
mittee on causes and prevention of blindness, Trans. Am. Oph. Soc,
1890, p. 531.
BIBLIOGRAPHY.
Treatises.
Mackenzie: Treatise on Diseases of the Eye. London, 1840.
Jaeger: Grauen Staar, etc. Inaug. Dissert., 1844.
Dalrymple: Pathology of the Human Eye. London, 1852.
Sichel, J.: Iconographie Ophthalmologique. Paris, 1852-1859.
Jaeger •. Beitrage zur Pathologie des Auges. Wien, 1856.
Hasner: Beitrage uber Augenheilkunde. Prag, 1860.
Jaeger: Ueber die Einstellung des Dioptrischen Apparates im menschlichen
Auge. Wien, 1861.
Liebreich: Atlas d'Ophthalmoscopie. Paris, 1863.
Schweigger: Vorlesungen tiber den Gebrauch des Augenspiegels. Berlin,
1864.
Donders: Accommodation and Refraction of the Eye. London, 1864.
Helmholtz: Physiologische Optik. Leipzig, 1867.
Lawson: Injuries of the Eye, Orbit, and Eyelids. London, 1867.
Knapp: Die intraocul&ren Geschwtllste. Carlsruhe, 1868.
Albutt: The Ophthalmoscope in Diseases of the Nervous System and of the
Kidneys. London, 1871. t
Anstie: Neuralgia. New York, 1872.
Cohn: Schussverletzungen des Auges. Erlangen, 1872.
Magnus: Ophthalmoscopischer Atlas. Leipzig, 1872.
Hybord: Du Zona Ophthahnique. Paris, 1872.
Mauthner: Vorlesungen tiber die Optischen Fehler der Augen. Wien, 1872.
Stellwag: On the Eye. • Fourth Am. Ed. New York, 1873.
Fick: Handbuch der Physiologic Wien, 1874.
Magnus: Die Sehnervenblutungen. Leipzig, 1874.
Erichsen: On Railway and Other Injuries of the Nervous System. London,
1866; New York, 1875.
GalezoAvski: Traite des Maladies des Yeux. Paris, 1875.
Pagenstecher und Genth: Atlas der pathologischen Anatomie des Augapfels.
Wiesbaden, 1875.
Abadie: Traite des Maladies des Yeux. Paris, 1876.
Bouchut: Ophthalmoscopic M<5dicale. Paris, 1876.
Carter: On the Eye. Edited by Dr. Green. Philadelphia, 1876.
GalezoAvski: Traite^lOphthalmoscopic. Paris. 1876.
Dickinson: Diseases of the Kidney. London, 1877.
KUhne: Untersuchungen aus dem physiologischen Institut der Universitat
Heidelberg, 1877-1880.
Gowers: Medical Opthtalmoscopy. London, 1879.
BIBLIOGRAPHY.
Horner: Krankheiten des Auges im Kindesalter. Handbuch der Kinder-
krankheiten. Gerhardt, 1879.
Klein: Lehrbuch der Augenheilkunde. Wien, 1879.
Landolt: Examination of the Eyes. Translated by Dr. Burnett. Philadel-
phia, 1879.
Perrin et Poncet: Atlas des Maladies profondes de loeil. Paris, 1879.
Rood: Modern Chromatics. New York, 1879.
Sichel flls: Traite d'Ophthalmologie. Paris, 1879.
Yetsch: Ueber den Frtihjahrscatarrh der Conjunctiva. Inaugural Disserta-
tion. Zurich, 1879.
Wecker and Landolt: Traits complet d'Ophthalmologie. Tome premier, pre-
miere partie. Paris, 1879.
Zehender: Lehrbuch der Augenheilkunde. Stuttgart, 1879.
Alt: Lectures on the Human Eye (Histology and Pathology). New York,
1880.
Arlt: Klinische Darstellung der Krankheiten des Auges. Wien, 1881.
Bernhardt. Hirngeschwiilste. Berlin, 1881.
Cohn: Real-Encyclopedia der Heilkunde. Wien, 1880.
Emmert: Auge und Schaedel. Berlin, 1880.
Flint: Practice of Medicine. Philadelphia, 1881.
Gerlach: Beitrage zur Normalen Anatomie des menschlichen Auges. Leip-
zig, 1880.
Graefe and Saemisch (abbreviated to G. and S.): Handbuch der gesammten
Augenheilkunde, in 7 vols. Leipzig, 1880. (The names of the contribu-
tors are Profs. Arlt, Arnold, Jr., Aubert, Becker, Berlin, Fdrster, Alfred
Graefe, Hirsch, Iwanoff, Landolt, Leber, Leuckart, Manz, Merkel, Michel,
Nagel, Saemisch, Sattler, Schirmer, Schmidt, Snellen, Schwalbe, Wal-
deyer, v. Wecker.)
Jeffries: Color blindness. Boston, 1880.
Le Conte ■. Sight. New York. 1881.
Meyer: Maladies des Yeux. Paris, 1880.
Munk: Ueber die Functionen der Grosshirnrinde. Berlin, 1881.
Nagel: Mittheilungen aus der Ophthalmiatrischen Klinik in Tubingen. 1880.
Nettleship: Diseases of the Eye. Am. Ed. Philadelphia, 1880.
A. Robin: Des Troubles oculaires dans les Maladies de l'Encephale. Paris,
1880.
Schweigger: Handbuch der Augenheilkunde. Fourth Edition. Berlin, 1880.
Soelberg Wells . On the Eye. Edited by Dr. Bull. - Philadelphia, 1880.
Wilbrand: Hemianopsia. Berlin, 1881.
Fuchs. Das Sarcom des Uvealtractus. Wiesbaden, 1882.
Haussmann: Die Bindehaut-Infection der Neugeborenen. Stuttgart, 1882.
Moore: Funf Lustren Ophthalmologischer Wirksamkeit. Wiesbaden, 1882.
Rothmund: Mittheilungen aus der Klinik zur Munehen. 1882.
Magnus: Die Blindheit. Breslau, 1883.
Ross • Diseases of the Nervous System. New York, 1883.
Wernicke: Lehrbuch der Gehirnkrankheiten. Cassel. 1881-1883.
Aschman: Wounds of the Optic Nerve. Inaug. Thesis. Zurich, 1884.
Bull: The Ophthalmoscope and Lues. Christiana, 1884.
Juler: Handbook of Ophthalmic Science and Practice. London 1884.
Meynert: Psychiatric Wien, 1884.
Michel • Lehrbuch der Augenheilkunde. Wiesbaden, 1884.
Schmidt-Rimpler. Augenheilkunde und Ophthalmoscopie. Braunschweig,
1884. Am. Ed., Wm. Wood & Co., 1889.
BIBLIOGRAPHY.
779
Swanzy: Handbook of Diseases of the Eye. London, 1884.
Walter: Klinische Studien tiber Netzhautablosungen. Inaug. Dissert. Zu-
rich, 1884.
Edinger: Zehn Vorlesungen tiber den Bau der nervOsen Centralorganc
Leipzig, 1885.
Fuchs: Die Ursachen und die Verhtltung der Blindheit. Wiesbaden, 1885.
Ziegler: Haab: Pathologische Anatomie des Auges. Jena, 1885.
Landolt: The Refraction and Accommodation of the Eye. Translated by
Culver. Edinburgh, 1886.
Schwalbe: Anatomie der Sinnesorganc Erlangen, 1886.
Loring: Text-book of Ophthalmoscopy. Parti. New York, 1886.
Magnus: Die Jugendblindheit. Wiesbaden, 1886.
Da Gama Pinto: Untersuchungen tiber intraoculare Tumoren. Wiesbaden,
1886.
Wedl und Bock: Pathologische Anatomie des Auges. Atlas. Wien, 1886.
Bunge: Ueber Exenteration des Auges. Halle, 1887.
Gowers: Diseases of the Brain. Philadelphia, 1887.
Nordenson: Die NetzhautablOsung. Wiesbaden, 1887.
Stilling: Untersuchungen tiber die Entstehung der Kurzsichtigkeit. Wies-
baden, 1887.
Herzog Karl Theodor: Ein Beitrag zur pathologischen Anatomie des Auges.
Miinchen, 1887.
Wilbrand: Die Seelenblindheit. Wiesbaden, 1887.
Alexander: Syphilis und Auge. Wiesbaden, 1888.
Gowers: Diseases of the Nervous System. Phila., 1888.
Obersteiner: Anleitung beim Studium des Baues der nervOsen Centralorganc
AVien, 1888.
Vossius: Grundriss der Augenheilkunde. Leipzig und Wien, 1888.
Fuchs: Lehrbuch der Augenheilkunde. Leipzig und Wien, 1889.
Magnus: Die Entstehung der reflectorischen Pupillenbewegung. Breslau,
1889.
Mauthner: Vortrage der Augenheilkunde. Wiesbaden, 1879-1889.
Wilbrand: Die hemianopische Gesichtsfeld-Formen und das optische Wahr-
nehmungszentrum, Atlas, Wiesbaden, 1890.
Berger und Tyrman: Die Krankheiten der Keilbein-Hohle und des Sieb-
bein-Labyrinthes, etc., Wiesbaden, 1886.
M.Foster: Text-book of Physiology. 5th edition. Macmillan, London, 189L
Dana : Text-book of Nervous Diseases. Wm. Wood & Co., 1892.
Ferrier : The Localization of Cerebral Disease. 1879.
M. P. Jacobi: Hysteria and Brain Tumors. Putnam Sons, New York, 1888.
Berger : Anatomie Normal et Pathologique de TCEil. Paris, 1893.
Charcot: Clinique des Maladies du Systeme JNerveux, T. 1. New York, 1892.
Reference Handbook of the Medical Sciences. Vols. I. to IX. Wm. Wood
& Co., 1886-1893.
Starr : Brain Surgery. Wm. Wood & Co., 1893.
Gray : Nervous and Mental Diseases. Lea Brothers & Co., Philadelphia, 1893.
Berger: Les Maladies des Yeux dans leurs Rapports avec le Pathologic
Generate. Paris, 1892.
De Schweinitz : Diseases of the Eye. W. B. Saunders, Philadelphia, 1892.
Herter : The Diagnosis of Diseases of the Nervous System. 1892.
Knies : Das Sehorgan und seine Erkrankungen. Wiesbaden, 1893.
Cohn : Uterus und Auge. Wiesbaden. 1890.
Norris and Oliver : A Text-book of Ophthalmology. Lea Brothers & Co.,
Philadelphia, 1893.
780
DISEASES OF THE EYE.
Wilbrand: Ophthalmiatrische Beitrage zur Diagnostik der Gehirn-Krank-
heiten. Wiesbaden, 1884.
Wilbrand and Saenger : Ueber SehstSrungen bei functionellen Nerven-
leiden. Leipzig, 1892.
Cohn : Lehrbuch der Hygiene des Auges. Wien und Leipzig, 1892.
Henschen : Klinische und Anatomische Beitrage zur Pathologie des Gehirns.
Erster und zweiter Theilen. Upsala, 1890, 1892.
Fischer: Ueber die Embolie des Arteria Centralis Retinae. Leipzig, 1891.
Journals, Reports, Transactions, etc
A. v. Graefe's Archiv fur Ophthalmologic Berlin.
Annales d'Oculistiaue. Brussels, Belgium.
Klinische Monatsblatter fur Augenheilkunde. Zehender. Stuttgart.
Centralblatt ftir Augenheilkunde. Hirschberg. Leipzig.
Archives of Ophthalmology. Knapp and Hirschberg. New York and Wies-
baden.
Archives d'Ophthalmologie. Panas, Landolt, Poncet. Paris.
Transactions of the American Ophthalmological Society. New York.
Ophthalmic Hospital Reports. London.
Brain: A Journal of Neurology. London.
American Journal of Medical Sciences. Lea & Co. Philadelphia.
Medical Record. Wm. Wood & Co. New York.
Medical Journal. D. Appleton & Co. New York.
Jahresbericht der Augenheilkunde. Nagel, Vols. I to VII., 1870-1876. Mi-
chel, Vols. VIII. to XVIII., 1877-1888.
Trans, of the Fifth International Med. Congress, Phila., 1877.
Medical News, Philadelphia.
American Practitioner.
Zeitschrift ftir Heilkunde.
Journal of Nervous and Mental Disease. New York.
American Journal of Ophthalmology. St. Louis.
Ophthalmic RevieAv. Manchester, Eng.
St. Thomas Hospital Reports. London.
Transactions of the Ophthalmological Society of the United Kingdom.
Churchill. London.
Recueil d'Ophthalmologie. Paris.
Brooklyn Medical Journal. Brooklyn, N. Y.
Bericht der Ophthalmologischen Versammlung zu Heidelberg.
Boston Medical and Surgical Journal. Boston.
Transactions of the Medical Society of the State of New York. Albanv
N. Y.
Birmingham Medical RevieAv. Birmingham, Eng.
Bulletins et Memoires de la Societe1 Francaise d'Ophthalmologie.
Medico-Chirurgical Transactions. London.
Archiv ftir Psychiatrie.
Berliner klinische Wochenschrift. Berlin.
British Medical Journal.
Medical Press and Circular. London.
Trans. Am. Med. Association. Chicago, 111.
The Ophthalmic Record. Nashville, Tenn.
Annals of Ophthalmology and Otology. St. Louis. Mo.
The Review of Insanity and Nervous Disease. Milwaukee Wis.
Many books and treatises and other sources of information are referred to
in the text which are omitted in the above lists.
INDEX.
Abducens, nucleus of, 155
Abscess, cerebral, 163, 671
intra-cranial, 726
of antrum, 726
of brain, 163, 671
of ethmoid, 726
of frontal sinus, 726
of lachrymal sac, 295
of orbit, 731, 732
pre-lachrymal, 306
Abrine, 355
Abrus precatorius, 354
Accommodation, 16 et seq., 61
and convergence, relation between,
85
binocular, 18
changes in crystalline lens in, 17
errors of, 61
mechanism of, 16 et seq.
monocular, 18
paralysis of, 66
paresis of, 66
range of, 17 et seq.
relative, 18 et seq., 138
spasm of (see Spasm of accommo-
dation)
table showing range of, at different
ages, 18
Acetate of lead, 234
in blepharitis, 253
Actual cautery, in corneal inflamma-
tion, 233
in hypopyum keratitis, 399,
401
in trachoma, 233
Acuity of vision, 21, 24, 37, 70
improved by glasses, 90
peripheral, 26
Adenoma of Meibomian glands, 260
Adenoid tissue of conjunctiva, 309
Advancement, capsular, 224
in muscular asthenopia, 225, 229
in ocular paralysis, 167 et seq.
in strabismus, 190
of ocular muscles, 169 et seq.
author's method, 169 et seq.,
229, 230
various methods, 169
of Tenon's capsule, 190
versus tenotomy, 196
After-images, 32
Agraphia, 656, 657, 703, 714
Albinism, 574
Albugo, 378, 411
Albuminuria, 612, 672, 716
of pregnancy, 613, 620, 621, 635
induction of labor in, 622
Alcoholic amblyopia, defective color
sense in, 28
Alcoholism, 66
Alexia, 656, 703, 714
Alternating anaesthesia, 149
motor paralysis, 149
Amaurosis (see Amblyopia)
congenita, statistical tables, 772,
773
due to fifth nerve injury, 754
fugax, 702
in young children, 718
Amblyopia, 694 et seq.
alcoholic, 28, 678, 682 et seq.
central, 181
congenital, 609, 699
crossed, 645, 714
ex abusu, 28, 678, 682 et seq.
ex anopsia, 699
following orbital fracture, 695, 757,
759
from general hemorrhage, 698
from irritation of teeth, 704
from lead, 700
from osmic acid, 699
from quinine, 700
from salicylic acid, 700
from suppression of menstruation,
701
from tobacco, 28, 435, 678, 682 et
seq.
glycosuric, 700
hysterical, 681, 701
in spinal cord concussion, 696, 697
medial, 181
monocular, congenital, 181 et seq.
in strabismus convergens, 180,
181
without squint, 181 et seq.
of diabetes, 622
of epilepsy, 701
of pregnancy, 701
toxic, 662, 678, 682 et seq., 699
traumatic, 694 et seq.
treatment of, 705
uraemic, 700
varieties of, 694
without ophthalmoscopic lesions,
732, 759
782
INDEX.
Ametropia, 69, 129
Amimia, 656, 657
Amnesia, 714
Amotio retinae, 630
Amyloid degeneration of conjunctiva,
357, 362
tumors, 260
Anaemia, 66, 208
Anaesthesia, alternating, 149
of cornea in glaucoma, 551, 559
Anaesthetics, 235 et seq.
in examination of eyes, 37
Anatomy of base of brain, 146 et seq.
of brain, 145 et seq., 653 et seq.
of choroid, 573
of conjunctiva, 247 et seq., 308 et
seq.
of cornea, 371 et seq.
of cranium, 145
of crystalline lens, 471
of eyelid, 247 et seq.
of frontal bone, 145
of globe, 1
of iris, 430 et seq.
of lachrymal apparatus, 292 et seq.
of medulla, 147 et seq.
of ocular muscles, 132 et seq.
of optic nerve, 641 et seq.
of optic nerve, compared with
ophthalmoscopic picture, 48
of orbit, 144, 249, 722 et seq.
of pons, 147 et seq.
of retina, minute, 594 et seq.
of sclera, 424
of visual path (see Hemianopia)
of vitreous body, 512
Ankyloblepharon, 291
Aneurism, 163, 437
orbital, 744, 746
Angioma of choroid, 593
of conjunctiva. 357, 360
of orbit, 735, 736, 744
Angle alpha, 14, 15, 114, 178
in hyperopia, 83
gamma, 14, 114, 178
in hyperopia, 83
in myopia, 102
of iris, 430
secretory, 8
Aniridia with glaucoma, 562
Anisometropia, 86, 101, 128 et seq.
Anophthalmia, atrophy of visual ap-
paratus in, 646
congenital, 646
Anterior chamber, 2, 371, 430
in glaucoma, 54, 551
ciliarv arteries, 7, 573
perforated space, 147, 641, 642, 644
principal focus, 11
pyramid of medulla, 146
Antero-posterior diameter of globe, 1
Antimetropia, 129
Antisepsis, 233
Antiseptics in operative work, 234 et
seq.
Antrum, tumors of, 736, 750
Aphasia, 175, 714
mixed, 657
motor, 656, 657
sensory, 656
Aphemia, 656, 657
Apoplexy, 435, 606, 715
Apraxia, 649, 656
Aqueductus Sylvii, 146, 147, 150, 152,
641
Aqueous chamber, 2
humor, 2
derivation of, 4
effects of section of cervical
sympathetic on secretion of, 9
Aquo-capsulitis, 442
Arciform fibres, 148
Arcus senilis, 374, 410
lentis, 473
Area of Martegiani, 512
Argyll-Robertson pupil, 162, 435, 688
Argyria conjunctivae, 318
Aristol, 234
Arteria centralis retinae, 6
embolism of, 601
Arterial pulsation, 598
in glaucoma, 549, 554
Arteries, anterior ciliary, 7, 424
at base of brain, 145
long ciliary, 7, 424, 573
posterior ciliary, 6, 424
short ciliary, 6, 424
Arterio-sclerosis, general, 620
Arteritis obliterans, 163
Artery, hyaloid, 8
internal carotid, 145
Artificial eyes, use of, 768 et seq.
pupil, in corneal opacity, 412
vitreous, 418, 545, 769
Asepsis, 233
Asthenopia, 139, 197 et seq.
accommodative, 85, 88, 197
due to nasal catarrh, 217
in anisometropia, 129
in myopia, 94
muscular, 197, 202 et seq.
cases of, 227 et seq.
exciting causes of, 208 et seq.
general considerations, 206 et
seq.
in myopia, 108
myotic, 207
neurotic, 207
objective symptoms, 209 et seq.
operations for, 224 et seq.
prisms in, 223
refraction in, 221 et seq.
spasm of muscles in, 225, 227
subjective symptoms, 209
treatment of, 219 et seq.
without refractive error, 221
neurasthenic, 197
reflex, 197
subdivisions of, 197
Asthenopic symptoms in astigmatism,
127
in hyperopia, 87
INDEX. 783
Astigmatism, 69, 111 et seq., 375
acquired, 111
against the rule, 114
apparatus for demonstrating, 113
caused by pterygium, 370
compound, 112, 114
hyperopic, 112
myopic, 112
congenital, 111
correction of, 73 et seq.
diagnosis of, 118 et seq.
diagrams illustrating, 113
due to pressure of lids, 125
following cataract extraction, 502
Green's tests for, 118 et seq.
hvperopic, 112
irregular, 111, 126
lenticular, 114
in cataract, 480
mathematical theory of, 113
methods of testing, 121 et seq.
mixed, 112, 115
myopic, 112
ophthalmoscopic picture in, 73 et
seq.
Pray's test types for, 121
regular, 111 et seq.
simple, 112, 114
symptoms of, 117
varieties of, 112
Asymmetry of skull, 724
Atrophic ring in myopia, 99
Atropia, 240, 241
in asthenopia, 198
in astigmatism, 120 et seq.
in castor oil, 240
in correction of errors of refrac-
tion, 70 et seq.
in hyperopia, 84, 86, 89
in muscular asthenopia, 219
in myopia, 106, 107
in spasm of accommodation, 65
in strabismus, 186, 187
toxic effects of, 240 et seq., 446, 447
Atropine conjunctivitis, 314
Axis of cornea, 14, 178
of globe, 1, 14
Bandages, 231, 232
Basal paralysis of ocular muscles, 158
Basedow's disease, 163, 251, 764 et seq.
treatment of, 766 et seq.
Base of brain, anatomy of, 146 et seq.
Benzoate of sodium, 233
Bident, Agnew's, for dislocated lens,
476
Binocular accommodation, 18
fusion, cerebral element in, 195
vision, 132 et seq.
conditions of, 135 et seq.
in anisometropia, 129
limitations of, 138
Blenorrhua, 319
chronic, 340, 350, 351
neonatorum, 775
Blepharitis marginalia, 252,256
Blepharo-adenitis, 252
Blepharophimosis, 276, 278
Blepharoplasty, 271 et seq.
author's methods, 272 et seq.
Blepharospasm, 155, 281
section of supra-orbital nerve& in.
695
Blind headache, 599
spot, 20, 181
size of, 26
Blindness caused by pressure on globe,
695
cerebral, 694
congenital, 775
statistical tables, 772, 773
mental. 694. 703
mind, 656, 714
monocular, undiscovered, 718
psychic, 649, 654, 655, 656, 657, 703
simulated, 719
methods of detecting, 720, 721
uraemic, 616
Blisters, 244
in ocular paralysis, 166
Blood-vessels, coumnctival, 7
of retina, 49
Blue light after cataract extraction,
31
Boiled water in operative work, 234
Bony growths in conjunctiva, 357
Borax, 233
Boric acid, 233
in operative work, 234
Bowman's director, 299, 300
membrane, 371, 372
Braehium, 150, 643, 646
anticum, 644, 646
posticum, 645, 646
Brain, abscess of, 671
anatomy of, 145 et seq., 653 et seq.
arteries at base of, 145
diseases, 692
lesions, 688 (see also Hemianopia)
localizing areas of, 654 et seq.
physiology of, 653 et seq.
tumor, 159, 163, 664, 665, 670, 671,
672, 675, ($76, 677, 678, 689, 715
(see also Hemianopia)
Broca's convolution, 657, 703
Bromide of potash before operations,
238
Briicke's dissecting spectacles, 215
lenses, 36
Bulbar paralysis, 155, 162
Buphthalmus, 553
Burns of eye, 287 et seq., 529, 5S0
Calabar bean, 241
Calcareous deposit in conjunctiva, 357,
414
in cornea, 414
Calcarine fissure, 647, 649
Canal of Cloquet, 8, 512, 558
of Fontana, 4
of Petit, 8, 462, 472, 512
hemorrhage into, 513
784
INDEX.
Canal of Schlemm,. 4, 6, 8, 373, 463, 558,
568
of Sylvius, 146, 147
Canaliculi, 292
chalky concretions in, 294
double, 294
leptothrix in, 294
occlusion of, 294
slitting of, 299
stricture of, 294
Canalis opticus, fracture of, 757, 759
section of skull through, 758
Cancer of cornea., 423
Cancerous disease of conjunctiva, 357,
359
Canities, 261
Cantholysis, 278, 388
in trachoma, 354, 357
Canthoplasty, 266. 276, 278, 388
in trachoma, 356, 357
Canthotomy, 265
in purulent conjunctivitis, 329
in trachoma, 354
Capsular advancement, 224
Capsule of lens, 2
of Tenon, 1, 168, 169, 424
Capsulitis in iritis, 445
Capsulotomy in cataract extraction, 499
peripheral, 501
Carbolic acid as a disinfectant, 233,
235
Cardinal points, 11, 12
table of, 13
Caries in dacryocystitis, 295, 296, 305,
306
of cranial bones, 584
Caruncle, 169, 247, 308
retraction of, 189, 190
Caruncula lachrymalis (see Caruncle)
Cataract, 478 et seq.
absorption of, 489, 490
anterior polar, 478, 482
artificial ripening of, 494, 495
black, 481
capsular, 478, 501
capsulo-lenticular, 478
causes of, 479
complicated, 478, 480, 486
complications in, 485
congenital, 478, 479, 480, 482, 483,
484, 490, 491, 775
diabetic, 479, 488
diagnosis of, 484 et seq.
discission of, 490, 491
disturbance of vision in, 480
electricity in, 488
extraction, 234 et seq., 489, 492 et
seq.
after-treatment in, 504 et seq.
antisepsis in, 496
astigmatism following, 502
complications following, 506
et seq.
danger of too small wound in,
499
delirium after, 232
Cataract extraction, entanglement of
capsule after, 503
Graefe's linear, 495
incarceration of iris after, 502
instruments for, 238 et seq.,
503 et seq.
intra-ocular hemorrhage after,
489
iridectomy in, 499
iritis following, 506, 507
Lebrun's incision in, 495
Liebreich's incision in, 495
prolapse of iris after, 502, 505
purulent infiltration of wound
after, 506
removal of capsule in, 501
Ring's mask after, 505
simple, 501 et seq.
simple linear, 492, 493
statistics of, 503
striped keratitis following, 507
toilet of eye after, 500
use of syringe in, 496, 500
various sections in, 496
fluid, 478
focal illumination of, 481
following iridectomy, 567
galvanism in, 488
glasses, 58, 510, 511
hard, 478, 479, 480, 495
hypermature, 479, 480
idiopathic, 480, 481
incipient, 481
inflammatory, 478, 479
in glaucoma, 551
in myopia, 101
juvenile, 478
lamellar (laminated), 474, 480, 482,
490, 492
lenticular, 478
astigmatism in, 480
light preception in, 485
massage in, 488
membranous (see secondary)
mixed, 480
Morgagnian, 479, 480
myopia produced by, 480
objective symptoms in, 480 et seq.
panophthalmitis following discis-
sion of, 491
partial, 478, 480
pathology of, 478 et seq.
percentage of success in operations
for, 488
posterior polar, 480, 482
prognosis in, 486
progressive, 480
rate of progress of, 480, 487
reclination (couching) of, 490
ripe, 480
secondary, 507 et seq.
complicated, 508, 509
senile, 478, 480, 481, 485
simple, 480
soft, 478, 479, 480, 486, 492
spontaneous cure of, 488
INDEX. 785
Cataract, spontaneous improvement of
vision in, 487
spontaneous luxation of lens in,
488
stage of irritation in, 487
subjective symptoms in, 480
symptoms of, 480 et seq.
traumatic, 478, 480, 482, 520, 522
zonular, 474, 480, 482, 490, 492
Catarrho-rheumatic ophthalmia, 314
treatment of, 318
Catoptric test, 474
Cauterization in conical cornea, 422
in trachoma, 350, 354, 356
Cavernous sinus, 144, 145
rupture of carotid in, 746
thrombosis of, 730, 732, 733,744
tumors of orbit, 735, 736, 744
Cellulitis, orbital, 729 et seq.
Centre of motion, 14
Cerebellum, 146, 658
lesions of, 658
peduncles of, 147
tumor of, 671, 675, 709
Cerebral apoplexy, 435, 606
blindness, 694
embolism, 435
hemorrhage, 163, 609
localization, 156, 157, 653 et seq.,
675
tumor (see Brain tumor)
Cerebro spinal meningitis, 435, 514,
516, 584, 585, 683
Cervical sympathetic (see Sympathetic,
cervical)
Chalazion, 254
forceps, 254
Chalky concretions in canaliculi, 294
Chamber, anterior, 2
aqueous, 2
posterior, 2
Chancre of conjunctiva, 358
of evelid. 259, 358
Chemosis, 327, 401
Cherry red spot, 661
in embolism of central retinal
artery, 602, 604
Cheyne-Stokes respiration, 438
Chiasm, optic, 145, 147, 641, 642, 643,
644, 646, 654, 715
arrangement of fibres in, 643,
652
Chloroform anaesthesia, 237
Chlorosis, 672, 677
Choked disc, 663, 674
Cholera, 600, 660
Cholesterin in vitreous, 513
Chondroma of choroid, 593
Chorio-capillaris, 559, 573
Choroid, 3, 5
anatomy of, 573
colloid degeneration of, 50, 575,
577, 616
coloboma of, 574
detachment of, 588
embolism in, 576, 584
50
Choroid, hemorrhages of, 576, 587
laceration of, 587
ossification of, 593
rupture of, 520
sarcoma of, 592, 593, 632, 639
supratraction of, 97 et seq.
tubercle of, 578, 588
tumor of, 589 et seq.
vessels of, 52
visibility of, 52
Choroidal atrophy, 578, 579
senile, 582
changes in myopia, 581 et seq.
crescent, 96 et seq., 105
in myopia, 96 et seq., 105
mode of formation of, 97
ring in glaucoma, 559
Choroidea (see Choroid)
Choroiditis, 575 et seq.
acute, 580
chronic, 580
circumscripta, 580
connective-tissue changes in, 577,
578, 580
disseminata, 579, 581, 624
exudative, 578
guttate, 580
hemorrhagic, 576
Hutchinson's classification of, 583
in myopia, 95
metastatic, 514, 584, 585
pathology of, 575 et seq.
prognosis in, 581
proliferation of pigment in, 577
suppurative, 584
symptoms of, 578 et seq.
syphilitic, 577, 579, 580
treatment of, 583
vascular changes in, 577
visual acuity in, 581
Cilia, 248
ensnaring of, 262
Ciliary body, 3, 5, 462 et seq.
glands of, 464
gumma of, 467
morbid growths in, 470
pars non plicata, 5
pars plicata, 5
ganglion, 17, 424, 432, 464, 724
muscle, 6, 462, 463
Ciliary muscle, action of, 6
in myopia, 100
mode of action of, 16
structure of, 63
nerves, 17, 464, 574
regeneration of, 542
processes, 3, 5, 462
region, wounds of, 521, 522
vessels, 463
Cilio-retinal vessels, 49
Cilio-spinal centre, 163
Circle of Haller, 6, 573
of Willis, 145
Circular venous*sinus, 4
Circum-corneal hypertrophy of con-
junctiva, 362, 363
'786
INDEX.
Citrine ointment, 256
in blepharitis, 252
Cloquet's canal, 8, 512, 558
Cocaine, 235 et seq., 240, 243
antidotes to, 237
corneal bleb produced by, 236
effect of, on tension, 236
in determining errors of refraction.
71
in facial neuralgia, 237
in glaucoma, 236
in hyperopia, 89
in operative work, 236
in tenotomy, 188, 224, 226
summary of effects of, 235 et seq.
toxic effects of, 237
with atropine, 237
with eserine, 237
Cold applications, 244
in iritis, 448
in purulent conjunctivitis, 323
douches in muscular asthenopia,
220
Collodion and cotton dressing, 266
Colloid bodies in optic papilla, 669
degeneration of choroid, 50, 575,
577, 616
Collyria, 245
Coloboma lentis, 473, 474
of choroid, 574
of eyelids, 280
of iris, 439, 775
of macula lutea, 105, 597
of sheath of optic nerve, 105, 658
Color-blindness, 28 et seq., 700
heredity in, 28, 29
methods of testing, 29, 30
Color chart, 27
fields, 27 et seq.
half-blindness, 712
perception in glaucoma, 549, 550
sense, 27 et seq.
congenital defect of, 28
in optic nerve atrophy, 686, 689
in optic neuritis, 667
Colored rings in g'aucoma, 551, 559
Columns of Burdach, 148
of Goll, 148
Commissure of Gudden, 643, 644, 645,
646, 647, 652, 715
of Meynert, 643, 644, 645, 646, 652,
657, 715
Conarium, 148
Condyloma of iris, 443
Congenital paralysis of ocular muscles,
173
Conical cornea,, 126, 375, 419 et seq., 461
Conjugate foci, 15
deviation, 161, 165, 175, 654, 655
cases of, 175, 176
movements of eyes, 157
Conjunctiva, 1, 247 et seq., 308 et seq.
adenoid tissue of, 309
amyloid degeneration of, 357, 362
anatomy of, 247 et seq., 308 et seq.
angioma of, 357, 360
Conjunctiva, blood-vessels of, 309, 310
bony growths in, 357
calcareous deposits in, 357
cancerous disease of, 357, 359
chancre of, 358
circum-corneal hypertrophy of,
362, 363
congenital fibrous growths in, 357,
359
cystoid growths in, 357, 359
dermoid growths in, 359
diseases of, 310 et seq.
epithelial disease of, 357, 359
epithelium of, 309
foreign bodies in, 367
foreign bodies on, 523
granuloma of, 358, 360
gummy tumor of, 358
herpes of, 363, 366
hyaline degeneration of, 343
hypertrophy of, 325, 331
laceration of, 520
lupoid disease of, 357, 358, 359
lymphoid tissue of, 309
mucous patches on, 358
nerves of, 310
nitrate of silver staining of, 318
papilloma of, 357, 358
pemphigus of, 362, 366
physiology of, 308 et seq.
pigment patches in, 357, 360
sarcoma of, 359
syphilitic lesions of, 357, 358
tubercular deposits in, 357, 358
vernal catarrh of, 362, 363
xerosis of, 360 et seq.
Conjunctival diseases, frequency of,
310
sac, germs in, 234
vessels, 7
Conjunctivitis, 311 et seq.
amyloid, 343
catarrhalis, 313 et seq.
complications of, 315
duration of, 315
sequelae of, 315
treatment of, 316 et seq.
croupous, 332 et seq.
diphtheritic, 332 et seq.
due to atropine, 314
follicular, 314, 340
granular (see Trachoma)
in eczema, 363
metastatic, in gonorrhoea, 315
cedematosa, 313 et seq.
papillary, 314
phlyctenular, 363, 364
plastic, 332 et seq.
treatment of, 336 et seq.
purulent, 319, 325 et seq. J
contagious nature of, 326
corneal complications in, 327,
330, 331
grattage in, 330, 331
protection of fellow eye in, 332
symptoms of, 327
INDEX.
787
Conjunctivitis, purulent, treatment of,
328 et seq.
vaseline treatment of, 328
vertical division of lid in, 329
secondary, 315
simplex, 313 et seq.
subdivisions of, 311
symptomatic, 315
syphilitic, 343
traumatic, 367
tuberculous, 343
Contusions of globe, 519
Conus in myopia, 96
Convergence, amplitude of, 85
Coquilles, 231
Corelysis, 449, 454, 461
Cornea, 1, 2, 35
actual cautery in ulceration of, 233
alteration of curve of, by ptery-
gium, 370
anaesthesia of, in glaucoma, 551,
559
anatomy of, 371 et seq.
angle of aperture of, 374
anterior elastic lamina of, 371, 372
anterior principal focus of, 12
axis of, 14, 178
blunted sensibility of, in malarial
keratitis, 388
calcareous deposit in, 414
canals of, 372, 373
cancerous ulceration of, 423
cell proliferation of, 375
conical, 375, 419 et seq., 461
operative treatment of, 421 et
seq.
curetting of, in trachoma, 354
in vascular keratitis, 388
curve of, 14
divisions of, 371
eczema of, 379 et seq.
epithelioma of, 423
epithelium of, 372
fibroma of, 423
fistula, of, 406, 411, 414
focal distance of, 374
foreign bodies in, 523
herpes of, 383
index of refraction of, 10, 374
inflammation of, 375 et seq. (see
Keratitis)
irregular astigmatism of margin
of, 412
lacunae of, 372
layers of, 371
limbus of, 3, 371
lymph channels of, 8
marginal vessels of, 371
morbid growths of, 423
nerves of, 374
nodal point of, 12
nutrition of, 373, 374
opacities of, 377, 378. 411 et seq.
detection of, by ophthalmo-
meter, 126
optical centre of, 12
Cornea, papilloma of, 423
paracentesis of, 426
parenchyma of, 372
pathology of, 375 et seq.
pemphigus of, 385
perforation of, 405
in purulent conjunctivitis, 324,
327, 331
physiology of, 374 et seq.
posterior elastic lamina of, 371, 373,
377
posterior principal focus of, 12
radius of curve of, 371
refractive index of, 10, 374
ring ulcer of, 406
sarcoma of, 423
sclerosis of, 410, 427
sensibility of, 36
sloughing of, in plastic conjunc-
tivitis, 337
staphyloma of, 411, 414
tattooing of, 412, 413, 461
transplantation of, 422
tubercular disease of, 423
ulceration of, 376, 377, 394, 405 et
seq.
wounds of, 521
Corneal corpuscles, 372, 373
irregularity, after trachoma, 357
opacity, methods of detecting, 126,
377, 378
Corpora albicantia (mammillaria), 145,
642
candicantia (mammillaria), 147,
641, 642
geniculata, 145, 147, 645, 715
mammillaria (candicantia), 145,
147, 641, 642, 643, 644
quadrigemina, 146, 147, 641, 643,
644, 645, 646, 652, 657, 715
quadrigemina, lesions of, 164
quadrigemina, section through, 150
Corpus ciliare, 462
geniculatum externum (laterale),
641, 642, 643, 644, 646, 657, 715
geniculatum internum (mediale),
150, 641, 642, 643, 645, 657
striatum, 657
subthalamicum, 146, 150
vitreum, 2, 512
Corrosive sublimate as a disinfectant,
233
in operative work, 234
strong solutions of, in purr
lent conjunctivitis, 330
strong solutions of, in trcv
choma, 351 et seq.
Cortical areas of localization, 156, 15?
paralvsis of ocular muscles, 164,
165
Crab's-eyes, 367
Cranial nerves, origins of, 152
relations of, at base of brain,
152
Cranium, anatomy of, 145
Cretes' prism, 210
788
INDEX.
Crossed cylinders, 118, 128
paralysis of third nerve, 159
Croupous and diphtheritic inflamma-
tion, 334
Crura cerebri, 145, 146
Crusta, 146, 147
Crystalline lens (see Lens)
Cul-de-sac, 308
Cuneus, 646, 647, 648, 649, 715
Cupping in ocular paralysis, 166
Curetting of cornea in vascular kerati-
tis, 388
Cyclitis, 407, 464 et seq.
plastic, 465, 467
prognosis in, 468
purulent, 467
sero-fibrinous, 465
serous, 442, 466
acute, 466
symptoms of, 466
traumatic, 468
treatment of, 468
varieties of, 466
Cylindrical glasses, 53, 54
Cysticercus cellulosae, 514, 518
in anterior chamber, 543
in orbit, 735
in vitreous, 514, 518
Cystic tumors of lids, 254
Cystitome, 499
Cystoid cicatrix after iridectomy, 567
Cysts of conjunctiva, 357, 359
of iris, 452
of orbit, 735
Dacryocystitis, 294 et seq., 315
caries in, 295, 296, 305, 306
causes of, 298
diagnosis of, 297
phlegmonous, 295
prognosis in, 298
symptoms of, 295, 296
treatment of, 298 et seq.
use of probes in, 299 et seq.
Daltonism, 28
Daturia, 240, 241, 243
Decentration of lenses, 202
Decussation of fibres of nuclei of third
nerve, 154
of Forel, 150
of Meynert, 150
of pyramids, 146
Delirium after cataract extraction,
232
Dermoid growths of conjunctiva, 359
Descemet's membrane, 371, 373, 377
Descemitis, 407, 442, 575
Detachment of choroid, 588
of retina, 630 et seq.
in myopia, 101
pathogenesis of, 632
prognosis in, 634
spontaneous recovery in, 634
treatment of, 634 et seq.
Deviation of eyes as a diagnostic fea-
ture, 165
Diabetes, 66, 67, 164, 235, 445, 487, 612,
622, 683, 684, 689, 716
mellitus, 163, 479
due to lesion of fourth ven-
tricle, 150
Diachylon ointment, 253
in eczema of lid, 256
Diagrammatic eye of Donders, 15
Dialysis of iris, 520
Dilator pupillae, 431
Dioptric interval, 56
series, equivalent in inches, 57
Dioptry, 55
Diphtheria, 67, 84, 163, 183, 333, 434,
613, 620, 689
paralysis of accommodation after,
66
Diplococci, 326
Diplopia, due to fracture at apex of
orbit, 754
monocular, 697
vertical, 141
Direct method of using ophthalmo-
scope, 39, 41
Discission in secondary cataract, 508
et seq.
Diseases of the eye, general treatment
of, 231 et seq.
Disinfection, 233
Dislocation of globe, 752
of lens, 473 et seq., 520
congenital, 473, 475
in cataract, 488
traumatic, 475, 477
treatment of, 475 et seq.
Dispersion circles, 15
Distichiasis, 261
Distortion of images by cylinders, 128
Dobell's solution, 298
Donders' diagrammatic eye, 15
Double images, 139
after tenotomy, 191
non-recognition of, 160
vision in strabismus, 177
Drusen, 575
Dry cups in myopia, 110
heat, 245
Duboisia, 240, 241
in correction of errors of refrac-
tion, 71
in hyperopia, 89
in spasm of accommodation, 65, 66
Dura mater, arrangement of, 654
Dyer's method of systematic exercise
of eyes, 219, 220, 223
Dynamic squint, 202 et seq., 213
Dyslexia, 703
Ecchymosis of lids, 280
sub-conjunctival, 368, 519
Echinococcus in orbit, 735
Ectopia lentis, 473, 475
Ectropium, 267
flap operations in, 268 et seq., 271
et seq.
hypertrophic, 267
INDEX.
7,80
Ectropium, mechanical treatment of
325
stitching of lids together in, 325
treatment of, 268 et seq.
Eczema, 256, 315
acute phlegmonous, 365
corneas, 379 et seq.
of lids, 256
Egyptian ophthalmia, 339
Electricity in ocular paralyses, 166
Electrolysis in trichiasis, 262
in vascular tumors of orbit, 738
Electro magnet in removal of foreign
bodies from vitreous, 525 et seq.
Embolism, cerebral, 604, 715
in choroid, 576, 584
of central retinal arterv, 601, 689
of retinal vessels, 600 et seq.
Empyema of orbit, 754, 759
Encanthus, 280
Endocarditis, 584
Enophthalmus, 436, 469
traumatic, 748
Entropium, 262 et seq.
after trachoma, 357
operations for, 263 et seq.
senile, 263
spasmodic, 263
Enucleation, danger to life after, 545
et seq.
in panophthalmitis, 586
in staphyloma of sclera, 428
in sympathetic ophthalmia, 538,
540
of eye, 402
method of operating, 544
of staphylomatous globe, 417
Epicanthus, 280
Epilation, 253, 262
Epilepsy, 64, 175, 435, 660, 688
amblyopia of, 701
of retina, 598
Epiphora, 293, 296
Episcleral tissue, 424
Episcleritis, 425
Epistaxis, 95
Epithelial cancer, 259
disease of conjunctiva, 357, 359
Epithelioma of cornea, 423
Equator, 1
Ervsipelas, 315, 395, 584, 671, 676, 689,
729, 730, 731
Eserine, 241
after cataract extraction, 503
and cocaine in glaucoma, 563
in asthenopia, 198
in glaucoma, 563
in muscular asthenopia, 219
in paralysis of accommodation, 67
Esophoria, 214
Ether anaesthesia, 237
inhaler, author's, 238
Ethmoid cells, distention of, 750
fracture of, 757, 759
Evisceratio bulbi, 401, 402
Evisceration, inefficacy of, as a pro-
tection from sympathetic oph-
thalmia, 538
in panophthalmitis, 587
in staphyloma of sclera, 428
in sympathetic ophthalmia, 532,
- 540, 544
Examination of eye, 35 et seq.
anaesthetics in, 37
Excavation of optic nerve in glaucoma,
549, 550, 551, 554, 559
physiological, 555
resembling glaucoma, 556
Excision in trachoma, 350
of apex in conical cornea, 422
of follicles in trichiasis, 262
of pupil, 442, 444, 451
Exenteration (see Evisceration)
of orbit, 741
Exophoria, 214
Exophthalmic goitre, 163, 251, 764 et
■seq.
treatment of, 766 et seq.
Exophthalmus, 37, 141, 401, 436
pulsating, 743 et seq.
treatment of, 747
Expression in trachoma, 352
Eye diseases, relative frequency of, 771
statistics of, 771 et seq.
Eye-douches, 244
Eyelashes, 248
diseases of, 261 et seq.
evulsion of, 253
Eyelid, chancre of, 358
coloboma of, 280
phlegmon of, 255
vertical division of, in purulent
conjunctivitis, 329
Eyelids, 35, 247 et seq.
affections of muscles of, 281 et seq.
anatomy of, 247 et seq.
ecchymosis of, 280
gangrene of, 255
glands of, 250
injuries of, 278 et seq.
lacerations of, 278
muscles of, 248 et seq.
wounds of, 278 et seq.
Eye-shades, 231
Eye-stones, 367
Facial and hypoglossal nerves, rela-
tions of, 156
nerve, nucleus of, 155
tic, 281
False passage by probe, 301
Far point, 17
Fascia, oculo-orbital, 1, 168, 169, 724
inflammation of, 728
Fasciculus cruciatus, 715
non-cruciatus, 715
opticus, 715
retroflexus, 150
Fever, intermittent, 383, 672
puerperal, 584, 729
recurrent, 66, 67, 584
790
INDEX.
Fever, relapsing, 514
scarlet, 67, 183, 315, 584, 613, 615,
616, 620, 672, 689
typhoid, 66, 67, 584, 672, 689
typhus, 383, 672, 689, 729
Fibres of Miiller, 463, 594, 617, 620
Fibroid tumors of conjunctiva, 357,
359
Fibroma of choroid, 593
of cornea, 423
Field of vision, 21, 2ietseq., 653
by reduced light, 26
defective, in asthenopia, 216
incorrect projection of, 140
in glaucoma, 549, 550, 551, 554,
555, 559
in optic nerve atrophy, 686,688,
689
in optic neuritis, 666, 667
in toxic amblyopia, 701
methods of taking, 24 et seq.
sector-like defects in, 667, 712
spiral restriction of, 704
temporary limitation of, 182
Fifth nerve, effect of irritation of, on
intra-ocular tension, 470
influence of, on intra-ocular
secretion, 9
nuclear origin of, 155
Filamentous keratis, 384
Filaria in anterior chamber, 453
Fillet, 148, 150, 643, 646, 657
Filtration angle, 558
First principal focus, 11
Fistula corneae, 406, 411, 414
lachrymal, 296, 305
Flap extraction, 495
from forehead, 272, 274
from temple, 272
from zygoma, 272, 273
naso-buccal, 272, 273, 274
operation, Knapp's, 273
Wolfe's, 271, 274
operations, author's methods, 272
et seq.
in ectropium, 268 et seq., 271
et seq.
in symblepharon, 290
with pedicle, 271
Flint's tablets, formula for, 677
Flocculus of cerebellum, 146
Foci of glasses, 55 et seq.
Focus, anterior principal, 11
of cornea, 12
first principal, 11
posterior principal, 11
of cornea, 12
principal, of crystalline lens, 12
second principal, 11
Folliculosis, 340, 341
Fontana's canal, 4
Foreign bodies in conjunctiva, 367
in cornea, 523
in globe, 523 et seq.
in globe, general considera-
tions, 529
Foreign bodies in orbit, 761 et seq.
in vitreous, 523 et seq.
in vitreous, statistics of, 528
on conjunctiva, 523
Formatio reticularis, 148 et seq.
Form sense, 21
Fornix, 308
Forster's operation for ripening cata-
ract, 494
Fossa patellaris, 472, 512
Fourth ventricle, lesions in vicinity of,
658
Fovea centralis, 5, 595
appearances of, 50, 52
detachment of, 631
minute structure of, 20
vascularity of, 48
Fracture of canalis opticus, 757, 759
of ethmoid, 757, 759
of lamina cribrosa, 757
of orbit, 754 et seq.
Frontal bone, anatomy of, 145
sinus, distention of, 749
emphysema, 671, 749
Fundus oculi as seen by ophthalmo-
scope, 47 et seq.
Fungus haematodes of eye, 637 et seq.
Galvanic current in paralysis of ac-
commodation, 67
Galvanism in exophthalmic goitre, 766,
767
in muscular asthenopia, 220
Galvano-cautery, 233
Ganglion, ciliary, 17, 424, 432, 464, 724
of Gasser, 9, 145, 146
of Luys, 644, 652
of Soemmering, 146
Gangrene of lid, 255
Gasserian ganglion, 9, 145, 146
Gelsemine, 242
General paresis, 67, 162
Germs in conjunctival sac, 234
Gerontoxon, 410
Glands of Henle, 250
of Krause, 250, 309
of lids, 250
of Moll, 250, 308
Glass, French, German, 56
index of refraction of, 55, 56
shell, after symblepharon onera-
tion, 289
Glasses, 53 et seq.
arrangement of, 54 et sec
bi-cylindric, 58
blue, in myopia, 111
cataract, 58, 510, 511
colored, 68, 231
after use of atropia, 65
in nystagmus, 175
comparison of old and new sys-
tems of numbering, 56, 57
cylindrical, 53, 54
decentering of, 90, 202
effect of change of position on
power of, 56, 57
INDEX.
791
Glasses, foci of, 55 et seq.
for aphakia, 58, 510, 511
Franklinic, 87
hyperbolic, 53
incidental effects of, 131
in paralysis of accommodation,
68
metric system of numbering, 56
nomenclature of, 54 et seq.
old method of numbering, 56
properties of, 53 et seq.
protective. 231
radii of curvature of, 55
smoked, in myopia, 110
spherical, 53
sphero-cylindric, 59, 118, 128
varieties of, 53 et seq.
Glaucoma, 548 et seq., 776
absolute, 551, 553, 567
acute inflammatory, 556
after cataract extraction, 562
anterius, 570
central vision in, 554
chronic, 551 et seq., 556
cocaine in, 236
diagnosis of, 554 et seq.
double, 552
etiology of, 557 et seq.
hemorrhagic, 553, 607
heredity of, 557
in microphthalmus, 562
in myopia, 549, 569, 570
in second eye following iridectomy
on first, 566
iridectomy in, 564 et seq.
malignant, 567
operative treatment of, based on
pathological examinations, 570
et seq.
optico-ciliary neurectomy in, 565
paracentesis in, 570
pathogenesis of, 557 et seq.
pathology of, 559 et seq., 570 et seq.
prognosis in, 556 et seq.
retention theory of, 563
sclerotomy in, 564 et seq.
secondarium, 553
secondary degenerations in, 553
simplex, 548
stretching of infra-trochlear nerve
in, 566
subacute, 552
subjective symptoms of, 550
symptoms of, 548 et seq.
treatment of, 563 et seq.
value of operations in, 565, 570
with absence of iris, 562
with deep anterior chamber, 570
with inflammation, 551, 552
Glioma retinae, 467, 514, 637 et seq.
Globe, anatomy of, 1
antero-posterior diameter of, 1
axis of, 1
lymph circulation of, 8
measurements of, 2
nutrition of, 8
Globe, poles of, 1
protrusion of, 37
vessels of. 6
Glycosuria, 672. 700 (see also Diabetes)
Gonococcus of Neisser, 322, 326, 376,
445
Gonorrhceal ophthalmia, 325 et seq.
Gout, 378, 445, 449, 557, 570, 606, 677,
692
Graduated tenotomy, 224
Graefe's equilibrium test, 206, 214
Grafting, Reverdin's method, 271
Thiersch's method, 271, 272, 290
Grafts in symblepharon, 290
Granuloma of conjunctiva, 358, 360
of iris, 452
Grattage, Gibson's eversion forceps for,
352, 353
in purulent conjunctivitis, 330, 331
in trachoma, 352 et seq.
scarificator for, 352
Graves' disease, 163, 251, 764 et seq.
Gray tubercle of Rolando, 155
Gumma of ciliary body, 467
of iris, 443
Gummata, 163
Gummy tumors of conjunctiva, 358
Gun-shot wounds of orbit, 753
Gymnastic prisms, 220, 223
Hallucinations, hemianopic, 712
Halo around macula lutea, 50
in glaucoma, 551, 559
Hebetudo visus, 197
Hebra's ointment in blepharitis, 253
Hemeralopia, 627, 703
Hemi-achromatopsia, 712
Hemiamblyopia, 181
Hemianaesthesia, 162, 657
Hemianopia, 216, 644, 647, 649, 657,
701, 707 et seq.
binasal, 707, 709 et seq.
bitemporal, 647, 707, 709, 710
boundary line in, 711, 712
central, 707 et seq.
cortical, 713, 714
diagnosis of, 715 et seq.
due to aneurism of internal caro-
tid, 708
due to lesion of angular gyrus,
714
of calcerine fissure, 714
of chiasm, 709, 710
of crus, 713
of'cuneus, 714
of internal capsule, 157, 713
of optic radiations, 714
of orbit, 708
of primary ganglia, 713
of tractus, 712 et seq.
due to localized meningitis, 713
tumor at base of brain, 713
epitome of symptoms of (Seguin),
716
for color, 712
792
INDEX.
Hemianopia, homonymous, 644, 647,
657
homonymous, lateral, 707, 710
diagram illustrating, 715
homonymous quadrants of, 712
lesions producing, 708 et seq.
location of lesion in, 644
monocular, 708
peripheral, 707 et seq.
peripheral limitation of visual
fields in, 714
prognosis in, 717
pupillary reaction in, 433
treatment of, 717
Wernicke's pupillary symptom in,
708, 709, 713, 714
with retrobulbar neuritis, 708
Hemianopic hallucinations, 712
Hemianopsia (see Hemianopia)
Hemiataxia, 162, 164
Hemiopic pupillary inaction, 708, 709,
713, 714
Hemiplegia, 64, 157, 159, 162, 175, 469,
657
crossed, 160
Hemorrhage, amblyopia from, 698
beneath conjunctiva, 519
cerebral, 163, 609
following iridectomy for glaucoma,
566, 569
into anterior chamber, 519
into canal of Petit, 513
into choroid, 587
into orbit, 751
into retina, 605 et seq.
into sheath of optic nerve, 759
into vitreous, 513, 517, 520
Hemorrhages in myopia, 95, 101
Herpes corneae, 380, 383
treatments of, 384 et seq.
of conjunctiva, 363, 366
zoster frontalis, 315
ophthalmicus, 256, 257, 258,
366, 383
Herpetiform keratitis, 385
Heterophoria, 214
Hexagonal pigment-epithelium of
retina, 5
Hippus, 434, 438
Hodgkins' disease, 736
Homatropia, 240, 241
in correction of errors of refraction,
71
in hyperopia, 89
in spasm of accommodation, 68
Hooks, Snellen's, 22
Hordeolum, 253
Horizontal meridian, 1
Horner's disease, 469, 748
muscle, 169, 250
Horny growths, 260
Horopter, 134
Hot applications, 244
in glaucoma, 564
in interstitial keratitis, 393
in iritis, 447, 448
Hot applications in purulent conjunc-
tivitis, 330, 331
Humor, aqueous, 2
vitreous, 2
Hyalitis, 512 et seq.
diagnosis of, 515
in soft cataract, 517
prognosis in, 516 et seq.
purulent, 513, 514, 517
treatment of, 516 et seq.
Hyaloid artery, 8, 512, 513, 518
membrane, 512
Hydrocephalus, 671
internus, 644
Hydrochinon, 233
Hydrophthalmus, 553
in myopia, 102
Hydrotherapy in ocular paralyses, 166
Hyoscine, 240, 241
in spasm of accommodation, 65
Hyperaemia palpebralis, 311, 312
Hyperaesthesia retinae, 197
Hyperbolic glasses, 421
Hyperesophoria, 214
Hyperexophoria, 214
Hypermetropia (see Hyperopia)
Hyperopia, 69, 82 et seq.
anatomical characteristics of, 83
axial, 91
complications of, 86
diagnosis of, 86
extreme, 90
facultative, 84
in strabismus convergens, 179 et
seq.
mydriatics in, 89
prognosis in, 86
size of image in, 72
statistics of, 90 et seq.
treatment of, 87
Hyperphoria, 213, 214, 216, 223, 229
Hypoglossus, relations of, 156
Hypopyum, 375, 440
keratitis, 395, 397 et seq.
course and symptoms of, 398
etiology of, 397
treatment of, 398 et seq.
Hypotony, 466, 468
Hysteria, 64
Illumination by plane mirror, 36
focal, 35
oblique, 35
Images, determination of size of, 12, 15
produced by glasses, 54
Inch, Austrian, 55
English, 55, 56
equivalent in dioptries, 57
Paris, 55, 56
Prussian, 55
Index of refraction, 10
of cornea, 10, 374
of crystalline lens, 10, 12
of glass, 55, 56
Indirect method of using ophthalmo-
scope, 40, 42
INDEX.
793
Infundibulum, 641, 642
Injuries of globe, 519 et seq.
Inoculation with blenorrhoeal pus in
trachoma, 354
Insanity, 67
Internal capsule, 146, 148, 157, 657, 715
Inter-pupillary distance, 137, 433
Intra-ocular tension (see Tension)
Inverted image, method of using oph-
thalmoscope by, 40
Iodine injections in detachment of
retina, 636
Iodoform, 233
Iridavulsion, 460
Iridectomy, 245, 454
cystoid cicatrix after, 567
explanation of effects of, in glau-
coma, 568
followed by cataract, 567
for therapeutic purposes, 454
for visual purposes, 454
in cataract extraction, 499
in conical cornea, 421
in corneal affections, 388
in corneal opacity, 412
in detachment of retina, 636
in glaucoma, 564 et seq.
statistics of, 572
in iritis, 450, 451
in pterygium, 370
in zonular cataract, 492
instruments, 455, 456
method of operating, 456 et seq.. 569
value of, in glaucoma, 570
Irideremia, 439
Iridesis in conical cornea, 422
Irido-cyclitis, 465
Iridodesis, 454, 461
Irido-dialysis,460
Irido-donesis, 439
Irido-rhexis, 454, 460
Iridotomy, 246, 412, 454, 459
Iris, 2, 35, 430 et seq.
absence of pigment in, 439
anatomy of, 430 et seq.
angle of, 430
avulsion of, 416
coloboma of, 439, 775
complete removal of, 246
condyloma of, 443
congenital defects of, 439
cysts in, 452
dialysis of, 520
functional diseases of, 434 et seq.
pathology of, 437
prognosis in, 437
treatment of, 437
granuloma of, 452
incarceration of, after cataract ex-
traction, 502
inflammation of, 440 et seq.
lymphomata of, 452
operations on, 454 et seq.
pigment of, 431
pillars of, 430
physiology of, 430 et seq.
Iris, prolapse of, 521
after cataract contraction, 502,
505
sarcoma of, 452
sphincter of, 5
tremulous, 439
tubercle of, 443, 452
tumors of, 451 et seq.
vascular growths in, 452
Iritis, 440 et seq.
causes of, 445
chronic, 450
complications of, 443
constitutional treatment of, 448
following cataract extraction, 506,
507
gonorrhoea! 445, 449
gouty, treatment, 449
gummy, 442, 443
iridectomy in, 450, 451
paracentesis in, 447, 450
plastic, 442
prognosis in, 446
quiet, 443
rheumatic, 443, 445, 447
treatment of, 449
sequelae of, 444 et seq.
serous, 407, 442, 466, 533, 575
signs of, 440 et seq.
spongy, 442, 570
suppurative, 442, 443
traumatic, 442
treatment of, 446 et seq.
varieties of, 442
Iritomy, 459
in zonular cataract, 492
Ischaemia of retina, 600
Jaborandi, 241, 242
Jequirity in trachoma, 354, 355
in vascular keratitis, 388
Karyokinesis, 372, 375
Keratitis, 378 et seq.
bullosa, 385
dendritica, exulcerans, mycotica,
409
diffusa, 389 et seq.
filamentosa, 384
herpetiformis, 385
interstitialis, 389 et seq.
consecutiva, 386
etiology of, 389 et seq.
prognosis in, 392
symptoms of, 390 et seq.
treatment of, 393
iridectomy in, 388
malarial, 388, 389
mycotica, 376, 395
dendritica, 376, 389
neuro-paralytica, 395, 402 et seq.
nummularis, 385, 410
parenchymatosa, 389 et seq.
phlyctenularis, 379 et seq.
treatment of, 381 et seq.
postica, 407 et seq.
794
INDEX.
Keratitis, postica, pathology of, 408
scrofulosa, 380
sequelae of, 411 et seq.
subdivisions of, 378
superficialis purulenta discreta, 396
suppurativa, 394 et seq.
symptoms of, 378 et seq.
vasculosa, 386 et seq.
xerotica, 395
zonularis, following glaucoma, 410
Kerato-malacia, 376, 394, 395, 404
Keratoscopy (see Shadow test)
Kidney disease, 609, 612, 613, 615, 616,
620, 621, 622, 688
Kopiopia, 197
Labarraque's solution, 233
in epithelioma, 259
Labio-glosso-laryngeal paralysis, 175
Laceration of choroid, 587
Lachrymal abscess, 295
apparatus, anatomy of, 292 et seq.
diseases of, 292 et seq.
fistula, 305
gland, 292
diseases of, 293
dislocation of, 294
extirpation of, 307
tumors of, 735
gouge, 303
knife, Agnew's, 299, 300
author's, 299, 300
probes, 299 et seq.
sac, 35, 292
abscess of, 295
catarrh of, 294 et seq.
chronic distention of, 294
excision of, 303
micro-organisms in, 295
obliteration of, 303, 304
phlegmonous inflammation of,
295, 304
syringing of, 302, 306
trouble, danger from, in cataract
extraction, 295
Lachrymation, excessive, 307
Lachrymo-nasal duct, 292
Lagophthalmus, 283
La grippe, 163, 682
Lamina cinerea terminalis, 644
cribrosa, 424, 650
fracture of, 757
fusca, 573
Lanolin, 237
Lead poisoning, 163, 612, 672, 683, 688
Leech, artificial, 243
Heurteloup's, 243
Leeches, 243
in myopia, 109
in ocular paralysis, 166
in spasm of accommodation, 65
Lemniscus, 148, 150, 643, 646, 657
Lens, Bnicke's, 36
crystalline, 2, 35, 471 et seq.
anatomy of, 471
capsule of, 2
Lens, crystalline, changes in, in ac-
commodation, 17
coloboma of, 473, 474
concussion of, 697
development of, 472
dislocation of, 473 et seq.
congenital, 473, 475
in cataract, 488
prognosis in, 475
symptoms of, 474
traumatic, 475, 477
treatment of, 475 et seq.
increase of refractive index of,
in deeper layers, 15
increase of size of, with age,
561
index of refraction of, 10, 12
luxation of, 520
optical centre of, 12
principal focus of, 12
radii of curvature of surfaces
of, 12
removal of, from vitreous, with
spoon, 501
sclerosis of, 479, 481, 485
size of, 498
suspensory ligament of, 5, 430,
462, 472, 512
toric, 59
Lenses (see Glasses)
forms of, 54
properties of, 10
Lenticular ganglion, 17
Lepra, 452
Leptothrix lachrymalis, 294, 306, 307
Lesser circles, 1
Leucocythemia, 452, 612, 622
Leucoma, 378
adhaerens, 378, 405
corneae, 411
Ligament, pectiniform, 4
Ligamentum annulare, 6
Light sense, 30
in optic nerve atrophy, 686
streak, explanation of, on retinal
vessels, 50 et seq.
subjective sensations of, 31
Lightning stroke, loss of sight by, 698
Limbus corneae, 3, 371
Line of fixation, 178
Lippitudo, 252
Liquor plumbi subacetatis, 253
Listing's reduced eye, 12
Localization, cerebral, 653 et seq.
Locomotor ataxia, 66, 67, 162 434 436
645, 686 ' '
Long ciliary arteries, 7, 424, 573
Lupoid growths, 259
Lupus non exedens, 259
of conjunctiva, 357, 358, 359
treatment of, 260
Luy's body, 146, 150
Lymph channels of cornea, 8
of eye, 557, 558
circulation of globe, 8
current, intraocular, 8
INDEX.
795
Lymph space, intra-vaginal, of optic
sheath, 8
spaces, perivascular, 8
Lymphoid tissue of conjunctiva, 309
Lymphomata, 163
of iris, 452
Macula corneae, 378, 411
lutea, 5, 595
coloboma of, 105, 597
halo around, 50
vascularity of, 48
Madarosis, 253, 261
Maddox prism, 210, 212
rod, 210, 212
Magnet, Bradford's,525
Gruening's, 525
Hirschberg's, 525
in removal of foreign bodies from
vitreous, 525 et seq.
Magnifying power of ophthalmoscope,
Malar bone, dislocation of, 754
Malaria, 208, 378, 445
Malarial keratitis, 388, 389
Malingering, 719
Mariotte's blind spot, 181
Massage in cataract, 488
in muscular asthenopia, 219
in retinal embolism, 605
Masturbation, 66
Measles, 183, 315, 613, 620, 672
Measurements of globe, 2
Mechanical exercise in ocular paral-
yses, 166
Medulla oblongata, 146
anatomy of, 147 et seq.
tracts of, 147
"Medusa head" nerve, 614, 663
Megalophthalmus, 775
Megalopsia, 581, 624
Megrims, 598 et seq.
Meibomian glands, 250, 308
Membrana limitans, 50
externa, 595
interna., 595
pupillaris perseverans, 439
Meningitis, 163. 516, 665, 672, 673, 675,
676, 689, 715, 718, 730, 740, 742
cerebrospinal, 672, 673, 676
following enucleation, 545 et seq.
tubercular, 163, 671, 672, 677
Menstrual disorders, 95, 609, 612
Mental blindness, 694, 703 (see Mind
b. and Psychic b.)
Mercurials in iritis, 448
Meridians of globe, 1
Metamorphopsia, 101, 581, 587, 609,624,
630, 634, 695
Methyl violet, 234
Metric angle, 203
of convergence, 433
system of numbering glasses, 56
Meynert's bundle, 150
Microbes in conjunctival sac, 234
Micro-organisms in corneal ulceration,
394, 395
in dacryo-cystitis, 295
in pterygium, 369
Microphthalmus, 439, 468, 775
Micropsia, 581, 624
Microsporon trachomatosum, 344
Migraine, 599, 702
Miliary aneurisms of retina, 610
Milium, 260
Mind blindness, 656, 714 (see Mental b.
and Psychic b.)
Miner's nystagmus, 174
Mobility of eye, 36
Moles, 261
Molluscum contagiosum, 256, 257
Monocular accommodation, 18
Moon blindness, 704
Motor areas of brain, 156, 157, 6o±etseq.
tracts, 147 et seq.
Mucous patches on conjunctiva, 358
Multiple sclerosis, 164, 672, 683
Mumps, 584
Muscae volitantes, 100, 486, 513
Muscarine, 242
Muscle, ciliary, 6
of Bowman, 463
of Briicke, 463
of Horner, 169, 250
of Miiller, 436, 724, 748
ofRiolani, 248
Muscles, methods of testing, 204, 205,
210 et seq.
ocular (see Ocular muscles)
of eyelids, 248 et seq.
Muscular apparatus, normal status of,
204 et seq.
asthenopia, 197, 202 et seq.
insufficiency, 202 et seq.
general considerations, 206 et
seq.
vertical, 216, 223, 229
relations, reversal of, for distance
and near, 218
Music deafness, 656
Mydriasis, 434
Mydriatics, 240, 241, et seq.
danger from, in glaucoma, 564
in astigmatism, 118
in hyperopia, 89
Myelitis, 469
acute, accompanied by optic neu-
ritis, 672, 673
Myopia, 69, 92 et seq.
atrophic changes in, 110
axial, 92
cataract in, 101
causes of, 93 et seq.
central choroidal changes in, 101
centre of motion in, 102
choroidal changes in, 101, 581
complications in, 97 et seq.
congenital, 92, 95, 103
constitutional causes of, 95
detachment of retina in, 93, 101.
110
796
INDEX.
Myopia, diagnosis of, 103
due to occupation, 104
functional disturbances in, 96
hemorrhages in, 95, 101, 110
hereditary, 103
hydrophthalmus in, 102
inflammatory, 95, 109
influence of muscles in producing,
94 et seq.
in school children, 104
malignant, 581
monocular, 96
muscular asthenopia in, 108
incapacity in, 101
ophthalmoscopic picture in, 96 et
seq., 105
optic nerve atrophy in, 582
pathological anatomy of, 96 et seq.
prevalence of, 93
prisms in, 109
prognosis in, 102 et seq.
prophylaxis, 103
scotoma in, 110
selection of glasses in, 106 et seq.
size of image in, 72
slowly progressive, 107
spasm of accommodation in, 104
stationary, 107
statistics of, 103 et seq.
tenotomy in, 227
treatment of, 106 et seq.
with cataract, 480
Myosis, 434, 435
from irritation of cerebral centre,
435
paralytic, 435, 438
Myotics, 240, 241
general conclusions, 243
NtEVI, 260, 261
Nasal catarrh as a cause of asthenopia,
217
duct, 292
catarrh of, 294
obstruction of, 294
stricture of, 299
Near point, 17
Nebula, 278
Needles for advancement, 229
Nerve, optic (see Optic nerve)
Nerves, cranial, 145 et seq.
diagram showing origins of, 152
relations of, at base of brain,
152
long ciliary, 425, 432, 433
ocular, 133, 147 et seq.
short ciliary, 425, 432, 433
Neuralgia, dental, 66
facial, cocaine in, 237
Neuritis optica, 662 et seq.
after orbital affections, 676
albuminurica, 677
anatomical characteristics of,
667 et seq.
apoplectica, 665
axial, 678, 679, 680, 681, 683, 684
Neuritis optica, axial, congenital, 686
brain symptoms in, 675
chemical irritation theory of,
674
descendens, 662, 664 et seq.
diagnosis of, 675
etiology of, 670
germ theory of, 674
glycosurica, 677
hemorrhagica, 665
idiopathica, 672
interstitialis, 668
monocularis, 664
partial, 662
pathogenesis of, 673 et seq.
pathology of, 684, 685
peri-axial, 681
prognosis in, 676
relation of, to intra-cranial dis-
ease, 670 et seq.
retrobulbars, 662, 665, 678 et
seq., 732
cases of, 680, 681
causes of, 681, 683
due to alcohol, 678, 682 et
seq.
la grippe, 682
tobacco, 678,682 et seq.
toxic, causes of, 683
opthalmoscopic pic-
ture in, 682
treatment of, 682, 684
with hemianopia, 708
subjective symptoms of, 666 et
seq.
syphilitica, 676
treatment of, 677 et seq.
with dropping of fluid from
nose, 666
Neuro-retinitis descendens, 662, 664 et
seq.
following sunstroke, 672
pigmentosa, 629
sympathetica, 535
syphilitica, 623
with brain tumor, 665
Nicotine paralysis, 162
poisoning, 163
Night blindness, 703
Nitrate of silver, 234
in blepharitis, 252
in conjunctival inflamma-
tions, 318, 322, 323, 324, 326
329, 336, 350, 351
in eczema of lids, 256
in lachrymal troubles, 298, 299,
302, 307
in trachoma, 350, 351
Nodal point, 11
of cornea, 12
Nubecula, 378, 411
Nuclear paralysis of ocular muscles,
Nuclei of fifth nerve, 155
of third nerve, 152 et seq.
Nucleus cuneatus, 148
INDEX.
797
Nucleus gracilis, 148
of facial nerve, 155
of sixth nerve, 155
oftrochlearis, 153, 154
Nummular keratitis, 385
Nutrition of globe, 8
Nyctalopia, 705
Nystagmus, HZetseq., 474, 574
etiology of, 174 et seq.
in congenital cataract, 484
monocular, 173, 174
treatment of, 175
Oblique illumination, 35
Occlusion of pupil, 442, 444, 451, 465
Ocular muscles, actions of, 133
advancement of, 169 et seq.
anatomy of, 132 et seq.
diagram of origins of, 755
physiology of, 132 et seq.
spasm of, 173 et seq.
paralyses, etiology of, 163 et seq.
prognosis in, 166
treatment of, 166 et seq.
Oculo-motorius, 152
Oculo-orbital fascia, 1, 168, 169, 724
inflammation of, 728
CEdema of retina, 606, 608
Olivary bodies, 146
Opacities in vitreous, 513, 514, 515,
516
Opaque nerve fibres, 597, 658
Operating mask, 239
Operative interference in muscular as-
thenopia, 224
work, antiseptics in, 234 et seq.
general considerations, 238 et
seq.
Ophthalmia migratoria, 536
neonatorum, 319 et seq.
corneal complications in, 324
prophylaxis, 321, 322
Crede's method, 322
statistics of, 321
treatment of, 322 et seq.
varieties of, 319, 320
tarsi, 252
Ophthalmomalacia, 466, 468
Ophthalmometer, description of, 123
et seq.
Helmholtz's, 16, 378
in detection of conical cornea, 419
of corneal opacities, 378
in determining astigmatism, 121
JavalV 70, 103, 357, 378
method of using. 123 et seq.
Ophthalmometry, 121, 123
Ophthalmoplegia externa, 155, 160
Ophthalmoscope, 39 et seq.
as an optometer, 74
author's, 43. 45
binocular, 44
direct method of employing, 39, 41
in determining refraction, 71 et
seq., 121 et seq.
Ophthalmoscope, indirect method of
emploj'ing, 40, 42
magnifying power of, 42
varieties of, 43 et seq.
Ophthalmoscopic picture, 47 et seq.
Optic centre, 656
centres, primary, 715
chiasm, 145, 147
disc, anatomically oval, 122, 659
congestion of, 611
'' woolly,'' 666, 676
lobes, 715
nerve, 2, 641 et seq.
anaemia of, 660
anatomy of, 641 et seq.
anomalies of, 105
arrangement of fibres in, 651,
652
atrophies following destruc-
tion of, 645
atrophy of, 164, 686 et seq.
causes of, 687 et set/.
congenital, 609, 687
experimental, 690
from cerebral causes, 689
hereditary, 687
in myopia, 585
interstitial, 690, 691
medullary, 689
morbid anatomy of, 689 et
seq.
ophthalmoscopic picture
in, 686 et seq.
parenchymatous, 688
partial, 686
primary, 689
prognosis in, 691
subdivision of, 687
symptoms of, 686 et seq.
treatment of, 692 et seq.
capillary congestion of, 610
colloid degeneration of, 616
coloboma of sheath of, 105, 658
congenital abnormalities of,
180 et seq., 658
congenital defects of, 180, 658
connective-tissue septa of, 650
crossing fibres of, 643, 647, 651,
652
direct fibres of, 642, 647, 651,
652
diseases of, 653 et seq.
emergence of, 147
excavation of, in glaucoma,
549, 550, 551, 554, 559
fasciculi of, 650
hemorrhage into, 660, 661, 663
into sheath of, 661, 662,759
hyperaemia of, 659
inter-vaginal space of, 650
intra-cranial portion of, 649 et
seq.
lymph spaces of, 650
macular fibres of, 651
number of fibres in, 651
orbital portion of, 649 et seq.
798
INDEX.
Optic nerve, physiological excavation
of, 555
physiology of, 641 et seq.
pupillary fibres of, 651
sheath, 649, 650, 673, 724
dropsy of, 674
inter-vaginal lymph space
of, 8
stretching of, in atrophy, 693
tumor of, 734, 736 et seq.
wounds of, 696, 760, 761
neuritis (see Neuritis optica)
papilla, 650
colloid bodies in, 669
connective tissue on, 659
radiations, 644, 645, 647, 715
thalamus, 146, 147, 150, 641, 642,
644, 645, 646, 657, 715
tract, 145, 146, 150, 642, 643, 644,
645, 646, 654, 715
arrangement of fibres in, 652
roots of, 644, 645, 646
Optical axis, length of, 13
centre of cornea, 12
of crystalline lens, 12
Optico-ciliary neurectomy, 592, 540,
542
in glaucoma, 565
vessels, 424
Optometers, 80 et seq.
Ora serrata, 5, 48, 462, 596
Orbicularis, paralysis of, 283, 294
Orbit, abscess communicating with,
726
abscess of, 731, 732
anatomy of, 144, 249, 722 et seq.
axis of, 724
bony tumors of, 736, 739
caries of, 726, 728
cerebral symptoms following in-
jury of, 756
diseases of, 725 et seq.
distention of cavities adjacent to,
749
emphysema of, 754
exenteration of, 741
foreign bodies in, 761 et seq.
fracture of apex of, 754, 755
fracture of, 520, 695. 696, 754 et seq.
gun-shot wounds of, 753
hemorrhage into, 751
injuries of, 66
lesions of, 696
origins of ocular muscles at apex
of, 755
penetrating wounds of, 755, 756
periostitis of, 689, 725 et seq.
treatment of, 727
phlegmonous inflammation of, 546,
665, 726, 729 et seq.
tumors of, 671, 733 et seq.
prognosis in, 743
recurrence of, 743
symptoms of, 737
treatment of, 738 et seq.
varieties of, 735, 736
Orbit, vascular tumors of, 735, 736, 743,
744, 745
wounds and injuries of, 752 etseq.,
759 et seq.
Orbital cellulitis, 401, 729 et seq.
following evisceration, 546
deep incisions in, 731
effects of, on vision, 732
treatment of, 730 et seq.
cysts, 735
fracture, amblyopia following, 757
muscle of Miiller, 724, 748
paralyses, 158, 163
symmetrical, 158
periostitis, 689, 725 et seq.
tumors, 671, 733 et seq.
veins, thrombosis of, 730, 732, 733
Orthophoria, 214
Ossification of choroid, 593
Oxide of zinc, 256
in blepharitis, 252
Pachymeningitis, 163
Painful vision, 197
Palpebral fissure, 247
ligaments, 247
Panas' solution in operative work, 234
Pannus, 338, 346, 352, 377, 387, 388
crassus, 338
tenuis, 338
Panophthalmitis, 401, 402
enucleation in, 586
evisceration in, 587
following discission of cataract,
491
intra-uterine, 775
suppurative, 588 et seq.
Papillitis, 616, 662, 663, 664, 666, 667
668, 669, 670, 671, 673, 674, 675
diagnosis of, 675
etiology of, 670 et seq.
relation of, to intracranial dis-
ease, 670 et seq.
with brain tumor, 664
Papilloma of conjunctiva, 357, 358
of cornea, 423
Papillomata, 260
Paquelin's cautery, 233
Paracentesis corneae, 245, 246, 247
in myopia, 110
effects of, on intra-ocular tension, 9
in conical cornea, 421
in glaucoma, 570
in iritis, 447, 450
Paralexia, 703
Parallax, 75
of vessels in glaucoma, 549
test, 210
Paralysis, bulbar, 162
motor, alternating, 149
nicotine, 162
of accommodation, 66
treatment of, 67
of ocular muscles, 139 et seq
basal, 158
collateral symptoms in, 159
INDEX.
799
Paralysis of ocular muscles, congenital,
cortical, 164, 165
diagnosis of, 140 et seq., 158 et
seq.
diagrams illustrating, 142
etiology of, 163 et seq.
intracranial, 144
location of lesion in, 144 et seq.,
nuclear, 160 et seq.
peripheral, 163
position of head in, 140
prognosis in, 166
recurrent, 158
rheumatic, 158, 163
sub-cortical, 464, 165
symptoms of, 139 et seq.
treatment of, 166 et seq.
of orbicularis, 283, 294
of superior oblique, 196
of third nerve, crossed, 159
orbital, 158. 163
Paraphasia, 656
Paraphimosis, 325
Paresis of accommodation, 66
treatment of, 67
of ocular muscles, 139 et seq.
Pars non plicata of ciliary body, 462
Pars plicata of ciliary body, 462
Partial tenotomy, 224, 226, 227
Pectiniform ligament, 4, 373
Pediculus pubis, 261
Peduncles, 145 et seq.
of cerebellum, 147
Pedunculus cerebri, 641, 643, 645
Pemphigus of conjunctiva, 362, 366
of cornea,, 385
Perimeter, 25, 26
forms of, 25
Schweigger's hand, 653
Perineuritis, 602, 665
following meningitis, 673
Periostitis of orbit, 689, 725 et seq.
Peripheral paralyses of ocular muscles,
163
Peritomy in trachoma, 354
in vascular keratitis, 388
Perivasculitis, 687
Pernicious anaemia, 609
Pes pedunculi, 146, 147, 150
Petit's canal, 8
Phantom image of retinal vessels, 32
Phenacetine after tenotomy, 227
Phlebitis of ophthalmic vein, 746
of retinal vessels, 603
Phlegmon of lid, 255
of orbit, 729 et seq.
Phlyctenular conjunctivitis, 363, 364
keratitis, 379 et seq.
Phorometer, 210, 211, 213
Phosphorus in muscular asthenopia, 219
poisoning, 612
Photometer, 31
Photophobia, 37
cocaine in, 381
Phtheiriasis, 261
Phthisis, 437, 766
bulbi, 406, 466
essential, 470
Physiology, general, of eye, 10 et seq.
of brain, 653 et seq.
of conjunctiva, 308 et seq.
of cornea, 374 et seq.
of iris, 430 et seq.
of ocular muscles, 132 et seq., 134
of optic nerve, 641 et seq.
Pigment patches in conjunctiva, 357,
360
proliferation in choroiditis, 577
Pigmentation of retina, 50
Pilocarpine, 241
constitutional effects of, -242
hypodermic injection of, 242
in asthenopia, 198
in muscular asthenopia, 219
in myopia, 109
in paralysis of accommodation, 67
injections in detachment of retina,
635
Pince-ciseaux of Wecker, 459, 492
Pineal gland, 148
Pinguecula, 360
Pink eye, 315
bacillus of, 315
Pituitary body, 146, 642
Placido's disc, 126, 378, 419, 420
Pleurisy, 435
Plica semilunaris, 308
Pneumonia, 672
Poles of globe, 1
Polio-encephalitis, 164
Polycoria, 440
Pons Varolii, 145 et seq.
anatomy of, 147 et seq.
lesions of, 149, 160, 161
Pooley's operation for ripening cata-
ract, 494
Posterior chamber, 2, 430
ciliary arteries, 6, 573
perforated space, 146, 642
principal focus, 11
Poultices, 244
Powder burns, 278, 523
Pregnancy, amblyopia of, 701
Pre-lachrymal abscess, 306
Presbyopia, 61 et seq.
reserve accommodation in, 62
selection of glasses in, 61 et seq.
Pressure, effects of, on intra-ocular
tension, 9
Primary ganglia, 709
Principal meridians, 1
point, 11
Prism, Cretes', 210
dioptry, 59, 201
holder, 210, 213
pile, 210, 211
Prisms, 53, 59
angle of deviation of, 59
refraction of, 59
degree of displacement by, 59
in muscular asthenopia, 223
800
INDEX.
Prisms, in myopia, 109
in ocular paralysis, 167
linear displacement by, 59
nomenclature of, 59, 201
properties of, 198 et seq.
Probe, author's, 302
Becker's conical, 299
Bowman's, 302
false passage by, 301
perforated, 302
Theobald's, 301, 302. 303
Weber's conical, 301
Williams', 301
Probes in dacryocystitis, 299 et seq.
Prothesis oculi, 768 et seq.
Protrusion of globe, 37, 141, 401, 436
Pseudo-glioma, 516, 585
Psychic blindness, 649, 654, 655, 656,
657, 703 (see Mental b. and Mind b.)
Pterygium, 368 et seq.
change of corneal curve by, 370
crassum, 368
following scleritis, 426
micro-organisms in, 369
pathology of, 369
removal of, before cataract ex-
traction, 487, 496
tenue, 368
treatment of, 369 et seq.
Ptosis, 289, 469
as a symptom of sympathetic dis-
ease, 287
congenital, 160
double, 164
nuclear, 160
due to lipomata, 287
in trachoma, 343
in tumors of orbit, 753
operations, 285 et seq.
partial, 357
single, 160
treatment of, 285 et seq.
Pulsation of retinal arteries, 598
vessels, 51
in glaucoma, 549, 554
Pulvinar, 146, 147, 641, 642, 644, 657,
715
Puncta lachrymalia, 247, 292, 293
eversion of, 294
stoppage of, 294
Punctum agendi, 89, 205
proximum, 17
remotum, 17
Puncture of cornea, 245, 246, 247
Pupil, 2, 35
consensual action of, 433
contraction of, 431, 433
in accommodation, 433
in convergence, 432, 433
diameter of, 432, 433
dilatation of, 431, 433
exclusion of, 442, 444, 451
extensive adhesions of, 450
in glaucoma, 549, 551, 559
in various diseases, 434 et seq.
occlusion of, 442, 444, 451, 465
Pupil, reflex action of, 433, 434, 435
seclusion of, 465
Pupillary centre (cerebral), 432
reaction in hemianopia, 433
reaction, 645
Pupiloscopy (see Shadow test)
Purkinje's images, 16, 472, 474
Pustular ophthalmia, 380
Pyoktanin, 234
Pyorrhea, 319
Quinine amblyopia, 700
Rabbit's conjunctiva transplantation
of, 290
Rachitic teeth, 483
Radiating visual fibres of Gratiolet,
644, 645, 646, 647, 715
Radii of curvature of glasses, 55
of surfaces of crystalline lens,
12
Radix descendens, 645, 646
Rami communicantes, 6
Range of accommodation, 17 et seq.
table showing, at different
ages, 18
Recurrent paralysis of ocular muscles,
158
Red nucleus, 146, 150, 658
Reduced eye of Listing, 12
Refraction, correction of, in muscular
asthenopia, 220, 221
Cuignet's test for, 76
errors of, 69 et seq.
Schmidt-Rimpler's test for, 71
Refractive errors, diagnosis of, 70 et
seq.
index (see Index of refraction)
Relative accommodations, 18 et seq.
Resorcin, 233
Respiratory centre, 150
Retina, 2, 5, 594 et seq.
anaemia of, 598
anaesthesia of, 216, 704
anomaly of vessels of, 49
blood-vessels of, 49
capillary aneurism of, 606
cerebral layers of, 595
cystoid degeneration of, 560, 632
detachment of, 630 et seq.
diagnosis of, 633
etiology of, 632
evacuation of fluid in, 111
in myopia, 93, 101, 110
pathogenesis of, 632
prognosis in, 634
treatment of, 634 et seq.
diseases of, 597 et seq.
epilepsy of, 598 et seq.
epithelial layer of, 595
functions of, 20
glioma of, 467, 514, 637 et seq.
hexagonal pigment epithelium of,
5, 595
lryperaemia of, 597
INDEX.
801
Ketina, hyperaesthesia of, 197, 204, 705
et seq.
hysterical, 706
ischaemia of, 600
laceration of, 631, 633
layers of, 594 et seq.
miliary aneurisms of, 610
minute anatomy of, 594 et seq.
oedema of, 606, 608
after blows, 612
Purkinje's phantom image of ves-
sels of, 32
sclerosis of, 626
serous effusion in, 611
structure of, 20
torpor of, 703, 704
visibility of, dependent on pig-
ment, 49
whitish opacity of, after blows, 695
Retinal arteries, spasm of, 598 et seq.
hemorrhage, 605 et seq.
in new-born children, 606, 608
phlebitis, 603
reflexes, 50
vessels, influence of cervical sym-
pathetic on, 598, 599
spasm of, 702
Retinitis, 610 et seq.
acute traumatic, 612
albescens punctata, 626
album inurica, 613 et seq
cause and development of, 615
diagnosis of, 615
induction of labor in, 621
in pregnancy, 615
pathology of, 617 et seq.
prognosis in, 616
statistics of, 616
treatment of, 621
apoplectica, 607 et seq.
centralis, 611
circum-papillaris, 662, 665
circumscripta, 625
due to oxaluria, 626
phosphorus poisoning, 626
etiology of, 612
glycosurica, 622
hemorrhagica, 607 et seq., 622
leucocythaemica, 622
pigmentosa, 579, 626 et seq.
diagnosis of, 628
field of vision of, 627
ophthalmoscopic picture in,
50, 627 et seq.
pathology of, 626 et seq.
prognosis in, 629
subjective symptoms, 627
treatment of, 629 •
plastica, 611
proliferans, 514, 612
purulenta, 612
simplex, 613
splenica, 606
symptoms of, 610 et seq.
syphilitica, 623 et seq.
prognosis in, 626
51
Retinitis syphilitica, treatment of, 625
visual disturbance in, 624
traumatica, 695
Retino-choroiditis, 535
Retinoscopy (see Shadow test)
Rheumatic paralysis of ocular muscles,
158, 163
Rheumatism, 66, 95, 235, 445, 449, 570,
606, 677, 681, 692, 725
Rickets, 483
Rods and cones, 20, 595
Rupture of choroid, 520
Saemisch's incision in hypopyum kera-
titis, 400
in ulcus rodens, 406
Sarcoma of choroid, 592, 593, 632, 639
of conjunctiva, 359
of cornea, 423
of iris, 452
Scalping of lids, 267
Scarification in purulent conjunctivi-
tis, 329, 330
in trachoma, 347, 352
Scarpa's nail, 302
Scheiner's test, 81 et seq.
Thomson's modification of, 70,
81 etseq., 121, 420
Schematic eye, 13
measurements of, 13
Schlemm's canal, 4, 6, 8
Sclera, 1, 3, 424 et seq.
anatomy of, 424
staphyloma of, 427
Scleral puncture in detachment of
retina, 635
ring in glaucoma, 551
Scleritis, 425 et seq.
circumscripta, 426
diffusa acuta, 426
Sclero-choroidal ring, 559, 560
Sclero-keratitis, 427
Sclerosis, cerebro-spinal, 66
disseminate, 67, 175
of cornea, 410
of lens, 479, 481, 485
Sclerotica (see Sclera)
Sclerotomy, 245, 416, 538
in glaucoma, 564 et seq.
posterior, 566, 567
Scotoma, 31, 609, 625, 629
absolute, 181
central, 604, 605, 606, 623, 667, 678,
679, 686, 689, 700
for color, 28, 89, 181, 622, 667,
678, 679, 681, 684, 686, 689,
700
in glaucoma, 555
in myopia, 110
negative, 31, 625
paracentral, in glaucoma, 555
peripheral, 678, 688
positive, 31, 625
ring-shaped, 625, 627, 629, 680, 684
scintillans, 702
temporary, 182
802
INDEX.
Scotomata in glaucoma, 549, 555, 559
Scrofula, 445, 483, 692, 725
Scrofulous teeth, 391
Seclusion of pupil, 465
Second principal focus, 11
sight, 63, 480
Secondary deviation, 139
Secretory angle, 8
Sella turcica, 145
Semilunar fold, 169
Sensitive nerves, effects of irritation
of, on intra-ocular tension and pu-
pil, 9
Sensory crossing, 148
tract, 148
Septicaemia, 729
Shadow test, 70, 76 et seq., 86, 103, 121
et seq.
Short ciliary arteries, 6, 573
Shot-silk appearance of retina, 610
Shrinking of tissues after trachoma, 357
Simple optical system, 11
Sinus, cavernous, 144, 145
circular, venous, 4
Sinuses, cerebral, 145
Sixth nerve, nucleus of, 155
Skiascopy (see Shadow test)
Skin diseases, 256
Skin flaps, 271 et seq.
grafts, 271 et seq.
transportations, 271
Snow blindness, 389, 704
Spasm of accommodation, 63, 68, 215
causes of, 64
diagnosis of, 65
in myopia, 104
methods of detecting, 210, 215
treatment of, 65
of muscles in muscular asthenopia,
225, 227
of ocular muscles, 173 et seq.
of orbicularis, 281
Speech area, 657
motor, 656
Sphenoidal fissure, 144, 145
Spherical glasses, 53
Sphincter iridis, 5
special nucleus for, 432
pupillae, 431
Spinal cord, amblyopia in concussion
of, 696
columns of, 146
decussation of fibres of, 148
disease, 162, 692
in ocular paralyses, 164
effect of irritation of, on intra-
ocular tension, 9
effect of section of, on intra-
ocular tension, 469
irritation of, 64
lesions, 688
meningitis, 434
Staphyloma, 406
intercalary, 427
of cornea, 411, 414
excision of, 417 et seq.
Staphyloma of cornea, treatment of,
416 et seq.
of sclera, 427, 467
treatment of, 428
pellucid um, 419
posterior, 97 et seq.
Stenopaic slit, 411, 420
spherical lens, 210
Stereoscope, practice with, after ten-
otomy, 191
Stimulating applications in muscular
asthenopia, 220
Stokes' lens, 118
Strabismus, 177 et seq.
alternating, 177
bilateral, 177
concomitans, 177
convergens, 177
classification as to refraction,
185
in hyperopia, 85
relation of abduction to adduc-
tion in, 182 et seq.
course of, 184 et seq.
deorsum-vergens, 177, 196
divergens, 177
in myopia, 186
operation for, 191
etiology of, 179
in myopia, 94
intermittent, 177, 196
method of operating for, 188 et seq.
method of measuring, 178, 179
monolateral, 177
operative treatment of, 187 et seq.
paralyticus, 177
periodic, 195
permanent, 177
phenomena of vision in, 183 etseq.
statistics, 182 et seq.
sursum-vergens, 177, 196
treatment of, 186
varieties of, 177
Stratum zonulare, 643, 644
Stretching of infra-trochlear nerve in
glaucoma, 566
Striped keratitis, 507
Strumous ophthalmia, 380
Strychnia in muscular asthenopia, 219
in ocular paralyses, 166
in spasm of accommodation, 65, 66
Stye, 253
Style, 302
Sub-conjunctival ecchymosis, 368
Sub-cortical lesions, 157
paralysis of ocular muscles, 164,165
Subjective sensations of light, 31
"Sublatio retinae, 630
Sub-retinal effusion in choroiditis, 580
Substantia nigra, 146, 148, 150
Sulcus orbito-palpebralis, 248
palpebro-malaris, 248
Sulphur as a disinfectant, 233
Sunstroke followed by neuro-retinitis,
672
Superior oblique, paralysis of, 196
INDEX.
803
Suppression of image, 137
in strabismus, 184
Supra-choroidea, 8, 559
Supratraction of choroid, 97 et seq.
Suspensory ligament of lens, 5, 430, 462
472, 512
Symblepharon, 288 et seq.
operation, Arlt's, 289
Knapp's, 290
Teale's, 290
skin flaps in, 290
skin grafts in, 290
transplantation of rabbit's con-
junctiva in, 290
treatment of, 289 et seq.
Sympathetic, cervical, effect of divi-
sion of, on pupil, 438
on secretion of aque-
ous, 9
effect of irritation of, on pupil,
436, 438
section or paresis of, on
intra-ocular tension, 469
effect of lesions of, 748
influence of, on intra-ocular
tension, 8, 9
on pupil, 9
on retinal vessels, 598, 599
irritation of, 64
lesions due to disease of, 436 et
seq.
symptoms of irritation of, 436
inflammation (see Sympathetic
ophthalmia)
irritation, 64, 531, 532, 533
ophthalmia, 531 et seq., 776
after ciliary wounds, 522
causes of, 531
ciliary nerve theory of, 536, 537
enucleation in, 538
evisceration in, 532, 540, 544
following cataract extraction,
505, 531
germ theory of, 536, 537
injections of corrosive subli-
mate into vitreous in, 541
irritative, type of, 532, 533
mode of transmission of, 536 et
seq.
pathology of, 535 et seq.
period of latency in, 532
prognosis in, 533
statistics of, 532
treatment of, 538 et seq.
Synchisis, 513
scintillans, 513
Syndesmitis, 311
Synechia, anterior, 405
posterior, 440, 441, 444, 449, 450
totalis, 465
Syphilis, 66, 67, 162, 235, 256, 285, 304,
378, 389, 390, 391, 392, 403, 445,
448, 483, 517, 570, 577, 579, 580,
583, 606, 611, 612, 623, 624, 625,
628, 667, 672, 676, 681, 683, 692,
715, 718, 725, 728, 749
Syphilis, as a cause of ocular paralyses,
163
sloughing of flaps in, 268
Syphilitic lesions of conjunctiva, 357,
358
teeth, 391
ulcerations, 259
Syringe, use of, in cataract extraction,
494, 500
Tabes dorsalis (see Locomotor ataxia)
Tactile bodies of Krause, 310
Tapetum, 574
Tarsitis, 255
Tarsoraphy, 278, 284
Tarsus, 247
inflammation of, 255 et seq.
subcutaneous division of, 357
Tattooing in conical cornea, 421
of cornea, 412, 413, 461
Tegmentum, 146, 148, 643
Teleangiectatic tumors, 260
treatment of, 260, 261
Tendo oculi, 293
Tenonitis, 158, 728
Tenon's capsule, 1, 168, 169, 424
advancement of, 190
Tenotomy, graduated, 224
improvement of vision after, 193
et seq.
in muscular asthenopia, 224 et seq.
in myopia, 227
in nystagmus, 175
in ocular paralyses, 167 et seq.
in strabismus, 188 et seq.
instruments for, 188 et seq.
mechanism of, 192 et seq.
partial, 224, 226, 227
ultimate results of, 192 et seq.
Tension, intra-ocular, 8, 9, 548
determination of, by manom-
eter, 9
effect of irritation of fifth
nerve on, 470, 557
effect of mydriatics on, 240
effect of section of cord on, 469
effect of section or paresis of
cervical sympathetic on, 469
examination of, 36
in cataract, 480
in detachment of retina, 633
in glaucoma, 548, 551, 554
methods of reducing, 246
nervous control over, 8, 9
periodic variation of, in glau-
coma, 549
Tensor choroideae, 463
Tentorium cerebelli, 145
Test letters, order of legibility of, 23
Test types, 22 et seq.
Burchardt's, 23
Green's, 22, 23
Little's, 117
Monoyer's, 22, 33
Pfliiger's, 23
Pray's, 121
804
INDEX.
Test types, Snellen's, 22, 23, 24, 33
Tests for simulated blindness, 720, 721
Thalamus opticus (see Optic thalamus)
Theobald's hooks for tenotomy, 189
Third nerve, decussation of fibres of
nuclei of, 154
nuclei of, 64, 152 et seq.
Thrombosis of cavernous sinus, 730,
732, 733, 744
of orbital veins, 730, 732, 733
of retinal vessels, £00 et seq.
Thymol, 233
Tobacco amblyopia, 28, 435, 678, 682
et seq.
Toric lenses, 59
Tortuous scleral vessels in glaucoma,
549, 551, 559
Trachoma, 337 et seq.
actual cautery in, 233, 350, 354,
356
acute, 340
cantholysis in, 354, 357
canthoplasty in, 356, 357
canthotomy in, 354
chronic, 340, 342
coccus, 340, 344
conditions favoring, 338 et seq.
contagiousness of, 339
corneal irregularity after, 357
curetting of cornea in, 354
duration of, 343
excision in, 350
expression in, 348 et seq.
follicular, 340, 341, 348
forceps, author's, 348, 349
Knapp's roller, 350
Prince's ring, 350
geographical limitations of, 338,
339
grattage in, 352 et seq.
hygienic conditions in, 350, 351
hypertrophic, 343
inoculation with blenorrhoeal pus
in, 354
isolated deposits of, 342
jequirity in, 354, 355
lymphoid infiltration in, 342
micro-organisms in, 339, 340, 344,
351
mixed, 342, 351
papillary, 340, 348
pathological anatomy of, 344 et seq.
peritomy in, 354
prophylaxis, 347
ptosis in, 343
racial susceptibility to, 339
sequelae of, 340, 343, 344, 357
Strong bichloride solutions in, 351
et seq.
subdivisions of, 337, 338, 340
treatment of, 347 et seq.
vegetable fungus in, 344, 345
Tractus opticus (see Optic tract)
Transverse axis, 1
Tremulous iris, 439
Trephining in conical cornea, 422
Trial cases, 54
varieties of, 57
frames, 58
glasses, 38
Trichiasis, 261, 262
after trachoma, 357
partial, 262, 264
treatment of, 262 et seq.
Trichinosis, 66
Trigeminus, motor root of, 146
Trochlearis nucleus, 153, 154
Tuber cinereum, 146, 641, 642, 644
Tubercle of choroid, 578, 588
of cornea, 423
of iris,' 443, 452
Tubercular disease of conjunctiva, 357,
358
Tuberculosis, 445, 589, 715, 718
Tumor of brain (see Brain tumor)
of cerebellum, 671, 675, 709
of choroid, 589 etseq.
diagnosis of, 591
prognosis in, 592
treatment of, 591
of iris, 451 et seq.
of optic nerve, 734, 736 et seq.
of orbit, 671, 733 et seq.
prognosis in, 743
recurrence of, 743
symptoms of, 737
treatment of, 738 et seq.
vascular, 735, 736, 743, 744,
745
Turkish bath, 242
in muscular asthenopia, 219
Tylosis, 253, 261
Tyrrell's hook in iridectomy, 412
Ulcers of cornea, 405 et seq.
Ulcus corneae serpens, 377, 395, 397 et
seq.
rodens, 377, 406
Unstriped fibres of Miiller, 251
Uraemic blindness, 616
Uterine disease, 84, 127, 208, 672, 677,
683, 689
Uvea, 6, 424, 430, 431, 573
Uveitis anterior, 407 et seq., 442, 466
maligna, 531
serosa, 442, 531, 533, 534, 575
Valve of Vieussens, 146
Varicella, 315
Variola, 315, 445
lesions of eye in, 365
Vascular growths in iris, 452
Velum medullare anticum, 146
Venae vorticosae, 573
Venous pulsation, 598
in glaucoma, 549, 554
Ventricle, third, 145, 146
Verbal deafness, 656
Vernal catarrh of conjunctiva, 362, 363
409
Vertical axis, 1
meridian, 1
INDEX.
805
Vertical muscular insufficiency, 216,
223, 229
Vessels of globe, 6
Vision, average, 21, 22
effect of illumination on, 23, 24
sudden obscurity of, in glaucoma,
551
Visual acuity, 21, 24, 37, 70
improved by glasses, 90
normal, 21
peripheral, 26
apparatus, scheme of central, 646
area, 647, 649
diagrams showing, 648
centres, 647, 653, 654
primary, 645
field (see Field of vision)
image, suppression of, 137, 184
line, 14, 178
memories, 649, 654, 714
path, anatomy of (see Hemiano-
pia)
purple, 32 et seq., 595
radiations, 644, 645, 646, 647, 715
rose, 32
Vitreous, 2, 512
artificial, 418, 769
blood-vessels in, 514
body (see Vitreous)
cholesterin in, 513
connective tissue in, 513
cysticercus in, 514, 518
degeneration of, in myopia, 101
detachment of, 513
fluid, 513
in choroiditis, 578, 581
foreign bodies in, 514, 518, 523 et
seq.
hemorrhage into, 513, 517, 520
humor (see Vitreous)
inflammation of (see Hyalitis)
Vitreous, membranes in, 514, 517, 520,
581
opacities, 513, 514, 515, 516
in choroiditis. 581
prolapse of, 515, 522
pus in, 513, 514, 517
Warts, 260
Watch-glass in purulent conjunctivi-
tis, 332
Wecker's scissors, 459, 492
in secondary cataract opera-
tions, 510
Wernicke's pupillary symptom, 708,
709, 713, 714
Wharton Jones' sliding flap, 268, 270
White precipitate ointment, 256
Whooping cough, 383, 600
Word blindness, 657
deafness, 656
Wounds and injuries of orbit, 759 et
seq.
of globe, 519 et seq.
complicated, 523 et seq.
of optic nerve, 760, 761
Xanthelasma, 256, 257
Xeroma, 356
Xerophthalmus, 361
Xerosis, 342, 344, 360 ei seq.
idiopathic, 361
Yellow ointment in blepharitis, 252,
253
oxide of mercury, 234
spot, 5
vascularity of, 48
Zona ophthalmique, 257
Zonula of Zinn, 5, 6, 43, 462, 472
INDEX OF AUTHORS.
Abadie. 75, 224, 352, 541, 566
Axlamuck, 635
Agnew, 84. 169, 173, 232, 296, 299, 303,
476, 505, 509
Alexander 392, 445
Alt, 370, 464, 468, 470, 532, 535, 536,561,
665, 669
v. Ammon. 270, 276, 281
Anagnostakis, 263, 266
Andrews, 500, 545, 736
Angelucci, 601, 603
Annuske, 671
Argyll-Robertson, 435, 688
Arlt, 95, 248, 264, 267, 289, 365, 386, 390,
397, 441, 443, 483, 534, 545, 635
Arnaut, 351
Aschmann, 760
Atkinson, 700
Aub, 695
Aubert, 21. 25, 32, 216
Ayers, 32, 33, 423
Badal, 80, 566
Bartholow, 766
Barwinkel, 469
Batten, 95
Baumann, 745
Baumgarten, 359
Bechterew, 155
Becker, 2, 16, 33, 48, 244, 299, 472, 511,
545, 546, 589, 591, 599, 765
Benedict, 673
Benson, 545
Berger, 657, 666, 711, 757
Berlin, 104, 131, 511, 524, 603, 612, 695,
703, 735, 739, 751, 754, 756, 758, 759
Bernhardt, 671
Bernheimer, 643
Berry, 555, 632
Berthold, 435
Bezold, 361
Bibber, 287
Birdsall, 155
Birnbacher, 560
Bjerrum, 31, 555
Bock, 513
Boe, 541
Boland, 697
Boll, 32
Bonnet, 224
Bouchut, 664
Bouillaud, 656
Bowman, 120, 299, 302, 371, 372, 373,
422 461, 474, 494, 508, 539, 635
Bradford, 525
Brailey, 487, 531, 535, 536, 576, 635
Bremer, 773
Brettauer, 488
Broca, 656, 657
Brugger, 386
Brunner, 700
Budge, 436
Bulkley, 765
Bull, C. S., 166. 256, 306, 358, 359, 362,
476, 502, 557, 568, 570, 572, 616, 637,
638, 666, 671, 709. 728, 736, 739
Bull, OleB., 623, 679
Buller, 332, 698
Bunge, 544, 545, 586, 595, 596, 651, 685
Burchardt, 23
Burke, 44
Burnett, 14, 30, 201, 230, 339, 371, 374,
664
Burow, 80
Carl Theodor, Herzog, 97, 100, 620,
Carmalt, 640
Canon duVillard, 745
Carter, 492, 761, 770
Cattell, 23, 24
Chamberlain, 244
Charcot, 645, 697, 701, 714
Cheatham, 355
Chibret, 339, 401, 402
Chisholm, 505, 672
Claude-Bernard, 438
Coccius, 16, 559
Cohn, 6, 22, 93, 103, 104, 107, 310, 344,
347, 366, 447, 535, 771
Collins, 163, 165, 166, 362, 439, 464, 466,
470, 473, 474, 559, 561, 562, 563, 570,
571
Conheim, 589, 608
Cooper, White, 366
Coppez, 355
Coppin, 636
Couper, 43
Courtaix, 705
Crede, 322, 775
Critchett, 169, 329, 335, 418, 422, 461,
532, 539, 562
Cross, 532, 540, 544, 770
Cuignet, 76, 86, 103, 121, 702
Culver, 12
Cunier, 224
Cusco, 44
Czermak, 384, 385, 560
808
INDEX OF AUTHORS.
Da Gama Pinto, 375, 407, 639
Dagunet, 259
Dalrymple, 736
Dana, 658, 675, 697
Davis, 51
De Beck, 358
Dechambre, 184
Decker, 385
Dejerine, 657
Delafield, 63-*
Dennett, 201, 202
Derby, H, 24, 94, 540, 629
Derby, R. H, 255, 335
De Schweinitz, 201, 403, 440, 604, 613,
623, 667, 700
Desmarres, 254, 460
Despaernet, 705
Deutschmann, 326, 535, 536, 537, 541,
545, 674, 675
Dianoux, 264
Dickinson, 615
Dieffenbach, 188
Dimmer, 51, 576
Dobrowlsky, 83, 86, 114
D'Oench, 545
Donaldson, 649
Donders, 9, 15, 17, 18, 19, 29, 30, F\
83, 84, 85, 90, 91, 93, 100, 102, lla,
115, 128, 134, 178, 179, 180, 184, 197,
250, 411, 472, 532, 536, 555
Dracoulides, 533
Dransart, 636
Duane, 219
Duchenne, 162
Dunglison, 367
Dunn, 705
Diirr, 104
Dyer, 25, 219, 220
Eales, 604
Earles, 615
Edes, 672
Edinger, 147, 151, 153, 154, 155, 645
Edmunds, 671, 673, 674
Elsching, 588
Ely, 705
Emmert, 64, 241, 375, 389, 409, 765
Eperon, 196
Erb, 672
Erichsen, 697
Erisman, 107
Etters, 164
Eulenberg, 438
Eversbusch, 431, 658
Exner, 657
Ferguson, 123
Ferrier, 157. 165, 656
Fick, 26, 112, 234
Fieuzal, 664
Filehne, 684
Fischer, 601
Fisher, 605
Fitzgerald, 766
Flarer, 264
Flemming, 372, 375
Flint, 334
Fontan, 162, 408
Forel, 150
Forster, 25, 26, 31, 157, 494, 580, 625,
683, 711
Foster, F. P., 307
Foster, M., 34, 645
Franke, 447
Frankel, 589
Freyer, 330
Fries, 698
Frost, 534
Fuchs, 93, 104, 105, 164, 254, 370, 385,
431, 465, 557, 561, 589, 590, 591, 592,
684, 776
Galezowski, 162, 349, 506, 622, 636,
665, 688, 704, 705
Galleuga, 557
Garlick, 671
Gamier, 736
Gaule, 403
Gauss, 12
Gayet, 234, 264, 505
Geissler, 488
Genth, 667
Gerhardt, 437
Gerlach, 6, 169, 472
German, 345
Gessner, 749
Gibson, 352
Gifford, 536, 537, 558, 605
Giraud-Teulon, 44, 58, 74
Goldzieher, 164, 536
Gowers, 160, 161, 165, 175, 281, 282
284, 598, 603, 622, 645, 664, 667, 674'
697, 699, 712, 766
Gradle, 210
Graefe, v., 2, 19, 80, 95, 97, 139, 154
158, 159, 160, 184, 187, 189, 192, 202'
205, 213, 214, 224, 225, 240, 243, 263,
297, 307, 333, 337, 360, 382, 383, 400
410, 422, 423, 461, 470, 488, 490, 495'
505, 548, 555, 562, 564, 565, 566, 567'
569, 572, 600, 601, 623, 635, 660 674'
679, 695, 764, 767
Graefe, Alfred, 175, 182, 184, 196 224
362, 540, 544, 545, 586, 600, 635, 720 '
Grahamer, 775
Graser, 240
Gratiolet, 644, 645
Gray, 692
Green, John, 22, 33, 57, 59, 118 192
240, 265, 266, 267, 507, 510
Griffith, 593, 730
Groenow, 711
Grossmann, 328
Grosz, 636
Gruening, 191, 236, 287, 354, 525, 566
569, 570, 665, 700, 737
Grimhagen, 431, 557
Gudden, 154, 643, 644, 645, 646 647
652, 690, 715 '
Guerin, 224
Gunn, 545
Guthrie, 745
INDEX OF AUTHORS.
809
Haab, 376, 383, 395, 589, 646
Haas, 696
Hall, 154
Haltenhoff, 315
Hamilton, 176
Hancock, 568
Hansen, 195
Hansen-Grut, 376, 389, 409
Harlan, 196, 290, 321, 702 721 744
Hasner, 385, 386
Haussmann, 321, 775
Hay, 421
Heil, 473
Heisrath, 352
Helmholtz, 16, 20, 21, 39, 112 378 472
Henle, 145, 146, 148, 309
Henschen, 647, 648, 649, 651, 652 657
675, 708, 709, 711, 713, 714, 718
Hensen, 438
Herter, 672, 703
Hess, 385
Hewitt, 758
Heyl, 330
Heymann, 765
Higgins, 403, 635
Hilbert, 255
Hildebrand, 528
Hippel, 94, 104, 348, 351, 352, 403, 422,
557
Hirschberg, 29, 80, 178, 179, 241, 321
360, 365, 422, 447, 518, 525, 528, 538,
539, 605, 606, 616, 622, 635, 638, 672,
697, 699, 773
Hirshfeld, 337
Hjort, 16
Hock, 679
Holden, 689
Holder, 695, 751, 758
Holmes, 737
Holmgren, 29
Holtz, 672
Holtze, 240
Horner, 28, 93, 110, 169, 287, 321, 361,
363, 364, 366, 374, 377, 380, 383, 384,
390, 392, 395, 397, 436, 438, 450, 452,
469, 470, 491, 632, 700, 748
Horsley, 157, 164
Horstmann, 2
Hotz, 265, 266, 267, 287
Howard, 163
Howe, 339, 621, 775
Hubbell, 156
Hughes, 587
Hulke, 666
Hun, 648, 718
Hunnius, 165
Hutchinson, 66, 257, 390, 392, 394, 437,
438, 443, 579, 583, 616, 683, 705
Iodko, 700
Isler. 185
Iwanoff, 83, 380, 632, 665
Jaarsma, 241
Jackson, E., 43, 201, 612
Jackson, Hughlings, 664, 671, 673
Jacobson, 837, 560
Jaeger, 2, 96, 97, 99, 580, 582, 613, 690
Jaesche, 264
Janeway, 176, 283, 766, 767
Jany, 406
Jatzow, 710
Javal, 55, 56, 70, 74, 103, 104, 114, 116
121, 123, 124, 126, 184, 192, 357, 378!
419, 557, 562
Jeffries, B. Joy, 30, 259
Jeffries, J. A., 165
Johnson, G. Lindsay, 594
Johnson, W. B., 736, 737
Jones, Wharton, 268, 270
Juler, 335
Kahler, 152, 153
Kalish, 488
Kansocki, 343
Katz, 773, 776
Kendall, 366
Kerschbaumer, 502
Kipp, 388, 389, 518
Klein, 293, 372
Knapp, H., 58, 113, 190, 192, 196, £54,
260, 273, 290, 350, 401, 418, 442 476
501, 502, 509, 525, 557, 581, 589, 590'
600, 603, 638, 684, 695, 700, 710, 728
730, 737, 739, 740, 750, 758
Knapp, P. C, 697
Knies, 8, 152, 153, 154, 157, 164, 361
362, 403, 442, 536, 560, 561, 652, 654
Kohler, 744, 747
Roller, 235, 635
Kolliker, 472, 595
Kollock, 361
Konig, 29
Konigshofer, 24
Krause, 542, 651
Krenchel, 195
Kubli, 362
Kiihne, 32, 33
Kuhnt, 595, 673
Kuschbert, 361
Laker, 558, 689
Landesberg, 386, 733, 773
Landolt, 12, 13, 14, 18, 27, 83, 91, 103
178, 179, 182, 184, 202, 203, 225, 241,
330, 701
Lang, 717, 770
Lange, 470
Langley, 30
Laqueur, 125, 557, 563
Laurence, 307
Lawford, 671
Lawson, 278, 532, 538, 539
Leber, 4, 6, 48, 361, 374, 384, 395, 535
536, 541, 542, 561, 622, 626, 627, 632'
633, 634, 666, 673, 674, 675, 679, 684
686, 687, 698, 699, 701, 705, 736
Lebrun, 495
Leconte, 32
Leeser, 434
Lennox, 472 |
Leube, 162
810
INDEX OF AUTHORS.
Liddell, 737, 738
Liebreich, 44, 105, 495, 597, 598, 602,
622, 631, 684
Lippincott, 494, 500
Listing, 12
Litter, 699
Loiseau, 76
Loring, 39, 43, 45, 48, 50, 51, 57, 96,
103, 104, 105, 168, 227, 360, 459, 510,
511, 597, 601, 604, 605, 608, 613, 621,
622, 623, 673, 701
Luys, 644, 652
Mackenzie, 536, 548, 736, 750
Maddox, 202, 210, 212
Magnus, 155, 432, 436, 601, 602, 661,
664, 718, 771, 773, 776
Manby, 766
Mandelstamm, 698
Manz, 597, 623, 673
Marchand, 712
Marpman, 234
Mascart, 80
Mathewson, 259, 666, 671
Mathiessen, 113
Mauthner, 17, 23, 56, 63, 158, 159, 161,
163, 392, 532, 538, 605, 708
McHardy, 25
McKeown, 494, 525
Meigs, 608, 621
Meissner, 134
Merkle, 2, 132, 471, 755
Meyer, Adolf, 576
Meyer, E., 271, 540
Meynert, 150, 643, 644, 645, 646, 652,
657, 715
Michel, 156, 166, 340, 344, 469, 550, 577,
578, 588, 589, 590, 598, 603, 605, 620,
626, 652, 684, 689, 703, 717, 736, 737
Miles, 534
Mills, 157, 165
Minor, 388, 684
Mitchell, Weir, 117, 438, 709
Mittendorf, 241, 248
Mobius, 437, 438
Monoyer, 22, 33
Moore, 315, 621, 701, 702
Mooren, 66, 377, 383, 389, 392, 406, 495,
557, 626, 672
Morton, 474
Motais, 168, 169, 546
Mules, 418, 482, 545, 603, 605, 769
Miiller, 436, 536, 559, 561, 576, 724, 748
Munk, 165, 649, 703
Muttermilch, 344, 345
Nagel, 2, 18, 23, 55, 56, 65, 80, 82, 97,
202, 203, 433, 692
Natanson, 562
Naumhoff, 606, 608
Neese 52s
Neisser, 322, 361, 376, 445
Nettleship, 43, 48, 49, 77, 80, 333, 335,
336, 410, 540, 545, 570, 579, 580, 583,
602, 604, 605, 651, £66, 680, 684, 685,
687, 691
Neumann, 334
Nicati, 438, 464, 469
Niedeke, 536
Nieden, 217, 744, 747, 749
Noiszewski, 344
Nordenson, 633
Norris, 237, 710, 712, 713
Nothnagel, 159
Nuel, 385
Obersteiner, 147, 155
Obriga, 165
Oeller, 673
Ogle, 438
Oglesby, 174, 175
Ohlemann, 538
Oliver, 28, 121, 709, 713, 714
Oppenheim, 697
Oppenheimer, 382, 776
Pagenstecher, 561, 664, 667, 668, 730
Panas, 234, 500, 501, 531
Parinaud, 664, 673, 674
Passavant, 449
Pell, 164
Perlia, 152, 153, 154, 161
Perrin, 80
Peters, 708
Peterson, 166
Pflilger, 23, 160, 570
Pick, 152, 153
Picque, 675
Plehn, 80, 81
Pollak, 479
Poncet, 596, 622, 632, 690
Pooley, 494, 526, 534, 621, 664, 736
Pope, 265
Porteau, 438
Porterfield, 81
Posch, 24
Pray, 121
Prentice, 59, 201, 203
Prevost, 165
Prince, 169, 173, 350, 370
Prout, 254, 260, 362
Purkinje, 32, 472, 474
Purtscher, 659
Querenghi, 432, 433, 464
Raab, 639
Raehlmann, 175, 341, 343, 344, 362, 421,
434
Randall, 83, 90
Randolph, 536, 537
Ranney, 209
Recklinghausen, 765
Reich, 341, 344, 664, 671, 697
Reid, 344
Reinhardt, 647
Reiss, 700
Reuss, 2
Reute, 39
Reverdin, 271
Richet, 271
INDEX OF AUTHORS.
811
Ring, 505
Risley, 487
Rivington, 744
Robin, 174
Rockwell, 767
Rohmer, 541
Rood, 32, 431
Roosa, 57, 85, 252, 355, 452, 700
Ross, 165, 436
Rossander, 532
Ruppell, 447
Rushmore, 255
Ryerson, 370
Saemisch, 264, 377, 395, 400, 406, 409,
601, 602
Salzer, 651
Salzmann, 26
Samelsohn, 367, 651, 678, 701
Sands, 601
Sanson, 474
Sargent, Elizabeth, 606
Sattler, 340, 344, 348, 356, 376, 406, 573,
576, 744, 745, 746, 747, 765, 766
Savage, 212
Schaefer, 157, 164, 165
Schapringer, 748
Scheiner, 70, 81, 121, 420
Schell, 182, 183, 287
Schiess, 540, 544. 545, 636, 639, 737
Schiess-Gemuseus, 474, 532, 537, 736
Schiff, 175
Schirk, 25
Schirmer, 85, 252
Schlaefke, 744
Schmeichler, 435
Schmidt, 604, 615
Schmidt-Rimpler, 71, 76, 337, 406, 651,
673, 708, 773
Schnabel, 96, 443, 514
Schobl, 372
Schoeler, 8, 9, 163, 321, 636, 637
Schoen, 51, 374, 560, 562, 574
Schueller, 51
Schulten, 8
Schultze, 20
Schwalbe, 4, 250, 372, 431, 512, 604,
650, 673, 724
Schweigger, 26, 84, 181, 182, 183, 185,
194 197, 447, 501, 503, 505, 532, 541,
543, 555, 556, 561, 566, 601, 653, 711,
718
Scott, 158
Seabrook, 488
Seeligmuller, 438
Seggel, 23, 24, 91, 94, 160, 440, 679, 759
Segger, 182
Seggert, 22
Seguin, 672, 709, 715, 716, 718
Seidelmann, 773
Sichel, 635, 736
Siemerling, 152, 155
Silex, 597
Smith, Priestley, 25, 26, 63, 179, 216,
"38 47° 477 479, 560, 561, 562, 567,
568' 571,' 603,' 604, 605, 662, 666, 668
Snellen, 21, 22, 33, 80, 254, 260, 265
267, 290, 535, 561, 562, 572, 721, 765
Soemmering, 146, 150
Sous, 80
SpauldiDg, 534, 535, 730
Spitzka, 147, 154, 155
Starr, 147, 148, 149, 152, 153, 157, 161,
438, 654, 657, 701, 702, 703, 718
Steffan, 672
Stellwag, 338, 380, 410, 673, 764, 765
Stevens, 25, 172, 210, 213, 216, 218, 224,
226, 229, 330
Stewart, 216
Stilling, 8, 30, 92, 94, 95, 99, 301, 401,
558, 645, 650, 672, 679, 721
St. John, 174, 528
Stolte, 772
Stdlting, 560
Story, 51, 766
Straub, 160, 736
Strawbridge, 423, 737
Streatfield, 265, 449
Strieker, 20
Stromeyer, 395
Strumpel, 697
Sturm, 113
Sulzer, 571
Swanzy, 165, 264, 435, 718
Symons, 392
Sziklai, 242
Tangeman, 513
Tay, 583
Teale, 290
Terson, 452
Theobald, 189, 301, 302, 303, 475, 699
Thiersch, 271, 272, 290
Thomson, 30, 70, 81, 82, 121, 420
Trousseau, 510
True, 339
Tscherning, 92, 93, 103
Tweedy, 317
Tyrmann, 666, 757
Tyrrell, 329
Uhthoff, 159, 161, 651, 679, 682, 685,
687, 773
Ulrich, 86, 182, 183, 186, 667
Van Aurep, 243
Van Duyse, 506
Vetsch, 363, 638
Volkers, 438
Vossius, 343, 372, 651, 665, 672, 678, 685,
730, 736, 737
Wadsworth, 39, 182, 615, 616, 639
Wagner, 644
Walter, 632
Walton, 656
Warlomont, 76
Weber, 169, 301, 560, 561, 567
Webster, 531, 535, 545
Wecker, 169, 173, 190, 224, 264, 287,
354, 355, 413, 492, 501, 510, 541, 565,
635, 693
812
INDEX OF AUTHORS.
Wedl, 513, 559
Weeks, 315, 323, 617, 620, 622, 682
Weigert, 334
Weiss, 84, 97, 98, 99, 102, 589
Wells, 736
Wernicke, 148, 164, 165, 665, 703, 708
White, 604, 766
Wicherkiewitz, 570
Widmark, 355, 704
Wiethe, 709
Wilbrand, 182, 216, 438, 704, 708, 709,
710, 711, 712, 713, 714, 717, 718
Wilde, 163, 165, 166
Willemer, 735, 737
Williams, E., 301, 399, 401, 529, 534
Williams, H. W., 174, 301
Williams, R., 707
Wilson, 328
Woinow, 30, 178, 432
Wolfe, 271, 274, 635, 636
Wolfheim, 736
Wolfring, 337
Wreden, 732, 733
Young, Thomas, 118
Zehender, 141, 698, 736
Ziegler, 589, 699
Zimmerman, 691
NOYES ON THE EYE.
PLATE IV.
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NOYES ON THE EYE.
PLATE V.
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NOYES ON THE EYE.
PLATE VI.
NOYES ON THE EYE
PLATE VI
DESCRIPTION" OF OHROMO-LITHOGEAPHS.
Fig. 1.—Normal fundus oculi: moderate degree of pigment. Jaeger.
Fig. 2.—Atrophy of optic nerve and retina. Jaeger, PI. x., fig. 50. Man,
25. No perception of light; cause obscure. Had frequent and severe head-
aches, repeated attacks of intermittent fever. Blindness within nine months.
Disc slightly excavated, color grayish-green, texture somewhat opaque. Reti-
nal arteries very small, with thickened walls, veins disproportionately large,
and both they and the arteries bordered with white lines of connective tissue.
Fig. 3.—Neuro-retinitis apoplectica. Jaeger, PI. xiv., fig. 65. Man, 53.
V = perception of light. Trouble of sight gradually coming on for months,
grew suddenly worse within a week, just recovered from delirium tremens.
Nerve deeply red, oedematous, outline obliterated. Arteries very small,
thready; veins flexuous, large, their tortuosity often in the vertical plane, as
indicated by the darker and fainter portions of the curves; numerous hemor-
rhages in streaks proceeding from capillaries and small vessels. Retina oede-
matous, especially near the disc.
Fig. 4.—Retinitis albuminurica. Man, 19. Nerve a little flushed, but
practically normal. The region of the macula surrounded by numerous bril-
liant, glistening, white spots arranged in a somewhat radiating manner.
Among them a few pigment specks. In the vicinity are other similar white
spots less distinct. (Amount of albumin very large, was dropsical.) Retina
otherwise normal, vessels normal.
Fig. 5.—Myopia, with congenital excavation of optic nerve, etc. Jaeger, PI.
xxvii., fig. 119. Man, 21. Myopia—\. Depth of excavation not given, but it
covers the entire surface of the nerve and the vessels are pushed to the nasal
side. The pigment epithelium has disappeared from the surface of the choroid
over the lower part of the fundus, exposing its vessels. Ten years later it is
said no other change had occurred than the removal of the pigment epithelium
over the whole fundus; vision continued nearly normal. The case presents
all the signs of glaucomatous excavation in a myopic eye. No mention is
made whether arteries would pulsate under light pressure.
Yia. 6.—Atrophy of optic nerve. Jaeger, PI. x., fig. 51. Woman, 46. Ery-
sipelas of face five weeks previous, severely affecting the lids and orbitai tis-
sues. V=0; the eye normal. Atrophy succeeding neuro-retinitis. Nerve
opaque, retinal arteries either obliterated or reduced.
Yia. 7.—Neuro-retinitis serosa. Jaeger, PI. xiii., fig. 62. Woman, 27.. Loss
of sight following childbirth; suffered extremely from headache, health very
poor. History does not indicate cerebral tumor. Other eye has similar con-
dition in less degree; nerve swollen, deeply red, infiltrated, outline abolished;
optic fibres conspicuous as radiating lines. Arteries small; veins engorged,
very tortuous.
814 DESCRIPTION OF CHROMO-LITHOCRAPHS.
Fig. 8.—Papillitis with both serous and plastic infiltration of the nerve.
Swelling very marked, edge blurred and diameter increased; arteries very
tortuous, marked by infiltration; veins swollen, very dark. Lesion confined
chiefly to nerve, sometimes called "choked disc." Condition found in cere-
bral tumor.
Fig. 9.—Papillitis with more intense plastic infiltration than in Fig. 8,
attended by hemorrhages in both nerve and retina. Striation of nerve
strongly pronounced.
Fig. 10.—White atrophy of optic nerve following meningitis. Liebreich.
Female, 19. Nerve white, opaque, border sharply defined and exhibiting a
distinct scleral ring, shallow excavation. Arteries small; veins broad, not
tortuous, bordered for short distance from disc with connective tissue.
Fig. 11.—Gray atrophy of optic nerve following tabes dorsalis. Liebreich.
Man, 40. Absolute blindness. Nerve grayish, slightly excavated, border
sharply defined, tissue opaque, lamina cribrosa distinct; arteries small; veins
broad. (Atrophy of nerve with spinal-cord lesion does not invariably present
the gray color of this picture.)
Fig. 12.—Atrophy of optic nerve and retina. Jaeger, PL ix., fig. 48. Fe-
male, 46. Became blind after successive childbirths, each attended by several
hemorrhages. For six years had the usual appearances of simple white atro-
phy (see Fig. 10), but after an illness, which seems to have been meningitis,
further degeneration took place in the optic nerves, giving rise to its peculiar
hue of gray yellow-green, with dense opacity and slight excavation. Vessels
much attenuated.
Fig. 13.—Colloid deposits upon optic nerve and on the adjacent retina.
Lawson, Trans. Oph. Soc. United Kingdom, vol. hi., PI. viii., fig. 1. Man, 23.
Had syphilitic choroido-retinitis with pigment patches at periphery; retina
hazy, floating bodies in vitreous. The masses consist of white, translucent,
beaded-like bodies heaped together to the height of 2.5 D. On the nerve
they are less individualized and of duller hue. Spangles of cholesterin occur
on the surface. There has been neuritis. (See p. 563, where a similar deposit
is referred to after neuro-retinitis.)
Fig. 14.—Colloid deposits in region of macula. Nettleship, Trans. Oph.
Soc. United Kingdom, vol. iv., PI. ii., fig. 2. Female, 41. V=ffl Hm. 5D.
Had no power of accommodation, spots grayish yellow, some better defined
than others, none sharply cut. " The most defined ones are surrounded by a
shaded gray ring, such as might be produced by a slightly prominent nodule."
This statement describes the true growth and character of these formations.
Compare Fig. 194, p. 526.
Fig. 15.—Tubercles of choroid and neuritis optica. Male. Trans. Oph.
Soc. United Kingdom, vol. iv., p. 160. Girl, 10. Acute miliary tuberculosis,
and double optic neuritis. Course of disease very rapid, viz., 14 days from
apparent beginning of illness. Some 8 to 10 nodules present, yellowish-white
at centre, shading into color of normal choroid, in which they were imbed-
ded ; three of them beneath retinal vessels. At autopsy abundant tubercles
in lungs, kidneys, liver, and spleen, and scattered in the vessels in the Sylvian
fissure. Attempt to discover bacilli in the choroidal tubercles did not suc-
ceed, while giant cells and patches of retrograde tubercle were easily de-
tected. The eye lesions resemble closely the early stage of choroiditis dis-
seminata, and are to be certainly distinguished only by the general symptoms
of the case.
DESCRIPTION OF CHROMO-LITHOGRAPHS. 81.")
Fig. 16.—Syphilitic retinitis with hemorrhages and new blood-vessels in
the vitreous. Nettleship, Trans. Oph. Soc. United King., vol. iv., p. 150.
Had chancre a year previously and secondary symptoms. Had iritis, hyalitis,
hemorrhage into retina both at periphery and about macula. From edge of
nerve, which was pale and hazy, a flat, transparent, vascular membrane grew
out into the vitreous, its vessels looped and numerous, and one large one
formed its anterior free border. At a later time more vessels developed.
After a year, the vitreous became clear, the hemorrhages were absorbed, the
vascular membrane remained. V = I2n°T.
Fig. 17.—Retinitis (after possible embolism). Jaeger, PL xv., fig. 70. Man,
32. Had had some rheumatic pains for six months. Had insufficiency of the
mitral valve and slight contraction of the left auriculo-ventricular opening.
Five days before examination, at 9 A.M., the left eye became suddenly and
completely blind as by a black cloud coming over it, without pain or other
symptoms. There is no perception of light. Other eye normal. A faint
grayish haze overspreads the optic nerve and the surrounding retina for
a distance beyond the macula, it follows the principal vessels going up
and down, but it gradually fades out toward the periphery. All the blood-
vessels are pervious, but reduced in size—it is not stated whether by pres-
Bure the blood current could easily be stopped—at the macula a minute red
spot said to be a hemorrhage (?). The periphery of the optic nerve very red,
at its centre a physiological excavation. Two and a half months later there
was complete atrophy of the nerve, retinal vessels still more reduced, and
bordered by connective tissue, giving the appearance seen in Fig. 6. The
diffused opacity had disappeared. The suddenness of the attack, the instant
and permanent blindness, the kind of diffused opacity, the minute red spots.
near, if not in the fovea centralis (as is probable), and the consecutive atrophy
suggest embolism, and to this the signs of heart disease add further proba-
bility. The partial continuance of the retinal circulation and the absence of
any unusual co-ordinate vessels make it probable that the stoppage of the
arteria centralis was incomplete, although sufficient to destroy the function
of the retina. On this subject see p. 548 and Fig. 202.
Fig. 18.—Case of epithelioma, which, beginning upon the lower lid, after
many years invaded the conjunctiva bulbi and the cornea, and compelled
evisceration of the orbit. The ulceration of the skin has been replaced by
cicatricial tissue, the eyelids shrunken and adherent to the globe, and the new
growth covers the outer and lower part of the cornea, as an exuberant granu-
lating mass.—Noyes.
Fig. 19.—Extra papillary coloboma. Dr. G. L. Johnson, Archiv f. Augen-
heilkunde, XXL, 291, 1890. This picture represents what by some is called
coloboma at the macula, and is one of a series of plates of cases which are in-
terpreted to be congenital defects and not illustrations of choroido-retinitis.
In his description, Dr. J. speaks of the white annular patch subdivided by
lines of pigment which concentrate upon a central red mass of choroidal
vessels and pigment, which he likens to a nsevus of the skin. The visual
field excessively small, viz., 30° X 23° V = ?%. No central scotoma. The
sclera is exposed, and its excessive whiteness is thought by Dr. J. to dis-
tinguish cases of coloboma from atrophy due to choroiditis, in which he says
a yellowish tinge will be found. He also claims that the deposition of pig-
ment above rather than below the retinal vessels is distinctive. The paper
is worthy of study, and if one does not concur fully in the interpretation of
the cases, the subject is ably handled. The picture from this point of view
has peculiar interest. See Loring, "The Ophthalmoscope," Vol. I., p. 95.
Most observers would class the cases as instances of choroiditis congenita.
816 DESCRIPTION OF CHROMO-LITHOGRAPHS.
Figs. 20, 21, 22, 24, 25, from Dr. G. Lindslay Johnson, Archiv fiir Ophthal.,
XXL, p. 1, 1892, represent various appearances found at the macula lutea in
normal eyes. Fig. 20 is the usual picture—not intending to mean that the
fine radiating red streaks around the fovea are blood-vessels, but the dark
coloration often present. Fig. 21 presents an annular halo of reflection seen
most perfectly by the inverted image, but also visible in the upright
method. It flickers and changes its breadth as the light falls in different
directions. The yellowish dots grouped in and around the fovea are quite
common, do not affect vision, and are probably colloid. Fig. 22 is an illus-
tration of an extreme degree of surface reflection from a large region near
the macula. Such a picture is found in dark eyes and in young subjects.
The evanescent glittering quality of the appearance cannot be perfectly
rendered, but is fairly suggested. Again the same yellow dots appear at the
fovea, which is deep red rather than yellow. Note the shading along the
retinal arteries as evidence of the unusual amount of connective tissue.
Fig. 24 is meant to show an elongated reflex proceeding from the fovea, and
which will change both length and direction as the point of view varies.
Fig. 25 considerably resembles Fig. 21, but is far less frequent. The ring is
to be interpreted in both figures as being a reflex from an annular elevation
of the retina. The crescent at the fovea is not an uncommon type of reflex.
(See p. 47.)
Fig. 23 —Neuro-retinitis from thrombosis.—Noyes. The picture was
drawn about a week after the onset of sudden blindness. The nerve is
swollen, infiltrated, marked by a hemorrhage, the arteries slender, the veins
turgid. Exudation, which at the outset covered most of the central part of
the fundus, appears now in masses between the nerve and the macula, and
follows as well as conceals the vessels. It also extends on the nasal side in
small patches and in a general infiltration. The fovea is cherry red. The
distinction from embolism is in the continuance of a feeble circulation in-
stead of its entire suspension, in the absence of the white infiltration sur-
rounding the fovea on all sides, and in the papillitis and hemorrhages.
Central vision was at first almost abolished. When the drawing was made
V = fingers at 3 feet. Visual field at the firs^ barely included the macula
and comprised a narrow ellipse reaching out on the temporal side to about
40°. This increased about 10° in all directions after the lapse of a week.
Fig. 26.—Vaccinia of the eyelids, girl five years old, in the fifth day of the
eye affection and ten days after vaccination on the arm. In a week the
crusts had fallen, the ulceration healed, the lid normal. At no time was
the cornea implicated, and the conjunctiva but slightly. The sore on the
arm was very large and phlegmonous.—Noyes. (See Zimmermann, Archiv
fiir Ophthalmology, XXL, p. 215, 1890.)
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