
<oai_dc:dc xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
  <dc:title>Hemodynamic Alterations in Hypertensive Patients Due to Chlorothiazide</dc:title>
  <dc:subject>Hypertension</dc:subject>
  <dc:subject>Antihypertensive Agents</dc:subject>
  <dc:subject>Chlorothiazide</dc:subject>
  <dc:subject>Hemodynamics</dc:subject>
  <dc:description>In this article, Freis&apos;s team reported that chlorothiazide--which reduces extracellular blood volume--appeared to also reduce cardiac output, arterial pressure, and pressure in the right atrium, suggesting that the diuretic imposed a wide antihypertensive effect.. NOTE: Scanned item is a reprint. Original article is on pp. 1261-1263.</dc:description>
  <dc:publisher>Massachusetts Medical Society, 23 June 1960</dc:publisher>
  <dc:publisher>The Massachusetts Medical Society, 23 June 1960</dc:publisher>
  <dc:contributor>New England Journal of Medicine</dc:contributor>
  <dc:contributor>Frohlich, Edward D.</dc:contributor>
  <dc:contributor>Schnaper, Harold W.</dc:contributor>
  <dc:contributor>Wilson, Ilse M.</dc:contributor>
  <dc:contributor>Freis, Edward D.</dc:contributor>
  <dc:type>Articles</dc:type>
  <dc:format>Archival Materials</dc:format>
  <dc:format>Text</dc:format>
  <dc:format>3 pages</dc:format>
  <dc:identifier>nlm:nlmuid-101584929X87-doc</dc:identifier>
  <dc:identifier>101584929X87</dc:identifier>
  <dc:identifier>http://resource.nlm.nih.gov/101584929X87</dc:identifier>
  <dc:identifier>Profiles ID: XFBBGF</dc:identifier>
  <dc:language>English</dc:language>
  <dc:relation>Profiles in Science</dc:relation>
</oai_dc:dc>